PMID-sentid	Pub_year	Sent_text	compound_name	comp_offset	prot_official_name	organism	prot_offset
26129954-5	2015	Statistical analysis was performed, which indicated that BAI-1 expression levels in the normal bladder mucosa group were significantly higher than those in the BTCC group and were associated with clinical staging.	S-Butyrylthiocholine chloride	160-164	adhesion G protein-coupled receptor B1	Homo sapiens	57-62
32864010-3	2020	This study aimed to investigate the function of DRAM2 in regulating gemcitabine resistance of BTCC.	S-Butyrylthiocholine chloride	94-98	DNA damage regulated autophagy modulator 2	Homo sapiens	48-53
32864010-10	2020	Conclusions: Our data suggested that downregulation of DRAM2 rescued the sensitivity of T24-GEM cells to gemcitabine, providing an appropriate therapeutic target for BTCC treatment.	S-Butyrylthiocholine chloride	166-170	DNA damage regulated autophagy modulator 2	Homo sapiens	55-60
25286682-5	2014	CONCLUSION: DAB2IP may be one of the important inhibitory factors during the occurrence and progression of BTCC.	S-Butyrylthiocholine chloride	107-111	DAB2 interacting protein	Homo sapiens	12-18
24434421-5	2014	These effects were abolished by pre-incubation of cells with concentrations of BTCC or PTCC that maximize HO-1 induction and were reversed by the inhibitor of heme oxygenase activity tin protoporphyrin IX (SnPPIX).	S-Butyrylthiocholine chloride	79-83	heme oxygenase 1	Rattus norvegicus	106-110
34434217-5	2021	Finally, survival analysis of these hub genes found that three of them (CCNB1, ASPM, and ACTC1) were conspicuously related to the prognosis of patients with BTCC (p < 0.05).	S-Butyrylthiocholine chloride	157-161	cyclin B1	Homo sapiens	72-77
34434217-5	2021	Finally, survival analysis of these hub genes found that three of them (CCNB1, ASPM, and ACTC1) were conspicuously related to the prognosis of patients with BTCC (p < 0.05).	S-Butyrylthiocholine chloride	157-161	assembly factor for spindle microtubules	Homo sapiens	79-83
34434217-5	2021	Finally, survival analysis of these hub genes found that three of them (CCNB1, ASPM, and ACTC1) were conspicuously related to the prognosis of patients with BTCC (p < 0.05).	S-Butyrylthiocholine chloride	157-161	actin alpha cardiac muscle 1	Homo sapiens	89-94
12934404-4	2003	RESULTS: The content of total MMP9 in the BTCC group was significantly higher than that in the other groups, and the content increased with the tumor grade and stage aggravation.	S-Butyrylthiocholine chloride	42-46	matrix metallopeptidase 9	Homo sapiens	30-34