PMID-sentid	Pub_year	Sent_text	compound_name	comp_offset	prot_official_name	organism	prot_offset
34389797-10	2021	Silencing of Arl2 or overexpression miR-214-3p reversed the effects of XIST on inflammation and pyroptosis.	mir-214-3p	36-46	inactive X specific transcripts	Mus musculus	71-75
27823969-3	2016	MiR-214-3p was markedly decreased in the fibrotic myocardium of a mouse Ang-II infusion model, but was upregulated in Ang-II-treated mouse myofibroblasts.	mir-214-3p	0-10	angiotensinogen (serpin peptidase inhibitor, clade A, member 8)	Mus musculus	72-78
27823969-3	2016	MiR-214-3p was markedly decreased in the fibrotic myocardium of a mouse Ang-II infusion model, but was upregulated in Ang-II-treated mouse myofibroblasts.	mir-214-3p	0-10	angiotensinogen (serpin peptidase inhibitor, clade A, member 8)	Mus musculus	118-124
34427853-4	2022	Here, we report that miR-214-3p, one of the downregulated miRNAs identified in the skin of psoriatic patients and imiquimod (IMQ)-induced mouse models, can negatively regulate the expression of forkhead box M1 (FOXM1).	mir-214-3p	21-31	forkhead box M1	Mus musculus	194-209
34427853-4	2022	Here, we report that miR-214-3p, one of the downregulated miRNAs identified in the skin of psoriatic patients and imiquimod (IMQ)-induced mouse models, can negatively regulate the expression of forkhead box M1 (FOXM1).	mir-214-3p	21-31	forkhead box M1	Mus musculus	211-216
34804162-9	2021	Overexpression of LINC00665 led to significant enhancements in cell viability and colony formation, whereas this effect was weakened when miR-214-3p was overexpressed or MAPK1 was downregulated.	mir-214-3p	138-148	long intergenic non-protein coding RNA 665	Homo sapiens	18-27
30720150-0	2019	MiR-214-3p delays fracture healing in rats with osteoporotic fracture through inhibiting BMP/Smad signaling pathway.	mir-214-3p	0-10	SMAD family member 4	Rattus norvegicus	93-97
30720150-13	2019	CONCLUSIONS: MiR-214-3p delays fracture healing in rats with osteoporotic fracture by inhibiting the BMP/Smad signaling pathway.	mir-214-3p	13-23	SMAD family member 4	Rattus norvegicus	105-109
29364467-8	2018	CONCLUSIONS: MiR-214-3p delays the fracture healing by inhibiting the Wnt/beta-catenin signaling pathway.	mir-214-3p	13-23	catenin (cadherin associated protein), beta 1	Mus musculus	74-86
34820454-0	2021	LncRNA PVT1 Promotes Hypoxia-Induced Cardiomyocyte Injury by Inhibiting miR-214-3p.	mir-214-3p	72-82	Pvt1 oncogene	Rattus norvegicus	7-11
34820454-9	2021	In addition, miR-214-3p inhibitors reversed the viability of H9c2 cells with PVT1 knockout and promoted apoptosis.	mir-214-3p	13-23	Pvt1 oncogene	Rattus norvegicus	77-81
34296787-0	2021	MiR-214-3p plays a protective role in diabetic neuropathic rats by regulating Nav1.3 and TLR4.	mir-214-3p	0-10	sodium voltage-gated channel alpha subunit 3	Rattus norvegicus	78-84
34296787-0	2021	MiR-214-3p plays a protective role in diabetic neuropathic rats by regulating Nav1.3 and TLR4.	mir-214-3p	0-10	toll-like receptor 4	Rattus norvegicus	89-93
34360002-7	2021	We found that FGFR1 is a functional target of miR-214-3p, preventing the signaling of the anti-fibrotic FGF2 pathway during FAP fibrogenesis.	mir-214-3p	46-56	fibroblast growth factor receptor 1	Homo sapiens	14-19
34137063-16	2021	However, the inhibition of miR-214-3p attenuated the effect of lncRNA Miat downregulation on HAECs.	mir-214-3p	27-37	myocardial infarction associated transcript	Homo sapiens	70-74
34336642-14	2021	Dual-luciferase reporter gene assay showed that SNHG3 and ASF1B directly bound to miR-214-3p.	mir-214-3p	82-92	small nucleolar RNA host gene 3	Homo sapiens	48-53
34336642-14	2021	Dual-luciferase reporter gene assay showed that SNHG3 and ASF1B directly bound to miR-214-3p.	mir-214-3p	82-92	anti-silencing function 1B histone chaperone	Homo sapiens	58-63
34360002-7	2021	We found that FGFR1 is a functional target of miR-214-3p, preventing the signaling of the anti-fibrotic FGF2 pathway during FAP fibrogenesis.	mir-214-3p	46-56	fibroblast growth factor 2	Homo sapiens	104-108
33714889-0	2021	Decreased miR-214-3p activates NF-kappaB pathway and aggravates osteoarthritis progression.	mir-214-3p	10-20	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	31-40
35481549-9	2022	Further functional assays suggested that CDIP1-plasmid reversed the effects of miR-214-3p mimic on MPP+-stimulated SH-SY5Y cells.	mir-214-3p	79-89	cell death inducing p53 target 1	Homo sapiens	41-46
34327240-0	2021	Inhibition of miR-214-3p Protects Endothelial Cells from ox-LDL-Induced Damage by Targeting GPX4.	mir-214-3p	14-24	glutathione peroxidase 4	Homo sapiens	92-96
34327240-8	2021	Our data demonstrated that ox-LDL could induce upregulation of miR-214-3p and result in suppression of GPX4 in VECs.	mir-214-3p	63-73	glutathione peroxidase 4	Homo sapiens	103-107
34327240-9	2021	Downregulation of miR-214-3p could protect VECs from ROS-induced endothelial dysfunction by reversing its inhibitory effect on GPX4 expression.	mir-214-3p	18-28	glutathione peroxidase 4	Homo sapiens	127-131
35433428-8	2022	MiR-214-3p and miR-21-5p were significantly upregulated in serum of epithelioid cell melanoma patients compared to spindle-shaped melanoma patients and miR-132-3p and, conversely, were significantly downregulated in serum of epithelioid cell melanoma patients.	mir-214-3p	0-10	microRNA 1323	Homo sapiens	152-162
33935961-6	2020	Consequently, miR-214-3p was downregulated in AS fibroblasts, with enhanced ALP activity and calcium nodules, which were reversed by miR-214-3p overexpression.	mir-214-3p	14-24	alkaline phosphatase, placental	Homo sapiens	76-79
33935961-6	2020	Consequently, miR-214-3p was downregulated in AS fibroblasts, with enhanced ALP activity and calcium nodules, which were reversed by miR-214-3p overexpression.	mir-214-3p	133-143	alkaline phosphatase, placental	Homo sapiens	76-79
33714889-11	2021	INTERPRETATION: Decreased miR-214-3p activates the NF-kappaB signaling pathway and aggravates OA development through targeting IKKbeta, suggesting miR-214-3p may be a novel therapeutic target for OA.	mir-214-3p	26-36	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	51-60
33714889-11	2021	INTERPRETATION: Decreased miR-214-3p activates the NF-kappaB signaling pathway and aggravates OA development through targeting IKKbeta, suggesting miR-214-3p may be a novel therapeutic target for OA.	mir-214-3p	26-36	conserved helix-loop-helix ubiquitous kinase	Mus musculus	127-134
33714889-11	2021	INTERPRETATION: Decreased miR-214-3p activates the NF-kappaB signaling pathway and aggravates OA development through targeting IKKbeta, suggesting miR-214-3p may be a novel therapeutic target for OA.	mir-214-3p	147-157	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	51-60
33714889-11	2021	INTERPRETATION: Decreased miR-214-3p activates the NF-kappaB signaling pathway and aggravates OA development through targeting IKKbeta, suggesting miR-214-3p may be a novel therapeutic target for OA.	mir-214-3p	147-157	conserved helix-loop-helix ubiquitous kinase	Mus musculus	127-134
33197504-14	2021	The miR-214-3p inhibitor reversed the protective effects of sh BACE1-AS and sh ATG5 against Abeta1-42-induced cell injury.	mir-214-3p	4-14	autophagy related 5	Homo sapiens	79-83
33048316-5	2021	This study aimed to explore the role of exosomal miR-214-3p in endometriosis fibrosis and the relationship between CCN2 and miR-214-3p in endometriosis fibrosis.	mir-214-3p	124-134	cellular communication network factor 2	Homo sapiens	115-119
33048316-7	2021	Exosomal miR-214-3p can inhibit fibrosis in EMs through targeting CCN2.	mir-214-3p	9-19	cellular communication network factor 2	Homo sapiens	66-70
32863772-8	2020	Conclusion: MiR-214-3p plays a pivotal role in CSCs properties by targeting YAP1, which provides a potential treatment strategy for LSCC patients.	mir-214-3p	12-22	Yes1 associated transcriptional regulator	Homo sapiens	76-80
32352854-9	2020	The up-regulation of miR-214-3p inhibited the protein expression of LIVIN and suppressed the activation of the NF-kappaB signaling pathway.	mir-214-3p	21-31	baculoviral IAP repeat containing 7	Homo sapiens	68-73
32352854-9	2020	The up-regulation of miR-214-3p inhibited the protein expression of LIVIN and suppressed the activation of the NF-kappaB signaling pathway.	mir-214-3p	21-31	nuclear factor kappa B subunit 1	Homo sapiens	111-120
32352854-11	2020	The expression of LIVIN and the activation of the NF-kappaB signaling pathway were suppressed by down-regulating DNAJC3-AS1, while these effects could be restored by miR-214-3p inhibitor.	mir-214-3p	166-176	baculoviral IAP repeat containing 7	Homo sapiens	18-23
32352854-11	2020	The expression of LIVIN and the activation of the NF-kappaB signaling pathway were suppressed by down-regulating DNAJC3-AS1, while these effects could be restored by miR-214-3p inhibitor.	mir-214-3p	166-176	nuclear factor kappa B subunit 1	Homo sapiens	50-59
33312221-5	2020	Both BBR and miR-214-3p mimic reduced the cell viability, repressed migration and invasion capacities, increased rates of total apoptotic cells and ratio of Bax/Bcl-2, and increased the percentage of G2/M cells of MCF-7 and MDA-MB-231 cells by colony formation and CKK8 assay, scratch wound healing and gelatin-based 3D conformation assay, transwell invasion assay, and cell cycle analysis, respectively.	mir-214-3p	13-23	BCL2 associated X, apoptosis regulator	Homo sapiens	157-160
33312221-5	2020	Both BBR and miR-214-3p mimic reduced the cell viability, repressed migration and invasion capacities, increased rates of total apoptotic cells and ratio of Bax/Bcl-2, and increased the percentage of G2/M cells of MCF-7 and MDA-MB-231 cells by colony formation and CKK8 assay, scratch wound healing and gelatin-based 3D conformation assay, transwell invasion assay, and cell cycle analysis, respectively.	mir-214-3p	13-23	BCL2 apoptosis regulator	Homo sapiens	161-166
33071725-13	2020	Furthermore, miR-214-3p overexpression alleviated MPP+-stimulated damage in SK-N-SH cells by downregulating KLF4.	mir-214-3p	13-23	Kruppel like factor 4	Homo sapiens	108-112
32681443-12	2020	Additionally, miR-214-3p silencing reversed the influence of BACE1-AS knockdown on isoflurane-mediated proliferation, apoptosis and autophagy in Abeta-induced SK-N-SH and SK-N-AS cells.	mir-214-3p	14-24	BACE1 antisense RNA	Homo sapiens	61-69
32681443-12	2020	Additionally, miR-214-3p silencing reversed the influence of BACE1-AS knockdown on isoflurane-mediated proliferation, apoptosis and autophagy in Abeta-induced SK-N-SH and SK-N-AS cells.	mir-214-3p	14-24	amyloid beta precursor protein	Homo sapiens	145-150
32681443-13	2020	In conclusion, BACE1-AS aggravated isoflurane-induced neurotoxicity to AD via sponging miR-214-3p.	mir-214-3p	87-97	beta-secretase 1	Homo sapiens	15-20
31434695-0	2019	The deficiency of miR-214-3p exacerbates cardiac fibrosis via miR-214-3p/NLRC5 axis.	mir-214-3p	18-28	NLR family, CARD domain containing 5	Mus musculus	73-78
32714974-0	2020	miR-214-3p Attenuates Sepsis-Induced Myocardial Dysfunction in Mice by Inhibiting Autophagy through PTEN/AKT/mTOR Pathway.	mir-214-3p	0-10	phosphatase and tensin homolog	Mus musculus	100-104
32714974-0	2020	miR-214-3p Attenuates Sepsis-Induced Myocardial Dysfunction in Mice by Inhibiting Autophagy through PTEN/AKT/mTOR Pathway.	mir-214-3p	0-10	thymoma viral proto-oncogene 1	Mus musculus	105-108
32714974-0	2020	miR-214-3p Attenuates Sepsis-Induced Myocardial Dysfunction in Mice by Inhibiting Autophagy through PTEN/AKT/mTOR Pathway.	mir-214-3p	0-10	mechanistic target of rapamycin kinase	Mus musculus	109-113
32413870-12	2020	MiR-214-3p suppressed the malignant behaviors of colorectal cancer by regulating the PLAGL2/MYH9 axis.	mir-214-3p	0-10	PLAG1 like zinc finger 2	Homo sapiens	85-91
32413870-12	2020	MiR-214-3p suppressed the malignant behaviors of colorectal cancer by regulating the PLAGL2/MYH9 axis.	mir-214-3p	0-10	myosin heavy chain 9	Homo sapiens	92-96
32073886-8	2020	Furthermore, miR-214-3p inhibited the expression of irisin/FNDC5, and promoted the migration, invasion, and epithelial/mesenchymal transition (EMT) of U2OS cell through targeting FNDC5.	mir-214-3p	13-23	fibronectin type III domain containing 5	Homo sapiens	52-58
32073886-8	2020	Furthermore, miR-214-3p inhibited the expression of irisin/FNDC5, and promoted the migration, invasion, and epithelial/mesenchymal transition (EMT) of U2OS cell through targeting FNDC5.	mir-214-3p	13-23	fibronectin type III domain containing 5	Homo sapiens	59-64
32073886-8	2020	Furthermore, miR-214-3p inhibited the expression of irisin/FNDC5, and promoted the migration, invasion, and epithelial/mesenchymal transition (EMT) of U2OS cell through targeting FNDC5.	mir-214-3p	13-23	fibronectin type III domain containing 5	Homo sapiens	179-184
32073886-9	2020	Conclusions: Irisin/FNDC5 could inhibit the viability, migration, invasion, and EMT of osteosarcoma cells, and miR-214-3p could target FNDC5 to release its antitumor effects.	mir-214-3p	111-121	fibronectin type III domain containing 5	Homo sapiens	135-140