PMID-sentid	Pub_year	Sent_text	compound_name	comp_offset	prot_official_name	organism	prot_offset
34977182-12	2021	miR-153-3p mainly regulates cardiomyocyte apoptosis by regulating the expression of caspase7, further elucidating the importance of apoptosis in heart development.	mir-153-3p	0-10	caspase 7	Rattus norvegicus	84-92
34324029-15	2022	MiR-153-3p was found to modulate breast cancer cell carcinogenesis via targeting CFL2.	mir-153-3p	0-10	cofilin 2	Homo sapiens	81-85
34546850-11	2021	Also, we found that circPIP5K1A regulated TMSB4X level via interaction with miR-153-3p in EM cells.	mir-153-3p	76-86	thymosin beta 4 X-linked	Homo sapiens	42-48
34519634-5	2021	The interaction between miR-153-3p and TWF1 or FGD5-AS1 was determined by luciferase reporter and RIP assays.	mir-153-3p	24-34	twinfilin actin binding protein 1	Homo sapiens	39-43
34197715-10	2021	MiR-153-3p was revealed to bind with circ-RBM4 and directly targeted nuclear factor I/C (NFIC) in mouse podocytes.	mir-153-3p	0-10	RNA binding motif protein 4	Mus musculus	42-46
34519634-5	2021	The interaction between miR-153-3p and TWF1 or FGD5-AS1 was determined by luciferase reporter and RIP assays.	mir-153-3p	24-34	FYVE, RhoGEF and PH domain containing 5	Homo sapiens	47-51
34519634-5	2021	The interaction between miR-153-3p and TWF1 or FGD5-AS1 was determined by luciferase reporter and RIP assays.	mir-153-3p	24-34	prostaglandin D2 receptor	Homo sapiens	52-55
34519634-8	2021	Moreover, FGD5-AS1 directly bound to miR-153-3p and FGD5-AS1 addition neutralized the inhibitory impacts of miR-153-3p supplementation on DDP resistance in the HCC cells.	mir-153-3p	37-47	FYVE, RhoGEF and PH domain containing 5	Homo sapiens	10-14
34519634-8	2021	Moreover, FGD5-AS1 directly bound to miR-153-3p and FGD5-AS1 addition neutralized the inhibitory impacts of miR-153-3p supplementation on DDP resistance in the HCC cells.	mir-153-3p	37-47	prostaglandin D2 receptor	Homo sapiens	15-18
34519634-8	2021	Moreover, FGD5-AS1 directly bound to miR-153-3p and FGD5-AS1 addition neutralized the inhibitory impacts of miR-153-3p supplementation on DDP resistance in the HCC cells.	mir-153-3p	108-118	FYVE, RhoGEF and PH domain containing 5	Homo sapiens	10-14
34519634-8	2021	Moreover, FGD5-AS1 directly bound to miR-153-3p and FGD5-AS1 addition neutralized the inhibitory impacts of miR-153-3p supplementation on DDP resistance in the HCC cells.	mir-153-3p	108-118	prostaglandin D2 receptor	Homo sapiens	15-18
34519634-8	2021	Moreover, FGD5-AS1 directly bound to miR-153-3p and FGD5-AS1 addition neutralized the inhibitory impacts of miR-153-3p supplementation on DDP resistance in the HCC cells.	mir-153-3p	108-118	FYVE, RhoGEF and PH domain containing 5	Homo sapiens	52-56
34519634-8	2021	Moreover, FGD5-AS1 directly bound to miR-153-3p and FGD5-AS1 addition neutralized the inhibitory impacts of miR-153-3p supplementation on DDP resistance in the HCC cells.	mir-153-3p	108-118	prostaglandin D2 receptor	Homo sapiens	57-60
34519634-9	2021	In addition, knockdown of TWF1 inhibited DDP resistance of HCC cells, which was reversed by miR-153-3p deletion.	mir-153-3p	92-102	twinfilin actin binding protein 1	Homo sapiens	26-30
34519634-10	2021	Lastly, FGD5-AS1 interference decreased TWF1 expression level, which was rescued by miR-153-3p inhibition.	mir-153-3p	84-94	FYVE, RhoGEF and PH domain containing 5	Homo sapiens	8-12
34519634-10	2021	Lastly, FGD5-AS1 interference decreased TWF1 expression level, which was rescued by miR-153-3p inhibition.	mir-153-3p	84-94	twinfilin actin binding protein 1	Homo sapiens	40-44
34627697-10	2021	MiR-153-3p overexpression attenuated the enhanced effects of PVT1 on ox-LDL-induced cell damage.	mir-153-3p	0-10	Pvt1 oncogene	Homo sapiens	61-65
34627697-11	2021	GRB2 overexpression reversed the mitigating effects of miR-153-3p on ox-LDL-caused injury.	mir-153-3p	55-65	growth factor receptor bound protein 2	Mus musculus	0-4
34643245-11	2021	A549 cells were transfected with HHIP-AS1 overexpression vector and miR-153-3p mimic, and the miR-153-3p mimic had a mitigating effect on HHIP-AS1 inhibition (P<0.001).	mir-153-3p	68-78	HHIP antisense RNA 1	Homo sapiens	138-146
34643245-11	2021	A549 cells were transfected with HHIP-AS1 overexpression vector and miR-153-3p mimic, and the miR-153-3p mimic had a mitigating effect on HHIP-AS1 inhibition (P<0.001).	mir-153-3p	94-104	HHIP antisense RNA 1	Homo sapiens	138-146
34197715-10	2021	MiR-153-3p was revealed to bind with circ-RBM4 and directly targeted nuclear factor I/C (NFIC) in mouse podocytes.	mir-153-3p	0-10	nuclear factor I/C	Mus musculus	69-87
34197715-10	2021	MiR-153-3p was revealed to bind with circ-RBM4 and directly targeted nuclear factor I/C (NFIC) in mouse podocytes.	mir-153-3p	0-10	nuclear factor I/C	Mus musculus	89-93
34295807-8	2021	The binding relationship between miR-153-3p and circ-MMP11 or ANLN was predicted by circinteractome or starbase, and then verified by a dual-luciferase reporter assay and RNA pull-down assay.	mir-153-3p	33-43	anillin, actin binding protein	Homo sapiens	62-66
34247992-0	2021	MiR-153-3p reduces extracellular matrix accumulation in high glucose-stimulated human glomerular mesangial cells via targeting PAQR3 in diabetic nephropathy.	mir-153-3p	0-10	progestin and adipoQ receptor family member 3	Homo sapiens	127-132
34247992-5	2021	The bioinformatics method and dual-luciferase reporter assay were employed together to anticipate and check the targeting relationship between miR-153-3p and PAQR3.	mir-153-3p	143-153	progestin and adipoQ receptor family member 3	Homo sapiens	158-163
34247992-6	2021	Western blot assays were applied to check the PAQR3, PI3K and AKT expression after miR-153-3p transfection or PAQR3 administration.	mir-153-3p	83-93	progestin and adipoQ receptor family member 3	Homo sapiens	46-51
34247992-6	2021	Western blot assays were applied to check the PAQR3, PI3K and AKT expression after miR-153-3p transfection or PAQR3 administration.	mir-153-3p	83-93	AKT serine/threonine kinase 1	Homo sapiens	62-65
34247992-10	2021	Finally, the fact that miR-153-3p regulates the PI3K/AKT pathway by PAQR3 was confirmed.	mir-153-3p	23-33	AKT serine/threonine kinase 1	Homo sapiens	53-56
34247992-10	2021	Finally, the fact that miR-153-3p regulates the PI3K/AKT pathway by PAQR3 was confirmed.	mir-153-3p	23-33	progestin and adipoQ receptor family member 3	Homo sapiens	68-73
34247992-11	2021	CONCLUSION: MiR-153-3p regulates the PI3K/AKT pathway through PAQR3, thereby playing a role in regulating cell proliferation and ECM accumulation in high glucose-stimulated MCs.	mir-153-3p	12-22	AKT serine/threonine kinase 1	Homo sapiens	42-45
34247992-11	2021	CONCLUSION: MiR-153-3p regulates the PI3K/AKT pathway through PAQR3, thereby playing a role in regulating cell proliferation and ECM accumulation in high glucose-stimulated MCs.	mir-153-3p	12-22	progestin and adipoQ receptor family member 3	Homo sapiens	62-67
34981843-7	2022	For mechanism investigation, circRNF121 could affect activating transcription factor 2 (ATF2) expression by decoying miR-153-3p, thereby accelerating CC cell development.	mir-153-3p	117-127	activating transcription factor 2	Homo sapiens	53-86
35635053-9	2022	In addition, circ_0008068 bound to miR-153-3p to modulate the expression of its target AGK.	mir-153-3p	35-45	acylglycerol kinase	Mus musculus	87-90
35635053-10	2022	Besides, miR-153-3p was validated to act as a tumor suppressor in OSCC tumorigenesis by suppressing AGK.	mir-153-3p	9-19	acylglycerol kinase	Mus musculus	100-103
35305977-7	2022	miR-203a-3p and miR-153-3p improved the cognitive deficits through targeting SRC.	mir-153-3p	16-26	SRC proto-oncogene, non-receptor tyrosine kinase	Rattus norvegicus	77-80
35305977-8	2022	Moreover, miR-203a-3p and miR-153-3p relieved the apoptosis, decreases NLRP3 inflammasome activity, decreases oxidative stress and inflammation in hippocampal neuron through targeting SRC.	mir-153-3p	26-36	SRC proto-oncogene, non-receptor tyrosine kinase	Rattus norvegicus	184-187
35305977-10	2022	Thus, miR-203a-3p and miR-153-3p confers neuroprotective effects against ischemic stroke via attenuation of apoptosis, oxidative stress and inflammatory pathways through inhibiting SRC-dependent MAPK signaling pathway in vivo and in vitro, suggesting new therapeutic targets for the prevention and treatment of stroke.	mir-153-3p	22-32	SRC proto-oncogene, non-receptor tyrosine kinase	Rattus norvegicus	181-184
34981843-7	2022	For mechanism investigation, circRNF121 could affect activating transcription factor 2 (ATF2) expression by decoying miR-153-3p, thereby accelerating CC cell development.	mir-153-3p	117-127	activating transcription factor 2	Homo sapiens	88-92
35232557-0	2022	MiR-153-3p reduces extracellular matrix accumulation in high glucose-stimulated human glomerular mesangial cells via targeting PAQR3 in diabetic nephropathy.	mir-153-3p	0-10	progestin and adipoQ receptor family member 3	Homo sapiens	127-132
35088224-0	2022	miR-153-3p Attenuates the Development of Gastric Cancer by Suppressing SphK2.	mir-153-3p	0-10	sphingosine kinase 2	Homo sapiens	71-76
35088224-8	2022	The binding ability of miR-153-3p and SphK2 was determined by dual-luciferase reporter assays.	mir-153-3p	23-33	sphingosine kinase 2	Homo sapiens	38-43
35088224-14	2022	Rescue assays showed that miR-153-3p could reverse the effect of SphK2 on the cell growth, metastasis, and the apoptosis of GC cells.	mir-153-3p	26-36	sphingosine kinase 2	Homo sapiens	65-70
35088224-15	2022	In conclusion, this study showed that miR-153-3p suppressed the growth and metastasis in GC cells by regulating SphK2, which might facilitate the search for novel biomarkers to treat GC.	mir-153-3p	38-48	sphingosine kinase 2	Homo sapiens	112-117
35426067-0	2022	miR-153-3p inhibited osteogenic differentiation of human DPSCs through CBFbeta signaling.	mir-153-3p	0-10	nuclear transcription factor Y subunit alpha	Homo sapiens	71-78
35426067-8	2022	A luciferase reporter assay confirmed that miR-153-3p directly targeted to CBFbeta.	mir-153-3p	43-53	nuclear transcription factor Y subunit alpha	Homo sapiens	75-82
35426067-12	2022	miR-153-3p negatively regulated the osteogenic differentiation, and overexpression of CBFbeta could offset the negative effect of miR-153-3p.	mir-153-3p	0-10	nuclear transcription factor Y subunit alpha	Homo sapiens	86-93
35426067-12	2022	miR-153-3p negatively regulated the osteogenic differentiation, and overexpression of CBFbeta could offset the negative effect of miR-153-3p.	mir-153-3p	130-140	nuclear transcription factor Y subunit alpha	Homo sapiens	86-93
35232557-5	2022	The bioinformatics method and dual-luciferase reporter assay were employed together to anticipate and check the targeting relationship between miR-153-3p and PAQR3.	mir-153-3p	143-153	progestin and adipoQ receptor family member 3	Homo sapiens	158-163
35232557-6	2022	Western blot assays were applied to check the PAQR3, PI3K and AKT expression after miR-153-3p transfection or PAQR3 administration.	mir-153-3p	83-93	progestin and adipoQ receptor family member 3	Homo sapiens	46-51
35232557-6	2022	Western blot assays were applied to check the PAQR3, PI3K and AKT expression after miR-153-3p transfection or PAQR3 administration.	mir-153-3p	83-93	AKT serine/threonine kinase 1	Homo sapiens	62-65
35232557-10	2022	Finally, the fact that miR-153-3p regulates the PI3K/AKT pathway by PAQR3 was confirmed.	mir-153-3p	23-33	AKT serine/threonine kinase 1	Homo sapiens	53-56
35232557-10	2022	Finally, the fact that miR-153-3p regulates the PI3K/AKT pathway by PAQR3 was confirmed.	mir-153-3p	23-33	progestin and adipoQ receptor family member 3	Homo sapiens	68-73
35232557-11	2022	CONCLUSION: MiR-153-3p regulates the PI3K/AKT pathway through PAQR3, thereby playing a role in regulating cell proliferation and ECM accumulation in high glucose-stimulated MCs.	mir-153-3p	12-22	AKT serine/threonine kinase 1	Homo sapiens	42-45
35232557-11	2022	CONCLUSION: MiR-153-3p regulates the PI3K/AKT pathway through PAQR3, thereby playing a role in regulating cell proliferation and ECM accumulation in high glucose-stimulated MCs.	mir-153-3p	12-22	progestin and adipoQ receptor family member 3	Homo sapiens	62-67
33368310-0	2021	MiR-153-3p inhibits osteogenic differentiation of periodontal ligament stem cells through KDM6A-induced demethylation of H3K27me3.	mir-153-3p	0-10	lysine demethylase 6A	Homo sapiens	90-95
33368310-8	2021	A luciferase reporter assay was used to confirm the interaction between KDM6A and miR-153-3p.	mir-153-3p	82-92	lysine demethylase 6A	Homo sapiens	72-77
33469676-8	2021	Furthermore, circ_0008537 indirectly affected the expression levels of pro-survival protein myeloid cell leukemia 1 (MCL1) and snail family zinc finger 1 (Snail1) via miR-153-3p in liver cancer cells.	mir-153-3p	167-177	MCL1 apoptosis regulator, BCL2 family member	Homo sapiens	92-115
33368310-11	2021	miR-153-3p downregulation or KDM6A overexpression promoted the osteogenic differentiation of PDLSCs, as demonstrated by increases in ALP activity, matrix mineralization, and ALP, Runx2, and OPN expression.	mir-153-3p	0-10	alkaline phosphatase, placental	Homo sapiens	133-136
33368310-11	2021	miR-153-3p downregulation or KDM6A overexpression promoted the osteogenic differentiation of PDLSCs, as demonstrated by increases in ALP activity, matrix mineralization, and ALP, Runx2, and OPN expression.	mir-153-3p	0-10	alkaline phosphatase, placental	Homo sapiens	174-177
33368310-11	2021	miR-153-3p downregulation or KDM6A overexpression promoted the osteogenic differentiation of PDLSCs, as demonstrated by increases in ALP activity, matrix mineralization, and ALP, Runx2, and OPN expression.	mir-153-3p	0-10	RUNX family transcription factor 2	Homo sapiens	179-184
33368310-11	2021	miR-153-3p downregulation or KDM6A overexpression promoted the osteogenic differentiation of PDLSCs, as demonstrated by increases in ALP activity, matrix mineralization, and ALP, Runx2, and OPN expression.	mir-153-3p	0-10	secreted phosphoprotein 1	Homo sapiens	190-193
33368310-12	2021	KDM6A was confirmed to be a target of miR-153-3p, and KDM6A overexpression reversed the inhibitory effect of miR-153-3p mimic on PDLSC osteogenesis.	mir-153-3p	38-48	lysine demethylase 6A	Homo sapiens	0-5
33368310-12	2021	KDM6A was confirmed to be a target of miR-153-3p, and KDM6A overexpression reversed the inhibitory effect of miR-153-3p mimic on PDLSC osteogenesis.	mir-153-3p	109-119	lysine demethylase 6A	Homo sapiens	0-5
33368310-12	2021	KDM6A was confirmed to be a target of miR-153-3p, and KDM6A overexpression reversed the inhibitory effect of miR-153-3p mimic on PDLSC osteogenesis.	mir-153-3p	109-119	lysine demethylase 6A	Homo sapiens	54-59
33368310-14	2021	CONCLUSION: miR-153-3p inhibited PDLSC osteogenesis by targeting KDM6A and inhibiting ALP, Runx2, and OPN transcription.	mir-153-3p	12-22	lysine demethylase 6A	Homo sapiens	65-70
33368310-14	2021	CONCLUSION: miR-153-3p inhibited PDLSC osteogenesis by targeting KDM6A and inhibiting ALP, Runx2, and OPN transcription.	mir-153-3p	12-22	alkaline phosphatase, placental	Homo sapiens	86-89
33368310-14	2021	CONCLUSION: miR-153-3p inhibited PDLSC osteogenesis by targeting KDM6A and inhibiting ALP, Runx2, and OPN transcription.	mir-153-3p	12-22	RUNX family transcription factor 2	Homo sapiens	91-96
33368310-14	2021	CONCLUSION: miR-153-3p inhibited PDLSC osteogenesis by targeting KDM6A and inhibiting ALP, Runx2, and OPN transcription.	mir-153-3p	12-22	secreted phosphoprotein 1	Homo sapiens	102-105
33469676-8	2021	Furthermore, circ_0008537 indirectly affected the expression levels of pro-survival protein myeloid cell leukemia 1 (MCL1) and snail family zinc finger 1 (Snail1) via miR-153-3p in liver cancer cells.	mir-153-3p	167-177	MCL1 apoptosis regulator, BCL2 family member	Homo sapiens	117-121
33469676-8	2021	Furthermore, circ_0008537 indirectly affected the expression levels of pro-survival protein myeloid cell leukemia 1 (MCL1) and snail family zinc finger 1 (Snail1) via miR-153-3p in liver cancer cells.	mir-153-3p	167-177	snail family transcriptional repressor 1	Homo sapiens	127-153
33469676-8	2021	Furthermore, circ_0008537 indirectly affected the expression levels of pro-survival protein myeloid cell leukemia 1 (MCL1) and snail family zinc finger 1 (Snail1) via miR-153-3p in liver cancer cells.	mir-153-3p	167-177	snail family transcriptional repressor 1	Homo sapiens	155-161
33500627-15	2021	The level of miR-153-3p was negatively related with TNM stage.	mir-153-3p	13-23	teneurin transmembrane protein 1	Homo sapiens	52-55
33015775-11	2020	Furthermore, inhibition of miR-153-3p counteracted the effects of LINC00858 knockdown on proliferation and invasion of HCC cells.	mir-153-3p	27-37	long intergenic non-protein coding RNA 858	Homo sapiens	66-75
32902234-0	2020	Enhanced Serum miR-153-3p Promotes the Development of Knee Osteoarthritis by Targeting SOST.	mir-153-3p	15-25	sclerostin	Homo sapiens	87-91
32902234-4	2020	The relationship between serum miR-153-3p level and C-reactive protein (CRP) and erythrocyte sedimentation rate (ESR) was analyzed by Pearson"s correlation assay.	mir-153-3p	31-41	C-reactive protein	Homo sapiens	52-70
32902234-4	2020	The relationship between serum miR-153-3p level and C-reactive protein (CRP) and erythrocyte sedimentation rate (ESR) was analyzed by Pearson"s correlation assay.	mir-153-3p	31-41	C-reactive protein	Homo sapiens	72-75
32902234-7	2020	A positive correlation was found between the serum miR-153-3p level and ESR/CRP levels.	mir-153-3p	51-61	C-reactive protein	Homo sapiens	76-79
32902234-11	2020	Pearson"s correlation assay showed a negative correlation between serum miR-153-3p and serum SOST levels.	mir-153-3p	72-82	sclerostin	Homo sapiens	93-97
33488947-9	2021	ApoE epsilon4 subjects showed a reduction in plasma AOC and an increase in the Nrf2 blocker, miR-153-3p, contributing to an enhancement of the erythrocyte concentration of Abeta.	mir-153-3p	93-103	apolipoprotein E	Homo sapiens	0-4
33488947-9	2021	ApoE epsilon4 subjects showed a reduction in plasma AOC and an increase in the Nrf2 blocker, miR-153-3p, contributing to an enhancement of the erythrocyte concentration of Abeta.	mir-153-3p	93-103	NFE2 like bZIP transcription factor 2	Homo sapiens	79-83
33488947-9	2021	ApoE epsilon4 subjects showed a reduction in plasma AOC and an increase in the Nrf2 blocker, miR-153-3p, contributing to an enhancement of the erythrocyte concentration of Abeta.	mir-153-3p	93-103	amyloid beta precursor protein	Homo sapiens	172-177
33164880-10	2021	Mechanistically, miR-153-3p regulates the VEGF/VEGFR2/PI3K/Akt/eNOS pathways by targeting ANGPT1.	mir-153-3p	17-27	vascular endothelial growth factor A	Homo sapiens	42-46
33164880-10	2021	Mechanistically, miR-153-3p regulates the VEGF/VEGFR2/PI3K/Akt/eNOS pathways by targeting ANGPT1.	mir-153-3p	17-27	kinase insert domain receptor	Homo sapiens	47-53
33164880-10	2021	Mechanistically, miR-153-3p regulates the VEGF/VEGFR2/PI3K/Akt/eNOS pathways by targeting ANGPT1.	mir-153-3p	17-27	AKT serine/threonine kinase 1	Homo sapiens	59-62
33164880-10	2021	Mechanistically, miR-153-3p regulates the VEGF/VEGFR2/PI3K/Akt/eNOS pathways by targeting ANGPT1.	mir-153-3p	17-27	nitric oxide synthase 3	Homo sapiens	63-67
33164880-10	2021	Mechanistically, miR-153-3p regulates the VEGF/VEGFR2/PI3K/Akt/eNOS pathways by targeting ANGPT1.	mir-153-3p	17-27	angiopoietin 1	Homo sapiens	90-96
33164880-12	2021	Collectively, MSCs-derived exosomes with low-expressed miR-153-3p notably promotes the activation of ANGPT1 and the VEGF/VEGFR2 /PI3K/Akt/eNOS pathways, thereby preventing the damages endothelial cells and cardiomyocytes against hypoxia.	mir-153-3p	55-65	angiopoietin 1	Homo sapiens	101-107
33164880-12	2021	Collectively, MSCs-derived exosomes with low-expressed miR-153-3p notably promotes the activation of ANGPT1 and the VEGF/VEGFR2 /PI3K/Akt/eNOS pathways, thereby preventing the damages endothelial cells and cardiomyocytes against hypoxia.	mir-153-3p	55-65	vascular endothelial growth factor A	Homo sapiens	116-120
33164880-12	2021	Collectively, MSCs-derived exosomes with low-expressed miR-153-3p notably promotes the activation of ANGPT1 and the VEGF/VEGFR2 /PI3K/Akt/eNOS pathways, thereby preventing the damages endothelial cells and cardiomyocytes against hypoxia.	mir-153-3p	55-65	kinase insert domain receptor	Homo sapiens	121-127
33164880-12	2021	Collectively, MSCs-derived exosomes with low-expressed miR-153-3p notably promotes the activation of ANGPT1 and the VEGF/VEGFR2 /PI3K/Akt/eNOS pathways, thereby preventing the damages endothelial cells and cardiomyocytes against hypoxia.	mir-153-3p	55-65	AKT serine/threonine kinase 1	Homo sapiens	134-137
33164880-12	2021	Collectively, MSCs-derived exosomes with low-expressed miR-153-3p notably promotes the activation of ANGPT1 and the VEGF/VEGFR2 /PI3K/Akt/eNOS pathways, thereby preventing the damages endothelial cells and cardiomyocytes against hypoxia.	mir-153-3p	55-65	nitric oxide synthase 3	Homo sapiens	138-142
33182030-13	2020	Transfecting miR-153-3p mimics into trophoblast cells directly inhibited IDO genes, which suppressed STAT3 pathway activation, regulating the biological behavior of trophoblast cells.	mir-153-3p	13-23	indoleamine 2,3-dioxygenase 1	Homo sapiens	73-76
33182030-13	2020	Transfecting miR-153-3p mimics into trophoblast cells directly inhibited IDO genes, which suppressed STAT3 pathway activation, regulating the biological behavior of trophoblast cells.	mir-153-3p	13-23	signal transducer and activator of transcription 3	Homo sapiens	101-106
32912499-9	2020	Knockdown of KCNQ1OT1 or overexpression of miR-153-3p weakened OGD/R-induced neuronal injury and regulated Foxo3 expressions.	mir-153-3p	43-53	forkhead box O3	Mus musculus	107-112
33015775-12	2020	In addition, the overexpression of Rabl3 rescued the effects of miR-153-3p on cell proliferation and invasion of HCC cells.	mir-153-3p	64-74	RAB, member of RAS oncogene family like 3	Homo sapiens	35-40
32675387-4	2020	FGD5-AS1 acted as a sponge of miR-153-3p, and MCL1 was direct target of miR-153-3p.	mir-153-3p	30-40	prostaglandin D2 receptor	Homo sapiens	5-8
32675387-4	2020	FGD5-AS1 acted as a sponge of miR-153-3p, and MCL1 was direct target of miR-153-3p.	mir-153-3p	72-82	FYVE, RhoGEF and PH domain containing 5	Homo sapiens	0-4
32675387-4	2020	FGD5-AS1 acted as a sponge of miR-153-3p, and MCL1 was direct target of miR-153-3p.	mir-153-3p	72-82	prostaglandin D2 receptor	Homo sapiens	5-8
32675387-4	2020	FGD5-AS1 acted as a sponge of miR-153-3p, and MCL1 was direct target of miR-153-3p.	mir-153-3p	72-82	MCL1 apoptosis regulator, BCL2 family member	Homo sapiens	46-50
32675387-0	2020	LncRNA FGD5-AS1 promotes tumor growth by regulating MCL1 via sponging miR-153-3p in oral cancer.	mir-153-3p	70-80	FGD5 antisense RNA 1	Homo sapiens	7-15
32675387-0	2020	LncRNA FGD5-AS1 promotes tumor growth by regulating MCL1 via sponging miR-153-3p in oral cancer.	mir-153-3p	70-80	MCL1 apoptosis regulator, BCL2 family member	Homo sapiens	52-56
32675387-5	2020	Forced expression of miR-153-3p or inhibition of MCL1 reversed the promoted role of FGD5-AS1 on OC cells" migration and invasion.	mir-153-3p	21-31	FYVE, RhoGEF and PH domain containing 5	Homo sapiens	84-88
32675387-4	2020	FGD5-AS1 acted as a sponge of miR-153-3p, and MCL1 was direct target of miR-153-3p.	mir-153-3p	30-40	FYVE, RhoGEF and PH domain containing 5	Homo sapiens	0-4
32675387-5	2020	Forced expression of miR-153-3p or inhibition of MCL1 reversed the promoted role of FGD5-AS1 on OC cells" migration and invasion.	mir-153-3p	21-31	prostaglandin D2 receptor	Homo sapiens	89-92
32321315-0	2020	MiR-153-3p induces immune dysregulation by inhibiting PELI1 expression in umbilical cord-derived mesenchymal stem cells in patients with systemic lupus erythematosus.	mir-153-3p	0-10	pellino E3 ubiquitin protein ligase 1	Homo sapiens	54-59
32342278-8	2020	Bcl-2 restoration reversed the increased sensitivity to IM induced by miR-153-3p-mimic transfection in IM-resistant CML cells.	mir-153-3p	70-80	BCL2 apoptosis regulator	Homo sapiens	0-5
32342278-7	2020	Dual-luciferase reporter assays confirmed that Bcl-2 is a direct target of miR-153-3p.	mir-153-3p	75-85	BCL2 apoptosis regulator	Homo sapiens	47-52
32321315-10	2020	We also found that PELI1 overexpression abolished the function of miR-153-3p on UC-MSCs.	mir-153-3p	66-76	pellino E3 ubiquitin protein ligase 1	Homo sapiens	19-24
31836665-10	2020	Elevated miR-153-3p levels correlated with LRP5/6 and FZD10, and miR-153-3p sequestration via a plasmid-based miR inhibitor system attenuated Wnt signaling.	mir-153-3p	9-19	low density lipoprotein receptor-related protein 5	Mus musculus	43-49
32314711-0	2020	[miR-153-3p inhibits proliferation and migration of breast cancer cells via down-regulating ROCK1].	mir-153-3p	1-11	Rho associated coiled-coil containing protein kinase 1	Homo sapiens	92-97
32314711-9	2020	The miR-153-3p mimics co-transfected with ROCK1 wild-type reporter vector significantly decreased the relative activity of luciferase in MDA-MB-231 cells.	mir-153-3p	4-14	Rho associated coiled-coil containing protein kinase 1	Homo sapiens	42-47
32314711-12	2020	Conclusion The miR-153-3p can inhibit the proliferation, migration and invasion of breast cancer cells via down-regulation of ROCK1.	mir-153-3p	15-25	Rho associated coiled-coil containing protein kinase 1	Homo sapiens	126-131
32380843-9	2020	At the same time, NEAT1 directly interacts with miR-153-3p in NSCLC.	mir-153-3p	48-58	nuclear paraspeckle assembly transcript 1	Homo sapiens	18-23
32380843-10	2020	In addition, upregulation of miR-153-3p inhibits the cell progression, and miR-153-3p inhibitor recovers the inhibition effect of si-NEAT1 in NSCLC cell lines.	mir-153-3p	75-85	nuclear paraspeckle assembly transcript 1	Homo sapiens	133-138
32380843-11	2020	Subsequently, si-NEAT1 inhibits Wnt/beta-catenin signaling pathway, which is reactivated by miR-153-3p inhibitor.	mir-153-3p	92-102	nuclear paraspeckle assembly transcript 1	Homo sapiens	17-22
32380843-11	2020	Subsequently, si-NEAT1 inhibits Wnt/beta-catenin signaling pathway, which is reactivated by miR-153-3p inhibitor.	mir-153-3p	92-102	catenin beta 1	Homo sapiens	36-48
32267161-4	2020	Results: KTN1-AS1, a competing endogenous RNA, promoted cell proliferation, migration, invasion and epithelial-mesenchymal transition by sponging miR-153-3p in HNSCC.	mir-153-3p	146-156	kinectin 1	Homo sapiens	9-13
32267161-4	2020	Results: KTN1-AS1, a competing endogenous RNA, promoted cell proliferation, migration, invasion and epithelial-mesenchymal transition by sponging miR-153-3p in HNSCC.	mir-153-3p	146-156	prostaglandin D2 receptor	Homo sapiens	14-17
32267161-5	2020	Dysregulation of SNAI1 and ZEB2 mediated the effect of KTN1-AS1 due to miR-153-3p exhaustion.	mir-153-3p	71-81	snail family transcriptional repressor 1	Homo sapiens	17-22
32267161-5	2020	Dysregulation of SNAI1 and ZEB2 mediated the effect of KTN1-AS1 due to miR-153-3p exhaustion.	mir-153-3p	71-81	zinc finger E-box binding homeobox 2	Homo sapiens	27-31
32267161-5	2020	Dysregulation of SNAI1 and ZEB2 mediated the effect of KTN1-AS1 due to miR-153-3p exhaustion.	mir-153-3p	71-81	kinectin 1	Homo sapiens	55-59
32267161-5	2020	Dysregulation of SNAI1 and ZEB2 mediated the effect of KTN1-AS1 due to miR-153-3p exhaustion.	mir-153-3p	71-81	prostaglandin D2 receptor	Homo sapiens	60-63
31836665-10	2020	Elevated miR-153-3p levels correlated with LRP5/6 and FZD10, and miR-153-3p sequestration via a plasmid-based miR inhibitor system attenuated Wnt signaling.	mir-153-3p	9-19	frizzled class receptor 10	Mus musculus	54-59
31907031-14	2020	Finally, SNHG1 was found to activate PTEN/AKT/mTOR signaling pathway in SH-SY5Y cells by targeting miR-153-3p.	mir-153-3p	99-109	AKT serine/threonine kinase 1	Homo sapiens	42-45
31138034-8	2019	Moreover, inhibitor of growth protein 2 was validated as a direct target of miR-153-3p and the overexpression of inhibitor of growth protein 2 reversed the suppressive effects of miR-153-3p on acute lymphoblastic leukemia cell behaviors.	mir-153-3p	76-86	inhibitor of growth family member 2	Homo sapiens	10-39
31907031-0	2020	SNHG1 promotes MPP+-induced cytotoxicity by regulating PTEN/AKT/mTOR signaling pathway in SH-SY5Y cells via sponging miR-153-3p.	mir-153-3p	117-127	small nucleolar RNA host gene 1	Homo sapiens	0-5
31907031-11	2020	Furthermore, miR-153-3p-mediated suppression of MPP+-induced cytotoxicity was abated following SNHG1 up-regulation.	mir-153-3p	13-23	small nucleolar RNA host gene 1	Homo sapiens	95-100
31907031-14	2020	Finally, SNHG1 was found to activate PTEN/AKT/mTOR signaling pathway in SH-SY5Y cells by targeting miR-153-3p.	mir-153-3p	99-109	small nucleolar RNA host gene 1	Homo sapiens	9-14
31907031-14	2020	Finally, SNHG1 was found to activate PTEN/AKT/mTOR signaling pathway in SH-SY5Y cells by targeting miR-153-3p.	mir-153-3p	99-109	phosphatase and tensin homolog	Homo sapiens	37-41
31907031-14	2020	Finally, SNHG1 was found to activate PTEN/AKT/mTOR signaling pathway in SH-SY5Y cells by targeting miR-153-3p.	mir-153-3p	99-109	mechanistic target of rapamycin kinase	Homo sapiens	46-50
31907031-15	2020	CONCLUSION: SNHG1 aggravates MPP+-induced cellular toxicity in SH-SY5Y cells by regulating PTEN/AKT/mTOR signaling via sponging miR-153-3p, indicating the potential of SNHG1 as a promising therapeutic target for PD.	mir-153-3p	128-138	small nucleolar RNA host gene 1	Homo sapiens	12-17
31297886-1	2019	The study of either miR-153-3p or MCL1 gene in ovarian carcinoma (OVC) has been reported; however, the interaction between miR-153-3p and MCL1 gene in OVC as well as the influence of their interaction on OVC progression has not been reported yet.	mir-153-3p	20-30	Oncogene OVC (ovarian adenocarcinoma oncogene)	Homo sapiens	66-69
31297886-9	2019	miR-153-3p was found to target MCL1 mRNA and interfered OVC progression.	mir-153-3p	0-10	Oncogene OVC (ovarian adenocarcinoma oncogene)	Homo sapiens	56-59
31297886-10	2019	The repression of MCL1 gene expression caused by either miR-153-3p or small interfering RNA technique led to significantly reduced OVC cell growth and invasion in vitro.	mir-153-3p	56-66	Oncogene OVC (ovarian adenocarcinoma oncogene)	Homo sapiens	131-134
31297886-13	2019	In this study, we revealed the role of miR-153-3p on OVC progression by targeting MCL1 gene.	mir-153-3p	39-49	Oncogene OVC (ovarian adenocarcinoma oncogene)	Homo sapiens	53-56
31138034-8	2019	Moreover, inhibitor of growth protein 2 was validated as a direct target of miR-153-3p and the overexpression of inhibitor of growth protein 2 reversed the suppressive effects of miR-153-3p on acute lymphoblastic leukemia cell behaviors.	mir-153-3p	179-189	inhibitor of growth family member 2	Homo sapiens	10-39
27340781-8	2016	In a GVHD model, recipient mice injected with a miR-153-3p antagomir exhibited higher IDO expression levels at the early stage after transplantation, as well as delayed aGVHD and longer survival, indicating that the miR-153-3p level at +7 d post-transplant is a good predictor of aGVHD.	mir-153-3p	48-58	indoleamine 2,3-dioxygenase 1	Mus musculus	86-89