PMID-sentid	Pub_year	Sent_text	compound_name	comp_offset	prot_official_name	organism	prot_offset
28938540-6	2017	We then found that miR-128-3p caused translational inhibition of SPTAN1, reducing its protein level.	mir-128-3p	19-29	spectrin alpha, non-erythrocytic 1	Homo sapiens	65-71
26783552-11	2015	Furthermore loss- and gain-of-function studies identified miR-128-3p as a central modulator of the effects of EVs on PPAR-gamma inhibition and HSC activation.	mir-128-3p	58-68	peroxisome proliferator activated receptor gamma	Homo sapiens	117-127
34263426-2	2021	In this study, we postulated that NEAT1 may act as a miR-128-3p sponge.	mir-128-3p	53-63	nuclear paraspeckle assembly transcript 1	Homo sapiens	34-39
34916848-8	2021	Further study also stated that FOXD3-AS1 interacted with miR-128-3p and SZRD1 was the target gene of miR-128-3p.	mir-128-3p	57-67	forkhead box D3	Homo sapiens	31-36
34916848-8	2021	Further study also stated that FOXD3-AS1 interacted with miR-128-3p and SZRD1 was the target gene of miR-128-3p.	mir-128-3p	57-67	prostaglandin D2 receptor	Homo sapiens	37-40
34916848-8	2021	Further study also stated that FOXD3-AS1 interacted with miR-128-3p and SZRD1 was the target gene of miR-128-3p.	mir-128-3p	57-67	SUZ RNA binding domain containing 1	Homo sapiens	72-77
34916848-8	2021	Further study also stated that FOXD3-AS1 interacted with miR-128-3p and SZRD1 was the target gene of miR-128-3p.	mir-128-3p	101-111	forkhead box D3	Homo sapiens	31-36
34916848-8	2021	Further study also stated that FOXD3-AS1 interacted with miR-128-3p and SZRD1 was the target gene of miR-128-3p.	mir-128-3p	101-111	prostaglandin D2 receptor	Homo sapiens	37-40
34916848-8	2021	Further study also stated that FOXD3-AS1 interacted with miR-128-3p and SZRD1 was the target gene of miR-128-3p.	mir-128-3p	101-111	SUZ RNA binding domain containing 1	Homo sapiens	72-77
34916848-9	2021	Moreover, overexpression of miR-128-3p restrained the cell proliferation and metastasis of glioma, and reduced the SZRD1 level.	mir-128-3p	28-38	SUZ RNA binding domain containing 1	Homo sapiens	115-120
34916848-10	2021	Rescue assay illustrated that miR-128-3p inhibitor could reverse the suppressive impact of si-FOXD3-AS1 on the glioma progression.	mir-128-3p	30-40	forkhead box D3	Homo sapiens	94-99
34916848-10	2021	Rescue assay illustrated that miR-128-3p inhibitor could reverse the suppressive impact of si-FOXD3-AS1 on the glioma progression.	mir-128-3p	30-40	prostaglandin D2 receptor	Homo sapiens	100-103
34916848-11	2021	Similarly, SZRD1 overexpression could neutralize the influences of miR-128-3p mimic on glioma progression.	mir-128-3p	67-77	SUZ RNA binding domain containing 1	Homo sapiens	11-16
25962360-0	2015	miR-128-3p suppresses hepatocellular carcinoma proliferation by regulating PIK3R1 and is correlated with the prognosis of HCC patients.	mir-128-3p	0-10	phosphoinositide-3-kinase regulatory subunit 1	Homo sapiens	75-81
32797712-9	2020	Further testing in an in vivo middle cerebral artery occlusion (MCAO) mouse model of ischemic stroke showed that inhibiting miR-9-5p or miR-128-3p significantly decreases MCAO-induced infraction volume and inhibited apoptotic response as revealed by decreased cleaved Caspase-3 protein expression in immunohistochemical analysis.	mir-128-3p	136-146	caspase 3	Mus musculus	268-277
34718247-14	2021	miR-128-3p knockdown annulled the inhibitory effect of inhibiting lncRNA GAS5 on mitochondrial apoptosis.	mir-128-3p	0-10	growth arrest specific 5	Rattus norvegicus	73-77
34151664-7	2021	The PCR and western blotting showed that the expression levels of Bax was significantly inhibited by omentin via the upregulation of miR-128-3p at its 3"-UTR.	mir-128-3p	133-143	BCL2 associated X, apoptosis regulator	Rattus norvegicus	66-69
34895074-9	2021	miR-128-3p negatively regulated RUNX1 and subsequently downregulated multidrug resistance-associated protein 1 (MRP1).	mir-128-3p	0-10	RUNX family transcription factor 1	Homo sapiens	32-37
34895074-9	2021	miR-128-3p negatively regulated RUNX1 and subsequently downregulated multidrug resistance-associated protein 1 (MRP1).	mir-128-3p	0-10	ATP binding cassette subfamily C member 1	Homo sapiens	69-110
34895074-9	2021	miR-128-3p negatively regulated RUNX1 and subsequently downregulated multidrug resistance-associated protein 1 (MRP1).	mir-128-3p	0-10	ATP binding cassette subfamily C member 1	Homo sapiens	112-116
34895074-10	2021	Together, the present study indicates that RUNX1 confers TMZ resistance in GBM by upregulating MRP1, which is negatively regulated by miR-128-3p.	mir-128-3p	134-144	RUNX family transcription factor 1	Homo sapiens	43-48
34895074-10	2021	Together, the present study indicates that RUNX1 confers TMZ resistance in GBM by upregulating MRP1, which is negatively regulated by miR-128-3p.	mir-128-3p	134-144	ATP binding cassette subfamily C member 1	Homo sapiens	95-99
34895074-11	2021	Targeting miR-128-3p/RUNX1/MRP1 axis provides a potential strategy to overcome TMZ resistance in GBM.	mir-128-3p	10-20	RUNX family transcription factor 1	Homo sapiens	21-26
34895074-11	2021	Targeting miR-128-3p/RUNX1/MRP1 axis provides a potential strategy to overcome TMZ resistance in GBM.	mir-128-3p	10-20	ATP binding cassette subfamily C member 1	Homo sapiens	27-31
34592882-8	2021	Mechanistically, miR-128-3p, which targeted SIRT1, was hobbled by OIP5-AS1.	mir-128-3p	17-27	sirtuin 1	Rattus norvegicus	44-49
34592882-8	2021	Mechanistically, miR-128-3p, which targeted SIRT1, was hobbled by OIP5-AS1.	mir-128-3p	17-27	Opa interacting protein 5	Rattus norvegicus	66-74
34730612-6	2021	Logistic regression analysis was used to analyze the correlation between miR-9-5p and miR-128-3p levels and patient prognosis.	mir-128-3p	86-96	microRNA 95	Homo sapiens	73-81
34263426-7	2021	By binding to anti-oncogene miR-128-3p in the nucleus, NEAT1 enhanced tumorigenesis and glioma development.	mir-128-3p	28-38	nuclear paraspeckle assembly transcript 1	Homo sapiens	55-60
34263426-8	2021	Further experiments suggested that ITGA5 expression was increased in glioma tissues and was found to be connected with miR-128-3p.	mir-128-3p	119-129	integrin subunit alpha 5	Homo sapiens	35-40
34263426-9	2021	Additionally, NEAT1 facilitated ITGA5 expression via competitively binding to miR-128-3p.	mir-128-3p	78-88	nuclear paraspeckle assembly transcript 1	Homo sapiens	14-19
34263426-9	2021	Additionally, NEAT1 facilitated ITGA5 expression via competitively binding to miR-128-3p.	mir-128-3p	78-88	integrin subunit alpha 5	Homo sapiens	32-37
34493213-7	2021	The binding activity between miR-128-3p and circ-ABCB10 or zinc finger E-box binding homeobox 1 (ZEB1) was explored through pull-down assay or luciferase reporter assay.	mir-128-3p	29-39	ATP binding cassette subfamily B member 10	Homo sapiens	49-55
34533066-0	2022	SKA3, negatively regulated by miR-128-3p, promotes the progression of non-small-cell lung cancer.	mir-128-3p	30-40	spindle and kinetochore associated complex subunit 3	Mus musculus	0-4
34533066-8	2022	miR-128-3p repressed SKA3 expression by targeting it.	mir-128-3p	0-10	spindle and kinetochore associated complex subunit 3	Mus musculus	21-25
34533066-9	2022	Conclusion: miR-128-3p inhibited the progression of NSCLC through targeting SKA3.	mir-128-3p	12-22	spindle and kinetochore associated complex subunit 3	Mus musculus	76-80
34493213-7	2021	The binding activity between miR-128-3p and circ-ABCB10 or zinc finger E-box binding homeobox 1 (ZEB1) was explored through pull-down assay or luciferase reporter assay.	mir-128-3p	29-39	zinc finger E-box binding homeobox 1	Homo sapiens	59-95
34493213-7	2021	The binding activity between miR-128-3p and circ-ABCB10 or zinc finger E-box binding homeobox 1 (ZEB1) was explored through pull-down assay or luciferase reporter assay.	mir-128-3p	29-39	zinc finger E-box binding homeobox 1	Homo sapiens	97-101
34493213-12	2021	Loss of circ-ABCB10 inhibited ZEB1 expression by serving as a sponge of miR-128-3p in CC cells.	mir-128-3p	72-82	ATP binding cassette subfamily B member 10	Homo sapiens	13-19
34493213-12	2021	Loss of circ-ABCB10 inhibited ZEB1 expression by serving as a sponge of miR-128-3p in CC cells.	mir-128-3p	72-82	zinc finger E-box binding homeobox 1	Homo sapiens	30-34
34493213-13	2021	Circ-ABCB10 sponged miR-128-3p to enhance cell proliferation, invasion, EMT and inhibit apoptosis in CC cells.	mir-128-3p	20-30	ATP binding cassette subfamily B member 10	Homo sapiens	5-11
34331612-13	2021	HCP5 promoted cell proliferation and tumor formation of MM cells by activating the Wnt/beta-catenin/CCND1 signaling pathway by sponging miR-128-3p to increase PLAGL2 expression.	mir-128-3p	136-146	HLA complex P5	Homo sapiens	0-4
34368861-9	2021	The mechanistic study revealed that SNHG22 bound to miR-128-3p and attenuated its inhibitory effects on E2F transcription factor 3 (E2F3) expression levels and activity.	mir-128-3p	52-62	small nucleolar RNA host gene 22	Homo sapiens	36-42
34368861-9	2021	The mechanistic study revealed that SNHG22 bound to miR-128-3p and attenuated its inhibitory effects on E2F transcription factor 3 (E2F3) expression levels and activity.	mir-128-3p	52-62	E2F transcription factor 3	Homo sapiens	104-130
34368861-9	2021	The mechanistic study revealed that SNHG22 bound to miR-128-3p and attenuated its inhibitory effects on E2F transcription factor 3 (E2F3) expression levels and activity.	mir-128-3p	52-62	E2F transcription factor 3	Homo sapiens	132-136
34422901-19	2021	Conclusion: CircFTO promotes angiogenesis and impairs the blood-retinal barrier in vitro and in vivo by binding with miR-128-3p to upregulate TXNIP in DR.	mir-128-3p	117-127	thioredoxin interacting protein	Mus musculus	142-147
34331612-13	2021	HCP5 promoted cell proliferation and tumor formation of MM cells by activating the Wnt/beta-catenin/CCND1 signaling pathway by sponging miR-128-3p to increase PLAGL2 expression.	mir-128-3p	136-146	catenin beta 1	Homo sapiens	87-99
34331612-13	2021	HCP5 promoted cell proliferation and tumor formation of MM cells by activating the Wnt/beta-catenin/CCND1 signaling pathway by sponging miR-128-3p to increase PLAGL2 expression.	mir-128-3p	136-146	cyclin D1	Homo sapiens	100-105
34331612-13	2021	HCP5 promoted cell proliferation and tumor formation of MM cells by activating the Wnt/beta-catenin/CCND1 signaling pathway by sponging miR-128-3p to increase PLAGL2 expression.	mir-128-3p	136-146	PLAG1 like zinc finger 2	Homo sapiens	159-165
34236821-8	2021	MiR-128-3p reduced inflammation factor levels (tumor necrosis factor alpha, interleukin (IL)-6, IL-1beta, and IL-18).	mir-128-3p	0-10	tumor necrosis factor	Homo sapiens	47-74
34236821-8	2021	MiR-128-3p reduced inflammation factor levels (tumor necrosis factor alpha, interleukin (IL)-6, IL-1beta, and IL-18).	mir-128-3p	0-10	interleukin 1 alpha	Homo sapiens	96-104
34236821-8	2021	MiR-128-3p reduced inflammation factor levels (tumor necrosis factor alpha, interleukin (IL)-6, IL-1beta, and IL-18).	mir-128-3p	0-10	interleukin 18	Homo sapiens	110-115
34236821-9	2021	CONCLUSION: Thus, lncRNA AF131217.1 promoted inflammation in the regulated CSF via KLF4 by miR-128-3p.	mir-128-3p	91-101	Kruppel like factor 4	Homo sapiens	83-87
34108447-7	2021	Moreover, miR-128-3p was downregulated after Bmp1 overexpression, and upregulation of miR-128-3p reversed the effects of Bmp1 overexpression in IEC-6 cells.	mir-128-3p	10-20	bone morphogenetic protein 1	Rattus norvegicus	45-49
34173806-12	2021	TXNIP was the target gene of miR-128-3p and TXNIP overexpression abolished the miR-128-3p-mediated effects after HG treatment.	mir-128-3p	79-89	thioredoxin interacting protein	Homo sapiens	0-5
34173806-12	2021	TXNIP was the target gene of miR-128-3p and TXNIP overexpression abolished the miR-128-3p-mediated effects after HG treatment.	mir-128-3p	79-89	thioredoxin interacting protein	Homo sapiens	44-49
34306331-10	2021	Different from miR-128-3p mimic, miR-128-3p inhibitor promoted malignant phenotype of retinoblastoma cells, and partially reversed the inhibitory effect of siZNRD1-AS1.	mir-128-3p	15-25	RB transcriptional corepressor 1	Homo sapiens	86-100
34306331-10	2021	Different from miR-128-3p mimic, miR-128-3p inhibitor promoted malignant phenotype of retinoblastoma cells, and partially reversed the inhibitory effect of siZNRD1-AS1.	mir-128-3p	15-25	prostaglandin D2 receptor	Homo sapiens	164-167
34306331-10	2021	Different from miR-128-3p mimic, miR-128-3p inhibitor promoted malignant phenotype of retinoblastoma cells, and partially reversed the inhibitory effect of siZNRD1-AS1.	mir-128-3p	33-43	RB transcriptional corepressor 1	Homo sapiens	86-100
34306331-10	2021	Different from miR-128-3p mimic, miR-128-3p inhibitor promoted malignant phenotype of retinoblastoma cells, and partially reversed the inhibitory effect of siZNRD1-AS1.	mir-128-3p	33-43	prostaglandin D2 receptor	Homo sapiens	164-167
34306331-11	2021	MiR-128-3p mimic down-regulated BMI1, PNCA, N-Cadherin expressions, and up-regulated p16 and E-Cadherin expressions.	mir-128-3p	0-10	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	32-36
34306331-11	2021	MiR-128-3p mimic down-regulated BMI1, PNCA, N-Cadherin expressions, and up-regulated p16 and E-Cadherin expressions.	mir-128-3p	0-10	cadherin 2	Homo sapiens	44-54
34306331-11	2021	MiR-128-3p mimic down-regulated BMI1, PNCA, N-Cadherin expressions, and up-regulated p16 and E-Cadherin expressions.	mir-128-3p	0-10	cyclin dependent kinase inhibitor 2A	Homo sapiens	85-88
34306331-11	2021	MiR-128-3p mimic down-regulated BMI1, PNCA, N-Cadherin expressions, and up-regulated p16 and E-Cadherin expressions.	mir-128-3p	0-10	cadherin 1	Homo sapiens	93-103
34306331-12	2021	The regulatory effect of miR-128-3p was partially reversed by BMI1.	mir-128-3p	25-35	BMI1 proto-oncogene, polycomb ring finger	Homo sapiens	62-66
35634846-3	2022	Suppression of miR-128-3p may complement and enhance the adverse effects of TERT overexpression.	mir-128-3p	15-25	telomerase reverse transcriptase	Homo sapiens	76-80
35634846-4	2022	OBJECTIVE: The study aimed at evaluation of prognostic significance of the miR-128-3p/TERT expression in patients with primary neuroblastoma.	mir-128-3p	75-85	telomerase reverse transcriptase	Homo sapiens	86-90
34430706-5	2021	Pellino homolog 2 (PELI2) was predicted to be a direct target of miR-128-3p via luciferase reporter assay.	mir-128-3p	65-75	pellino 2	Mus musculus	0-17
34430706-5	2021	Pellino homolog 2 (PELI2) was predicted to be a direct target of miR-128-3p via luciferase reporter assay.	mir-128-3p	65-75	pellino 2	Mus musculus	19-24
34430706-12	2021	PEL12 upregulation in MPVECs alleviated miR-128-3p-induced caspase-3 activity inhibition and pro-inflammatory factor production.	mir-128-3p	40-50	caspase 3	Mus musculus	59-68
34092219-11	2021	Mechanistically, SNHG16 targeted miR-128-3p and attenuated its expression, while miR-128-3p targeted the 3" untranslated region of HMGB3.	mir-128-3p	81-91	high mobility group box 3	Rattus norvegicus	131-136
35358540-3	2022	MiR-128-3p was negatively regulated by SNHG16 and exerted anti-tumor effects.	mir-128-3p	0-10	small nucleolar RNA host gene 16	Homo sapiens	39-45
35358540-5	2022	SNHG16 competitively bound with miR-128-3p against CASC3, thus positively regulating CASC3 expression.	mir-128-3p	32-42	small nucleolar RNA host gene 16	Homo sapiens	0-6
35358540-5	2022	SNHG16 competitively bound with miR-128-3p against CASC3, thus positively regulating CASC3 expression.	mir-128-3p	32-42	CASC3 exon junction complex subunit	Homo sapiens	85-90
35587369-6	2022	Moreover, we confirmed that miR-128-3p directly targeted nuclear factor of activated T cell 5 (NFAT5), a protein that combines with osteoprotegerin and thus regulates osteoclastogenesis with the presence of the receptor activator of nuclear factor kappaB ligand.	mir-128-3p	28-38	nuclear factor of activated T cells 5	Mus musculus	57-93
35587369-6	2022	Moreover, we confirmed that miR-128-3p directly targeted nuclear factor of activated T cell 5 (NFAT5), a protein that combines with osteoprotegerin and thus regulates osteoclastogenesis with the presence of the receptor activator of nuclear factor kappaB ligand.	mir-128-3p	28-38	nuclear factor of activated T cells 5	Mus musculus	95-100
35587369-6	2022	Moreover, we confirmed that miR-128-3p directly targeted nuclear factor of activated T cell 5 (NFAT5), a protein that combines with osteoprotegerin and thus regulates osteoclastogenesis with the presence of the receptor activator of nuclear factor kappaB ligand.	mir-128-3p	28-38	tumor necrosis factor receptor superfamily, member 11b (osteoprotegerin)	Mus musculus	132-147
35587369-7	2022	Furthermore, we demonstrated that both the knockdown of KCNQ1OT1 and the overexpression of miR-128-3p attenuate the expression of NFAT5, while upregulating the osteoclastogenesis markers c-Fos, NFATc1, and Ctsk.	mir-128-3p	91-101	nuclear factor of activated T cells 5	Mus musculus	130-135
35587369-7	2022	Furthermore, we demonstrated that both the knockdown of KCNQ1OT1 and the overexpression of miR-128-3p attenuate the expression of NFAT5, while upregulating the osteoclastogenesis markers c-Fos, NFATc1, and Ctsk.	mir-128-3p	91-101	FBJ osteosarcoma oncogene	Mus musculus	187-192
35587369-7	2022	Furthermore, we demonstrated that both the knockdown of KCNQ1OT1 and the overexpression of miR-128-3p attenuate the expression of NFAT5, while upregulating the osteoclastogenesis markers c-Fos, NFATc1, and Ctsk.	mir-128-3p	91-101	nuclear factor of activated T cells, cytoplasmic, calcineurin dependent 1	Mus musculus	194-200
35587369-7	2022	Furthermore, we demonstrated that both the knockdown of KCNQ1OT1 and the overexpression of miR-128-3p attenuate the expression of NFAT5, while upregulating the osteoclastogenesis markers c-Fos, NFATc1, and Ctsk.	mir-128-3p	91-101	cathepsin K	Mus musculus	206-210
35587369-9	2022	Finally, we found that the inhibition of osteoclast differentiation by KCNQ1OT1 overexpression could be rescued using a miR-128-3p mimic, while the enhancement of migration and osteoclast differentiation by si-NFAT5 could be reversed with a miR-128-3p inhibitor.	mir-128-3p	120-130	KCNQ1 overlapping transcript 1	Mus musculus	71-79
35587369-9	2022	Finally, we found that the inhibition of osteoclast differentiation by KCNQ1OT1 overexpression could be rescued using a miR-128-3p mimic, while the enhancement of migration and osteoclast differentiation by si-NFAT5 could be reversed with a miR-128-3p inhibitor.	mir-128-3p	241-251	KCNQ1 overlapping transcript 1	Mus musculus	71-79
35587369-9	2022	Finally, we found that the inhibition of osteoclast differentiation by KCNQ1OT1 overexpression could be rescued using a miR-128-3p mimic, while the enhancement of migration and osteoclast differentiation by si-NFAT5 could be reversed with a miR-128-3p inhibitor.	mir-128-3p	241-251	nuclear factor of activated T cells 5	Mus musculus	210-215
34108447-7	2021	Moreover, miR-128-3p was downregulated after Bmp1 overexpression, and upregulation of miR-128-3p reversed the effects of Bmp1 overexpression in IEC-6 cells.	mir-128-3p	10-20	bone morphogenetic protein 1	Rattus norvegicus	121-125
34108447-7	2021	Moreover, miR-128-3p was downregulated after Bmp1 overexpression, and upregulation of miR-128-3p reversed the effects of Bmp1 overexpression in IEC-6 cells.	mir-128-3p	86-96	bone morphogenetic protein 1	Rattus norvegicus	121-125
34108447-8	2021	Phf6 was observed to be a target of miR-128-3p.	mir-128-3p	36-46	PHD finger protein 6	Rattus norvegicus	0-4
35587748-9	2022	Rescue experiments manifested that decreased miR-128-3p level reversed CRC cell inhibition by silencing LINC00467.	mir-128-3p	45-55	long intergenic non-protein coding RNA 467	Homo sapiens	104-113
35587748-10	2022	Furthermore, vascular endothelial growth factor C (VEGFC) was identified as a target of miR-128-3p that could reverse the inhibition of cell growth that is mediated by miR-128-3p.	mir-128-3p	88-98	vascular endothelial growth factor C	Homo sapiens	13-49
35587748-10	2022	Furthermore, vascular endothelial growth factor C (VEGFC) was identified as a target of miR-128-3p that could reverse the inhibition of cell growth that is mediated by miR-128-3p.	mir-128-3p	88-98	vascular endothelial growth factor C	Homo sapiens	51-56
35187800-5	2022	The binding between miR-128-3p and BNIP3P1 (or BNIP3) was explored by luciferase reporter assays.	mir-128-3p	20-30	BCL2 interacting protein 3 pseudogene 1	Homo sapiens	35-42
35187800-5	2022	The binding between miR-128-3p and BNIP3P1 (or BNIP3) was explored by luciferase reporter assays.	mir-128-3p	20-30	BCL2 interacting protein 3	Homo sapiens	47-52
35486287-0	2022	Targeting specificity protein 1 with miR-128-3p overcomes TGF-beta1 mediated epithelial-mesenchymal transition in breast cancer: An in vitro study.	mir-128-3p	37-47	Sp1 transcription factor	Homo sapiens	10-31
35486287-4	2022	Herein, we evaluated the role of miR-128-3p mimics in suppressing EMT of breast cancer cell lines by regulating the TGF-beta1/SP1 axis.	mir-128-3p	33-43	transforming growth factor beta 1	Homo sapiens	116-125
35486287-9	2022	miR-128-3p overexpression impeded the SP1 mediated EMT markers in TGF-beta1 stimulated cells by inhibiting the SP1 nuclear function.	mir-128-3p	0-10	transforming growth factor beta 1	Homo sapiens	66-75
35486287-10	2022	Further, treatment with miR-128-3p mimics significantly reduced the migration, invasion and spreading capability of TGF-beta1 stimulated cells.	mir-128-3p	24-34	transforming growth factor beta 1	Homo sapiens	116-125
35486287-12	2022	CONCLUSIONS: Upregulated miR-128-3p inhibited SP1, thereby limiting the TGF-beta1 induced EMT in MCF-7 and MDA-MB-231 cell lines for the first time.	mir-128-3p	25-35	transforming growth factor beta 1	Homo sapiens	72-81
35186455-7	2022	The binding relationship between miR-128-3p and ZEB1 was examined by bioinformatics analysis and dual-luciferase assay, and verified by RT-qPCR and Western blot.	mir-128-3p	33-43	zinc finger E-box binding homeobox 1	Homo sapiens	48-52
35252056-10	2022	Among eleven miRNA candidates, miR-128-3p displayed the most vigorous activity to negatively regulate c-Met and was downregulated in LR-HCC cells.	mir-128-3p	31-41	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	102-107
35252056-12	2022	MiR-128-3p and c-Met participate in the mechanisms underlying lenvatinib resistance through regulating Akt that mediates the apoptotic pathway and ERK (extracellular-signal-regulated kinase) modulating cell cycle progression.	mir-128-3p	0-10	AKT serine/threonine kinase 1	Homo sapiens	103-106
35252056-12	2022	MiR-128-3p and c-Met participate in the mechanisms underlying lenvatinib resistance through regulating Akt that mediates the apoptotic pathway and ERK (extracellular-signal-regulated kinase) modulating cell cycle progression.	mir-128-3p	0-10	mitogen-activated protein kinase 1	Homo sapiens	147-150
35252056-12	2022	MiR-128-3p and c-Met participate in the mechanisms underlying lenvatinib resistance through regulating Akt that mediates the apoptotic pathway and ERK (extracellular-signal-regulated kinase) modulating cell cycle progression.	mir-128-3p	0-10	mitogen-activated protein kinase 1	Homo sapiens	152-189
35186455-11	2022	PANC-1 cells transfected with miR-128-3p mimic or si-ZEB1 showed upregulated E-cadherin and downregulated N-cadherin, and transformed from mesenchymal phenotypes to epithelial phenotypes, with decreased invasion and migration, while opposite results occurred in AsPC-1 cells transfected with miR-128-3p inhibitor or oe-ZEB1.	mir-128-3p	30-40	cadherin 1	Homo sapiens	77-87
35186455-11	2022	PANC-1 cells transfected with miR-128-3p mimic or si-ZEB1 showed upregulated E-cadherin and downregulated N-cadherin, and transformed from mesenchymal phenotypes to epithelial phenotypes, with decreased invasion and migration, while opposite results occurred in AsPC-1 cells transfected with miR-128-3p inhibitor or oe-ZEB1.	mir-128-3p	30-40	cadherin 2	Homo sapiens	106-116
35186455-11	2022	PANC-1 cells transfected with miR-128-3p mimic or si-ZEB1 showed upregulated E-cadherin and downregulated N-cadherin, and transformed from mesenchymal phenotypes to epithelial phenotypes, with decreased invasion and migration, while opposite results occurred in AsPC-1 cells transfected with miR-128-3p inhibitor or oe-ZEB1.	mir-128-3p	30-40	zinc finger E-box binding homeobox 1	Homo sapiens	319-323
35186455-11	2022	PANC-1 cells transfected with miR-128-3p mimic or si-ZEB1 showed upregulated E-cadherin and downregulated N-cadherin, and transformed from mesenchymal phenotypes to epithelial phenotypes, with decreased invasion and migration, while opposite results occurred in AsPC-1 cells transfected with miR-128-3p inhibitor or oe-ZEB1.	mir-128-3p	292-302	zinc finger E-box binding homeobox 1	Homo sapiens	53-57
35186455-12	2022	miR-128-3p targeted ZEB1.	mir-128-3p	0-10	zinc finger E-box binding homeobox 1	Homo sapiens	20-24
35186455-13	2022	oe-ZEB1 antagonized the inhibition of miR-128-3p mimic on PANC-1 cell EMT, invasion, and migration, while si-ZEB1 reversed the facilitation of miR-128-3p inhibitor in AsPC-1 cells.	mir-128-3p	38-48	zinc finger E-box binding homeobox 1	Homo sapiens	3-7
35186455-13	2022	oe-ZEB1 antagonized the inhibition of miR-128-3p mimic on PANC-1 cell EMT, invasion, and migration, while si-ZEB1 reversed the facilitation of miR-128-3p inhibitor in AsPC-1 cells.	mir-128-3p	38-48	zinc finger E-box binding homeobox 1	Homo sapiens	109-113
35186455-13	2022	oe-ZEB1 antagonized the inhibition of miR-128-3p mimic on PANC-1 cell EMT, invasion, and migration, while si-ZEB1 reversed the facilitation of miR-128-3p inhibitor in AsPC-1 cells.	mir-128-3p	143-153	zinc finger E-box binding homeobox 1	Homo sapiens	109-113
35186455-14	2022	In conclusion, miR-128-3p inhibited PC cell EMT, invasion, and migration by targeting ZEB1.	mir-128-3p	15-25	zinc finger E-box binding homeobox 1	Homo sapiens	86-90
35088229-10	2022	Meanwhile, miR-128-3p was negatively associated with blood lipid level (LDL-C), inflammatory cytokines (TNF-alpha, IL-1beta, IL-6), cell adhesion molecules (VCAM-1, ICAM-1), and Gensini score (all P < 0.05) in CHD patients.	mir-128-3p	11-21	tumor necrosis factor	Homo sapiens	104-113
35130573-9	2022	The detection of luciferase reporter gene showed that lncRNA PVT1 targeted miR-128-3p, and there was a negative correlation in the serum of DR patients.	mir-128-3p	75-85	Pvt1 oncogene	Homo sapiens	61-65
35088229-10	2022	Meanwhile, miR-128-3p was negatively associated with blood lipid level (LDL-C), inflammatory cytokines (TNF-alpha, IL-1beta, IL-6), cell adhesion molecules (VCAM-1, ICAM-1), and Gensini score (all P < 0.05) in CHD patients.	mir-128-3p	11-21	interleukin 1 alpha	Homo sapiens	115-123
35088229-10	2022	Meanwhile, miR-128-3p was negatively associated with blood lipid level (LDL-C), inflammatory cytokines (TNF-alpha, IL-1beta, IL-6), cell adhesion molecules (VCAM-1, ICAM-1), and Gensini score (all P < 0.05) in CHD patients.	mir-128-3p	11-21	interleukin 6	Homo sapiens	125-129
35088229-10	2022	Meanwhile, miR-128-3p was negatively associated with blood lipid level (LDL-C), inflammatory cytokines (TNF-alpha, IL-1beta, IL-6), cell adhesion molecules (VCAM-1, ICAM-1), and Gensini score (all P < 0.05) in CHD patients.	mir-128-3p	11-21	vascular cell adhesion molecule 1	Homo sapiens	157-163
35088229-10	2022	Meanwhile, miR-128-3p was negatively associated with blood lipid level (LDL-C), inflammatory cytokines (TNF-alpha, IL-1beta, IL-6), cell adhesion molecules (VCAM-1, ICAM-1), and Gensini score (all P < 0.05) in CHD patients.	mir-128-3p	11-21	intercellular adhesion molecule 1	Homo sapiens	165-171
35088229-11	2022	CONCLUSION: Lnc-HULC and its target miR-128-3p relate to lipid level, stenosis degree, inflammatory cytokines, and cell adhesion molecules in CHD patients.	mir-128-3p	36-46	hepatocellular carcinoma up-regulated long non-coding RNA	Homo sapiens	16-20
34968167-7	2022	In addition, GC cells were cocultured with T lymphocytes, and the subsequent flow cytometric analysis showed that overexpression of miR-128-3p in tumor cells decreased the percentages of CD4+ CD25+ Foxp3+ Tregs by downregulating IL16 expression in GC, whereas miR-128-3p inhibition had the opposite effect.	mir-128-3p	132-142	CD4 molecule	Homo sapiens	187-190
34990303-4	2022	Additionally, miR-128-3p expression was negatively correlated with ZEB1.	mir-128-3p	14-24	zinc finger E-box binding homeobox 1	Homo sapiens	67-71
34990303-5	2022	miR-128-3p overexpression or ZEB1 silencing attenuated extracellular matrix degradation and cell apoptosis, and increased the proliferation of IL-1beta-activated CHON-001 cells.	mir-128-3p	0-10	interleukin 1 alpha	Homo sapiens	143-151
34990303-6	2022	Furthermore, ZEB1 was directly targeted by miR-128-3p.	mir-128-3p	43-53	zinc finger E-box binding homeobox 1	Homo sapiens	13-17
34990303-8	2022	Overall, our findings suggested that miR-128-3p might regulate the development of OA via targeting ZEB1.	mir-128-3p	37-47	zinc finger E-box binding homeobox 1	Homo sapiens	99-103
34968167-7	2022	In addition, GC cells were cocultured with T lymphocytes, and the subsequent flow cytometric analysis showed that overexpression of miR-128-3p in tumor cells decreased the percentages of CD4+ CD25+ Foxp3+ Tregs by downregulating IL16 expression in GC, whereas miR-128-3p inhibition had the opposite effect.	mir-128-3p	132-142	interleukin 2 receptor subunit alpha	Homo sapiens	192-196
34968167-7	2022	In addition, GC cells were cocultured with T lymphocytes, and the subsequent flow cytometric analysis showed that overexpression of miR-128-3p in tumor cells decreased the percentages of CD4+ CD25+ Foxp3+ Tregs by downregulating IL16 expression in GC, whereas miR-128-3p inhibition had the opposite effect.	mir-128-3p	132-142	forkhead box P3	Homo sapiens	198-203
34968167-7	2022	In addition, GC cells were cocultured with T lymphocytes, and the subsequent flow cytometric analysis showed that overexpression of miR-128-3p in tumor cells decreased the percentages of CD4+ CD25+ Foxp3+ Tregs by downregulating IL16 expression in GC, whereas miR-128-3p inhibition had the opposite effect.	mir-128-3p	132-142	interleukin 16	Homo sapiens	229-233
34968167-8	2022	Moreover, the recombinant IL16 reversed the effects of miR-128-3p overexpression, and a competitive antibody against the IL16 receptor CD4 also reversed the effects of miR-128-3p knockdown.	mir-128-3p	55-65	interleukin 16	Homo sapiens	26-30
34968167-8	2022	Moreover, the recombinant IL16 reversed the effects of miR-128-3p overexpression, and a competitive antibody against the IL16 receptor CD4 also reversed the effects of miR-128-3p knockdown.	mir-128-3p	55-65	CD4 molecule	Homo sapiens	135-138
34968167-8	2022	Moreover, the recombinant IL16 reversed the effects of miR-128-3p overexpression, and a competitive antibody against the IL16 receptor CD4 also reversed the effects of miR-128-3p knockdown.	mir-128-3p	168-178	interleukin 16	Homo sapiens	121-125
34968167-8	2022	Moreover, the recombinant IL16 reversed the effects of miR-128-3p overexpression, and a competitive antibody against the IL16 receptor CD4 also reversed the effects of miR-128-3p knockdown.	mir-128-3p	168-178	CD4 molecule	Homo sapiens	135-138
33611674-8	2021	The interaction between miR-128-3p and GAS5 or HDAC4 was predicted by ENCORI or TargetScan Human and verified by the dual-luciferase reporter, RNA Immunoprecipitation (RIP), and RNA pull-down assays.	mir-128-3p	24-34	growth arrest specific 5	Homo sapiens	39-43
35110470-0	2022	The protective effects of miR-128-3p on sevoflurane-induced progressive neurotoxicity in rats by targeting NOVA1.	mir-128-3p	26-36	NOVA alternative splicing regulator 1	Rattus norvegicus	107-112
35110470-7	2022	Dual luciferase assay was used to identify the targeted binding relationship between miR-128-3p and NOVA1.	mir-128-3p	85-95	NOVA alternative splicing regulator 1	Rattus norvegicus	100-105
35110470-13	2022	Overexpressed miR-128-3p partially reversed the role of sevoflurane treatment in triggering cell apoptosis, enhancing the expression of Bax and cleaved caspase-3 and inhibiting Bcl-2 expression obviously.	mir-128-3p	14-24	BCL2 associated X, apoptosis regulator	Rattus norvegicus	136-139
35110470-13	2022	Overexpressed miR-128-3p partially reversed the role of sevoflurane treatment in triggering cell apoptosis, enhancing the expression of Bax and cleaved caspase-3 and inhibiting Bcl-2 expression obviously.	mir-128-3p	14-24	caspase 3	Rattus norvegicus	152-161
35110470-13	2022	Overexpressed miR-128-3p partially reversed the role of sevoflurane treatment in triggering cell apoptosis, enhancing the expression of Bax and cleaved caspase-3 and inhibiting Bcl-2 expression obviously.	mir-128-3p	14-24	BCL2, apoptosis regulator	Rattus norvegicus	177-182
35110470-14	2022	Overexpressed miR-128-3p partially reversed the role of sevoflurane treatment in promoting the expression of NOX1and NOX4, and inflammatory cytokine levels by targeting with NOVA1.	mir-128-3p	14-24	NADPH oxidase 1	Rattus norvegicus	109-113
35110470-14	2022	Overexpressed miR-128-3p partially reversed the role of sevoflurane treatment in promoting the expression of NOX1and NOX4, and inflammatory cytokine levels by targeting with NOVA1.	mir-128-3p	14-24	NADPH oxidase 4	Rattus norvegicus	117-121
35110470-14	2022	Overexpressed miR-128-3p partially reversed the role of sevoflurane treatment in promoting the expression of NOX1and NOX4, and inflammatory cytokine levels by targeting with NOVA1.	mir-128-3p	14-24	NOVA alternative splicing regulator 1	Rattus norvegicus	174-179
35110470-15	2022	MiR-128-3p might be a potential therapeutic target for the prevention or treatment of sevoflurane-induced neurotoxicity by targeting with NOVA1.	mir-128-3p	0-10	NOVA alternative splicing regulator 1	Rattus norvegicus	138-143
33611674-8	2021	The interaction between miR-128-3p and GAS5 or HDAC4 was predicted by ENCORI or TargetScan Human and verified by the dual-luciferase reporter, RNA Immunoprecipitation (RIP), and RNA pull-down assays.	mir-128-3p	24-34	histone deacetylase 4	Homo sapiens	47-52
33611674-11	2021	Rescue assay confirmed that miR-128-3p overexpression or HDAC4 knockdown weakened the inhibitory effect of GAS5 or anti-miR-128-3p on RA development.	mir-128-3p	28-38	growth arrest specific 5	Homo sapiens	107-111
33611674-11	2021	Rescue assay confirmed that miR-128-3p overexpression or HDAC4 knockdown weakened the inhibitory effect of GAS5 or anti-miR-128-3p on RA development.	mir-128-3p	120-130	histone deacetylase 4	Homo sapiens	57-62
34007135-11	2021	Mechanistically, TP73-AS1 was validated to promote PC progression through GOLM1 upregulation by competitively binding to miR-128-3p.	mir-128-3p	121-131	tumor protein p73	Homo sapiens	17-21
34042286-5	2021	Effect of miR-128-3p overexpression on levels of TNF-alpha, IL-1beta, ICAM-1, and MCP-1 was tested by ELISA assay.	mir-128-3p	10-20	mast cell protease 1-like 1	Rattus norvegicus	82-87
34042286-11	2021	By western blot, we discovered miR-128-3p activates NF-kappaB by targeting TRAF6.	mir-128-3p	31-41	TNF receptor associated factor 6	Rattus norvegicus	75-80
34042286-12	2021	Our data reveal a novel mechanism that a decreased miR-128-3p level in TNBS-induced colitis could inhibit production of inflammatory factors, which activates NF-kappaB signaling by targeting TRAF6.	mir-128-3p	51-61	TNF receptor associated factor 6	Rattus norvegicus	191-196
34051661-0	2021	LncRNA OIP5-AS1 inhibits ferroptosis in prostate cancer with long-term cadmium exposure through miR-128-3p/SLC7A11 signaling.	mir-128-3p	96-106	OIP5 antisense RNA 1	Homo sapiens	7-15
34051661-8	2021	Mechanistically, we demonstrated that OIP5-AS1 served as an endogenous sponge of miR-128-3p to regulate the expression of SLC7A11, a surrogate marker of ferroptosis.	mir-128-3p	81-91	OIP5 antisense RNA 1	Homo sapiens	38-46
34051661-8	2021	Mechanistically, we demonstrated that OIP5-AS1 served as an endogenous sponge of miR-128-3p to regulate the expression of SLC7A11, a surrogate marker of ferroptosis.	mir-128-3p	81-91	solute carrier family 7 member 11	Homo sapiens	122-129
34042286-0	2021	MiR-128-3p alleviates TNBS-induced colitis through inactivating TRAF6/NF-kappaB signaling pathway in rats.	mir-128-3p	0-10	TNF receptor associated factor 6	Rattus norvegicus	64-69
34042286-5	2021	Effect of miR-128-3p overexpression on levels of TNF-alpha, IL-1beta, ICAM-1, and MCP-1 was tested by ELISA assay.	mir-128-3p	10-20	tumor necrosis factor	Rattus norvegicus	49-58
34042286-5	2021	Effect of miR-128-3p overexpression on levels of TNF-alpha, IL-1beta, ICAM-1, and MCP-1 was tested by ELISA assay.	mir-128-3p	10-20	interleukin 1 alpha	Rattus norvegicus	60-68
34042286-5	2021	Effect of miR-128-3p overexpression on levels of TNF-alpha, IL-1beta, ICAM-1, and MCP-1 was tested by ELISA assay.	mir-128-3p	10-20	intercellular adhesion molecule 1	Rattus norvegicus	70-76
34007135-11	2021	Mechanistically, TP73-AS1 was validated to promote PC progression through GOLM1 upregulation by competitively binding to miR-128-3p.	mir-128-3p	121-131	prostaglandin D2 receptor	Homo sapiens	22-25
34007135-11	2021	Mechanistically, TP73-AS1 was validated to promote PC progression through GOLM1 upregulation by competitively binding to miR-128-3p.	mir-128-3p	121-131	golgi membrane protein 1	Homo sapiens	74-79
33867828-5	2021	Additionally, miR-128-3p expression was decreased and negatively correlated with SIRT1 level in mouse CaOx nephrocalcinosis model following TF treatment.	mir-128-3p	14-24	sirtuin 1	Mus musculus	81-86
33760158-8	2021	Dual-luciferase reporter assays were conducted to verify the binding between miR-128-3p and FOXD3-AS1.	mir-128-3p	77-87	forkhead box D3	Homo sapiens	92-97
33760158-8	2021	Dual-luciferase reporter assays were conducted to verify the binding between miR-128-3p and FOXD3-AS1.	mir-128-3p	77-87	prostaglandin D2 receptor	Homo sapiens	98-101
33760158-12	2021	Moreover, the results demonstrated that FOXD3-AS1 targeted and negatively regulated miR-128-3p, which indirectly upregulated LIMK1 expression.	mir-128-3p	84-94	forkhead box D3	Homo sapiens	40-45
33760158-12	2021	Moreover, the results demonstrated that FOXD3-AS1 targeted and negatively regulated miR-128-3p, which indirectly upregulated LIMK1 expression.	mir-128-3p	84-94	prostaglandin D2 receptor	Homo sapiens	46-49
33760158-12	2021	Moreover, the results demonstrated that FOXD3-AS1 targeted and negatively regulated miR-128-3p, which indirectly upregulated LIMK1 expression.	mir-128-3p	84-94	LIM domain kinase 1	Homo sapiens	125-130
33760158-13	2021	Therefore, the present study demonstrated that FOXD3-AS1 upregulated LIMK1 expression via competitively sponging miR-128-3p in CC cells, promoting CC progression.	mir-128-3p	113-123	forkhead box D3	Homo sapiens	47-52
33760158-13	2021	Therefore, the present study demonstrated that FOXD3-AS1 upregulated LIMK1 expression via competitively sponging miR-128-3p in CC cells, promoting CC progression.	mir-128-3p	113-123	prostaglandin D2 receptor	Homo sapiens	53-56
33760158-13	2021	Therefore, the present study demonstrated that FOXD3-AS1 upregulated LIMK1 expression via competitively sponging miR-128-3p in CC cells, promoting CC progression.	mir-128-3p	113-123	LIM domain kinase 1	Homo sapiens	69-74
33991762-0	2021	MiR-128-3p suppresses tumor proliferation and metastasis via targeting CDC6 in hepatocellular carcinoma cells.	mir-128-3p	0-10	cell division cycle 6	Homo sapiens	71-75
33991762-5	2021	The potential binding sites of miR-128-3p on CDC6 were predicted with Targetscan 7.2 and confirmed by dual-luciferase reporter assay.	mir-128-3p	31-41	cell division cycle 6	Homo sapiens	45-49
33991762-11	2021	CDC6 was targeted by miR-128-3p and had higher expression in HCC tissue.	mir-128-3p	21-31	cell division cycle 6	Homo sapiens	0-4
33991762-12	2021	The promotive effects of overexpressed CDC6 on HCC cell viability, migration and invasion were reversed by up-regulating miR-128-3p.	mir-128-3p	121-131	cell division cycle 6	Homo sapiens	39-43
33991762-14	2021	CONCLUSION: MiR-128-3p may suppress HCC cell proliferation and metastasis via targeting CDC6.	mir-128-3p	12-22	cell division cycle 6	Homo sapiens	88-92
33986789-0	2021	Inhibition of miR-128-3p Attenuated Doxorubicin-Triggered Acute Cardiac Injury in Mice by the Regulation of PPAR-gamma.	mir-128-3p	14-24	peroxisome proliferator activated receptor gamma	Mus musculus	108-118
33986789-12	2021	Moreover, the protective effects provided by miR-128-3p inhibition were abolished by a PPAR-gamma antagonist in vivo and in vitro.	mir-128-3p	45-55	peroxisome proliferator activated receptor gamma	Mus musculus	87-97
33867828-8	2021	In addition, miR-128-3p inhibited SIRT1 expression by directly binding its 3"-untranslated region (UTR).	mir-128-3p	13-23	sirtuin 1	Mus musculus	34-39
33089633-12	2021	Downregulated miR-128-3p expression induces the apoptosis and inhibits the proliferation of spermatogenic cells by promoting MAPK14 phosphorylation.	mir-128-3p	14-24	mitogen-activated protein kinase 14	Mus musculus	125-131
33089633-0	2021	The role of miR-128-3p through MAPK14 activation in the apoptosis of GC2 spermatocyte cell line following heat stress.	mir-128-3p	12-22	mitogen-activated protein kinase 14	Mus musculus	31-37
33089633-0	2021	The role of miR-128-3p through MAPK14 activation in the apoptosis of GC2 spermatocyte cell line following heat stress.	mir-128-3p	12-22	guanylate cyclase 2f	Mus musculus	69-72
33089633-3	2021	OBJECTIVES: This study aimed to verify whether the target gene of miR-128-3p is MAPK14, which affects spermatogenic cell proliferation and apoptosis under testicular hyperthermia.	mir-128-3p	66-76	mitogen-activated protein kinase 14	Mus musculus	80-86
33089633-9	2021	In the GC2-spd cell line in vitro, miR-128-3p inhibitors were found to upregulate p-MAPK14 expression, reduce cell proliferation activity, and increase apoptosis, consistent with the results obtained in the heat treatment alone.	mir-128-3p	35-45	guanylate cyclase 2f	Mus musculus	7-10
33089633-9	2021	In the GC2-spd cell line in vitro, miR-128-3p inhibitors were found to upregulate p-MAPK14 expression, reduce cell proliferation activity, and increase apoptosis, consistent with the results obtained in the heat treatment alone.	mir-128-3p	35-45	mitogen-activated protein kinase 14	Mus musculus	84-90
33089633-10	2021	Furthermore, miR-128-3p mimics transfected in the GC2 cells after heat stress reduced p-MAPK14 expression, alleviated the decrease in cell proliferation, and decreased the apoptosis level.	mir-128-3p	13-23	guanylate cyclase 2f	Mus musculus	50-53
33089633-10	2021	Furthermore, miR-128-3p mimics transfected in the GC2 cells after heat stress reduced p-MAPK14 expression, alleviated the decrease in cell proliferation, and decreased the apoptosis level.	mir-128-3p	13-23	mitogen-activated protein kinase 14	Mus musculus	88-94
33867828-9	2021	Furthermore, miR-128-3p activation partially reversed the acceerative effect of TF on SIRT1 expression.	mir-128-3p	13-23	sirtuin 1	Mus musculus	86-91
33373887-14	2021	SUMMARY: NEAT1 affects AQP4 signaling pathway to alleviate the spinal cord injury-induced NP via promoting miR-128-3p expression.	mir-128-3p	107-117	aquaporin 4	Rattus norvegicus	23-27
33552270-0	2021	miR-128-3p serves as an oncogenic microRNA in osteosarcoma cells by downregulating ZC3H12D.	mir-128-3p	0-10	zinc finger CCCH-type containing 12D	Homo sapiens	83-90
33634112-0	2021	Exosomal miR-128-3p Promotes Epithelial-to-Mesenchymal Transition in Colorectal Cancer Cells by Targeting FOXO4 via TGF-beta/SMAD and JAK/STAT3 Signaling.	mir-128-3p	9-19	forkhead box O4	Homo sapiens	106-111
33634112-0	2021	Exosomal miR-128-3p Promotes Epithelial-to-Mesenchymal Transition in Colorectal Cancer Cells by Targeting FOXO4 via TGF-beta/SMAD and JAK/STAT3 Signaling.	mir-128-3p	9-19	transforming growth factor alpha	Homo sapiens	116-124
33634112-0	2021	Exosomal miR-128-3p Promotes Epithelial-to-Mesenchymal Transition in Colorectal Cancer Cells by Targeting FOXO4 via TGF-beta/SMAD and JAK/STAT3 Signaling.	mir-128-3p	9-19	signal transducer and activator of transcription 3	Homo sapiens	138-143
33634112-7	2021	Mechanistically, miR-128-3p overexpression downregulated the expression of FOXO4 and induced the activation of TGF-beta/SMAD and JAK/STAT3 signaling in CRC cells and xenografted tumors, which led to EMT.	mir-128-3p	17-27	forkhead box O4	Homo sapiens	75-80
33634112-7	2021	Mechanistically, miR-128-3p overexpression downregulated the expression of FOXO4 and induced the activation of TGF-beta/SMAD and JAK/STAT3 signaling in CRC cells and xenografted tumors, which led to EMT.	mir-128-3p	17-27	transforming growth factor alpha	Homo sapiens	111-119
33634112-7	2021	Mechanistically, miR-128-3p overexpression downregulated the expression of FOXO4 and induced the activation of TGF-beta/SMAD and JAK/STAT3 signaling in CRC cells and xenografted tumors, which led to EMT.	mir-128-3p	17-27	signal transducer and activator of transcription 3	Homo sapiens	133-138
33623823-0	2021	miR-128-3p inhibits apoptosis and inflammation in LPS-induced sepsis by targeting TGFBR2.	mir-128-3p	0-10	transforming growth factor beta receptor 2	Homo sapiens	82-88
33623823-12	2021	Moreover, TGFBR2 was a direct target of miR-128-3p, and its overexpression reversed the inhibitory effects of miR-128-3p overexpression on inflammation and apoptosis in LPS-induced HK2 cells.	mir-128-3p	40-50	transforming growth factor beta receptor 2	Homo sapiens	10-16
33623823-12	2021	Moreover, TGFBR2 was a direct target of miR-128-3p, and its overexpression reversed the inhibitory effects of miR-128-3p overexpression on inflammation and apoptosis in LPS-induced HK2 cells.	mir-128-3p	110-120	transforming growth factor beta receptor 2	Homo sapiens	10-16
33623823-13	2021	Besides, overexpression of miR-128-3p downregulated TGFBR2 to suppress the activation of the Smad signaling pathway.	mir-128-3p	27-37	transforming growth factor beta receptor 2	Homo sapiens	52-58
33623823-14	2021	Conclusion: miR-128-3p could inhibit apoptosis and inflammation by targeting TGFBR2 in LPS-induced HK2 cells, which might provide therapeutic strategy for the treatment of sepsis.	mir-128-3p	12-22	transforming growth factor beta receptor 2	Homo sapiens	77-83
33552270-9	2021	The results demonstrated that miR-128-3p directly targeted ZC3H12D and downregulated its expression, thereby promoting cell proliferation and migration.	mir-128-3p	30-40	zinc finger CCCH-type containing 12D	Homo sapiens	59-66
33575204-4	2020	Our current survey disclosed a complementary binding between miR-128-3p and the NTRK3 3" untranslated regions (3"-UTR), while luciferase activities of NTRK3 3"-UTR were restrained by miR-128-3p in 293T cells.	mir-128-3p	61-71	neurotrophic receptor tyrosine kinase 3	Homo sapiens	80-85
33575204-4	2020	Our current survey disclosed a complementary binding between miR-128-3p and the NTRK3 3" untranslated regions (3"-UTR), while luciferase activities of NTRK3 3"-UTR were restrained by miR-128-3p in 293T cells.	mir-128-3p	61-71	neurotrophic receptor tyrosine kinase 3	Homo sapiens	151-156
33575204-4	2020	Our current survey disclosed a complementary binding between miR-128-3p and the NTRK3 3" untranslated regions (3"-UTR), while luciferase activities of NTRK3 3"-UTR were restrained by miR-128-3p in 293T cells.	mir-128-3p	183-193	neurotrophic receptor tyrosine kinase 3	Homo sapiens	80-85
33575204-4	2020	Our current survey disclosed a complementary binding between miR-128-3p and the NTRK3 3" untranslated regions (3"-UTR), while luciferase activities of NTRK3 3"-UTR were restrained by miR-128-3p in 293T cells.	mir-128-3p	183-193	neurotrophic receptor tyrosine kinase 3	Homo sapiens	151-156
33575204-7	2020	These findings established that miR-128-3p can function as a tumor suppressor by inhibiting carcinogenesis of the oncogene, NTRK3.	mir-128-3p	32-42	neurotrophic receptor tyrosine kinase 3	Homo sapiens	124-129
33074219-13	2021	CONCLUSION: GAS5 knockdown promoted the growth of ox-LDL-induced THP-1 cells through down-modulating FBLN2 and increasing miR-128-3p, suggesting the potential value of GAS5 for treatment of AS.	mir-128-3p	122-132	growth arrest specific 5	Homo sapiens	12-16
33147048-15	2021	To conclude, the circ-FBXW7 delivery by exosomes could ameliorate chemoresistance to oxaliplatin in CRC by directly binding to miR-128-3p, suggesting a promising therapeutic strategy for oxaliplatin-resistant CRC patients.	mir-128-3p	127-137	F-box and WD repeat domain containing 7	Homo sapiens	22-27
33424542-3	2020	Herein, whether miR-128-3p contributes to spinal cord I/R injury by regulating specificity protein 1 (SP1) was assessed.	mir-128-3p	16-26	Sp1 transcription factor	Rattus norvegicus	79-100
33174052-14	2020	These findings indicated that miR-128-3p may inhibit the osteoblast differentiation of BM-MSCs by downregulation of these 6 genes, particularly RUNX1, YWHAB and NTRK2.	mir-128-3p	30-40	RUNX family transcription factor 1	Rattus norvegicus	144-149
33437366-0	2020	SCAMP3 is regulated by miR-128-3p and promotes the metastasis of hepatocellular carcinoma cells through EGFR-MAPK p38 signaling pathway.	mir-128-3p	23-33	secretory carrier membrane protein 3	Homo sapiens	0-6
33437366-8	2020	SCAMP3 up-regulation or miR-128-3p down-regulation could promote HCC cell proliferation, migration, invasion, and transcription and protein levels of EGFR, p-EGFR, MAPK p38, p-MAPK p38, N-cadherin and vimentin, and inhibit HCC cell apoptosis and transcription and protein levels of E-cadherin.	mir-128-3p	24-34	epidermal growth factor receptor	Homo sapiens	150-154
33437366-8	2020	SCAMP3 up-regulation or miR-128-3p down-regulation could promote HCC cell proliferation, migration, invasion, and transcription and protein levels of EGFR, p-EGFR, MAPK p38, p-MAPK p38, N-cadherin and vimentin, and inhibit HCC cell apoptosis and transcription and protein levels of E-cadherin.	mir-128-3p	24-34	epidermal growth factor receptor	Homo sapiens	158-162
33437366-12	2020	CONCLUSION: SCAMP3 can be controlled by miR-128-3p and can mediate the EGFR-MAPK p38 signaling pathway to inhibit HCC cell metastasis, which is expected to become a promising therapeutic target for HCC.	mir-128-3p	40-50	secretory carrier membrane protein 3	Mus musculus	12-18
33296418-12	2020	Transfection with miR-128-3p mimics, but not the control, significantly mitigated the Peg13 or Sox13-regulated luciferase expression in 293T cells.	mir-128-3p	18-28	paternally expressed 13	Homo sapiens	86-91
33296418-12	2020	Transfection with miR-128-3p mimics, but not the control, significantly mitigated the Peg13 or Sox13-regulated luciferase expression in 293T cells.	mir-128-3p	18-28	SRY-box transcription factor 13	Homo sapiens	95-100
33296418-13	2020	Peg13 over-expression significantly reduced the sevoflurane-related neurotoxicity and increased Sox13 expression in NSCs, which were mitigated by miR-128-3p transfection.	mir-128-3p	146-156	paternally expressed 13	Homo sapiens	0-5
33296418-14	2020	CONCLUSION: Such data indicated that Peg13 mitigated the sevoflurane-related neurotoxicity by sponging miR-128-3p to preserve Sox13 expression in NSCs.	mir-128-3p	103-113	paternally expressed 13	Homo sapiens	37-42
32556677-6	2020	The regulatory actions of miR-128-3p on both MIAT and PELI3 were interrogated by luciferase reporter assay.	mir-128-3p	26-36	myocardial infarction associated transcript	Homo sapiens	45-49
32556677-6	2020	The regulatory actions of miR-128-3p on both MIAT and PELI3 were interrogated by luciferase reporter assay.	mir-128-3p	26-36	pellino E3 ubiquitin protein ligase family member 3	Homo sapiens	54-59
32556677-8	2020	We further uncovered the negative correlation among MIAT, PELI3, and miR-128-3p.	mir-128-3p	69-79	myocardial infarction associated transcript	Homo sapiens	52-56
32556677-10	2020	Application of miR-128-3p inhibitor significantly stimulated luciferase activities driven by both MIAT and PELI3 promoter and phenotypically promoted cell viability, proliferation, migration, and invasion.	mir-128-3p	15-25	myocardial infarction associated transcript	Homo sapiens	98-102
32556677-10	2020	Application of miR-128-3p inhibitor significantly stimulated luciferase activities driven by both MIAT and PELI3 promoter and phenotypically promoted cell viability, proliferation, migration, and invasion.	mir-128-3p	15-25	pellino E3 ubiquitin protein ligase family member 3	Homo sapiens	107-112
32090639-0	2020	Nanocomplexes loaded with miR-128-3p for enhancing chemotherapy effect of colorectal cancer through dual-targeting silence the activity of PI3K/AKT and MEK/ERK pathway.	mir-128-3p	26-36	AKT serine/threonine kinase 1	Homo sapiens	144-147
32090639-0	2020	Nanocomplexes loaded with miR-128-3p for enhancing chemotherapy effect of colorectal cancer through dual-targeting silence the activity of PI3K/AKT and MEK/ERK pathway.	mir-128-3p	26-36	mitogen-activated protein kinase kinase 7	Homo sapiens	152-155
32090639-0	2020	Nanocomplexes loaded with miR-128-3p for enhancing chemotherapy effect of colorectal cancer through dual-targeting silence the activity of PI3K/AKT and MEK/ERK pathway.	mir-128-3p	26-36	mitogen-activated protein kinase 1	Homo sapiens	156-159
33174038-10	2020	HULC knockdown protected HMEC-1 cells from LPS-induced injury by upregulating miR-128-3p.	mir-128-3p	78-88	hepatocellular carcinoma up-regulated long non-coding RNA	Homo sapiens	0-4
33174052-14	2020	These findings indicated that miR-128-3p may inhibit the osteoblast differentiation of BM-MSCs by downregulation of these 6 genes, particularly RUNX1, YWHAB and NTRK2.	mir-128-3p	30-40	tyrosine 3-monooxygenase/tryptophan 5-monooxygenase activation protein, beta	Rattus norvegicus	151-156
33174052-14	2020	These findings indicated that miR-128-3p may inhibit the osteoblast differentiation of BM-MSCs by downregulation of these 6 genes, particularly RUNX1, YWHAB and NTRK2.	mir-128-3p	30-40	neurotrophic receptor tyrosine kinase 2	Rattus norvegicus	161-166
32500307-0	2020	miR-128-3p Inhibits NRP1 Expression and Promotes Inflammatory Response to Acute Kidney Injury in Sepsis.	mir-128-3p	0-10	neuropilin 1	Mus musculus	20-24
33238881-0	2020	MiR-128-3p inhibits vascular smooth muscle cell proliferation and migration by repressing FOXO4/MMP9 signaling pathway.	mir-128-3p	0-10	forkhead box O4	Mus musculus	90-95
33238881-0	2020	MiR-128-3p inhibits vascular smooth muscle cell proliferation and migration by repressing FOXO4/MMP9 signaling pathway.	mir-128-3p	0-10	matrix metallopeptidase 9	Mus musculus	96-100
33238881-10	2020	Further experiments confirmed the interaction between miR-128-3p and FOXO4.	mir-128-3p	54-64	forkhead box O4	Mus musculus	69-74
33238881-11	2020	Augmentation of FOXO4 or MMP9 reversed the effects of miR-128-3p.	mir-128-3p	54-64	forkhead box O4	Mus musculus	16-21
33238881-11	2020	Augmentation of FOXO4 or MMP9 reversed the effects of miR-128-3p.	mir-128-3p	54-64	matrix metallopeptidase 9	Mus musculus	25-29
33238881-13	2020	CONCLUSION: MiR-128-3p repressed the proliferation and migration of VSMCs through inhibiting the expressions of FOXO4 and MMP9.	mir-128-3p	12-22	forkhead box O4	Mus musculus	112-117
33238881-13	2020	CONCLUSION: MiR-128-3p repressed the proliferation and migration of VSMCs through inhibiting the expressions of FOXO4 and MMP9.	mir-128-3p	12-22	matrix metallopeptidase 9	Mus musculus	122-126
32958647-4	2020	Selective in vivo inhibition of miR-128-3p in FMRP-deficient astroglia sufficiently rescues decreased mGluR5 function, while astroglial overexpression of miR-128-3p strongly and selectively diminishes developmental astroglial mGluR5 signaling.	mir-128-3p	32-42	glutamate receptor, ionotropic, kainate 1	Mus musculus	102-108
33116599-12	2020	Bioinformatics analysis and luciferase reporter assay were proceeded to clarify whether miR-128-3p directly binds with lncRNA DLEU2.	mir-128-3p	88-98	deleted in lymphocytic leukemia, 2	Mus musculus	126-131
33116599-16	2020	Further, LncRNA DLEU2 is one of the targets of miR-128-3p.	mir-128-3p	47-57	deleted in lymphocytic leukemia, 2	Mus musculus	16-21
33116599-17	2020	miR-128-3p inhibitor abrogated the cell proliferation suppressed by knockdown of DLEU2, apoptosis induced by knockdown of DLEU2 and reversed the expression of cell cycle hallmarks regulated by knockdown of DLEU2.	mir-128-3p	0-10	deleted in lymphocytic leukemia, 2	Mus musculus	81-86
33116599-17	2020	miR-128-3p inhibitor abrogated the cell proliferation suppressed by knockdown of DLEU2, apoptosis induced by knockdown of DLEU2 and reversed the expression of cell cycle hallmarks regulated by knockdown of DLEU2.	mir-128-3p	0-10	deleted in lymphocytic leukemia, 2	Mus musculus	122-127
33116599-17	2020	miR-128-3p inhibitor abrogated the cell proliferation suppressed by knockdown of DLEU2, apoptosis induced by knockdown of DLEU2 and reversed the expression of cell cycle hallmarks regulated by knockdown of DLEU2.	mir-128-3p	0-10	deleted in lymphocytic leukemia, 2	Mus musculus	122-127
33116599-18	2020	Conclusion: Taken together, these results suggested knockdown of DLEU2 inhibited cervical cancer progression via targeting miR-128-3p.	mir-128-3p	123-133	deleted in lymphocytic leukemia, 2	Mus musculus	65-70
32500307-10	2020	miR-128-3p targets NRP1 for cell degradation, promotes inflammatory cell infiltration, increases expression of inflammatory factors, decreases renal cell viability, and increases apoptosis in LPS-induced septic acute renal injury.	mir-128-3p	0-10	neuropilin 1	Mus musculus	19-23
32663367-7	2020	The interaction between miR-128-3p and LINC01005 or Kruppel-like factor 4 (KLF4) was analyzed by luciferase reporter assay.	mir-128-3p	24-34	long intergenic non-protein coding RNA 1005	Homo sapiens	39-48
32663367-10	2020	Furthermore, LINC01005 acted as a sponge of miR-128-3p to upregulate KLF4 expression.	mir-128-3p	44-54	long intergenic non-protein coding RNA 1005	Homo sapiens	13-22
32663367-10	2020	Furthermore, LINC01005 acted as a sponge of miR-128-3p to upregulate KLF4 expression.	mir-128-3p	44-54	Kruppel like factor 4	Homo sapiens	69-73
32594289-0	2020	miR-128-3p enhances the protective effect of dexmedetomidine on acute lung injury in septic mice by targeted inhibition of MAPK14.	mir-128-3p	0-10	mitogen-activated protein kinase 14	Mus musculus	123-129
33015801-12	2020	Moreover, miR-128-3p bound to SZRD1 3"-UTR in a sequence-specific manner.	mir-128-3p	10-20	SUZ RNA binding domain containing 1	Homo sapiens	30-35
33015801-13	2020	In addition, LINC00346 knockdown significantly inhibited the expression of SZRD1 and the inhibition could be reversed by miR-128-3p mimics.	mir-128-3p	121-131	p53 regulated carcinoma associated Stat3 activating long intergenic non-protein coding transcript	Homo sapiens	13-22
33015801-13	2020	In addition, LINC00346 knockdown significantly inhibited the expression of SZRD1 and the inhibition could be reversed by miR-128-3p mimics.	mir-128-3p	121-131	SUZ RNA binding domain containing 1	Homo sapiens	75-80
33015801-14	2020	Furthermore, cell proliferation and apoptosis affected by LINC00346 were partially rescued by modulating miR-128-3p or SZRD1 expression.	mir-128-3p	105-115	p53 regulated carcinoma associated Stat3 activating long intergenic non-protein coding transcript	Homo sapiens	58-67
32594289-6	2020	Compared with the Model group, the contents of MDA, MPO, inflammatory factors in the DEX group and miR-128-3p mimic group were significantly decreased, and the content SOD was significantly increased, however, opposite results were occurred in oe-MAPK14 group (all p < 0.05).	mir-128-3p	99-109	mitogen-activated protein kinase 14	Mus musculus	247-253
32594289-8	2020	Compared with the miR-128-3p mimic group, all the indicators were deteriorated in the miR-128-3p mimic+oe-MAPK14 group (all p < 0.05).	mir-128-3p	86-96	mitogen-activated protein kinase 14	Mus musculus	106-112
32594289-10	2020	miR-128-3p can further enhance the protective effect of dexmedetomidine on acute lung injury in septic mice by targeting and inhibiting MAPK14 expression.	mir-128-3p	0-10	mitogen-activated protein kinase 14	Mus musculus	136-142
32278027-10	2020	Meanwhile, some common pro-inflammatory cytokines were significantly reduced while anti-inflammatory cytokine IL-10 was increased by miR-128-3p.	mir-128-3p	133-143	interleukin 10	Rattus norvegicus	110-115
32558224-9	2020	Overexpression of miR-128-3p was found to inhibit proliferation, migration, invasion, self-renewal in vitro and tumorigenicity in vivo of BCSCs, which was further validated to be achieved through inhibition of Wnt signalling pathway by down-regulating NEK2.	mir-128-3p	18-28	NIMA related kinase 2	Homo sapiens	252-256
32558224-10	2020	In summary, this study indicates that miR-128-3p inhibits the stem-like cell features of BCSCs via inhibition of the Wnt signalling pathway by down-regulating NEK2, which provides a new target for breast cancer treatment.	mir-128-3p	38-48	NIMA related kinase 2	Homo sapiens	159-163
32486927-9	2020	Overall, our study results suggest that repression of miR-128-3p can alleviate liver I/R injury through the miR-128-3p/Rnd3/NF-kappaB axis and may facilitate the development of novel protective approaches against liver I/R injury.	mir-128-3p	54-64	Rho family GTPase 3	Mus musculus	119-123
32486927-9	2020	Overall, our study results suggest that repression of miR-128-3p can alleviate liver I/R injury through the miR-128-3p/Rnd3/NF-kappaB axis and may facilitate the development of novel protective approaches against liver I/R injury.	mir-128-3p	54-64	nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105	Mus musculus	124-133
32278027-11	2020	Here, in the current investigation, by utilizing dual-luciferase reporter assays, ZEB1 was proved as a direct target of miR-128-3p.	mir-128-3p	120-130	zinc finger E-box binding homeobox 1	Rattus norvegicus	82-86
32278027-14	2020	miR-128-3p rescued the effects of ZEB1 on neuropathic pain progression via inhibiting neuroinflammation.	mir-128-3p	0-10	zinc finger E-box binding homeobox 1	Rattus norvegicus	34-38
32278027-15	2020	Taken these together, we implied miR-128-3p alleviated the progression of neuropathic pain via modulating ZEB1.	mir-128-3p	33-43	zinc finger E-box binding homeobox 1	Rattus norvegicus	106-110
32528036-11	2020	The target-relationship between miR-128-3p and c-Met and PDGFRalpha was verified by dual luciferase reporter gene.	mir-128-3p	32-42	MET proto-oncogene, receptor tyrosine kinase	Homo sapiens	47-52
32022984-8	2020	Taken together, these results showed that gypenoside LI could inhibit human melanoma cells through inducing apoptosis, arresting cell cycle at the S phase and suppressing the Wnt/beta-catenin signaling pathway in a miR-128-3p dependent manner.	mir-128-3p	215-225	catenin beta 1	Homo sapiens	179-191
32528036-11	2020	The target-relationship between miR-128-3p and c-Met and PDGFRalpha was verified by dual luciferase reporter gene.	mir-128-3p	32-42	platelet derived growth factor receptor alpha	Homo sapiens	57-67
32528036-15	2020	Overexpression of miR-128-3p down-regulated the expression levels of EMT-transformed proteins (c-Met, PDGFRalpha, Notch1 and Slug) to enhance the effect of TMZ.	mir-128-3p	18-28	met proto-oncogene	Mus musculus	95-100
32528036-15	2020	Overexpression of miR-128-3p down-regulated the expression levels of EMT-transformed proteins (c-Met, PDGFRalpha, Notch1 and Slug) to enhance the effect of TMZ.	mir-128-3p	18-28	platelet derived growth factor receptor alpha	Homo sapiens	102-112
32528036-15	2020	Overexpression of miR-128-3p down-regulated the expression levels of EMT-transformed proteins (c-Met, PDGFRalpha, Notch1 and Slug) to enhance the effect of TMZ.	mir-128-3p	18-28	notch receptor 1	Homo sapiens	114-120
32528036-15	2020	Overexpression of miR-128-3p down-regulated the expression levels of EMT-transformed proteins (c-Met, PDGFRalpha, Notch1 and Slug) to enhance the effect of TMZ.	mir-128-3p	18-28	snail family transcriptional repressor 2	Homo sapiens	125-129
32528036-16	2020	In addition, we found that miR-128-3p targeted and bound c-Met.	mir-128-3p	27-37	met proto-oncogene	Mus musculus	57-62
32528036-20	2020	miR-128-3p is capable of enhancing the sensitivity of glioblastoma to TMZ through regulating c-Met/EMT.	mir-128-3p	0-10	met proto-oncogene	Mus musculus	93-98
32377170-13	2020	In conclusion, MIR4435-2HG knockdown suppressed the progression of OC cells through downregulating CDK14 expression by the promotion of miR-128-3p.	mir-128-3p	136-146	MIR4435-2 host gene	Homo sapiens	15-26
31328440-9	2019	Moreover, HOTTIP was capable of positively modulating HOXA13 expression via the competitive binding to miR-128-3p.	mir-128-3p	103-113	HOXA distal transcript antisense RNA	Homo sapiens	10-16
32830547-0	2020	MiR-128-3p Post-Transcriptionally Inhibits WISP1 to Suppress Apoptosis and Inflammation in Human Articular Chondrocytes via the PI3K/AKT/NF-kappaB Signaling Pathway.	mir-128-3p	0-10	cellular communication network factor 4	Homo sapiens	43-48
32830547-0	2020	MiR-128-3p Post-Transcriptionally Inhibits WISP1 to Suppress Apoptosis and Inflammation in Human Articular Chondrocytes via the PI3K/AKT/NF-kappaB Signaling Pathway.	mir-128-3p	0-10	AKT serine/threonine kinase 1	Homo sapiens	133-136
32830547-0	2020	MiR-128-3p Post-Transcriptionally Inhibits WISP1 to Suppress Apoptosis and Inflammation in Human Articular Chondrocytes via the PI3K/AKT/NF-kappaB Signaling Pathway.	mir-128-3p	0-10	nuclear factor kappa B subunit 1	Homo sapiens	137-146
32270599-0	2020	LINC00963 targeting miR-128-3p promotes acute kidney injury process by activating JAK2/STAT1 pathway.	mir-128-3p	20-30	Janus kinase 2	Rattus norvegicus	82-86
32270599-0	2020	LINC00963 targeting miR-128-3p promotes acute kidney injury process by activating JAK2/STAT1 pathway.	mir-128-3p	20-30	signal transducer and activator of transcription 1	Rattus norvegicus	87-92
32270599-13	2020	LINC00963 could target miR-128-3p to reduce G1 arrest and apoptosis through JAK2/STAT1 pathway to promote the progression of AKI.	mir-128-3p	23-33	signal transducer and activator of transcription 1	Rattus norvegicus	81-86
31328440-9	2019	Moreover, HOTTIP was capable of positively modulating HOXA13 expression via the competitive binding to miR-128-3p.	mir-128-3p	103-113	homeobox A13	Homo sapiens	54-60
31112750-7	2019	Furthermore, double-luciferase reporter assay, and co-transfection test showed that miR-128-3p directly targeted Sirt1.	mir-128-3p	84-94	sirtuin 1	Mus musculus	113-118
31102936-12	2019	MiR-128-3p was predicted to be a target gene of HCP5.	mir-128-3p	0-10	HLA complex P5	Homo sapiens	48-52
31112750-12	2019	In mice, Dox-induced liver damage was deteriorated by miR-128-3p agomir via increasing the levels of ALT, AST, MDA, and down-regulating the protein levels of Sirt1, Nrf2, Sirt3, NQO1 and HO-1.	mir-128-3p	54-64	transmembrane protease, serine 11d	Mus musculus	106-109
31112750-9	2019	The protein levels of Sirt1, Nrf2, Sirt3, NQO1 and HO-1 in miR-128-3p mimic + Dox group were decreased compared with Dox group.	mir-128-3p	59-69	sirtuin 1	Mus musculus	22-27
31112750-9	2019	The protein levels of Sirt1, Nrf2, Sirt3, NQO1 and HO-1 in miR-128-3p mimic + Dox group were decreased compared with Dox group.	mir-128-3p	59-69	nuclear factor, erythroid derived 2, like 2	Mus musculus	29-33
31112750-12	2019	In mice, Dox-induced liver damage was deteriorated by miR-128-3p agomir via increasing the levels of ALT, AST, MDA, and down-regulating the protein levels of Sirt1, Nrf2, Sirt3, NQO1 and HO-1.	mir-128-3p	54-64	sirtuin 1	Mus musculus	158-163
31112750-9	2019	The protein levels of Sirt1, Nrf2, Sirt3, NQO1 and HO-1 in miR-128-3p mimic + Dox group were decreased compared with Dox group.	mir-128-3p	59-69	sirtuin 3	Mus musculus	35-40
31112750-12	2019	In mice, Dox-induced liver damage was deteriorated by miR-128-3p agomir via increasing the levels of ALT, AST, MDA, and down-regulating the protein levels of Sirt1, Nrf2, Sirt3, NQO1 and HO-1.	mir-128-3p	54-64	nuclear factor, erythroid derived 2, like 2	Mus musculus	165-169
31112750-9	2019	The protein levels of Sirt1, Nrf2, Sirt3, NQO1 and HO-1 in miR-128-3p mimic + Dox group were decreased compared with Dox group.	mir-128-3p	59-69	NAD(P)H dehydrogenase, quinone 1	Mus musculus	42-46
31112750-12	2019	In mice, Dox-induced liver damage was deteriorated by miR-128-3p agomir via increasing the levels of ALT, AST, MDA, and down-regulating the protein levels of Sirt1, Nrf2, Sirt3, NQO1 and HO-1.	mir-128-3p	54-64	sirtuin 3	Mus musculus	171-176
31112750-9	2019	The protein levels of Sirt1, Nrf2, Sirt3, NQO1 and HO-1 in miR-128-3p mimic + Dox group were decreased compared with Dox group.	mir-128-3p	59-69	heme oxygenase 1	Mus musculus	51-55
31112750-12	2019	In mice, Dox-induced liver damage was deteriorated by miR-128-3p agomir via increasing the levels of ALT, AST, MDA, and down-regulating the protein levels of Sirt1, Nrf2, Sirt3, NQO1 and HO-1.	mir-128-3p	54-64	NAD(P)H dehydrogenase, quinone 1	Mus musculus	178-182
31112750-12	2019	In mice, Dox-induced liver damage was deteriorated by miR-128-3p agomir via increasing the levels of ALT, AST, MDA, and down-regulating the protein levels of Sirt1, Nrf2, Sirt3, NQO1 and HO-1.	mir-128-3p	54-64	heme oxygenase 1	Mus musculus	187-191
31112750-12	2019	In mice, Dox-induced liver damage was deteriorated by miR-128-3p agomir via increasing the levels of ALT, AST, MDA, and down-regulating the protein levels of Sirt1, Nrf2, Sirt3, NQO1 and HO-1.	mir-128-3p	54-64	glutamic pyruvic transaminase, soluble	Mus musculus	101-104
31112750-13	2019	While, miR-128-3p antagomir alleviated liver injury via decreasing the levels of ALT, AST, MDA, and up-regulating the protein levels of Sirt1, Nrf2, Sirt3, NQO1 and HO-1.	mir-128-3p	7-17	glutamic pyruvic transaminase, soluble	Mus musculus	81-84
31112750-13	2019	While, miR-128-3p antagomir alleviated liver injury via decreasing the levels of ALT, AST, MDA, and up-regulating the protein levels of Sirt1, Nrf2, Sirt3, NQO1 and HO-1.	mir-128-3p	7-17	transmembrane protease, serine 11d	Mus musculus	86-89
30906475-8	2019	Conversely, transfection with miR-128-3p inhibitor significantly increased the levels of p-IRS-1, PI3K and p-AKT, accompanied by an elevated proliferation rate of the cells.	mir-128-3p	30-40	insulin receptor substrate 1	Homo sapiens	91-96
31112750-13	2019	While, miR-128-3p antagomir alleviated liver injury via decreasing the levels of ALT, AST, MDA, and up-regulating the protein levels of Sirt1, Nrf2, Sirt3, NQO1 and HO-1.	mir-128-3p	7-17	sirtuin 1	Mus musculus	136-141
31112750-13	2019	While, miR-128-3p antagomir alleviated liver injury via decreasing the levels of ALT, AST, MDA, and up-regulating the protein levels of Sirt1, Nrf2, Sirt3, NQO1 and HO-1.	mir-128-3p	7-17	nuclear factor, erythroid derived 2, like 2	Mus musculus	143-147
31112750-13	2019	While, miR-128-3p antagomir alleviated liver injury via decreasing the levels of ALT, AST, MDA, and up-regulating the protein levels of Sirt1, Nrf2, Sirt3, NQO1 and HO-1.	mir-128-3p	7-17	sirtuin 3	Mus musculus	149-154
31112750-13	2019	While, miR-128-3p antagomir alleviated liver injury via decreasing the levels of ALT, AST, MDA, and up-regulating the protein levels of Sirt1, Nrf2, Sirt3, NQO1 and HO-1.	mir-128-3p	7-17	NAD(P)H dehydrogenase, quinone 1	Mus musculus	156-160
31112750-13	2019	While, miR-128-3p antagomir alleviated liver injury via decreasing the levels of ALT, AST, MDA, and up-regulating the protein levels of Sirt1, Nrf2, Sirt3, NQO1 and HO-1.	mir-128-3p	7-17	heme oxygenase 1	Mus musculus	165-169
30906475-0	2019	miR-128-3p inhibits glioma cell proliferation and differentiation by targeting NPTX1 through IRS-1/PI3K/AKT signaling pathway.	mir-128-3p	0-10	neuronal pentraxin 1	Homo sapiens	79-84
31078043-0	2019	MiR-128-3p suppresses breast cancer cellular progression via targeting LIMK1.	mir-128-3p	0-10	LIM domain kinase 1	Homo sapiens	71-76
31078043-4	2019	In addition, we found that overexpression of miR-128-3p arrested breast cancer cells in G0/G1 phase by affecting expression of CDK4/CDK6/Cyclin D1 and CDK2/Cyclin E1.	mir-128-3p	45-55	cyclin dependent kinase 4	Homo sapiens	127-131
31078043-4	2019	In addition, we found that overexpression of miR-128-3p arrested breast cancer cells in G0/G1 phase by affecting expression of CDK4/CDK6/Cyclin D1 and CDK2/Cyclin E1.	mir-128-3p	45-55	cyclin dependent kinase 6	Homo sapiens	132-136
31078043-4	2019	In addition, we found that overexpression of miR-128-3p arrested breast cancer cells in G0/G1 phase by affecting expression of CDK4/CDK6/Cyclin D1 and CDK2/Cyclin E1.	mir-128-3p	45-55	cyclin D1	Homo sapiens	137-146
31078043-4	2019	In addition, we found that overexpression of miR-128-3p arrested breast cancer cells in G0/G1 phase by affecting expression of CDK4/CDK6/Cyclin D1 and CDK2/Cyclin E1.	mir-128-3p	45-55	cyclin dependent kinase 2	Homo sapiens	151-155
31078043-4	2019	In addition, we found that overexpression of miR-128-3p arrested breast cancer cells in G0/G1 phase by affecting expression of CDK4/CDK6/Cyclin D1 and CDK2/Cyclin E1.	mir-128-3p	45-55	cyclin E1	Homo sapiens	156-165
31078043-7	2019	Our findings demonstrated that miR-128-3p could regulate cellular progression of breast cancer via regulating the LIMK1/CFL1 signaling pathway, and this new avenue could broaden existing versions of molecular mechanisms in breast cancer and perhaps represent potential novel direction of breast cancer treatment in the future.	mir-128-3p	31-41	LIM domain kinase 1	Homo sapiens	114-119
31078043-7	2019	Our findings demonstrated that miR-128-3p could regulate cellular progression of breast cancer via regulating the LIMK1/CFL1 signaling pathway, and this new avenue could broaden existing versions of molecular mechanisms in breast cancer and perhaps represent potential novel direction of breast cancer treatment in the future.	mir-128-3p	31-41	cofilin 1	Homo sapiens	120-124
30906475-0	2019	miR-128-3p inhibits glioma cell proliferation and differentiation by targeting NPTX1 through IRS-1/PI3K/AKT signaling pathway.	mir-128-3p	0-10	insulin receptor substrate 1	Homo sapiens	93-98
30906475-6	2019	In the U251 human glioma cell line, transfection with miR-128-3p mimics increased the levels of phosphorylated insulin receptor substrate 1 (p-IRS-1), phosphoinositide-3 kinase (PI3K) and p-AKT, as demonstrated by western blot analysis.	mir-128-3p	54-64	insulin receptor substrate 1	Homo sapiens	111-139
30906475-6	2019	In the U251 human glioma cell line, transfection with miR-128-3p mimics increased the levels of phosphorylated insulin receptor substrate 1 (p-IRS-1), phosphoinositide-3 kinase (PI3K) and p-AKT, as demonstrated by western blot analysis.	mir-128-3p	54-64	insulin receptor substrate 1	Homo sapiens	143-148
30906475-6	2019	In the U251 human glioma cell line, transfection with miR-128-3p mimics increased the levels of phosphorylated insulin receptor substrate 1 (p-IRS-1), phosphoinositide-3 kinase (PI3K) and p-AKT, as demonstrated by western blot analysis.	mir-128-3p	54-64	phosphatidylinositol-4,5-bisphosphate 3-kinase catalytic subunit delta	Homo sapiens	151-176
30402839-10	2018	Mechanistically, PTEN/PI3K/Akt signaling pathway was found to participate in the miR-128-3p induced sensitivity to sorafenib via DJ-1.	mir-128-3p	81-91	phosphatase and tensin homolog	Homo sapiens	17-21
30341898-0	2019	MiR-128-3p accelerates cardiovascular calcification and insulin resistance through ISL1-dependent Wnt pathway in type 2 diabetes mellitus rats.	mir-128-3p	0-10	ISL LIM homeobox 1	Rattus norvegicus	83-87
30341898-0	2019	MiR-128-3p accelerates cardiovascular calcification and insulin resistance through ISL1-dependent Wnt pathway in type 2 diabetes mellitus rats.	mir-128-3p	0-10	Wnt family member 1	Rattus norvegicus	98-101
30420640-7	2018	Notably, miR-33a-5p and miR-128-3p in whole blood of lung cancer patients or early-stage lung cancer patients (TNM stage I-II) were lowly expressed as compared with that in healthy controls.	mir-128-3p	24-34	teneurin transmembrane protein 1	Homo sapiens	111-114
30402839-10	2018	Mechanistically, PTEN/PI3K/Akt signaling pathway was found to participate in the miR-128-3p induced sensitivity to sorafenib via DJ-1.	mir-128-3p	81-91	Parkinsonism associated deglycase	Homo sapiens	129-133
29777777-0	2018	MiR-128-3p directly targets VEGFC/VEGFR3 to modulate the proliferation of lymphatic endothelial cells through Ca2+ signaling.	mir-128-3p	0-10	vascular endothelial growth factor C	Homo sapiens	28-33
29777777-0	2018	MiR-128-3p directly targets VEGFC/VEGFR3 to modulate the proliferation of lymphatic endothelial cells through Ca2+ signaling.	mir-128-3p	0-10	fms related receptor tyrosine kinase 4	Homo sapiens	34-40
30223934-11	2018	Besides, miR-128-3p inhibition remarkably alleviated the inhibitory effects of HCl on the activation of ERK and PI3K/AKT pathways, which were further reversed after inhibition of miR-128-3p and E2F3 at the same time.	mir-128-3p	9-19	mitogen-activated protein kinase 1	Homo sapiens	104-107
30223934-11	2018	Besides, miR-128-3p inhibition remarkably alleviated the inhibitory effects of HCl on the activation of ERK and PI3K/AKT pathways, which were further reversed after inhibition of miR-128-3p and E2F3 at the same time.	mir-128-3p	9-19	AKT serine/threonine kinase 1	Homo sapiens	117-120
30223934-11	2018	Besides, miR-128-3p inhibition remarkably alleviated the inhibitory effects of HCl on the activation of ERK and PI3K/AKT pathways, which were further reversed after inhibition of miR-128-3p and E2F3 at the same time.	mir-128-3p	9-19	E2F transcription factor 3	Homo sapiens	194-198
28627514-0	2017	Simultaneous overactivation of Wnt/beta-catenin and TGFbeta signalling by miR-128-3p confers chemoresistance-associated metastasis in NSCLC.	mir-128-3p	74-84	catenin beta 1	Homo sapiens	35-47
28627514-0	2017	Simultaneous overactivation of Wnt/beta-catenin and TGFbeta signalling by miR-128-3p confers chemoresistance-associated metastasis in NSCLC.	mir-128-3p	74-84	transforming growth factor beta 1	Homo sapiens	52-59
28627514-4	2017	Mechanistically, miR-128-3p induces mesenchymal and stemness-like properties through downregulating multiple inhibitors of Wnt/beta-catenin and TGF-beta pathways, leading to their overactivation.	mir-128-3p	17-27	catenin beta 1	Homo sapiens	127-139
28627514-4	2017	Mechanistically, miR-128-3p induces mesenchymal and stemness-like properties through downregulating multiple inhibitors of Wnt/beta-catenin and TGF-beta pathways, leading to their overactivation.	mir-128-3p	17-27	transforming growth factor beta 1	Homo sapiens	144-152