PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15208781-4	2004	We show that the risk allele, which is present in approximately 17% of white individuals from the general population and in approximately 28% of white individuals with RA, disrupts the P1 proline-rich motif that is important for interaction with Csk, potentially altering these proteins" normal function as negative regulators of T-cell activation.	Proline	188-195	C-terminal Src kinase	Homo sapiens	246-249