PMID-sentid	Pub_year	Sent_text	compound_name	comp_offset	prot_official_name	organism	prot_offset
34973275-0	2022	miR-142-3p simultaneously targets HMGA1, HMGA2, HMGB1, and HMGB3 and inhibits tumorigenic properties and in-vivo metastatic potential of human cervical cancer cells.	mir-142-3p	0-10	high mobility group AT-hook 1	Homo sapiens	34-39
34973275-0	2022	miR-142-3p simultaneously targets HMGA1, HMGA2, HMGB1, and HMGB3 and inhibits tumorigenic properties and in-vivo metastatic potential of human cervical cancer cells.	mir-142-3p	0-10	high mobility group AT-hook 2	Homo sapiens	41-46
34973275-0	2022	miR-142-3p simultaneously targets HMGA1, HMGA2, HMGB1, and HMGB3 and inhibits tumorigenic properties and in-vivo metastatic potential of human cervical cancer cells.	mir-142-3p	0-10	high mobility group box 3	Homo sapiens	59-64
34973275-3	2022	MAIN METHODS: 3" UTR luciferase reporter assays were performed to validate HMGA1, A2, B1, and B3 as targets of miR-142-3p in human cervical cancer cells.	mir-142-3p	111-121	high mobility group AT-hook 1	Homo sapiens	75-80
34973275-0	2022	miR-142-3p simultaneously targets HMGA1, HMGA2, HMGB1, and HMGB3 and inhibits tumorigenic properties and in-vivo metastatic potential of human cervical cancer cells.	mir-142-3p	0-10	high mobility group box 1	Homo sapiens	48-53
34973275-3	2022	MAIN METHODS: 3" UTR luciferase reporter assays were performed to validate HMGA1, A2, B1, and B3 as targets of miR-142-3p in human cervical cancer cells.	mir-142-3p	111-121	immunoglobulin kappa variable 7-3 (pseudogene)	Homo sapiens	86-96
35378380-3	2022	Indeed, while miR-223 has little effect on EC responses, we show that the induced expression of miR-142-3p and miR-451 in ECs results in profound cell damage, especially in inflammatory conditions, characterized by a dramatic increase in cell apoptosis, impaired angiogenic repair responses, and the induction of IL-6, IL-8, CXCL10 and CXCL11 expression.	mir-142-3p	96-106	microRNA 223	Homo sapiens	14-21
34973275-8	2022	In addition, miR-142-3p expression decreased phospho-p65 and EMT-related proteins in cervical cancer cells and their in vivo metastatic potential upon implantation in zebrafish.	mir-142-3p	13-23	v-rel avian reticuloendotheliosis viral oncogene homolog A	Danio rerio	53-56
34659494-9	2021	Further investigation indicated that the tumor-inhibitory effect of ST8SIA6-AS1 silencing was reversed by miR-142-3p depletion.	mir-142-3p	106-116	ST8 alpha-N-acetyl-neuraminide alpha-2,8-sialyltransferase 6	Homo sapiens	68-75
34659494-9	2021	Further investigation indicated that the tumor-inhibitory effect of ST8SIA6-AS1 silencing was reversed by miR-142-3p depletion.	mir-142-3p	106-116	prostaglandin D2 receptor	Homo sapiens	76-79
34659494-10	2021	In conclusion, ST8SIA6-AS1 was indicated to exert an oncogenic function in liver cancer by competitively sponging miR-142-3p.	mir-142-3p	114-124	ST8 alpha-N-acetyl-neuraminide alpha-2,8-sialyltransferase 6	Homo sapiens	15-22
34659494-10	2021	In conclusion, ST8SIA6-AS1 was indicated to exert an oncogenic function in liver cancer by competitively sponging miR-142-3p.	mir-142-3p	114-124	prostaglandin D2 receptor	Homo sapiens	23-26
34619495-7	2021	Furthermore, miR-142-3p negatively-regulated expression of ATG16L1 and ATG4c by directly targeting its 3"-UTR, and meaningfully abated the level of autophagy.	mir-142-3p	13-23	autophagy related 16-like 1 (S. cerevisiae)	Mus musculus	59-66
34619495-7	2021	Furthermore, miR-142-3p negatively-regulated expression of ATG16L1 and ATG4c by directly targeting its 3"-UTR, and meaningfully abated the level of autophagy.	mir-142-3p	13-23	autophagy related 4C, cysteine peptidase	Mus musculus	71-76
34619495-8	2021	CONCLUSION: These findings suggested that miR-142-3p inhibited M. tuberculosis-induced activation of autophagy and promoted H37Ra survival in RAW264.7 cells by targeting ATG16L1 and ATG4c.	mir-142-3p	42-52	autophagy related 16-like 1 (S. cerevisiae)	Mus musculus	170-177
34619495-8	2021	CONCLUSION: These findings suggested that miR-142-3p inhibited M. tuberculosis-induced activation of autophagy and promoted H37Ra survival in RAW264.7 cells by targeting ATG16L1 and ATG4c.	mir-142-3p	42-52	autophagy related 4C, cysteine peptidase	Mus musculus	182-187
34753133-0	2022	miR-142-3p Regulates Tumor Cell Autophagy and Promotes Colon Cancer Progression by Targeting TP53INP2.	mir-142-3p	0-10	tumor protein p53 inducible nuclear protein 2	Homo sapiens	93-101
34753133-14	2022	Rescue experiments revealed that influence of miR-142-3p inhibitor on CC cell proliferation and apoptosis could be reversed by silencing TP53INP2.	mir-142-3p	46-56	tumor protein p53 inducible nuclear protein 2	Homo sapiens	137-145
34753133-15	2022	CONCLUSION: miR-142-3p hampered tumor cell autophagy and promoted CC progression via targeting TP53INP2, which will offer a fresh research orientation for the diagnosis of CC.	mir-142-3p	12-22	tumor protein p53 inducible nuclear protein 2	Homo sapiens	95-103
34109430-6	2021	Dual-luciferase reporter gene assays were performed to investigate the association between miR-142-3p and Rac family small GTPase 1 (RAC1).	mir-142-3p	91-101	Rac family small GTPase 1	Homo sapiens	106-109
34109430-6	2021	Dual-luciferase reporter gene assays were performed to investigate the association between miR-142-3p and Rac family small GTPase 1 (RAC1).	mir-142-3p	91-101	Rac family small GTPase 1	Homo sapiens	133-137
34109430-11	2021	RAC1 was a possible target of miR-142-3p, which was confirmed by dual-luciferase reporter assay.	mir-142-3p	30-40	Rac family small GTPase 1	Homo sapiens	0-4
34109430-12	2021	Transfection of miR-142-3p mimics decreased the levels of RAC1 and suppressed epithelial-to-mesenchymal transition in colon cancer cells.	mir-142-3p	16-26	Rac family small GTPase 1	Homo sapiens	58-62
34109430-13	2021	The phosphorylation of extraceullar signal-regulated kinase (ERK) was decreased significantly by the inhibition of RAC1 or transfection of miR-142-3p mimics in colon cancer cells.	mir-142-3p	139-149	mitogen-activated protein kinase 1	Homo sapiens	23-59
34109430-13	2021	The phosphorylation of extraceullar signal-regulated kinase (ERK) was decreased significantly by the inhibition of RAC1 or transfection of miR-142-3p mimics in colon cancer cells.	mir-142-3p	139-149	mitogen-activated protein kinase 1	Homo sapiens	61-64
34336555-0	2021	miR-142-3p Modulates Cell Invasion and Migration via PKM2-Mediated Aerobic Glycolysis in Colorectal Cancer.	mir-142-3p	0-10	pyruvate kinase M1/2	Homo sapiens	53-57
34336555-6	2021	Dual-luciferase reporter assays were performed to confirm the correlation between miR-142-3p and pyruvate kinase isozyme M2 (PKM2).	mir-142-3p	82-92	pyruvate kinase M1/2	Homo sapiens	125-129
34336555-10	2021	About the underlying mechanism, we found that miR-142-3p modulated aerobic glycolysis via targeting pyruvate kinase M2 (PKM2).	mir-142-3p	46-56	pyruvate kinase M1/2	Homo sapiens	100-118
34336555-10	2021	About the underlying mechanism, we found that miR-142-3p modulated aerobic glycolysis via targeting pyruvate kinase M2 (PKM2).	mir-142-3p	46-56	pyruvate kinase M1/2	Homo sapiens	120-124
34336555-11	2021	In addition, we demonstrated PKM2 and PKM2-mediated aerobic glycolysis contributes to miR-142-3p-mediated colorectal cancer cell invasion and migration.	mir-142-3p	86-96	pyruvate kinase M1/2	Homo sapiens	29-33
34336555-11	2021	In addition, we demonstrated PKM2 and PKM2-mediated aerobic glycolysis contributes to miR-142-3p-mediated colorectal cancer cell invasion and migration.	mir-142-3p	86-96	pyruvate kinase M1/2	Homo sapiens	38-42
34145485-0	2021	MiR-142-3p ameliorates high glucose-induced renal tubular epithelial cell injury by targeting BOD1.	mir-142-3p	0-10	biorientation of chromosomes in cell division 1	Homo sapiens	94-98
34145485-8	2021	The binding between miR-142-3p and biorientation of chromosomes in cell division 1 (BOD1) was validated by a luciferase reporter assay.	mir-142-3p	20-30	biorientation of chromosomes in cell division 1	Homo sapiens	84-88
34145485-11	2021	Mechanistically, BOD1 was confirmed to be targeted by miR-142-3p in HK-2 cells.	mir-142-3p	54-64	biorientation of chromosomes in cell division 1	Homo sapiens	17-21
34145485-12	2021	Moreover, BOD1 overexpression reversed the suppressive effect of miR-142-3p overexpression on the apoptosis and oxidative stress of HK-2 cells treated with HG.	mir-142-3p	65-75	biorientation of chromosomes in cell division 1	Homo sapiens	10-14
34145485-13	2021	CONCLUSION: MiR-142-3p ameliorates HG-induced renal tubular epithelial cell injury by targeting BOD1.	mir-142-3p	12-22	biorientation of chromosomes in cell division 1	Homo sapiens	96-100
34153134-7	2021	The luciferase gene reporter assay was performed to validate that miR-142-3p is a TUG1 target.	mir-142-3p	66-76	taurine upregulated gene 1	Mus musculus	82-86
34153134-8	2021	Accordingly, the effects of miR-142-3p knockdown on TUG1-induced MA-C apoptosis were determined using flow cytometry.	mir-142-3p	28-38	taurine upregulated gene 1	Mus musculus	52-56
34153134-14	2021	Inhibition of OGD/R-induced increases in TUG1 expression that in turn reduces miR-142-3p upregulation may suppress reperfusion-induced losses in cell viability.	mir-142-3p	78-88	taurine upregulated gene 1	Mus musculus	41-45
34461809-7	2021	The expression of SIRT1 induced by MALAT1 overexpression was obviously abolished by the introduction of miR-142-3p mimic.	mir-142-3p	104-114	sirtuin 1	Rattus norvegicus	18-23
34461809-7	2021	The expression of SIRT1 induced by MALAT1 overexpression was obviously abolished by the introduction of miR-142-3p mimic.	mir-142-3p	104-114	metastasis associated lung adenocarcinoma transcript 1 (non-coding RNA)	Mus musculus	35-41
34362486-0	2021	(Effect of MiR-142-3p Targeting HOXA5 on Proliferation, Cycle Arrest and Apoptosis of Acute B Lymphocytic Leukemia Cells).	mir-142-3p	11-21	homeobox A5	Homo sapiens	32-37
34362486-1	2021	OBJECTIVE: To investigate the effect and molecular mechanism of miR-142-3p to the proliferation, cycle and apoptosis of acute B lymphocytic leukemia (B-ALL) cells by regulating the homeobox gene 5 (HOXA5) expression.	mir-142-3p	64-74	homeobox A5	Homo sapiens	198-203
34362486-10	2021	The proliferation of Nalm6 cells and the number of cell clones could be inhibited by miR-142-3p mimic after 48 and 72 hours of transfection (P<0.05), which causing G1 phase arrest of Nalm6 cells and inhibiting the expression of CyclinD1 and CDK4 protein (P<0.01), promoting the apoptosis of Nalm6 cells, and inhibiting the expression of BCL-2 protein, moreover, promoting the expression of Bax and Caspase-3 protein (P<0.05).	mir-142-3p	85-95	cyclin D1	Homo sapiens	228-236
34362486-10	2021	The proliferation of Nalm6 cells and the number of cell clones could be inhibited by miR-142-3p mimic after 48 and 72 hours of transfection (P<0.05), which causing G1 phase arrest of Nalm6 cells and inhibiting the expression of CyclinD1 and CDK4 protein (P<0.01), promoting the apoptosis of Nalm6 cells, and inhibiting the expression of BCL-2 protein, moreover, promoting the expression of Bax and Caspase-3 protein (P<0.05).	mir-142-3p	85-95	cyclin dependent kinase 4	Homo sapiens	241-245
34362486-10	2021	The proliferation of Nalm6 cells and the number of cell clones could be inhibited by miR-142-3p mimic after 48 and 72 hours of transfection (P<0.05), which causing G1 phase arrest of Nalm6 cells and inhibiting the expression of CyclinD1 and CDK4 protein (P<0.01), promoting the apoptosis of Nalm6 cells, and inhibiting the expression of BCL-2 protein, moreover, promoting the expression of Bax and Caspase-3 protein (P<0.05).	mir-142-3p	85-95	BCL2 apoptosis regulator	Homo sapiens	337-342
34362486-10	2021	The proliferation of Nalm6 cells and the number of cell clones could be inhibited by miR-142-3p mimic after 48 and 72 hours of transfection (P<0.05), which causing G1 phase arrest of Nalm6 cells and inhibiting the expression of CyclinD1 and CDK4 protein (P<0.01), promoting the apoptosis of Nalm6 cells, and inhibiting the expression of BCL-2 protein, moreover, promoting the expression of Bax and Caspase-3 protein (P<0.05).	mir-142-3p	85-95	BCL2 associated X, apoptosis regulator	Homo sapiens	390-393
34362486-11	2021	CONCLUSION: MiR-142-3p can inhibit the proliferation of Nalm6 cells by targeting down-regulation the expression of HOXA5, arrest the G1 phase of cells, and promote apoptosis of the cells.	mir-142-3p	12-22	homeobox A5	Homo sapiens	115-120
35452863-0	2022	miR-142-3p suppresses porcine reproductive and respiratory syndrome virus (PRRSV) infection by directly targeting Rac1.	mir-142-3p	0-10	Rac family small GTPase 1	Homo sapiens	114-118
35452863-6	2022	Importantly, we verified that miR-142-3p inhibited PRRSV entry into PAMs and accordingly suppressed PRRSV infection by downregulating Rac1 expression.	mir-142-3p	30-40	Rac family small GTPase 1	Homo sapiens	134-138
35378380-4	2022	We show that the strong deleterious effect of miR-142-3p may be due in part to its ability to block the activation of ERK1/2 and eNOS-mediated signals in ECs.	mir-142-3p	46-56	mitogen-activated protein kinase 3	Homo sapiens	118-124
35378380-4	2022	We show that the strong deleterious effect of miR-142-3p may be due in part to its ability to block the activation of ERK1/2 and eNOS-mediated signals in ECs.	mir-142-3p	46-56	nitric oxide synthase 3	Homo sapiens	129-133
35378380-5	2022	miR-142-3p also inhibits the expression of RAC1, ROCK2 and CLIC4, three genes that are critical for EC migration and angiogenic responses.	mir-142-3p	0-10	Rac family small GTPase 1	Homo sapiens	43-47
35414011-0	2022	Possible implication of miR-142-3p in coronary microembolization induced myocardial injury via ATXN1L/HDAC3/NOL3 axis.	mir-142-3p	24-34	ataxin 1-like	Rattus norvegicus	95-101
35378380-5	2022	miR-142-3p also inhibits the expression of RAC1, ROCK2 and CLIC4, three genes that are critical for EC migration and angiogenic responses.	mir-142-3p	0-10	Rho associated coiled-coil containing protein kinase 2	Homo sapiens	49-54
35414011-0	2022	Possible implication of miR-142-3p in coronary microembolization induced myocardial injury via ATXN1L/HDAC3/NOL3 axis.	mir-142-3p	24-34	histone deacetylase 3	Rattus norvegicus	102-107
35378380-5	2022	miR-142-3p also inhibits the expression of RAC1, ROCK2 and CLIC4, three genes that are critical for EC migration and angiogenic responses.	mir-142-3p	0-10	chloride intracellular channel 4	Homo sapiens	59-64
35279674-8	2022	miR-142-3p was elevated with aging, which downregulated Bmal1 and diminished the osteogenic potential of BMSCs.	mir-142-3p	0-10	aryl hydrocarbon receptor nuclear translocator-like	Mus musculus	56-61
35414011-0	2022	Possible implication of miR-142-3p in coronary microembolization induced myocardial injury via ATXN1L/HDAC3/NOL3 axis.	mir-142-3p	24-34	nucleolar protein 3	Rattus norvegicus	108-112
35414011-1	2022	This study aims to explore the mechanism underlying miR-142-3p regulating myocardial injury induced by coronary microembolization (CME) through ATXN1L.	mir-142-3p	52-62	ataxin 1-like	Rattus norvegicus	144-150
35414011-6	2022	The interaction between miR-142-3p and ATXN1L as well as the binding between HDAC3 and histone 3 (H3) was identified.	mir-142-3p	24-34	ataxin 1-like	Rattus norvegicus	39-45
35414011-13	2022	miR-142-3p regulated ATXN1L expression in a targeted manner.	mir-142-3p	0-10	ataxin 1-like	Rattus norvegicus	21-27
35414011-14	2022	In the cellular context, miR-142-3p overexpression attenuated apoptosis and pyroptosis in cardiomyocytes, which was partly counteracted by ATXN1L overexpression.	mir-142-3p	25-35	ataxin 1-like	Rattus norvegicus	139-145
35414011-17	2022	In vivo and in vitro evidence in this study supported that miR-142-3p could attenuate CME-induced myocardial injury via ATXN1L/HDAC3/NOL3.	mir-142-3p	59-69	ataxin 1-like	Rattus norvegicus	120-126
35414011-17	2022	In vivo and in vitro evidence in this study supported that miR-142-3p could attenuate CME-induced myocardial injury via ATXN1L/HDAC3/NOL3.	mir-142-3p	59-69	histone deacetylase 3	Rattus norvegicus	127-132
35414011-17	2022	In vivo and in vitro evidence in this study supported that miR-142-3p could attenuate CME-induced myocardial injury via ATXN1L/HDAC3/NOL3.	mir-142-3p	59-69	nucleolar protein 3	Rattus norvegicus	133-137
35489022-14	2022	Our results concluded that miR-142-3p and its targets gene FAM83D may be potential diagnostic and prognostic biomarkers for patients with OC.	mir-142-3p	27-37	family with sequence similarity 83 member D	Homo sapiens	59-65
35557596-0	2022	Exosomal miR-142-3p secreted by hepatitis B virus (HBV)-hepatocellular carcinoma (HCC) cells promotes ferroptosis of M1-type macrophages through SLC3A2 and the mechanism of HCC progression.	mir-142-3p	9-19	solute carrier family 3 member 2	Homo sapiens	145-151
35557596-12	2022	Conclusions: Our findings indicated that miR-142-3p promoted HBV-infected M1-type macrophage ferroptosis through SLC3A2, affecting the production of GSH, MDA, and Fe2+ and accelerating the development of HCC.	mir-142-3p	41-51	solute carrier family 3 member 2	Homo sapiens	113-119
35246019-9	2022	However, miR-142-3p inhibitor reverses the effect of circHECTD1 on all the above-mentioned aspects, including HMGB1 expression.	mir-142-3p	9-19	high mobility group box 1	Homo sapiens	110-115
35106926-8	2022	The predicted binding relationship between miR-142-3p and circ-RAD23B or MAP4K3 was verified by dual-luciferase reporter assay.	mir-142-3p	43-53	mitogen-activated protein kinase kinase kinase kinase 3	Homo sapiens	73-79
35106926-14	2022	Rescue experiments presented that miR-142-3p inhibition reversed the effects of circ-RAD23B knockdown, and MAP4K3 overexpression abolished the effects of miR-142-3p restoration.	mir-142-3p	34-44	RAD23 homolog B, nucleotide excision repair protein	Homo sapiens	85-91
35106926-14	2022	Rescue experiments presented that miR-142-3p inhibition reversed the effects of circ-RAD23B knockdown, and MAP4K3 overexpression abolished the effects of miR-142-3p restoration.	mir-142-3p	154-164	mitogen-activated protein kinase kinase kinase kinase 3	Homo sapiens	107-113
34986757-13	2022	Overexpressing miR-142-3p accelerated apoptosis and suppressed the PI3K/AKT/HIF-1alpha signal.	mir-142-3p	15-25	AKT serine/threonine kinase 1	Homo sapiens	72-75
35082279-0	2022	Correction to: The mechanism of miR-142-3p in coronary microembolization-induced myocardiac injury via regulating target gene IRAK-1.	mir-142-3p	32-42	interleukin 1 receptor associated kinase 1	Homo sapiens	126-132
35006556-12	2022	In addition, it was confirmed by dual-luciferase reporter assay that MALAT1 interacted with miR-142-3p directly.	mir-142-3p	92-102	metastasis associated lung adenocarcinoma transcript 1	Homo sapiens	69-75
35006556-14	2022	Finally, miR-142-3p interacted with HMGB1 directly and inhibition of HMGB1 protein expression mediated by MALAT1 knockdown was reversed by miR-142-3p inhibitor.	mir-142-3p	9-19	high mobility group box 1	Homo sapiens	36-41
35006556-14	2022	Finally, miR-142-3p interacted with HMGB1 directly and inhibition of HMGB1 protein expression mediated by MALAT1 knockdown was reversed by miR-142-3p inhibitor.	mir-142-3p	9-19	high mobility group box 1	Homo sapiens	69-74
35006556-14	2022	Finally, miR-142-3p interacted with HMGB1 directly and inhibition of HMGB1 protein expression mediated by MALAT1 knockdown was reversed by miR-142-3p inhibitor.	mir-142-3p	9-19	metastasis associated lung adenocarcinoma transcript 1	Homo sapiens	106-112
35006556-14	2022	Finally, miR-142-3p interacted with HMGB1 directly and inhibition of HMGB1 protein expression mediated by MALAT1 knockdown was reversed by miR-142-3p inhibitor.	mir-142-3p	139-149	high mobility group box 1	Homo sapiens	36-41
35006556-14	2022	Finally, miR-142-3p interacted with HMGB1 directly and inhibition of HMGB1 protein expression mediated by MALAT1 knockdown was reversed by miR-142-3p inhibitor.	mir-142-3p	139-149	high mobility group box 1	Homo sapiens	69-74
35006556-14	2022	Finally, miR-142-3p interacted with HMGB1 directly and inhibition of HMGB1 protein expression mediated by MALAT1 knockdown was reversed by miR-142-3p inhibitor.	mir-142-3p	139-149	metastasis associated lung adenocarcinoma transcript 1	Homo sapiens	106-112
34986757-13	2022	Overexpressing miR-142-3p accelerated apoptosis and suppressed the PI3K/AKT/HIF-1alpha signal.	mir-142-3p	15-25	hypoxia inducible factor 1 subunit alpha	Homo sapiens	76-86
34986757-15	2022	PIK3CG overexpression dampened the anti-tumor effect of miR-142-3p.	mir-142-3p	56-66	phosphatidylinositol-4,5-bisphosphate 3-kinase catalytic subunit gamma	Homo sapiens	0-6
34986757-16	2022	miR-142-3p repressed HCC invasion and intensified apoptosis to restrain HCC by abating the PIK3CG-mediated PI3K/AKT/HIF-1alpha pathway.	mir-142-3p	0-10	phosphatidylinositol-4,5-bisphosphate 3-kinase catalytic subunit gamma	Homo sapiens	91-97
34986757-16	2022	miR-142-3p repressed HCC invasion and intensified apoptosis to restrain HCC by abating the PIK3CG-mediated PI3K/AKT/HIF-1alpha pathway.	mir-142-3p	0-10	AKT serine/threonine kinase 1	Homo sapiens	112-115
34986757-16	2022	miR-142-3p repressed HCC invasion and intensified apoptosis to restrain HCC by abating the PIK3CG-mediated PI3K/AKT/HIF-1alpha pathway.	mir-142-3p	0-10	hypoxia inducible factor 1 subunit alpha	Homo sapiens	116-126
33410156-12	2021	CircWHSC1 functioned as the sponge for miR-142-3p, which directly targeted HOXA1.	mir-142-3p	39-49	nuclear receptor binding SET domain protein 2	Homo sapiens	0-9
33785332-0	2021	MiR-142-3p targets HMGA2 and suppresses breast cancer malignancy.	mir-142-3p	0-10	high mobility group AT-hook 2	Homo sapiens	19-24
33785332-9	2021	MiR-142-3p also decreases cell proliferation through inhibition of the ERK/AKT/STAT3 signaling pathways.	mir-142-3p	0-10	mitogen-activated protein kinase 1	Homo sapiens	71-74
33785332-9	2021	MiR-142-3p also decreases cell proliferation through inhibition of the ERK/AKT/STAT3 signaling pathways.	mir-142-3p	0-10	AKT serine/threonine kinase 1	Homo sapiens	75-78
33785332-9	2021	MiR-142-3p also decreases cell proliferation through inhibition of the ERK/AKT/STAT3 signaling pathways.	mir-142-3p	0-10	signal transducer and activator of transcription 3	Homo sapiens	79-84
33410156-12	2021	CircWHSC1 functioned as the sponge for miR-142-3p, which directly targeted HOXA1.	mir-142-3p	39-49	homeobox A1	Homo sapiens	75-80
33410156-13	2021	Inhibition of miR-142-3p ameliorated the effects of circWHSC1 knockdown on HCC cell proliferation and metastasis.	mir-142-3p	14-24	nuclear receptor binding SET domain protein 2	Homo sapiens	52-61
33952723-7	2021	In vitro, miR-142-3p overexpression in human microglia cells (HMC3) modulated microglia activation, as shown by CD68 levels.	mir-142-3p	10-20	CD68 molecule	Homo sapiens	112-116
33555315-7	2021	Using a combination of TRAP-qPCR, miR-142-3p overexpression in vitro, and miR-142-null mice, we show that miR-142-3p negatively regulates cortical myelination.	mir-142-3p	106-116	microRNA 142	Mus musculus	34-41
33955243-11	2021	Knockdown of the miR-142-3p inhibited the expression level of HMGB1, the proliferation and migration of CEP cells, but it promoted apoptosis of CEP cells.	mir-142-3p	17-27	high mobility group box 1	Mus musculus	62-67
33500702-0	2021	miR-142-3p reduces the viability of human cervical cancer cells by negatively regulating the cytoplasmic localization of HMGB1.	mir-142-3p	0-10	high mobility group box 1	Homo sapiens	121-126
33500702-9	2021	Transfection with a miR-142-3p inhibitor increased cytoplasmic HMGB1 expression in HeLa cells, as shown by western blot analysis, while transfection with miR-142-3p mimics decreased the cytoplasmic expression of HMGB1 in HeLa cells.	mir-142-3p	20-30	high mobility group box 1	Homo sapiens	63-68
33500702-9	2021	Transfection with a miR-142-3p inhibitor increased cytoplasmic HMGB1 expression in HeLa cells, as shown by western blot analysis, while transfection with miR-142-3p mimics decreased the cytoplasmic expression of HMGB1 in HeLa cells.	mir-142-3p	20-30	high mobility group box 1	Homo sapiens	212-217
33500702-9	2021	Transfection with a miR-142-3p inhibitor increased cytoplasmic HMGB1 expression in HeLa cells, as shown by western blot analysis, while transfection with miR-142-3p mimics decreased the cytoplasmic expression of HMGB1 in HeLa cells.	mir-142-3p	154-164	high mobility group box 1	Homo sapiens	212-217
33500702-10	2021	Therefore, miR-142-3p negatively regulated HMGB1 levels in cervical cancer cells.	mir-142-3p	11-21	high mobility group box 1	Homo sapiens	43-48
33500702-11	2021	These findings indicated that miR-142-3p inhibited the proliferation of human cervical cancer cells, at least in part, by negatively regulating the cytoplasmic localization of HMGB1.	mir-142-3p	30-40	high mobility group box 1	Homo sapiens	176-181
33633466-7	2021	Besides, the interaction between miR-142-3p and circ_0020123 or ZFX was confirmed by dual-luciferase reporter assay and RNA immunoprecipitation (RIP) assay.	mir-142-3p	33-43	zinc finger protein X-linked	Homo sapiens	64-67
33633466-11	2021	ZFX could be targeted by miR-142-3p.	mir-142-3p	25-35	zinc finger protein X-linked	Homo sapiens	0-3
33633466-12	2021	The silencing of ZFX could hinder the progression of NSCLC and abolish the promotion effect of miR-142-3p inhibitor on NSCLC progression.	mir-142-3p	95-105	zinc finger protein X-linked	Homo sapiens	17-20
33621197-8	2021	CONCLUSION: miR-142-3p promotes the development of diabetes by inhibiting SPRED2-mediated autophagy, including inducing cell apoptosis, aggravating cellular oxidative stress and secretion of inflammatory factors, and resveratrol improves this effect.	mir-142-3p	12-22	sprouty related EVH1 domain containing 2	Homo sapiens	74-80
33461070-0	2021	Aberrantly up-regulated miR-142-3p inhibited the proliferation and invasion of trophoblast cells by regulating FOXM1.	mir-142-3p	24-34	forkhead box M1	Homo sapiens	111-116
33416140-0	2021	miR-142-3p targets AC9 to regulate sciatic nerve injury-induced neuropathic pain by regulating the cAMP/AMPK signalling pathway.	mir-142-3p	0-10	cathelicidin antimicrobial peptide	Rattus norvegicus	99-103
33416140-0	2021	miR-142-3p targets AC9 to regulate sciatic nerve injury-induced neuropathic pain by regulating the cAMP/AMPK signalling pathway.	mir-142-3p	0-10	protein kinase AMP-activated catalytic subunit alpha 2	Rattus norvegicus	104-108
33643894-9	2020	Interestingly, overexpression of miR-142-3p reduced the level and nuclear accumulation of beta-catenin.	mir-142-3p	33-43	catenin beta 1	Homo sapiens	90-102
33643894-10	2020	We further observed that miR-142-3p remarkably inhibited the transcriptional activity of beta-catenin gene (CTNNB1).	mir-142-3p	25-35	catenin beta 1	Homo sapiens	89-101
33643894-10	2020	We further observed that miR-142-3p remarkably inhibited the transcriptional activity of beta-catenin gene (CTNNB1).	mir-142-3p	25-35	catenin beta 1	Homo sapiens	108-114
33643894-11	2020	However, mutations in the predicted binding sites blocked this inhibition, suggesting that miR-142-3p may directly bind to the mRNA of beta-catenin.	mir-142-3p	91-101	catenin beta 1	Homo sapiens	135-147
33643894-12	2020	Conclusion: In conclusion, we identified miR-142-3p exerts its function as a tumor suppressor through blocking the activation of Wnt signaling by directly targeting to CTNNB1.	mir-142-3p	41-51	catenin beta 1	Homo sapiens	168-174
33461070-5	2021	The interaction between miR-142-3p and FOXM1 was confirmed by dual-luciferase reporter assay.	mir-142-3p	24-34	forkhead box M1	Homo sapiens	39-44
33461070-10	2021	Additionally, we found that miR-142-3p targeted FOXM1.	mir-142-3p	28-38	forkhead box M1	Homo sapiens	48-53
33461070-11	2021	Moreover, FOXM1 expression was negatively regulated by miR-142-3p.	mir-142-3p	55-65	forkhead box M1	Homo sapiens	10-15
33461070-13	2021	Reverse experiments determined that overexpression of FOXM1 reversed the suppressive effects of miR-142-3p on cell proliferation and migration.	mir-142-3p	96-106	forkhead box M1	Homo sapiens	54-59
33461070-14	2021	DISCUSSION: Our results demonstrated that miR-142-3p regulated cell proliferation and migration through targeting FOXM1 in trophoblast cells, providing a novel therapeutic target and extending the pathogenesis of preeclampsia.	mir-142-3p	42-52	forkhead box M1	Homo sapiens	114-119
33206362-6	2020	Finally, the effect of CD44 silencing on the expression of VEGF, CXCR4, MMP9, and MiR-142-3p was measured.	mir-142-3p	82-92	CD44 molecule (Indian blood group)	Homo sapiens	23-27
33272847-14	2021	miR-26a and miR-142-3p promoted CD4 + T cells proliferation in vitro through targeting Pten.	mir-142-3p	12-22	CD4 antigen	Mus musculus	32-35
33272847-14	2021	miR-26a and miR-142-3p promoted CD4 + T cells proliferation in vitro through targeting Pten.	mir-142-3p	12-22	phosphatase and tensin homolog	Mus musculus	87-91
33054738-0	2020	USP6NL mediated by LINC00689/miR-142-3p promotes the development of triple-negative breast cancer.	mir-142-3p	29-39	USP6 N-terminal like	Homo sapiens	0-6
32911017-0	2020	MiR-142-3p inhibits adipogenic differentiation and autophagy in obesity through targeting KLF9.	mir-142-3p	0-10	Kruppel-like factor 9	Mus musculus	90-94
32911017-11	2020	KLF9 targeting miR-142-3p was suppressed by miR-142-3p.	mir-142-3p	15-25	Kruppel like factor 9	Homo sapiens	0-4
32911017-11	2020	KLF9 targeting miR-142-3p was suppressed by miR-142-3p.	mir-142-3p	44-54	Kruppel like factor 9	Homo sapiens	0-4
32911017-12	2020	KLF9 overexpression partially reversed the inhibitory effect of miR-142-3p mimic on adipogenic differentiation and the expressions of autophagy related-genes in 3T3-L1 cells.	mir-142-3p	64-74	Kruppel-like factor 9	Mus musculus	0-4
32911017-14	2020	CONCLUSION: Overexpressed miR-142-3p inhibited adipogenic differentiation and autophagy through targeting KLF9.	mir-142-3p	26-36	Kruppel-like factor 9	Mus musculus	106-110
33177878-10	2020	Moreover, miR-142-3p mimic enhanced HCC cell proliferation, migration, invasion and cell cycle, while its effects were abolished by Bach-1 overexpression.	mir-142-3p	10-20	BTB domain and CNC homolog 1	Homo sapiens	132-138
32866906-14	2020	Moreover, histone acetyltransferase p300 suppressed miR-142-3p expression.	mir-142-3p	52-62	E1A binding protein p300	Homo sapiens	36-40
33054738-11	2020	Further, USP6NL was proved as the target of a tumor-inhibitor miR-142-3p, and LINC00689 augmented USP6NL expression by absorbing miR-142-3p.	mir-142-3p	62-72	USP6 N-terminal like	Homo sapiens	9-15
33054738-11	2020	Further, USP6NL was proved as the target of a tumor-inhibitor miR-142-3p, and LINC00689 augmented USP6NL expression by absorbing miR-142-3p.	mir-142-3p	62-72	long intergenic non-protein coding RNA 689	Homo sapiens	78-87
33054738-12	2020	Importantly, miR-142-3p deficiency or USP6NL overexpression fully abolished the inhibitory effect of LINC00689 silence on TNBC cellular behaviors.	mir-142-3p	13-23	long intergenic non-protein coding RNA 689	Homo sapiens	101-110
33061617-14	2020	Besides, miR-142-3p bound to ARL2, and the inhibitory effects of miR-142-3p restoration on cell proliferation, cycle, migration and invasion were counteracted by ARL2 overexpression.	mir-142-3p	9-19	ADP ribosylation factor like GTPase 2	Homo sapiens	29-33
33061617-14	2020	Besides, miR-142-3p bound to ARL2, and the inhibitory effects of miR-142-3p restoration on cell proliferation, cycle, migration and invasion were counteracted by ARL2 overexpression.	mir-142-3p	9-19	ADP ribosylation factor like GTPase 2	Homo sapiens	162-166
33061617-14	2020	Besides, miR-142-3p bound to ARL2, and the inhibitory effects of miR-142-3p restoration on cell proliferation, cycle, migration and invasion were counteracted by ARL2 overexpression.	mir-142-3p	65-75	ADP ribosylation factor like GTPase 2	Homo sapiens	29-33
33061617-14	2020	Besides, miR-142-3p bound to ARL2, and the inhibitory effects of miR-142-3p restoration on cell proliferation, cycle, migration and invasion were counteracted by ARL2 overexpression.	mir-142-3p	65-75	ADP ribosylation factor like GTPase 2	Homo sapiens	162-166
32635856-10	2020	Additionally, MSCs exosomes inhibited NPCs apoptosis and MAPK signaling by delivering miR-142-3p that targets mixed lineage kinase 3 (MLK3).	mir-142-3p	86-96	mitogen-activated protein kinase kinase kinase 11	Homo sapiens	110-132
32824678-6	2020	On the functional side, miR-142-3p upregulation significantly reduced ST-T1b cell size, the size of vinculin plaques, migration through fibronectin-coated transwell filters, and the ability of ST-T1b and primary endometriotic stroma cells to contract collagen I gels.	mir-142-3p	24-34	vinculin	Homo sapiens	100-108
32824678-6	2020	On the functional side, miR-142-3p upregulation significantly reduced ST-T1b cell size, the size of vinculin plaques, migration through fibronectin-coated transwell filters, and the ability of ST-T1b and primary endometriotic stroma cells to contract collagen I gels.	mir-142-3p	24-34	fibronectin 1	Homo sapiens	136-147
32325105-0	2020	Repression of miR-142-3p alleviates psoriasis-like inflammation by repressing proliferation and promoting apoptosis of keratinocytes via targeting Sema3A.	mir-142-3p	14-24	semaphorin 3A	Homo sapiens	147-153
32325105-5	2020	In addition, inhibition of miR-142-3p dramatically restricted cell proliferation and enhanced apoptosis in HaCaT cells exposed to M5, as exemplified by a decrease in the antiapoptotic Bcl-2 protein, concomitant with an increase in the proapoptotic proteins Bax.	mir-142-3p	27-37	BCL2 apoptosis regulator	Homo sapiens	184-189
32325105-5	2020	In addition, inhibition of miR-142-3p dramatically restricted cell proliferation and enhanced apoptosis in HaCaT cells exposed to M5, as exemplified by a decrease in the antiapoptotic Bcl-2 protein, concomitant with an increase in the proapoptotic proteins Bax.	mir-142-3p	27-37	BCL2 associated X, apoptosis regulator	Homo sapiens	257-260
32325105-8	2020	Mechanistically, silencing of Sema3A effectively abolished the anti-proliferative, apoptosis-promoting, and anti-inflammatory effects of miR-142-3p inhibition in keratinocytes.	mir-142-3p	137-147	semaphorin 3A	Homo sapiens	30-36
32635856-10	2020	Additionally, MSCs exosomes inhibited NPCs apoptosis and MAPK signaling by delivering miR-142-3p that targets mixed lineage kinase 3 (MLK3).	mir-142-3p	86-96	mitogen-activated protein kinase kinase kinase 11	Homo sapiens	134-138
32635856-12	2020	The results confirmed that bone marrow MSCs-derived exosomes-packaged miR-142-3p alleviates NPCs injury through suppressing MAPK signaling by targeting MLK3.	mir-142-3p	70-80	mitogen-activated protein kinase kinase kinase 11	Homo sapiens	152-156
32642410-0	2020	MiR-142-3p enhances chemosensitivity of breast cancer cells and inhibits autophagy by targeting HMGB1.	mir-142-3p	0-10	high mobility group box 1	Homo sapiens	96-101
32560222-12	2020	Notably, an increased level of the tumor suppressor, miR-142-3p, whose targets include LGR5, IL-6, and ABCG2, was detected in association with MSI-N1014 treatment.	mir-142-3p	53-63	leucine rich repeat containing G protein-coupled receptor 5	Homo sapiens	87-91
32560222-12	2020	Notably, an increased level of the tumor suppressor, miR-142-3p, whose targets include LGR5, IL-6, and ABCG2, was detected in association with MSI-N1014 treatment.	mir-142-3p	53-63	interleukin 6	Homo sapiens	93-97
32560222-12	2020	Notably, an increased level of the tumor suppressor, miR-142-3p, whose targets include LGR5, IL-6, and ABCG2, was detected in association with MSI-N1014 treatment.	mir-142-3p	53-63	ATP binding cassette subfamily G member 2 (Junior blood group)	Homo sapiens	103-108
32642410-6	2020	Moreover, overexpression of HMGB1 dramatically reversed the promotion of apoptosis and inhibition of autophagy mediated by miR-142-3p up-regulation.	mir-142-3p	123-133	high mobility group box 1	Homo sapiens	28-33
32642410-7	2020	In conclusion, miR-142-3p overexpression may inhibit autophagy and promote the drug sensitivity of breast cancer cells to DOX by targeting HMGB1.	mir-142-3p	15-25	high mobility group box 1	Homo sapiens	139-144
32642410-8	2020	The miR-142-3p/HMGB1 axis might be a novel target to regulate the drug resistance of breast cancer patients.	mir-142-3p	4-14	high mobility group box 1	Homo sapiens	15-20
31619579-5	2019	SBF2-AS1 inhibited the expression of TWF1 by competitively binding with miR-142-3p in pancreatic cancer.	mir-142-3p	72-82	SBF2 antisense RNA 1	Homo sapiens	0-8
32269710-12	2020	miR-142-3p was expressed at low levels in LSECs and negatively regulated autophagy and EndMT by reducing ATG5 expression.	mir-142-3p	0-10	autophagy related 5	Rattus norvegicus	105-109
31836002-14	2019	MiR-142-3p over-expression played an opposite role and attenuated the effects of MCM3AP-AS1 over-expression.	mir-142-3p	0-10	MCM3AP antisense RNA 1	Homo sapiens	81-91
32508597-10	2020	Overexpression of miR-142-3p in microglia was found to decrease the expression of CAMK2A and subsequently BDNF through regulation of CREB phosphorylation.	mir-142-3p	18-28	calcium/calmodulin dependent protein kinase II alpha	Homo sapiens	82-88
32508597-10	2020	Overexpression of miR-142-3p in microglia was found to decrease the expression of CAMK2A and subsequently BDNF through regulation of CREB phosphorylation.	mir-142-3p	18-28	brain derived neurotrophic factor	Homo sapiens	106-110
32508597-10	2020	Overexpression of miR-142-3p in microglia was found to decrease the expression of CAMK2A and subsequently BDNF through regulation of CREB phosphorylation.	mir-142-3p	18-28	cAMP responsive element binding protein 1	Homo sapiens	133-137
33335754-6	2020	Results: It was evidenced that the expression of the number of miRs was altered on the 14th day post-SCI, and miR-142-3p was found to be the most significantly suppressed miR.	mir-142-3p	110-120	membrane associated ring-CH-type finger 8	Rattus norvegicus	63-66
33335754-9	2020	The findings suggested that miR-142-3p may inhibit the MAP by inhibiting the expression of cleaved-caspase-3/-9 and Bax in SCI rats.	mir-142-3p	28-38	BCL2 associated X, apoptosis regulator	Rattus norvegicus	116-119
32327611-0	2020	Activated CD4+ T cells-derived exosomal miR-142-3p boosts post-ischemic ventricular remodeling by activating myofibroblast.	mir-142-3p	40-50	CD4 molecule	Homo sapiens	10-13
32327611-4	2020	Mechanistically, miR-142-3p that was enriched in CD4-activated Exos recapitulated the pro-fibrotic effects of CD4-activated Exos in cardiac fibroblasts, and vice versa.	mir-142-3p	17-27	CD4 molecule	Homo sapiens	49-52
32327611-4	2020	Mechanistically, miR-142-3p that was enriched in CD4-activated Exos recapitulated the pro-fibrotic effects of CD4-activated Exos in cardiac fibroblasts, and vice versa.	mir-142-3p	17-27	CD4 molecule	Homo sapiens	110-113
32327611-5	2020	Furthermore, miR-142-3p directly targeted and inhibited the expression of Adenomatous Polyposis Coli (APC), a negative WNT signaling pathway regulator, contributing to the activation of WNT signaling pathway and cardiac fibroblast activation.	mir-142-3p	13-23	APC regulator of WNT signaling pathway	Homo sapiens	74-100
32327611-7	2020	Targeting miR-142-3p in CD4-activated Exos may hold promise for treating cardiac remodeling post-MI.	mir-142-3p	10-20	CD4 molecule	Homo sapiens	24-27
31511910-3	2019	As miR-142-3p has recently been reported to be a ligand and activator of TLR7, a detector of exogenous and endogenous single-stranded RNA, we evaluated TLR7 expression and downstream IL23A activation in surgically resected TSC brain tissue.	mir-142-3p	3-13	toll like receptor 7	Homo sapiens	73-77
31425004-4	2019	Overexpression of miR-142-3p or knockdown of KLF9 significantly suppressed CRL-7566 cell proliferation and metastasis, induced cell apoptosis, and decreased both cell autophagy and vascularization.	mir-142-3p	18-28	interleukin 31 receptor A	Homo sapiens	75-78
31619579-5	2019	SBF2-AS1 inhibited the expression of TWF1 by competitively binding with miR-142-3p in pancreatic cancer.	mir-142-3p	72-82	twinfilin actin binding protein 1	Homo sapiens	37-41
31619579-6	2019	CONCLUSION: Our study demonstrates that knock-down of SBF2-AS1 inhibits the expression of TWF1 by competitively binding with miR-142-3p to induce gemcitabine resistance in pancreatic cancer.	mir-142-3p	125-135	SBF2 antisense RNA 1	Homo sapiens	54-62
31619579-6	2019	CONCLUSION: Our study demonstrates that knock-down of SBF2-AS1 inhibits the expression of TWF1 by competitively binding with miR-142-3p to induce gemcitabine resistance in pancreatic cancer.	mir-142-3p	125-135	twinfilin actin binding protein 1	Homo sapiens	90-94
30480817-0	2019	miR-142-3p as tumor suppressor miRNA in the regulation of tumorigenicity, invasion and migration of human breast cancer by targeting Bach-1 expression.	mir-142-3p	0-10	BTB domain and CNC homolog 1	Homo sapiens	133-139
31114934-6	2019	The regulatory effect of miR-142-3p on FAM98A was interrogated by luciferase reporter assay.	mir-142-3p	25-35	family with sequence similarity 98 member A	Homo sapiens	39-45
31114934-11	2019	Mechanistically, it was demonstrated that miR-142-3p directly targeted FAM98A, and modulated its expression.	mir-142-3p	42-52	family with sequence similarity 98 member A	Homo sapiens	71-77
31239367-10	2019	miR-142-3p inhibitor significantly decreased the cell viability, the number of cell clones, the migration rate, the number of invasive cells, the contents and expression of IL-6 and MMP-3, and increased the apoptosis rate and the expressions of Bax and Bad, and decreased Bcl-2 expression of TNF-alpha-treated RA-HFLSs.	mir-142-3p	0-10	interleukin 6	Homo sapiens	173-177
31239367-10	2019	miR-142-3p inhibitor significantly decreased the cell viability, the number of cell clones, the migration rate, the number of invasive cells, the contents and expression of IL-6 and MMP-3, and increased the apoptosis rate and the expressions of Bax and Bad, and decreased Bcl-2 expression of TNF-alpha-treated RA-HFLSs.	mir-142-3p	0-10	matrix metallopeptidase 3	Homo sapiens	182-187
31239367-10	2019	miR-142-3p inhibitor significantly decreased the cell viability, the number of cell clones, the migration rate, the number of invasive cells, the contents and expression of IL-6 and MMP-3, and increased the apoptosis rate and the expressions of Bax and Bad, and decreased Bcl-2 expression of TNF-alpha-treated RA-HFLSs.	mir-142-3p	0-10	BCL2 associated X, apoptosis regulator	Homo sapiens	245-248
31239367-10	2019	miR-142-3p inhibitor significantly decreased the cell viability, the number of cell clones, the migration rate, the number of invasive cells, the contents and expression of IL-6 and MMP-3, and increased the apoptosis rate and the expressions of Bax and Bad, and decreased Bcl-2 expression of TNF-alpha-treated RA-HFLSs.	mir-142-3p	0-10	BCL2 apoptosis regulator	Homo sapiens	272-277
31239367-10	2019	miR-142-3p inhibitor significantly decreased the cell viability, the number of cell clones, the migration rate, the number of invasive cells, the contents and expression of IL-6 and MMP-3, and increased the apoptosis rate and the expressions of Bax and Bad, and decreased Bcl-2 expression of TNF-alpha-treated RA-HFLSs.	mir-142-3p	0-10	tumor necrosis factor	Homo sapiens	292-301
31239367-11	2019	MiR-142-3p inhibitor significantly reversed TNF-alpha-induced up-regulation of IRAK1, TLR4, and p-NF-kappaB p65 in TNF-alpha-treated RA-HFLSs.	mir-142-3p	0-10	tumor necrosis factor	Homo sapiens	44-53
31239367-11	2019	MiR-142-3p inhibitor significantly reversed TNF-alpha-induced up-regulation of IRAK1, TLR4, and p-NF-kappaB p65 in TNF-alpha-treated RA-HFLSs.	mir-142-3p	0-10	interleukin 1 receptor associated kinase 1	Homo sapiens	79-84
31239367-11	2019	MiR-142-3p inhibitor significantly reversed TNF-alpha-induced up-regulation of IRAK1, TLR4, and p-NF-kappaB p65 in TNF-alpha-treated RA-HFLSs.	mir-142-3p	0-10	toll like receptor 4	Homo sapiens	86-90
31239367-11	2019	MiR-142-3p inhibitor significantly reversed TNF-alpha-induced up-regulation of IRAK1, TLR4, and p-NF-kappaB p65 in TNF-alpha-treated RA-HFLSs.	mir-142-3p	0-10	RELA proto-oncogene, NF-kB subunit	Homo sapiens	108-111
31239367-11	2019	MiR-142-3p inhibitor significantly reversed TNF-alpha-induced up-regulation of IRAK1, TLR4, and p-NF-kappaB p65 in TNF-alpha-treated RA-HFLSs.	mir-142-3p	0-10	tumor necrosis factor	Homo sapiens	115-124
30353102-0	2019	EZH2-DNMT1-mediated epigenetic silencing of miR-142-3p promotes metastasis through targeting ZEB2 in nasopharyngeal carcinoma.	mir-142-3p	44-54	enhancer of zeste 2 polycomb repressive complex 2 subunit	Homo sapiens	0-4
30353102-0	2019	EZH2-DNMT1-mediated epigenetic silencing of miR-142-3p promotes metastasis through targeting ZEB2 in nasopharyngeal carcinoma.	mir-142-3p	44-54	DNA methyltransferase 1	Homo sapiens	5-10
30353102-0	2019	EZH2-DNMT1-mediated epigenetic silencing of miR-142-3p promotes metastasis through targeting ZEB2 in nasopharyngeal carcinoma.	mir-142-3p	44-54	zinc finger E-box binding homeobox 2	Homo sapiens	93-97
30353102-4	2019	miR-142 locus hypermethylation was observed and found to be associated with miR-142-3p downregulation in metastatic NPC.	mir-142-3p	76-86	microRNA 142	Homo sapiens	0-7
30353102-5	2019	Furthermore, miR-142-3p was epigenetically silenced by EZH2-recruited DNMT1 and suppressed NPC cell metastasis and EMT.	mir-142-3p	13-23	enhancer of zeste 2 polycomb repressive complex 2 subunit	Homo sapiens	55-59
30353102-5	2019	Furthermore, miR-142-3p was epigenetically silenced by EZH2-recruited DNMT1 and suppressed NPC cell metastasis and EMT.	mir-142-3p	13-23	DNA methyltransferase 1	Homo sapiens	70-75
30353102-9	2019	This study identifies miR-142-3p as a key suppressive regulator in NPC metastasis and reveals a DNMT1-mediated epigenetic mechanism for miR-142-3p silencing, providing a potential prognostic marker and therapeutic target to combat NPC metastasis.	mir-142-3p	136-146	DNA methyltransferase 1	Homo sapiens	96-101
31322790-8	2019	These include TGFBR1 and PICALM, of which their derepression in the brain due to reduced expression levels of miR-142-3p may reduce the risk of AD.	mir-142-3p	110-120	transforming growth factor beta receptor 1	Homo sapiens	14-20
31322790-8	2019	These include TGFBR1 and PICALM, of which their derepression in the brain due to reduced expression levels of miR-142-3p may reduce the risk of AD.	mir-142-3p	110-120	phosphatidylinositol binding clathrin assembly protein	Homo sapiens	25-31
31145943-0	2019	Inhibition of lncRNA TUG1 upregulates miR-142-3p to ameliorate myocardial injury during ischemia and reperfusion via targeting HMGB1- and Rac1-induced autophagy.	mir-142-3p	38-48	taurine up-regulated 1	Homo sapiens	21-25
31145943-0	2019	Inhibition of lncRNA TUG1 upregulates miR-142-3p to ameliorate myocardial injury during ischemia and reperfusion via targeting HMGB1- and Rac1-induced autophagy.	mir-142-3p	38-48	high mobility group box 1	Homo sapiens	127-132
31145943-0	2019	Inhibition of lncRNA TUG1 upregulates miR-142-3p to ameliorate myocardial injury during ischemia and reperfusion via targeting HMGB1- and Rac1-induced autophagy.	mir-142-3p	38-48	Rac family small GTPase 1	Homo sapiens	138-142
31145943-12	2019	The function of TUG1 were achieved by sponging miR-142-3p and releasing the suppression of the putative targets of miR-142-3p, HMGB1 and Rac1.	mir-142-3p	47-57	taurine up-regulated 1	Homo sapiens	16-20
31145943-12	2019	The function of TUG1 were achieved by sponging miR-142-3p and releasing the suppression of the putative targets of miR-142-3p, HMGB1 and Rac1.	mir-142-3p	115-125	taurine up-regulated 1	Homo sapiens	16-20
30480817-11	2019	Furthermore, replacement of miR-142-3p could inhibit the proliferation, invasion, and migration in breast cancer potentially by targeting of Bach-1 mRNA and subsequent inhibition of CXCR4, MMP9, and VEGFR protein expressions.	mir-142-3p	28-38	BTB domain and CNC homolog 1	Homo sapiens	141-147
30480817-11	2019	Furthermore, replacement of miR-142-3p could inhibit the proliferation, invasion, and migration in breast cancer potentially by targeting of Bach-1 mRNA and subsequent inhibition of CXCR4, MMP9, and VEGFR protein expressions.	mir-142-3p	28-38	C-X-C motif chemokine receptor 4	Homo sapiens	182-187
30480817-11	2019	Furthermore, replacement of miR-142-3p could inhibit the proliferation, invasion, and migration in breast cancer potentially by targeting of Bach-1 mRNA and subsequent inhibition of CXCR4, MMP9, and VEGFR protein expressions.	mir-142-3p	28-38	matrix metallopeptidase 9	Homo sapiens	189-193
30480817-11	2019	Furthermore, replacement of miR-142-3p could inhibit the proliferation, invasion, and migration in breast cancer potentially by targeting of Bach-1 mRNA and subsequent inhibition of CXCR4, MMP9, and VEGFR protein expressions.	mir-142-3p	28-38	kinase insert domain receptor	Homo sapiens	199-204
30480817-12	2019	In addition, induction of miR-142-3p could upregulate tumor suppressor miRNAs, including miR-330, miR-145, and miR34a.	mir-142-3p	26-36	microRNA 330	Homo sapiens	89-96
30480817-12	2019	In addition, induction of miR-142-3p could upregulate tumor suppressor miRNAs, including miR-330, miR-145, and miR34a.	mir-142-3p	26-36	microRNA 145	Homo sapiens	98-105
30480817-12	2019	In addition, induction of miR-142-3p could upregulate tumor suppressor miRNAs, including miR-330, miR-145, and miR34a.	mir-142-3p	26-36	microRNA 34a	Homo sapiens	111-117
30480817-13	2019	CONCLUSION: For the first time, our results revealed that miR-142-3p could target Bach-1in breast cancer cells leading to the reduction of EMT-related proteins and reduced cell proliferation, invasion, and migration.	mir-142-3p	58-68	BTB domain and CNC homolog 1	Homo sapiens	82-88
30480817-6	2019	miR-142-3p targeting of Bach-1 expression in MCF-7 and MDA-MB-468 breast cancer cells was evaluated using bioinformatics, qRT-PCR and western blot analyses.	mir-142-3p	0-10	BTB domain and CNC homolog 1	Homo sapiens	24-30
30244433-0	2018	Mononuclear-cell-derived microparticles attenuate endothelial inflammation by transfer of miR-142-3p in a CD39 dependent manner.	mir-142-3p	90-100	ectonucleoside triphosphate diphosphohydrolase 1	Mus musculus	106-110
30362621-5	2019	We demonstrate that transfer of miR-142-3p in LAC EVs to endothelial cells promotes angiogenesis through inhibition of TGFbetaR1.	mir-142-3p	32-42	lactase	Homo sapiens	46-49
30362621-7	2019	These findings suggest that miR-142-3p within LAC EVs can be transferred from LAC cells to both endothelial and fibroblast cells to promote tumor associated changes.	mir-142-3p	28-38	lactase	Homo sapiens	46-49
30362621-7	2019	These findings suggest that miR-142-3p within LAC EVs can be transferred from LAC cells to both endothelial and fibroblast cells to promote tumor associated changes.	mir-142-3p	28-38	lactase	Homo sapiens	78-81
30683933-0	2019	The mechanism of miR-142-3p in coronary microembolization-induced myocardiac injury via regulating target gene IRAK-1.	mir-142-3p	17-27	interleukin 1 receptor associated kinase 1	Homo sapiens	111-117
29459719-0	2018	miR-142-3p regulates autophagy by targeting ATG16L1 in thymic-derived regulatory T cell (tTreg).	mir-142-3p	0-10	autophagy related 16 like 1	Homo sapiens	44-51
29459719-6	2018	We demonstrate that miR-142-3p downregulates ATG16L1 mRNA and production of ATG16L1, that has been linked to autoimmune diseases.	mir-142-3p	20-30	autophagy related 16 like 1	Homo sapiens	45-52
29459719-6	2018	We demonstrate that miR-142-3p downregulates ATG16L1 mRNA and production of ATG16L1, that has been linked to autoimmune diseases.	mir-142-3p	20-30	autophagy related 16 like 1	Homo sapiens	76-83
29459719-8	2018	In aggregate, these studies provide a new approach that uses miR-142-3p knockdown to increase tTreg cell efficacy by increasing ATG16L1 mRNA and protein and the autophagy process.	mir-142-3p	61-71	autophagy related 16 like 1	Homo sapiens	128-135
30092578-8	2018	miR-142-3p was found to be a prognostic factor of HCC, and the mechanism by which TUG1 upregulated ZEB1 was via direct binding to miR-142-3p.	mir-142-3p	0-10	taurine upregulated gene 1	Mus musculus	82-86
29360449-9	2018	miR-142-3p-induced deduction of aerobic glycolysis and proliferation were reversed by LDHA overexpression.	mir-142-3p	0-10	lactate dehydrogenase A	Homo sapiens	86-90
30092578-8	2018	miR-142-3p was found to be a prognostic factor of HCC, and the mechanism by which TUG1 upregulated ZEB1 was via direct binding to miR-142-3p.	mir-142-3p	130-140	taurine upregulated gene 1	Mus musculus	82-86
30092578-8	2018	miR-142-3p was found to be a prognostic factor of HCC, and the mechanism by which TUG1 upregulated ZEB1 was via direct binding to miR-142-3p.	mir-142-3p	130-140	zinc finger E-box binding homeobox 1	Mus musculus	99-103
29742504-0	2018	MiR-142-3p Enhances Cell Viability and Inhibits Apoptosis by Targeting CDKN1B and TIMP3 Following Sciatic Nerve Injury.	mir-142-3p	0-10	cyclin-dependent kinase inhibitor 1B	Rattus norvegicus	71-77
30513513-0	2018	miR-142-3p Suppresses Cell Growth by Targeting CDK4 in Colorectal Cancer.	mir-142-3p	0-10	cyclin-dependent kinase 4	Mus musculus	47-51
29590647-10	2018	RESULTS: We revealed a reversion of TGF-beta3-induced BTB impairment after miR-142-3p treatment both in vitro and in vivo.	mir-142-3p	75-85	transforming growth factor, beta 3	Rattus norvegicus	36-45
29742504-0	2018	MiR-142-3p Enhances Cell Viability and Inhibits Apoptosis by Targeting CDKN1B and TIMP3 Following Sciatic Nerve Injury.	mir-142-3p	0-10	TIMP metallopeptidase inhibitor 3	Rattus norvegicus	82-87
29115575-11	2018	Furthermore, overexpression of HMGB1 significantly reversed the inhibitory effect of miR-142-3p on neuroinflammation and neuropathic pain development (P&lt;0.05).	mir-142-3p	85-95	high mobility group box 1	Mus musculus	31-36
29949787-14	2018	Finally, systemic treatment with miR-142-3p antagomir attenuated endothelial apoptosis and retarded the progression of atherosclerosis in the aorta of ApoE-/- mice.	mir-142-3p	33-43	apolipoprotein E	Mus musculus	151-155
30198432-0	2018	miR-142-3p Inhibits the Metastasis of Hepatocellular Carcinoma Cells by Regulating HMGB1 Gene Expression.	mir-142-3p	0-10	high mobility group box 1	Homo sapiens	83-88
28823564-0	2017	MiR-142-3p blocks TGF-beta-induced activation of hepatic stellate cells through targeting TGFbetaRI.	mir-142-3p	0-10	transforming growth factor beta 1	Homo sapiens	18-26
28823564-0	2017	MiR-142-3p blocks TGF-beta-induced activation of hepatic stellate cells through targeting TGFbetaRI.	mir-142-3p	0-10	transforming growth factor beta receptor 1	Homo sapiens	90-99
28100738-15	2017	miR-142-3p was upregulated in the CSF of MS patients and in EAE cerebellum.	mir-142-3p	0-10	colony stimulating factor 2	Homo sapiens	34-37
28146434-6	2017	Conversely, increased excretion of miR-142-3p via donor cell SEVs and uptake by recipient endothelial cells was found to reduce TGFBR1 activity and cause tumor-promoting changes in these cells in vitro and in vivo.	mir-142-3p	35-45	transforming growth factor beta receptor 1	Homo sapiens	128-134
28100738-6	2017	We propose miR-142-3p as a molecular mediator of the IL-1beta-dependent downregulation of the glial glutamate-aspartate transporter (GLAST), which causes an enhancement of the glutamatergic transmission in the EAE cerebellum.	mir-142-3p	11-21	interleukin 1 beta	Homo sapiens	53-61
25976503-6	2015	Overexpression of FZD7 expression was able to reverse the inhibitory effects induced by miR-142-3p.	mir-142-3p	88-98	frizzled class receptor 7	Homo sapiens	18-22
28100738-6	2017	We propose miR-142-3p as a molecular mediator of the IL-1beta-dependent downregulation of the glial glutamate-aspartate transporter (GLAST), which causes an enhancement of the glutamatergic transmission in the EAE cerebellum.	mir-142-3p	11-21	solute carrier family 1 member 3	Homo sapiens	133-138
28025989-10	2016	Taken together, the findings of the present study demonstrate that miR-142-3p inhibits H/R-induced apoptosis and fibrosis of cardiomyocytes, partly at least, by the direct inhibition of HMGB1 expression.	mir-142-3p	67-77	high mobility group box 1	Mus musculus	186-191
27527863-7	2016	Overexpression of Bod1 inhibited radiation- and miR-142-3p-induced premature chromatid separation and increased resistance to radiation in 786-O and A549 cells.	mir-142-3p	48-58	biorientation of chromosomes in cell division 1	Homo sapiens	18-22
28656050-7	2017	We further demonstrated that miR-142-3p targeted the 3"-untranslated region of Mef2c.	mir-142-3p	29-39	myocyte enhancer factor 2C	Mus musculus	79-84
28275790-3	2016	The target relationship between miR-142-3p and interleukin-1 receptor- associated kinase 1 (IRAK1) was detected by dual-luciferase reporter assay.	mir-142-3p	32-42	interleukin 1 receptor associated kinase 1	Homo sapiens	47-90
28275790-3	2016	The target relationship between miR-142-3p and interleukin-1 receptor- associated kinase 1 (IRAK1) was detected by dual-luciferase reporter assay.	mir-142-3p	32-42	interleukin 1 receptor associated kinase 1	Homo sapiens	92-97
27113904-0	2016	MiR-142-3p is a RANKL-dependent inducer of cell death in osteoclasts.	mir-142-3p	0-10	TNF superfamily member 11	Homo sapiens	16-21
24361879-0	2014	miR-142-3p enhances FcepsilonRI-mediated degranulation in mast cells.	mir-142-3p	0-10	Fc epsilon receptor Ia	Homo sapiens	20-31
24558198-0	2014	MiR-142-3p represses TGF-beta-induced growth inhibition through repression of TGFbetaR1 in non-small cell lung cancer.	mir-142-3p	0-10	transforming growth factor beta 1	Homo sapiens	21-29
23152889-9	2012	Inhibiting endogenous miR-142-3p in CAD cells increased endogenous D1 receptor protein expression levels.	mir-142-3p	22-32	dopamine receptor D1	Mus musculus	67-78
23635652-11	2013	This is the first description of an miRNA-mediated mechanism of HSP70 regulation in cancer, making miR-142-3p an attractive target for PDAC therapeutic intervention.	mir-142-3p	99-109	heat shock protein family A (Hsp70) member 4	Homo sapiens	64-69