PMID-sentid	Pub_year	Sent_text	compound_name	comp_offset	prot_official_name	organism	prot_offset
34111561-6	2021	Accordingly, ablation of the ALCAT1 gene or pharmacological inhibition of the ALCAT1 enzyme by Dafaglitapin (Dafa), a potent and highly specific ALCAT1 inhibitor, not only restored TLCL levels, but also mitochondrial respiration by attenuating signal transduction pathways mediated by HIF-1alpha.	dafaglitapin	95-107	lysocardiolipin acyltransferase 1	Homo sapiens	29-35
34111561-6	2021	Accordingly, ablation of the ALCAT1 gene or pharmacological inhibition of the ALCAT1 enzyme by Dafaglitapin (Dafa), a potent and highly specific ALCAT1 inhibitor, not only restored TLCL levels, but also mitochondrial respiration by attenuating signal transduction pathways mediated by HIF-1alpha.	dafaglitapin	95-107	lysocardiolipin acyltransferase 1	Homo sapiens	78-84
34111561-6	2021	Accordingly, ablation of the ALCAT1 gene or pharmacological inhibition of the ALCAT1 enzyme by Dafaglitapin (Dafa), a potent and highly specific ALCAT1 inhibitor, not only restored TLCL levels, but also mitochondrial respiration by attenuating signal transduction pathways mediated by HIF-1alpha.	dafaglitapin	95-107	lysocardiolipin acyltransferase 1	Homo sapiens	145-151
34111561-6	2021	Accordingly, ablation of the ALCAT1 gene or pharmacological inhibition of the ALCAT1 enzyme by Dafaglitapin (Dafa), a potent and highly specific ALCAT1 inhibitor, not only restored TLCL levels, but also mitochondrial respiration by attenuating signal transduction pathways mediated by HIF-1alpha.	dafaglitapin	95-107	hypoxia inducible factor 1 subunit alpha	Homo sapiens	285-295