PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
12646656-4	2003	However, pretreatment of MBP-primed T cells with NaPA markedly inhibited its ability to induce microglial expression of iNOS and activation of NF-kappaB.	Acecainide	49-53	myelin basic protein	Mus musculus	25-28	
12646656-4	2003	However, pretreatment of MBP-primed T cells with NaPA markedly inhibited its ability to induce microglial expression of iNOS and activation of NF-kappaB.	Acecainide	49-53	nitric oxide synthase 2, inducible	Mus musculus	120-124	
12646656-6	2003	Furthermore, MBP-primed T cells isolated from NaPA-treated donor mice were also less efficient than MBP-primed T cells isolated from normal donor mice in inducing iNOS in microglial cells and transferring EAE to recipient mice.	Acecainide	46-50	myelin basic protein	Homo sapiens	13-16	
12646656-6	2003	Furthermore, MBP-primed T cells isolated from NaPA-treated donor mice were also less efficient than MBP-primed T cells isolated from normal donor mice in inducing iNOS in microglial cells and transferring EAE to recipient mice.	Acecainide	46-50	nitric oxide synthase 2, inducible	Mus musculus	163-167	
12646656-9	2003	Histological and immunocytochemical analysis showed that NaPA inhibited EAE-induced spinal cord mononuclear cell invasion and normalized iNOS, nitrotyrosine, and p65 (the RelA subunit of NF-kappaB) expression within the spinal cord.	Acecainide	57-61	nitric oxide synthase 2, inducible	Mus musculus	137-141	
12646656-9	2003	Histological and immunocytochemical analysis showed that NaPA inhibited EAE-induced spinal cord mononuclear cell invasion and normalized iNOS, nitrotyrosine, and p65 (the RelA subunit of NF-kappaB) expression within the spinal cord.	Acecainide	57-61	v-rel reticuloendotheliosis viral oncogene homolog A (avian)	Mus musculus	162-165