PMID-sentid	Pub_year	Sent_text	compound_name	comp_offset	prot_official_name	organism	prot_offset
22613960-6	2012	Moreover, membrane-depolarizing             agents, including K+ and ATP-sensitive K+ (KATP) channel inhibitors glibenclamide             and U37883A enhanced TRAIL-induced apoptosis.	U 37883A	142-149	TNF superfamily member 10	Homo sapiens	159-164
22425821-9	2012	Pressor responses to norepinephrine and PNU-37883A (a vascular adenosine triphosphate-sensitive potassium channel inhibitor acting on the Kir6.1 pore-forming subunit) were measured at 6 or 24 hrs, in the absence or presence of the autonomic ganglion blocker, pentolinium.	U 37883A	40-50	potassium inwardly-rectifying channel, subfamily J, member 8	Rattus norvegicus	138-144
10516647-0	1999	Block of human aorta Kir6.1 by the vascular KATP channel inhibitor U37883A.	U 37883A	67-74	potassium inwardly rectifying channel subfamily J member 8	Homo sapiens	21-27
10516647-7	1999	K+-current carried by hKir6.1+SUR1 was inhibited by the putative vascular-selective KATP channel inhibitor U37883A (IC50 32 microM) whereas current carried by Kir6.2+SUR1 or Shaker K+ channels was unaffected.	U 37883A	107-114	potassium inwardly rectifying channel subfamily J member 8	Homo sapiens	22-29
10516647-7	1999	K+-current carried by hKir6.1+SUR1 was inhibited by the putative vascular-selective KATP channel inhibitor U37883A (IC50 32 microM) whereas current carried by Kir6.2+SUR1 or Shaker K+ channels was unaffected.	U 37883A	107-114	ATP binding cassette subfamily C member 8	Homo sapiens	30-34
9392837-4	1997	Vasodilator responses to T-kinin and bradykinin were attenuated by the nitric oxide synthase inhibitor, N omega Nitro-L-arginine methyl ester (L-NAME), but were not altered by the cyclooxygenase inhibitor, sodium meclofenamate, or the K+ ATP channel antagonist, U37883A.	U 37883A	262-269	kininogen 1	Homo sapiens	37-47
7955144-5	1994	Pulmonary vasodilator responses under elevated-tone conditions were inhibited by N omega-nitro-L-arginine methyl ester, suggesting that des-Arg9-bradykinin stimulates the release of nitric oxide, whereas meclofenamate and U-37883A, a nonsulfonylurea ATP-sensitive K+ channel antagonist, did not alter vasodilator responses to the B1 receptor agonist.	U 37883A	222-230	kininogen 1	Homo sapiens	145-155
24117345-4	2014	KEY RESULTS: During conventional whole-cell recording, pinacidil elicited an inward current that was suppressed by glibenclamide, a sulfonylurea agent, and by U-37883A, a selective K(IR)6.1 blocker.	U 37883A	159-167	potassium inwardly-rectifying channel, subfamily J, member 8	Mus musculus	181-189