PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
12668615-0	2003	A naturally occurring mutation in an ATP-binding domain of the recombination repair gene XRCC3 ablates its function without causing cancer susceptibility.	Adenosine Triphosphate	37-40	X-ray repair cross complementing 3	Homo sapiens	89-94	
12668615-4	2003	One of these variants, D213N, occurs in a highly conserved ATP-binding domain and completely abrogates the ability of the transfected gene to correct the phenotype of XRCC3 deficient cells.	Adenosine Triphosphate	59-62	X-ray repair cross complementing 3	Homo sapiens	167-172