PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 11404398-3 2001 Using reporter constructs and electromobility shift assays, we found that cotreatment of astrocyte cultures with ATP (1-100 microm) significantly potentiated IL-1beta-mediated activation of NF-kappaB and AP-1 and that ATP alone activated AP-1. Adenosine Triphosphate 113-116 interleukin 1 beta Homo sapiens 158-166 11404398-3 2001 Using reporter constructs and electromobility shift assays, we found that cotreatment of astrocyte cultures with ATP (1-100 microm) significantly potentiated IL-1beta-mediated activation of NF-kappaB and AP-1 and that ATP alone activated AP-1. Adenosine Triphosphate 113-116 JunB proto-oncogene, AP-1 transcription factor subunit Homo sapiens 204-208 11404398-3 2001 Using reporter constructs and electromobility shift assays, we found that cotreatment of astrocyte cultures with ATP (1-100 microm) significantly potentiated IL-1beta-mediated activation of NF-kappaB and AP-1 and that ATP alone activated AP-1. Adenosine Triphosphate 113-116 JunB proto-oncogene, AP-1 transcription factor subunit Homo sapiens 238-242 11404398-5 2001 A role for ATP in modulating IL-1beta-mediated inflammatory gene expression was supported further by the observation that ATP potentiated the IL-1beta-induced expression of IL-8 mRNA and protein but strongly downregulated IP-10 expression. Adenosine Triphosphate 11-14 interleukin 1 beta Homo sapiens 29-37 11404398-5 2001 A role for ATP in modulating IL-1beta-mediated inflammatory gene expression was supported further by the observation that ATP potentiated the IL-1beta-induced expression of IL-8 mRNA and protein but strongly downregulated IP-10 expression. Adenosine Triphosphate 11-14 interleukin 1 beta Homo sapiens 142-150 11404398-5 2001 A role for ATP in modulating IL-1beta-mediated inflammatory gene expression was supported further by the observation that ATP potentiated the IL-1beta-induced expression of IL-8 mRNA and protein but strongly downregulated IP-10 expression. Adenosine Triphosphate 11-14 C-X-C motif chemokine ligand 8 Homo sapiens 173-177 11404398-5 2001 A role for ATP in modulating IL-1beta-mediated inflammatory gene expression was supported further by the observation that ATP potentiated the IL-1beta-induced expression of IL-8 mRNA and protein but strongly downregulated IP-10 expression. Adenosine Triphosphate 122-125 interleukin 1 beta Homo sapiens 29-37 11404398-5 2001 A role for ATP in modulating IL-1beta-mediated inflammatory gene expression was supported further by the observation that ATP potentiated the IL-1beta-induced expression of IL-8 mRNA and protein but strongly downregulated IP-10 expression. Adenosine Triphosphate 122-125 interleukin 1 beta Homo sapiens 142-150 11404398-5 2001 A role for ATP in modulating IL-1beta-mediated inflammatory gene expression was supported further by the observation that ATP potentiated the IL-1beta-induced expression of IL-8 mRNA and protein but strongly downregulated IP-10 expression. Adenosine Triphosphate 122-125 C-X-C motif chemokine ligand 8 Homo sapiens 173-177 11404398-5 2001 A role for ATP in modulating IL-1beta-mediated inflammatory gene expression was supported further by the observation that ATP potentiated the IL-1beta-induced expression of IL-8 mRNA and protein but strongly downregulated IP-10 expression. Adenosine Triphosphate 122-125 C-X-C motif chemokine ligand 10 Homo sapiens 222-227 11404398-7 2001 ADP, a selective agonist for P2Y(1), produced results similar to or greater than those obtained using ATP, whereas 2"-3"-O-(4-benzoyl-benzoyl)-ATP, a selective agonist for P2X(7), was less effective than ATP. Adenosine Diphosphate 0-3 purinergic receptor P2Y1 Homo sapiens 29-35 11404398-7 2001 ADP, a selective agonist for P2Y(1), produced results similar to or greater than those obtained using ATP, whereas 2"-3"-O-(4-benzoyl-benzoyl)-ATP, a selective agonist for P2X(7), was less effective than ATP. 2"-3"-o-(4-benzoyl-benzoyl)-atp 115-146 purinergic receptor P2X 7 Homo sapiens 172-178 11404398-7 2001 ADP, a selective agonist for P2Y(1), produced results similar to or greater than those obtained using ATP, whereas 2"-3"-O-(4-benzoyl-benzoyl)-ATP, a selective agonist for P2X(7), was less effective than ATP. Adenosine Triphosphate 143-146 purinergic receptor P2X 7 Homo sapiens 172-178