PMID-sentid	Pub_year	Sent_text	compound_name	comp_offset	prot_official_name	organism	prot_offset
11050174-5	2000	Complexes between endogenously expressed hER and AIB1 were detected in estradiol-treated cells and to a much lesser extent in cells treated with the partial agonist, monohydroxytamoxifen.	monohydroxytamoxifen	166-186	ANIB1	Homo sapiens	49-53
6708522-0	1984	The inhibition of prolactin synthesis in GH3 rat pituitary tumor cells by monohydroxytamoxifen is associated with changes in the properties of the estrogen receptor.	monohydroxytamoxifen	74-94	estrogen receptor 1	Rattus norvegicus	147-164
1829955-2	1991	The c-myc mRNA levels were differentially regulated by the synthetic progestin, medroxyprogesterone acetate and the nonsteroidal antiestrogen, monohydroxytamoxifen, in both cell lines.	monohydroxytamoxifen	143-163	MYC proto-oncogene, bHLH transcription factor	Homo sapiens	4-9
3347055-3	1988	By contrast, the timing of the ODC activity peak induced by tamoxifen and monohydroxytamoxifen was highly dependent upon the dosing conditions and was delayed to 18 h at lower tamoxifen doses.	monohydroxytamoxifen	74-94	ornithine decarboxylase 1	Rattus norvegicus	31-34
3347055-4	1988	In immature rats, tamoxifen and monohydroxytamoxifen induced two peaks of uterine ODC activity resembling those induced by estradiol.	monohydroxytamoxifen	32-52	ornithine decarboxylase 1	Rattus norvegicus	82-85
3347055-7	1988	Thus, when dose-related temporal shifts are taken into account, tamoxifen and monohydroxytamoxifen are complete agonists with respect to induction of uterine weight gain and ODC activity.	monohydroxytamoxifen	78-98	ornithine decarboxylase 1	Rattus norvegicus	174-177
2721449-0	1989	Serum and monohydroxytamoxifen inhibit progesterone receptor concentrations in primary rat uterine cells grown in serum-free medium.	monohydroxytamoxifen	10-30	progesterone receptor	Rattus norvegicus	39-60
2721449-5	1989	Addition of serum or monohydroxytamoxifen for 24 h decreases PgR levels by about 50%, and estrogen can completely overcome the inhibition caused by either serum or monohydroxytamoxifen.	monohydroxytamoxifen	21-41	progesterone receptor	Rattus norvegicus	61-64
6184101-9	1982	Although monohydroxytamoxifen has a high binding affinity for the estrogen receptor, the metabolic activation of tamoxifen is an advantage rather than a requirement for antiestrogenic activity.	monohydroxytamoxifen	9-29	estrogen receptor 1	Homo sapiens	66-83
491598-0	1979	Effect of oestradiol benzoate, tamoxifen and monohydroxytamoxifen on immature rat uterine progesterone receptor synthesis and endometrial cell division.	monohydroxytamoxifen	45-65	progesterone receptor	Rattus norvegicus	90-111
207880-2	1978	One derivative, monohydroxytamoxifen, was found to be a potent antiestrogen in the rat, with a high affinity for the estrogen receptor.	monohydroxytamoxifen	16-36	estrogen receptor 1	Rattus norvegicus	117-134