PMID-sentid	Pub_year	Sent_text	compound_name	comp_offset	prot_official_name	organism	prot_offset
24940695-4	2014	We have demonstrated using the human Burkitt"s lymphoma B-cell line, Raji, that p53, p63 and p73 all accumulate in the nucleus, following treatment of cells with fludarabine nucleoside (2-FaraA).	2-faraa	186-193	tumor protein p53	Homo sapiens	80-83
26556860-2	2015	We previously showed that the Hsp90 inhibitor, SNX-7081, synergizes with and restores sensitivity to fludarabine nucleoside (2-FaraA) in human chronic lymphocytic leukemia (CLL) cells with lesions in the p53 pathway (Best OG, et al., Leukemia Lymphoma 53:1367-75, 2012).	2-faraa	125-132	heat shock protein 90 alpha family class A member 1	Homo sapiens	30-35
26556860-8	2015	These results provide valuable insight into the synergistic mechanism between SNX-7081 and 2-FaraA that may provide an alternative treatment for CLL patients with p53 mutations, for whom therapeutic options are currently limited.	2-faraa	91-98	tumor protein p53	Homo sapiens	163-166
24940695-4	2014	We have demonstrated using the human Burkitt"s lymphoma B-cell line, Raji, that p53, p63 and p73 all accumulate in the nucleus, following treatment of cells with fludarabine nucleoside (2-FaraA).	2-faraa	186-193	tumor protein p63	Homo sapiens	85-88
24940695-4	2014	We have demonstrated using the human Burkitt"s lymphoma B-cell line, Raji, that p53, p63 and p73 all accumulate in the nucleus, following treatment of cells with fludarabine nucleoside (2-FaraA).	2-faraa	186-193	tumor protein p73	Homo sapiens	93-96
24940695-8	2014	The levels of a number of proteins found on the outer plasma membrane change on cells treated with 2-FaraA, suggesting that signaling from the B-cell antigen receptor (BCR) is stimulated, resulting in induction of apoptosis through the intrinsic pathway.	2-faraa	99-106	BCR activator of RhoGEF and GTPase	Homo sapiens	143-166
24940695-8	2014	The levels of a number of proteins found on the outer plasma membrane change on cells treated with 2-FaraA, suggesting that signaling from the B-cell antigen receptor (BCR) is stimulated, resulting in induction of apoptosis through the intrinsic pathway.	2-faraa	99-106	BCR activator of RhoGEF and GTPase	Homo sapiens	168-171