PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 10727403-1 2000 In the biosynthesis of heparin and heparan sulphate, D-glucuronic acid residues are converted into L-iduronic acid (IdoA) units by C-5 epimerization, at the polymer level. Iduronic Acid 99-114 complement C5 Bos taurus 131-134 10727403-1 2000 In the biosynthesis of heparin and heparan sulphate, D-glucuronic acid residues are converted into L-iduronic acid (IdoA) units by C-5 epimerization, at the polymer level. Iduronic Acid 116-120 complement C5 Bos taurus 131-134 25043515-2 2014 Maize ABP1 was simulated with the natural auxin indole-3-acetic acid (IAA) and the synthetic analog naphthalen-1-acetic acid (NAA), to elucidate the role of the KDEL sequence and the helix at the C-terminus. 1-naphthaleneacetic acid 126-129 auxin-binding protein 1 Zea mays 6-10 25043515-10 2014 Our simulations confirm the asymmetric behavior of the two monomers, the stronger interaction of NAA than IAA and offers insight into the possible mechanism of ABP1 as an auxin receptor. 1-naphthaleneacetic acid 97-100 auxin-binding protein 1 Zea mays 160-164 25046756-0 2014 Effect of 1-naphthaleneacetic acid on organic acid exudation by the roots of white lupin plants grown under phosphorus-deficient conditions. 1-naphthaleneacetic acid 10-34 5'-nucleotidase, cytosolic IIIA Homo sapiens 83-88 25046756-1 2014 The effect of NAA (1-naphthaleneacetic acid) on organic acid exudation in white lupin plants grown under phosphorus deficiency was investigated. 1-naphthaleneacetic acid 14-17 5'-nucleotidase, cytosolic IIIA Homo sapiens 80-85 25046756-1 2014 The effect of NAA (1-naphthaleneacetic acid) on organic acid exudation in white lupin plants grown under phosphorus deficiency was investigated. 1-naphthaleneacetic acid 19-43 5'-nucleotidase, cytosolic IIIA Homo sapiens 80-85 25046756-4 2014 The exogenous addition of NAA led to an increase in organic acid exudation, but this response was not proportional to the concentration of the dose applied, noticing the largest increments with NAA 10(-8)M. In contrast the increase in root weight was proportional to the dose applied, which shows that with higher doses the roots produced are not of proteoid type. 1-naphthaleneacetic acid 26-29 N-alpha-acetyltransferase 10, NatA catalytic subunit Homo sapiens 194-200 25056921-6 2014 RD20 also confers tolerance against stress induced by Paraquat, Rose Bengal, heavy metal, and the synthetic auxins 1-naphthaleneacetic acid and 2,4-dichlorophenoxyacetic acid. 1-naphthaleneacetic acid 115-139 Caleosin-related family protein Arabidopsis thaliana 0-4 24800738-2 2014 1-Naphtoxyacetic acid (1-NOA), an analog of the synthetic auxin 1-N-naphtalene acetic acid (NAA), inhibits the IAA influx carrier AUX1. 1-naphthaleneacetic acid 92-95 Transmembrane amino acid transporter family protein Arabidopsis thaliana 130-134 25255415-11 2014 When accounting for the other variables, the association between NAA and RLS prevalence was statistically significant (P = 0.002). 1-naphthaleneacetic acid 65-68 RLS1 Homo sapiens 73-76 25255415-14 2014 We found the association between NAA race and RLS prevalence to be statistically significant. 1-naphthaleneacetic acid 33-36 RLS1 Homo sapiens 46-49 24887377-4 2014 In the present study, we incorporated an alkyne-bearing NAA into an enzyme, murine dihydrofolate reductase (mDHFR), in high cell density cultivation of Escherichia coli, and performed CuAAC conjugation with fluorescent azide dyes to evaluate enzyme compatibility of various CuAAC conditions comprising combination of commercially available Cu(I)-chelating ligands and reductants. 1-naphthaleneacetic acid 56-59 dihydrofolate reductase Mus musculus 108-113 24625793-6 2014 AbetaPP-PS1 mice exhibit a significant reduction in the level of NAA and increase in level of myo-inositol. 1-naphthaleneacetic acid 65-68 presenilin 1 Mus musculus 8-11 24366686-3 2014 Herein, we revealed that exogenous auxin, 1-naphthylacetic acid (NAA), could simultaneously stimulate Arabidopsis HO-1 (HY1) gene expression and H2O2 generation. 1-naphthaleneacetic acid 42-63 Plant heme oxygenase (decyclizing) family protein Arabidopsis thaliana 120-123 24366686-3 2014 Herein, we revealed that exogenous auxin, 1-naphthylacetic acid (NAA), could simultaneously stimulate Arabidopsis HO-1 (HY1) gene expression and H2O2 generation. 1-naphthaleneacetic acid 65-68 Plant heme oxygenase (decyclizing) family protein Arabidopsis thaliana 120-123 24366686-5 2014 NAA-induced HY1 expression is dependent on H2O2. 1-naphthaleneacetic acid 0-3 Plant heme oxygenase (decyclizing) family protein Arabidopsis thaliana 12-15 24366686-6 2014 This conclusion was supported by analyzing the removal of H2O2 with ascorbic acid (AsA) and dimethylthiourea (DMTU), both of which could block NAA-induced HY1 expression and LR formation. 1-naphthaleneacetic acid 143-146 Plant heme oxygenase (decyclizing) family protein Arabidopsis thaliana 155-158 24366686-8 2014 Simultaneously, HY1 is required for NAA-mediated H2O2 generation, since Znpp inhibition of HY1 blocked the NAA-induced H2O2 production and LR formation. 1-naphthaleneacetic acid 36-39 Plant heme oxygenase (decyclizing) family protein Arabidopsis thaliana 16-19 24366686-8 2014 Simultaneously, HY1 is required for NAA-mediated H2O2 generation, since Znpp inhibition of HY1 blocked the NAA-induced H2O2 production and LR formation. 1-naphthaleneacetic acid 107-110 Plant heme oxygenase (decyclizing) family protein Arabidopsis thaliana 16-19 24366686-8 2014 Simultaneously, HY1 is required for NAA-mediated H2O2 generation, since Znpp inhibition of HY1 blocked the NAA-induced H2O2 production and LR formation. 1-naphthaleneacetic acid 107-110 Plant heme oxygenase (decyclizing) family protein Arabidopsis thaliana 91-94 24366686-9 2014 Genetic data demonstrated that hy1-100 was significantly impaired in H2O2 production and LR formation in response to NAA, compared with wild-type plants. 1-naphthaleneacetic acid 117-120 Plant heme oxygenase (decyclizing) family protein Arabidopsis thaliana 31-34 24306962-1 2014 BACKGROUND: Whether Native American ancestry (NAA) is associated with COPD or lung function in a racially admixed Hispanic population is unknown. 1-naphthaleneacetic acid 46-49 COPD Homo sapiens 70-74 24306962-9 2014 After adjustment for current smoking and other covariates, NAA was inversely associated with COPD (OR per 10% increment, 0.55; 95% CI, 0.41-0.75) but positively associated with FEV1, FVC, and FEV1/FVC. 1-naphthaleneacetic acid 59-62 COPD Homo sapiens 93-97 24306962-10 2014 After additional adjustment for pack-years of smoking, the association between NAA and COPD or lung function measures was slightly attenuated. 1-naphthaleneacetic acid 79-82 COPD Homo sapiens 87-91 24306962-11 2014 We found that about 31% of the estimated effect of NAA on COPD is mediated by pack-years of smoking. 1-naphthaleneacetic acid 51-54 COPD Homo sapiens 58-62 24306962-12 2014 CONCLUSIONS: NAA is inversely associated with COPD but positively associated with FEV1 or FVC in Costa Ricans. 1-naphthaleneacetic acid 13-16 COPD Homo sapiens 46-50 24164597-7 2014 Moreover, the rel3 mutant was more resistant than wild-type to 1-naphthaleneacetic acid (NAA) and N-1-naphthylphthalamic acid (NPA) in root growth, and exhibited insensitivity to auxins but greater sensitivity to auxin transport inhibitors during nodulation. 1-naphthaleneacetic acid 63-87 REL3 Lotus japonicus 14-18 24164597-7 2014 Moreover, the rel3 mutant was more resistant than wild-type to 1-naphthaleneacetic acid (NAA) and N-1-naphthylphthalamic acid (NPA) in root growth, and exhibited insensitivity to auxins but greater sensitivity to auxin transport inhibitors during nodulation. 1-naphthaleneacetic acid 89-92 REL3 Lotus japonicus 14-18 23562458-3 2013 However, the catalytic enzyme glutamate carboxypeptidase II (GCP II) rapidly hydrolyzes NAAG into NAA and glutamate. 1-naphthaleneacetic acid 88-91 folate hydrolase 1 Rattus norvegicus 30-59 23562458-3 2013 However, the catalytic enzyme glutamate carboxypeptidase II (GCP II) rapidly hydrolyzes NAAG into NAA and glutamate. 1-naphthaleneacetic acid 88-91 folate hydrolase 1 Rattus norvegicus 61-67 23562458-4 2013 Inhibition of the GCP II enzyme with NAAG peptidase inhibitors reduces the concentration of glutamate both by increasing the duration of NAAG activity on mGluR3 and by reducing degradation into NAA and glutamate resulting in reduced cell death in models of TBI and TBI with hypoxia. 1-naphthaleneacetic acid 37-40 folate hydrolase 1 Rattus norvegicus 18-24 23562458-4 2013 Inhibition of the GCP II enzyme with NAAG peptidase inhibitors reduces the concentration of glutamate both by increasing the duration of NAAG activity on mGluR3 and by reducing degradation into NAA and glutamate resulting in reduced cell death in models of TBI and TBI with hypoxia. 1-naphthaleneacetic acid 37-40 glutamate receptor, ionotropic, AMPA3 (alpha 3) Mus musculus 154-160 23503757-5 2013 Stress experiments showed that the AtRDR1 promoter has a broad-spectrum response to various stresses and is sensitive to 1-naphthaleneacetic acid, abscisic acid, and salicylic acid. 1-naphthaleneacetic acid 121-145 RNA-dependent RNA polymerase 1 Arabidopsis thaliana 35-41 23289750-9 2013 Similar to previous findings in an Arabidopsis thaliana SGT1 mutant (atsgt1b), degradation of AUX/IAA proteins was retarded in cyp2 mutants treated with exogenous 1-naphthylacetic acid. 1-naphthaleneacetic acid 163-184 UDP-glucosyltransferase 74F2 Arabidopsis thaliana 56-60 22540348-10 2012 Activation of the auxin-regulated synthetic DR5 promoter and of auxin response genes was strongly repressed in seedlings overexpressing PIN8 when exposed to 1-naphthalene acetic acid. 1-naphthaleneacetic acid 157-182 Auxin efflux carrier family protein Arabidopsis thaliana 136-140 23293348-6 2013 Following treatment with the synthetic auxin 1-naphthaleneacetic acid (NAA), DR5:GUS expression in aux1(rcr1) and aux1-T occurred mainly in the root apex and mature zone. 1-naphthaleneacetic acid 45-69 Transmembrane amino acid transporter family protein Arabidopsis thaliana 99-103 23293348-6 2013 Following treatment with the synthetic auxin 1-naphthaleneacetic acid (NAA), DR5:GUS expression in aux1(rcr1) and aux1-T occurred mainly in the root apex and mature zone. 1-naphthaleneacetic acid 45-69 Transmembrane amino acid transporter family protein Arabidopsis thaliana 114-118 23293348-6 2013 Following treatment with the synthetic auxin 1-naphthaleneacetic acid (NAA), DR5:GUS expression in aux1(rcr1) and aux1-T occurred mainly in the root apex and mature zone. 1-naphthaleneacetic acid 71-74 Transmembrane amino acid transporter family protein Arabidopsis thaliana 99-103 23293348-7 2013 NAA-induced DR5:GUS expression in the root apex was markedly prevented by ethylene in genotypes with aux1(rcr1) but not in aux1-T genotypes and the wild type. 1-naphthaleneacetic acid 0-3 Transmembrane amino acid transporter family protein Arabidopsis thaliana 101-105 23293348-7 2013 NAA-induced DR5:GUS expression in the root apex was markedly prevented by ethylene in genotypes with aux1(rcr1) but not in aux1-T genotypes and the wild type. 1-naphthaleneacetic acid 0-3 Transmembrane amino acid transporter family protein Arabidopsis thaliana 123-127 23250625-4 2013 Here, we report the identification and characterization of an Arabidopsis (Arabidopsis thaliana) leaf senescence-associated gene named SAG201, which is highly up-regulated during leaf senescence and can be induced by 1-naphthaleneacetic acid, a synthetic auxin. 1-naphthaleneacetic acid 217-241 SAUR-like auxin-responsive protein family Arabidopsis thaliana 135-141 23622383-11 2013 An increased concentration of NAA in the urine is sufficient to diagnose Canavan disease, which is due to mutations of the ASPA gene. 1-naphthaleneacetic acid 30-33 aspartoacylase Homo sapiens 123-127 22750589-4 2012 However, extracellular NAAG is rapidly converted into NAA and glutamate by the catalytic enzyme glutamate carboxypeptidase II (GCPII) reducing presynaptic inhibition. 1-naphthaleneacetic acid 23-26 folate hydrolase 1 Homo sapiens 96-125 22750589-4 2012 However, extracellular NAAG is rapidly converted into NAA and glutamate by the catalytic enzyme glutamate carboxypeptidase II (GCPII) reducing presynaptic inhibition. 1-naphthaleneacetic acid 23-26 folate hydrolase 1 Homo sapiens 127-132 22849963-7 2012 IP-10, MCP-4, and MIP-1beta were significantly associated with CMRs in the right basal ganglia with (1) lower concentrations of IP-10 correlating with higher N-acetyl aspartate to creatine ratios (NAA/Cr) and (2) higher concentrations of MCP-4 and MIP-1beta correlating with higher myoinositol to creatine (mI/Cr) ratios. 1-naphthaleneacetic acid 197-200 C-X-C motif chemokine ligand 10 Homo sapiens 128-133 22722629-5 2012 RESULTS: Compared to controls, NAA was significantly reduced in the putamen (p = 0.012) and in the midbrain of GBA-PD (p = 0.05). 1-naphthaleneacetic acid 31-34 glucosylceramidase beta Homo sapiens 111-114 22284711-4 2012 We further discovered that N-1-naphthylacetic acid (NAA), haemin, and CO aqueous solution, led to the induction of ZmHO-1 expression as well as the thereafter promotion of lateral root development. 1-naphthaleneacetic acid 52-55 HO-1 Zea mays 115-121 22381655-6 2012 Real-time RT-PCR analysis revealed that TaEXPB23 transcript expression was upregulated by exogenous methyl jasmonate (MeJA) and salt stress, but downregulated by exogenous gibberellins (GA3), ethylene (ET), indole-3-acetic acid (IAA) and alpha-naphthlcetic acid (NAA). 1-naphthaleneacetic acid 263-266 EXPB23 Triticum aestivum 40-48 22407922-5 2012 We found that the expression of NAA in MRL-lpr mice prevented proteinuria and reduced kidney immune complex formation. 1-naphthaleneacetic acid 32-35 Fas (TNF receptor superfamily member 6) Mus musculus 43-46 22407922-7 2012 Administration of the IgM NAA to MRL-lpr mice also delayed the onset of nephritis. 1-naphthaleneacetic acid 26-29 Fas (TNF receptor superfamily member 6) Mus musculus 37-40 22407922-11 2012 Furthermore, the NAA B cells produced large amounts of IL-10 upon TLR stimulation. 1-naphthaleneacetic acid 17-20 interleukin 10 Mus musculus 55-60 21673093-4 2011 Furthermore, the application of lower doses of NaCl (10 mM) and polyethylene glycol (PEG) (2%) mimicked the inducible effects of naphthylacetic acid and the HO-1 inducer haemin on the up-regulation of BnHO1 and subsequent lateral root (LR) formation. 1-naphthaleneacetic acid 129-148 heme oxygenase 1, chloroplastic-like Brassica napus 201-206 21679158-5 2012 Semi-quantitative RT-PCR (reverse transcription-PCR) showed that GhRDR6 was up-regulated by the application of various phytohormones, including MeJA [methyl JA (jasmonate)], ABA (abscisic acid), JA, alpha-naphthylacetic acid, gibberellins and ET (ethylene). 1-naphthaleneacetic acid 199-224 RNA-dependent RNA polymerase 6-like Gossypium hirsutum 65-71 22304711-8 2012 GCPII (glutamate carboxypeptidase II) cleaves the prevalent neuropeptide NAAG into NAA and glutamate and there is widespread evidence and belief that targeting the glutamate derived from this enzymatic action may be a promising therapeutic route. 1-naphthaleneacetic acid 73-76 folate hydrolase 1 Homo sapiens 0-5 22304711-8 2012 GCPII (glutamate carboxypeptidase II) cleaves the prevalent neuropeptide NAAG into NAA and glutamate and there is widespread evidence and belief that targeting the glutamate derived from this enzymatic action may be a promising therapeutic route. 1-naphthaleneacetic acid 73-76 folate hydrolase 1 Homo sapiens 7-36 23118909-8 2012 The NAA/Cr and Cho/Cr were lower in MSA-C or SCA2 comparing with SCA3 or SCA6. 1-naphthaleneacetic acid 4-7 ataxin 2 Homo sapiens 45-49 23118909-10 2012 The cerebellar NAA/Cho was lower in SCA2 than SCA1, SCA3 or SCA6 (p<0.04). 1-naphthaleneacetic acid 15-18 ataxin 2 Homo sapiens 36-40 23118909-10 2012 The cerebellar NAA/Cho was lower in SCA2 than SCA1, SCA3 or SCA6 (p<0.04). 1-naphthaleneacetic acid 15-18 ataxin 1 Homo sapiens 46-50 23118909-12 2012 In the early stages of diseases (SARA score<10), significant lower NAA/Cr and NAA/Cho in SCA2, SCA3, SCA6 or MSA-C were observed comparing with healthy controls (p<0.017). 1-naphthaleneacetic acid 67-70 ataxin 2 Homo sapiens 89-93 23118909-12 2012 In the early stages of diseases (SARA score<10), significant lower NAA/Cr and NAA/Cho in SCA2, SCA3, SCA6 or MSA-C were observed comparing with healthy controls (p<0.017). 1-naphthaleneacetic acid 78-81 ataxin 2 Homo sapiens 89-93 23118909-14 2012 Patients with MSA-C and SCA2 had lower NAA/Cr and Cho/Cr than SCA3 or SCA6 (p<0.016). 1-naphthaleneacetic acid 39-42 ataxin 2 Homo sapiens 24-28 21224416-4 2011 The highest fidelity NAA systems derived from the Methanocaldococcus jannaschii tyrosyl AARS feature specific mutations to two residues reported to interact with the hydroxyl group of the substrate tyrosine. 1-naphthaleneacetic acid 21-24 alanyl-tRNA synthetase 1 Homo sapiens 88-92 21477123-9 2011 Both elevated CO(2) and NAA application increased expressions of caprice, triptychon and rho-related protein from plants 2, and decreased expressions of werewolf, GLABRA2, GLABRA3 and the transparent testa glabra 1, genes related to root-hair development, while 1-NOA and NPA application had an opposite effect. 1-naphthaleneacetic acid 24-27 HD-ZIP IV family of homeobox-leucine zipper protein with lipid-binding START domain-containing protein Arabidopsis thaliana 163-170 21477123-9 2011 Both elevated CO(2) and NAA application increased expressions of caprice, triptychon and rho-related protein from plants 2, and decreased expressions of werewolf, GLABRA2, GLABRA3 and the transparent testa glabra 1, genes related to root-hair development, while 1-NOA and NPA application had an opposite effect. 1-naphthaleneacetic acid 24-27 basic helix-loop-helix (bHLH) DNA-binding superfamily protein Arabidopsis thaliana 172-179 21477123-9 2011 Both elevated CO(2) and NAA application increased expressions of caprice, triptychon and rho-related protein from plants 2, and decreased expressions of werewolf, GLABRA2, GLABRA3 and the transparent testa glabra 1, genes related to root-hair development, while 1-NOA and NPA application had an opposite effect. 1-naphthaleneacetic acid 24-27 myb domain protein 0 Arabidopsis thaliana 206-214 21459102-6 2011 Adult visceral obesity and insulin level correlated with right>left shift in hippocampal NAA concentrations, controlling for age and denominator. 1-naphthaleneacetic acid 92-95 insulin Cricetulus griseus 27-34 21372002-2 2011 In the current study, the immune characteristics of MPO-NAA and MPO-ANCA were examined and compared with the aim to investigate the pathogenesis of MPO-ANCA. 1-naphthaleneacetic acid 56-59 myeloperoxidase Homo sapiens 52-55 21372002-7 2011 The MPO-NAA-induced production of reactive oxygen species was assessed using oxidation of dihydrorhodamine (DHR) to rhodamine. 1-naphthaleneacetic acid 8-11 myeloperoxidase Homo sapiens 4-7 21372002-8 2011 RESULTS: MPO-NAA recognized epitope(s) in the heavy chains of MPO with conformation-dependent structure, the same as MPO-ANCA. 1-naphthaleneacetic acid 13-16 myeloperoxidase Homo sapiens 9-12 21372002-8 2011 RESULTS: MPO-NAA recognized epitope(s) in the heavy chains of MPO with conformation-dependent structure, the same as MPO-ANCA. 1-naphthaleneacetic acid 13-16 myeloperoxidase Homo sapiens 62-65 21372002-8 2011 RESULTS: MPO-NAA recognized epitope(s) in the heavy chains of MPO with conformation-dependent structure, the same as MPO-ANCA. 1-naphthaleneacetic acid 13-16 myeloperoxidase Homo sapiens 62-65 21372002-9 2011 The median titre of MPO-NAA was lower than that of MPO-ANCA (1 : 40 vs 1 : 4800, P < 0.001). 1-naphthaleneacetic acid 24-27 myeloperoxidase Homo sapiens 20-23 21372002-10 2011 The median avidity of MPO-NAA was lower than that of MPO-ANCA (2.2 x 10(7) vs 8.7 x 10(7)/M, P = 0.014). 1-naphthaleneacetic acid 26-29 myeloperoxidase Homo sapiens 22-25 21372002-11 2011 The IgG subclasses of MPO-NAA were mainly restricted to IgG1 (100%) and lack of IgG3. 1-naphthaleneacetic acid 26-29 myeloperoxidase Homo sapiens 22-25 21372002-11 2011 The IgG subclasses of MPO-NAA were mainly restricted to IgG1 (100%) and lack of IgG3. 1-naphthaleneacetic acid 26-29 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 80-84 21372002-12 2011 The inhibition effect on the binding between ceruloplasmin and MPO was lower for MPO-NAA than MPO-ANCA (P = 0.046). 1-naphthaleneacetic acid 85-88 myeloperoxidase Homo sapiens 63-66 21372002-12 2011 The inhibition effect on the binding between ceruloplasmin and MPO was lower for MPO-NAA than MPO-ANCA (P = 0.046). 1-naphthaleneacetic acid 85-88 myeloperoxidase Homo sapiens 81-84 21372002-12 2011 The inhibition effect on the binding between ceruloplasmin and MPO was lower for MPO-NAA than MPO-ANCA (P = 0.046). 1-naphthaleneacetic acid 85-88 myeloperoxidase Homo sapiens 81-84 21372002-13 2011 The MPO-NAA-induced respiratory burst of neutrophils was significantly weaker than that of MPO-ANCA (P = 0.036). 1-naphthaleneacetic acid 8-11 myeloperoxidase Homo sapiens 4-7 21372002-14 2011 CONCLUSION: The lower titre, lower avidity and lack of IgG3 subclass compared with MPO-ANCA may contribute to the non-pathogenic co-existence of MPO-NAA with MPO in serum. 1-naphthaleneacetic acid 149-152 myeloperoxidase Homo sapiens 145-148 21372002-14 2011 CONCLUSION: The lower titre, lower avidity and lack of IgG3 subclass compared with MPO-ANCA may contribute to the non-pathogenic co-existence of MPO-NAA with MPO in serum. 1-naphthaleneacetic acid 149-152 myeloperoxidase Homo sapiens 145-148 21502189-7 2011 Quantification of auxin export from mesophyll protoplasts revealed that naphthalene-1-acetic acid but not indole-3-acetic acid transport is affected by the rol1-2 mutation. 1-naphthaleneacetic acid 72-97 rhamnose biosynthesis 1 Arabidopsis thaliana 156-162 21502189-8 2011 Inhibition of flavonol biosynthesis in rol1-2 fls1-3 restores naphthalene-1-acetic acid transport to wild-type levels, indicating a very specific mode of action of flavonols on the auxin transport machinery. 1-naphthaleneacetic acid 62-87 rhamnose biosynthesis 1 Arabidopsis thaliana 39-45 21502189-8 2011 Inhibition of flavonol biosynthesis in rol1-2 fls1-3 restores naphthalene-1-acetic acid transport to wild-type levels, indicating a very specific mode of action of flavonols on the auxin transport machinery. 1-naphthaleneacetic acid 62-87 flavonol synthase 1 Arabidopsis thaliana 46-50 21494695-8 2011 There was an inverse correlation between brain NAA/Cr (neuronal injury) and circulating CD14+CD16+ and CD14(lo)CD16+ monocytes. 1-naphthaleneacetic acid 47-50 CD14 molecule Homo sapiens 88-92 21494695-8 2011 There was an inverse correlation between brain NAA/Cr (neuronal injury) and circulating CD14+CD16+ and CD14(lo)CD16+ monocytes. 1-naphthaleneacetic acid 47-50 Fc gamma receptor IIIa Homo sapiens 93-97 21494695-8 2011 There was an inverse correlation between brain NAA/Cr (neuronal injury) and circulating CD14+CD16+ and CD14(lo)CD16+ monocytes. 1-naphthaleneacetic acid 47-50 CD14 molecule Homo sapiens 103-107 21494695-8 2011 There was an inverse correlation between brain NAA/Cr (neuronal injury) and circulating CD14+CD16+ and CD14(lo)CD16+ monocytes. 1-naphthaleneacetic acid 47-50 Fc gamma receptor IIIa Homo sapiens 111-115 21412048-3 2011 Although the conditional FAB1A/B knockdown mutant revealed an auxin-resistant phenotype to a membrane impermeable auxin, 2,4-dichlorophenoxyacetic acid (2,4-D), the mutant did not exhibit this phenotype to a membrane-permeable artificial auxin, naphthalene 1-acetic acid (NAA). 1-naphthaleneacetic acid 245-270 1-phosphatidylinositol-3-phosphate 5-kinase FAB1A Arabidopsis thaliana 25-32 21412048-3 2011 Although the conditional FAB1A/B knockdown mutant revealed an auxin-resistant phenotype to a membrane impermeable auxin, 2,4-dichlorophenoxyacetic acid (2,4-D), the mutant did not exhibit this phenotype to a membrane-permeable artificial auxin, naphthalene 1-acetic acid (NAA). 1-naphthaleneacetic acid 272-275 1-phosphatidylinositol-3-phosphate 5-kinase FAB1A Arabidopsis thaliana 25-32 20709516-6 2010 NAA and AA patients showed higher IFN-gamma and EDN levels compared to AC and NC, with no differences in IL-4, IL-5, or IL-13 levels among the four groups. 1-naphthaleneacetic acid 0-3 interferon gamma Homo sapiens 34-43 20709516-6 2010 NAA and AA patients showed higher IFN-gamma and EDN levels compared to AC and NC, with no differences in IL-4, IL-5, or IL-13 levels among the four groups. 1-naphthaleneacetic acid 0-3 ribonuclease A family member 2 Homo sapiens 48-51 20709516-8 2010 In NAA, AA, and AC patients, % eosinophils and EDN levels correlated positively with IFN-gamma, GM-CSF, eotaxin, and RANTES, but not with IL-5 levels. 1-naphthaleneacetic acid 3-6 ribonuclease A family member 2 Homo sapiens 47-50 19655147-6 2009 Furthermore, the NAA-response region was found to be located in the -1,482/-1204 fragment, while the element(s) for the sucrose-responsive expression may be present in the -247/-1 region in the GhCesA4 promoter. 1-naphthaleneacetic acid 17-20 cellulose synthase A catalytic subunit 8 [UDP-forming] Gossypium hirsutum 194-201 19217667-6 2009 Anti-MUC1 IgM was found in 5/47 (10.6%) of normals, 5/45 (11.1%) of NAA, 7/47 (14.9%) of AA, and 4/20 (20.0%) of CRC (p=0.70). 1-naphthaleneacetic acid 68-71 mucin 1, cell surface associated Homo sapiens 5-9 19217667-7 2009 The prevalence of anti-MUC1 IgG was 8/47 (17.0%) of normals, 14/45 (31.1%) of NAA, 14/47 (29.8%) of AA, and 6/20 (30.0%) of CRC (p=0.36). 1-naphthaleneacetic acid 78-81 mucin 1, cell surface associated Homo sapiens 23-27 19217667-9 2009 However, in an exploratory analysis, the median normalized anti-MUC1 IgG OD level of the combined abnormal groups (NAA, AA, CRC) was significantly higher than the normals (0.045 OD vs. 0.030 OD; p=0.017). 1-naphthaleneacetic acid 115-118 mucin 1, cell surface associated Homo sapiens 64-68 19442440-3 2009 Owing to the specific fiber-like morphology and chemical composition, zeolite-like calcosilicate CAS-1 can effectively intercept the particles and thus reduces the TSNA level of mainstream smoke in the range of 30-60% once it is added into the cigarette filter, which is superior to zeolite NaA additive with the same amount. 1-naphthaleneacetic acid 291-294 cycloartenol Synthase-like Nicotiana tabacum 97-102 19061772-12 2008 In the 23 responders, the rCBF after challenge were greater than 20 mL/min per 100 g (P=.008), had a significantly better CRC in the anterior CSWM than the nonresponders (1.40 vs 1.06), and had normal NAA/Cre ratio in the anterior, middle, and posterior CSWM in MRSI study. 1-naphthaleneacetic acid 201-204 CCAAT/enhancer binding protein zeta Rattus norvegicus 26-30 19236511-6 2009 Cyclin D1, p21 and cyclin A were overexpressed in 58.6%, 51.7% and 31.8% of the NAA cases, respectively, whereas 81.3%, 62.5% and 41.7% of the AAA cases showed overexpression of cyclin D1, p21 and cyclin A, respectively. 1-naphthaleneacetic acid 80-83 cyclin D1 Homo sapiens 0-9 19236511-6 2009 Cyclin D1, p21 and cyclin A were overexpressed in 58.6%, 51.7% and 31.8% of the NAA cases, respectively, whereas 81.3%, 62.5% and 41.7% of the AAA cases showed overexpression of cyclin D1, p21 and cyclin A, respectively. 1-naphthaleneacetic acid 80-83 cyclin dependent kinase inhibitor 1A Homo sapiens 11-14 19236511-6 2009 Cyclin D1, p21 and cyclin A were overexpressed in 58.6%, 51.7% and 31.8% of the NAA cases, respectively, whereas 81.3%, 62.5% and 41.7% of the AAA cases showed overexpression of cyclin D1, p21 and cyclin A, respectively. 1-naphthaleneacetic acid 80-83 cyclin A2 Homo sapiens 19-27 17852346-9 2008 Furthermore, the levels of rice PIG-F transcription were up-regulated by growth hormones including GA(3), NAA and kinetin. 1-naphthaleneacetic acid 106-109 phosphatidylinositol glycan anchor biosynthesis class F Sus scrofa 32-37 24256959-4 2008 While single voxel proton spectroscopy (MRS) of the basal ganglia at the beginning was normal, the follow-up MRS showed moderate to severe NAA and mI decrease. 1-naphthaleneacetic acid 139-142 MROS Homo sapiens 109-112 18069944-10 2008 Accordingly, treatments with sucrose, gibberellic acid and NAA induced upregulation of AtCel2, whereas ABA triggered downregulation of both AtCel2 and KOR3 in roots. 1-naphthaleneacetic acid 59-62 cellulase 2 Arabidopsis thaliana 87-93 18069944-10 2008 Accordingly, treatments with sucrose, gibberellic acid and NAA induced upregulation of AtCel2, whereas ABA triggered downregulation of both AtCel2 and KOR3 in roots. 1-naphthaleneacetic acid 59-62 cellulase 2 Arabidopsis thaliana 140-146 18069944-10 2008 Accordingly, treatments with sucrose, gibberellic acid and NAA induced upregulation of AtCel2, whereas ABA triggered downregulation of both AtCel2 and KOR3 in roots. 1-naphthaleneacetic acid 59-62 glycosyl hydrolase 9A3 Arabidopsis thaliana 151-155 16337204-3 2005 NtPDR3 was found to be induced in suspension cells treated with methyl jasmonate, salicylic acid, 1-naphthalene acetic acid, or cembrene, a macrocyclic diterpene. 1-naphthaleneacetic acid 98-123 pleiotropic drug resistance protein 3 Nicotiana tabacum 0-6 17337532-7 2007 Addition of the polarly transported auxins indole-3-acetic acid and 1-naphthyl acetic acid (but not 2,4-dichlorophenoxyacetic acid) restored both the normal organization of actin and the synchrony of cell division. 1-naphthaleneacetic acid 68-90 actin Nicotiana tabacum 173-178 16525894-5 2006 It was found that CEG was expressed abundantly in vascular tissues of the primary root, but not in newly formed lateral root primordia and the root meristem, and induced by exogenous auxin NAA (alpha-naphthalene acetic acid). 1-naphthaleneacetic acid 189-192 F-box and associated interaction domains-containing protein Arabidopsis thaliana 18-21 16525894-5 2006 It was found that CEG was expressed abundantly in vascular tissues of the primary root, but not in newly formed lateral root primordia and the root meristem, and induced by exogenous auxin NAA (alpha-naphthalene acetic acid). 1-naphthaleneacetic acid 194-223 F-box and associated interaction domains-containing protein Arabidopsis thaliana 18-21 16525894-6 2006 In addition, the ceg mutant was hyposensitive to NAA, IAA (indole-3-acetic acid) and 2,4-D (2,4-dichlorophenoxyacetic acid), as well as the auxin transport inhibitor TIBA (3,3,5-triiodobenzoic acid), showing that CEG is an auxin-inducible gene. 1-naphthaleneacetic acid 49-52 F-box and associated interaction domains-containing protein Arabidopsis thaliana 17-20 16212599-6 2005 Expression of AtPGP1 in yeast and in the standard mammalian expression system used to analyze human MDR-type proteins results in enhanced efflux of indole-3-acetic acid (IAA) and the synthetic auxin 1-naphthalene acetic acid (1-NAA), but not the inactive auxin 2-NAA. 1-naphthaleneacetic acid 199-224 phosphatidylglycerolphosphate synthase 1 Arabidopsis thaliana 14-20 16359384-6 2005 In addition to the salt induction, the AtNAC2 can also be induced by abscisic acid (ABA), ACC and NAA. 1-naphthaleneacetic acid 98-101 NAC domain containing protein 2 Arabidopsis thaliana 39-45 16212599-6 2005 Expression of AtPGP1 in yeast and in the standard mammalian expression system used to analyze human MDR-type proteins results in enhanced efflux of indole-3-acetic acid (IAA) and the synthetic auxin 1-naphthalene acetic acid (1-NAA), but not the inactive auxin 2-NAA. 1-naphthaleneacetic acid 226-231 phosphatidylglycerolphosphate synthase 1 Arabidopsis thaliana 14-20 16463701-15 2005 In case of all the 1H MRS spectra obtained from the subcortical nuclei regions significant decrease in the NAA/tCr ratio was observed which could reflect the reduced concentration of N-acetyloaspartate, known as a neurons marker. 1-naphthaleneacetic acid 107-110 T cell receptor beta variable 20/OR9-2 (non-functional) Homo sapiens 111-114 16100961-3 2005 RESULTS: Statistically significant decrease (p < 0.01) only of Naa/Cr--at HCR among the CDR0, CDR1+CDR2, and CDR3, and at PCA between CDR0 and CDR1+CDR2 in relation to CDR3. 1-naphthaleneacetic acid 63-66 coiled-coil alpha-helical rod protein 1 Homo sapiens 74-77 16100961-3 2005 RESULTS: Statistically significant decrease (p < 0.01) only of Naa/Cr--at HCR among the CDR0, CDR1+CDR2, and CDR3, and at PCA between CDR0 and CDR1+CDR2 in relation to CDR3. 1-naphthaleneacetic acid 63-66 cerebellar degeneration related protein 1 Homo sapiens 94-98 16100961-3 2005 RESULTS: Statistically significant decrease (p < 0.01) only of Naa/Cr--at HCR among the CDR0, CDR1+CDR2, and CDR3, and at PCA between CDR0 and CDR1+CDR2 in relation to CDR3. 1-naphthaleneacetic acid 63-66 cerebellar degeneration related protein 2 Homo sapiens 99-103 16100961-3 2005 RESULTS: Statistically significant decrease (p < 0.01) only of Naa/Cr--at HCR among the CDR0, CDR1+CDR2, and CDR3, and at PCA between CDR0 and CDR1+CDR2 in relation to CDR3. 1-naphthaleneacetic acid 63-66 CDR3 Homo sapiens 109-113 16100961-3 2005 RESULTS: Statistically significant decrease (p < 0.01) only of Naa/Cr--at HCR among the CDR0, CDR1+CDR2, and CDR3, and at PCA between CDR0 and CDR1+CDR2 in relation to CDR3. 1-naphthaleneacetic acid 63-66 cerebellar degeneration related protein 1 Homo sapiens 143-147 16100961-3 2005 RESULTS: Statistically significant decrease (p < 0.01) only of Naa/Cr--at HCR among the CDR0, CDR1+CDR2, and CDR3, and at PCA between CDR0 and CDR1+CDR2 in relation to CDR3. 1-naphthaleneacetic acid 63-66 cerebellar degeneration related protein 2 Homo sapiens 148-152 16100961-3 2005 RESULTS: Statistically significant decrease (p < 0.01) only of Naa/Cr--at HCR among the CDR0, CDR1+CDR2, and CDR3, and at PCA between CDR0 and CDR1+CDR2 in relation to CDR3. 1-naphthaleneacetic acid 63-66 CDR3 Homo sapiens 168-172 16100961-4 2005 CONCLUSION: The HCR is the first to show Naa reduction (CDR1). 1-naphthaleneacetic acid 41-44 coiled-coil alpha-helical rod protein 1 Homo sapiens 16-19 16100961-4 2005 CONCLUSION: The HCR is the first to show Naa reduction (CDR1). 1-naphthaleneacetic acid 41-44 cerebellar degeneration related protein 1 Homo sapiens 56-60 15908594-4 2005 In addition, we showed that the atmdr1-100 and atmdr1-100/atpgp1-100 mutants are defective in multiple aspects of root development, including increased root-growth sensitivity to 1-naphthalene acetic acid (1-NAA), and decreased sensitivity to naphthylphthalamic acid (NPA)-mediated inhibition of root elongation. 1-naphthaleneacetic acid 179-204 ATP binding cassette subfamily B19 Arabidopsis thaliana 32-38 15908594-4 2005 In addition, we showed that the atmdr1-100 and atmdr1-100/atpgp1-100 mutants are defective in multiple aspects of root development, including increased root-growth sensitivity to 1-naphthalene acetic acid (1-NAA), and decreased sensitivity to naphthylphthalamic acid (NPA)-mediated inhibition of root elongation. 1-naphthaleneacetic acid 179-204 ATP binding cassette subfamily B19 Arabidopsis thaliana 47-53 15908594-4 2005 In addition, we showed that the atmdr1-100 and atmdr1-100/atpgp1-100 mutants are defective in multiple aspects of root development, including increased root-growth sensitivity to 1-naphthalene acetic acid (1-NAA), and decreased sensitivity to naphthylphthalamic acid (NPA)-mediated inhibition of root elongation. 1-naphthaleneacetic acid 179-204 ATP binding cassette subfamily B1 Arabidopsis thaliana 58-64 15908594-4 2005 In addition, we showed that the atmdr1-100 and atmdr1-100/atpgp1-100 mutants are defective in multiple aspects of root development, including increased root-growth sensitivity to 1-naphthalene acetic acid (1-NAA), and decreased sensitivity to naphthylphthalamic acid (NPA)-mediated inhibition of root elongation. 1-naphthaleneacetic acid 206-211 ATP binding cassette subfamily B19 Arabidopsis thaliana 32-38 15908594-4 2005 In addition, we showed that the atmdr1-100 and atmdr1-100/atpgp1-100 mutants are defective in multiple aspects of root development, including increased root-growth sensitivity to 1-naphthalene acetic acid (1-NAA), and decreased sensitivity to naphthylphthalamic acid (NPA)-mediated inhibition of root elongation. 1-naphthaleneacetic acid 206-211 ATP binding cassette subfamily B19 Arabidopsis thaliana 47-53 15908594-4 2005 In addition, we showed that the atmdr1-100 and atmdr1-100/atpgp1-100 mutants are defective in multiple aspects of root development, including increased root-growth sensitivity to 1-naphthalene acetic acid (1-NAA), and decreased sensitivity to naphthylphthalamic acid (NPA)-mediated inhibition of root elongation. 1-naphthaleneacetic acid 206-211 ATP binding cassette subfamily B1 Arabidopsis thaliana 58-64 15908594-5 2005 Consistent with the proposed role of AtMDR1 in basipetal auxin transport, we found that expression of the auxin responsive DR5::GUS reporter gene in the central elongation zone is significantly reduced in the atmdr1-100 mutant roots treated with 1-NAA at the root tips, compared to similarly treated wild-type plants. 1-naphthaleneacetic acid 246-251 ATP binding cassette subfamily B19 Arabidopsis thaliana 209-215 15739773-2 2004 NAA shape up, in biological fluids, as normal human endogenous low- and high-weight substances, e.g. angiotensin 2, bradykinin and others, and play the regulatory role in homeostasis. 1-naphthaleneacetic acid 0-3 kininogen 1 Homo sapiens 116-126 15739773-5 2004 We determined, within our case study, the level of NAA to e-NOS in blood and tears of patients with diabetic retinopathy and in donors. 1-naphthaleneacetic acid 51-54 nitric oxide synthase 3 Homo sapiens 58-63 15739773-6 2004 The level of NAA to e-NOS was found to be twice higher in tears versus blood in all groups of patients. 1-naphthaleneacetic acid 13-16 nitric oxide synthase 3 Homo sapiens 20-25 15460601-1 2004 OBJECTIVE: To determine whether men who relapse after neoadjuvant androgen ablation (NAA) and high-dose radiation therapy (RT) have faster PSA doubling times(PSAdt) than those who are treated with RT alone. 1-naphthaleneacetic acid 85-88 aminopeptidase puromycin sensitive Homo sapiens 139-142 15460601-6 2004 Patients treated with NAA had higher pre-treatment Gleason scores (p<O.001), PSA (p<O.001), and T stage (p<0.001). 1-naphthaleneacetic acid 22-25 aminopeptidase puromycin sensitive Homo sapiens 80-83 15460601-8 2004 Rising PSA profiles occurred in 78% of the RT-only group and 70% of the NAA group. 1-naphthaleneacetic acid 72-75 aminopeptidase puromycin sensitive Homo sapiens 7-10 13677801-9 2003 The longitudinal decrease of whole brain and hippocampal volumes on MR imaging, NAA levels on 1H MRS, cerebral glucose metabolism on PET, and cerebral blood flow on SPECT are associated with rate of cognitive decline in patients with AD. 1-naphthaleneacetic acid 80-83 MROS Homo sapiens 97-100 12836546-11 2003 The best NAA concentration was 0.01 mg.L-1. 1-naphthaleneacetic acid 9-12 immunoglobulin kappa variable 1-16 Homo sapiens 39-42 12502072-1 2003 The degradation of phenol by tyrosinase immobilized on chemically modified sodium aluminosilicate (NaA), calcium aluminosilicate (CaA), and silica gel was studied. 1-naphthaleneacetic acid 99-102 tyrosinase Homo sapiens 29-39 12586904-1 2003 Exogenously supplied auxin (1-naphthaleneacetic acid) inhibited light-induced activity increase of polyamine oxidase (PAO), a hydrogen peroxide-producing enzyme, in the outer tissues of maize (Zea mays) mesocotyl. 1-naphthaleneacetic acid 28-52 polyamine oxidase 1 Zea mays 99-116 12586904-1 2003 Exogenously supplied auxin (1-naphthaleneacetic acid) inhibited light-induced activity increase of polyamine oxidase (PAO), a hydrogen peroxide-producing enzyme, in the outer tissues of maize (Zea mays) mesocotyl. 1-naphthaleneacetic acid 28-52 polyamine oxidase 1 Zea mays 118-121 12502072-3 2003 Tyrosinase immobilized on CaA and on NaA could be re-used repeatedly without any decrease in performance. 1-naphthaleneacetic acid 37-40 tyrosinase Homo sapiens 0-10