PMID-sentid	Pub_year	Sent_text	compound_name	comp_offset	prot_official_name	organism	prot_offset
34630643-12	2021	Overall, the present results suggested that miR-24 improves myocardial injury in rats by inhibiting the NF-kappaB/TNF-alpha pathway.	mir-24	44-50	tumor necrosis factor	Rattus norvegicus	114-123
34412561-12	2022	Furthermore, the impact of applying miR-24-3p inhibitor on PsA and hypoxia-treated cells could be reversed by si-p16.	mir-24	36-42	cyclin-dependent kinase inhibitor 2A	Rattus norvegicus	113-116
34581696-0	2021	miR-24 alleviates MI/RI by blocking the S100A8/TLR4/MyD88/NF-kappaB pathway.	mir-24	0-6	S100 calcium binding protein A8	Rattus norvegicus	40-46
34581696-0	2021	miR-24 alleviates MI/RI by blocking the S100A8/TLR4/MyD88/NF-kappaB pathway.	mir-24	0-6	toll-like receptor 4	Rattus norvegicus	47-51
34581696-0	2021	miR-24 alleviates MI/RI by blocking the S100A8/TLR4/MyD88/NF-kappaB pathway.	mir-24	0-6	MYD88, innate immune signal transduction adaptor	Rattus norvegicus	52-57
34581696-6	2021	In contrast to the level of S100A8, the level of miR-24 in myocardial tissue was significantly reduced after 30 min of ischemia followed by reperfusion for 2 h. Overexpression of miR-24 reduced the myocardial infarction area and improved the heart function of rats 3 days after MI/RI.	mir-24	49-55	S100 calcium binding protein A8	Rattus norvegicus	28-34
34581696-6	2021	In contrast to the level of S100A8, the level of miR-24 in myocardial tissue was significantly reduced after 30 min of ischemia followed by reperfusion for 2 h. Overexpression of miR-24 reduced the myocardial infarction area and improved the heart function of rats 3 days after MI/RI.	mir-24	179-185	S100 calcium binding protein A8	Rattus norvegicus	28-34
34581696-8	2021	Interestingly, miR-24 upregulation reduced S100A8 expression, followed by inhibition of TLR4/MyD-88/NF-kappaB signaling activation.	mir-24	15-21	S100 calcium binding protein A8	Rattus norvegicus	43-49
34581696-8	2021	Interestingly, miR-24 upregulation reduced S100A8 expression, followed by inhibition of TLR4/MyD-88/NF-kappaB signaling activation.	mir-24	15-21	toll-like receptor 4	Rattus norvegicus	88-92
34581696-8	2021	Interestingly, miR-24 upregulation reduced S100A8 expression, followed by inhibition of TLR4/MyD-88/NF-kappaB signaling activation.	mir-24	15-21	MYD88, innate immune signal transduction adaptor	Rattus norvegicus	93-99
34581696-9	2021	In conclusion, miR-24 can alleviate MI/RI via inactivation of the S100A8/TLR4/MyD-88/NF-kappaB signaling pathway.	mir-24	15-21	S100 calcium binding protein A8	Rattus norvegicus	66-72
34581696-9	2021	In conclusion, miR-24 can alleviate MI/RI via inactivation of the S100A8/TLR4/MyD-88/NF-kappaB signaling pathway.	mir-24	15-21	toll-like receptor 4	Rattus norvegicus	73-77
34581696-9	2021	In conclusion, miR-24 can alleviate MI/RI via inactivation of the S100A8/TLR4/MyD-88/NF-kappaB signaling pathway.	mir-24	15-21	MYD88, innate immune signal transduction adaptor	Rattus norvegicus	78-84
34858468-12	2021	A sexually dimorphic response of miR-24 may thus be at the basis of the sex difference in Sox6 expression changes following exposure to the three chemicals.	mir-24	33-39	SRY-box transcription factor 6	Rattus norvegicus	90-94
33032910-5	2020	A luciferase assay using a plasmid containing the 3"-UTR of OATP2B1 gene demonstrated that only miR-24 significantly reduced its expression.	mir-24	96-102	solute carrier organic anion transporter family member 2B1	Homo sapiens	60-67
33032910-6	2020	The overexpression of miR-24 decreased the expression of OATP2B1 mRNA and protein in HEK/OATP2B1 and Caco-2 cells and uptake of [3H]estrone-3-sulfate by HEK/OATP2B1 cells.	mir-24	22-28	solute carrier organic anion transporter family member 2B1	Homo sapiens	57-64
33032910-6	2020	The overexpression of miR-24 decreased the expression of OATP2B1 mRNA and protein in HEK/OATP2B1 and Caco-2 cells and uptake of [3H]estrone-3-sulfate by HEK/OATP2B1 cells.	mir-24	22-28	EPH receptor A3	Homo sapiens	85-88
33032910-6	2020	The overexpression of miR-24 decreased the expression of OATP2B1 mRNA and protein in HEK/OATP2B1 and Caco-2 cells and uptake of [3H]estrone-3-sulfate by HEK/OATP2B1 cells.	mir-24	22-28	solute carrier organic anion transporter family member 2B1	Homo sapiens	89-96
33032910-6	2020	The overexpression of miR-24 decreased the expression of OATP2B1 mRNA and protein in HEK/OATP2B1 and Caco-2 cells and uptake of [3H]estrone-3-sulfate by HEK/OATP2B1 cells.	mir-24	22-28	EPH receptor A3	Homo sapiens	153-156
33032910-6	2020	The overexpression of miR-24 decreased the expression of OATP2B1 mRNA and protein in HEK/OATP2B1 and Caco-2 cells and uptake of [3H]estrone-3-sulfate by HEK/OATP2B1 cells.	mir-24	22-28	solute carrier organic anion transporter family member 2B1	Homo sapiens	89-96
33032910-8	2020	In conclusion, it was found that miR-24 negatively regulates OATP2B1 expression, resulting in suppression of OATP2B1 activity, while its contribution to regulation of apparent expression of OATP2B1 is considered to depend on tissues and cell types.	mir-24	33-39	solute carrier organic anion transporter family member 2B1	Homo sapiens	61-68
33032910-8	2020	In conclusion, it was found that miR-24 negatively regulates OATP2B1 expression, resulting in suppression of OATP2B1 activity, while its contribution to regulation of apparent expression of OATP2B1 is considered to depend on tissues and cell types.	mir-24	33-39	solute carrier organic anion transporter family member 2B1	Homo sapiens	109-116
33032910-8	2020	In conclusion, it was found that miR-24 negatively regulates OATP2B1 expression, resulting in suppression of OATP2B1 activity, while its contribution to regulation of apparent expression of OATP2B1 is considered to depend on tissues and cell types.	mir-24	33-39	solute carrier organic anion transporter family member 2B1	Homo sapiens	109-116
34639051-3	2021	We aimed to target miR-24, a suppressor upstream of junctophilin-2 (JP-2), which is required to affix the sarcoplasmic reticulum to T-tubules, and hence the release of Ca2+ in excitation-contraction coupling using pachymic acid (PA) and/or losartan (LN).	mir-24	19-25	junctophilin 2	Rattus norvegicus	52-66
34639051-3	2021	We aimed to target miR-24, a suppressor upstream of junctophilin-2 (JP-2), which is required to affix the sarcoplasmic reticulum to T-tubules, and hence the release of Ca2+ in excitation-contraction coupling using pachymic acid (PA) and/or losartan (LN).	mir-24	19-25	junctophilin 2	Rattus norvegicus	68-72
34639051-10	2021	In conclusion, HF progression following DOX administration can be prevented or even delayed by targeting miR-24 and its downstream JP-2.	mir-24	105-111	junctophilin 2	Rattus norvegicus	131-135
34591940-4	2021	Expression of the gene encoding miR-24 is induced by the transcription factor AP1 and activated by MAP kinases in HSV1-infected cells.	mir-24	32-38	JunB proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	78-81
34298972-0	2021	Role of miR-24 in Multiple Endocrine Neoplasia Type 1: A Potential Target for Molecular Therapy.	mir-24	8-14	menin 1	Homo sapiens	18-53
34298972-6	2021	Here, we review the current knowledge on the post-transcriptional regulation of MEN1 and menin expression by miR-24, and its possible direct role in MEN1 syndrome, describing the possibility and the potential approaches to target and silence this miRNA, to permit the correct expression of the wild type menin, and thereby prevent the development of cancers in the target tissues.	mir-24	109-115	menin 1	Homo sapiens	80-84
34298972-6	2021	Here, we review the current knowledge on the post-transcriptional regulation of MEN1 and menin expression by miR-24, and its possible direct role in MEN1 syndrome, describing the possibility and the potential approaches to target and silence this miRNA, to permit the correct expression of the wild type menin, and thereby prevent the development of cancers in the target tissues.	mir-24	109-115	menin 1	Homo sapiens	89-94
34298972-6	2021	Here, we review the current knowledge on the post-transcriptional regulation of MEN1 and menin expression by miR-24, and its possible direct role in MEN1 syndrome, describing the possibility and the potential approaches to target and silence this miRNA, to permit the correct expression of the wild type menin, and thereby prevent the development of cancers in the target tissues.	mir-24	109-115	menin 1	Homo sapiens	149-153
34298972-6	2021	Here, we review the current knowledge on the post-transcriptional regulation of MEN1 and menin expression by miR-24, and its possible direct role in MEN1 syndrome, describing the possibility and the potential approaches to target and silence this miRNA, to permit the correct expression of the wild type menin, and thereby prevent the development of cancers in the target tissues.	mir-24	109-115	menin 1	Homo sapiens	304-309
35462096-6	2022	Also, miRNA pull-down and mass spectrophotometry analysis of PMA-treated U937 cells revealed that miR-24 was specifically associated with alpha-tubulin and hnRNP-E1 proteins.	mir-24	98-104	tubulin alpha 1b	Homo sapiens	138-151
34107258-0	2021	miR-24 controls the regenerative competence of hair follicle progenitors by targeting Plk3.	mir-24	0-6	polo like kinase 3	Homo sapiens	86-90
34107258-5	2021	Mechanistically, we find that miR-24 limits the intrinsic growth competence of HF progenitor by directly targeting Plk3, whose downregulation leads to reduced expression of CCNE1, a key cyclin for cell-cycle entry.	mir-24	30-36	polo like kinase 3	Homo sapiens	115-119
34107258-5	2021	Mechanistically, we find that miR-24 limits the intrinsic growth competence of HF progenitor by directly targeting Plk3, whose downregulation leads to reduced expression of CCNE1, a key cyclin for cell-cycle entry.	mir-24	30-36	cyclin E1	Homo sapiens	173-178
35462096-6	2022	Also, miRNA pull-down and mass spectrophotometry analysis of PMA-treated U937 cells revealed that miR-24 was specifically associated with alpha-tubulin and hnRNP-E1 proteins.	mir-24	98-104	poly(rC) binding protein 1	Homo sapiens	156-164
35462096-8	2022	We conclude that miR-24 is transported into exosomes from activated macrophages with the support of alpha-tubulin and hnRNP-E1.	mir-24	17-23	tubulin alpha 1b	Homo sapiens	100-113
35462096-8	2022	We conclude that miR-24 is transported into exosomes from activated macrophages with the support of alpha-tubulin and hnRNP-E1.	mir-24	17-23	poly(rC) binding protein 1	Homo sapiens	118-126
35506414-12	2022	HSD11B2 reversed miR-24-3p"s repression on HTR/SVneo cell advancement.	mir-24	17-23	hydroxysteroid 11-beta dehydrogenase 2	Homo sapiens	0-7
35385441-0	2022	miR-24 Alleviates MI/RI by Blocking the S100A8/TLR4/MyD88/NF-kB Pathway: Erratum.	mir-24	0-6	S100 calcium binding protein A8	Homo sapiens	40-46
35385441-0	2022	miR-24 Alleviates MI/RI by Blocking the S100A8/TLR4/MyD88/NF-kB Pathway: Erratum.	mir-24	0-6	toll like receptor 4	Homo sapiens	47-51
35385441-0	2022	miR-24 Alleviates MI/RI by Blocking the S100A8/TLR4/MyD88/NF-kB Pathway: Erratum.	mir-24	0-6	MYD88 innate immune signal transduction adaptor	Homo sapiens	52-57
35154347-4	2022	In addition, the study explored the possible mechanism through which SYD could reduce myocardial ischemia apoptosis and regulate the expression of the miR-24/Bim pathway.	mir-24	151-157	Bcl2-like 11	Rattus norvegicus	158-161
35154347-13	2022	Dual-luciferase reporter assay and western blot analysis confirmed that Bim was a direct target of miR-24.	mir-24	99-105	Bcl2-like 11	Rattus norvegicus	72-75
33907980-9	2021	The bioinformatics analysis predicted PVT1 negatively regulates miR-24 and KLF6 is a direct target of miR-24.	mir-24	64-70	Pvt1 oncogene	Homo sapiens	38-42
35173828-6	2022	MiR-24 targets and regulates numerous genes in various cancer types and enhances the expression of several oncogenes (e.g., cMyc, BCL2 and HIF1), which are challenging in terms of druggability.	mir-24	0-6	MYC proto-oncogene, bHLH transcription factor	Homo sapiens	124-128
35173828-6	2022	MiR-24 targets and regulates numerous genes in various cancer types and enhances the expression of several oncogenes (e.g., cMyc, BCL2 and HIF1), which are challenging in terms of druggability.	mir-24	0-6	BCL2 apoptosis regulator	Homo sapiens	130-134
35173828-6	2022	MiR-24 targets and regulates numerous genes in various cancer types and enhances the expression of several oncogenes (e.g., cMyc, BCL2 and HIF1), which are challenging in terms of druggability.	mir-24	0-6	hypoxia inducible factor 1 subunit alpha	Homo sapiens	139-143
35173828-7	2022	In contrast, several tumor suppressor proteins (p21 and p53) have been reported to be downregulated by miR-24.	mir-24	103-109	H3 histone pseudogene 16	Homo sapiens	48-51
35173828-7	2022	In contrast, several tumor suppressor proteins (p21 and p53) have been reported to be downregulated by miR-24.	mir-24	103-109	tumor protein p53	Homo sapiens	56-59
33502561-7	2021	Overexpression of APEX1 and miR-24 potentiates ESC proliferation and inhibits apoptosis, while those effects could be reversed by APEX1 and miR-24 silencing.	mir-24	140-146	apurinic/apyrimidinic endodeoxyribonuclease 1	Homo sapiens	18-23
33907980-11	2021	PVT1 knockdown could alleviate LPS-induced biological behavior of cardiac fibroblasts through sponging miR-24 in vitro.	mir-24	103-109	Pvt1 oncogene	Homo sapiens	0-4
33540664-0	2021	miR-24 Targets the Transmembrane Glycoprotein Neuropilin-1 in Human Brain Microvascular Endothelial Cells.	mir-24	0-6	neuropilin 1	Homo sapiens	46-58
33841640-0	2021	miR-24 targets HMOX1 to regulate inflammation and neurofunction in rats with cerebral vasospasm after subarachnoid hemorrhage.	mir-24	0-6	heme oxygenase 1	Rattus norvegicus	15-20
33841640-5	2021	Dual-luciferase reporter assay was conducted to verify the relationship between miR-24 and HMOX1.	mir-24	80-86	heme oxygenase 1	Rattus norvegicus	91-96
33841640-7	2021	RESULTS: miR-24 could negatively regulate HMOX1 expression.	mir-24	9-15	heme oxygenase 1	Rattus norvegicus	42-47
33841640-9	2021	Inhibiting miR-24 expression or enhancing the expression of its down streaming target, HMOX1, could partly reverse the increased oxidation and inflammation as well as functional deficits in the rats.	mir-24	11-17	heme oxygenase 1	Rattus norvegicus	87-92
33841640-10	2021	Moreover, the effects of miR-24 inhibitor could be reversed by inhibiting HMOX1 expression.	mir-24	25-31	heme oxygenase 1	Rattus norvegicus	74-79
33841640-11	2021	CONCLUSION: miR-24 downregulation can promote HMOX1 expression, thereby decreasing the inflammatory response and improving the neurological function of rats with CVS after SAH.	mir-24	12-18	heme oxygenase 1	Rattus norvegicus	46-51
33564755-0	2021	miR-24 targets SARS-CoV-2 co-factor Neuropilin-1 in human brain microvascular endothelial cells: Insights for COVID-19 neurological manifestations.	mir-24	0-6	neuropilin 1	Homo sapiens	36-48
33564755-4	2021	By combining bioinformatic and functional approaches, we identified miR-24 as a regulator of Neuropilin-1 transcription.	mir-24	68-74	neuropilin 1	Homo sapiens	93-105
33564755-5	2021	Since Neuropilin-1 has been shown to play a key role in the endothelium-mediated regulation of the blood-brain barrier, we validated miR-24 as a functional modulator of Neuropilin-1 in human brain microvascular endothelial cells (hBMECs), which are the most suitable cell line for an in vitro blooda$"brain barrier model.	mir-24	133-139	neuropilin 1	Homo sapiens	6-18
33564755-5	2021	Since Neuropilin-1 has been shown to play a key role in the endothelium-mediated regulation of the blood-brain barrier, we validated miR-24 as a functional modulator of Neuropilin-1 in human brain microvascular endothelial cells (hBMECs), which are the most suitable cell line for an in vitro blooda$"brain barrier model.	mir-24	133-139	neuropilin 1	Homo sapiens	169-181
33907980-0	2021	PVT1 knockdown inhibited the biological behavior of LPS-induced cardiac fibroblasts by regulating miR-24.	mir-24	98-104	Pvt1 oncogene	Homo sapiens	0-4
33258087-9	2021	HMC-EVs-carried miR-24 could target AQP4 to activate the P38 MAPK/ERK1/2/P13K/AKT pathway, and thus promoted the proliferation and migration of SH-SY5Y cells after H/R injury, which were reversed by LY294002 and PD98095.	mir-24	16-22	aquaporin 4	Homo sapiens	36-40
33258087-9	2021	HMC-EVs-carried miR-24 could target AQP4 to activate the P38 MAPK/ERK1/2/P13K/AKT pathway, and thus promoted the proliferation and migration of SH-SY5Y cells after H/R injury, which were reversed by LY294002 and PD98095.	mir-24	16-22	mitogen-activated protein kinase 3	Homo sapiens	66-72
33258087-9	2021	HMC-EVs-carried miR-24 could target AQP4 to activate the P38 MAPK/ERK1/2/P13K/AKT pathway, and thus promoted the proliferation and migration of SH-SY5Y cells after H/R injury, which were reversed by LY294002 and PD98095.	mir-24	16-22	AKT serine/threonine kinase 1	Homo sapiens	78-81
33258087-10	2021	Taken together, HMC-EVs-carried miR-24 played protective roles in I/R injury, possibly by targeting AQP4 and activating the P38 MAPK/ERK1/2/P13K/AKT pathway.	mir-24	32-38	aquaporin 4	Homo sapiens	100-104
33258087-10	2021	Taken together, HMC-EVs-carried miR-24 played protective roles in I/R injury, possibly by targeting AQP4 and activating the P38 MAPK/ERK1/2/P13K/AKT pathway.	mir-24	32-38	mitogen-activated protein kinase 3	Homo sapiens	133-139
33258087-10	2021	Taken together, HMC-EVs-carried miR-24 played protective roles in I/R injury, possibly by targeting AQP4 and activating the P38 MAPK/ERK1/2/P13K/AKT pathway.	mir-24	32-38	AKT serine/threonine kinase 1	Homo sapiens	145-148
33540664-4	2021	By combining bioinformatic and functional approaches, we identified miR-24 as a regulator of Neuropilin-1 transcription.	mir-24	68-74	neuropilin 1	Homo sapiens	93-105
33540664-5	2021	Since Neuropilin-1 has been shown to play a key role in the endothelium-mediated regulation of the blood-brain barrier, we validated miR-24 as a functional modulator of Neuropilin-1 in human brain microvascular endothelial cells (hBMECs), which are the most suitable cell line for an in vitro blood-brain barrier model.	mir-24	133-139	neuropilin 1	Homo sapiens	6-18
33540664-5	2021	Since Neuropilin-1 has been shown to play a key role in the endothelium-mediated regulation of the blood-brain barrier, we validated miR-24 as a functional modulator of Neuropilin-1 in human brain microvascular endothelial cells (hBMECs), which are the most suitable cell line for an in vitro blood-brain barrier model.	mir-24	133-139	neuropilin 1	Homo sapiens	169-181
32898528-0	2020	G6PD, bond by miR-24, regulates mitochondrial dysfunction and oxidative stress in phenylephrine-induced hypertrophic cardiomyocytes.	mir-24	14-20	glucose-6-phosphate dehydrogenase	Homo sapiens	0-4
33631962-11	2021	Moreover, MIR22HG knockdown inhibited the NF-kappaB pathway by upregulating miR-24 in AC16 cardiomyocytes.	mir-24	76-82	MIR22 host gene	Homo sapiens	10-17
33631962-11	2021	Moreover, MIR22HG knockdown inhibited the NF-kappaB pathway by upregulating miR-24 in AC16 cardiomyocytes.	mir-24	76-82	nuclear factor kappa B subunit 1	Homo sapiens	42-51
33631962-12	2021	Inhibition of miR-24 resisted the effects of MIR22HG silencing on hypoxia-induced injury in AC16 cardiomyocytes.	mir-24	14-20	MIR22 host gene	Homo sapiens	45-52
33238281-0	2021	LncRNA PMS2L2 downregulates miR-24 through methylation to suppress cell apoptosis in ulcerative colitis.	mir-24	28-34	PMS1 homolog 2, mismatch repair system component pseudogene 2	Homo sapiens	7-13
33238281-14	2021	PMS2L2 overexpression rescued the detrimental effect of miR-24 on cell apoptosis.	mir-24	56-62	PMS1 homolog 2, mismatch repair system component pseudogene 2	Homo sapiens	0-6
33238281-15	2021	CONCLUSION: PMS2L2 may downregulate miR-24 via methylation to suppress cell apoptosis in UC.	mir-24	36-42	PMS1 homolog 2, mismatch repair system component pseudogene 2	Homo sapiens	12-18
32898528-13	2020	MiR-24 was found to directly bind to G6PD at the motif of CUGAGCC and regulated its expression, furtherly, influenced the G6PD-mediated mitochondrial dysfunction and oxidative stress of CH cells.	mir-24	0-6	glucose-6-phosphate dehydrogenase	Homo sapiens	37-41
32898528-13	2020	MiR-24 was found to directly bind to G6PD at the motif of CUGAGCC and regulated its expression, furtherly, influenced the G6PD-mediated mitochondrial dysfunction and oxidative stress of CH cells.	mir-24	0-6	glucose-6-phosphate dehydrogenase	Homo sapiens	122-126
32898528-14	2020	SIGNIFICANCE: Generally, our study demonstrated that miR-24/G6PD regulates mitochondrial dysfunction and oxidative stress in CH cells, representing a new sight for CH therapy.	mir-24	53-59	glucose-6-phosphate dehydrogenase	Homo sapiens	60-64
32929392-8	2020	RESULTS: Here, we demonstrated that activated WNT signaling and downregulated TGF-beta pathways under the control of decreased mir-24 which are involved in myogenic differentiation are differentially expressed in WJ-MSC.	mir-24	127-133	transforming growth factor alpha	Homo sapiens	78-86
32413244-0	2020	The opposite functions of miR-24 in the osteogenesis and adipogenesis of adipose-derived mesenchymal stem cells are mediated by the HOXB7/beta-catenin complex.	mir-24	26-32	catenin beta 1	Rattus norvegicus	138-150
32725119-0	2020	Expression of Concern: von Willebrand factor rescued by miR-24 inhibition facilitates the proliferation and migration of osteosarcoma cells in vitro.	mir-24	56-62	von Willebrand factor	Homo sapiens	23-44
32602343-12	2020	In HepaRG cells, miR-24 decreased the OATP2B1 and HNF4alpha expression levels.	mir-24	17-23	solute carrier organic anion transporter family member 2B1	Homo sapiens	38-45
32602343-12	2020	In HepaRG cells, miR-24 decreased the OATP2B1 and HNF4alpha expression levels.	mir-24	17-23	hepatocyte nuclear factor 4 alpha	Homo sapiens	50-59
32602343-13	2020	These results suggest that miR-24 represses not only the translation of OATP2B1 but also the transcription of OATP2B1 by HNF4alpha mRNA degradation.	mir-24	27-33	solute carrier organic anion transporter family member 2B1	Homo sapiens	72-79
32602343-13	2020	These results suggest that miR-24 represses not only the translation of OATP2B1 but also the transcription of OATP2B1 by HNF4alpha mRNA degradation.	mir-24	27-33	solute carrier organic anion transporter family member 2B1	Homo sapiens	110-117
32602343-13	2020	These results suggest that miR-24 represses not only the translation of OATP2B1 but also the transcription of OATP2B1 by HNF4alpha mRNA degradation.	mir-24	27-33	hepatocyte nuclear factor 4 alpha	Homo sapiens	121-130
32413244-0	2020	The opposite functions of miR-24 in the osteogenesis and adipogenesis of adipose-derived mesenchymal stem cells are mediated by the HOXB7/beta-catenin complex.	mir-24	26-32	homeo box B7	Rattus norvegicus	132-137
32413244-5	2020	Overexpression of HOXB7 could partly halt the inhibitory effect of miR-24 on osteogenesis, and downregulation of HOXB7 could also partly suppress the positive effect of miR-24 on adipogenesis.	mir-24	67-73	homeo box B7	Rattus norvegicus	18-23
32413244-5	2020	Overexpression of HOXB7 could partly halt the inhibitory effect of miR-24 on osteogenesis, and downregulation of HOXB7 could also partly suppress the positive effect of miR-24 on adipogenesis.	mir-24	169-175	homeo box B7	Rattus norvegicus	113-118
32413855-16	2020	miR-24 inhibitor transfection increased PHD1 expression (p < 0.05).	mir-24	0-6	egl-9 family hypoxia-inducible factor 2	Rattus norvegicus	40-44
32413855-21	2020	CONCLUSIONS: The present study suggests that thrombin reduces HIF-1alpha degradation and initiates angiogenesis by increasing miR-24, which targets PHD1 after ICH.	mir-24	126-132	coagulation factor II	Rattus norvegicus	45-53
32413855-21	2020	CONCLUSIONS: The present study suggests that thrombin reduces HIF-1alpha degradation and initiates angiogenesis by increasing miR-24, which targets PHD1 after ICH.	mir-24	126-132	egl-9 family hypoxia-inducible factor 2	Rattus norvegicus	148-152
31533001-8	2020	miR-24 enhanced the viability of rat hippocampal neurons by targeting p27kip1.	mir-24	0-6	cyclin-dependent kinase inhibitor 1B	Rattus norvegicus	70-77
32323758-3	2020	The aim of the present study was to investigate whether adenovirus-mediated miR-24 overexpression can suppress the NOD-like receptor family pyrin domain-containing 3 (NLRP3)-related inflammatory signaling pathway and attenuate diabetic vascular remodeling.	mir-24	76-82	NLR family, pyrin domain containing 3	Rattus norvegicus	167-172
32323758-7	2020	The results of the present study demonstrated that miR-24 upregulation suppressed neointimal hyperplasia and accelerated reendothelialization in the injured arteries, lowered the expression of NLRP3, apoptosis-associated speck-like protein, caspase-1, proliferating cell nuclear antigen, CD45, interleukin (IL)-1beta, IL-18 and tumor necrosis factor-alpha, and increased the expression of CD31, smooth muscle (SM) alpha-actin and SM-myosin heavy chain.	mir-24	51-57	NLR family, pyrin domain containing 3	Rattus norvegicus	193-198
32323758-7	2020	The results of the present study demonstrated that miR-24 upregulation suppressed neointimal hyperplasia and accelerated reendothelialization in the injured arteries, lowered the expression of NLRP3, apoptosis-associated speck-like protein, caspase-1, proliferating cell nuclear antigen, CD45, interleukin (IL)-1beta, IL-18 and tumor necrosis factor-alpha, and increased the expression of CD31, smooth muscle (SM) alpha-actin and SM-myosin heavy chain.	mir-24	51-57	caspase 1	Rattus norvegicus	241-250
32323758-7	2020	The results of the present study demonstrated that miR-24 upregulation suppressed neointimal hyperplasia and accelerated reendothelialization in the injured arteries, lowered the expression of NLRP3, apoptosis-associated speck-like protein, caspase-1, proliferating cell nuclear antigen, CD45, interleukin (IL)-1beta, IL-18 and tumor necrosis factor-alpha, and increased the expression of CD31, smooth muscle (SM) alpha-actin and SM-myosin heavy chain.	mir-24	51-57	proliferating cell nuclear antigen	Rattus norvegicus	252-286
32323758-7	2020	The results of the present study demonstrated that miR-24 upregulation suppressed neointimal hyperplasia and accelerated reendothelialization in the injured arteries, lowered the expression of NLRP3, apoptosis-associated speck-like protein, caspase-1, proliferating cell nuclear antigen, CD45, interleukin (IL)-1beta, IL-18 and tumor necrosis factor-alpha, and increased the expression of CD31, smooth muscle (SM) alpha-actin and SM-myosin heavy chain.	mir-24	51-57	protein tyrosine phosphatase, receptor type, C	Rattus norvegicus	288-292
32323758-7	2020	The results of the present study demonstrated that miR-24 upregulation suppressed neointimal hyperplasia and accelerated reendothelialization in the injured arteries, lowered the expression of NLRP3, apoptosis-associated speck-like protein, caspase-1, proliferating cell nuclear antigen, CD45, interleukin (IL)-1beta, IL-18 and tumor necrosis factor-alpha, and increased the expression of CD31, smooth muscle (SM) alpha-actin and SM-myosin heavy chain.	mir-24	51-57	interleukin 1 alpha	Rattus norvegicus	294-316
32323758-7	2020	The results of the present study demonstrated that miR-24 upregulation suppressed neointimal hyperplasia and accelerated reendothelialization in the injured arteries, lowered the expression of NLRP3, apoptosis-associated speck-like protein, caspase-1, proliferating cell nuclear antigen, CD45, interleukin (IL)-1beta, IL-18 and tumor necrosis factor-alpha, and increased the expression of CD31, smooth muscle (SM) alpha-actin and SM-myosin heavy chain.	mir-24	51-57	interleukin 18	Rattus norvegicus	318-323
32323758-7	2020	The results of the present study demonstrated that miR-24 upregulation suppressed neointimal hyperplasia and accelerated reendothelialization in the injured arteries, lowered the expression of NLRP3, apoptosis-associated speck-like protein, caspase-1, proliferating cell nuclear antigen, CD45, interleukin (IL)-1beta, IL-18 and tumor necrosis factor-alpha, and increased the expression of CD31, smooth muscle (SM) alpha-actin and SM-myosin heavy chain.	mir-24	51-57	tumor necrosis factor	Rattus norvegicus	328-355
32323758-7	2020	The results of the present study demonstrated that miR-24 upregulation suppressed neointimal hyperplasia and accelerated reendothelialization in the injured arteries, lowered the expression of NLRP3, apoptosis-associated speck-like protein, caspase-1, proliferating cell nuclear antigen, CD45, interleukin (IL)-1beta, IL-18 and tumor necrosis factor-alpha, and increased the expression of CD31, smooth muscle (SM) alpha-actin and SM-myosin heavy chain.	mir-24	51-57	platelet and endothelial cell adhesion molecule 1	Rattus norvegicus	389-393
32323758-8	2020	These data indicated that miR-24 overexpression can attenuate vascular remodeling in a diabetic rat model through suppressing the NLRP3/caspase-1/IL-1beta signaling pathway.	mir-24	26-32	NLR family, pyrin domain containing 3	Rattus norvegicus	130-135
32323758-8	2020	These data indicated that miR-24 overexpression can attenuate vascular remodeling in a diabetic rat model through suppressing the NLRP3/caspase-1/IL-1beta signaling pathway.	mir-24	26-32	caspase 1	Rattus norvegicus	136-145
32323758-8	2020	These data indicated that miR-24 overexpression can attenuate vascular remodeling in a diabetic rat model through suppressing the NLRP3/caspase-1/IL-1beta signaling pathway.	mir-24	26-32	interleukin 1 alpha	Rattus norvegicus	146-154
31533001-9	2020	To conclude, this study demonstrated that miR-24 could attenuate isoflurane-induced neurotoxicity in rat hippocampus via anti-oxidative stress function and inhibiting p27kip1 expression.	mir-24	42-48	cyclin-dependent kinase inhibitor 1B	Rattus norvegicus	167-174
32521898-11	2020	In silico analysis together with the dual luciferase assay revealed RNA binding protein DND1 to be the target of miR-24.	mir-24	113-119	DND microRNA-mediated repression inhibitor 1	Homo sapiens	88-92
32271404-10	2020	Upregulation of miR-24 promoted the expression of PDGFRa and NG2, decreased MBP and ADM level, and increased IL-6 and TNF-alpha secretion.	mir-24	16-22	platelet derived growth factor receptor alpha	Rattus norvegicus	50-56
32271404-10	2020	Upregulation of miR-24 promoted the expression of PDGFRa and NG2, decreased MBP and ADM level, and increased IL-6 and TNF-alpha secretion.	mir-24	16-22	chondroitin sulfate proteoglycan 4	Rattus norvegicus	61-64
32271404-10	2020	Upregulation of miR-24 promoted the expression of PDGFRa and NG2, decreased MBP and ADM level, and increased IL-6 and TNF-alpha secretion.	mir-24	16-22	myelin basic protein	Rattus norvegicus	76-79
32271404-10	2020	Upregulation of miR-24 promoted the expression of PDGFRa and NG2, decreased MBP and ADM level, and increased IL-6 and TNF-alpha secretion.	mir-24	16-22	interleukin 6	Rattus norvegicus	109-113
32271404-10	2020	Upregulation of miR-24 promoted the expression of PDGFRa and NG2, decreased MBP and ADM level, and increased IL-6 and TNF-alpha secretion.	mir-24	16-22	tumor necrosis factor	Rattus norvegicus	118-127
32226248-8	2020	MiR-24 showed significant inverse correlation with BMI, glucose, insulin, FIRI, HOMA, LH, testosterone, TG, and LH:FSH ratio whereas HDL levels showed significant positive association with miR-24 and miR-29a.	mir-24	0-6	insulin	Homo sapiens	65-72
31312880-8	2020	Mechanistically, microRNA sequencing identifies miR-24 as a major component of the exosomes from B2M-UMSCs.	mir-24	48-54	beta-2-microglobulin	Homo sapiens	97-100
32062612-10	2020	CONCLUSIONS: miR-24-3p/PIM-2/XIAP signaling contributes to BBR-mediated leukemia mitigation in p53-defect ALL, which should be further developed as a treatment strategy in ALL patients with p53 deficiency.	mir-24	13-19	Pim-2 proto-oncogene, serine/threonine kinase	Homo sapiens	23-28
32062612-10	2020	CONCLUSIONS: miR-24-3p/PIM-2/XIAP signaling contributes to BBR-mediated leukemia mitigation in p53-defect ALL, which should be further developed as a treatment strategy in ALL patients with p53 deficiency.	mir-24	13-19	X-linked inhibitor of apoptosis	Homo sapiens	29-33
32062612-10	2020	CONCLUSIONS: miR-24-3p/PIM-2/XIAP signaling contributes to BBR-mediated leukemia mitigation in p53-defect ALL, which should be further developed as a treatment strategy in ALL patients with p53 deficiency.	mir-24	13-19	tumor protein p53	Homo sapiens	95-98
32062612-10	2020	CONCLUSIONS: miR-24-3p/PIM-2/XIAP signaling contributes to BBR-mediated leukemia mitigation in p53-defect ALL, which should be further developed as a treatment strategy in ALL patients with p53 deficiency.	mir-24	13-19	tumor protein p53	Homo sapiens	190-193
32728001-0	2020	MiR-24 Protects Cardiomyocytes Against Hypoxia/Reoxygenation-Induced Injury Through Regulating Mitogen-Activated Protein Kinase 14.	mir-24	0-6	mitogen activated protein kinase 14	Rattus norvegicus	95-130
32728001-6	2020	A dual-luciferase reporter gene assay was conducted to verify whether Mapk14 is indeed a target of miR-24.	mir-24	99-105	mitogen activated protein kinase 14	Rattus norvegicus	70-76
32728001-13	2020	The mRNA level of Mapk14 and its protein (p38 MAPK) level were negatively affected by miR-24.	mir-24	86-92	mitogen activated protein kinase 14	Rattus norvegicus	18-24
32728001-14	2020	Furthermore, we discovered that depletion of Mapk14 reduced the promoting effect of the miR-24 inhibitor on cell apoptosis.Overall, our results illustrated that miR-24 could attenuate H/R-induced injury partly by regulating Mapk14.	mir-24	88-94	mitogen activated protein kinase 14	Rattus norvegicus	45-51
32728001-14	2020	Furthermore, we discovered that depletion of Mapk14 reduced the promoting effect of the miR-24 inhibitor on cell apoptosis.Overall, our results illustrated that miR-24 could attenuate H/R-induced injury partly by regulating Mapk14.	mir-24	88-94	mitogen activated protein kinase 14	Rattus norvegicus	224-230
32728001-14	2020	Furthermore, we discovered that depletion of Mapk14 reduced the promoting effect of the miR-24 inhibitor on cell apoptosis.Overall, our results illustrated that miR-24 could attenuate H/R-induced injury partly by regulating Mapk14.	mir-24	161-167	mitogen activated protein kinase 14	Rattus norvegicus	45-51
32728001-14	2020	Furthermore, we discovered that depletion of Mapk14 reduced the promoting effect of the miR-24 inhibitor on cell apoptosis.Overall, our results illustrated that miR-24 could attenuate H/R-induced injury partly by regulating Mapk14.	mir-24	161-167	mitogen activated protein kinase 14	Rattus norvegicus	224-230
31562981-8	2019	MiR-24 could negatively regulate the expression of LINC00472 and JP2 by directly binding to them.	mir-24	0-6	long intergenic non-protein coding RNA 472	Homo sapiens	51-60
31562981-8	2019	MiR-24 could negatively regulate the expression of LINC00472 and JP2 by directly binding to them.	mir-24	0-6	T cell receptor gamma joining P2	Homo sapiens	65-68
31539718-6	2019	RESULTS: The expression of miR-24 was decreased in HG-stimulated VSMCs and balloon-injured carotid arteries of diabetic rats, which was accompanied by increased expression of Ogt and Keap1 and decreased expression of Nrf2 and Ho-1.	mir-24	27-33	O-linked N-acetylglucosamine (GlcNAc) transferase	Rattus norvegicus	175-178
31588805-8	2019	Furthermore, miR-24 inhibitor declined LBPs-induced change in Wnt/beta-catenin and JAK1/STAT3 pathways in OGD-injuried cells.	mir-24	13-19	catenin beta 1	Rattus norvegicus	66-78
31588805-8	2019	Furthermore, miR-24 inhibitor declined LBPs-induced change in Wnt/beta-catenin and JAK1/STAT3 pathways in OGD-injuried cells.	mir-24	13-19	Janus kinase 1	Rattus norvegicus	83-87
31588805-8	2019	Furthermore, miR-24 inhibitor declined LBPs-induced change in Wnt/beta-catenin and JAK1/STAT3 pathways in OGD-injuried cells.	mir-24	13-19	signal transducer and activator of transcription 3	Rattus norvegicus	88-93
31539718-6	2019	RESULTS: The expression of miR-24 was decreased in HG-stimulated VSMCs and balloon-injured carotid arteries of diabetic rats, which was accompanied by increased expression of Ogt and Keap1 and decreased expression of Nrf2 and Ho-1.	mir-24	27-33	Kelch-like ECH-associated protein 1	Rattus norvegicus	183-188
31539718-6	2019	RESULTS: The expression of miR-24 was decreased in HG-stimulated VSMCs and balloon-injured carotid arteries of diabetic rats, which was accompanied by increased expression of Ogt and Keap1 and decreased expression of Nrf2 and Ho-1.	mir-24	27-33	NFE2 like bZIP transcription factor 2	Rattus norvegicus	217-221
31539718-6	2019	RESULTS: The expression of miR-24 was decreased in HG-stimulated VSMCs and balloon-injured carotid arteries of diabetic rats, which was accompanied by increased expression of Ogt and Keap1 and decreased expression of Nrf2 and Ho-1.	mir-24	27-33	heme oxygenase 1	Rattus norvegicus	226-230
31539718-9	2019	CONCLUSIONS: The up-regulation of miR-24 significantly promoted endothelial repair after balloon injury through inhibition of oxidative stress by activating the Nrf2/Ho-1 signaling pathway.	mir-24	34-40	NFE2 like bZIP transcription factor 2	Rattus norvegicus	161-165
31539718-9	2019	CONCLUSIONS: The up-regulation of miR-24 significantly promoted endothelial repair after balloon injury through inhibition of oxidative stress by activating the Nrf2/Ho-1 signaling pathway.	mir-24	34-40	heme oxygenase 1	Rattus norvegicus	166-170
30307120-0	2018	Inhibition of miR-24 suppresses malignancy of human non-small cell lung cancer cells by targeting WWOX in vitro and in vivo.	mir-24	14-20	WW domain containing oxidoreductase	Homo sapiens	98-102
31491426-2	2019	In this study, we first demonstrated that miR-24 plays an important role in maintaining retinal structure and visual function of rats by targeting chitinase-3-like protein 1 (CHI3L1).	mir-24	42-48	chitinase 3 like 1	Rattus norvegicus	147-173
31491426-2	2019	In this study, we first demonstrated that miR-24 plays an important role in maintaining retinal structure and visual function of rats by targeting chitinase-3-like protein 1 (CHI3L1).	mir-24	42-48	chitinase 3 like 1	Rattus norvegicus	175-181
31316620-0	2019	beta-catenin regulates effects of miR-24 on the viability and autophagy of glioma cells.	mir-24	34-40	catenin beta 1	Homo sapiens	0-12
31316620-4	2019	The current study aimed to investigate the role of beta-catenin in regulating the effects of miR-24 on the cell viability and autophagy of glioma cells.	mir-24	93-99	catenin beta 1	Homo sapiens	51-63
31316620-13	2019	XAV-939 attenuated the miR-24-induced decrease of the protein expression of LC3B and Beclin1, and decreased the stimulatory effects of miR-24 mimics on cell viability.	mir-24	23-29	microtubule associated protein 1 light chain 3 beta	Homo sapiens	76-80
31316620-13	2019	XAV-939 attenuated the miR-24-induced decrease of the protein expression of LC3B and Beclin1, and decreased the stimulatory effects of miR-24 mimics on cell viability.	mir-24	23-29	beclin 1	Homo sapiens	85-92
31316620-15	2019	To the best of our knowledge, the present study is the first to demonstrate whether beta-catenin regulates the intracellular effects of miR-24 on the viability and autophagy of glioma cells.	mir-24	136-142	catenin beta 1	Homo sapiens	84-96
31307174-3	2019	Partial correlation analysis was used to explore the correlation be-tween the expression of miR-24 in serum and hs-CRP and MMP-9.	mir-24	92-98	matrix metallopeptidase 9	Homo sapiens	123-128
31005375-8	2019	The expression of miR-24 was decreased in HG-stimulated VSMCs and balloon-injured carotid arteries of diabetic rats, accompanied by increased mRNA expression of PIK3R1.	mir-24	18-24	phosphoinositide-3-kinase regulatory subunit 1	Rattus norvegicus	161-167
31005375-9	2019	The up-regulation of miR-24 suppressed VSMCs proliferation, migration, collagen deposition not only induced by HG in vitro, but also in balloon-injured diabetic rats, which were related to inactivation of PI3K/Akt signaling pathway.	mir-24	21-27	AKT serine/threonine kinase 1	Rattus norvegicus	210-213
31005375-10	2019	CONCLUSION: The up-regulation of miR-24 significantly attenuated vascular remodeling both in balloon-injured diabetic rats and HG-stimulated VSMCs via suppression of proliferation, migration and collagen deposition by acting on PIK3R1 gene that modulated the PI3K/Akt/mTOR axes.	mir-24	33-39	phosphoinositide-3-kinase regulatory subunit 1	Rattus norvegicus	228-234
31005375-10	2019	CONCLUSION: The up-regulation of miR-24 significantly attenuated vascular remodeling both in balloon-injured diabetic rats and HG-stimulated VSMCs via suppression of proliferation, migration and collagen deposition by acting on PIK3R1 gene that modulated the PI3K/Akt/mTOR axes.	mir-24	33-39	AKT serine/threonine kinase 1	Rattus norvegicus	264-267
31005375-10	2019	CONCLUSION: The up-regulation of miR-24 significantly attenuated vascular remodeling both in balloon-injured diabetic rats and HG-stimulated VSMCs via suppression of proliferation, migration and collagen deposition by acting on PIK3R1 gene that modulated the PI3K/Akt/mTOR axes.	mir-24	33-39	mechanistic target of rapamycin kinase	Rattus norvegicus	268-272
30600184-0	2019	MiR-24 inhibits inflammatory responses in LPS-induced acute lung injury of neonatal rats through targeting NLRP3.	mir-24	0-6	NLR family, pyrin domain containing 3	Rattus norvegicus	107-112
30600184-8	2019	Direct binding of miR-24 and pyrin domain-containing 3(NLRP3) were determined by dual luciferase assay.	mir-24	18-24	NLR family, pyrin domain containing 3	Rattus norvegicus	55-60
30600184-11	2019	Overexpression of miR-24 significantly reduced LPS-induced lung damage and decreased the release of proinflammatory cytokine TNF-alpha, IL-6, IL-1beta and SP-A, SP-D expression induced by LPS.	mir-24	18-24	tumor necrosis factor	Rattus norvegicus	125-134
30600184-11	2019	Overexpression of miR-24 significantly reduced LPS-induced lung damage and decreased the release of proinflammatory cytokine TNF-alpha, IL-6, IL-1beta and SP-A, SP-D expression induced by LPS.	mir-24	18-24	interleukin 6	Rattus norvegicus	136-140
30600184-11	2019	Overexpression of miR-24 significantly reduced LPS-induced lung damage and decreased the release of proinflammatory cytokine TNF-alpha, IL-6, IL-1beta and SP-A, SP-D expression induced by LPS.	mir-24	18-24	interleukin 1 beta	Rattus norvegicus	142-150
30600184-11	2019	Overexpression of miR-24 significantly reduced LPS-induced lung damage and decreased the release of proinflammatory cytokine TNF-alpha, IL-6, IL-1beta and SP-A, SP-D expression induced by LPS.	mir-24	18-24	surfactant protein A1	Rattus norvegicus	155-159
30600184-11	2019	Overexpression of miR-24 significantly reduced LPS-induced lung damage and decreased the release of proinflammatory cytokine TNF-alpha, IL-6, IL-1beta and SP-A, SP-D expression induced by LPS.	mir-24	18-24	surfactant protein D	Rattus norvegicus	161-165
30600184-12	2019	In addition, miR-24 inhibited the expression of NLRP3 by directly targeting to the CDS region of NLRP3 mRNA.	mir-24	13-19	NLR family, pyrin domain containing 3	Rattus norvegicus	48-53
30600184-12	2019	In addition, miR-24 inhibited the expression of NLRP3 by directly targeting to the CDS region of NLRP3 mRNA.	mir-24	13-19	NLR family, pyrin domain containing 3	Rattus norvegicus	97-102
30600184-14	2019	These data show that miR-24 alleviated inflammatory responses in LPS-induced ALI via targeting NLRP3.	mir-24	21-27	NLR family, pyrin domain containing 3	Rattus norvegicus	95-100
30307120-6	2018	RESULTS: According to our experimental data, miR-24 inhibition could induce apoptosis by activating caspase 3 and suppress the viability and proliferation of NSCLC cells in vitro and in vivo.	mir-24	45-51	caspase 3	Homo sapiens	100-109
30307120-7	2018	MiR-24 downregulation could reduce the invasive ability of NSCLC cells by downregulating MMP9.	mir-24	0-6	matrix metallopeptidase 9	Homo sapiens	89-93
30307120-8	2018	WWOX was identified as a functional target of miR-24.	mir-24	46-52	WW domain containing oxidoreductase	Homo sapiens	0-4
30307120-9	2018	WWOX overexpression generated the same effect with antagonizing miR-24, while blocking WWOX counteracted the tumor suppressive effect caused by miR-24 inhibition.	mir-24	64-70	WW domain containing oxidoreductase	Homo sapiens	0-4
30307120-9	2018	WWOX overexpression generated the same effect with antagonizing miR-24, while blocking WWOX counteracted the tumor suppressive effect caused by miR-24 inhibition.	mir-24	144-150	WW domain containing oxidoreductase	Homo sapiens	87-91
30570867-0	2018	MiR-24 promotes migration and invasion of non-small cell lung cancer by targeting ZNF367.	mir-24	0-6	zinc finger protein 367	Homo sapiens	82-88
30279208-4	2018	The interaction between miR-24 and vWF was identified by dual luciferase reporter assay.	mir-24	24-30	von Willebrand factor	Homo sapiens	35-38
30279208-8	2018	vWF was further validated as the target of miR-24 in MG-63 and U2OS cells.	mir-24	43-49	von Willebrand factor	Homo sapiens	0-3
30279208-10	2018	However, the migration-inhibiting activity of miR-24 was predominantly attenuated by vWF overexpression.	mir-24	46-52	von Willebrand factor	Homo sapiens	85-88
30570867-3	2018	The aim of this study was to investigate whether MiR-24 can regulate cell proliferation of NSCLC by targeting ZNF367.	mir-24	49-55	zinc finger protein 367	Homo sapiens	110-116
29494963-0	2018	miR-24 inhibited the killing effect of natural killer cells to colorectal cancer cells by downregulating Paxillin.	mir-24	0-6	paxillin	Homo sapiens	105-113
29787826-3	2018	Here, we have dissected the function of a novel miR-24 on SR-B1 expression, HDL uptake and lipid metabolism.	mir-24	48-54	scavenger receptor class B member 1	Homo sapiens	58-63
29787826-8	2018	Taken together, we demonstrated that obesity induced miR-24 repressed HDL uptake, steroid hormone synthesis and lipid metabolism by targeting SR-B1.	mir-24	53-59	scavenger receptor class B member 1	Homo sapiens	142-147
29845232-4	2018	A luciferase reporter assay performed to investigate the regulatory association between miR-24 and NOS3 revealed that miR-24 bound to the NOS3 3" untranslated region and inhibited NOS3 expression.	mir-24	88-94	nitric oxide synthase 3	Homo sapiens	99-103
29845232-4	2018	A luciferase reporter assay performed to investigate the regulatory association between miR-24 and NOS3 revealed that miR-24 bound to the NOS3 3" untranslated region and inhibited NOS3 expression.	mir-24	88-94	nitric oxide synthase 3	Homo sapiens	138-142
29845232-4	2018	A luciferase reporter assay performed to investigate the regulatory association between miR-24 and NOS3 revealed that miR-24 bound to the NOS3 3" untranslated region and inhibited NOS3 expression.	mir-24	88-94	nitric oxide synthase 3	Homo sapiens	138-142
29845232-4	2018	A luciferase reporter assay performed to investigate the regulatory association between miR-24 and NOS3 revealed that miR-24 bound to the NOS3 3" untranslated region and inhibited NOS3 expression.	mir-24	118-124	nitric oxide synthase 3	Homo sapiens	99-103
29845232-4	2018	A luciferase reporter assay performed to investigate the regulatory association between miR-24 and NOS3 revealed that miR-24 bound to the NOS3 3" untranslated region and inhibited NOS3 expression.	mir-24	118-124	nitric oxide synthase 3	Homo sapiens	138-142
29845232-4	2018	A luciferase reporter assay performed to investigate the regulatory association between miR-24 and NOS3 revealed that miR-24 bound to the NOS3 3" untranslated region and inhibited NOS3 expression.	mir-24	118-124	nitric oxide synthase 3	Homo sapiens	138-142
29845232-7	2018	Vascular smooth muscle cells (VSMCs) transfected with an miR-24 inhibitor exhibited increased expression levels of NOS3, whereas those transfected with an miR-24 mimic or NOS3 small interfering RNA exhibited reduced expression levels of NOS3, compared with the control.	mir-24	57-63	nitric oxide synthase 3	Homo sapiens	115-119
29845232-8	2018	These results indicated a negative regulatory association between miR-24 and NOS3.	mir-24	66-72	nitric oxide synthase 3	Homo sapiens	77-81
29845232-9	2018	Downregulation of NOS3 may induce vasospasm following SAH, which may be due to the upregualtion of miR-24 in VSMCs.	mir-24	99-105	nitric oxide synthase 3	Homo sapiens	18-22
29559348-9	2018	Up-regulation of miR-24 could regulate vascular remodeling effectively, lower the level of inflammatory factors, inhibit the expression of mRNA and protein levels of JNK1/2, ERK1/2, RAS, PDGF-R, AP-1, P27, PCNA.	mir-24	17-23	mitogen activated protein kinase 3	Rattus norvegicus	174-180
29559348-9	2018	Up-regulation of miR-24 could regulate vascular remodeling effectively, lower the level of inflammatory factors, inhibit the expression of mRNA and protein levels of JNK1/2, ERK1/2, RAS, PDGF-R, AP-1, P27, PCNA.	mir-24	17-23	transmembrane p24 trafficking protein 7	Rattus norvegicus	201-204
29559348-9	2018	Up-regulation of miR-24 could regulate vascular remodeling effectively, lower the level of inflammatory factors, inhibit the expression of mRNA and protein levels of JNK1/2, ERK1/2, RAS, PDGF-R, AP-1, P27, PCNA.	mir-24	17-23	proliferating cell nuclear antigen	Rattus norvegicus	206-210
29786048-13	2018	The overexpression of miR-24 in NRCMs led to the decreased expression of p27 (t = 4.400, P &lt; 0.01), and decreased G0/G1 arrest in cell cycle and cardiomyocyte hypertrophy.	mir-24	22-28	cyclin-dependent kinase inhibitor 1B	Rattus norvegicus	73-76
29786048-14	2018	Conclusion: MiR-24 promotes cardiac hypertrophy partly by affecting the cell cycle through down-regulation of p27 expression.	mir-24	12-18	cyclin-dependent kinase inhibitor 1B	Rattus norvegicus	110-113
29494963-5	2018	Luciferase reporter assay was conducted to confirm the regulation of miR-24 on Paxillin.	mir-24	69-75	paxillin	Homo sapiens	79-87
29494963-11	2018	Moreover, overexpression of miR-24 inhibited secretions of IFN-gamma and TNF-alpha, and decreased cytotoxicity by downregulating Paxillin expression.	mir-24	28-34	interferon gamma	Homo sapiens	59-68
29494963-11	2018	Moreover, overexpression of miR-24 inhibited secretions of IFN-gamma and TNF-alpha, and decreased cytotoxicity by downregulating Paxillin expression.	mir-24	28-34	tumor necrosis factor	Homo sapiens	73-82
29494963-11	2018	Moreover, overexpression of miR-24 inhibited secretions of IFN-gamma and TNF-alpha, and decreased cytotoxicity by downregulating Paxillin expression.	mir-24	28-34	paxillin	Homo sapiens	129-137
29494963-13	2018	CONCLUSION: Overexpression of miR-24 supressed the killing effect of NK cells to colorectal cancer cells by downregulating Paxillin expression.	mir-24	30-36	paxillin	Homo sapiens	123-131
28189676-0	2017	miR-24 represses metastasis of human osteosarcoma cells by targeting Ack1 via AKT/MMPs pathway.	mir-24	0-6	tyrosine kinase non receptor 2	Homo sapiens	69-73
28929922-0	2018	Elevated levels of serum MiR-152 and miR-24 in uterine sarcoma: potential for inducing autophagy via SIRT1 and deacetylated LC3.	mir-24	37-43	sirtuin 1	Homo sapiens	101-106
28929922-0	2018	Elevated levels of serum MiR-152 and miR-24 in uterine sarcoma: potential for inducing autophagy via SIRT1 and deacetylated LC3.	mir-24	37-43	microtubule associated protein 1 light chain 3 alpha	Homo sapiens	124-127
28929922-9	2018	The mimics of miR-152 and miR-24 induced autophagy by increasing the level of SIRT1, which deacetylated LC3.	mir-24	26-32	sirtuin 1	Homo sapiens	78-83
28929922-9	2018	The mimics of miR-152 and miR-24 induced autophagy by increasing the level of SIRT1, which deacetylated LC3.	mir-24	26-32	microtubule associated protein 1 light chain 3 alpha	Homo sapiens	104-107
28742197-0	2017	MiR-24 alleviates cardiomyocyte apoptosis after myocardial infarction via targeting BIM.	mir-24	0-6	Bcl2-like 11	Rattus norvegicus	84-87
28742197-5	2017	This study aims to investigate the role of miR-24 in mediating BIM expression and CM apoptosis.	mir-24	43-49	Bcl2-like 11	Rattus norvegicus	63-66
28742197-13	2017	MiR-24 targeted suppressed BIM expression.	mir-24	0-6	Bcl2-like 11	Rattus norvegicus	27-30
28742197-14	2017	RESULTS: MiR-24 mimic and/or si-BIM transfection significantly declined the BIM expression, inhibited caspase-9 and caspase-3 activities, and reduced cell apoptosis in H9C2 cells.	mir-24	9-15	Bcl2-like 11	Rattus norvegicus	76-79
28742197-14	2017	RESULTS: MiR-24 mimic and/or si-BIM transfection significantly declined the BIM expression, inhibited caspase-9 and caspase-3 activities, and reduced cell apoptosis in H9C2 cells.	mir-24	9-15	caspase 9	Rattus norvegicus	102-111
28742197-14	2017	RESULTS: MiR-24 mimic and/or si-BIM transfection significantly declined the BIM expression, inhibited caspase-9 and caspase-3 activities, and reduced cell apoptosis in H9C2 cells.	mir-24	9-15	caspase 3	Rattus norvegicus	116-125
28742197-18	2017	MiR-24 up-regulation plays a critical role in decreasing BIM expression, reducing CM apoptosis, and improving cardiac function after AMI.	mir-24	0-6	Bcl2-like 11	Rattus norvegicus	57-60
29565463-0	2018	miR-24 may be a negative regulator of menin in lung cancer.	mir-24	0-6	menin 1	Homo sapiens	38-43
29565463-6	2018	In the present study, miR-24 was found to be associated with menin, affecting the activity of the SMAD3 pathway in lung cancer by inhibiting menin expression.	mir-24	22-28	menin 1	Homo sapiens	61-66
29565463-6	2018	In the present study, miR-24 was found to be associated with menin, affecting the activity of the SMAD3 pathway in lung cancer by inhibiting menin expression.	mir-24	22-28	SMAD family member 3	Homo sapiens	98-103
29565463-6	2018	In the present study, miR-24 was found to be associated with menin, affecting the activity of the SMAD3 pathway in lung cancer by inhibiting menin expression.	mir-24	22-28	menin 1	Homo sapiens	141-146
29565463-7	2018	miR-24 may promote the growth and metastasis and inhibit the apoptosis of lung cancer cells by targeting menin.	mir-24	0-6	menin 1	Homo sapiens	105-110
29393409-4	2018	Overexpression of miR-24 induced p53 expression and p53 was verified as a direct target of miR-24.	mir-24	18-24	tumor protein p53	Homo sapiens	33-36
29393409-4	2018	Overexpression of miR-24 induced p53 expression and p53 was verified as a direct target of miR-24.	mir-24	91-97	tumor protein p53	Homo sapiens	52-55
29393409-5	2018	Overexpression of miR-24 enhanced LEC death by directly targeting p53.	mir-24	18-24	C-C motif chemokine ligand 16	Homo sapiens	34-37
29393409-5	2018	Overexpression of miR-24 enhanced LEC death by directly targeting p53.	mir-24	18-24	tumor protein p53	Homo sapiens	66-69
29393409-7	2018	These results suggest that the miR-24-p53 signaling pathway is involved in a novel mechanism of age-associated cataractogenesis and miR-24 may be a useful therapeutic target for age-associated cataracts.	mir-24	31-37	tumor protein p53	Homo sapiens	38-41
29393409-7	2018	These results suggest that the miR-24-p53 signaling pathway is involved in a novel mechanism of age-associated cataractogenesis and miR-24 may be a useful therapeutic target for age-associated cataracts.	mir-24	132-138	tumor protein p53	Homo sapiens	38-41
29457789-6	2018	Here, we show that miR-24 potently suppresses SCN5A expression and that rs1805126 modulates this regulation.	mir-24	19-25	sodium voltage-gated channel alpha subunit 5	Homo sapiens	46-51
29352987-7	2018	Further experiments demonstrated that miR-24 participated in the XBP1s-induced eNOSup-regulation and EC migration.	mir-24	38-44	X-box binding protein 1	Homo sapiens	65-70
28404635-3	2017	In this article, we show that miR-24 drives the production of IFN-gamma and IL-17 in T cells at least in part through targeting TCF1, a transcription factor known for its role in limiting Th1 and Th17 immunity.	mir-24	30-36	interferon gamma	Homo sapiens	62-71
28404635-3	2017	In this article, we show that miR-24 drives the production of IFN-gamma and IL-17 in T cells at least in part through targeting TCF1, a transcription factor known for its role in limiting Th1 and Th17 immunity.	mir-24	30-36	interleukin 17A	Homo sapiens	76-81
28404635-3	2017	In this article, we show that miR-24 drives the production of IFN-gamma and IL-17 in T cells at least in part through targeting TCF1, a transcription factor known for its role in limiting Th1 and Th17 immunity.	mir-24	30-36	transcription factor 7	Homo sapiens	128-132
28404635-3	2017	In this article, we show that miR-24 drives the production of IFN-gamma and IL-17 in T cells at least in part through targeting TCF1, a transcription factor known for its role in limiting Th1 and Th17 immunity.	mir-24	30-36	negative elongation factor complex member C/D	Homo sapiens	188-191
28189676-0	2017	miR-24 represses metastasis of human osteosarcoma cells by targeting Ack1 via AKT/MMPs pathway.	mir-24	0-6	AKT serine/threonine kinase 1	Homo sapiens	78-81
28189676-6	2017	Moreover, Ack1 expression levels were inversely correlated with that of miR-24 in osteosarcoma tissues.	mir-24	72-78	tyrosine kinase non receptor 2	Homo sapiens	10-14
28189676-7	2017	Furthermore, functional assay showed that miR-24 significantly suppressed osteosarcoma progression partially mediated by inhibiting Ack1 expression.	mir-24	42-48	tyrosine kinase non receptor 2	Homo sapiens	132-136
28189676-8	2017	Finally, western bolt assay revealed that miR-24 regulate AKT/MMPs pathway via Ack1 in osteosarcoma cells.	mir-24	42-48	AKT serine/threonine kinase 1	Homo sapiens	58-61
28189676-8	2017	Finally, western bolt assay revealed that miR-24 regulate AKT/MMPs pathway via Ack1 in osteosarcoma cells.	mir-24	42-48	tyrosine kinase non receptor 2	Homo sapiens	79-83
28189676-9	2017	In conclusion, our study demonstrated the suppression of miR-24 on osteosarcoma metastasis by targeting Ack1 via AKT/MMPs pathways, providing a novel strategy for the diagnosis and treatment of osteosarcoma patients.	mir-24	57-63	tyrosine kinase non receptor 2	Homo sapiens	104-108
28189676-9	2017	In conclusion, our study demonstrated the suppression of miR-24 on osteosarcoma metastasis by targeting Ack1 via AKT/MMPs pathways, providing a novel strategy for the diagnosis and treatment of osteosarcoma patients.	mir-24	57-63	AKT serine/threonine kinase 1	Homo sapiens	113-116
27824038-5	2016	IRAK-M deficiency in turn leads to elevated miR-24 levels, sustains disruption of monocyte homeostasis and aggravates atherosclerosis.	mir-24	44-50	interleukin 1 receptor associated kinase 3	Homo sapiens	0-6
28055991-3	2017	A previous study has shown the inhibitory role on TGF-beta1 by microRNA-24 (miR-24) via targeting Furin.	mir-24	76-82	transforming growth factor, beta 1	Rattus norvegicus	50-59
28055991-3	2017	A previous study has shown the inhibitory role on TGF-beta1 by microRNA-24 (miR-24) via targeting Furin.	mir-24	76-82	furin (paired basic amino acid cleaving enzyme)	Rattus norvegicus	98-103
28694557-0	2017	Overexpression of miR-24 Is Involved in the Formation of Hypocoagulation State after Severe Trauma by Inhibiting the Synthesis of Coagulation Factor X.	mir-24	18-24	coagulation factor X	Homo sapiens	130-150
27157611-6	2016	Furthermore, silencing Jab1/CSN5 phenocopied the function of miR-24 in NPC cells after ionizing radiation treatment, resulting in increased apoptosis.	mir-24	61-67	COP9 signalosome subunit 5	Homo sapiens	23-27
27157611-6	2016	Furthermore, silencing Jab1/CSN5 phenocopied the function of miR-24 in NPC cells after ionizing radiation treatment, resulting in increased apoptosis.	mir-24	61-67	COP9 signalosome subunit 5	Homo sapiens	28-32
27157611-9	2016	Together, our findings indicate that miR-24 inhibits NPC tumor growth and increases NPC radiosensitivity by directly regulating Jab1/CSN5 and that both miR-24 and Jab1/CSN5 can serve as prognostic markers for NPC recurrence; this, in turn, may provide a promising therapeutic strategy for reversing NPC radioresistance.	mir-24	37-43	COP9 signalosome subunit 5	Homo sapiens	128-132
27157611-9	2016	Together, our findings indicate that miR-24 inhibits NPC tumor growth and increases NPC radiosensitivity by directly regulating Jab1/CSN5 and that both miR-24 and Jab1/CSN5 can serve as prognostic markers for NPC recurrence; this, in turn, may provide a promising therapeutic strategy for reversing NPC radioresistance.	mir-24	37-43	COP9 signalosome subunit 5	Homo sapiens	133-137
26748253-0	2016	mir-24 activity propagates stress-induced senescence by down regulating DNA topoisomerase 1.	mir-24	0-6	DNA topoisomerase I	Homo sapiens	72-91
27279639-0	2016	Targeted Silencing of S100A8 Gene by miR-24 to Increase Chemotherapy Sensitivity of Endometrial Carcinoma Cells to Paclitaxel.	mir-24	37-43	S100 calcium binding protein A8	Homo sapiens	22-28
27279639-11	2016	The regulation effects of miR-24 enhancement on cell proliferation and chemotherapy sensitivity were largely reversed by S100A8 up-regulation.	mir-24	26-32	S100 calcium binding protein A8	Homo sapiens	121-127
26847530-0	2016	miR-24 regulates CDKN1B/p27 expression in prostate cancer.	mir-24	0-6	cyclin dependent kinase inhibitor 1B	Homo sapiens	17-23
26847530-0	2016	miR-24 regulates CDKN1B/p27 expression in prostate cancer.	mir-24	0-6	transmembrane p24 trafficking protein 7	Homo sapiens	24-27
26967561-3	2016	Here we show that miR-24, which has been demonstrated to target genes involved in the DNA repair process, targets p38, p53, PML and H2AX simultaneously.	mir-24	18-24	mitogen-activated protein kinase 14	Homo sapiens	114-117
26967561-3	2016	Here we show that miR-24, which has been demonstrated to target genes involved in the DNA repair process, targets p38, p53, PML and H2AX simultaneously.	mir-24	18-24	tumor protein p53	Homo sapiens	119-122
26967561-3	2016	Here we show that miR-24, which has been demonstrated to target genes involved in the DNA repair process, targets p38, p53, PML and H2AX simultaneously.	mir-24	18-24	PML nuclear body scaffold	Homo sapiens	124-127
26967561-3	2016	Here we show that miR-24, which has been demonstrated to target genes involved in the DNA repair process, targets p38, p53, PML and H2AX simultaneously.	mir-24	18-24	H2A.X variant histone	Homo sapiens	132-136
27350307-0	2016	miR-24 promotes the proliferation, migration and invasion in human tongue squamous cell carcinoma by targeting FBXW7.	mir-24	0-6	F-box and WD repeat domain containing 7	Homo sapiens	111-116
27350307-6	2016	Furthermore, miR-24 repressed FBXW7 expression by directly binding to the 3-untranslated region of FBXW7.	mir-24	13-19	F-box and WD repeat domain containing 7	Homo sapiens	30-35
27350307-6	2016	Furthermore, miR-24 repressed FBXW7 expression by directly binding to the 3-untranslated region of FBXW7.	mir-24	13-19	F-box and WD repeat domain containing 7	Homo sapiens	99-104
27350307-7	2016	Moreover, the suppression of FBXW7 increased the proliferation, migration and invasion of TSCC cells, and the restoration of FBXW7 substantially attenuated the oncogenic effects of miR-24.	mir-24	181-187	F-box and WD repeat domain containing 7	Homo sapiens	125-130
27103442-6	2016	Overexpression of miR-24 significantly promoted 3T3-L1 adipogenesis, as evidenced by its ability to increase the expression of PPAR-gamma and SREBP1, lipid droplet formation and triglyceride (TG) accumulation.	mir-24	18-24	peroxisome proliferator activated receptor gamma	Homo sapiens	127-137
27103442-6	2016	Overexpression of miR-24 significantly promoted 3T3-L1 adipogenesis, as evidenced by its ability to increase the expression of PPAR-gamma and SREBP1, lipid droplet formation and triglyceride (TG) accumulation.	mir-24	18-24	sterol regulatory element binding transcription factor 1	Homo sapiens	142-148
27103442-8	2016	Finally, we demonstrated that miR-24 plays the modulational role by directly repressing MAPK7, a key number in the MAPK signaling pathway.	mir-24	30-36	mitogen-activated protein kinase 7	Homo sapiens	88-93
27135744-7	2016	However, miR-24 and miR-34a levels of the low (10th) and high (90th) percentiles of those liver samples were inversely correlated with HNF4A and almost all hemostatic factors expression levels.	mir-24	9-15	hepatocyte nuclear factor 4 alpha	Homo sapiens	135-140
26503504-0	2015	MiR-24 functions as a tumor suppressor in nasopharyngeal carcinoma through targeting FSCN1.	mir-24	0-6	fascin actin-bundling protein 1	Homo sapiens	85-90
26365986-7	2015	CONCLUSIONS/SIGNIFICANCE: In conclusion, our results demonstrated that deregulation of miR-24 is a recurrent event in human tongue squamous cell carcinoma and associate with tumor progression and that miR-24 induces cell survival and cisplatin resistance primarily through targeting PTEN/Akt pathway.	mir-24	87-93	phosphatase and tensin homolog	Homo sapiens	283-287
26365986-7	2015	CONCLUSIONS/SIGNIFICANCE: In conclusion, our results demonstrated that deregulation of miR-24 is a recurrent event in human tongue squamous cell carcinoma and associate with tumor progression and that miR-24 induces cell survival and cisplatin resistance primarily through targeting PTEN/Akt pathway.	mir-24	87-93	AKT serine/threonine kinase 1	Homo sapiens	288-291
26657215-9	2015	IFN-lambda1 induced Stat 3 phosphorylation reduces the expression of hepatocyte nuclear factor 4 alpha (HNF4alpha) through miR-24 in R4-GFP cells.	mir-24	123-129	interferon lambda 1	Homo sapiens	0-11
26657215-9	2015	IFN-lambda1 induced Stat 3 phosphorylation reduces the expression of hepatocyte nuclear factor 4 alpha (HNF4alpha) through miR-24 in R4-GFP cells.	mir-24	123-129	signal transducer and activator of transcription 3	Homo sapiens	20-26
26657215-9	2015	IFN-lambda1 induced Stat 3 phosphorylation reduces the expression of hepatocyte nuclear factor 4 alpha (HNF4alpha) through miR-24 in R4-GFP cells.	mir-24	123-129	hepatocyte nuclear factor 4 alpha	Homo sapiens	69-102
26657215-9	2015	IFN-lambda1 induced Stat 3 phosphorylation reduces the expression of hepatocyte nuclear factor 4 alpha (HNF4alpha) through miR-24 in R4-GFP cells.	mir-24	123-129	hepatocyte nuclear factor 4 alpha	Homo sapiens	104-113
26253191-7	2015	Treatment with exogenous TGF-beta suppressed miR-24 expression and induced KLF6.	mir-24	45-51	transforming growth factor beta 1	Homo sapiens	25-33
26252200-6	2015	Moreover, the low level of miR-24 was associated with increased expression of CARMA3 in bladder cancer cells.	mir-24	27-33	caspase recruitment domain family member 10	Homo sapiens	78-84
26252200-10	2015	Further study by luciferase reporter assay demonstrated that miR-24 could directly target CARMA3.	mir-24	61-67	caspase recruitment domain family member 10	Homo sapiens	90-96
26252200-11	2015	Overexpression of CARMA3 in bladder cancer cells transfected with miR-24 mimic partially reversed the inhibitory effect of miR-24.	mir-24	66-72	caspase recruitment domain family member 10	Homo sapiens	18-24
26252200-11	2015	Overexpression of CARMA3 in bladder cancer cells transfected with miR-24 mimic partially reversed the inhibitory effect of miR-24.	mir-24	123-129	caspase recruitment domain family member 10	Homo sapiens	18-24
26252200-12	2015	In conclusion, miR-24 inhibited cell proliferation, invasion and EMT in bladder cancer cells by downregulation of CARMA3, and that downregulation of CARMA3 was essential for the miR-24-inhibited cell proliferation, invasion and EMT in bladder cancer cells.	mir-24	15-21	caspase recruitment domain family member 10	Homo sapiens	114-120
26252200-12	2015	In conclusion, miR-24 inhibited cell proliferation, invasion and EMT in bladder cancer cells by downregulation of CARMA3, and that downregulation of CARMA3 was essential for the miR-24-inhibited cell proliferation, invasion and EMT in bladder cancer cells.	mir-24	178-184	caspase recruitment domain family member 10	Homo sapiens	149-155
24301787-9	2013	In addition, miR-24 significantly inhibited the expression of FABP4, while it upregulated AP-1 expression, but had no effect on the level of FABP4 mRNA.	mir-24	13-19	fatty acid binding protein 4	Homo sapiens	62-67
26313654-6	2015	Silencing of NKX3.1 could coincide with the effects of miR-24 overexpression.	mir-24	55-61	NK3 homeobox 1	Homo sapiens	13-19
25234642-6	2015	Using miR-24 mimics, we demonstrated a robust decrease in both furin mRNA levels and intracellular furin activity in A549 cells.	mir-24	6-12	furin, paired basic amino acid cleaving enzyme	Homo sapiens	63-68
25234642-6	2015	Using miR-24 mimics, we demonstrated a robust decrease in both furin mRNA levels and intracellular furin activity in A549 cells.	mir-24	6-12	furin, paired basic amino acid cleaving enzyme	Homo sapiens	99-104
25234642-8	2015	Our results suggest that viral-specific downregulation of furin-directed microRNAs such as miR-24 during the life cycle of HP infA viruses may represent a novel regulatory mechanism that governs furin-mediated proteolytic activation of HA0 glycoproteins and production of infectious virions.	mir-24	91-97	furin, paired basic amino acid cleaving enzyme	Homo sapiens	58-63
25234642-8	2015	Our results suggest that viral-specific downregulation of furin-directed microRNAs such as miR-24 during the life cycle of HP infA viruses may represent a novel regulatory mechanism that governs furin-mediated proteolytic activation of HA0 glycoproteins and production of infectious virions.	mir-24	91-97	furin, paired basic amino acid cleaving enzyme	Homo sapiens	195-200
25073511-0	2014	miR-24 promotes the proliferation and invasion of HCC cells by targeting SOX7.	mir-24	0-6	SRY-box transcription factor 7	Homo sapiens	73-77
25073511-6	2014	Forced expression of SOX7 substantially attenuated the oncogenic effects of miR-24.	mir-24	76-82	SRY-box transcription factor 7	Homo sapiens	21-25
25073511-7	2014	Those results strongly suggest that miR-24 plays important role in HCC development partially by targeting SOX7.	mir-24	36-42	SRY-box transcription factor 7	Homo sapiens	106-110
25120807-0	2014	Association between mir-24 and mir-378 in formalin-fixed paraffin-embedded tissues of breast cancer.	mir-24	20-26	microRNA 378a	Homo sapiens	31-38
24301787-9	2013	In addition, miR-24 significantly inhibited the expression of FABP4, while it upregulated AP-1 expression, but had no effect on the level of FABP4 mRNA.	mir-24	13-19	JunB proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	90-94
24301787-11	2013	AP-1 silencing could, at least partially, reverse the inhibitory effect of miR-24 on FABP4 expression.	mir-24	75-81	JunB proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	0-4
24301787-11	2013	AP-1 silencing could, at least partially, reverse the inhibitory effect of miR-24 on FABP4 expression.	mir-24	75-81	fatty acid binding protein 4	Homo sapiens	85-90
23578572-5	2013	Overexpression of miR-24 down-regulated LPAATbeta expression in osteosarcoma cells.	mir-24	18-24	1-acylglycerol-3-phosphate O-acyltransferase 2	Homo sapiens	40-49
23688438-0	2013	A functional variant at miR-24 binding site in B7-H2 alters susceptibility to gastric cancer in a Chinese Han population.	mir-24	24-30	inducible T cell costimulator ligand	Homo sapiens	47-52
23688438-6	2013	Dual-luciferase reporter assays showed that miR-24 inhibited the expression of B7-H2 through binding with the B7-H2 3"-UTR, and this inhibitory role of miR-24 was impacted by rs4819388.	mir-24	44-50	inducible T cell costimulator ligand	Homo sapiens	79-84
23688438-6	2013	Dual-luciferase reporter assays showed that miR-24 inhibited the expression of B7-H2 through binding with the B7-H2 3"-UTR, and this inhibitory role of miR-24 was impacted by rs4819388.	mir-24	44-50	inducible T cell costimulator ligand	Homo sapiens	110-115
23553486-0	2013	MicroRNA miR-24 promotes cell proliferation by targeting the CDKs inhibitors p27Kip1 and p16INK4a.	mir-24	9-15	cyclin dependent kinase inhibitor 1B	Homo sapiens	77-84
23553486-0	2013	MicroRNA miR-24 promotes cell proliferation by targeting the CDKs inhibitors p27Kip1 and p16INK4a.	mir-24	9-15	cyclin dependent kinase inhibitor 2A	Homo sapiens	89-97
23553486-7	2013	In this report we describe that miR-24 directly targets p27(Kip1) and p16(Ink4a) in primary keratinocyte and in different cancer derived cell lines promoting their proliferation, suggesting that miR-24 is involved in cyclin-dependent kinase inhibitors post-transcriptional regulation and that upregulation of miR-24 may play a role in carcinogenesis.	mir-24	32-38	interferon alpha inducible protein 27	Homo sapiens	56-59
23553486-7	2013	In this report we describe that miR-24 directly targets p27(Kip1) and p16(Ink4a) in primary keratinocyte and in different cancer derived cell lines promoting their proliferation, suggesting that miR-24 is involved in cyclin-dependent kinase inhibitors post-transcriptional regulation and that upregulation of miR-24 may play a role in carcinogenesis.	mir-24	32-38	cyclin dependent kinase inhibitor 1B	Homo sapiens	60-64
23553486-7	2013	In this report we describe that miR-24 directly targets p27(Kip1) and p16(Ink4a) in primary keratinocyte and in different cancer derived cell lines promoting their proliferation, suggesting that miR-24 is involved in cyclin-dependent kinase inhibitors post-transcriptional regulation and that upregulation of miR-24 may play a role in carcinogenesis.	mir-24	32-38	cyclin dependent kinase inhibitor 2A	Homo sapiens	70-73
23553486-7	2013	In this report we describe that miR-24 directly targets p27(Kip1) and p16(Ink4a) in primary keratinocyte and in different cancer derived cell lines promoting their proliferation, suggesting that miR-24 is involved in cyclin-dependent kinase inhibitors post-transcriptional regulation and that upregulation of miR-24 may play a role in carcinogenesis.	mir-24	32-38	cyclin dependent kinase inhibitor 2A	Homo sapiens	74-79
23578572-6	2013	Specifically, overexpression of miR-24 inhibited osteosarcoma cell proliferation, however, such effect was blocked when LPAATbeta activity was inhibited.	mir-24	32-38	1-acylglycerol-3-phosphate O-acyltransferase 2	Homo sapiens	120-129
23677957-9	2013	In the residuum stored at 4 C up to 5 days, the relative quantification of miR-106a normalized with miR-24 in colorectal cancer patients was significantly higher than those in healthy volunteers (P &lt; 0.05).	mir-24	100-106	microRNA 106a	Homo sapiens	75-83
22733138-4	2013	Using a heuristic algorithm of bioinformatics analysis and in vitro screening, we identified miR-24 as a candidate regulator of XIAP expression.	mir-24	93-99	X-linked inhibitor of apoptosis	Homo sapiens	128-132
24195361-6	2013	Overexpression of miR-24 in K562 cells promoted the epsilon- and gamma-globin gene expression during hemin-induced erythroid differentiation through targeting the negative globin regulator Sp1.	mir-24	18-24	hemoglobin subunit epsilon 1	Homo sapiens	52-77
23483452-9	2013	MiR-24 was only significantly up-regulated in NF1 MPNST patients.	mir-24	0-6	neurofibromin 1	Homo sapiens	46-49
22733138-9	2013	In summary, our data suggest a novel mechanism by which miR-24 directly modulates XIAP expression level and consequently the apoptosis threshold in cancer cells.	mir-24	56-62	X-linked inhibitor of apoptosis	Homo sapiens	82-86
23142218-0	2013	MiR-24 regulates the proliferation and invasion of glioma by ST7L via beta-catenin/Tcf-4 signaling.	mir-24	0-6	suppression of tumorigenicity 7 like	Homo sapiens	61-65
23142218-0	2013	MiR-24 regulates the proliferation and invasion of glioma by ST7L via beta-catenin/Tcf-4 signaling.	mir-24	0-6	catenin beta 1	Homo sapiens	70-82
23142218-0	2013	MiR-24 regulates the proliferation and invasion of glioma by ST7L via beta-catenin/Tcf-4 signaling.	mir-24	0-6	transcription factor 4	Homo sapiens	83-88
23142218-7	2013	Mechanistic investigation revealed that the deletion of miR-24 suppressed beta-catenin/Tcf-4 transcription activity by targeting ST7L.	mir-24	56-62	catenin beta 1	Homo sapiens	74-86
23142218-7	2013	Mechanistic investigation revealed that the deletion of miR-24 suppressed beta-catenin/Tcf-4 transcription activity by targeting ST7L.	mir-24	56-62	transcription factor 4	Homo sapiens	87-92
23142218-7	2013	Mechanistic investigation revealed that the deletion of miR-24 suppressed beta-catenin/Tcf-4 transcription activity by targeting ST7L.	mir-24	56-62	suppression of tumorigenicity 7 like	Homo sapiens	129-133
23213373-8	2012	The level of miR-24, a microRNA targeting HNF-4alpha, was reduced in spherical FLC-4 cells.	mir-24	13-19	hepatocyte nuclear factor 4 alpha	Homo sapiens	42-52
22435726-0	2012	Upregulation of miR-24 is associated with a decreased DNA damage response upon etoposide treatment in highly differentiated CD8(+) T cells sensitizing them to apoptotic cell death.	mir-24	16-22	CD8a molecule	Homo sapiens	124-127
22891046-0	2012	Mir-24 regulates junctophilin-2 expression in cardiomyocytes.	mir-24	0-6	junctophilin 2	Homo sapiens	17-31
22891046-5	2012	METHODS AND RESULTS: Bioinformatic analysis predicted 2 potential binding sites of miR-24 in the 3"-untranslated regions of JP2 mRNA.	mir-24	83-89	junctophilin 2	Homo sapiens	124-127
22891046-6	2012	Luciferase assays confirmed that miR-24 suppressed JP2 expression by binding to either of these sites.	mir-24	33-39	junctophilin 2	Homo sapiens	51-54
22891046-8	2012	Adenovirus-directed overexpression of miR-24 in cardiomyocytes decreased JP2 expression and reduced Ca(2+) transient amplitude and E-C coupling gain.	mir-24	38-44	junctophilin 2	Homo sapiens	73-76
22891046-9	2012	CONCLUSIONS: MiR-24-mediated suppression of JP2 expression provides a novel molecular mechanism for E-C coupling regulation in heart cells and suggests a new target against heart failure.	mir-24	13-19	junctophilin 2	Homo sapiens	44-47
22435726-4	2012	The increased expression of miR-24 in CD8(+) CD28(-)  T cells is associated with decreased expression of the histone variant H2AX, a protein that plays a key role in the DNA damage response (DDR).	mir-24	28-34	H2A.X variant histone	Homo sapiens	125-129
22260784-8	2012	By performing microarray analyses and bioinformatics analyses, we found furin to be a potential target for miR-24 in fibrosis (furin is a protease which controls latent TGF-beta activation processing).	mir-24	107-113	furin, paired basic amino acid cleaving enzyme	Homo sapiens	72-77
22260784-8	2012	By performing microarray analyses and bioinformatics analyses, we found furin to be a potential target for miR-24 in fibrosis (furin is a protease which controls latent TGF-beta activation processing).	mir-24	107-113	furin, paired basic amino acid cleaving enzyme	Homo sapiens	127-132
22260784-8	2012	By performing microarray analyses and bioinformatics analyses, we found furin to be a potential target for miR-24 in fibrosis (furin is a protease which controls latent TGF-beta activation processing).	mir-24	107-113	transforming growth factor beta 1	Homo sapiens	169-177
22260784-9	2012	Finally, we demonstrated that protein and mRNA levels of furin were regulated by miR-24 in CFs.	mir-24	81-87	furin, paired basic amino acid cleaving enzyme	Homo sapiens	57-62
22260784-10	2012	These findings suggest that miR-24 has a critical role in CF function and cardiac fibrosis after MI through a furin-TGF-beta pathway.	mir-24	28-34	furin, paired basic amino acid cleaving enzyme	Homo sapiens	110-115
22260784-10	2012	These findings suggest that miR-24 has a critical role in CF function and cardiac fibrosis after MI through a furin-TGF-beta pathway.	mir-24	28-34	transforming growth factor beta 1	Homo sapiens	116-124
22435726-4	2012	The increased expression of miR-24 in CD8(+) CD28(-)  T cells is associated with decreased expression of the histone variant H2AX, a protein that plays a key role in the DNA damage response (DDR).	mir-24	28-34	CD8a molecule	Homo sapiens	38-41
20945401-4	2011	CMS induced the expression of miR-24 that led to the down regulation of the subtilisin-like proprotein convertase FURIN, which is known to play a major role in the processing of TGFbeta1.	mir-24	30-36	furin, paired basic amino acid cleaving enzyme	Homo sapiens	114-119
22387692-6	2012	Overexpression of miR-24 into HuH-7 and HepG2 cells significantly decreased the ARNT protein level, but not the ARNT mRNA level, indicating translational repression.	mir-24	18-24	MIR7-3 host gene	Homo sapiens	30-35
22387692-6	2012	Overexpression of miR-24 into HuH-7 and HepG2 cells significantly decreased the ARNT protein level, but not the ARNT mRNA level, indicating translational repression.	mir-24	18-24	aryl hydrocarbon receptor nuclear translocator	Homo sapiens	80-84
22387692-6	2012	Overexpression of miR-24 into HuH-7 and HepG2 cells significantly decreased the ARNT protein level, but not the ARNT mRNA level, indicating translational repression.	mir-24	18-24	aryl hydrocarbon receptor nuclear translocator	Homo sapiens	112-116
22387692-10	2012	Finally, it was demonstrated that the miR-24 levels in a panel of 26 human livers were inversely correlated with the protein levels or the translational efficiency of ARNT.	mir-24	38-44	aryl hydrocarbon receptor nuclear translocator	Homo sapiens	167-171
22387692-12	2012	miR-24 would be one of the factors underlying the mechanisms by which ARNT protein is decreased by reactive oxygen species.	mir-24	0-6	aryl hydrocarbon receptor nuclear translocator	Homo sapiens	70-74
22139384-0	2012	miR-24 functions as a tumor suppressor in Hep2 laryngeal carcinoma cells             partly through down-regulation of the S100A8 protein.	mir-24	0-6	S100 calcium binding protein A8	Homo sapiens	123-129
22139384-3	2012	Ectopic expression of miR-24 in Hep2 cells significantly induced cell morphology changes and inhibited cell proliferation and invasion ability in vitro by targeting S100A8 at the translational level.	mir-24	22-28	S100 calcium binding protein A8	Homo sapiens	165-171
22139384-4	2012	Meanwhile, miR-24 could significantly inhibit Hep2 cell invasion after S100A8 protein blockade.	mir-24	11-17	S100 calcium binding protein A8	Homo sapiens	71-77
22139384-5	2012	In conclusion, our results suggest that miR-24 may function as a tumor suppressor in LSCC through down-regulation of S100A8, which suggests that miR-24 could serve as a novel potential maker for LSCC therapy.	mir-24	40-46	S100 calcium binding protein A8	Homo sapiens	117-123
22139384-5	2012	In conclusion, our results suggest that miR-24 may function as a tumor suppressor in LSCC through down-regulation of S100A8, which suggests that miR-24 could serve as a novel potential maker for LSCC therapy.	mir-24	145-151	S100 calcium binding protein A8	Homo sapiens	117-123
22336108-11	2012	miR-24, a microRNA that represses p14ARF expression, is expressed in retinoblastoma cell lines and correlates with lower protein expression when compared to other cell lines with high p14ARF mRNA.	mir-24	0-6	cyclin dependent kinase inhibitor 2A	Homo sapiens	34-40
22336108-11	2012	miR-24, a microRNA that represses p14ARF expression, is expressed in retinoblastoma cell lines and correlates with lower protein expression when compared to other cell lines with high p14ARF mRNA.	mir-24	0-6	cyclin dependent kinase inhibitor 2A	Homo sapiens	184-190
22336108-12	2012	Transient over-expression of siRNA against miR-24 led to elevated p14ARF protein in retinoblastoma cells.	mir-24	43-49	cyclin dependent kinase inhibitor 2A	Homo sapiens	66-72
21986943-6	2012	Furthermore, we identified the microRNA miR-24 as a novel post-transcriptional regulator of Net1A expression.	mir-24	40-46	neuroepithelial cell transforming 1	Homo sapiens	92-97
21986943-8	2012	Finally, miR-24 was found to be implicated in the regulation of the EMT program in response to TGF-beta and was shown to be directly involved in the TGF-beta-induced breast cancer cell invasiveness through Net1A regulation.	mir-24	9-15	transforming growth factor beta 1	Homo sapiens	95-103
21986943-8	2012	Finally, miR-24 was found to be implicated in the regulation of the EMT program in response to TGF-beta and was shown to be directly involved in the TGF-beta-induced breast cancer cell invasiveness through Net1A regulation.	mir-24	9-15	transforming growth factor beta 1	Homo sapiens	149-157
21986943-8	2012	Finally, miR-24 was found to be implicated in the regulation of the EMT program in response to TGF-beta and was shown to be directly involved in the TGF-beta-induced breast cancer cell invasiveness through Net1A regulation.	mir-24	9-15	neuroepithelial cell transforming 1	Homo sapiens	206-211
23098654-9	2012	Overall, this study showed a protective role for miR-24 against myocardial ischemia by inhibiting BCL2L11, and may represent a potential novel treatment for ischemic heart disease.	mir-24	49-55	Bcl2-like 11	Rattus norvegicus	98-105
20945401-4	2011	CMS induced the expression of miR-24 that led to the down regulation of the subtilisin-like proprotein convertase FURIN, which is known to play a major role in the processing of TGFbeta1.	mir-24	30-36	transforming growth factor beta 1	Homo sapiens	178-186
20945401-5	2011	FURIN was confirmed as a novel target of miR-24 by 3" UTR luciferase assay and western blot.	mir-24	41-47	furin, paired basic amino acid cleaving enzyme	Homo sapiens	0-5
20945401-6	2011	Overexpression of miR-24 resulted in a significant decrease in activated TGFbeta1.	mir-24	18-24	transforming growth factor beta 1	Homo sapiens	73-81
20945401-8	2011	Conversely, inhibition of miR-24 expression with a specific antagomir led to a small but significant increase in TGFbeta1.	mir-24	26-32	transforming growth factor beta 1	Homo sapiens	113-121
20945401-9	2011	Furthermore, the increase in active TGFbeta1 induced by CMS in HTM cells was prevented by miR-24.	mir-24	90-96	transforming growth factor beta 1	Homo sapiens	36-44
20945401-11	2011	Specifically, miR-24 might play an important role in modulating the induction of TGFbeta1 mediated by CMS through direct targeting of FURIN.	mir-24	14-20	transforming growth factor beta 1	Homo sapiens	81-89
20945401-11	2011	Specifically, miR-24 might play an important role in modulating the induction of TGFbeta1 mediated by CMS through direct targeting of FURIN.	mir-24	14-20	furin, paired basic amino acid cleaving enzyme	Homo sapiens	134-139
20062076-6	2010	Transfection of an miR-24 into gastric cancer cells reduced the elevation of the AE1 protein, which resulted in return of AE1-sequestrated p16 to the nucleus, thereby inhibiting proliferation of the cells.	mir-24	19-25	solute carrier family 4 member 1 (Diego blood group)	Homo sapiens	81-84
21383058-0	2011	miR-24 inhibits apoptosis and represses Bim in mouse cardiomyocytes.	mir-24	0-6	BCL2-like 11 (apoptosis facilitator)	Mus musculus	40-43
21383058-5	2011	miR-24 suppresses cardiomyocyte apoptosis, in part by direct repression of the BH3-only domain-containing protein Bim, which positively regulates apoptosis.	mir-24	0-6	BCL2-like 11 (apoptosis facilitator)	Mus musculus	114-117
20816961-3	2010	MiR-24 up-regulation reduced the expression of RNA-binding protein dead end 1 (DND1).	mir-24	0-6	DND microRNA-mediated repression inhibitor 1	Homo sapiens	79-83
20816961-6	2010	Furthermore, the miR-24-mediated change in DND1 expression suppressed the expression of cyclin-dependent kinase inhibitor 1B (CDKN1B), and also led to enhanced proliferation and reduced apoptosis in TSCC cells.	mir-24	17-23	DND microRNA-mediated repression inhibitor 1	Homo sapiens	43-47
20816961-6	2010	Furthermore, the miR-24-mediated change in DND1 expression suppressed the expression of cyclin-dependent kinase inhibitor 1B (CDKN1B), and also led to enhanced proliferation and reduced apoptosis in TSCC cells.	mir-24	17-23	cyclin dependent kinase inhibitor 1B	Homo sapiens	88-124
20816961-6	2010	Furthermore, the miR-24-mediated change in DND1 expression suppressed the expression of cyclin-dependent kinase inhibitor 1B (CDKN1B), and also led to enhanced proliferation and reduced apoptosis in TSCC cells.	mir-24	17-23	cyclin dependent kinase inhibitor 1B	Homo sapiens	126-132
20062076-6	2010	Transfection of an miR-24 into gastric cancer cells reduced the elevation of the AE1 protein, which resulted in return of AE1-sequestrated p16 to the nucleus, thereby inhibiting proliferation of the cells.	mir-24	19-25	solute carrier family 4 member 1 (Diego blood group)	Homo sapiens	122-125
20062076-6	2010	Transfection of an miR-24 into gastric cancer cells reduced the elevation of the AE1 protein, which resulted in return of AE1-sequestrated p16 to the nucleus, thereby inhibiting proliferation of the cells.	mir-24	19-25	cyclin dependent kinase inhibitor 2A	Homo sapiens	139-142
20062076-7	2010	Furthermore, the miR-24 inhibitor cooperated with hemin to induce the expression of AE1 in K562 cells and differentiation of the cells, which is consistent with results obtained from the cells cultured at pH 7.6 or from forced stable expression of AE1.	mir-24	17-23	solute carrier family 4 member 1 (Diego blood group)	Homo sapiens	84-87
20062076-7	2010	Furthermore, the miR-24 inhibitor cooperated with hemin to induce the expression of AE1 in K562 cells and differentiation of the cells, which is consistent with results obtained from the cells cultured at pH 7.6 or from forced stable expression of AE1.	mir-24	17-23	solute carrier family 4 member 1 (Diego blood group)	Homo sapiens	248-251
20062076-8	2010	These findings establish a novel regulation of miR-24-related AE1 expression in gastric carcinogenesis and erythropoiesis.	mir-24	47-53	solute carrier family 4 member 1 (Diego blood group)	Homo sapiens	62-65
17906079-0	2008	MicroRNA miR-24 inhibits erythropoiesis by targeting activin type I receptor ALK4.	mir-24	9-15	activin A receptor type 1B	Homo sapiens	77-81
20195546-0	2010	miR-24 regulates apoptosis by targeting the open reading frame (ORF) region of FAF1 in cancer cells.	mir-24	0-6	Fas associated factor 1	Homo sapiens	79-83
20195546-6	2010	CONCLUSIONS/SIGNIFICANCE: We found that miR-24 regulates apoptosis by targeting FAF1 in cancer cells.	mir-24	40-46	Fas associated factor 1	Homo sapiens	80-84
20041160-0	2009	MiR-24 tumor suppressor activity is regulated independent of p53 and through a target site polymorphism.	mir-24	0-6	tumor protein p53	Homo sapiens	61-64
20041160-5	2009	MiR-24 over expression in cells with wt-p53 upregulated TP53 and p21 protein; however, in p53-null cells miR-24 still induced cell cycle arrest without the involvement of p21.	mir-24	0-6	tumor protein p53	Homo sapiens	40-43
20041160-5	2009	MiR-24 over expression in cells with wt-p53 upregulated TP53 and p21 protein; however, in p53-null cells miR-24 still induced cell cycle arrest without the involvement of p21.	mir-24	0-6	tumor protein p53	Homo sapiens	56-60
20041160-5	2009	MiR-24 over expression in cells with wt-p53 upregulated TP53 and p21 protein; however, in p53-null cells miR-24 still induced cell cycle arrest without the involvement of p21.	mir-24	0-6	H3 histone pseudogene 16	Homo sapiens	65-68
20041160-9	2009	A novel function for miR-24 as a p53-independent cell cycle inhibitory miRNA is proposed.	mir-24	21-27	tumor protein p53	Homo sapiens	33-36
19748357-4	2009	miR-24 directly regulates MYC and E2F2 and some genes that they transactivate.	mir-24	0-6	MYC proto-oncogene, bHLH transcription factor	Homo sapiens	26-29
19748357-4	2009	miR-24 directly regulates MYC and E2F2 and some genes that they transactivate.	mir-24	0-6	E2F transcription factor 2	Homo sapiens	34-38
19748357-5	2009	Enhanced proliferation from antagonizing miR-24 is abrogated by knocking down E2F2, but not MYC, and cell proliferation, inhibited by miR-24 overexpression, is rescued by miR-24-insensitive E2F2.	mir-24	41-47	E2F transcription factor 2	Homo sapiens	78-82
19748357-5	2009	Enhanced proliferation from antagonizing miR-24 is abrogated by knocking down E2F2, but not MYC, and cell proliferation, inhibited by miR-24 overexpression, is rescued by miR-24-insensitive E2F2.	mir-24	134-140	E2F transcription factor 2	Homo sapiens	78-82
19377482-0	2009	miR-24-mediated downregulation of H2AX suppresses DNA repair in terminally differentiated blood cells.	mir-24	0-6	H2A.X variant histone	Homo sapiens	34-38
19377482-5	2009	miR-24-mediated suppression of H2AX renders cells hypersensitive to gamma-irradiation and genotoxic drugs, a phenotype that is fully rescued by overexpression of miR-24-insensitive H2AX.	mir-24	0-6	H2A.X variant histone	Homo sapiens	31-35
19377482-5	2009	miR-24-mediated suppression of H2AX renders cells hypersensitive to gamma-irradiation and genotoxic drugs, a phenotype that is fully rescued by overexpression of miR-24-insensitive H2AX.	mir-24	0-6	H2A.X variant histone	Homo sapiens	181-185
19377482-5	2009	miR-24-mediated suppression of H2AX renders cells hypersensitive to gamma-irradiation and genotoxic drugs, a phenotype that is fully rescued by overexpression of miR-24-insensitive H2AX.	mir-24	162-168	H2A.X variant histone	Homo sapiens	31-35
18365017-0	2008	p16(INK4a) translation suppressed by miR-24.	mir-24	37-43	cyclin dependent kinase inhibitor 2A	Homo sapiens	0-3
18365017-0	2008	p16(INK4a) translation suppressed by miR-24.	mir-24	37-43	cyclin dependent kinase inhibitor 2A	Homo sapiens	4-9
18365017-6	2008	CONCLUSIONS/SIGNIFICANCE: Together, our results suggest that miR-24 represses the initiation and elongation phases of p16 translation.	mir-24	61-67	cyclin dependent kinase inhibitor 2A	Homo sapiens	118-121
19826043-4	2009	miR-24 directly downregulates mitogen-activated protein kinase (MAPK) phosphatase-7 and enhances phosphorylation of both c-jun-NH(2)-kinase and p38 kinases.	mir-24	0-6	mitogen-activated protein kinase 14	Homo sapiens	144-147
17906079-2	2008	Here we report that miR-24 regulates erythroid differentiation by influencing the expression of human activin type I receptor ALK4 (hALK4).	mir-24	20-26	activin A receptor type 1B	Homo sapiens	126-130
17906079-2	2008	Here we report that miR-24 regulates erythroid differentiation by influencing the expression of human activin type I receptor ALK4 (hALK4).	mir-24	20-26	activin A receptor type 1B	Homo sapiens	132-137
17906079-3	2008	Ectopic expression of miR-24 reduces the mRNA and protein levels of hALK4 by targeting the 3"-untranslated region of hALK4 mRNA and interferes with activin-induced Smad2 phosphorylation and reporter expression.	mir-24	22-28	activin A receptor type 1B	Homo sapiens	68-73
17906079-3	2008	Ectopic expression of miR-24 reduces the mRNA and protein levels of hALK4 by targeting the 3"-untranslated region of hALK4 mRNA and interferes with activin-induced Smad2 phosphorylation and reporter expression.	mir-24	22-28	activin A receptor type 1B	Homo sapiens	117-122
17906079-3	2008	Ectopic expression of miR-24 reduces the mRNA and protein levels of hALK4 by targeting the 3"-untranslated region of hALK4 mRNA and interferes with activin-induced Smad2 phosphorylation and reporter expression.	mir-24	22-28	SMAD family member 2	Homo sapiens	164-169
17906079-4	2008	Furthermore, miR-24 represses the activin-mediated accumulation of hemoglobin, an erythroid differentiation marker, in erythroleukemic K562 cells and decreases erythroid colony-forming and burst-forming units of CD34+ hematopoietic progenitor cells.	mir-24	13-19	CD34 molecule	Homo sapiens	212-216
17906079-6	2008	Thus, our findings define a regulation mode of miR-24 on erythropoiesis by impeding ALK4 expression.	mir-24	47-53	activin A receptor type 1B	Homo sapiens	84-88