PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
1437149-1	1992	Transcription factor AP-1 is constituted by the various products of the fos and jun proto-oncogene family members, which associate as dimers to bind with variable efficiency to 12-O-tetradecanoyl phorbol 13-acetate (TPA)-responsive promoter elements (TREs).	Tetradecanoylphorbol Acetate	177-214	Jun proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	0-25	
1437149-1	1992	Transcription factor AP-1 is constituted by the various products of the fos and jun proto-oncogene family members, which associate as dimers to bind with variable efficiency to 12-O-tetradecanoyl phorbol 13-acetate (TPA)-responsive promoter elements (TREs).	Tetradecanoylphorbol Acetate	177-214	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	72-75	
1437149-1	1992	Transcription factor AP-1 is constituted by the various products of the fos and jun proto-oncogene family members, which associate as dimers to bind with variable efficiency to 12-O-tetradecanoyl phorbol 13-acetate (TPA)-responsive promoter elements (TREs).	Tetradecanoylphorbol Acetate	216-219	Jun proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	0-25	
1437149-1	1992	Transcription factor AP-1 is constituted by the various products of the fos and jun proto-oncogene family members, which associate as dimers to bind with variable efficiency to 12-O-tetradecanoyl phorbol 13-acetate (TPA)-responsive promoter elements (TREs).	Tetradecanoylphorbol Acetate	216-219	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	72-75	
1437149-5	1992	IP-1 activity is blocked by stimulation of the protein kinase C (PKC) signal transduction pathway, achieved by treating HeLa cells with phorbol esters or with a diacylglycerol analog.	Phorbol Esters	136-150	inhibitor of nuclear factor kappa B kinase regulatory subunit gamma	Homo sapiens	0-4	
1437149-5	1992	IP-1 activity is blocked by stimulation of the protein kinase C (PKC) signal transduction pathway, achieved by treating HeLa cells with phorbol esters or with a diacylglycerol analog.	Diglycerides	161-175	inhibitor of nuclear factor kappa B kinase regulatory subunit gamma	Homo sapiens	0-4	
1437149-6	1992	We observed an increase in AP-1-DNA binding after treatment of the cells with either the calcium ionophore A-23187 or dibutyryl cAMP; this could be ascribed to inhibition of IP-1 activity.	Calcium	89-96	Jun proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	27-31	
1437149-6	1992	We observed an increase in AP-1-DNA binding after treatment of the cells with either the calcium ionophore A-23187 or dibutyryl cAMP; this could be ascribed to inhibition of IP-1 activity.	Calcium	89-96	inhibitor of nuclear factor kappa B kinase regulatory subunit gamma	Homo sapiens	174-178	
1437149-6	1992	We observed an increase in AP-1-DNA binding after treatment of the cells with either the calcium ionophore A-23187 or dibutyryl cAMP; this could be ascribed to inhibition of IP-1 activity.	Cyclic AMP	128-132	Jun proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	27-31	
1437149-6	1992	We observed an increase in AP-1-DNA binding after treatment of the cells with either the calcium ionophore A-23187 or dibutyryl cAMP; this could be ascribed to inhibition of IP-1 activity.	Cyclic AMP	128-132	inhibitor of nuclear factor kappa B kinase regulatory subunit gamma	Homo sapiens	174-178	
1437149-10	1992	We also analysed IP-1 regulation upon differentiation of P19 embryonal carcinoma cells by retinoic acid.	Tretinoin	90-103	inhibitor of nuclear factor kappa B kinase regulatory subunit gamma	Homo sapiens	17-21