PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10051731-9	1999	Histopathological studies revealed that erythromycin markedly decreased neutrophils in the lung and skin lesions and myeloperoxidase in the lung, simultaneously with inhibiting ICAM-1 expression.	Erythromycin	40-52	intercellular adhesion molecule 1	Homo sapiens	177-183	
10051731-12	1999	CONCLUSIONS: The therapeutic dosage of erythromycin significantly suppressed acute neutrophil influx into the lung, intradermal Arthus reaction and the expression of ICAM-1 in the lesions of experimental EAA.	Erythromycin	39-51	intercellular adhesion molecule 1	Homo sapiens	166-172