PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
11001806-1	2000	Smith-Lemli-Opitz syndrome (SLOS), an autosomal recessive condition with multiple malformations, mental retardation, and growth failure, results from markedly reduced activity of the final enzyme in the cholesterol biosynthetic pathway, 7-dehydrocholesterol reductase (DHCR7).	Cholesterol	203-214	7-dehydrocholesterol reductase	Homo sapiens	237-267	
11001806-1	2000	Smith-Lemli-Opitz syndrome (SLOS), an autosomal recessive condition with multiple malformations, mental retardation, and growth failure, results from markedly reduced activity of the final enzyme in the cholesterol biosynthetic pathway, 7-dehydrocholesterol reductase (DHCR7).	Cholesterol	203-214	7-dehydrocholesterol reductase	Homo sapiens	269-274	
11001806-3	2000	The biochemical defect in SLOS is a deficiency of DHCR7, which results in an abnormally low cholesterol level, and increased amounts of intermediates of sterol biosynthesis.	Cholesterol	92-103	7-dehydrocholesterol reductase	Homo sapiens	50-55