PMID-sentid Pub_year Sent_text compound_name comp_offset prot_official_name organism prot_offset 24241852-3 1986 We show that the accumulation of beta-1,3-glucanase in cultured pith-parenchyma tissue is blocked by combinations of the auxin, alpha-naphthaleneacetic acid (NAA), and the cytokinin, kinetin. 1-naphthaleneacetic acid 128-156 glucan endo-1,3-beta-glucosidase, acidic-like Nicotiana tabacum 33-51 24201672-2 1989 The spatial distribution of the ABP in the maize (Zea mays L.) mesocotyl corresponds with the distribution of growth induced by naphthalene-1-acetic acid and with the distribution of Site I binding as previously shown by J.D. 1-naphthaleneacetic acid 128-153 auxin-binding protein 4 Zea mays 32-35 34071854-0 2021 Design and Evaluation of 223Ra-Labeled and Anti-PSMA Targeted NaA Nanozeolites for Prostate Cancer Therapy-Part II. 1-naphthaleneacetic acid 62-65 folate hydrolase 1 Homo sapiens 48-52 34071854-5 2021 In this paper, we extended our in vitro study of 223Ra-labeled and PSMA-targeted NaA nanozeolites (223RaA-silane-PEG-D2B) by undertaking comprehensive preclinical in vitro and in vivo research. 1-naphthaleneacetic acid 81-84 folate hydrolase 1 Homo sapiens 67-71 34570571-5 2021 In the patients with a stable disease, strong significant negative correlations between Cho/Cr and Cho/NAA with p53 mutation (-0.945 and -0.812 respectively, p < 0.05) and between Cho/Cr and IDH1, 2 mutation (-0.796, p < 0.05) were found. 1-naphthaleneacetic acid 103-106 tumor protein p53 Homo sapiens 112-115 34570571-6 2021 In the patients with tumour progression, a significant positive correlation of NAA/Cr with 1p19q codeletion (0.486, p < 0.05) and of Cho/Cr and Cho/NAA values with p53 mutation (0.477 and 0.416, p < 0.05) were identified. 1-naphthaleneacetic acid 148-151 tumor protein p53 Homo sapiens 164-167 35605136-0 2022 Interzeolite Conversion-Synthesized Sub-1 mum NaA Zeolite Membrane for C2H2/C2H4 Selective Separation. 1-naphthaleneacetic acid 46-49 SUB1 regulator of transcription Homo sapiens 36-41 35245283-6 2022 Physiological analysis illustrated that, unlike picloram, exogenous application of NAA can not restore these defective phenotypes, implying that PIN3-mediated polar auxin transport is required for the late ovule initiation and pistil length. 1-naphthaleneacetic acid 83-86 Auxin efflux carrier family protein Arabidopsis thaliana 145-149 24241852-3 1986 We show that the accumulation of beta-1,3-glucanase in cultured pith-parenchyma tissue is blocked by combinations of the auxin, alpha-naphthaleneacetic acid (NAA), and the cytokinin, kinetin. 1-naphthaleneacetic acid 158-161 glucan endo-1,3-beta-glucosidase, acidic-like Nicotiana tabacum 33-51 24241852-4 1986 When tissues pre-incubated for 7 d on complete medium containing 2.0 mg l(-1) NAA and 0.3 mg l(-1) kinetin are transferred onto medium without hormones or with either hormone added separately, the beta-1,3-glucanase content expressed per mg soluble protein increases approx. 1-naphthaleneacetic acid 78-81 glucan endo-1,3-beta-glucosidase, acidic-like Nicotiana tabacum 197-215 2991266-11 1985 The ABP has a sharp binding optimum at pH 5.5, and the KD was calculated to be 5.7 X 10(-8) M to [14C]NAA. 1-naphthaleneacetic acid 102-105 auxin-binding protein 4 Zea mays 4-7 6184589-3 1982 Nitroprusside (NP), hydroxylamine (HA) and sodium azide (NaA) increased c-GMP levels and also enhanced amylase release in a dose-dependent manner; cyclic-AMP (c-AMP) levels were not affected. 1-naphthaleneacetic acid 57-60 5'-nucleotidase, cytosolic II Mus musculus 74-77 19872676-15 1932 The formation of KA and NaA in the sap raises its osmotic pressure and water enters. 1-naphthaleneacetic acid 24-27 SH2 domain containing 1A Homo sapiens 35-38 1148239-1 1975 The minor bases present in the family of Drosophila tRNAs recognising codons of the type NAA or NAG have been studied. 1-naphthaleneacetic acid 89-92 transfer RNA:Serine-GCT 2-5 Drosophila melanogaster 52-57 33734402-9 2021 However, both NAA and IAA at low concentrations rapidly and specifically promoted endocytosis of photo-converted PIN2 from the PM. 1-naphthaleneacetic acid 14-17 telomeric repeat binding factor 1 Homo sapiens 113-117 33734402-10 2021 These analyses identify a specific effect of NAA and IAA on PIN2 endocytosis, thus contributing to its polarity maintenance and furthermore illustrate that high auxin levels have nonspecific effects on trafficking and endomembrane compartments. 1-naphthaleneacetic acid 45-48 telomeric repeat binding factor 1 Homo sapiens 60-64 33788431-11 2021 The effects of INA group were significantly superior to those of NAA group in reducing symptom score, SP and VIP expression, and serum IgE and IL-4 contents and up-regulating NPY expression and IFN-gammacontent (P<0.05, P<0.01). 1-naphthaleneacetic acid 65-68 interleukin-4 Oryctolagus cuniculus 143-147 31727633-9 2020 Finally, we present highly penetrant defects from NAA-mediated degradation of the FTZ-F1 nuclear hormone receptor, NHR-25, during C. elegans uterine-vulval development. 1-naphthaleneacetic acid 50-53 Nuclear hormone receptor family member nhr-25 Caenorhabditis elegans 115-121 32319527-6 2020 RESULTS: In tissue samples obtained from the CEL, elevated relative cerebral blood volume (rCBV) was associated with the presence of recurrent tumor pathology (p<0.03), while increases in normalized choline (nCho) and choline-to-NAA index (CNI) were associated with the presence of recurrent tumor pathology in NEL tissue samples (p<0.008). 1-naphthaleneacetic acid 229-232 carboxyl ester lipase Homo sapiens 45-48 32887308-0 2020 Design and Evaluation of 223Ra-Labeled and Anti-PSMA Targeted NaA Nanozeolites for Prostate Cancer Therapy-Part I. Prostate cancer is the second most frequent malignancy in men worldwide. 1-naphthaleneacetic acid 62-65 folate hydrolase 1 Homo sapiens 48-52 32335162-3 2020 Therefore, we developed a simple and highly accurate KE-2 test method named alpha-Sens that uses the dual luciferase assay system and attempted a further application of luciferase-based determination of cell viability to calculate the normalized Antioxidant response element (ARE)-mediated transcriptional activity, named normalized ARE Activity (nAA), to evaluate the sensitizing potential of chemicals. 1-naphthaleneacetic acid 347-350 prefoldin subunit 6 Homo sapiens 53-57 32335162-8 2020 Our results suggest that better prediction capacity could be achieved by using nAA as a classifier compared to other existing KE-2 test methods. 1-naphthaleneacetic acid 79-82 prefoldin subunit 6 Homo sapiens 126-130 32170435-4 2020 After that, human serum albumin (HSA) was immobilized into the NAA pores by using glutaraldehyde as a cross-linking agent. 1-naphthaleneacetic acid 63-66 albumin Homo sapiens 18-31 32211379-1 2020 A label-free electrochemical detection platform for the sensitive and rapid detection of Flightless I (Flii) protein, a biomarker of wound chronicity, has been developed using nanoporous anodic alumina (NAA) membranes modified with Flii antibody recognition sites. 1-naphthaleneacetic acid 203-206 FLII actin remodeling protein Homo sapiens 89-101 32211379-1 2020 A label-free electrochemical detection platform for the sensitive and rapid detection of Flightless I (Flii) protein, a biomarker of wound chronicity, has been developed using nanoporous anodic alumina (NAA) membranes modified with Flii antibody recognition sites. 1-naphthaleneacetic acid 203-206 FLII actin remodeling protein Homo sapiens 103-107 31879119-8 2020 RESULTS: MRS data which predict a poor neurological outcome (G2 and 3) include the following: decreased N-acetyl aspartate (NAA) (sensitivity 88%, specificity 100%), decreased creatine (47%, 100%), increased lactate (47%, 100%), and decreased glutamate (sensitivity 35%, specificity 100%). 1-naphthaleneacetic acid 124-127 crystallin gamma E, pseudogene Homo sapiens 61-69 33576334-3 2021 In addition, we established a new editor variant recognizing an NAA PAM, expanding the base editing possibilities in zebrafish. 1-naphthaleneacetic acid 64-67 peptidylglycine alpha-amidating monooxygenase Danio rerio 68-71 32878466-10 2020 Serum tumor necrosis factor alpha levels at 3 h after infusion were significantly higher in CAA group than in NAA group. 1-naphthaleneacetic acid 110-113 tumor necrosis factor Homo sapiens 6-33 31838195-4 2020 SCA1, 2, 3, 6, and FA patients showed overall decreased NAA/Cr compared to controls. 1-naphthaleneacetic acid 56-59 ataxin 1 Homo sapiens 0-4 31245362-3 2019 The sensing platform consists of an anti-Flii antibody (Ab1)-modified NAA membrane attached onto a gold electrode. 1-naphthaleneacetic acid 70-73 FLII actin remodeling protein Homo sapiens 41-45 31838195-7 2020 SCA2 subjects showed the lowest NAA/Cr and Cho/Cr in cerebellum and the highest mI/Cr compared to controls and other genotypes, and therefore the most promising results for a potential biomarker. 1-naphthaleneacetic acid 32-35 ataxin 2 Homo sapiens 0-4 30496625-5 2019 The root elongation of plc2 mutants was less sensitive to the high concentration of exogenous auxins, and the application of 1-naphthaleneacetic acid or the auxin transport inhibitor N-1-naphthylphthalamic acid could rescue the root hair growth of plc2 mutants. 1-naphthaleneacetic acid 125-149 phospholipase C 2 Arabidopsis thaliana 23-27 30496625-5 2019 The root elongation of plc2 mutants was less sensitive to the high concentration of exogenous auxins, and the application of 1-naphthaleneacetic acid or the auxin transport inhibitor N-1-naphthylphthalamic acid could rescue the root hair growth of plc2 mutants. 1-naphthaleneacetic acid 125-149 phospholipase C 2 Arabidopsis thaliana 248-252 30886224-4 2019 The primary acetylation site is K550, located in the Rev1-interacting region. 1-naphthaleneacetic acid 32-36 REV1 DNA directed polymerase Homo sapiens 53-57 31110507-7 2019 In squash, CpMAKR4 was up-regulated by naphthylacetic acid (NAA) and, similar to MAKR4 in Arabidopsis, indole-3-butyric acid (IBA). 1-naphthaleneacetic acid 39-58 membrane-associated kinase regulator Arabidopsis thaliana 13-18 31110507-7 2019 In squash, CpMAKR4 was up-regulated by naphthylacetic acid (NAA) and, similar to MAKR4 in Arabidopsis, indole-3-butyric acid (IBA). 1-naphthaleneacetic acid 60-63 membrane-associated kinase regulator Arabidopsis thaliana 13-18 31432034-9 2019 Adjustment of analysis based on gender revealed a significant correlation between CA-125 level and diagnosis of AA only in females (34.23 +- 39 U/mL in NAA versus 20.7 +- 26.7 U/mL in AA, p = 0.012). 1-naphthaleneacetic acid 152-155 mucin 16, cell surface associated Homo sapiens 82-88 29177310-4 2017 Given the swollen elastic modulus and the dependence of charge density on the equilibrium gel volume, it would seem that the latter factor is an important determinant of non-Gaussian elasticity of polyelectrolyte P(NIPA-co-NaA) hydrogels containing strongly dissociated groups. 1-naphthaleneacetic acid 223-226 zinc finger C3HC-type containing 1 Homo sapiens 215-219 30937140-9 2019 Results: GLCCI1 rs37973 homozygotes mutant genotype GG had a higher plasma epinephrine concentration (median concentration 27.032 pg/ml, nGG = 36; median concentration 23.149 pg/ml, nAA+AG = 146; P = 0.015) and cortisol concentration (median concentration 1.141 ng/ml, nGG = 36; median concentration 0.921 ng/ml, nAA+AG = 146; P = 0.013). 1-naphthaleneacetic acid 182-185 glucocorticoid induced 1 Homo sapiens 9-15 30937140-9 2019 Results: GLCCI1 rs37973 homozygotes mutant genotype GG had a higher plasma epinephrine concentration (median concentration 27.032 pg/ml, nGG = 36; median concentration 23.149 pg/ml, nAA+AG = 146; P = 0.015) and cortisol concentration (median concentration 1.141 ng/ml, nGG = 36; median concentration 0.921 ng/ml, nAA+AG = 146; P = 0.013). 1-naphthaleneacetic acid 313-316 glucocorticoid induced 1 Homo sapiens 9-15 29967167-5 2018 Guided by these phenotypes, we identify N-oleoyl-glutamine (C18:1-Gln) as a key PM20D1-regulated NAA. 1-naphthaleneacetic acid 97-100 peptidase M20 domain containing 1 Mus musculus 80-86 29967167-8 2018 These data demonstrate that PM20D1 is a dominant enzymatic regulator of NAA levels in vivo and elucidate physiologic functions for NAA signaling in metabolism and nociception. 1-naphthaleneacetic acid 72-75 peptidase M20 domain containing 1 Mus musculus 28-34 29967167-8 2018 These data demonstrate that PM20D1 is a dominant enzymatic regulator of NAA levels in vivo and elucidate physiologic functions for NAA signaling in metabolism and nociception. 1-naphthaleneacetic acid 131-134 peptidase M20 domain containing 1 Mus musculus 28-34 29604522-12 2018 NAA treatment induced expression of CYCB1, GH3.6 and ARF8. 1-naphthaleneacetic acid 0-3 CYCLIN B1;1 Arabidopsis thaliana 36-41 29604522-12 2018 NAA treatment induced expression of CYCB1, GH3.6 and ARF8. 1-naphthaleneacetic acid 0-3 auxin response factor 8 Arabidopsis thaliana 53-57 29509406-3 2018 In this work, the technique is applied after each step of different surface modification paths of the NAA pores: (i) electrostatic immobilization of bovine serum albumin (BSA), (ii) covalent attachment of streptavidin via (3-aminipropyl)-triethoxysilane and glutaraldehyde grafting, and (iii) immune complexation. 1-naphthaleneacetic acid 102-105 albumin Homo sapiens 156-169 30260266-6 2018 Moreover, root growth of SAUR53 overexpression seedlings is significantly insensitive to IAA and 2,4-D, while showing wild-type sensitivity to NAA, suggesting that elevated level of SAUR53 may interfere with normal auxin transport. 1-naphthaleneacetic acid 143-146 SAUR-like auxin-responsive protein family Arabidopsis thaliana 25-31 30260266-6 2018 Moreover, root growth of SAUR53 overexpression seedlings is significantly insensitive to IAA and 2,4-D, while showing wild-type sensitivity to NAA, suggesting that elevated level of SAUR53 may interfere with normal auxin transport. 1-naphthaleneacetic acid 143-146 SAUR-like auxin-responsive protein family Arabidopsis thaliana 182-188 30834110-8 2019 NAA displayed higher levels of matrix metalloproteinase-1 (MMP-1) compared with both NAC (p = 0.011) and AA (p = 0.001), and lower PD20 compared with NAC (p < 0.001). 1-naphthaleneacetic acid 0-3 interstitial collagenase Bos taurus 31-57 30834110-8 2019 NAA displayed higher levels of matrix metalloproteinase-1 (MMP-1) compared with both NAC (p = 0.011) and AA (p = 0.001), and lower PD20 compared with NAC (p < 0.001). 1-naphthaleneacetic acid 0-3 interstitial collagenase Bos taurus 59-64 31208285-6 2019 Promoter motif analysis of four maize PIN1 genes and their expression levels in response to NAA, low phosphate levels and PEG treatment indicated that ZmPIN1a and ZmPIN1b may contribute more than ZmPIN1c and ZmPIN1d to root growth regulation and abiotic stress response. 1-naphthaleneacetic acid 92-95 probable auxin efflux carrier component 1c Zea mays 151-158 31208285-6 2019 Promoter motif analysis of four maize PIN1 genes and their expression levels in response to NAA, low phosphate levels and PEG treatment indicated that ZmPIN1a and ZmPIN1b may contribute more than ZmPIN1c and ZmPIN1d to root growth regulation and abiotic stress response. 1-naphthaleneacetic acid 92-95 auxin efflux carrier component 1a Zea mays 163-170 30120882-0 2018 Crystal structure of phospholipase A2 in complex with 1-naphthaleneacetic acid. 1-naphthaleneacetic acid 54-78 phospholipase A2 group IB Homo sapiens 21-37 30120882-5 2018 In this study, the inhibitory activity of 1-napthaleneacetic acid (NAA) against porcine pancreatic PLA2 has been explained through isothermal titration calorimetry and enzyme kinetics studies. 1-naphthaleneacetic acid 67-70 phospholipase A2 group IB Homo sapiens 99-103 30120882-6 2018 The atomic level of interactions of NAA with PLA2 was also studied using X-ray crystallography. 1-naphthaleneacetic acid 36-39 phospholipase A2 group IB Homo sapiens 45-49 30041521-2 2018 The surface of NAA-PCs is chemically functionalized with gamma-globulin (GG), transferrin (TFN), and serum albumin (HSA), the major proteins present in human blood plasma. 1-naphthaleneacetic acid 15-18 transferrin Homo sapiens 78-89 30041521-2 2018 The surface of NAA-PCs is chemically functionalized with gamma-globulin (GG), transferrin (TFN), and serum albumin (HSA), the major proteins present in human blood plasma. 1-naphthaleneacetic acid 15-18 transferrin Homo sapiens 91-94 30041521-2 2018 The surface of NAA-PCs is chemically functionalized with gamma-globulin (GG), transferrin (TFN), and serum albumin (HSA), the major proteins present in human blood plasma. 1-naphthaleneacetic acid 15-18 albumin Homo sapiens 116-119 29415436-6 2018 The sensing performance of the prepared NAA platforms was examined by real-time screening of binding reactions between human serum albumin (HSA)-modified NAA (i.e., sensing element) and quercetin (i.e., analyte). 1-naphthaleneacetic acid 40-43 albumin Homo sapiens 125-138 29415436-6 2018 The sensing performance of the prepared NAA platforms was examined by real-time screening of binding reactions between human serum albumin (HSA)-modified NAA (i.e., sensing element) and quercetin (i.e., analyte). 1-naphthaleneacetic acid 154-157 albumin Homo sapiens 125-138 28973586-9 2017 In Arabidopsis ABP1 bound to IAA or NAA, glycosylation structures arranged around the protein, covering the putative site of entrance or egress of auxin. 1-naphthaleneacetic acid 36-39 endoplasmic reticulum auxin binding protein 1 Arabidopsis thaliana 15-19 29203794-2 2017 Recently, it has been reported that SHATI is N-acetyltransferase 8-like protein (NAT8L) that produces N-acetylaspatate (NAA) from aspartate and acetyl-CoA. 1-naphthaleneacetic acid 120-123 N-acetyltransferase 8-like Mus musculus 36-41 29203794-2 2017 Recently, it has been reported that SHATI is N-acetyltransferase 8-like protein (NAT8L) that produces N-acetylaspatate (NAA) from aspartate and acetyl-CoA. 1-naphthaleneacetic acid 120-123 N-acetyltransferase 8-like Mus musculus 45-79 29203794-2 2017 Recently, it has been reported that SHATI is N-acetyltransferase 8-like protein (NAT8L) that produces N-acetylaspatate (NAA) from aspartate and acetyl-CoA. 1-naphthaleneacetic acid 120-123 N-acetyltransferase 8-like Mus musculus 81-86 29203794-3 2017 We have generated SHATI/NAT8L knockout (Shati -/-) mouse which demonstrates behavioral deficits that are not rescued by single NAA supplementation, although the reason for which is still not clarified. 1-naphthaleneacetic acid 127-130 N-acetyltransferase 8-like Mus musculus 18-23 29203794-4 2017 It is possible that the developmental impairment results from deletion of SHATI/NAT8L in the mouse brain, because NAA is involved in myelination through lipid synthesis in oligodendrocytes. 1-naphthaleneacetic acid 114-117 N-acetyltransferase 8-like Mus musculus 74-79 29203794-4 2017 It is possible that the developmental impairment results from deletion of SHATI/NAT8L in the mouse brain, because NAA is involved in myelination through lipid synthesis in oligodendrocytes. 1-naphthaleneacetic acid 114-117 N-acetyltransferase 8-like Mus musculus 80-85 29203794-8 2017 These findings suggest that SHATI/NAT8L is involved in myelination in the juvenile mouse brain via supplementation of acetate derived from NAA. 1-naphthaleneacetic acid 139-142 N-acetyltransferase 8-like Mus musculus 28-33 29203794-8 2017 These findings suggest that SHATI/NAT8L is involved in myelination in the juvenile mouse brain via supplementation of acetate derived from NAA. 1-naphthaleneacetic acid 139-142 N-acetyltransferase 8-like Mus musculus 34-39 28782920-6 2017 Application of NAA inhibited akt1 root growth, but promoted wild-type root growth under LK conditions. 1-naphthaleneacetic acid 15-18 K+ transporter 1 Arabidopsis thaliana 29-33 28660363-5 2017 At 27 C, the ckrc1-1 root length is significantly shortened and the root gravity defect is enhanced, changes that can be restored with addition of 1-naphthaleneacetic acid, but not indole-3-acetic acid (IAA). 1-naphthaleneacetic acid 148-172 tryptophan aminotransferase of Arabidopsis 1 Arabidopsis thaliana 14-21 26613958-1 2016 Canavan disease (CD) is a rare fatal childhood neurological autosomal recessive genetic disease caused by mutations in the ASPA gene, which lead to catalytic deficiency of the ASPA enzyme that catalyzes the deacetylation of NAA. 1-naphthaleneacetic acid 224-227 aspartoacylase Homo sapiens 123-127 28744293-10 2017 Consistent with the effect of TOR overexpression, the addition of the auxin NAA inhibited autophagy during nutrient deficiency, salt and osmotic stress, but not during oxidative or ER stress. 1-naphthaleneacetic acid 76-79 target of rapamycin Arabidopsis thaliana 30-33 27128744-1 2016 In this study, we report an innovative approach aiming to assess the binding affinity between drug molecules and human serum albumin by combining nanoporous anodic alumina rugate filters (NAA-RFs) modified with human serum albumin (HSA) and reflectometric interference spectroscopy (RIfS). 1-naphthaleneacetic acid 188-191 albumin Homo sapiens 119-132 27876241-6 2017 At the temperature of 110 C (pressure 1.43bar), NaA formed with a mixture of NaP1 and Faujasite. 1-naphthaleneacetic acid 48-51 nucleosome assembly protein 1 like 1 Homo sapiens 77-81 27375614-1 2016 We review how polyreactive natural IgM autoantibodies (IgM-NAA) protect the host from invading micro-organisms and host neo-antigens that are constantly being produced by oxidation mechanisms and cell apoptosis. 1-naphthaleneacetic acid 59-62 Ig heavy chain (V-D-J region) Mus musculus 35-38 27375614-1 2016 We review how polyreactive natural IgM autoantibodies (IgM-NAA) protect the host from invading micro-organisms and host neo-antigens that are constantly being produced by oxidation mechanisms and cell apoptosis. 1-naphthaleneacetic acid 59-62 Ig heavy chain (V-D-J region) Mus musculus 55-58 27375614-2 2016 Second, we discuss how IgM-NAA and IgM anti-leukocyte antibodies (IgM-ALA) inhibits autoimmune inflammation by anti-idiotypic mechanisms, enhancing removal of apoptotic cells, masking neo-antigens, and regulating the function of dendritic cells (DC) and effector cells. 1-naphthaleneacetic acid 27-30 Ig heavy chain (V-D-J region) Mus musculus 23-26 27375614-5 2016 It is, therefore, not surprising why the host positively selects such autoreactive B1 cells that generate IgM-NAA, which are also evolutionarily conserved. 1-naphthaleneacetic acid 110-113 Ig heavy chain (V-D-J region) Mus musculus 106-109 27375614-7 2016 We also show how protective IgM-NAA can be rendered pathogenic under non-physiological conditions. 1-naphthaleneacetic acid 32-35 Ig heavy chain (V-D-J region) Mus musculus 28-31 27375614-8 2016 We also review IgG-NAA that are more abundant than IgM-NAA in plasma. 1-naphthaleneacetic acid 55-58 Ig heavy chain (V-D-J region) Mus musculus 51-54 27375614-9 2016 However, we need to understand if the (Fab)(2) region of IgG-NAA has physiological relevance in non-disease states, as in plasma, their functional activity is blocked by IgM-NAA having anti-idiotypic activity. 1-naphthaleneacetic acid 61-64 FA complementation group B Homo sapiens 39-42 27375614-9 2016 However, we need to understand if the (Fab)(2) region of IgG-NAA has physiological relevance in non-disease states, as in plasma, their functional activity is blocked by IgM-NAA having anti-idiotypic activity. 1-naphthaleneacetic acid 61-64 Ig heavy chain (V-D-J region) Mus musculus 170-173 27375614-9 2016 However, we need to understand if the (Fab)(2) region of IgG-NAA has physiological relevance in non-disease states, as in plasma, their functional activity is blocked by IgM-NAA having anti-idiotypic activity. 1-naphthaleneacetic acid 174-177 FA complementation group B Homo sapiens 39-42 27375614-9 2016 However, we need to understand if the (Fab)(2) region of IgG-NAA has physiological relevance in non-disease states, as in plasma, their functional activity is blocked by IgM-NAA having anti-idiotypic activity. 1-naphthaleneacetic acid 174-177 Ig heavy chain (V-D-J region) Mus musculus 170-173 27375614-12 2016 IgM-NAA has therapeutic potential. 1-naphthaleneacetic acid 4-7 Ig heavy chain (V-D-J region) Mus musculus 0-3 26613958-1 2016 Canavan disease (CD) is a rare fatal childhood neurological autosomal recessive genetic disease caused by mutations in the ASPA gene, which lead to catalytic deficiency of the ASPA enzyme that catalyzes the deacetylation of NAA. 1-naphthaleneacetic acid 224-227 aspartoacylase Homo sapiens 176-180 25255415-11 2014 When accounting for the other variables, the association between NAA and RLS prevalence was statistically significant (P = 0.002). 1-naphthaleneacetic acid 65-68 RLS1 Homo sapiens 73-76 25903543-5 2015 Expression levels of TAR2, YUC1, YUC2, YUC4, and YUC6 were downregulated in response to synthetic auxins [1-naphthaleneacetic acid (NAA) and 2,4-dichlorophenoxyacetic acid (2,4-D)] exogenously applied to Arabidopsis thaliana L. seedlings. 1-naphthaleneacetic acid 106-130 tryptophan aminotransferase related 2 Arabidopsis thaliana 21-25 25903543-5 2015 Expression levels of TAR2, YUC1, YUC2, YUC4, and YUC6 were downregulated in response to synthetic auxins [1-naphthaleneacetic acid (NAA) and 2,4-dichlorophenoxyacetic acid (2,4-D)] exogenously applied to Arabidopsis thaliana L. seedlings. 1-naphthaleneacetic acid 106-130 Flavin-binding monooxygenase family protein Arabidopsis thaliana 27-31 25903543-5 2015 Expression levels of TAR2, YUC1, YUC2, YUC4, and YUC6 were downregulated in response to synthetic auxins [1-naphthaleneacetic acid (NAA) and 2,4-dichlorophenoxyacetic acid (2,4-D)] exogenously applied to Arabidopsis thaliana L. seedlings. 1-naphthaleneacetic acid 106-130 Flavin-binding monooxygenase family protein Arabidopsis thaliana 33-37 25903543-5 2015 Expression levels of TAR2, YUC1, YUC2, YUC4, and YUC6 were downregulated in response to synthetic auxins [1-naphthaleneacetic acid (NAA) and 2,4-dichlorophenoxyacetic acid (2,4-D)] exogenously applied to Arabidopsis thaliana L. seedlings. 1-naphthaleneacetic acid 106-130 Flavin-binding monooxygenase family protein Arabidopsis thaliana 39-43 25903543-5 2015 Expression levels of TAR2, YUC1, YUC2, YUC4, and YUC6 were downregulated in response to synthetic auxins [1-naphthaleneacetic acid (NAA) and 2,4-dichlorophenoxyacetic acid (2,4-D)] exogenously applied to Arabidopsis thaliana L. seedlings. 1-naphthaleneacetic acid 106-130 Flavin-binding monooxygenase family protein Arabidopsis thaliana 49-53 25903543-5 2015 Expression levels of TAR2, YUC1, YUC2, YUC4, and YUC6 were downregulated in response to synthetic auxins [1-naphthaleneacetic acid (NAA) and 2,4-dichlorophenoxyacetic acid (2,4-D)] exogenously applied to Arabidopsis thaliana L. seedlings. 1-naphthaleneacetic acid 132-135 tryptophan aminotransferase related 2 Arabidopsis thaliana 21-25 25903543-5 2015 Expression levels of TAR2, YUC1, YUC2, YUC4, and YUC6 were downregulated in response to synthetic auxins [1-naphthaleneacetic acid (NAA) and 2,4-dichlorophenoxyacetic acid (2,4-D)] exogenously applied to Arabidopsis thaliana L. seedlings. 1-naphthaleneacetic acid 132-135 Flavin-binding monooxygenase family protein Arabidopsis thaliana 27-31 25903543-5 2015 Expression levels of TAR2, YUC1, YUC2, YUC4, and YUC6 were downregulated in response to synthetic auxins [1-naphthaleneacetic acid (NAA) and 2,4-dichlorophenoxyacetic acid (2,4-D)] exogenously applied to Arabidopsis thaliana L. seedlings. 1-naphthaleneacetic acid 132-135 Flavin-binding monooxygenase family protein Arabidopsis thaliana 33-37 25903543-5 2015 Expression levels of TAR2, YUC1, YUC2, YUC4, and YUC6 were downregulated in response to synthetic auxins [1-naphthaleneacetic acid (NAA) and 2,4-dichlorophenoxyacetic acid (2,4-D)] exogenously applied to Arabidopsis thaliana L. seedlings. 1-naphthaleneacetic acid 132-135 Flavin-binding monooxygenase family protein Arabidopsis thaliana 39-43 25903543-5 2015 Expression levels of TAR2, YUC1, YUC2, YUC4, and YUC6 were downregulated in response to synthetic auxins [1-naphthaleneacetic acid (NAA) and 2,4-dichlorophenoxyacetic acid (2,4-D)] exogenously applied to Arabidopsis thaliana L. seedlings. 1-naphthaleneacetic acid 132-135 Flavin-binding monooxygenase family protein Arabidopsis thaliana 49-53 25849901-7 2015 The obtained results reveal that optimized NAA-DBR photonic coatings can achieve an outstanding sensing performance for gold(iii) ions, with a sensitivity of 22.16 nm muM(-1), a low limit of detection of 0.156 muM (i.e. 30.7 ppb) and excellent linearity within the working range (0.9983). 1-naphthaleneacetic acid 43-46 PWWP domain containing 3A, DNA repair factor Homo sapiens 167-173 25738325-2 2015 Application of a synthetic auxin, NAA, and an inhibitor of polar auxin transport, NPA, increased and decreased respectively the magnitude of the phototropic response in the wild type, while in axr2 application of NAA reduced the negative phototropic response and NPA had no effect. 1-naphthaleneacetic acid 34-37 indole-3-acetic acid 7 Arabidopsis thaliana 193-197 25738325-2 2015 Application of a synthetic auxin, NAA, and an inhibitor of polar auxin transport, NPA, increased and decreased respectively the magnitude of the phototropic response in the wild type, while in axr2 application of NAA reduced the negative phototropic response and NPA had no effect. 1-naphthaleneacetic acid 213-216 indole-3-acetic acid 7 Arabidopsis thaliana 193-197 24360094-10 2014 We also found significant positive correlations between NAA/Cr ratio in PWM and IGFBP3 level, as well as with GH concentration in a stimulation test with glucagon. 1-naphthaleneacetic acid 56-59 insulin like growth factor binding protein 3 Homo sapiens 80-86 24080131-13 2014 Western blots of culture media showed a fourfold increase of MMP-9 (92 kD) protein only in AAA-SMCs/U937 but not in NAA-SMCs/U937 (P < .001) and a large increase in active-MMP2 (62 kD), which was less apparent in NAA-SMCs/U937 media (P < .01). 1-naphthaleneacetic acid 216-219 matrix metallopeptidase 9 Homo sapiens 61-66 25043515-2 2014 Maize ABP1 was simulated with the natural auxin indole-3-acetic acid (IAA) and the synthetic analog naphthalen-1-acetic acid (NAA), to elucidate the role of the KDEL sequence and the helix at the C-terminus. 1-naphthaleneacetic acid 126-129 auxin-binding protein 1 Zea mays 6-10 25043515-10 2014 Our simulations confirm the asymmetric behavior of the two monomers, the stronger interaction of NAA than IAA and offers insight into the possible mechanism of ABP1 as an auxin receptor. 1-naphthaleneacetic acid 97-100 auxin-binding protein 1 Zea mays 160-164 24938853-4 2014 Application of 1-naphthaleneacetic acid-containing lanolin to the stem tips enhanced the positive phototropism of WT, and reduced the negative phototropism of axr2. 1-naphthaleneacetic acid 15-39 indole-3-acetic acid 7 Arabidopsis thaliana 159-163 25056921-6 2014 RD20 also confers tolerance against stress induced by Paraquat, Rose Bengal, heavy metal, and the synthetic auxins 1-naphthaleneacetic acid and 2,4-dichlorophenoxyacetic acid. 1-naphthaleneacetic acid 115-139 Caleosin-related family protein Arabidopsis thaliana 0-4 26518975-3 2016 The biological assay indicated that replacing N(10) with carbon would significantly increase inhibitory activities against DHFR and cytotoxicities against cancer cell lines. 1-naphthaleneacetic acid 46-51 dihydrofolate reductase Homo sapiens 123-127 26448619-9 2015 Ammonia (NH4(+)) increases the expression and activity of the L-arginine (Arg) transporter (Arg/neutral amino acids [NAA] exchanger) y(+)LAT2 in cultured rat cortical astrocytes by a mechanism involving activation (nuclear translocation) of the transcription factor nuclear factor-Nuclear factor-kappaB (Nf-kappaB-p65). 1-naphthaleneacetic acid 117-120 linker for activation of T cells family, member 2 Rattus norvegicus 137-141 26151906-1 2015 Glutamate carboxypeptidase II (GCP-II) is a brain metallopeptidase that hydrolyzes the abundant neuropeptide N-acetyl-aspartyl-glutamate (NAAG) to NAA and glutamate. 1-naphthaleneacetic acid 138-141 folate hydrolase 1 Homo sapiens 0-29 26151906-1 2015 Glutamate carboxypeptidase II (GCP-II) is a brain metallopeptidase that hydrolyzes the abundant neuropeptide N-acetyl-aspartyl-glutamate (NAAG) to NAA and glutamate. 1-naphthaleneacetic acid 138-141 folate hydrolase 1 Homo sapiens 31-37 26572024-6 2015 NAA distinctly enhanced the activities of APX, POD, CAT, MDHAR, GPX, and ratios of AsA/DHA and GSH/GSSG, while decreased the levels of ROS and MDA. 1-naphthaleneacetic acid 0-3 peroxidase Glycine max 47-50 26572024-6 2015 NAA distinctly enhanced the activities of APX, POD, CAT, MDHAR, GPX, and ratios of AsA/DHA and GSH/GSSG, while decreased the levels of ROS and MDA. 1-naphthaleneacetic acid 0-3 catalase-3 Glycine max 52-55 26572024-6 2015 NAA distinctly enhanced the activities of APX, POD, CAT, MDHAR, GPX, and ratios of AsA/DHA and GSH/GSSG, while decreased the levels of ROS and MDA. 1-naphthaleneacetic acid 0-3 monodehydroascorbate reductase Glycine max 57-62 26572024-6 2015 NAA distinctly enhanced the activities of APX, POD, CAT, MDHAR, GPX, and ratios of AsA/DHA and GSH/GSSG, while decreased the levels of ROS and MDA. 1-naphthaleneacetic acid 0-3 probable phospholipid hydroperoxide glutathione peroxidase Glycine max 64-67 26402110-0 2015 A Critical Proton MR Spectroscopy Marker of Alzheimer"s Disease Early Neurodegenerative Change: Low Hippocampal NAA/Cr Ratio Impacts APOE e4 Mexico City Children and Their Parents. 1-naphthaleneacetic acid 112-115 apolipoprotein E Homo sapiens 133-137 26402110-6 2015 The NAA/Cr ratio in right hippocampus in controls versus APOE e4 MC children and in left hippocampus in MC APOE e4 parents versus their children was significantly different after adjusting for age, gender, and BMI (p = 0.027 and 0.01, respectively). 1-naphthaleneacetic acid 4-7 apolipoprotein E Homo sapiens 57-61 26402110-8 2015 Decreases in NAA/Cr correlate well with cognitive function, behavioral symptoms, and dementia severity; thus, since the progression of AD starts decades before clinical diagnosis, our findings support the hypothesis that under chronic exposures to fine particulate matter and ozone above the standards, neurodegenerative processes start in childhood and APOE e4 carriers are at higher risk. 1-naphthaleneacetic acid 13-16 apolipoprotein E Homo sapiens 354-358 26331041-5 2014 RESULTS: Significant reductions of NAA/Cr and NAA/Cho in the cerebellar hemispheres in all patients and lower Cho/Cr in the cerebellar hemispheres in patients with SCA2 or MSA-C were found at all times. 1-naphthaleneacetic acid 35-38 ataxin 2 Homo sapiens 164-168 26331041-6 2014 At initial assessments, patients with MSA-C or SCA2 tended to have lower NAA/Cr and Cho/Cr in the cerebellar hemispheres than those with SCA3 or SCA6. 1-naphthaleneacetic acid 73-76 ataxin 2 Homo sapiens 47-51 26331041-7 2014 At follow-ups, patients with SCA2 or MSA-C had a lower NAA/Cr in cerebellar hemispheres than those with SCA3 or SCA6. 1-naphthaleneacetic acid 55-58 ataxin 2 Homo sapiens 29-33 25046756-0 2014 Effect of 1-naphthaleneacetic acid on organic acid exudation by the roots of white lupin plants grown under phosphorus-deficient conditions. 1-naphthaleneacetic acid 10-34 5'-nucleotidase, cytosolic IIIA Homo sapiens 83-88 25046756-1 2014 The effect of NAA (1-naphthaleneacetic acid) on organic acid exudation in white lupin plants grown under phosphorus deficiency was investigated. 1-naphthaleneacetic acid 14-17 5'-nucleotidase, cytosolic IIIA Homo sapiens 80-85 25046756-1 2014 The effect of NAA (1-naphthaleneacetic acid) on organic acid exudation in white lupin plants grown under phosphorus deficiency was investigated. 1-naphthaleneacetic acid 19-43 5'-nucleotidase, cytosolic IIIA Homo sapiens 80-85 25046756-4 2014 The exogenous addition of NAA led to an increase in organic acid exudation, but this response was not proportional to the concentration of the dose applied, noticing the largest increments with NAA 10(-8)M. In contrast the increase in root weight was proportional to the dose applied, which shows that with higher doses the roots produced are not of proteoid type. 1-naphthaleneacetic acid 26-29 N-alpha-acetyltransferase 10, NatA catalytic subunit Homo sapiens 194-200 25255415-14 2014 We found the association between NAA race and RLS prevalence to be statistically significant. 1-naphthaleneacetic acid 33-36 RLS1 Homo sapiens 46-49 24887377-4 2014 In the present study, we incorporated an alkyne-bearing NAA into an enzyme, murine dihydrofolate reductase (mDHFR), in high cell density cultivation of Escherichia coli, and performed CuAAC conjugation with fluorescent azide dyes to evaluate enzyme compatibility of various CuAAC conditions comprising combination of commercially available Cu(I)-chelating ligands and reductants. 1-naphthaleneacetic acid 56-59 dihydrofolate reductase Mus musculus 108-113 24366686-3 2014 Herein, we revealed that exogenous auxin, 1-naphthylacetic acid (NAA), could simultaneously stimulate Arabidopsis HO-1 (HY1) gene expression and H2O2 generation. 1-naphthaleneacetic acid 42-63 Plant heme oxygenase (decyclizing) family protein Arabidopsis thaliana 120-123 24800738-2 2014 1-Naphtoxyacetic acid (1-NOA), an analog of the synthetic auxin 1-N-naphtalene acetic acid (NAA), inhibits the IAA influx carrier AUX1. 1-naphthaleneacetic acid 92-95 Transmembrane amino acid transporter family protein Arabidopsis thaliana 130-134 24366686-3 2014 Herein, we revealed that exogenous auxin, 1-naphthylacetic acid (NAA), could simultaneously stimulate Arabidopsis HO-1 (HY1) gene expression and H2O2 generation. 1-naphthaleneacetic acid 65-68 Plant heme oxygenase (decyclizing) family protein Arabidopsis thaliana 120-123 24366686-5 2014 NAA-induced HY1 expression is dependent on H2O2. 1-naphthaleneacetic acid 0-3 Plant heme oxygenase (decyclizing) family protein Arabidopsis thaliana 12-15 24366686-6 2014 This conclusion was supported by analyzing the removal of H2O2 with ascorbic acid (AsA) and dimethylthiourea (DMTU), both of which could block NAA-induced HY1 expression and LR formation. 1-naphthaleneacetic acid 143-146 Plant heme oxygenase (decyclizing) family protein Arabidopsis thaliana 155-158 24366686-8 2014 Simultaneously, HY1 is required for NAA-mediated H2O2 generation, since Znpp inhibition of HY1 blocked the NAA-induced H2O2 production and LR formation. 1-naphthaleneacetic acid 36-39 Plant heme oxygenase (decyclizing) family protein Arabidopsis thaliana 16-19 24366686-8 2014 Simultaneously, HY1 is required for NAA-mediated H2O2 generation, since Znpp inhibition of HY1 blocked the NAA-induced H2O2 production and LR formation. 1-naphthaleneacetic acid 107-110 Plant heme oxygenase (decyclizing) family protein Arabidopsis thaliana 16-19 24366686-8 2014 Simultaneously, HY1 is required for NAA-mediated H2O2 generation, since Znpp inhibition of HY1 blocked the NAA-induced H2O2 production and LR formation. 1-naphthaleneacetic acid 107-110 Plant heme oxygenase (decyclizing) family protein Arabidopsis thaliana 91-94 24366686-9 2014 Genetic data demonstrated that hy1-100 was significantly impaired in H2O2 production and LR formation in response to NAA, compared with wild-type plants. 1-naphthaleneacetic acid 117-120 Plant heme oxygenase (decyclizing) family protein Arabidopsis thaliana 31-34 24306962-1 2014 BACKGROUND: Whether Native American ancestry (NAA) is associated with COPD or lung function in a racially admixed Hispanic population is unknown. 1-naphthaleneacetic acid 46-49 COPD Homo sapiens 70-74 24306962-9 2014 After adjustment for current smoking and other covariates, NAA was inversely associated with COPD (OR per 10% increment, 0.55; 95% CI, 0.41-0.75) but positively associated with FEV1, FVC, and FEV1/FVC. 1-naphthaleneacetic acid 59-62 COPD Homo sapiens 93-97 24306962-10 2014 After additional adjustment for pack-years of smoking, the association between NAA and COPD or lung function measures was slightly attenuated. 1-naphthaleneacetic acid 79-82 COPD Homo sapiens 87-91 24306962-11 2014 We found that about 31% of the estimated effect of NAA on COPD is mediated by pack-years of smoking. 1-naphthaleneacetic acid 51-54 COPD Homo sapiens 58-62 23289750-9 2013 Similar to previous findings in an Arabidopsis thaliana SGT1 mutant (atsgt1b), degradation of AUX/IAA proteins was retarded in cyp2 mutants treated with exogenous 1-naphthylacetic acid. 1-naphthaleneacetic acid 163-184 UDP-glucosyltransferase 74F2 Arabidopsis thaliana 56-60 24306962-12 2014 CONCLUSIONS: NAA is inversely associated with COPD but positively associated with FEV1 or FVC in Costa Ricans. 1-naphthaleneacetic acid 13-16 COPD Homo sapiens 46-50 24625793-6 2014 AbetaPP-PS1 mice exhibit a significant reduction in the level of NAA and increase in level of myo-inositol. 1-naphthaleneacetic acid 65-68 presenilin 1 Mus musculus 8-11 23562458-3 2013 However, the catalytic enzyme glutamate carboxypeptidase II (GCP II) rapidly hydrolyzes NAAG into NAA and glutamate. 1-naphthaleneacetic acid 88-91 folate hydrolase 1 Rattus norvegicus 30-59 23562458-3 2013 However, the catalytic enzyme glutamate carboxypeptidase II (GCP II) rapidly hydrolyzes NAAG into NAA and glutamate. 1-naphthaleneacetic acid 88-91 folate hydrolase 1 Rattus norvegicus 61-67 23562458-4 2013 Inhibition of the GCP II enzyme with NAAG peptidase inhibitors reduces the concentration of glutamate both by increasing the duration of NAAG activity on mGluR3 and by reducing degradation into NAA and glutamate resulting in reduced cell death in models of TBI and TBI with hypoxia. 1-naphthaleneacetic acid 37-40 folate hydrolase 1 Rattus norvegicus 18-24 23562458-4 2013 Inhibition of the GCP II enzyme with NAAG peptidase inhibitors reduces the concentration of glutamate both by increasing the duration of NAAG activity on mGluR3 and by reducing degradation into NAA and glutamate resulting in reduced cell death in models of TBI and TBI with hypoxia. 1-naphthaleneacetic acid 37-40 glutamate receptor, ionotropic, AMPA3 (alpha 3) Mus musculus 154-160 24164597-7 2014 Moreover, the rel3 mutant was more resistant than wild-type to 1-naphthaleneacetic acid (NAA) and N-1-naphthylphthalamic acid (NPA) in root growth, and exhibited insensitivity to auxins but greater sensitivity to auxin transport inhibitors during nodulation. 1-naphthaleneacetic acid 63-87 REL3 Lotus japonicus 14-18 24164597-7 2014 Moreover, the rel3 mutant was more resistant than wild-type to 1-naphthaleneacetic acid (NAA) and N-1-naphthylphthalamic acid (NPA) in root growth, and exhibited insensitivity to auxins but greater sensitivity to auxin transport inhibitors during nodulation. 1-naphthaleneacetic acid 89-92 REL3 Lotus japonicus 14-18 23503757-5 2013 Stress experiments showed that the AtRDR1 promoter has a broad-spectrum response to various stresses and is sensitive to 1-naphthaleneacetic acid, abscisic acid, and salicylic acid. 1-naphthaleneacetic acid 121-145 RNA-dependent RNA polymerase 1 Arabidopsis thaliana 35-41 23293348-6 2013 Following treatment with the synthetic auxin 1-naphthaleneacetic acid (NAA), DR5:GUS expression in aux1(rcr1) and aux1-T occurred mainly in the root apex and mature zone. 1-naphthaleneacetic acid 45-69 Transmembrane amino acid transporter family protein Arabidopsis thaliana 99-103 23293348-6 2013 Following treatment with the synthetic auxin 1-naphthaleneacetic acid (NAA), DR5:GUS expression in aux1(rcr1) and aux1-T occurred mainly in the root apex and mature zone. 1-naphthaleneacetic acid 45-69 Transmembrane amino acid transporter family protein Arabidopsis thaliana 114-118 23293348-6 2013 Following treatment with the synthetic auxin 1-naphthaleneacetic acid (NAA), DR5:GUS expression in aux1(rcr1) and aux1-T occurred mainly in the root apex and mature zone. 1-naphthaleneacetic acid 71-74 Transmembrane amino acid transporter family protein Arabidopsis thaliana 99-103 23293348-7 2013 NAA-induced DR5:GUS expression in the root apex was markedly prevented by ethylene in genotypes with aux1(rcr1) but not in aux1-T genotypes and the wild type. 1-naphthaleneacetic acid 0-3 Transmembrane amino acid transporter family protein Arabidopsis thaliana 101-105 23293348-7 2013 NAA-induced DR5:GUS expression in the root apex was markedly prevented by ethylene in genotypes with aux1(rcr1) but not in aux1-T genotypes and the wild type. 1-naphthaleneacetic acid 0-3 Transmembrane amino acid transporter family protein Arabidopsis thaliana 123-127 23622383-11 2013 An increased concentration of NAA in the urine is sufficient to diagnose Canavan disease, which is due to mutations of the ASPA gene. 1-naphthaleneacetic acid 30-33 aspartoacylase Homo sapiens 123-127 23250625-4 2013 Here, we report the identification and characterization of an Arabidopsis (Arabidopsis thaliana) leaf senescence-associated gene named SAG201, which is highly up-regulated during leaf senescence and can be induced by 1-naphthaleneacetic acid, a synthetic auxin. 1-naphthaleneacetic acid 217-241 SAUR-like auxin-responsive protein family Arabidopsis thaliana 135-141 22722629-5 2012 RESULTS: Compared to controls, NAA was significantly reduced in the putamen (p = 0.012) and in the midbrain of GBA-PD (p = 0.05). 1-naphthaleneacetic acid 31-34 glucosylceramidase beta Homo sapiens 111-114 22750589-4 2012 However, extracellular NAAG is rapidly converted into NAA and glutamate by the catalytic enzyme glutamate carboxypeptidase II (GCPII) reducing presynaptic inhibition. 1-naphthaleneacetic acid 23-26 folate hydrolase 1 Homo sapiens 96-125 22750589-4 2012 However, extracellular NAAG is rapidly converted into NAA and glutamate by the catalytic enzyme glutamate carboxypeptidase II (GCPII) reducing presynaptic inhibition. 1-naphthaleneacetic acid 23-26 folate hydrolase 1 Homo sapiens 127-132 22849963-7 2012 IP-10, MCP-4, and MIP-1beta were significantly associated with CMRs in the right basal ganglia with (1) lower concentrations of IP-10 correlating with higher N-acetyl aspartate to creatine ratios (NAA/Cr) and (2) higher concentrations of MCP-4 and MIP-1beta correlating with higher myoinositol to creatine (mI/Cr) ratios. 1-naphthaleneacetic acid 197-200 C-X-C motif chemokine ligand 10 Homo sapiens 128-133 22540348-10 2012 Activation of the auxin-regulated synthetic DR5 promoter and of auxin response genes was strongly repressed in seedlings overexpressing PIN8 when exposed to 1-naphthalene acetic acid. 1-naphthaleneacetic acid 157-182 Auxin efflux carrier family protein Arabidopsis thaliana 136-140 22304711-8 2012 GCPII (glutamate carboxypeptidase II) cleaves the prevalent neuropeptide NAAG into NAA and glutamate and there is widespread evidence and belief that targeting the glutamate derived from this enzymatic action may be a promising therapeutic route. 1-naphthaleneacetic acid 73-76 folate hydrolase 1 Homo sapiens 0-5 22381655-6 2012 Real-time RT-PCR analysis revealed that TaEXPB23 transcript expression was upregulated by exogenous methyl jasmonate (MeJA) and salt stress, but downregulated by exogenous gibberellins (GA3), ethylene (ET), indole-3-acetic acid (IAA) and alpha-naphthlcetic acid (NAA). 1-naphthaleneacetic acid 263-266 EXPB23 Triticum aestivum 40-48 22407922-5 2012 We found that the expression of NAA in MRL-lpr mice prevented proteinuria and reduced kidney immune complex formation. 1-naphthaleneacetic acid 32-35 Fas (TNF receptor superfamily member 6) Mus musculus 43-46 22407922-7 2012 Administration of the IgM NAA to MRL-lpr mice also delayed the onset of nephritis. 1-naphthaleneacetic acid 26-29 Fas (TNF receptor superfamily member 6) Mus musculus 37-40 22407922-11 2012 Furthermore, the NAA B cells produced large amounts of IL-10 upon TLR stimulation. 1-naphthaleneacetic acid 17-20 interleukin 10 Mus musculus 55-60 21679158-5 2012 Semi-quantitative RT-PCR (reverse transcription-PCR) showed that GhRDR6 was up-regulated by the application of various phytohormones, including MeJA [methyl JA (jasmonate)], ABA (abscisic acid), JA, alpha-naphthylacetic acid, gibberellins and ET (ethylene). 1-naphthaleneacetic acid 199-224 RNA-dependent RNA polymerase 6-like Gossypium hirsutum 65-71 22284711-4 2012 We further discovered that N-1-naphthylacetic acid (NAA), haemin, and CO aqueous solution, led to the induction of ZmHO-1 expression as well as the thereafter promotion of lateral root development. 1-naphthaleneacetic acid 52-55 HO-1 Zea mays 115-121 22304711-8 2012 GCPII (glutamate carboxypeptidase II) cleaves the prevalent neuropeptide NAAG into NAA and glutamate and there is widespread evidence and belief that targeting the glutamate derived from this enzymatic action may be a promising therapeutic route. 1-naphthaleneacetic acid 73-76 folate hydrolase 1 Homo sapiens 7-36 23118909-8 2012 The NAA/Cr and Cho/Cr were lower in MSA-C or SCA2 comparing with SCA3 or SCA6. 1-naphthaleneacetic acid 4-7 ataxin 2 Homo sapiens 45-49 23118909-10 2012 The cerebellar NAA/Cho was lower in SCA2 than SCA1, SCA3 or SCA6 (p<0.04). 1-naphthaleneacetic acid 15-18 ataxin 2 Homo sapiens 36-40 23118909-10 2012 The cerebellar NAA/Cho was lower in SCA2 than SCA1, SCA3 or SCA6 (p<0.04). 1-naphthaleneacetic acid 15-18 ataxin 1 Homo sapiens 46-50 23118909-12 2012 In the early stages of diseases (SARA score<10), significant lower NAA/Cr and NAA/Cho in SCA2, SCA3, SCA6 or MSA-C were observed comparing with healthy controls (p<0.017). 1-naphthaleneacetic acid 67-70 ataxin 2 Homo sapiens 89-93 23118909-12 2012 In the early stages of diseases (SARA score<10), significant lower NAA/Cr and NAA/Cho in SCA2, SCA3, SCA6 or MSA-C were observed comparing with healthy controls (p<0.017). 1-naphthaleneacetic acid 78-81 ataxin 2 Homo sapiens 89-93 23118909-14 2012 Patients with MSA-C and SCA2 had lower NAA/Cr and Cho/Cr than SCA3 or SCA6 (p<0.016). 1-naphthaleneacetic acid 39-42 ataxin 2 Homo sapiens 24-28 21459102-6 2011 Adult visceral obesity and insulin level correlated with right>left shift in hippocampal NAA concentrations, controlling for age and denominator. 1-naphthaleneacetic acid 92-95 insulin Cricetulus griseus 27-34 21477123-9 2011 Both elevated CO(2) and NAA application increased expressions of caprice, triptychon and rho-related protein from plants 2, and decreased expressions of werewolf, GLABRA2, GLABRA3 and the transparent testa glabra 1, genes related to root-hair development, while 1-NOA and NPA application had an opposite effect. 1-naphthaleneacetic acid 24-27 HD-ZIP IV family of homeobox-leucine zipper protein with lipid-binding START domain-containing protein Arabidopsis thaliana 163-170 21477123-9 2011 Both elevated CO(2) and NAA application increased expressions of caprice, triptychon and rho-related protein from plants 2, and decreased expressions of werewolf, GLABRA2, GLABRA3 and the transparent testa glabra 1, genes related to root-hair development, while 1-NOA and NPA application had an opposite effect. 1-naphthaleneacetic acid 24-27 basic helix-loop-helix (bHLH) DNA-binding superfamily protein Arabidopsis thaliana 172-179 21477123-9 2011 Both elevated CO(2) and NAA application increased expressions of caprice, triptychon and rho-related protein from plants 2, and decreased expressions of werewolf, GLABRA2, GLABRA3 and the transparent testa glabra 1, genes related to root-hair development, while 1-NOA and NPA application had an opposite effect. 1-naphthaleneacetic acid 24-27 myb domain protein 0 Arabidopsis thaliana 206-214 21502189-7 2011 Quantification of auxin export from mesophyll protoplasts revealed that naphthalene-1-acetic acid but not indole-3-acetic acid transport is affected by the rol1-2 mutation. 1-naphthaleneacetic acid 72-97 rhamnose biosynthesis 1 Arabidopsis thaliana 156-162 21372002-2 2011 In the current study, the immune characteristics of MPO-NAA and MPO-ANCA were examined and compared with the aim to investigate the pathogenesis of MPO-ANCA. 1-naphthaleneacetic acid 56-59 myeloperoxidase Homo sapiens 52-55 21372002-7 2011 The MPO-NAA-induced production of reactive oxygen species was assessed using oxidation of dihydrorhodamine (DHR) to rhodamine. 1-naphthaleneacetic acid 8-11 myeloperoxidase Homo sapiens 4-7 21372002-8 2011 RESULTS: MPO-NAA recognized epitope(s) in the heavy chains of MPO with conformation-dependent structure, the same as MPO-ANCA. 1-naphthaleneacetic acid 13-16 myeloperoxidase Homo sapiens 9-12 21372002-8 2011 RESULTS: MPO-NAA recognized epitope(s) in the heavy chains of MPO with conformation-dependent structure, the same as MPO-ANCA. 1-naphthaleneacetic acid 13-16 myeloperoxidase Homo sapiens 62-65 21372002-8 2011 RESULTS: MPO-NAA recognized epitope(s) in the heavy chains of MPO with conformation-dependent structure, the same as MPO-ANCA. 1-naphthaleneacetic acid 13-16 myeloperoxidase Homo sapiens 62-65 21372002-9 2011 The median titre of MPO-NAA was lower than that of MPO-ANCA (1 : 40 vs 1 : 4800, P < 0.001). 1-naphthaleneacetic acid 24-27 myeloperoxidase Homo sapiens 20-23 21372002-10 2011 The median avidity of MPO-NAA was lower than that of MPO-ANCA (2.2 x 10(7) vs 8.7 x 10(7)/M, P = 0.014). 1-naphthaleneacetic acid 26-29 myeloperoxidase Homo sapiens 22-25 21372002-11 2011 The IgG subclasses of MPO-NAA were mainly restricted to IgG1 (100%) and lack of IgG3. 1-naphthaleneacetic acid 26-29 myeloperoxidase Homo sapiens 22-25 21372002-11 2011 The IgG subclasses of MPO-NAA were mainly restricted to IgG1 (100%) and lack of IgG3. 1-naphthaleneacetic acid 26-29 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 80-84 21372002-12 2011 The inhibition effect on the binding between ceruloplasmin and MPO was lower for MPO-NAA than MPO-ANCA (P = 0.046). 1-naphthaleneacetic acid 85-88 myeloperoxidase Homo sapiens 63-66 21372002-12 2011 The inhibition effect on the binding between ceruloplasmin and MPO was lower for MPO-NAA than MPO-ANCA (P = 0.046). 1-naphthaleneacetic acid 85-88 myeloperoxidase Homo sapiens 81-84 21372002-12 2011 The inhibition effect on the binding between ceruloplasmin and MPO was lower for MPO-NAA than MPO-ANCA (P = 0.046). 1-naphthaleneacetic acid 85-88 myeloperoxidase Homo sapiens 81-84 21372002-13 2011 The MPO-NAA-induced respiratory burst of neutrophils was significantly weaker than that of MPO-ANCA (P = 0.036). 1-naphthaleneacetic acid 8-11 myeloperoxidase Homo sapiens 4-7 21372002-14 2011 CONCLUSION: The lower titre, lower avidity and lack of IgG3 subclass compared with MPO-ANCA may contribute to the non-pathogenic co-existence of MPO-NAA with MPO in serum. 1-naphthaleneacetic acid 149-152 myeloperoxidase Homo sapiens 145-148 21372002-14 2011 CONCLUSION: The lower titre, lower avidity and lack of IgG3 subclass compared with MPO-ANCA may contribute to the non-pathogenic co-existence of MPO-NAA with MPO in serum. 1-naphthaleneacetic acid 149-152 myeloperoxidase Homo sapiens 145-148 21502189-8 2011 Inhibition of flavonol biosynthesis in rol1-2 fls1-3 restores naphthalene-1-acetic acid transport to wild-type levels, indicating a very specific mode of action of flavonols on the auxin transport machinery. 1-naphthaleneacetic acid 62-87 rhamnose biosynthesis 1 Arabidopsis thaliana 39-45 21502189-8 2011 Inhibition of flavonol biosynthesis in rol1-2 fls1-3 restores naphthalene-1-acetic acid transport to wild-type levels, indicating a very specific mode of action of flavonols on the auxin transport machinery. 1-naphthaleneacetic acid 62-87 flavonol synthase 1 Arabidopsis thaliana 46-50 21412048-3 2011 Although the conditional FAB1A/B knockdown mutant revealed an auxin-resistant phenotype to a membrane impermeable auxin, 2,4-dichlorophenoxyacetic acid (2,4-D), the mutant did not exhibit this phenotype to a membrane-permeable artificial auxin, naphthalene 1-acetic acid (NAA). 1-naphthaleneacetic acid 245-270 1-phosphatidylinositol-3-phosphate 5-kinase FAB1A Arabidopsis thaliana 25-32 21673093-4 2011 Furthermore, the application of lower doses of NaCl (10 mM) and polyethylene glycol (PEG) (2%) mimicked the inducible effects of naphthylacetic acid and the HO-1 inducer haemin on the up-regulation of BnHO1 and subsequent lateral root (LR) formation. 1-naphthaleneacetic acid 129-148 heme oxygenase 1, chloroplastic-like Brassica napus 201-206 21494695-8 2011 There was an inverse correlation between brain NAA/Cr (neuronal injury) and circulating CD14+CD16+ and CD14(lo)CD16+ monocytes. 1-naphthaleneacetic acid 47-50 CD14 molecule Homo sapiens 88-92 21494695-8 2011 There was an inverse correlation between brain NAA/Cr (neuronal injury) and circulating CD14+CD16+ and CD14(lo)CD16+ monocytes. 1-naphthaleneacetic acid 47-50 Fc gamma receptor IIIa Homo sapiens 93-97 21494695-8 2011 There was an inverse correlation between brain NAA/Cr (neuronal injury) and circulating CD14+CD16+ and CD14(lo)CD16+ monocytes. 1-naphthaleneacetic acid 47-50 CD14 molecule Homo sapiens 103-107 21494695-8 2011 There was an inverse correlation between brain NAA/Cr (neuronal injury) and circulating CD14+CD16+ and CD14(lo)CD16+ monocytes. 1-naphthaleneacetic acid 47-50 Fc gamma receptor IIIa Homo sapiens 111-115 21412048-3 2011 Although the conditional FAB1A/B knockdown mutant revealed an auxin-resistant phenotype to a membrane impermeable auxin, 2,4-dichlorophenoxyacetic acid (2,4-D), the mutant did not exhibit this phenotype to a membrane-permeable artificial auxin, naphthalene 1-acetic acid (NAA). 1-naphthaleneacetic acid 272-275 1-phosphatidylinositol-3-phosphate 5-kinase FAB1A Arabidopsis thaliana 25-32 20709516-6 2010 NAA and AA patients showed higher IFN-gamma and EDN levels compared to AC and NC, with no differences in IL-4, IL-5, or IL-13 levels among the four groups. 1-naphthaleneacetic acid 0-3 interferon gamma Homo sapiens 34-43 21224416-4 2011 The highest fidelity NAA systems derived from the Methanocaldococcus jannaschii tyrosyl AARS feature specific mutations to two residues reported to interact with the hydroxyl group of the substrate tyrosine. 1-naphthaleneacetic acid 21-24 alanyl-tRNA synthetase 1 Homo sapiens 88-92 20709516-6 2010 NAA and AA patients showed higher IFN-gamma and EDN levels compared to AC and NC, with no differences in IL-4, IL-5, or IL-13 levels among the four groups. 1-naphthaleneacetic acid 0-3 ribonuclease A family member 2 Homo sapiens 48-51 20709516-8 2010 In NAA, AA, and AC patients, % eosinophils and EDN levels correlated positively with IFN-gamma, GM-CSF, eotaxin, and RANTES, but not with IL-5 levels. 1-naphthaleneacetic acid 3-6 ribonuclease A family member 2 Homo sapiens 47-50 19442440-3 2009 Owing to the specific fiber-like morphology and chemical composition, zeolite-like calcosilicate CAS-1 can effectively intercept the particles and thus reduces the TSNA level of mainstream smoke in the range of 30-60% once it is added into the cigarette filter, which is superior to zeolite NaA additive with the same amount. 1-naphthaleneacetic acid 291-294 cycloartenol Synthase-like Nicotiana tabacum 97-102 19217667-6 2009 Anti-MUC1 IgM was found in 5/47 (10.6%) of normals, 5/45 (11.1%) of NAA, 7/47 (14.9%) of AA, and 4/20 (20.0%) of CRC (p=0.70). 1-naphthaleneacetic acid 68-71 mucin 1, cell surface associated Homo sapiens 5-9 19217667-7 2009 The prevalence of anti-MUC1 IgG was 8/47 (17.0%) of normals, 14/45 (31.1%) of NAA, 14/47 (29.8%) of AA, and 6/20 (30.0%) of CRC (p=0.36). 1-naphthaleneacetic acid 78-81 mucin 1, cell surface associated Homo sapiens 23-27 19217667-9 2009 However, in an exploratory analysis, the median normalized anti-MUC1 IgG OD level of the combined abnormal groups (NAA, AA, CRC) was significantly higher than the normals (0.045 OD vs. 0.030 OD; p=0.017). 1-naphthaleneacetic acid 115-118 mucin 1, cell surface associated Homo sapiens 64-68 19061772-12 2008 In the 23 responders, the rCBF after challenge were greater than 20 mL/min per 100 g (P=.008), had a significantly better CRC in the anterior CSWM than the nonresponders (1.40 vs 1.06), and had normal NAA/Cre ratio in the anterior, middle, and posterior CSWM in MRSI study. 1-naphthaleneacetic acid 201-204 CCAAT/enhancer binding protein zeta Rattus norvegicus 26-30 19655147-6 2009 Furthermore, the NAA-response region was found to be located in the -1,482/-1204 fragment, while the element(s) for the sucrose-responsive expression may be present in the -247/-1 region in the GhCesA4 promoter. 1-naphthaleneacetic acid 17-20 cellulose synthase A catalytic subunit 8 [UDP-forming] Gossypium hirsutum 194-201 19236511-6 2009 Cyclin D1, p21 and cyclin A were overexpressed in 58.6%, 51.7% and 31.8% of the NAA cases, respectively, whereas 81.3%, 62.5% and 41.7% of the AAA cases showed overexpression of cyclin D1, p21 and cyclin A, respectively. 1-naphthaleneacetic acid 80-83 cyclin D1 Homo sapiens 0-9 19236511-6 2009 Cyclin D1, p21 and cyclin A were overexpressed in 58.6%, 51.7% and 31.8% of the NAA cases, respectively, whereas 81.3%, 62.5% and 41.7% of the AAA cases showed overexpression of cyclin D1, p21 and cyclin A, respectively. 1-naphthaleneacetic acid 80-83 cyclin dependent kinase inhibitor 1A Homo sapiens 11-14 19236511-6 2009 Cyclin D1, p21 and cyclin A were overexpressed in 58.6%, 51.7% and 31.8% of the NAA cases, respectively, whereas 81.3%, 62.5% and 41.7% of the AAA cases showed overexpression of cyclin D1, p21 and cyclin A, respectively. 1-naphthaleneacetic acid 80-83 cyclin A2 Homo sapiens 19-27 17337532-7 2007 Addition of the polarly transported auxins indole-3-acetic acid and 1-naphthyl acetic acid (but not 2,4-dichlorophenoxyacetic acid) restored both the normal organization of actin and the synchrony of cell division. 1-naphthaleneacetic acid 68-90 actin Nicotiana tabacum 173-178 24256959-4 2008 While single voxel proton spectroscopy (MRS) of the basal ganglia at the beginning was normal, the follow-up MRS showed moderate to severe NAA and mI decrease. 1-naphthaleneacetic acid 139-142 MROS Homo sapiens 109-112 17852346-9 2008 Furthermore, the levels of rice PIG-F transcription were up-regulated by growth hormones including GA(3), NAA and kinetin. 1-naphthaleneacetic acid 106-109 phosphatidylinositol glycan anchor biosynthesis class F Sus scrofa 32-37 18069944-10 2008 Accordingly, treatments with sucrose, gibberellic acid and NAA induced upregulation of AtCel2, whereas ABA triggered downregulation of both AtCel2 and KOR3 in roots. 1-naphthaleneacetic acid 59-62 cellulase 2 Arabidopsis thaliana 87-93 18069944-10 2008 Accordingly, treatments with sucrose, gibberellic acid and NAA induced upregulation of AtCel2, whereas ABA triggered downregulation of both AtCel2 and KOR3 in roots. 1-naphthaleneacetic acid 59-62 cellulase 2 Arabidopsis thaliana 140-146 18069944-10 2008 Accordingly, treatments with sucrose, gibberellic acid and NAA induced upregulation of AtCel2, whereas ABA triggered downregulation of both AtCel2 and KOR3 in roots. 1-naphthaleneacetic acid 59-62 glycosyl hydrolase 9A3 Arabidopsis thaliana 151-155 12502072-1 2003 The degradation of phenol by tyrosinase immobilized on chemically modified sodium aluminosilicate (NaA), calcium aluminosilicate (CaA), and silica gel was studied. 1-naphthaleneacetic acid 99-102 tyrosinase Homo sapiens 29-39 16525894-5 2006 It was found that CEG was expressed abundantly in vascular tissues of the primary root, but not in newly formed lateral root primordia and the root meristem, and induced by exogenous auxin NAA (alpha-naphthalene acetic acid). 1-naphthaleneacetic acid 189-192 F-box and associated interaction domains-containing protein Arabidopsis thaliana 18-21 16525894-5 2006 It was found that CEG was expressed abundantly in vascular tissues of the primary root, but not in newly formed lateral root primordia and the root meristem, and induced by exogenous auxin NAA (alpha-naphthalene acetic acid). 1-naphthaleneacetic acid 194-223 F-box and associated interaction domains-containing protein Arabidopsis thaliana 18-21 16525894-6 2006 In addition, the ceg mutant was hyposensitive to NAA, IAA (indole-3-acetic acid) and 2,4-D (2,4-dichlorophenoxyacetic acid), as well as the auxin transport inhibitor TIBA (3,3,5-triiodobenzoic acid), showing that CEG is an auxin-inducible gene. 1-naphthaleneacetic acid 49-52 F-box and associated interaction domains-containing protein Arabidopsis thaliana 17-20 16337204-3 2005 NtPDR3 was found to be induced in suspension cells treated with methyl jasmonate, salicylic acid, 1-naphthalene acetic acid, or cembrene, a macrocyclic diterpene. 1-naphthaleneacetic acid 98-123 pleiotropic drug resistance protein 3 Nicotiana tabacum 0-6 15908594-4 2005 In addition, we showed that the atmdr1-100 and atmdr1-100/atpgp1-100 mutants are defective in multiple aspects of root development, including increased root-growth sensitivity to 1-naphthalene acetic acid (1-NAA), and decreased sensitivity to naphthylphthalamic acid (NPA)-mediated inhibition of root elongation. 1-naphthaleneacetic acid 179-204 ATP binding cassette subfamily B19 Arabidopsis thaliana 32-38 15908594-4 2005 In addition, we showed that the atmdr1-100 and atmdr1-100/atpgp1-100 mutants are defective in multiple aspects of root development, including increased root-growth sensitivity to 1-naphthalene acetic acid (1-NAA), and decreased sensitivity to naphthylphthalamic acid (NPA)-mediated inhibition of root elongation. 1-naphthaleneacetic acid 179-204 ATP binding cassette subfamily B19 Arabidopsis thaliana 47-53 15908594-4 2005 In addition, we showed that the atmdr1-100 and atmdr1-100/atpgp1-100 mutants are defective in multiple aspects of root development, including increased root-growth sensitivity to 1-naphthalene acetic acid (1-NAA), and decreased sensitivity to naphthylphthalamic acid (NPA)-mediated inhibition of root elongation. 1-naphthaleneacetic acid 179-204 ATP binding cassette subfamily B1 Arabidopsis thaliana 58-64 15908594-4 2005 In addition, we showed that the atmdr1-100 and atmdr1-100/atpgp1-100 mutants are defective in multiple aspects of root development, including increased root-growth sensitivity to 1-naphthalene acetic acid (1-NAA), and decreased sensitivity to naphthylphthalamic acid (NPA)-mediated inhibition of root elongation. 1-naphthaleneacetic acid 206-211 ATP binding cassette subfamily B19 Arabidopsis thaliana 32-38 15908594-4 2005 In addition, we showed that the atmdr1-100 and atmdr1-100/atpgp1-100 mutants are defective in multiple aspects of root development, including increased root-growth sensitivity to 1-naphthalene acetic acid (1-NAA), and decreased sensitivity to naphthylphthalamic acid (NPA)-mediated inhibition of root elongation. 1-naphthaleneacetic acid 206-211 ATP binding cassette subfamily B19 Arabidopsis thaliana 47-53 15908594-4 2005 In addition, we showed that the atmdr1-100 and atmdr1-100/atpgp1-100 mutants are defective in multiple aspects of root development, including increased root-growth sensitivity to 1-naphthalene acetic acid (1-NAA), and decreased sensitivity to naphthylphthalamic acid (NPA)-mediated inhibition of root elongation. 1-naphthaleneacetic acid 206-211 ATP binding cassette subfamily B1 Arabidopsis thaliana 58-64 15908594-5 2005 Consistent with the proposed role of AtMDR1 in basipetal auxin transport, we found that expression of the auxin responsive DR5::GUS reporter gene in the central elongation zone is significantly reduced in the atmdr1-100 mutant roots treated with 1-NAA at the root tips, compared to similarly treated wild-type plants. 1-naphthaleneacetic acid 246-251 ATP binding cassette subfamily B19 Arabidopsis thaliana 209-215 16463701-15 2005 In case of all the 1H MRS spectra obtained from the subcortical nuclei regions significant decrease in the NAA/tCr ratio was observed which could reflect the reduced concentration of N-acetyloaspartate, known as a neurons marker. 1-naphthaleneacetic acid 107-110 T cell receptor beta variable 20/OR9-2 (non-functional) Homo sapiens 111-114 15460601-1 2004 OBJECTIVE: To determine whether men who relapse after neoadjuvant androgen ablation (NAA) and high-dose radiation therapy (RT) have faster PSA doubling times(PSAdt) than those who are treated with RT alone. 1-naphthaleneacetic acid 85-88 aminopeptidase puromycin sensitive Homo sapiens 139-142 15460601-6 2004 Patients treated with NAA had higher pre-treatment Gleason scores (p<O.001), PSA (p<O.001), and T stage (p<0.001). 1-naphthaleneacetic acid 22-25 aminopeptidase puromycin sensitive Homo sapiens 80-83 15460601-8 2004 Rising PSA profiles occurred in 78% of the RT-only group and 70% of the NAA group. 1-naphthaleneacetic acid 72-75 aminopeptidase puromycin sensitive Homo sapiens 7-10 12836546-11 2003 The best NAA concentration was 0.01 mg.L-1. 1-naphthaleneacetic acid 9-12 immunoglobulin kappa variable 1-16 Homo sapiens 39-42 12586904-1 2003 Exogenously supplied auxin (1-naphthaleneacetic acid) inhibited light-induced activity increase of polyamine oxidase (PAO), a hydrogen peroxide-producing enzyme, in the outer tissues of maize (Zea mays) mesocotyl. 1-naphthaleneacetic acid 28-52 polyamine oxidase 1 Zea mays 99-116 12586904-1 2003 Exogenously supplied auxin (1-naphthaleneacetic acid) inhibited light-induced activity increase of polyamine oxidase (PAO), a hydrogen peroxide-producing enzyme, in the outer tissues of maize (Zea mays) mesocotyl. 1-naphthaleneacetic acid 28-52 polyamine oxidase 1 Zea mays 118-121 16359384-6 2005 In addition to the salt induction, the AtNAC2 can also be induced by abscisic acid (ABA), ACC and NAA. 1-naphthaleneacetic acid 98-101 NAC domain containing protein 2 Arabidopsis thaliana 39-45 16212599-6 2005 Expression of AtPGP1 in yeast and in the standard mammalian expression system used to analyze human MDR-type proteins results in enhanced efflux of indole-3-acetic acid (IAA) and the synthetic auxin 1-naphthalene acetic acid (1-NAA), but not the inactive auxin 2-NAA. 1-naphthaleneacetic acid 199-224 phosphatidylglycerolphosphate synthase 1 Arabidopsis thaliana 14-20 16212599-6 2005 Expression of AtPGP1 in yeast and in the standard mammalian expression system used to analyze human MDR-type proteins results in enhanced efflux of indole-3-acetic acid (IAA) and the synthetic auxin 1-naphthalene acetic acid (1-NAA), but not the inactive auxin 2-NAA. 1-naphthaleneacetic acid 226-231 phosphatidylglycerolphosphate synthase 1 Arabidopsis thaliana 14-20 16100961-3 2005 RESULTS: Statistically significant decrease (p < 0.01) only of Naa/Cr--at HCR among the CDR0, CDR1+CDR2, and CDR3, and at PCA between CDR0 and CDR1+CDR2 in relation to CDR3. 1-naphthaleneacetic acid 63-66 coiled-coil alpha-helical rod protein 1 Homo sapiens 74-77 16100961-3 2005 RESULTS: Statistically significant decrease (p < 0.01) only of Naa/Cr--at HCR among the CDR0, CDR1+CDR2, and CDR3, and at PCA between CDR0 and CDR1+CDR2 in relation to CDR3. 1-naphthaleneacetic acid 63-66 cerebellar degeneration related protein 1 Homo sapiens 94-98 16100961-3 2005 RESULTS: Statistically significant decrease (p < 0.01) only of Naa/Cr--at HCR among the CDR0, CDR1+CDR2, and CDR3, and at PCA between CDR0 and CDR1+CDR2 in relation to CDR3. 1-naphthaleneacetic acid 63-66 cerebellar degeneration related protein 2 Homo sapiens 99-103 16100961-3 2005 RESULTS: Statistically significant decrease (p < 0.01) only of Naa/Cr--at HCR among the CDR0, CDR1+CDR2, and CDR3, and at PCA between CDR0 and CDR1+CDR2 in relation to CDR3. 1-naphthaleneacetic acid 63-66 CDR3 Homo sapiens 109-113 16100961-3 2005 RESULTS: Statistically significant decrease (p < 0.01) only of Naa/Cr--at HCR among the CDR0, CDR1+CDR2, and CDR3, and at PCA between CDR0 and CDR1+CDR2 in relation to CDR3. 1-naphthaleneacetic acid 63-66 cerebellar degeneration related protein 1 Homo sapiens 143-147 16100961-3 2005 RESULTS: Statistically significant decrease (p < 0.01) only of Naa/Cr--at HCR among the CDR0, CDR1+CDR2, and CDR3, and at PCA between CDR0 and CDR1+CDR2 in relation to CDR3. 1-naphthaleneacetic acid 63-66 cerebellar degeneration related protein 2 Homo sapiens 148-152 16100961-3 2005 RESULTS: Statistically significant decrease (p < 0.01) only of Naa/Cr--at HCR among the CDR0, CDR1+CDR2, and CDR3, and at PCA between CDR0 and CDR1+CDR2 in relation to CDR3. 1-naphthaleneacetic acid 63-66 CDR3 Homo sapiens 168-172 16100961-4 2005 CONCLUSION: The HCR is the first to show Naa reduction (CDR1). 1-naphthaleneacetic acid 41-44 coiled-coil alpha-helical rod protein 1 Homo sapiens 16-19 16100961-4 2005 CONCLUSION: The HCR is the first to show Naa reduction (CDR1). 1-naphthaleneacetic acid 41-44 cerebellar degeneration related protein 1 Homo sapiens 56-60 15739773-2 2004 NAA shape up, in biological fluids, as normal human endogenous low- and high-weight substances, e.g. angiotensin 2, bradykinin and others, and play the regulatory role in homeostasis. 1-naphthaleneacetic acid 0-3 kininogen 1 Homo sapiens 116-126 15739773-5 2004 We determined, within our case study, the level of NAA to e-NOS in blood and tears of patients with diabetic retinopathy and in donors. 1-naphthaleneacetic acid 51-54 nitric oxide synthase 3 Homo sapiens 58-63 15739773-6 2004 The level of NAA to e-NOS was found to be twice higher in tears versus blood in all groups of patients. 1-naphthaleneacetic acid 13-16 nitric oxide synthase 3 Homo sapiens 20-25 13677801-9 2003 The longitudinal decrease of whole brain and hippocampal volumes on MR imaging, NAA levels on 1H MRS, cerebral glucose metabolism on PET, and cerebral blood flow on SPECT are associated with rate of cognitive decline in patients with AD. 1-naphthaleneacetic acid 80-83 MROS Homo sapiens 97-100 12502072-3 2003 Tyrosinase immobilized on CaA and on NaA could be re-used repeatedly without any decrease in performance. 1-naphthaleneacetic acid 37-40 tyrosinase Homo sapiens 0-10 11266581-4 2001 By contrast, in the presence of 10 nM 1-naphthaleneacetic acid (NAA), the concentrations of ethylene required to induce I(50) in eir1-1 and aux1-7 roots were greatly reduced nearly to the level required in wild-type roots. 1-naphthaleneacetic acid 38-62 Auxin efflux carrier family protein Arabidopsis thaliana 129-135 12361388-10 2002 Because the free conformations of both CsH and N(10)-desmethylated cyclosporins differ from those of "classical" (N(10)-methylated, [L-MeVal(11)]-using) cyclosporins, these potent FPR inhibitory cyclosporins probably bind to FPR pharmacophores for which classical cyclosporins show little affinity. 1-naphthaleneacetic acid 47-52 formyl peptide receptor 1 Homo sapiens 180-183 12361388-10 2002 Because the free conformations of both CsH and N(10)-desmethylated cyclosporins differ from those of "classical" (N(10)-methylated, [L-MeVal(11)]-using) cyclosporins, these potent FPR inhibitory cyclosporins probably bind to FPR pharmacophores for which classical cyclosporins show little affinity. 1-naphthaleneacetic acid 47-52 formyl peptide receptor 1 Homo sapiens 225-228 12361388-11 2002 Moreover, because the conformations of the N(10)-desmethylated cyclosporins widely differ from the CsH one, they probably bind to different pharmacophores on the FPR molecules. 1-naphthaleneacetic acid 43-48 formyl peptide receptor 1 Homo sapiens 162-165 11910006-6 2002 Exogenous application of the synthetic auxin 1-naphthylacetic acid is able to rescue the aux1 lateral root phenotype, implying that root auxin levels are suboptimal for lateral root primordium initiation in the mutant. 1-naphthaleneacetic acid 45-66 Transmembrane amino acid transporter family protein Arabidopsis thaliana 89-93 21959764-1 2002 The peptide N-Acetylaspartylglutamate (NAAG) is hydrolyzed by N-Acetylated-alpha-linked-acidic dipeptidase (NAALADase, glutamate carboxypeptidase II) into N-Acetylated aspartate (NAA) and glutamate. 1-naphthaleneacetic acid 39-42 folate hydrolase 1B (pseudogene) Homo sapiens 62-106 21959764-1 2002 The peptide N-Acetylaspartylglutamate (NAAG) is hydrolyzed by N-Acetylated-alpha-linked-acidic dipeptidase (NAALADase, glutamate carboxypeptidase II) into N-Acetylated aspartate (NAA) and glutamate. 1-naphthaleneacetic acid 39-42 folate hydrolase 1B (pseudogene) Homo sapiens 108-117 21959764-1 2002 The peptide N-Acetylaspartylglutamate (NAAG) is hydrolyzed by N-Acetylated-alpha-linked-acidic dipeptidase (NAALADase, glutamate carboxypeptidase II) into N-Acetylated aspartate (NAA) and glutamate. 1-naphthaleneacetic acid 39-42 folate hydrolase 1 Homo sapiens 119-148 11590771-2 2001 The results showed that the ultraviolet light was more effective than fluorescent light in promoting degradation, and the degradation of NAA under ultraviolet light followed the first order kinetics with the photolysis rate constant of 1.15 x 10(-2) min-1 and half-life time (t1/2) of 60 min. 1-naphthaleneacetic acid 137-140 CD59 molecule (CD59 blood group) Homo sapiens 250-255 12113906-6 2002 Multiple regression analyses showed that the APOC1 polymorphism was the only variable which predicted NAA/Cr values in basal ganglia. 1-naphthaleneacetic acid 102-105 apolipoprotein C1 Homo sapiens 45-50 12065761-3 2002 In the current study, we discovered that inactivation of the neuronal isoform of NOS (nNOS) by NAA in vitro results in covalent alteration of the heme prosthetic group, in part, to products that contain an intact porphyrin ring and are either dissociable from or irreversibly bound to the protein. 1-naphthaleneacetic acid 95-98 nitric oxide synthase 1 Homo sapiens 86-90 12065761-9 2002 Because inactivation and alteration may trigger ubiquitination and proteasomal degradation of nNOS, NAA may be a useful biochemical tool for the study of these basic regulatory processes. 1-naphthaleneacetic acid 100-103 nitric oxide synthase 1 Homo sapiens 94-98 11814060-2 2001 Here we report that nicotinamide (NAA) can induce rapid and reversible reversion of aging phenotypes in human diploid fibroblasts in terms of cell morphology and senescence-associated beta-galactosidase activity. 1-naphthaleneacetic acid 34-37 galactosidase beta 1 Homo sapiens 184-202 11260147-8 2001 In contrast, C5a-triggered LTR was significantly higher in ASA (14.4 +/- 12.88 pg/105 cells) and NAA (22.9 +/- 22.61 pg/105 cells) than in AA (9.6 +/- 3.29 pg/105 cells) and controls (7.5 +/- 7.19 pg/105 cells) (P < 0.05). 1-naphthaleneacetic acid 97-100 complement C5a receptor 1 Homo sapiens 13-16 11260147-9 2001 This difference between ASA and NAA vs. AA and controls was even more pronounced when determining the quotient C5a-/anti-IgE-induced LTR (P < 0.001). 1-naphthaleneacetic acid 32-35 complement C5a receptor 1 Homo sapiens 111-114 11266581-4 2001 By contrast, in the presence of 10 nM 1-naphthaleneacetic acid (NAA), the concentrations of ethylene required to induce I(50) in eir1-1 and aux1-7 roots were greatly reduced nearly to the level required in wild-type roots. 1-naphthaleneacetic acid 64-67 Auxin efflux carrier family protein Arabidopsis thaliana 129-135 11097314-6 2000 RESULTS: The numbers of eosinophils and ECP concentration were increased in the sputum of AA and NAA compared with AC and NC (P < 0.05). 1-naphthaleneacetic acid 97-100 ribonuclease A family member 3 Homo sapiens 40-43 30759919-7 2001 The partial amino acid sequences from the purified NAA-induced pI-5.09 LOX are identical to those of LOX4. 1-naphthaleneacetic acid 51-54 seed linoleate 9S-lipoxygenase-3 Glycine max 71-74 30759919-7 2001 The partial amino acid sequences from the purified NAA-induced pI-5.09 LOX are identical to those of LOX4. 1-naphthaleneacetic acid 51-54 linoleate 9S-lipoxygenase-4 Glycine max 101-105 30759919-8 2001 RNA protection assays showed that NAA induces the expression of LOX4 and LOX5 mRNAs in cultured embryo cotyledons where they are not normally expressed. 1-naphthaleneacetic acid 34-37 linoleate 9S-lipoxygenase-4 Glycine max 64-68 30759919-9 2001 Soybean genotypes with a polymorphic variant of LOX4 in hypocotyls showed the same variation as NAA-induced LOXs in the embryo cotyledons. 1-naphthaleneacetic acid 96-99 linoleate 9S-lipoxygenase-4 Glycine max 48-52 30759919-10 2001 These results demonstrate that the NAA-induced pI-5.09 LOX is seedling LOX4 and also suggest that auxin might be directly or indirectly involved in seedling LOX expression during seed germination. 1-naphthaleneacetic acid 35-38 seed linoleate 9S-lipoxygenase-3 Glycine max 55-58 30759919-10 2001 These results demonstrate that the NAA-induced pI-5.09 LOX is seedling LOX4 and also suggest that auxin might be directly or indirectly involved in seedling LOX expression during seed germination. 1-naphthaleneacetic acid 35-38 linoleate 9S-lipoxygenase-4 Glycine max 71-75 30759919-10 2001 These results demonstrate that the NAA-induced pI-5.09 LOX is seedling LOX4 and also suggest that auxin might be directly or indirectly involved in seedling LOX expression during seed germination. 1-naphthaleneacetic acid 35-38 seed linoleate 9S-lipoxygenase-3 Glycine max 71-74 11097314-7 2000 The numbers of eotaxin mRNA+ and immunoreactive cells were increased in NAA, but not AA, when compared with controls (P < 0.05). 1-naphthaleneacetic acid 72-75 C-C motif chemokine ligand 11 Homo sapiens 15-22 11097314-8 2000 Eotaxin immunoreactive cells in NAA were significantly higher than in AA (P < 0.05). 1-naphthaleneacetic acid 32-35 C-C motif chemokine ligand 11 Homo sapiens 0-7 11097314-10 2000 In NAA, but not AA, the numbers of eotaxin mRNA+ cells were correlated with histamine PC20 (r = -0.81, P < 0.01) and eosinophil numbers in sputum (r = 0.7, P < 0.05). 1-naphthaleneacetic acid 3-6 C-C motif chemokine ligand 11 Homo sapiens 35-42 11053229-6 2000 Third, the magnitude of the abnormal dorsal PFC fMRI response was predicted by an assay of N-acetylaspartate concentrations (NAA) in dorsal PFC, a measure of neuronal pathology obtained using proton magnetic resonance spectroscopy. 1-naphthaleneacetic acid 125-128 complement factor properdin Homo sapiens 44-52 11053229-6 2000 Third, the magnitude of the abnormal dorsal PFC fMRI response was predicted by an assay of N-acetylaspartate concentrations (NAA) in dorsal PFC, a measure of neuronal pathology obtained using proton magnetic resonance spectroscopy. 1-naphthaleneacetic acid 125-128 complement factor properdin Homo sapiens 44-47 11053229-7 2000 Patients had significantly lower dorsal PFC NAA than controls and dorsal PFC NAA inversely predicted the fMRI response in dorsal PFC (areas 9, 46) to varying WM difficulty - supporting the assumption that abnormal PFC responses arose from abnormal PFC neurons. 1-naphthaleneacetic acid 77-80 complement factor properdin Homo sapiens 73-76 11053229-7 2000 Patients had significantly lower dorsal PFC NAA than controls and dorsal PFC NAA inversely predicted the fMRI response in dorsal PFC (areas 9, 46) to varying WM difficulty - supporting the assumption that abnormal PFC responses arose from abnormal PFC neurons. 1-naphthaleneacetic acid 77-80 complement factor properdin Homo sapiens 73-76 11053229-7 2000 Patients had significantly lower dorsal PFC NAA than controls and dorsal PFC NAA inversely predicted the fMRI response in dorsal PFC (areas 9, 46) to varying WM difficulty - supporting the assumption that abnormal PFC responses arose from abnormal PFC neurons. 1-naphthaleneacetic acid 77-80 complement factor properdin Homo sapiens 73-76 11053229-7 2000 Patients had significantly lower dorsal PFC NAA than controls and dorsal PFC NAA inversely predicted the fMRI response in dorsal PFC (areas 9, 46) to varying WM difficulty - supporting the assumption that abnormal PFC responses arose from abnormal PFC neurons. 1-naphthaleneacetic acid 77-80 complement factor properdin Homo sapiens 73-76 11721401-2 1999 METHODS: c-kit expression on the bone marrow mononuclear cells(MNC) and CD34 positive cells from 11 chronic aplastic anemia (CAA), 9 severe aplastic anemia(SAA), 10 non-aplastic anemia(NAA) patients (5 iron deficiency anemia and 5 autoimmune hemolytic anemia) and 5 healthy controls was measured with flow cytometric immunofluorescence assay and anti-CD117 monoclonal antibody. 1-naphthaleneacetic acid 185-188 KIT proto-oncogene, receptor tyrosine kinase Homo sapiens 9-14 11065138-2 2000 Among augmenting inspiratory (aug-I), postinspiratory (post-I), and augmenting expiratory (aug-E) neurons labeled, GAD-immunoreactivity was demonstrated only in those neurons that were not antidromically activated (NAA) by stimulation of the vagus nerve and the C2-C3 spinal cord. 1-naphthaleneacetic acid 215-218 glutamate decarboxylase 1 Felis catus 115-118 10712533-4 2000 AtPRP3/beta-glucuronidase (GUS) expression increased in roots of transgenic seedlings treated with either 1-aminocyclopropane-1-carboxylic acid (ACC) or alpha-naphthaleneacetic acid (alpha-NAA), compounds known to promote root hair formation. 1-naphthaleneacetic acid 153-181 proline-rich protein 3 Arabidopsis thaliana 0-6 10712533-4 2000 AtPRP3/beta-glucuronidase (GUS) expression increased in roots of transgenic seedlings treated with either 1-aminocyclopropane-1-carboxylic acid (ACC) or alpha-naphthaleneacetic acid (alpha-NAA), compounds known to promote root hair formation. 1-naphthaleneacetic acid 183-192 proline-rich protein 3 Arabidopsis thaliana 0-6 10712533-5 2000 In the presence of 1-alpha-(2-aminoethoxyvinyl)glycine (AVG), an inhibitor of ethylene biosynthesis, AtPRP3/GUS expression was strongly reduced, but could be rescued by co-addition of ACC or alpha-NAA to the growth medium. 1-naphthaleneacetic acid 191-200 proline-rich protein 3 Arabidopsis thaliana 101-107 10652297-11 2000 ZmSAUR1 gene expression began within 10 min, increased rapidly between 10 and 60 min, and peaked around 60 min after 10 microM alpha-naphthaleneacetic acid treatment. 1-naphthaleneacetic acid 127-155 auxin-responsive protein SAUR50 Zea mays 0-7 11543173-1 1999 The partially agravitropic growth habit of roots of an auxin-resistant mutant of Arabidopsis thaliana, axr4, was restored by the addition of 30-300 nM 1-naphthaleneacetic acid (NAA) to the growth medium. 1-naphthaleneacetic acid 151-175 alpha/beta-Hydrolases superfamily protein Arabidopsis thaliana 103-107 11543173-1 1999 The partially agravitropic growth habit of roots of an auxin-resistant mutant of Arabidopsis thaliana, axr4, was restored by the addition of 30-300 nM 1-naphthaleneacetic acid (NAA) to the growth medium. 1-naphthaleneacetic acid 177-180 alpha/beta-Hydrolases superfamily protein Arabidopsis thaliana 103-107 10570327-5 1999 Compared with AC and NC, the numbers of EG2+ cells and the cells expressing mRNA for eotaxin, eotaxin-2, RANTES, MCP-3, MCP-4, and CCR3 were significantly increased in AA and NAA (p < 0.01). 1-naphthaleneacetic acid 175-178 C-C motif chemokine receptor 3 Homo sapiens 131-135 10570327-7 1999 Significant correlations between the cells expressing eotaxin or CCR3 and EG2+ eosinophils in the bronchial tissue were also observed for both AA (p < 0.01) and NAA (p = 0.01). 1-naphthaleneacetic acid 164-167 C-C motif chemokine ligand 11 Homo sapiens 54-61 10570327-7 1999 Significant correlations between the cells expressing eotaxin or CCR3 and EG2+ eosinophils in the bronchial tissue were also observed for both AA (p < 0.01) and NAA (p = 0.01). 1-naphthaleneacetic acid 164-167 C-C motif chemokine receptor 3 Homo sapiens 65-69 10570327-8 1999 Moreover, in the total asthmatic group (AA + NAA) there was a significant inverse correlation between the expression of eotaxin and that of the histamine PC20 (p < 0.05). 1-naphthaleneacetic acid 45-48 C-C motif chemokine ligand 11 Homo sapiens 120-127 11721401-3 1999 RESULTS: The CD117 positive rates of MNC in CAA, SAA, NAA and control groups were (2.08 +/- 1.96)%, (0.89 +/- 0.32)%, (0.67 +/- 0.39)% and (0.45 +/- 0.16)%, respectively, and the rates were significantly higher in CAA and SAA groups than in NAA and control groups(P < 0.05). 1-naphthaleneacetic acid 54-57 KIT proto-oncogene, receptor tyrosine kinase Homo sapiens 13-18 11721401-3 1999 RESULTS: The CD117 positive rates of MNC in CAA, SAA, NAA and control groups were (2.08 +/- 1.96)%, (0.89 +/- 0.32)%, (0.67 +/- 0.39)% and (0.45 +/- 0.16)%, respectively, and the rates were significantly higher in CAA and SAA groups than in NAA and control groups(P < 0.05). 1-naphthaleneacetic acid 241-244 KIT proto-oncogene, receptor tyrosine kinase Homo sapiens 13-18 11721401-4 1999 The CD117 and CD34 double positive cells of MNC in CAA, SAA, NAA and control groups were (0.87 +/- 0.57)%, (0.65 +/- 0.41)%, (0.46 +/- 0.26)% and (0.41 +/- 0.15)%, respectively, and were significantly higher in CAA and SAA groups than in NAA and control groups (P < 0.05). 1-naphthaleneacetic acid 61-64 KIT proto-oncogene, receptor tyrosine kinase Homo sapiens 4-9 11721401-4 1999 The CD117 and CD34 double positive cells of MNC in CAA, SAA, NAA and control groups were (0.87 +/- 0.57)%, (0.65 +/- 0.41)%, (0.46 +/- 0.26)% and (0.41 +/- 0.15)%, respectively, and were significantly higher in CAA and SAA groups than in NAA and control groups (P < 0.05). 1-naphthaleneacetic acid 61-64 CD34 molecule Homo sapiens 14-18 9871369-3 1998 Vertically placed agr roots grow into agar medium containing IAA or naphthalene-1-acetic acid, but not into medium containing 2,4-D. 1-naphthaleneacetic acid 68-93 Auxin efflux carrier family protein Arabidopsis thaliana 18-21 11721401-5 1999 The CD117 positive cells of CD34 positive cells were also higher in CAA[(71.6 +/- 12.4)%] and SAA[(88.9 +/- 23.5)%] groups than in NAA[(41.8 +/- 10.3)%] and control [(46.3 +/- 9.7)%] groups (P < 0.01). 1-naphthaleneacetic acid 131-134 KIT proto-oncogene, receptor tyrosine kinase Homo sapiens 4-9 11721401-5 1999 The CD117 positive cells of CD34 positive cells were also higher in CAA[(71.6 +/- 12.4)%] and SAA[(88.9 +/- 23.5)%] groups than in NAA[(41.8 +/- 10.3)%] and control [(46.3 +/- 9.7)%] groups (P < 0.01). 1-naphthaleneacetic acid 131-134 CD34 molecule Homo sapiens 28-32 10205161-3 1999 We are able to bypass the defect within auxin uptake and restore the gravitropic root phenotype of aux1 by growing mutant seedlings in the presence of the membrane-permeable synthetic auxin, 1-naphthaleneacetic acid. 1-naphthaleneacetic acid 191-215 Transmembrane amino acid transporter family protein Arabidopsis thaliana 99-103 9838072-3 1998 PSMA is also expressed in the brain, and is involved in conversion of the major neurotransmitter NAAG (N-acetyl-aspartyl glutamate) to NAA and free glutamate, the levels of which are disrupted in several neurological disorders including multiple sclerosis, amyotrophic lateral sclerosis, Alzheimer"s disease and schizophrenia. 1-naphthaleneacetic acid 97-100 folate hydrolase 1 Homo sapiens 0-4 11477878-6 1998 A significant elevation in IL-5 release following HDM stimulation in PBMC and BALF cells was observed in the both asthmatic and atopic groups; but the value in AA was higher than that in NAA, and it was also higher in BALF cells than in PBMC. 1-naphthaleneacetic acid 187-190 interleukin 5 Homo sapiens 27-31 18726202-1 1998 The relationship among transfer and expression of auxin binding protein gene (abp), auxin (NAA)-induced plasmalemma hyperpolarity and sensibility to auxin during protoplast culture was studied by measuring transmembrane potential difference (Em) and culturing the protoplasts of sense and antisenseabp transgenic tobacco. 1-naphthaleneacetic acid 91-94 auxin-binding protein T85 Nicotiana tabacum 50-71 9697346-1 1998 The agravitropic nature of root growth of an auxin-resistant mutant of Arabidopsis, aux1, was restored when the synthetic auxin 1-naphthaleneacetic acid (NAA) was added to the growth medium; aux1 roots were not resistant to NAA. 1-naphthaleneacetic acid 128-152 Transmembrane amino acid transporter family protein Arabidopsis thaliana 84-88 9697346-1 1998 The agravitropic nature of root growth of an auxin-resistant mutant of Arabidopsis, aux1, was restored when the synthetic auxin 1-naphthaleneacetic acid (NAA) was added to the growth medium; aux1 roots were not resistant to NAA. 1-naphthaleneacetic acid 154-157 Transmembrane amino acid transporter family protein Arabidopsis thaliana 84-88 9697346-1 1998 The agravitropic nature of root growth of an auxin-resistant mutant of Arabidopsis, aux1, was restored when the synthetic auxin 1-naphthaleneacetic acid (NAA) was added to the growth medium; aux1 roots were not resistant to NAA. 1-naphthaleneacetic acid 154-157 Transmembrane amino acid transporter family protein Arabidopsis thaliana 191-195 9697346-1 1998 The agravitropic nature of root growth of an auxin-resistant mutant of Arabidopsis, aux1, was restored when the synthetic auxin 1-naphthaleneacetic acid (NAA) was added to the growth medium; aux1 roots were not resistant to NAA. 1-naphthaleneacetic acid 224-227 Transmembrane amino acid transporter family protein Arabidopsis thaliana 84-88 18726202-1 1998 The relationship among transfer and expression of auxin binding protein gene (abp), auxin (NAA)-induced plasmalemma hyperpolarity and sensibility to auxin during protoplast culture was studied by measuring transmembrane potential difference (Em) and culturing the protoplasts of sense and antisenseabp transgenic tobacco. 1-naphthaleneacetic acid 91-94 auxin-binding protein T85 Nicotiana tabacum 78-81 9426235-4 1997 Nt-gh3 mRNA accumulates within a short time after auxin treatment, responds to very low concentrations of NAA (as little as 10[-9] M) and specifically to active auxins. 1-naphthaleneacetic acid 106-109 probable indole-3-acetic acid-amido synthetase GH3.1 Nicotiana tabacum 3-6 9100453-4 1996 The SP-A/TP ratio in the NAA was similar to that in BLF but 1/24 of that in BALF. 1-naphthaleneacetic acid 25-28 surfactant protein A2 Homo sapiens 4-8 9407392-7 1997 These include near-field circulations in the brain and the eye, and a far-field systemic circulation involving the liver and kidney, the purpose of which in each case is apparently to regenerate aspartate (Asp) in order for it to be recycled into NAA as part of the still unknown function of the NAA cycle. 1-naphthaleneacetic acid 247-250 assembly factor for spindle microtubules Homo sapiens 206-209 9407392-7 1997 These include near-field circulations in the brain and the eye, and a far-field systemic circulation involving the liver and kidney, the purpose of which in each case is apparently to regenerate aspartate (Asp) in order for it to be recycled into NAA as part of the still unknown function of the NAA cycle. 1-naphthaleneacetic acid 296-299 assembly factor for spindle microtubules Homo sapiens 206-209 9407392-10 1997 Second, there is up to a 50-fold drop in the intercompartmental NAA gradient, and third, the ability of the brain to perform its normal intercompartmental cycling of NAA to Asp is terminated, and as a result, the only remaining long-term source of Asp for NAA synthesis is via nutritional supplementation of Asp or its metabolic precursors. 1-naphthaleneacetic acid 166-169 assembly factor for spindle microtubules Homo sapiens 173-176 9407392-10 1997 Second, there is up to a 50-fold drop in the intercompartmental NAA gradient, and third, the ability of the brain to perform its normal intercompartmental cycling of NAA to Asp is terminated, and as a result, the only remaining long-term source of Asp for NAA synthesis is via nutritional supplementation of Asp or its metabolic precursors. 1-naphthaleneacetic acid 166-169 assembly factor for spindle microtubules Homo sapiens 173-176 30727631-3 1997 Nodules were induced from leaf-base or chopped shoot-base explants on modified half-strength MS medium supplemented with 2.69-5.37 muM NAA and 4.44 muM BA and could be maintained long-term as nodules. 1-naphthaleneacetic acid 135-138 latexin Homo sapiens 131-134 30727631-8 1997 Many shoots formed roots in the same medium in which they were regenerated after 10 subcultures, but the best rooting occurred in medium containing 0.54 muM NAA and 0.44 muM BA. 1-naphthaleneacetic acid 157-160 latexin Homo sapiens 153-156 8728022-1 1996 The present study investigates synergistic effects of the TNF-alpha inhibitor thalidomide and the poly(ADP-ribose) polymerase (PARP)-inhibitor nicotinic acid amide (NAA) in male DBA/1 hybird mice suffering from type II collagen-induced arthritis. 1-naphthaleneacetic acid 165-168 poly (ADP-ribose) polymerase family, member 1 Mus musculus 98-125 8728022-1 1996 The present study investigates synergistic effects of the TNF-alpha inhibitor thalidomide and the poly(ADP-ribose) polymerase (PARP)-inhibitor nicotinic acid amide (NAA) in male DBA/1 hybird mice suffering from type II collagen-induced arthritis. 1-naphthaleneacetic acid 165-168 poly (ADP-ribose) polymerase family, member 1 Mus musculus 127-131 8742498-7 1996 Our results provide evidence that the AAP-induced hepatitis and its exacerbation by ethanol can either be reduced by end-product inhibition of PARP by NAA or by dietary depletion of the enzyme"s substrate NAD. 1-naphthaleneacetic acid 151-154 poly (ADP-ribose) polymerase family, member 1 Mus musculus 143-147 12232095-7 1994 In contract, sucrose-induced expression of Gus mediated by a sucrose-responsive domain of the VspB promoter was strongly inhibited by NAA. 1-naphthaleneacetic acid 134-137 stem 31 kDa glycoprotein Glycine max 94-98 7920716-7 1994 The level of AtsEH transcripts increased strongly after treatment with a plant hormone, auxin (2,4-dichlorophenoxyacetic acid, 2,4-D; naphthalene-acetic acid, NAA; and indole-3-acetic acid, IAA) in young, pre-bolting plants. 1-naphthaleneacetic acid 159-162 soluble epoxide hydrolase Arabidopsis thaliana 13-18 7972488-7 1994 The expressed ABP1 also appears to be active, since extracts of insect cells expressing ABP1 contain a saturable high-affinity 1-naphthylacetic acid-binding site, whereas no saturable auxin-binding activity is detected in extracts from control cells. 1-naphthaleneacetic acid 127-148 auxin-binding protein 1 Zea mays 14-18 7972488-7 1994 The expressed ABP1 also appears to be active, since extracts of insect cells expressing ABP1 contain a saturable high-affinity 1-naphthylacetic acid-binding site, whereas no saturable auxin-binding activity is detected in extracts from control cells. 1-naphthaleneacetic acid 127-148 auxin-binding protein 1 Zea mays 88-92 33874506-6 1994 Bulbil formation in the presence of NAA was strongly stimulated by 176 mM sucrose for both cultivars but there were genotypie differences in auxin responses, St Keverne showing good bulbil development with 0.54 muM NAA, 5.4 mu 1AA and 5.4 muM 1BA und Hawera responding only to 27 muM LAA. 1-naphthaleneacetic acid 36-39 PWWP domain containing 3A, DNA repair factor Homo sapiens 239-244 7823015-8 1994 Current work will elucidate whether any of the mCAT proteins interact with members of a newly identified family of single membrane-spanning proteins, such as rBAT, 4F2 and NAA-Tr, which are thought to modulate or activate y+L and/or bo,+ transport systems. 1-naphthaleneacetic acid 172-175 catalase Mus musculus 47-51 24193758-7 1994 This compact embryogenic tissue differentiated into normal somatic embryos when transferred onto regeneration medium containing NAA (15 muM) and ABA (2 muM). 1-naphthaleneacetic acid 128-131 latexin Homo sapiens 136-139 1324098-7 1992 The abolished AA and NAA were restored by 0.5 mg/kg morphine and 0.25 mg/kg ACTH, respectively. 1-naphthaleneacetic acid 21-24 proopiomelanocortin Homo sapiens 76-80 1301071-4 1992 Diaziquone (AZQ) was reduced enzymatically by 2e- using S9 cell fraction from MCF-7 cells which is rich in NAA(P)H:quinone-acceptor oxidoreductase (DT-diaphorase) (QAO) activity. 1-naphthaleneacetic acid 107-110 NAD(P)H quinone dehydrogenase 1 Homo sapiens 148-161 24196174-1 1993 Green Protocorm-like Bodies (PLB) with high multiplication capacity were induced from shoot tips of flower stalk buds having 1 or 2 leaf primordia using New Dogashima Medium (NDM) containing 0.1 mg l(-1) alpha-naphthaleneacetic acid (NAA) and 1 mg 1(-1) 6-benzylaminopurine (BAP). 1-naphthaleneacetic acid 202-232 phospholamban Homo sapiens 6-27 24196174-1 1993 Green Protocorm-like Bodies (PLB) with high multiplication capacity were induced from shoot tips of flower stalk buds having 1 or 2 leaf primordia using New Dogashima Medium (NDM) containing 0.1 mg l(-1) alpha-naphthaleneacetic acid (NAA) and 1 mg 1(-1) 6-benzylaminopurine (BAP). 1-naphthaleneacetic acid 202-232 phospholamban Homo sapiens 29-32 24196174-1 1993 Green Protocorm-like Bodies (PLB) with high multiplication capacity were induced from shoot tips of flower stalk buds having 1 or 2 leaf primordia using New Dogashima Medium (NDM) containing 0.1 mg l(-1) alpha-naphthaleneacetic acid (NAA) and 1 mg 1(-1) 6-benzylaminopurine (BAP). 1-naphthaleneacetic acid 234-237 phospholamban Homo sapiens 29-32 1324098-8 1992 Hence, beta-E and ACTH liberated from the pituitary gland by stimulation of an AP and NAP may act as positive feedback on the AA and NAA afferent pathways, respectively. 1-naphthaleneacetic acid 133-136 proopiomelanocortin Homo sapiens 18-22 1376778-13 1992 Nitric oxide synthesis by IL-1-activated smooth muscle cells is inhibited by NAA, NMA, and N omega-nitro-L-arginine (NNA) with ED50 (i.e., effective dose for 50% inhibition) values of 20, 60, and 1000 microM, respectively; this rank order of inhibition is characteristic of an agonist-unregulated, inducible isoform of nitric oxide synthase. 1-naphthaleneacetic acid 77-80 interleukin 1 beta Canis lupus familiaris 26-30 1657317-6 1991 Both NAA and NAPS responsive neuron activity that were abolished by hypophysectomy were restored by concurrent application of NAPS and intraperitoneal ACTH. 1-naphthaleneacetic acid 5-8 proopiomelanocortin Homo sapiens 151-155 1756590-1 1991 A new sensitive method for measuring aspartoacylase activity in human skin fibroblasts using [3H]N-acetyl-L-aspartic acid (NAA) is described. 1-naphthaleneacetic acid 123-126 aspartoacylase Homo sapiens 37-51 16668538-6 1991 The results revealed that at least one of the more acidic nonembryo LOX isozymes was induced by either alpha-naphthaleneacetic acid or indoleacetic acid but not by 2,4-dichlorophenoxyacetic acid after 4 days" exposure. 1-naphthaleneacetic acid 103-131 seed linoleate 9S-lipoxygenase-3 Glycine max 68-71 16668538-10 1991 The more acidic isozymes induced by alpha-naphthaleneacetic acid showed enzymatic activity and shared the same molecular mass and isoelectric point values as the germination-associated LOX isozymes found in hypocotyls and radicles, suggesting that those LOXs are involved in germination competency of soybean embryos. 1-naphthaleneacetic acid 36-64 seed linoleate 9S-lipoxygenase-3 Glycine max 185-188 1657317-8 1991 It was concluded that NAA production involves dopaminergic transmission in the HARN and ACTH acting presynaptically on this transmission. 1-naphthaleneacetic acid 22-25 proopiomelanocortin Homo sapiens 88-92 33762638-5 2021 In this study, we investigated the correlation of the Val66Met polymorphism of the BDNF gene with Glx/NAA in the pregenual anterior cingulate cortex (pgACC) using MRS at 3 Tesla (T) (n = 30, all males) and 7 T (n = 98, 40 females). 1-naphthaleneacetic acid 102-105 brain derived neurotrophic factor Homo sapiens 83-87 1676667-7 1991 The Kis for 1-naphthylacetic acid and 2-naphthylacetic acid as inhibitors of AST IV-catalyzed sulfation of 1-naphthalenemethanol were approximately 10-fold higher than those of the corresponding naphthoic acids. 1-naphthaleneacetic acid 12-33 sulfotransferase family 1A member 1 Rattus norvegicus 77-83 33762638-0 2021 Met carriers of the BDNF Val66Met polymorphism show reduced Glx/NAA in the pregenual ACC in two independent cohorts. 1-naphthaleneacetic acid 64-67 SAFB like transcription modulator Homo sapiens 0-3 33762638-6 2021 In both cohorts, Met carriers had lower Glx/NAA compared to Val homozygotes. 1-naphthaleneacetic acid 44-47 SAFB like transcription modulator Homo sapiens 17-20 33762638-0 2021 Met carriers of the BDNF Val66Met polymorphism show reduced Glx/NAA in the pregenual ACC in two independent cohorts. 1-naphthaleneacetic acid 64-67 brain derived neurotrophic factor Homo sapiens 20-24 33762638-7 2021 Follow-up analyses using absolute quantification revealed that the Met carriers do not show decreased pgACC glutamate or glutamine levels, but instead show increased NAA compared to the Val homozygotes. 1-naphthaleneacetic acid 166-169 SAFB like transcription modulator Homo sapiens 67-70 34937345-3 2022 Electrochemical impedance changes in a thrombin binding aptamer (TBA)-functionalized NAA/ITO/glass electrode due to specific binding of alpha-thrombin are monitored for protein detection. 1-naphthaleneacetic acid 85-88 coagulation factor II, thrombin Homo sapiens 39-47 34931817-6 2022 Importantly, we discovered that the n-10 isomers in C16:1 and C18:1 of aminophospholipids showed elevated contribution among other isomers, which correlated well with an increased transcription of the corresponding desaturase (FADS2) in the human breast cancer cell line (MDA-MB-231) relative to that in the normal cell line (HMEC). 1-naphthaleneacetic acid 36-40 fatty acid desaturase 2 Homo sapiens 227-232 34282445-9 2022 In multivariate analysis, interferon (IFN) gamma (gamma) levels (OR = 4.1; 95% 2.01-7.9) and depressive symptoms (OR = 1.9; 95%CI = 1.1-3.2) were associated with NAA/Cr ratio. 1-naphthaleneacetic acid 162-165 interferon gamma Homo sapiens 26-48 34946329-9 2021 C-reactive protein (CRP) increased significantly in the No-AA group throughout all time intervals, and from the first 24 h to the 25-72 h in the NAA and CAA groups. 1-naphthaleneacetic acid 145-148 C-reactive protein Homo sapiens 0-18 34946329-11 2021 CRP was significantly higher in the first 24 h in the CAA than in the No-AA group, and in the 24-48 h in the CAA group than in the No-AA and NAA groups. 1-naphthaleneacetic acid 141-144 C-reactive protein Homo sapiens 0-3 34631362-7 2021 Captopril, moexipril, benazepril, fosinopril, losartan, remdesivir, Sigma ACEI, NAA, and NAM interacted and docked at the interface of ACE2 and SARS-CoV-2 spike protein complex. 1-naphthaleneacetic acid 80-83 angiotensin converting enzyme 2 Homo sapiens 135-139 34631362-7 2021 Captopril, moexipril, benazepril, fosinopril, losartan, remdesivir, Sigma ACEI, NAA, and NAM interacted and docked at the interface of ACE2 and SARS-CoV-2 spike protein complex. 1-naphthaleneacetic acid 80-83 surface glycoprotein Severe acute respiratory syndrome coronavirus 2 155-160 34480752-7 2021 The exogenous IAA or auxin analog 1-naphthalene acetic acid (NAA) both rescued the root hair growth phenotype of hb24 mutants, but IBA did not, suggesting a role for HB24 in the IBA-to-IAA conversion. 1-naphthaleneacetic acid 34-59 homeobox protein 24 Arabidopsis thaliana 113-117 34480752-7 2021 The exogenous IAA or auxin analog 1-naphthalene acetic acid (NAA) both rescued the root hair growth phenotype of hb24 mutants, but IBA did not, suggesting a role for HB24 in the IBA-to-IAA conversion. 1-naphthaleneacetic acid 61-64 homeobox protein 24 Arabidopsis thaliana 113-117 34480752-7 2021 The exogenous IAA or auxin analog 1-naphthalene acetic acid (NAA) both rescued the root hair growth phenotype of hb24 mutants, but IBA did not, suggesting a role for HB24 in the IBA-to-IAA conversion. 1-naphthaleneacetic acid 61-64 homeobox protein 24 Arabidopsis thaliana 166-170 34628190-5 2021 NAA treatments showed that CHX23 was crucial for pollen auxin homeostasis. 1-naphthaleneacetic acid 0-3 cation/H+ exchanger 23 Arabidopsis thaliana 27-32 34628190-9 2021 PIN8 depends on CHX23 in regulating pollen morphology and response to NAA treatments. 1-naphthaleneacetic acid 70-73 Auxin efflux carrier family protein Arabidopsis thaliana 0-4 34628190-9 2021 PIN8 depends on CHX23 in regulating pollen morphology and response to NAA treatments. 1-naphthaleneacetic acid 70-73 cation/H+ exchanger 23 Arabidopsis thaliana 16-21 34660662-6 2021 Among the three SCFAs, NaA revealed the best efficacy at alleviating TGF-beta1-induced LX2 cell activation. 1-naphthaleneacetic acid 23-26 transforming growth factor beta 1 Homo sapiens 69-78 34660662-9 2021 The results of phosphokinase array kit and Western blot indicated that NaA increased the AMP-activated protein kinase (AMPK) activation and reduced the phosphorylation of c-Jun in cultured LX2 cells, and siRNA-peroxisome proliferator-activated receptor (PPAR) -gamma abolished the inhibitory effects of NaA against TGF-beta1-induced LX2 cell activation. 1-naphthaleneacetic acid 71-74 protein kinase AMP-activated non-catalytic subunit beta 1 Homo sapiens 89-117 34660662-9 2021 The results of phosphokinase array kit and Western blot indicated that NaA increased the AMP-activated protein kinase (AMPK) activation and reduced the phosphorylation of c-Jun in cultured LX2 cells, and siRNA-peroxisome proliferator-activated receptor (PPAR) -gamma abolished the inhibitory effects of NaA against TGF-beta1-induced LX2 cell activation. 1-naphthaleneacetic acid 71-74 protein kinase AMP-activated non-catalytic subunit beta 1 Homo sapiens 119-123 34660662-9 2021 The results of phosphokinase array kit and Western blot indicated that NaA increased the AMP-activated protein kinase (AMPK) activation and reduced the phosphorylation of c-Jun in cultured LX2 cells, and siRNA-peroxisome proliferator-activated receptor (PPAR) -gamma abolished the inhibitory effects of NaA against TGF-beta1-induced LX2 cell activation. 1-naphthaleneacetic acid 71-74 Jun proto-oncogene, AP-1 transcription factor subunit Homo sapiens 171-176 34660662-9 2021 The results of phosphokinase array kit and Western blot indicated that NaA increased the AMP-activated protein kinase (AMPK) activation and reduced the phosphorylation of c-Jun in cultured LX2 cells, and siRNA-peroxisome proliferator-activated receptor (PPAR) -gamma abolished the inhibitory effects of NaA against TGF-beta1-induced LX2 cell activation. 1-naphthaleneacetic acid 303-306 protein kinase AMP-activated non-catalytic subunit beta 1 Homo sapiens 89-117 34660662-9 2021 The results of phosphokinase array kit and Western blot indicated that NaA increased the AMP-activated protein kinase (AMPK) activation and reduced the phosphorylation of c-Jun in cultured LX2 cells, and siRNA-peroxisome proliferator-activated receptor (PPAR) -gamma abolished the inhibitory effects of NaA against TGF-beta1-induced LX2 cell activation. 1-naphthaleneacetic acid 303-306 Jun proto-oncogene, AP-1 transcription factor subunit Homo sapiens 171-176 34660662-9 2021 The results of phosphokinase array kit and Western blot indicated that NaA increased the AMP-activated protein kinase (AMPK) activation and reduced the phosphorylation of c-Jun in cultured LX2 cells, and siRNA-peroxisome proliferator-activated receptor (PPAR) -gamma abolished the inhibitory effects of NaA against TGF-beta1-induced LX2 cell activation. 1-naphthaleneacetic acid 303-306 peroxisome proliferator activated receptor gamma Homo sapiens 204-266 34660662-9 2021 The results of phosphokinase array kit and Western blot indicated that NaA increased the AMP-activated protein kinase (AMPK) activation and reduced the phosphorylation of c-Jun in cultured LX2 cells, and siRNA-peroxisome proliferator-activated receptor (PPAR) -gamma abolished the inhibitory effects of NaA against TGF-beta1-induced LX2 cell activation. 1-naphthaleneacetic acid 303-306 transforming growth factor beta 1 Homo sapiens 315-324 34660662-10 2021 In conclusion, this study showed that NaA inhibited LX2 cell activation by activating the AMPK/PPARgamma and blocking the c-Jun signaling pathways. 1-naphthaleneacetic acid 38-41 protein kinase AMP-activated non-catalytic subunit beta 1 Homo sapiens 90-94 34660662-10 2021 In conclusion, this study showed that NaA inhibited LX2 cell activation by activating the AMPK/PPARgamma and blocking the c-Jun signaling pathways. 1-naphthaleneacetic acid 38-41 peroxisome proliferator activated receptor gamma Homo sapiens 95-104 34660662-10 2021 In conclusion, this study showed that NaA inhibited LX2 cell activation by activating the AMPK/PPARgamma and blocking the c-Jun signaling pathways. 1-naphthaleneacetic acid 38-41 Jun proto-oncogene, AP-1 transcription factor subunit Homo sapiens 122-127 34489672-11 2021 In the frontal voxel, NAA was predicted by more IL-1B and less TNF-alpha, Cr by less TNF-alpha and more IL-5, and Glx by less TNF-alpha. 1-naphthaleneacetic acid 22-25 interleukin 1 beta Homo sapiens 48-53 34489672-11 2021 In the frontal voxel, NAA was predicted by more IL-1B and less TNF-alpha, Cr by less TNF-alpha and more IL-5, and Glx by less TNF-alpha. 1-naphthaleneacetic acid 22-25 tumor necrosis factor Homo sapiens 63-72 34489672-11 2021 In the frontal voxel, NAA was predicted by more IL-1B and less TNF-alpha, Cr by less TNF-alpha and more IL-5, and Glx by less TNF-alpha. 1-naphthaleneacetic acid 22-25 tumor necrosis factor Homo sapiens 85-94 34489672-11 2021 In the frontal voxel, NAA was predicted by more IL-1B and less TNF-alpha, Cr by less TNF-alpha and more IL-5, and Glx by less TNF-alpha. 1-naphthaleneacetic acid 22-25 interleukin 5 Homo sapiens 104-108 34489672-11 2021 In the frontal voxel, NAA was predicted by more IL-1B and less TNF-alpha, Cr by less TNF-alpha and more IL-5, and Glx by less TNF-alpha. 1-naphthaleneacetic acid 22-25 tumor necrosis factor Homo sapiens 126-135 34489672-12 2021 In the parietal voxel, MI was predicted by more IL-10 and IL-8 and less IL-2, Cho by more TNF-alpha and less IL-2, NAA by more IL-1B and TNF-alpha and less IL-13, IL-2, and IL-7, and Cr by more IL-10 and less IL-2. 1-naphthaleneacetic acid 115-118 interleukin 1 beta Homo sapiens 127-132 34489672-12 2021 In the parietal voxel, MI was predicted by more IL-10 and IL-8 and less IL-2, Cho by more TNF-alpha and less IL-2, NAA by more IL-1B and TNF-alpha and less IL-13, IL-2, and IL-7, and Cr by more IL-10 and less IL-2. 1-naphthaleneacetic acid 115-118 tumor necrosis factor Homo sapiens 137-146 34423210-7 2021 The Thomas, Yoon-Nelson, and bed depth service time models were applied to describe the breakthrough curves for Pb(II) ion removal by a fix-bed column using the MKG@NaA absorbent and to predict the column performance under different experimental conditions with good linearity (R 2 > 0.95). 1-naphthaleneacetic acid 165-168 submaxillary gland androgen regulated protein 3B Homo sapiens 112-118 34111481-7 2021 Molecular dynamics simulation analysis showed the binding energy between mLRAP and NAA were electrostatic forces and Van der Walls. 1-naphthaleneacetic acid 83-86 amelogenin, X-linked Mus musculus 73-78