PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
11512043-4	2001	METHODS: Two neuroendocrine challenge tests, measuring the PRL increases induced by acute administration of serotonergic (clomipramine, 25 mg i.v.)	Clomipramine	122-134	prolactin	Homo sapiens	59-62	
11512043-8	2001	PRL was estimated in blood samples taken every 15 min for 1 h for clomipramine and every 30 min for 2 h for haloperidol.	Clomipramine	66-78	prolactin	Homo sapiens	0-3	
11512043-12	2001	In contrast, the PRL increases obtained after clomipramine administration during neuroleptic treatment were preserved after treatment with risperidone.	Clomipramine	46-58	prolactin	Homo sapiens	17-20	
11512043-13	2001	Both PRL response patterns to clomipramine were similar to that of healthy controls.	Clomipramine	30-42	prolactin	Homo sapiens	5-8	
11512043-15	2001	CONCLUSIONS: During treatment with typical neuroleptics, the PRL responses to clomipramine are normal, and they are preserved after switch to risperidone in doses that cause complete dopamine receptor blockade.	Clomipramine	78-90	prolactin	Homo sapiens	61-64