PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10675738-1	2000	The involvement of nitric oxide (NO) formed by the inducible isoform of NO synthase (iNOS) has been investigated in the development of rat intestinal lesions following indomethacin administration.	Indomethacin	168-180	nitric oxide synthase 2	Rattus norvegicus	85-89	
10675738-2	2000	Over a 72-h period, indomethacin (10 mg kg(-1), s.c.) provoked a time-dependent increase in expression of iNOS (assessed by the conversion of radiolabelled L-arginine to citrulline) and enhancement of vascular leakage of radiolabelled human serum albumin in the jejunum which commenced 18 h after indomethacin.	Indomethacin	20-32	nitric oxide synthase 2	Rattus norvegicus	106-110	
10675738-2	2000	Over a 72-h period, indomethacin (10 mg kg(-1), s.c.) provoked a time-dependent increase in expression of iNOS (assessed by the conversion of radiolabelled L-arginine to citrulline) and enhancement of vascular leakage of radiolabelled human serum albumin in the jejunum which commenced 18 h after indomethacin.	Indomethacin	297-309	nitric oxide synthase 2	Rattus norvegicus	106-110	
10675738-5	2000	These findings suggest that indomethacin provokes a site-selective expression of iNOS in the rat jejunum which correlates with lesion formation and vascular leakage, whereas both the ileum and colon are spared.	Indomethacin	28-40	nitric oxide synthase 2	Rattus norvegicus	81-85