PMID-sentid Pub_year Sent_text compound_name comp_offset prot_official_name organism prot_offset 2094320-8 1990 By pre-incubating patient serum, collected during rhIL-2 treatment, with monoclonal antibodies to murine IL-5, or human granulocyte-macrophage colony stimulating factor (GM-CSF), a reduction of 80% and 38% respectively in eosinophil and GM colony production was found. gm 170-172 colony stimulating factor 2 Homo sapiens 120-168 1693295-5 1990 IL-3 treatment also increased the numbers of GM clusters and mature cells (including all myeloid cells and lymphocytes) 7.8- and 4.8-fold, respectively. gm 45-47 interleukin 3 Homo sapiens 0-4 2307855-3 1990 Expression of the TGF beta 1 gene was noted in a fibroblast subpopulation of the affected tissues from the patients with DF and GM. gm 128-130 transforming growth factor beta 1 Homo sapiens 18-28 2307855-5 1990 The results suggest that TGF beta 1 may play a role in the development of skin fibrosis in cases of DF and GM. gm 107-109 transforming growth factor beta 1 Homo sapiens 25-35 2224057-3 1990 IL-1 and IL-6 are also known to enhance GM colony formation. gm 40-42 interleukin 1 complex Mus musculus 0-4 2224057-3 1990 IL-1 and IL-6 are also known to enhance GM colony formation. gm 40-42 interleukin 6 Mus musculus 9-13 2224057-7 1990 The addition of IL-3 to GM colony assay system did not enhance but rather suppressed GM colony formation. gm 24-26 interleukin 3 Mus musculus 16-20 2224057-7 1990 The addition of IL-3 to GM colony assay system did not enhance but rather suppressed GM colony formation. gm 85-87 interleukin 3 Mus musculus 16-20 1688498-10 1990 The reduced capacity of these stromal cells to produce G-CSF is associated with a reduced capacity of the CM to sustain GM colony formation and may be associated with the inability of these patients to sustain their neutrophil counts in vivo. gm 120-122 colony stimulating factor 3 Homo sapiens 55-60 2312183-0 1990 Gm allotype genes and gene dosage affecting both IgG subclass and IgE levels in atopic patients. gm 0-2 immunoglobulin heavy constant epsilon Homo sapiens 66-69 2312183-6 1990 Also the IgE levels were influenced by the Gm allotypes with increased IgE levels in the G1m(f,f) patients compared to the G1m(a,a) patients. gm 43-45 immunoglobulin heavy constant epsilon Homo sapiens 9-12 2312183-6 1990 Also the IgE levels were influenced by the Gm allotypes with increased IgE levels in the G1m(f,f) patients compared to the G1m(a,a) patients. gm 43-45 immunoglobulin heavy constant epsilon Homo sapiens 71-74 33588608-2 2021 GM provides insulin dosing recommendations based on patient-specific blood glucose (BG) trends after providers select either a custom dose or weight-based multiplier as the initial dosing strategy for the first 24 hours. gm 0-2 insulin Homo sapiens 12-19 7579172-4 1995 We describe the complete nucleotide sequence of chs5, the identification of its immediate neighbors and the organization of the four hitherto identified chs clusters in the Gm genome. gm 173-175 chalcone synthase 5 Glycine max 48-52 34977285-4 2021 The amount of chemokine (C-C motif) ligand 5 (CCL5) secreted from the MSC2 aggregates incorporating GM. gm 100-102 C-C motif chemokine ligand 5 Homo sapiens 14-44 34977285-4 2021 The amount of chemokine (C-C motif) ligand 5 (CCL5) secreted from the MSC2 aggregates incorporating GM. gm 100-102 C-C motif chemokine ligand 5 Homo sapiens 46-50 34977285-6 2021 Results: The amount of CCL5 secreted for the 3D MSC2 aggregates incorporating GM was significantly higher than that of two-dimensional (2D) MSC, 2D MSC2, and 3D MSC aggregates incorporating GM. gm 78-80 C-C motif chemokine ligand 5 Homo sapiens 23-27 34448678-8 2021 Patients with GM had better OS (HR 0.49, p < 0.001) and PFS on first-line vascular endothelial growth factor receptor tyrosine kinase inhibitors (VEGFR-TKIs) (HR 0.64, p = 0.045) than patients with non-GM. gm 14-16 kinase insert domain receptor Homo sapiens 146-151 34448678-11 2021 CONCLUSIONS: Patients with m-ccRCC who develop GM are molecularly characterized by heightened angiogenesis, translating into better prognosis and better outcomes on VEGFR-TKIs, but not IO. gm 47-49 kinase insert domain receptor Homo sapiens 165-170 34501500-5 2021 Regarding phthalates, monoethyl phthalate GM was 55.0 ng/mL and diethyl hexyl phthalate (as the sum of five metabolites) GM was 60.6 ng/mL. gm 121-123 thrombopoietin Mus musculus 136-138 34348893-3 2021 GRB2-SH2 domain targets the GM complex to phosphorylated H2AX at DSBs. gm 28-30 growth factor receptor bound protein 2 Homo sapiens 0-4 34348893-3 2021 GRB2-SH2 domain targets the GM complex to phosphorylated H2AX at DSBs. gm 28-30 H2A.X variant histone Homo sapiens 57-61 34348893-5 2021 RBBP6 depletion results in prolonged GM complex and HDR defects. gm 37-39 RB binding protein 6, ubiquitin ligase Homo sapiens 0-5 35088923-9 2022 In particular, the combination GM + BC resulted to be more effective than GM + CS in the up-regulation of key genes such as collagen type 2A1 (COLII), SOX-9, and aggrecan). gm 31-33 SRY-box transcription factor 9 Homo sapiens 151-156 35531956-13 2022 Transient upregulation of CXCR4 on GM HSC-enriched cells, using a noncytotoxic portion of viral protein R (VPR) fused to CXCR4 delivered as a protein in lentiviral particles, resulted in higher homing and long-term engraftment of GM HSC transplanted either i.v. gm 35-37 C-X-C motif chemokine receptor 4 Homo sapiens 26-31 35531956-22 2022 Alternatively, CXCR4 expression on HSC, when transiently increased using a protein delivery method, improves homing and engraftment specifically of GM HSC. gm 148-150 C-X-C motif chemokine receptor 4 Homo sapiens 15-20 35467188-6 2022 Furthermore, the concentration of Cry1Ab and EPSPS in the gut was determined after digestion of GM maize. gm 96-98 3-phosphoshikimate 1-carboxyvinyltransferase 2 Zea mays 45-50 35447036-18 2022 It is vital to assess PRL level and reduce PRL to normal in patients with GM. gm 74-76 prolactin Homo sapiens 22-25 35447036-18 2022 It is vital to assess PRL level and reduce PRL to normal in patients with GM. gm 74-76 prolactin Homo sapiens 43-46 35386402-8 2022 When cp = 0.05, MTD1% and MTD0.1% could be greatly reduced within 19 m and 75 m. These data were helpful to provide scientific data to set the isolation distance between GM and non-GM maize and select the right place to produce the hybrid maize seeds. gm 170-172 MTD1 Zea mays 16-20 35582513-8 2022 Results: The CSA of GM, and qualitative EI of both muscles were found to be mild-to-moderately correlated to all clinical examination tools (p<0.01), whereas the CSA of gastrocnemius lateralis was mildly related to Modified Ashworth Scale (p=0.009). gm 20-22 ERCC excision repair 8, CSA ubiquitin ligase complex subunit Homo sapiens 13-16 35103257-5 2022 rGO was selected as the conductive element to endow the hydrogel with conductive properties, and the beta-CD unit in rGO allowed rGO to interact with GM to improve the dispersity of rGO. gm 150-152 ACD, shelterin complex subunit and telomerase recruitment factor Rattus norvegicus 101-108 35010101-7 2022 Cd2+ doping results in a two-photon absorption cross-section, reaching 2.6 x 106 Goeppert-Mayer (GM), which is among the highest reported for CsPbBr3 NCs. gm 97-99 CD2 molecule Homo sapiens 0-3 2573954-6 1989 Vagotomy resulted in an increase in gastrin compared with controls in both antrum (1062 +/- 176 pmol/gm vs 484 +/- 48 pmol/gm) and plasma (236 +/- 72 pmol/L vs 29 +/- 4 pmol/L; p less than 0.05). gm 101-103 gastrin Oryctolagus cuniculus 36-43 2556120-4 1989 Mr 25,000 ir-bFGF from regenerating rat liver, partially purified by heparin-affinity chromatography, induces plasminogen activator activity and cell proliferation in transformed fetal bovine aortic endothelial GM 7373 cells and competes with Mr 18,000 [125I]bFGF for the binding to high affinity bFGF receptors. gm 211-213 fibroblast growth factor 2 Rattus norvegicus 13-17 2665868-8 1989 Also observed was a synergistic effect on GM colony formation in which more My-10-positive PD-MMCs stimulated by hrGM-CSF and G-CSF could form GM colonies than the sum of those stimulated by each separately. gm 42-44 colony stimulating factor 3 Homo sapiens 126-131 2473792-3 1989 Cultures of unseparated bone marrow, harvested from patients four to six days after administration of 5-fluorouracil (5-FU), resulted in additive GM colony formation with GM-CSF plus G-CSF, GM-CSF plus IL-3, and G-CSF plus IL-3. gm 146-148 colony stimulating factor 2 Homo sapiens 171-177 2469502-4 1989 Furthermore, day 4 but not day 2 post-5-FU BM showed enhanced GM colony formation when stimulated with IL-6 plus interleukin-4 (IL-4) or granulocyte colony-stimulating factor (G-CSF). gm 62-64 interleukin 6 Mus musculus 103-107 2469502-4 1989 Furthermore, day 4 but not day 2 post-5-FU BM showed enhanced GM colony formation when stimulated with IL-6 plus interleukin-4 (IL-4) or granulocyte colony-stimulating factor (G-CSF). gm 62-64 interleukin 4 Mus musculus 128-132 2469502-4 1989 Furthermore, day 4 but not day 2 post-5-FU BM showed enhanced GM colony formation when stimulated with IL-6 plus interleukin-4 (IL-4) or granulocyte colony-stimulating factor (G-CSF). gm 62-64 colony stimulating factor 3 (granulocyte) Mus musculus 137-174 2651350-3 1989 Interleukin 3 also increased GM colony formation but to a lesser extent. gm 29-31 interleukin 3 Homo sapiens 0-13 2654150-4 1989 In particular, CFU-GM, CFU-GEMM, BFU-E, and HPP-CFC, the most immature colonies, were inhibited by TGF-beta 1, whereas the more differentiated unipotent CFU-G, CFU-M, and CFU-E were not affected. gm 19-21 transforming growth factor, beta 1 Mus musculus 99-109 2510419-4 1989 For example, HPR-labelled and FITC-labelled WGA was positively recorded from neurons in cases of GM 2-gangliosidoses and mucopolysaccharidoses, type HURLER. gm 97-99 haptoglobin-related protein Homo sapiens 13-16 3067866-7 1988 Genes on chromosome 14 may also influence susceptibility to RA, probably by interaction with MHC genes and there are different Gm associations for DR4-positive and collagen-antibody-positive rheumatoid subgroups. gm 127-129 major histocompatibility complex, class II, DR beta 4 Homo sapiens 147-150 3207373-2 1988 When serum samples from patients with rheumatoid arthritis (RA) were tested for RF specificity towards these IgG monoclonal anti-D antibodies the incidence and titre of reactivity towards an IgG3 monoclonal anti-D antibody was considerably greater than for a polyclonal anti-D antibody of the same Gm allotype, G3m(5). gm 298-300 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 191-195 3048443-3 1988 Media conditioned by multiply passaged endothelial cells cultured for three days with recombinant IL-1 alpha (ECMIL-1) stimulated erythroid burst and GM colony formation in cultures of human nonadherent T-lymphocyte-depleted marrow mononuclear cells. gm 150-152 interleukin 1 alpha Homo sapiens 98-108 2455571-6 1988 IL-3 induction of GM colonies depended on the presence of FBS, whereas the degree of GM-CSF induction of GM colonies in FBS-deprived cultures depended on the method by which adherent cells were removed. gm 18-20 interleukin 3 Homo sapiens 0-4 2831972-2 1988 The first equivalent of Cu2+ binds to HRG producing a type II electron paramagnetic resonance (EPR) spectrum with g[[ = 2.25, gm = 2.05, A[[ = 0.019 cm-1 (180 G), and superhyperfine along gm. gm 126-128 histidine rich glycoprotein Homo sapiens 38-41 2830255-5 1987 When the myosin prephosphorylated with MLCK was further phosphorylated with PK-C, PPi PAGE analysis showed only one band comigrating with GM, i.e., GMP1 and GMP2 migrated to the same position as GM. gm 138-140 myosin heavy chain 14 Homo sapiens 9-15 2830255-5 1987 When the myosin prephosphorylated with MLCK was further phosphorylated with PK-C, PPi PAGE analysis showed only one band comigrating with GM, i.e., GMP1 and GMP2 migrated to the same position as GM. gm 138-140 myosin light chain kinase Homo sapiens 39-43 2830255-5 1987 When the myosin prephosphorylated with MLCK was further phosphorylated with PK-C, PPi PAGE analysis showed only one band comigrating with GM, i.e., GMP1 and GMP2 migrated to the same position as GM. gm 148-150 myosin heavy chain 14 Homo sapiens 9-15 2830255-5 1987 When the myosin prephosphorylated with MLCK was further phosphorylated with PK-C, PPi PAGE analysis showed only one band comigrating with GM, i.e., GMP1 and GMP2 migrated to the same position as GM. gm 148-150 myosin light chain kinase Homo sapiens 39-43 3498640-8 1987 These data indicate that rG-CSF had a direct effect on the growth and development of GM progenitors at a late stage and a significant effect on multipotential hemopoietic precursors. gm 85-87 colony stimulating factor 3 Rattus norvegicus 25-31 3495596-6 1987 GM cells maintained as IL 3- or GM-CSF-dependent cells readily converted to a lymphokine-independent growth state when infected with v-src. gm 0-2 interleukin 3 Mus musculus 23-27 3495596-6 1987 GM cells maintained as IL 3- or GM-CSF-dependent cells readily converted to a lymphokine-independent growth state when infected with v-src. gm 0-2 colony stimulating factor 2 (granulocyte-macrophage) Mus musculus 32-38 3298286-5 1987 Dose-response studies revealed that maximal GM and blast cell colony formation is achieved with 100 U/ml GM-CSF. gm 44-46 colony stimulating factor 2 (granulocyte-macrophage) Mus musculus 105-111 3496057-4 1987 The Gm(zafngb) phenotype was found in 26% of DR3 or DR7 positive patients overall and only 9% of RA patients negative for these DR antigens (p less than 0.005), suggesting an interaction between HLA-DR3/7 and Gm(zafngb). gm 4-6 TNF receptor superfamily member 25 Homo sapiens 45-48 3496057-4 1987 The Gm(zafngb) phenotype was found in 26% of DR3 or DR7 positive patients overall and only 9% of RA patients negative for these DR antigens (p less than 0.005), suggesting an interaction between HLA-DR3/7 and Gm(zafngb). gm 4-6 TNF receptor superfamily member 25 Homo sapiens 199-202 2953314-9 1987 Within the patient group Gm(1,3;5,21) was found only in DR4 positive individuals. gm 25-27 major histocompatibility complex, class II, DR beta 4 Homo sapiens 56-59 3469412-8 1986 Based on these results, we suggest that the augmenting effect of E2 on GM colony formation is mediated by inducing the colony precursor cells to be more responsive to CSF. gm 71-73 colony stimulating factor 2 Homo sapiens 167-170 3113776-6 1986 Although Gm showed no association with eye disease, it modified the risk for ophthalmopathy associated with HLA-B8; the odds ratios were 20.9 for B8+ Gmfb homozygozity (fb+), 15.3 for B8+ fb- and 1.7 for B8- fb+ (B8- fb- = 1.00). gm 9-11 glia maturation factor beta Homo sapiens 150-154 3533587-3 1986 GM-CFU recovery in SNA- was equal or superior to that in the SBA- fraction. gm 0-2 snail family transcriptional repressor 1 Homo sapiens 19-22 3490183-0 1986 Association of Gm allotypes with the occurrence of ankylosing spondylitis in HLA-B27-positive anterior uveitis. gm 15-17 major histocompatibility complex, class I, B Homo sapiens 77-84 3493344-0 1986 Increased frequency of Gm(1,2;21) phenotype in HLA-DR4 positive seropositive rheumatoid arthritis. gm 23-25 major histocompatibility complex, class II, DR beta 4 Homo sapiens 51-54 3726861-5 1986 An increase in Gm(1,3;5) was observed in DR3 positive, DR4 negative patients. gm 15-17 TNF receptor superfamily member 25 Homo sapiens 41-44 3726861-5 1986 An increase in Gm(1,3;5) was observed in DR3 positive, DR4 negative patients. gm 15-17 major histocompatibility complex, class II, DR beta 4 Homo sapiens 55-58 3726861-6 1986 This association occurred predominantly in females (compared with DR4 and DR3/4 patients of the same Gm phenotype who were predominantly male). gm 101-103 TNF receptor superfamily member 25 Homo sapiens 74-77 3963065-4 1986 Oxytocin in corpus luteum increased from 14.0 +/- 1.8 ng/gm of wet weight in early to 30.8 +/- 0.9 ng/gm in midluteal phases (p = less than 0.001). gm 57-59 oxytocin/neurophysin I prepropeptide Homo sapiens 0-8 2417889-2 1986 The exceptional presence on gamma 1 and gamma 2 chains of Gm allotypes usually located on the CH3 domain of gamma 3 shows an unexpected clustering of base changes and subsequent identity of short DNA sequences in the CH3 exon of the non-allelic gamma 1, gamma 2 and gamma 3 genes. gm 58-60 tryptophanyl-tRNA synthetase 1 Homo sapiens 40-47 2417889-2 1986 The exceptional presence on gamma 1 and gamma 2 chains of Gm allotypes usually located on the CH3 domain of gamma 3 shows an unexpected clustering of base changes and subsequent identity of short DNA sequences in the CH3 exon of the non-allelic gamma 1, gamma 2 and gamma 3 genes. gm 58-60 tryptophanyl-tRNA synthetase 1 Homo sapiens 254-261 3484428-1 1986 Recombinant human granulocyte-macrophage colony-stimulating factor (rH GM-CSF) was purified to homogeneity from medium conditioned by COS cells transfected with a cloned human GM-CSF cDNA and shown to be an effective proliferative stimulus in human marrow cultures for GM and eosinophil colony formation. gm 71-73 colony stimulating factor 2 Homo sapiens 18-66 3867712-7 1985 When subdivided according to HLA type, a non-random distribution of Gm phenotypes was seen in HLA-B8/DR3 positive individuals with anti-IgA antibodies (HLA-B8/DR3 being the haplotype associated with IgA deficiency). gm 68-70 CD79a molecule Homo sapiens 136-139 2579983-4 1985 The extractable CGRP was 8.6 +/- 1.8 pmoles/gm of tissue in the iris and 44.0 +/- 8.1 pmoles/gm in the trigeminal ganglion. gm 44-46 calcitonin related polypeptide alpha Homo sapiens 16-20 2579983-5 1985 Following damage to the Gasserian ganglion a marked decrease of CGRP immunoreactivity was observed in the anterior uvea (control 11.3 +/- 1.6 pmoles/gm; operated 1.4 +/- 0.1 pmoles/gm) confirming the origin of the immunoreactive fibres from trigeminal primary sensory neurons. gm 149-151 calcitonin related polypeptide alpha Homo sapiens 64-68 2579983-5 1985 Following damage to the Gasserian ganglion a marked decrease of CGRP immunoreactivity was observed in the anterior uvea (control 11.3 +/- 1.6 pmoles/gm; operated 1.4 +/- 0.1 pmoles/gm) confirming the origin of the immunoreactive fibres from trigeminal primary sensory neurons. gm 181-183 calcitonin related polypeptide alpha Homo sapiens 64-68 3975854-3 1985 The highest concentrations of immunoreactive neurotensin were found in the mucosal extracts of the jejunum (422 +/- 68 ng/gm) and ileum (3025 +/- 289 ng/gm). gm 122-124 neurotensin Canis lupus familiaris 45-56 3975854-6 1985 The neurotensin concentration in extracts of the seromuscular layers of jejunum (73 +/- 14 ng/gm) and ileum (187 +/- 38 ng/gm) were statistically higher in comparison with other gut loci. gm 94-96 neurotensin Canis lupus familiaris 4-15 2939302-5 1985 Gc2/Gc1 + Gc2 with Gc1 = GAC + GAC1 and Gc2 = GM + GM1, GAC and GM being the dependent conductances on amniotic cavity (AC) and maternal (M) sides; GAC1 and GM1 being the leak conductances on AC and M. Gp and Gc are due to epithelial cell layer and Gs (conductance in a series) is due to the other layers. gm 46-48 solute carrier family 25 member 18 Homo sapiens 0-3 2939302-5 1985 Gc2/Gc1 + Gc2 with Gc1 = GAC + GAC1 and Gc2 = GM + GM1, GAC and GM being the dependent conductances on amniotic cavity (AC) and maternal (M) sides; GAC1 and GM1 being the leak conductances on AC and M. Gp and Gc are due to epithelial cell layer and Gs (conductance in a series) is due to the other layers. gm 46-48 olfactomedin 4 Homo sapiens 4-7 2939302-5 1985 Gc2/Gc1 + Gc2 with Gc1 = GAC + GAC1 and Gc2 = GM + GM1, GAC and GM being the dependent conductances on amniotic cavity (AC) and maternal (M) sides; GAC1 and GM1 being the leak conductances on AC and M. Gp and Gc are due to epithelial cell layer and Gs (conductance in a series) is due to the other layers. gm 46-48 solute carrier family 25 member 18 Homo sapiens 10-13 2939302-5 1985 Gc2/Gc1 + Gc2 with Gc1 = GAC + GAC1 and Gc2 = GM + GM1, GAC and GM being the dependent conductances on amniotic cavity (AC) and maternal (M) sides; GAC1 and GM1 being the leak conductances on AC and M. Gp and Gc are due to epithelial cell layer and Gs (conductance in a series) is due to the other layers. gm 46-48 solute carrier family 25 member 18 Homo sapiens 10-13 2939302-5 1985 Gc2/Gc1 + Gc2 with Gc1 = GAC + GAC1 and Gc2 = GM + GM1, GAC and GM being the dependent conductances on amniotic cavity (AC) and maternal (M) sides; GAC1 and GM1 being the leak conductances on AC and M. Gp and Gc are due to epithelial cell layer and Gs (conductance in a series) is due to the other layers. gm 51-53 solute carrier family 25 member 18 Homo sapiens 0-3 2939302-5 1985 Gc2/Gc1 + Gc2 with Gc1 = GAC + GAC1 and Gc2 = GM + GM1, GAC and GM being the dependent conductances on amniotic cavity (AC) and maternal (M) sides; GAC1 and GM1 being the leak conductances on AC and M. Gp and Gc are due to epithelial cell layer and Gs (conductance in a series) is due to the other layers. gm 51-53 olfactomedin 4 Homo sapiens 4-7 6334444-0 1984 Association of Gm allotypes with the occurrence of ankylosing spondylitis in HLA-B27-positive anterior uveitis. gm 15-17 major histocompatibility complex, class I, B Homo sapiens 77-84 6371846-8 1984 With a 125I-insulin infusion site at 5 mm depth in the subcutaneous tissue of rabbits, an overall linear proportionality was found between the results obtained with a NaI(Tl) detector and a GM detector raised 15 mm above the skin surface inside the detector housing. gm 190-192 insulin Oryctolagus cuniculus 12-19 6605825-5 1983 When human thymus cells were treated with A-22 antibody they showed a reduction of up to 70% in their capacity to attach to the GM-4762 lymphoblast cell line and the K-562 myeloid line. gm 128-130 immunoglobulin kappa variable 3-25 (pseudogene) Homo sapiens 42-46 6619556-1 1983 Gm allotypes were detected and quantitated by radioimmunoassay (RIA) in paired serum and CSF samples from patients suffering from various neurological diseases. gm 0-2 colony stimulating factor 2 Homo sapiens 89-92 6187855-6 1983 Further, both Early and Late Gm were able to generate NP-specific induction-phase suppressor cells (Ts1) when as few as 10(2) NP-coupled Gm were given i.v., the same as NP-SAC controls. gm 29-31 Trichinella spiralis resistance 1 Mus musculus 100-103 6187855-6 1983 Further, both Early and Late Gm were able to generate NP-specific induction-phase suppressor cells (Ts1) when as few as 10(2) NP-coupled Gm were given i.v., the same as NP-SAC controls. gm 137-139 Trichinella spiralis resistance 1 Mus musculus 100-103 6187855-7 1983 Finally, NP-specific effector-phase suppressor cells (Ts3) were equally induced by Early Gm, Late Gm, or SAC controls. gm 89-91 Trichinella spiralis resistance 3 Mus musculus 54-57 6797346-1 1981 In this study linkage between the loci for Gm (gamma-type heavy-chain immunoglobulin markers) and Pi (alpha 1-antitrypsin/alpha 1-protease inhibitor) has been shown in families segregating for the Pi M subtypes (M1, M2, M3 and Msal) as identified by separator isoelectric focusing . gm 43-45 serpin family A member 1 Homo sapiens 102-121 6797346-1 1981 In this study linkage between the loci for Gm (gamma-type heavy-chain immunoglobulin markers) and Pi (alpha 1-antitrypsin/alpha 1-protease inhibitor) has been shown in families segregating for the Pi M subtypes (M1, M2, M3 and Msal) as identified by separator isoelectric focusing . gm 43-45 serpin family A member 1 Homo sapiens 122-148 6253697-2 1980 99mTc-PYP uptake ratio, measured by the ratio of cpm/gm for the heart to cpm/gm for the skull, began to increase five days after virus inoculation and reached a maximum on the seventh day. gm 53-55 pyrophosphatase (inorganic) 1 Mus musculus 6-9 7395924-1 1980 Gm and Inv data--polymorphism for Gm3 and for Gm1,17,21 without Gm(26). gm 34-36 leishmanolysin like peptidase Homo sapiens 7-10 6245291-4 1980 The cyclic GMP concentrations in the coronary artery after the injection of saline, SG 75, and dilazep were 35.6 +/- 4.4 pmole/Gm, 40.4 +/- 3.3 pmole/Gm, 35.3 +/- 3.2 pmole/Gm, respectively. gm 127-129 5'-nucleotidase, cytosolic II Homo sapiens 11-14 6245291-4 1980 The cyclic GMP concentrations in the coronary artery after the injection of saline, SG 75, and dilazep were 35.6 +/- 4.4 pmole/Gm, 40.4 +/- 3.3 pmole/Gm, 35.3 +/- 3.2 pmole/Gm, respectively. gm 150-152 5'-nucleotidase, cytosolic II Homo sapiens 11-14 6245291-4 1980 The cyclic GMP concentrations in the coronary artery after the injection of saline, SG 75, and dilazep were 35.6 +/- 4.4 pmole/Gm, 40.4 +/- 3.3 pmole/Gm, 35.3 +/- 3.2 pmole/Gm, respectively. gm 150-152 5'-nucleotidase, cytosolic II Homo sapiens 11-14 383740-2 1979 The mean (+/- SE) concentration of beta-endorphin in pancreatic extracts of 5 non-diabetic adults was 13.5 +/- 9.8 ng/gm with a range of 2.1 to 52.8 ng/gm of tissue. gm 118-120 proopiomelanocortin Homo sapiens 35-49 112031-3 1979 The only associations discovered were the ABO and Gm polymorphisms: The incidence of O and Gm(-1) phenotypes in patients is obviously higher than in controls. gm 91-93 ABO, alpha 1-3-N-acetylgalactosaminyltransferase and alpha 1-3-galactosyltransferase Homo sapiens 42-45 365007-4 1979 Antral tissue gastrin also increased by 100 per cent (6.7 x 10(6) to 13.7 x 10(6) pg/gm tissue, p less than 0.05). gm 85-87 gastrin Canis lupus familiaris 14-21 702517-1 1978 This paper describes the effect of trinitrocresolate anions (TNC-) on the electrical conductance (Gm), and tracer-measured unidirectional Na and K fluxes (MNa and MK) across bilayers formed from sheep red cell lipids dissolved in decane. gm 98-100 tenascin Ovis aries 61-64 702517-3 1978 In the presence of TNC- (10(-2) M), Gm increased and TNC- was the main charge carrier in the system. gm 36-38 tenascin Ovis aries 19-22 702517-8 1978 In the presence of both valinomycin (10(-6) M) and TNC- (10(-2) M), this selectivity disappeared in that both Gm and MNa in the NaCl system were similar to the respective values in the KCl system. gm 110-112 tenascin Ovis aries 51-54 77655-0 1978 [Gm(28), a new allotypic marker on human IgG3: peculiar interest of its study within Negroid populations (author"s transl)]. gm 1-3 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 41-45 842652-6 1977 The contribution of gcl to gm was 60% in G and 30% in S in 14-day denervated muscles. gm 27-29 germ cell-less 1, spermatogenesis associated Rattus norvegicus 20-23 54236-5 1975 High IgG3 concentrations corresponded to the presence of Gm(b). gm 57-59 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 5-9 1228847-5 1975 The use of serum from the same animal for this purpose avoids the theoretical possibility that antiglobulin antibodies directed at subclass determinants such as Gm of Inv could be differentially inhibited due to possible subclass differences in the blocking sera employed. gm 161-163 inversin Homo sapiens 167-170 4133177-0 1974 Gm (Ray), a new allotypic marker on human IgG3. gm 0-2 SH3 and SYLF domain containing 1 Homo sapiens 4-7 4133177-0 1974 Gm (Ray), a new allotypic marker on human IgG3. gm 0-2 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 42-46 4623402-2 1972 Differences in Gm gene expression for G1 and G3 H chains in sera. gm 15-17 proline rich protein BstNI subfamily 3 Homo sapiens 39-51 4237446-0 1968 [Demonstration of trace Gm-Inv-antibodies in human serums by means of the multiple dosis technic. gm 24-26 inversin Homo sapiens 27-30 4235334-0 1968 [Demonstration of Gm-Inv-antibodies in concentrations below threshold in human sera by means of the multiple dosis technic. gm 18-20 inversin Homo sapiens 21-24 4880038-0 1968 [Variations of Gm ET Inv phenotypes associated with allogenic bone marrow grafts]. gm 15-17 inversin Homo sapiens 21-24 33515810-8 2021 It suggests that the risk of GM might be significantly increased by the XRCC3 Thr241Met polymorphism according to ethnicity and cancer types. gm 29-31 X-ray repair cross complementing 3 Homo sapiens 72-77 34038111-4 2021 Mechanistically, GM affected miRNA maturation by targeting TAR RNA-binding protein 2 (TRBP), with an efficacy higher than that of enoxacin, and promoted the binding of TRBP with Dicer. gm 17-19 TARBP2, RISC loading complex RNA binding subunit Mus musculus 59-84 34038111-4 2021 Mechanistically, GM affected miRNA maturation by targeting TAR RNA-binding protein 2 (TRBP), with an efficacy higher than that of enoxacin, and promoted the binding of TRBP with Dicer. gm 17-19 TARBP2, RISC loading complex RNA binding subunit Mus musculus 86-90 34038111-4 2021 Mechanistically, GM affected miRNA maturation by targeting TAR RNA-binding protein 2 (TRBP), with an efficacy higher than that of enoxacin, and promoted the binding of TRBP with Dicer. gm 17-19 TARBP2, RISC loading complex RNA binding subunit Mus musculus 168-172 33736248-6 2021 Here we employ Bayesian multilevel logistic regression to understand the associations between various well attributes and reported occurrences of GM in 0.6% of the 25,000 oil and gas wells in BC. gm 146-148 gastrin Homo sapiens 179-182 33736248-10 2021 We ascribe the spatial clustering of GM cases to the local geologic environment, and we speculate that there are links between particular intermediate gas-bearing formations and GM occurrence in the Fort Nelson Plains Area. gm 178-180 gastrin Homo sapiens 151-154 33850460-6 2021 Platelets on GM caused a continuous high release in both interleukin-10 and metalloproteinase-3 compared with PRP alone. gm 13-15 interleukin 10 Rattus norvegicus 57-71 33850460-6 2021 Platelets on GM caused a continuous high release in both interleukin-10 and metalloproteinase-3 compared with PRP alone. gm 13-15 proline rich protein 2-like 1 Rattus norvegicus 110-113 33341734-8 2021 After beta-glucuronidase hydrolysis, the GM of SG-corrected urinary 2,4-D was 0.15 mug/L, and the detection frequency was 100%. gm 41-43 glucuronidase beta Homo sapiens 6-24 33898740-5 2021 However, there was a significant interaction: In the presence of GM 3 (i.e., GM 3/3 and GM 3/17 subjects), the presence of the HLA-C allele was associated with a 4-fold increase in the likelihood of developing AD compared with its absence (hazard ratio [HR] 4.17, 95% CI, 1.28-13.58). gm 65-67 major histocompatibility complex, class II, DR beta 1 Homo sapiens 127-130 33898740-5 2021 However, there was a significant interaction: In the presence of GM 3 (i.e., GM 3/3 and GM 3/17 subjects), the presence of the HLA-C allele was associated with a 4-fold increase in the likelihood of developing AD compared with its absence (hazard ratio [HR] 4.17, 95% CI, 1.28-13.58). gm 77-79 major histocompatibility complex, class II, DR beta 1 Homo sapiens 127-130 33898740-5 2021 However, there was a significant interaction: In the presence of GM 3 (i.e., GM 3/3 and GM 3/17 subjects), the presence of the HLA-C allele was associated with a 4-fold increase in the likelihood of developing AD compared with its absence (hazard ratio [HR] 4.17, 95% CI, 1.28-13.58). gm 77-79 major histocompatibility complex, class II, DR beta 1 Homo sapiens 127-130 33401097-12 2021 These results indicate that in addition to drought stress, plants overexpressing AtAREB1 exhibited better performance under flooding when compared to the non-GM line, suggesting a cross-signaling response to both abiotic factors. gm 158-160 abscisic acid responsive elements-binding factor 2 Arabidopsis thaliana 81-88 33295908-3 2021 The GM could enter into the active site of alpha-glucosidase and bind with the catalytic amino acid residues through hydrogen bonding and pi-conjugation, thus playing an important role in the competitive inhibition of catechins and theaflavins. gm 4-6 sucrase-isomaltase Homo sapiens 43-60 33295908-5 2021 In addition, the in vivo hypoglycemic effects of galloylated polyphenols suggest that the GM may be considered as a pharmaceutical fragment for the alleviation of type II diabetes symptoms through alpha-glucosidase inhibition. gm 90-92 sucrase-isomaltase Homo sapiens 197-214 32434426-5 2020 In addition, the E-cadherin expression level decreased to a significantly great extent compared with that co-cultured with TAM aggregates incorporating GM, whereas the significantly higher expression of N-cadherin and Vimentin was observed. gm 152-154 cadherin 1 Homo sapiens 17-27 32434426-7 2020 The GM containing transforming growth factor-beta1 (TGF-beta1) were prepared to incorporate into 3D CAF (3D CAF-GM-TGF-beta1). gm 4-6 transforming growth factor beta 1 Homo sapiens 18-50 32434426-7 2020 The GM containing transforming growth factor-beta1 (TGF-beta1) were prepared to incorporate into 3D CAF (3D CAF-GM-TGF-beta1). gm 4-6 transforming growth factor beta 1 Homo sapiens 52-61 32434426-7 2020 The GM containing transforming growth factor-beta1 (TGF-beta1) were prepared to incorporate into 3D CAF (3D CAF-GM-TGF-beta1). gm 4-6 lysine acetyltransferase 2B Homo sapiens 100-103 32434426-7 2020 The GM containing transforming growth factor-beta1 (TGF-beta1) were prepared to incorporate into 3D CAF (3D CAF-GM-TGF-beta1). gm 112-114 lysine acetyltransferase 2B Homo sapiens 100-103 33473367-7 2020 The GM muscle of PM lambs showed higher ( P < 0.05) conjugated linoleic acid (CLA) content, as well as higher total polyunsaturated acids (PUFAs), PUFA n-6, and PUFA n-3 ( P < 0.05). gm 4-6 PUFA Ovis aries 140-144 33473367-7 2020 The GM muscle of PM lambs showed higher ( P < 0.05) conjugated linoleic acid (CLA) content, as well as higher total polyunsaturated acids (PUFAs), PUFA n-6, and PUFA n-3 ( P < 0.05). gm 4-6 PUFA Ovis aries 148-152 32817212-10 2020 Upon testing select candidate proteins, we discovered that XPO6, an exportin, is required for gM to be released from the nucleus toward the TGN. gm 94-96 exportin 6 Homo sapiens 59-63 32817212-15 2020 Analysis of select proteins revealed that XPO6 is required for gM to leave the nuclear membranes late in the infection. gm 63-65 exportin 6 Homo sapiens 42-46 32749395-4 2020 The LN distribution study showed that both DiR and FK506 were delivered into the LNs effectively via GM-mediated transport after 24 h and were present in the LNs for at least 48 h. The FK506-loaded GM (GM-FK506) significantly prolonged allograft survival compared with the PBS group (mean survival time, 17.8 +- 1.9 versus 7.3 +- 1.0 days; P < 0.01), and marked decreased the acute rejection grade. gm 101-103 arginine vasopressin receptor 2 Homo sapiens 43-46 32749395-4 2020 The LN distribution study showed that both DiR and FK506 were delivered into the LNs effectively via GM-mediated transport after 24 h and were present in the LNs for at least 48 h. The FK506-loaded GM (GM-FK506) significantly prolonged allograft survival compared with the PBS group (mean survival time, 17.8 +- 1.9 versus 7.3 +- 1.0 days; P < 0.01), and marked decreased the acute rejection grade. gm 198-200 arginine vasopressin receptor 2 Homo sapiens 43-46 32749395-5 2020 Furthermore, T cell infiltration, and secretion of IL-2 and IFN-gamma were dramatically reduced in the GM-FK506 group. gm 103-105 interleukin 2 Homo sapiens 51-55 32749395-5 2020 Furthermore, T cell infiltration, and secretion of IL-2 and IFN-gamma were dramatically reduced in the GM-FK506 group. gm 103-105 interferon gamma Homo sapiens 60-69 32124069-9 2020 CONCLUSIONS: An ACLP releasing the flap up to the mucogingival junction, with a >= 3-mm distance from the bone crest to the gingival margin can lead to a stable GM position at 42, 90, and 180 days. gm 161-163 AE binding protein 1 Homo sapiens 16-20 32982767-12 2020 However, when jumping from greater deformable surface, both GM muscle activity (P = 0.022, eta2 = 0.18, large) and peak shortening velocity of GM MTU (P = 0.042, eta2 = 0.10, medium) increased during the push-off phase. gm 60-62 DNA polymerase iota Homo sapiens 91-95 32058939-4 2020 Geometric means (GM) were highest for metabolites of DiBP (MiBP: 26.1 mug/L), DEP (MEP: 25.8 mug/L), DnBP (MnBP: 20.9 mug/L), and DEHP (cx-MEPP: 11.9 mug/L). gm 17-19 nicotinamide riboside kinase 2 Homo sapiens 59-63 32194748-6 2020 The infiltration of CD8+ lymphocytes was significantly higher in GM than in SM (P=0.0231). gm 65-67 CD8a molecule Homo sapiens 20-23 32132998-4 2020 In this experimental study with mice, we found GM feeding enhanced immunoglobulin production, antigen-specific (ovalbumin, OVA) immune responses, and phagocytosis activity. gm 47-49 serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus 112-121 31332929-2 2019 This study aimed to determine the performance of the flash GM system during daily-life glycaemic challenges such as carbohydrate-rich meals, bolus insulin-induced glycaemic disturbances and acute physical exercise in individuals with type 1 diabetes. gm 59-61 insulin Homo sapiens 147-154 31781634-9 2019 Four weeks after MSC-loaded GM treatment, we found that the mRNA levels of TNF-alpha and IL-6 were clearly reduced. gm 28-30 tumor necrosis factor Rattus norvegicus 75-84 31781634-9 2019 Four weeks after MSC-loaded GM treatment, we found that the mRNA levels of TNF-alpha and IL-6 were clearly reduced. gm 28-30 interleukin 6 Rattus norvegicus 89-93 31356511-5 2019 Barbell hip thrust showed higher GM activity than the SQ (effect size [ES] = 1.39, p = 0.038) but was not significantly different from RDL (ES = 0.49, p = 0.285) at 1RM. gm 33-35 hedgehog interacting protein Homo sapiens 8-11 31071302-6 2019 Moreover, over-expression and interference assays revealed that Smad3 has a different effect on C/EBPalpha and C/EBPbeta expression under GM versus DM conditions. gm 138-140 SMAD family member 3 Bos taurus 64-69 31071302-6 2019 Moreover, over-expression and interference assays revealed that Smad3 has a different effect on C/EBPalpha and C/EBPbeta expression under GM versus DM conditions. gm 138-140 CCAAT/enhancer-binding protein alpha Bos taurus 96-106 31071302-6 2019 Moreover, over-expression and interference assays revealed that Smad3 has a different effect on C/EBPalpha and C/EBPbeta expression under GM versus DM conditions. gm 138-140 CCAAT/enhancer-binding protein beta Bos taurus 111-120 31417557-14 2019 However, patients under DMF treatment with a stronger CD8+ T cell depletion present a more favorable response in terms of cortical integrity and GM and WM network responses. gm 145-147 CD8a molecule Homo sapiens 54-57 30832586-6 2019 By performing a GWAS for IMF content and composition traits recorded in the LD and GM muscles of 350 Duroc pigs, we identified the existence of one region on SSC14 (110-114 Mb) displaying significant associations with C18:0, C18:1(n-7), saturated and unsaturated fatty acid contents in both GM and LD muscles. gm 83-85 IMF Sus scrofa 25-28 30694168-3 2019 In this study, we addressed the impact of gM/UL49.5-specific regions on heterodimer formation, folding and trafficking from the endoplasmic reticulum (ER) to the trans-Golgi network (TGN) - events previously found to be responsible for abrogation of the UL49.5-mediated inhibition of the transporter associated with antigen processing (TAP). gm 42-44 tegument protein VP22 Bovine alphaherpesvirus 1 254-258 30694168-3 2019 In this study, we addressed the impact of gM/UL49.5-specific regions on heterodimer formation, folding and trafficking from the endoplasmic reticulum (ER) to the trans-Golgi network (TGN) - events previously found to be responsible for abrogation of the UL49.5-mediated inhibition of the transporter associated with antigen processing (TAP). gm 42-44 transporter 1, ATP binding cassette subfamily B member Homo sapiens 288-334 30694168-3 2019 In this study, we addressed the impact of gM/UL49.5-specific regions on heterodimer formation, folding and trafficking from the endoplasmic reticulum (ER) to the trans-Golgi network (TGN) - events previously found to be responsible for abrogation of the UL49.5-mediated inhibition of the transporter associated with antigen processing (TAP). gm 42-44 nuclear RNA export factor 1 Homo sapiens 336-339 30694168-8 2019 Finally, we found that leucine substitutions in putative glycine zipper motifs within TM helices of gM resulted in strong reduction of complex formation and decreased ability of gM to interfere with UL49.5-mediated major histocompatibility class I downregulation. gm 100-102 envelope glycoprotein N Bovine alphaherpesvirus 1 199-205 30694168-8 2019 Finally, we found that leucine substitutions in putative glycine zipper motifs within TM helices of gM resulted in strong reduction of complex formation and decreased ability of gM to interfere with UL49.5-mediated major histocompatibility class I downregulation. gm 178-180 envelope glycoprotein N Bovine alphaherpesvirus 1 199-205 30426567-9 2019 In addition, daily CD34+ cell/Kg yield was significantly higher in GM (2.08 x 10/Kg) compared with PM group (1.64 x 10/Kg, P = .019). gm 67-69 CD34 molecule Homo sapiens 19-23 30699960-5 2019 Of note, work that is more recent has now shown that RNA modifications in their naturally occurring context can have immune-modulatory functions: Gm, a naturally occurring ribose-methylation within tRNA resulted in a lack of TLR7 stimulation and within a defined sequence context acted as antagonist. gm 146-148 toll like receptor 7 Homo sapiens 225-229 30627906-3 2019 It was shown that activation of vanilloid type 1 receptors (TRPV1) induced by exogenous acidification of GM is not sufficient to potentiate the production of HCO3, including production depending on neuronal NO synthase. gm 105-107 transient receptor potential cation channel subfamily V member 1 Homo sapiens 60-65 30544623-9 2018 We also present evidence that GSK3 inhibition in GM/DCs reduced C/EBPbeta DNA binding activity and increased expression of costimulatory molecules in GM/DCs and their production of IL-10. gm 49-51 glycogen synthase kinase 3 beta Mus musculus 30-34 30544623-9 2018 We also present evidence that GSK3 inhibition in GM/DCs reduced C/EBPbeta DNA binding activity and increased expression of costimulatory molecules in GM/DCs and their production of IL-10. gm 49-51 CCAAT/enhancer binding protein (C/EBP), beta Mus musculus 64-73 30544623-9 2018 We also present evidence that GSK3 inhibition in GM/DCs reduced C/EBPbeta DNA binding activity and increased expression of costimulatory molecules in GM/DCs and their production of IL-10. gm 49-51 interleukin 10 Mus musculus 181-186 30544623-9 2018 We also present evidence that GSK3 inhibition in GM/DCs reduced C/EBPbeta DNA binding activity and increased expression of costimulatory molecules in GM/DCs and their production of IL-10. gm 150-152 glycogen synthase kinase 3 beta Mus musculus 30-34 30544623-11 2018 Our results highlight the importance of the p38MAPK-C/EBPbeta pathway in regulating phenotype and function of tolerogenic GM/DCs. gm 122-124 CCAAT/enhancer binding protein (C/EBP), beta Mus musculus 52-61 30209589-0 2018 A phase I/randomized phase II study of GM.CD40L vaccine in combination with CCL21 in patients with advanced lung adenocarcinoma. gm 39-41 CD40 ligand Homo sapiens 42-47 30209589-6 2018 During phase I, no dose-limiting toxicities were shown in three patients who received GM.CD40L.CCL21. gm 86-88 CD40 ligand Homo sapiens 89-94 30209589-6 2018 During phase I, no dose-limiting toxicities were shown in three patients who received GM.CD40L.CCL21. gm 86-88 C-C motif chemokine ligand 21 Homo sapiens 95-100 30209589-9 2018 For GM.CD40L versus GM.CD40L.CCL21, the most common treatment-related adverse events (TRAEs) were grade 1/2 injection site reaction (51.4% versus 61.1%) and grade 1/2 fatigue (35.1% versus 47.2%). gm 4-6 CD40 ligand Homo sapiens 7-12 30422398-6 2018 The interactions between this motif in the skeletal muscle-specific GM and PP1C have been revealed by structural studies. gm 68-70 protein phosphatase 1 catalytic subunit gamma Homo sapiens 75-79 30422398-8 2018 In this study, we found that residues next to the RVxF motif in GM also mediate interactions to PP1C and revealed the mechanism of the interaction by structural studies. gm 64-66 protein phosphatase 1 catalytic subunit gamma Homo sapiens 96-100 30422398-9 2018 Sequence analysis revealed that the PP1C-binding region in GM is highly conserved among G subunits. gm 59-61 protein phosphatase 1 catalytic subunit gamma Homo sapiens 36-40 30443599-7 2018 Our data provide the molecular basis for PP1 regulation by GM and reveal how PP1-mediated dephosphorylation is driven by scaffolding-based substrate recruitment. gm 59-61 inorganic pyrophosphatase 1 Homo sapiens 41-44 30443599-7 2018 Our data provide the molecular basis for PP1 regulation by GM and reveal how PP1-mediated dephosphorylation is driven by scaffolding-based substrate recruitment. gm 59-61 inorganic pyrophosphatase 1 Homo sapiens 77-80 30269275-8 2018 The GM ratios (90% confidence interval; CI) for Cmax, AUCinf, and AUClast were 1.015 (0.946-1.088), 0.979 (0.914-1.049), and 0.995 (0.941-1.053) for ABP 215 versus bevacizumab. gm 4-6 amine oxidase copper containing 1 Homo sapiens 149-152 30206828-8 2018 The GM for the antioxidant enzymes was 198.68 U/mL for GPx, 38.96 U/g Hb for GR, 94.78 U/mL for SOD, and 69.77 U/g Hb for CAT. gm 4-6 glutathione-disulfide reductase Homo sapiens 77-79 30206828-8 2018 The GM for the antioxidant enzymes was 198.68 U/mL for GPx, 38.96 U/g Hb for GR, 94.78 U/mL for SOD, and 69.77 U/g Hb for CAT. gm 4-6 catalase Homo sapiens 122-125 29707834-3 2018 PURPOSE: To evaluate the applicability of a semiautomatic algorithm provided by ITK-SNAP in classification mode (CLASS) for segmenting cervical spinal cord GM, WM in MRI images and analyzing DTI parameters. gm 156-158 IL2 inducible T cell kinase Homo sapiens 80-83 30420889-6 2018 Mycn is increased in multipotent progenitors and in the pre-GM compartment of myeloid progenitors in the ITD mice while the expression of several genes in the tumor suppressor Mxd family, including Mxd1, Mxd2, and Mxd4, is concomitantly downregulated, as well as the expression of the Mxd-related gene Mnt and the transcriptional activator Miz-1. gm 60-62 v-myc avian myelocytomatosis viral related oncogene, neuroblastoma derived Mus musculus 0-4 29909072-4 2018 In this study, we found that GM-DCs of inflammatory nature were more phagocytic, potent at inducing Th2 and Th17 differentiation, and had longer survival rate, whereas FL-DCs of steady state characters were stronger T cell activator and better at directing Th1 differentiation. gm 29-31 negative elongation factor complex member C/D Homo sapiens 108-111 30007720-8 2018 The results indicated that DPPIV enzyme hydrolyzed food-derived opioids (from 0.1 mM to 2 mM), BCM (33.42% for 2 mM), SM (83.81% for 2 mM), and GM (45.73% for 2 mM) in vitro. gm 144-146 dipeptidyl peptidase 4 Homo sapiens 27-32 29879453-8 2018 Several significant individual and epistatic effects of GM, KM, and FcgammaR genotypes on anti-GARP antibody responsiveness were noted in both patients and controls. gm 56-58 leucine rich repeat containing 32 Homo sapiens 95-99 29036524-7 2018 The new spectra were used to improve the photon to neutron dose equivalent ratios from some earlier work at NPL with GM tubes and EPDs. gm 117-119 N-acetylneuraminate pyruvate lyase Homo sapiens 108-111 30441263-5 2018 Results demonstrated significant group differences on GM and FC in hippocampus, temporal gyrus and cerebellum between SZ and HC, which are also significantly correlated with SNPs residing in genes like GABBR2, SATB2, CACNA1C, PDE4B, involved in pathways of cell junction, synapse and neuron projection. gm 54-56 gamma-aminobutyric acid type B receptor subunit 2 Homo sapiens 202-208 30441263-5 2018 Results demonstrated significant group differences on GM and FC in hippocampus, temporal gyrus and cerebellum between SZ and HC, which are also significantly correlated with SNPs residing in genes like GABBR2, SATB2, CACNA1C, PDE4B, involved in pathways of cell junction, synapse and neuron projection. gm 54-56 SATB homeobox 2 Homo sapiens 210-215 30441263-5 2018 Results demonstrated significant group differences on GM and FC in hippocampus, temporal gyrus and cerebellum between SZ and HC, which are also significantly correlated with SNPs residing in genes like GABBR2, SATB2, CACNA1C, PDE4B, involved in pathways of cell junction, synapse and neuron projection. gm 54-56 calcium voltage-gated channel subunit alpha1 C Homo sapiens 217-224 30441263-5 2018 Results demonstrated significant group differences on GM and FC in hippocampus, temporal gyrus and cerebellum between SZ and HC, which are also significantly correlated with SNPs residing in genes like GABBR2, SATB2, CACNA1C, PDE4B, involved in pathways of cell junction, synapse and neuron projection. gm 54-56 phosphodiesterase 4B Homo sapiens 226-231 29698786-4 2018 The ultrastructure of individual fibrinogen molecules was studied after heating or extended contact with the highly oriented pyrolytic graphite surface (HOPG) modified with oligoglycine-hydrocarbon graphite modifier (GM). gm 217-219 fibrinogen beta chain Homo sapiens 33-43 29500853-6 2018 For voriconazole and amphotericin B the GM-MIC values were acceptably low for the yeast phase (0.39 and 0.72 mug mL-1 ), while the mycelial phase showed values >=2-fold higher (8.76 and 1.88 mug mL-1 ), P < .05. gm 40-42 L1 cell adhesion molecule Mus musculus 113-117 29500853-6 2018 For voriconazole and amphotericin B the GM-MIC values were acceptably low for the yeast phase (0.39 and 0.72 mug mL-1 ), while the mycelial phase showed values >=2-fold higher (8.76 and 1.88 mug mL-1 ), P < .05. gm 40-42 L1 cell adhesion molecule Mus musculus 198-202 29954422-10 2018 Among them, PGK1 and G6PD were determined as optimal glucose metabolic (GM) markers for CTCs. gm 72-74 phosphoglycerate kinase 1 Homo sapiens 12-16 29954422-10 2018 Among them, PGK1 and G6PD were determined as optimal glucose metabolic (GM) markers for CTCs. gm 72-74 glucose-6-phosphate dehydrogenase Homo sapiens 21-25 29669828-0 2018 Bovine Herpesvirus 1 UL49.5 Interacts with gM and VP22 To Ensure Virus Cell-to-Cell Spread and Virion Incorporation: Novel Role for VP22 in gM-Independent UL49.5 Virion Incorporation. gm 43-45 envelope glycoprotein N Bovine alphaherpesvirus 1 21-27 29669828-0 2018 Bovine Herpesvirus 1 UL49.5 Interacts with gM and VP22 To Ensure Virus Cell-to-Cell Spread and Virion Incorporation: Novel Role for VP22 in gM-Independent UL49.5 Virion Incorporation. gm 140-142 envelope glycoprotein N Bovine alphaherpesvirus 1 21-27 29669828-0 2018 Bovine Herpesvirus 1 UL49.5 Interacts with gM and VP22 To Ensure Virus Cell-to-Cell Spread and Virion Incorporation: Novel Role for VP22 in gM-Independent UL49.5 Virion Incorporation. gm 140-142 envelope glycoprotein N Bovine alphaherpesvirus 1 155-161 29669828-4 2018 To identify which cysteine residue is required for the formation of the UL49.5/gM complex and to characterize the functional significance of the UL49.5/gM complex, we constructed and analyzed C42S and C78S substitution mutants in either a BHV-1 wild type (wt) or BHV-1 UL49.5 cytoplasmic tail-null (CT-null) virus background. gm 79-81 envelope glycoprotein N Bovine alphaherpesvirus 1 72-78 29669828-5 2018 The results demonstrated that BHV-1 UL49.5 residue C42 but not C78 was essential for the formation of the covalently linked functional UL49.5/gM complex, gM maturation in the Golgi compartment, and efficient cell-to-cell spread of the virus. gm 142-144 tegument protein VP22 Bovine alphaherpesvirus 1 36-40 29669828-5 2018 The results demonstrated that BHV-1 UL49.5 residue C42 but not C78 was essential for the formation of the covalently linked functional UL49.5/gM complex, gM maturation in the Golgi compartment, and efficient cell-to-cell spread of the virus. gm 142-144 tegument protein VP22 Bovine alphaherpesvirus 1 135-139 29669828-5 2018 The results demonstrated that BHV-1 UL49.5 residue C42 but not C78 was essential for the formation of the covalently linked functional UL49.5/gM complex, gM maturation in the Golgi compartment, and efficient cell-to-cell spread of the virus. gm 154-156 tegument protein VP22 Bovine alphaherpesvirus 1 36-40 29669828-11 2018 UL49.5 also forms a covalently linked complex with gM. gm 51-53 envelope glycoprotein N Bovine alphaherpesvirus 1 0-6 29669828-12 2018 The results of this study demonstrate that UL49.5 regulates gM maturation and virus cell-to-cell spread since gM maturation in the Golgi compartment depends on covalently linked UL49.5/gM complex. gm 60-62 envelope glycoprotein N Bovine alphaherpesvirus 1 43-49 29669828-12 2018 The results of this study demonstrate that UL49.5 regulates gM maturation and virus cell-to-cell spread since gM maturation in the Golgi compartment depends on covalently linked UL49.5/gM complex. gm 110-112 envelope glycoprotein N Bovine alphaherpesvirus 1 43-49 29669828-12 2018 The results of this study demonstrate that UL49.5 regulates gM maturation and virus cell-to-cell spread since gM maturation in the Golgi compartment depends on covalently linked UL49.5/gM complex. gm 110-112 envelope glycoprotein N Bovine alphaherpesvirus 1 178-184 29669828-12 2018 The results of this study demonstrate that UL49.5 regulates gM maturation and virus cell-to-cell spread since gM maturation in the Golgi compartment depends on covalently linked UL49.5/gM complex. gm 110-112 envelope glycoprotein N Bovine alphaherpesvirus 1 43-49 29669828-12 2018 The results of this study demonstrate that UL49.5 regulates gM maturation and virus cell-to-cell spread since gM maturation in the Golgi compartment depends on covalently linked UL49.5/gM complex. gm 110-112 envelope glycoprotein N Bovine alphaherpesvirus 1 178-184 29669828-16 2018 The putative UL49.5 CT-VP22 interaction is essential for a gM-independent UL49.5 virion incorporation and is revealed when UL49.5 and gM are not linked. gm 59-61 envelope glycoprotein N Bovine alphaherpesvirus 1 13-19 29669828-16 2018 The putative UL49.5 CT-VP22 interaction is essential for a gM-independent UL49.5 virion incorporation and is revealed when UL49.5 and gM are not linked. gm 59-61 envelope glycoprotein N Bovine alphaherpesvirus 1 74-80 29669828-16 2018 The putative UL49.5 CT-VP22 interaction is essential for a gM-independent UL49.5 virion incorporation and is revealed when UL49.5 and gM are not linked. gm 59-61 envelope glycoprotein N Bovine alphaherpesvirus 1 74-80 29669828-16 2018 The putative UL49.5 CT-VP22 interaction is essential for a gM-independent UL49.5 virion incorporation and is revealed when UL49.5 and gM are not linked. gm 134-136 envelope glycoprotein N Bovine alphaherpesvirus 1 13-19 29669828-16 2018 The putative UL49.5 CT-VP22 interaction is essential for a gM-independent UL49.5 virion incorporation and is revealed when UL49.5 and gM are not linked. gm 134-136 envelope glycoprotein N Bovine alphaherpesvirus 1 74-80 29669828-16 2018 The putative UL49.5 CT-VP22 interaction is essential for a gM-independent UL49.5 virion incorporation and is revealed when UL49.5 and gM are not linked. gm 134-136 envelope glycoprotein N Bovine alphaherpesvirus 1 74-80 29669828-17 2018 Therefore, UL49.5 virion incorporation is mediated by UL49.5-gM complex interaction and through a gM-independent interaction between UL49.5 and VP22. gm 61-63 envelope glycoprotein N Bovine alphaherpesvirus 1 11-17 29669828-17 2018 Therefore, UL49.5 virion incorporation is mediated by UL49.5-gM complex interaction and through a gM-independent interaction between UL49.5 and VP22. gm 61-63 envelope glycoprotein N Bovine alphaherpesvirus 1 54-60 29669828-17 2018 Therefore, UL49.5 virion incorporation is mediated by UL49.5-gM complex interaction and through a gM-independent interaction between UL49.5 and VP22. gm 61-63 envelope glycoprotein N Bovine alphaherpesvirus 1 54-60 29669828-17 2018 Therefore, UL49.5 virion incorporation is mediated by UL49.5-gM complex interaction and through a gM-independent interaction between UL49.5 and VP22. gm 98-100 envelope glycoprotein N Bovine alphaherpesvirus 1 11-17 29847803-3 2018 Through integrated analyses of enhancer dynamics, transcription factor binding, and proximal gene expression during successive stages of murine GM-lineage differentiation, we unravel the distinct kinetics by which PU.1 and CEBPA coordinate GM enhancer activity. gm 144-146 spleen focus forming virus (SFFV) proviral integration oncogene Mus musculus 214-218 29847803-3 2018 Through integrated analyses of enhancer dynamics, transcription factor binding, and proximal gene expression during successive stages of murine GM-lineage differentiation, we unravel the distinct kinetics by which PU.1 and CEBPA coordinate GM enhancer activity. gm 144-146 CCAAT/enhancer binding protein (C/EBP), alpha Mus musculus 223-228 29702737-4 2018 The ICT processes, responsible for the solvatochromism, also lead to interesting non-linear optical (NLO) properties: namely great two photon absorption cross-sections (hundreds of GM), investigated by the Two Photon Excited Fluorescence (TPEF) technique, and large second order hyperpolarizability coefficients, estimated through a convenient solvatochromic method. gm 181-183 transmembrane protein with EGF like and two follistatin like domains 2 Homo sapiens 239-243 29256086-10 2018 SEM revealed that D-KAT scores were directly associated with GM-ITE scores (ss = 0.37, 95% CI: 0.34-0.41) and indirectly associated with ED rotations (ss = 0.06, 95% CI: 0.02-0.10), inpatient caseload (ss = 0.04, 95% CI: 0.003-0.08), and average daily minutes of study (ss = 0.13, 95% CI: 0.09-0.17). gm 61-63 thiosulfate sulfurtransferase like domain containing 1 Homo sapiens 20-23 29307601-7 2018 However, some distinct differences in IgE binding patterns were observed among the non-GM commercial soybean lines and between different locations, highlighting the inherent variability in endogenous allergenic proteins. gm 87-89 immunoglobulin heavy constant epsilon Homo sapiens 38-41 29074302-6 2018 However, in patients and controls, particular GM, KM, and FcgammaR genotypes-individually or epistatically-were significantly associated with the levels of anti-MUC1 IgG antibodies in a racially restricted manner. gm 46-48 mucin 1, cell surface associated Homo sapiens 161-165 29093081-5 2018 Finally, we show that an HSV-1 gM mutant lacking the majority of the C-terminal domain (HA-gM[Delta345-473]) restricted neither gp160 processing nor the release of infectious virus. gm 31-33 Envelope surface glycoprotein gp160, precursor Human immunodeficiency virus 1 128-133 28760079-3 2018 BMP2 was dropped onto the GM freeze dried, followed by leaving at 25 C to obtain GM containing BMP2 (GM-BMP2). gm 82-84 bone morphogenetic protein 2 Mus musculus 102-109 29242837-7 2017 A genetic screen in C. albicans suggested that TOR is the molecular target of the antifungal molecule(s) of the GM. gm 112-114 RAR related orphan receptor C Homo sapiens 47-50 28811265-5 2017 Gm gamma-TMT belongs to Class I Methyl Transferases that have a Rossmann-like fold which consists of a seven-stranded beta sheet joined by alpha helices. gm 0-2 gamma-tocopherol methyltransferase Glycine max 3-12 28811265-11 2017 This study has laid the foundation to unveil the biochemical understanding of Gm gamma-TMT enzyme which can be further explored by studying its kinetic behaviour, substrate specificity and its interaction with other biomolecules. gm 78-80 gamma-tocopherol methyltransferase Glycine max 81-90 28922371-7 2017 High sensitivity C-reactive protein was the most elevated biomarker among current smokers when compared to never smokers [GM ratio = 1.39 (95% CI: 1.23, 1.57); p <0.001]. gm 122-124 C-reactive protein Homo sapiens 17-35 28744261-8 2017 The silver complexes were the most effective drugs (overall geometric mean of the minimum inhibitory concentration (GM-MIC) ranged from 0.26 to 2.16 muM), followed by the manganese (overall GM-MIC ranged from 0.87 to 10.71 muM) and copper (overall GM-MIC ranged from 3.37 to >72 muM) chelates. gm 116-118 latexin Homo sapiens 149-152 28495939-11 2017 CONCLUSIONS: APOE*E4-related GM loss in the posterior cingulate cortex (an area involved in Alzheimer disease pathology) was found only in those elderly controls who subsequently developed subtle cognitive decline but not in cognitively stable controls. gm 29-31 apolipoprotein E Homo sapiens 13-20 28648004-5 2017 Results: Compared with the other three groups, the muscle weights of GM and Sol in the BT-injection group significantly decreased.MyHC type ratioof GM and Sol on the right side in the BT-injection group also significantlychanged.According to subgroup comparisons, MyHC type ratio of GM and Sol on the injectedside in the 2w-BT subgroup was significant different from that of the 8w-BT subgroup.MyHC type ratios of GM on the non-injected side in the BT injection group also changed more than normal control group.The change of MyHC type ratio of non-injected GM was significantly higher in the 2 week-BT subgroup than that in the 8-week subgroup. gm 148-150 myosin heavy chain 13 Rattus norvegicus 130-134 28648004-5 2017 Results: Compared with the other three groups, the muscle weights of GM and Sol in the BT-injection group significantly decreased.MyHC type ratioof GM and Sol on the right side in the BT-injection group also significantlychanged.According to subgroup comparisons, MyHC type ratio of GM and Sol on the injectedside in the 2w-BT subgroup was significant different from that of the 8w-BT subgroup.MyHC type ratios of GM on the non-injected side in the BT injection group also changed more than normal control group.The change of MyHC type ratio of non-injected GM was significantly higher in the 2 week-BT subgroup than that in the 8-week subgroup. gm 148-150 myosin heavy chain 13 Rattus norvegicus 130-134 28648004-5 2017 Results: Compared with the other three groups, the muscle weights of GM and Sol in the BT-injection group significantly decreased.MyHC type ratioof GM and Sol on the right side in the BT-injection group also significantlychanged.According to subgroup comparisons, MyHC type ratio of GM and Sol on the injectedside in the 2w-BT subgroup was significant different from that of the 8w-BT subgroup.MyHC type ratios of GM on the non-injected side in the BT injection group also changed more than normal control group.The change of MyHC type ratio of non-injected GM was significantly higher in the 2 week-BT subgroup than that in the 8-week subgroup. gm 148-150 myosin heavy chain 13 Rattus norvegicus 130-134 28648004-5 2017 Results: Compared with the other three groups, the muscle weights of GM and Sol in the BT-injection group significantly decreased.MyHC type ratioof GM and Sol on the right side in the BT-injection group also significantlychanged.According to subgroup comparisons, MyHC type ratio of GM and Sol on the injectedside in the 2w-BT subgroup was significant different from that of the 8w-BT subgroup.MyHC type ratios of GM on the non-injected side in the BT injection group also changed more than normal control group.The change of MyHC type ratio of non-injected GM was significantly higher in the 2 week-BT subgroup than that in the 8-week subgroup. gm 148-150 myosin heavy chain 13 Rattus norvegicus 130-134 28727051-11 2017 The TT genotype at the gene increased MUFA ( < 0.05) in all tissues (21.4 vs. 19.5% in the liver, 55.0 vs. 53.1% in the LM, 53.9 vs. 51.7% in the GM, and 48.0 vs. 46.7% in SF for TT and CC genotypes, respectively). gm 149-151 Monounsaturated fatty acid percentage Sus scrofa 38-42 28402274-3 2017 Meantime, GM attenuated serum and HepG2 cell supernatant levels of TNF-alpha, IL-6, IL-1beta, SOD and MDA. gm 10-12 tumor necrosis factor Homo sapiens 67-76 28402274-3 2017 Meantime, GM attenuated serum and HepG2 cell supernatant levels of TNF-alpha, IL-6, IL-1beta, SOD and MDA. gm 10-12 interleukin 6 Homo sapiens 78-82 28402274-3 2017 Meantime, GM attenuated serum and HepG2 cell supernatant levels of TNF-alpha, IL-6, IL-1beta, SOD and MDA. gm 10-12 interleukin 1 beta Homo sapiens 84-92 28402274-5 2017 Taken together, our results suggested that GM showed beneficial effect on CP-induced liver injury through NF-kappaB-mediated inflammation and PI3K/Akt/mTOR/p70S6K/4EBP1 axis-mediated autophagy in vivo and in vitro. gm 43-45 nuclear factor kappa B subunit 1 Homo sapiens 106-115 28402274-5 2017 Taken together, our results suggested that GM showed beneficial effect on CP-induced liver injury through NF-kappaB-mediated inflammation and PI3K/Akt/mTOR/p70S6K/4EBP1 axis-mediated autophagy in vivo and in vitro. gm 43-45 AKT serine/threonine kinase 1 Homo sapiens 147-150 28402274-5 2017 Taken together, our results suggested that GM showed beneficial effect on CP-induced liver injury through NF-kappaB-mediated inflammation and PI3K/Akt/mTOR/p70S6K/4EBP1 axis-mediated autophagy in vivo and in vitro. gm 43-45 mechanistic target of rapamycin kinase Homo sapiens 151-155 28402274-5 2017 Taken together, our results suggested that GM showed beneficial effect on CP-induced liver injury through NF-kappaB-mediated inflammation and PI3K/Akt/mTOR/p70S6K/4EBP1 axis-mediated autophagy in vivo and in vitro. gm 43-45 ribosomal protein S6 kinase B1 Homo sapiens 156-162 28402274-5 2017 Taken together, our results suggested that GM showed beneficial effect on CP-induced liver injury through NF-kappaB-mediated inflammation and PI3K/Akt/mTOR/p70S6K/4EBP1 axis-mediated autophagy in vivo and in vitro. gm 43-45 eukaryotic translation initiation factor 4E binding protein 1 Homo sapiens 163-168 28124139-7 2017 The GM (and 95% confidence interval) concentration of the pro-inflammatory cytokine TNF-alpha decreased from 2.2 (2.0-2.4) at baseline to 2.0 pg/mL (1.8-2.2) (p = 0.02) at follow-up among the TCW. gm 4-6 tumor necrosis factor Homo sapiens 84-93 30271843-3 2017 EpH4 and/or 3T3L1 cells were cultured with or without the FN-treated GM in round U-bottom wells of 96-multiwell culture plates which had been coated with poly (vinyl alcohol) (PVA) to allow the cells to form their aggregates. gm 69-71 fibronectin 1 Mus musculus 58-60 30271843-4 2017 On the other hand, EpH4 cells were precultured with the FN-treated GM, and then continued to culture with 3T3L1 cells in the same condition described above. gm 67-69 fibronectin 1 Mus musculus 56-58 30271843-6 2017 When 3T3L1 cells were incubated with the original and GM-preincubated EpH4 cells in the presence of both the FN-treated GM, the number of alive cells in the aggregates was significantly high compared with that for the absence of FN-treated GM. gm 54-56 fibronectin 1 Mus musculus 109-111 30271843-6 2017 When 3T3L1 cells were incubated with the original and GM-preincubated EpH4 cells in the presence of both the FN-treated GM, the number of alive cells in the aggregates was significantly high compared with that for the absence of FN-treated GM. gm 54-56 fibronectin 1 Mus musculus 229-231 30271843-6 2017 When 3T3L1 cells were incubated with the original and GM-preincubated EpH4 cells in the presence of both the FN-treated GM, the number of alive cells in the aggregates was significantly high compared with that for the absence of FN-treated GM. gm 120-122 fibronectin 1 Mus musculus 109-111 30271843-6 2017 When 3T3L1 cells were incubated with the original and GM-preincubated EpH4 cells in the presence of both the FN-treated GM, the number of alive cells in the aggregates was significantly high compared with that for the absence of FN-treated GM. gm 120-122 fibronectin 1 Mus musculus 109-111 30271843-7 2017 In addition, higher beta-casein expression level of EpH4 cells in EpH4/3T3L1 cells aggregates in the presence of FN-treated GM was observed than that of cells in the absence of FN-treated GM. gm 124-126 fibronectin 1 Mus musculus 113-115 30271843-7 2017 In addition, higher beta-casein expression level of EpH4 cells in EpH4/3T3L1 cells aggregates in the presence of FN-treated GM was observed than that of cells in the absence of FN-treated GM. gm 188-190 fibronectin 1 Mus musculus 177-179 30271843-8 2017 Laminin secretion was also promoted for the cells aggregates cultured with FN-treated GM. gm 86-88 fibronectin 1 Mus musculus 75-77 30271843-9 2017 It is concluded that the presence of FN-treated GM in the EpH4/3T3L1 cells aggregates gave a better condition to cells, resulting in an enhanced generation of beta-casein from EpH4 cells in the aggregates. gm 48-50 fibronectin 1 Mus musculus 37-39 28159742-7 2017 Here, we address which CSF-1-activated pathways are involved in transmitting the lineage-instructive signal in primary bone marrow-derived GM progenitors. gm 139-141 colony stimulating factor 1 Homo sapiens 23-28 27873002-6 2017 Furthermore, these regioselective GM MS signals were also found to have highly heterogeneous distributions within the GM3-IHC staining. gm 34-36 granulocyte macrophage antigen 3 Mus musculus 118-121 28058445-10 2017 Cabazitaxel clearance normalized to body surface area (CL/BSA) was lower in C-1 (geometric mean [GM] 13.4 L/h/m2) than expected (26.4 L/h/m2), but similar in C-2 (23.5 L/h/m2) and C-3 (27.9 L/h/m2). gm 97-99 heterogeneous nuclear ribonucleoprotein C Homo sapiens 76-79 28058445-12 2017 Compared with C-2, CL/BSA increased 19% in C-3 (GM ratio 1.19; 90% CI 0.74-1.91), but decreased 23% in C-4 (0.77; 0.39-1.53). gm 48-50 complement C3 Homo sapiens 43-46 28060552-9 2017 These findings suggest a therapeutic potential of GM root extract against B19-NS1-exacerbated liver inflammation in SLE. gm 50-52 influenza virus NS1A binding protein Mus musculus 78-81 27653809-6 2017 Moreover, Kox was linearly correlated with gm and both were closely related to mesophyll structural traits. gm 43-45 NADPH oxidase 4 Homo sapiens 10-13 27452639-8 2016 GM analysis revealed that n-3 PUFA supplementation increased renal steroid hormone and proteolytic metabolite levels in PMW. gm 0-2 pumilio RNA binding family member 3 Homo sapiens 30-34 27734595-7 2016 Patients in the GM group displayed significantly higher CD56 and CD49e expression than those in the non-GM group (t-test, P = 0.027 and 0.042). gm 16-18 neural cell adhesion molecule 1 Homo sapiens 56-60 27734595-7 2016 Patients in the GM group displayed significantly higher CD56 and CD49e expression than those in the non-GM group (t-test, P = 0.027 and 0.042). gm 16-18 integrin subunit alpha 5 Homo sapiens 65-70 27678219-4 2016 Enforced expression of ID2 in GM-DCs (ID2-GM-DCs) suppressed their production of the proinflammatory cytokine tumor necrosis factor-alpha (TNF-alpha). gm 30-32 inhibitor of DNA binding 2 Mus musculus 23-26 27678219-4 2016 Enforced expression of ID2 in GM-DCs (ID2-GM-DCs) suppressed their production of the proinflammatory cytokine tumor necrosis factor-alpha (TNF-alpha). gm 30-32 inhibitor of DNA binding 2 Mus musculus 38-41 27678219-4 2016 Enforced expression of ID2 in GM-DCs (ID2-GM-DCs) suppressed their production of the proinflammatory cytokine tumor necrosis factor-alpha (TNF-alpha). gm 30-32 tumor necrosis factor Mus musculus 110-137 27678219-4 2016 Enforced expression of ID2 in GM-DCs (ID2-GM-DCs) suppressed their production of the proinflammatory cytokine tumor necrosis factor-alpha (TNF-alpha). gm 30-32 tumor necrosis factor Mus musculus 139-148 27220476-7 2016 In GM, we report somatic mutations in KIT and TP53. gm 3-5 KIT proto-oncogene, receptor tyrosine kinase Homo sapiens 38-41 27220476-7 2016 In GM, we report somatic mutations in KIT and TP53. gm 3-5 tumor protein p53 Homo sapiens 46-50 27100010-6 2016 Relatively high STC levels (0.06-2.35mug/g) detected in 52% of GM dust samples confirmed the presence of STC-producers, although this STC cannot be exclusively attributed to Aspergilli (Versicolores). gm 63-65 stanniocalcin 1 Homo sapiens 16-19 27100010-6 2016 Relatively high STC levels (0.06-2.35mug/g) detected in 52% of GM dust samples confirmed the presence of STC-producers, although this STC cannot be exclusively attributed to Aspergilli (Versicolores). gm 63-65 stanniocalcin 1 Homo sapiens 105-108 27100010-6 2016 Relatively high STC levels (0.06-2.35mug/g) detected in 52% of GM dust samples confirmed the presence of STC-producers, although this STC cannot be exclusively attributed to Aspergilli (Versicolores). gm 63-65 stanniocalcin 1 Homo sapiens 105-108 27334585-5 2016 In vaccinated animals, the GP85 mutant virus (GP85 DISC) induced an antibody response to important glycoprotein complexes considered neutralizing target antigens (gB, gH/gL/gO, and gM/gN). gm 181-183 GP85 Caviid betaherpesvirus 2 27-31 27334585-5 2016 In vaccinated animals, the GP85 mutant virus (GP85 DISC) induced an antibody response to important glycoprotein complexes considered neutralizing target antigens (gB, gH/gL/gO, and gM/gN). gm 181-183 GP85 Caviid betaherpesvirus 2 46-50 27317631-6 2016 Nevertheless, maximal force during tetanic contraction was ~40% greater in GM of Cav2(-/-) vs. WT mice (152 +- 14 vs. 110 +- 3 mN per square millimeter, respectively; P < 0.05). gm 75-77 caveolin 2 Mus musculus 81-85 27261579-5 2016 Likewise, GB and GM possess almost the same potency in attenuating LPS-induced expression and activation of nuclear factor kappa B (p65) and subsequent increases in tumor necrosis factor-alpha mRNA levels. gm 17-19 v-rel reticuloendotheliosis viral oncogene homolog A (avian) Mus musculus 132-135 27261579-8 2016 Finally, though GB and GM at equivalent dosages appear to reduce LPS-induced IL-1beta mRNA and protein levels and IL-10 protein levels to the same degree, GM is more potent than GB to attenuate the IL-10 mRNA levels. gm 23-25 interleukin 1 beta Mus musculus 77-85 27261579-8 2016 Finally, though GB and GM at equivalent dosages appear to reduce LPS-induced IL-1beta mRNA and protein levels and IL-10 protein levels to the same degree, GM is more potent than GB to attenuate the IL-10 mRNA levels. gm 23-25 interleukin 10 Mus musculus 114-119 27261579-8 2016 Finally, though GB and GM at equivalent dosages appear to reduce LPS-induced IL-1beta mRNA and protein levels and IL-10 protein levels to the same degree, GM is more potent than GB to attenuate the IL-10 mRNA levels. gm 23-25 interleukin 10 Mus musculus 198-203 27261579-8 2016 Finally, though GB and GM at equivalent dosages appear to reduce LPS-induced IL-1beta mRNA and protein levels and IL-10 protein levels to the same degree, GM is more potent than GB to attenuate the IL-10 mRNA levels. gm 155-157 interleukin 10 Mus musculus 198-203 27356476-4 2016 RESULTS: (1) Immunohistochemical detection showed the positive rate of Klotho protein was significantly higher in the placenta of GM (93%,28/30) than in the GN (73%,22/30; P<0.05). gm 130-132 klotho Homo sapiens 71-77 27356476-6 2016 (2) Real-time fluorescent quantitative PCR showed the Klotho mRNA expression was significantly higher in the placenta of GM (4.3 +- 3.1) than in the GN (2.1 +- 2.4; P<0.05). gm 121-123 klotho Homo sapiens 54-60 27356476-8 2016 (3) Western blot showed the Klotho protein expression was significantly higher in the placenta of GM (1.27+-0.90) than in the GN (0.64+-0.24; P<0.05). gm 98-100 klotho Homo sapiens 28-34 27255083-8 2016 Targeting TRPV1 using either GM mice or a pharmacological inhibitor tended to decrease IgE levels, airway inflammation and lung function changes. gm 29-31 transient receptor potential cation channel, subfamily V, member 1 Mus musculus 10-15 27050442-6 2016 RESULTS: MD was significantly higher in the cerebellar GM of ET total group (10.39 +- 0.87) in comparison with HC (9.90 +- 0.71) (p = 0.0027). gm 55-57 ret proto-oncogene Rattus norvegicus 61-63 27101723-8 2016 We concluded that the variability of gm offers a potential target for improving leaf AN and WUE simultaneously by the regulation of anatomical structure and GhAQP1. gm 37-39 aquaporin PIP1-3-like Gossypium hirsutum 157-163 26450539-4 2016 Here, we present an approach that allows obtaining CSF-suppressed images with improved contrast between lesions, WM and GM without strongly penalizing SNR. gm 120-122 colony stimulating factor 2 Homo sapiens 51-54 26450539-8 2016 RESULTS: The GM-WM contrast-to-noise ratio was by 133% higher in FLAIR(2) than in FLAIR and improved between lesions and WM by 31%, 93%, and 158% compared with T2, DIR, and FLAIR, respectively. gm 13-15 arginine vasopressin receptor 2 Homo sapiens 164-167 26773383-2 2016 We report here that the long predicted cytoplasmic tail of gM is not required for complex formation and that it interacts with the cellular protein p32, which has been reported to be involved in nuclear egress of human cytomegalovirus and herpes simplex virus. gm 59-61 inhibitor of growth family member 2 Homo sapiens 148-151 26725433-9 2016 YKS and some alkaloids (corynoxeine: CX, geissoschizine methyl ether: GM) in Uncaria hook, a constituent herb of YKS, also inhibited COMT activity, indicating that the augmenting effect of YKS on L-DOPA-induced DA production in 5-HT synthetic cells was due to the inhibition of COMT by CX and GM. gm 70-72 catechol-O-methyltransferase Rattus norvegicus 133-137 26725433-10 2016 Our results suggest that YKS facilitates the DA supplemental effect of L-DOPA, and that COMT inhibition by CX and GM contributes, at least in part, to the effects of YKS. gm 114-116 catechol-O-methyltransferase Rattus norvegicus 88-92 25977048-2 2015 Desiree) tubers, which have been genetically modified (GM) to reduce glycoalkaloid content, via the independent down-regulation of three genes SGT1, SGT2 and SGT3 known to be involved in glycoalkaloid biosynthesis. gm 55-57 protein SGT1 homolog Solanum tuberosum 143-147 25977048-2 2015 Desiree) tubers, which have been genetically modified (GM) to reduce glycoalkaloid content, via the independent down-regulation of three genes SGT1, SGT2 and SGT3 known to be involved in glycoalkaloid biosynthesis. gm 55-57 UDP-glucose flavonoid 3-O-glucosyltransferase 7-like Solanum tuberosum 149-153 25977048-2 2015 Desiree) tubers, which have been genetically modified (GM) to reduce glycoalkaloid content, via the independent down-regulation of three genes SGT1, SGT2 and SGT3 known to be involved in glycoalkaloid biosynthesis. gm 55-57 rhamnose:beta-solanine/beta-chaconine rhamnosyltransferase Solanum tuberosum 158-162 26371790-5 2015 After 4 weeks, TGF-beta1 release from GM within the cartilage phase promotes formation of a glycosaminoglycan- and type II collagen-rich matrix, and has a local inhibitory effect on osteogenesis. gm 38-40 transforming growth factor beta 1 Homo sapiens 15-24 26382248-0 2015 The decoy Fcgamma receptor encoded by the cytomegalovirus UL119-UL118 gene has differential affinity to IgG proteins expressing different GM allotypes. gm 138-140 Fc gamma receptor Ia Homo sapiens 10-26 26382248-0 2015 The decoy Fcgamma receptor encoded by the cytomegalovirus UL119-UL118 gene has differential affinity to IgG proteins expressing different GM allotypes. gm 138-140 membrane glycoprotein UL119 Human betaherpesvirus 5 58-63 25633990-10 2015 The CK positivity in GM may be due to CK molecules other than CK34BE12, CK5, CK6, CK7, CK8, CK14, CK18, CK19 and CK20. gm 21-23 keratin 20 Homo sapiens 113-117 25833054-4 2015 Furthermore, unlabeled gM, but not glycoprotein B, was detected by Western blotting in isolated axons during Us9-null PRV infection. gm 23-25 membrane protein US9 Suid alphaherpesvirus 1 109-112 25326496-8 2015 CONCLUSIONS: GM allotypes contribute to humoral immunity to EGFR in glioblastoma. gm 13-15 epidermal growth factor receptor Homo sapiens 60-64 26027394-3 2015 RESULTS: Comparison of the expression of uPA in the GM vascular endothelium and epithelial vessels revealed no significant differences in the patient groups. gm 52-54 plasminogen activator, urokinase Homo sapiens 41-44 26027394-4 2015 The level of PAI-1 in the GM vessels was statistically significantly higher in the control group than in the groups of patients with PHG and PH without PHG. gm 26-28 serpin family E member 1 Homo sapiens 13-18 26027394-7 2015 CONCLUSION: The predominance of uPA over PAI-1 in the GM vessels and epithelial cells can play a role in the development of GM bleeding. gm 54-56 plasminogen activator, urokinase Homo sapiens 32-35 26027394-7 2015 CONCLUSION: The predominance of uPA over PAI-1 in the GM vessels and epithelial cells can play a role in the development of GM bleeding. gm 54-56 serpin family E member 1 Homo sapiens 41-46 25102944-6 2015 Cultured JMML CD34(+)CD38(-) cells expressed CD117, CD116, c-mpl, CD123, CD90, but not CXCR4, and formed GM and erythroid colonies. gm 105-107 CD34 antigen Mus musculus 14-18 25102944-6 2015 Cultured JMML CD34(+)CD38(-) cells expressed CD117, CD116, c-mpl, CD123, CD90, but not CXCR4, and formed GM and erythroid colonies. gm 105-107 CD38 antigen Mus musculus 21-25 25949865-11 2015 Adv/GM treatment and they generated greater HER2-specific T-cell responses. gm 4-6 erb-b2 receptor tyrosine kinase 2 Mus musculus 44-48 26064086-5 2015 Then, the two principally studied strategies are discussed (i) GM-LAB producing antioxidant enzymes and (ii) GM-LAB producing the anti-inflammatory cytokine IL-10. gm 109-111 interleukin 10 Homo sapiens 157-162 25403201-6 2014 Genetic correlations of GM and LM with SM were much lower: 0.13 to 0.19 for IMF and 0.10 to 0.54 for fatty acids. gm 24-26 IMF Sus scrofa 76-79 25403201-9 2014 Selection for IMF and C18:1 in GM is expected to lead to positive responses in IMF and C18:1 in LM and vice versa, although this can entail genetic lags of 20 to 45% in the muscle not directly selected for. gm 31-33 IMF Sus scrofa 14-17 25403201-9 2014 Selection for IMF and C18:1 in GM is expected to lead to positive responses in IMF and C18:1 in LM and vice versa, although this can entail genetic lags of 20 to 45% in the muscle not directly selected for. gm 31-33 IMF Sus scrofa 79-82 25209806-3 2014 VAMP3 colocalized with the gM/gN complex at the trans-Golgi network and other compartments, possibly the late endosome in HHV-6-infected cells, and its expression gradually increased during the late phase of virus infection. gm 27-29 vesicle associated membrane protein 3 Homo sapiens 0-5 25209806-4 2014 Finally, VAMP3 was incorporated into mature virions and may be transported with the gM/gN complex. gm 84-86 vesicle associated membrane protein 3 Homo sapiens 9-14 25128573-1 2014 Recent studies in rat muscle fibres show that repetitive firing of action potentials causes changes in fibre resting membrane conductance (Gm) that reflect regulation of ClC-1 Cl(-) and KATP K(+) ion channels. gm 139-141 chloride voltage-gated channel 1 Rattus norvegicus 170-175 25016213-8 2014 B-Cd was significantly higher in women (GM 0.57mug/l) than in men (0.50mug/l) (p=0.007) and in smokers (GM 1.29mug/l) than in nonsmokers (GM 0.53mug/l) (p=<0.001) and in seniors from Prague (GM 0.60mug/l) compared to those from Teplice (GM 0.43mug/l) (p=<0.001). gm 40-42 cytochrome P450 family 4 subfamily V member 2 Homo sapiens 0-4 24965291-6 2014 The aim of the present study was to clarify the effects of GM adsorption to CA beads on TNF-alpha release in vitro. gm 59-61 tumor necrosis factor Homo sapiens 88-97 24965291-10 2014 The amount of TNF-alpha after incubation with CA beads positively correlated with GM adsorption ratio. gm 82-84 tumor necrosis factor Homo sapiens 14-23 24965291-11 2014 GM adsorption to CA beads induced a small amount of TNF-alpha release. gm 0-2 tumor necrosis factor Homo sapiens 52-61 24965291-12 2014 This is the first report on TNF-alpha release induced via GM adsorption stimuli. gm 58-60 tumor necrosis factor Homo sapiens 28-37 24965291-13 2014 The biological effects of TNF-alpha release during GM adsorption need to be clarified. gm 51-53 tumor necrosis factor Homo sapiens 26-35 24755879-3 2014 Simulation of "fluid" and "solid" inclusions were accurately detected on both the C1 and alpha 3D maps and in the C1 and alpha distributions over whole GM and WM. gm 152-154 heterogeneous nuclear ribonucleoprotein C Homo sapiens 114-126 24304136-3 2014 In this study, we aimed to determine whether particular GM and KM (kappa marker) allotypes were associated with antibody responsiveness to XAGE-1b, a highly immunogenic lung tumour-associated cancer-testis antigen. gm 56-58 X antigen family member 1A Homo sapiens 139-146 24304136-5 2014 The distribution of various GM phenotypes was significantly different between XAGE-1b antibody-positive and -negative patients (P = 0 023), as well as in the subgroup of XAGE-1b antigen-positive advanced NSCLC (P = 0 007). gm 28-30 X antigen family member 1A Homo sapiens 78-85 24462232-9 2014 Thus, an optimal PID controller gain set is successfully found within the GPMSOR and guarantees the OLUPTD processes with a pre-specified GM and PM as well as a minimum IAE or ISE. gm 138-140 metastasis associated 1 family member 2 Homo sapiens 17-20 24036099-4 2014 More importantly, GM-bilosomes were found capable of inducing mucosal immune response, i.e. sIgA titre in salivary and intestinal secretions as well as cell mediated immune response (IL-2 and IFN-gamma levels in spleen homogenate) which was not induced by i.m. gm 18-20 interleukin 2 Homo sapiens 183-187 24036099-4 2014 More importantly, GM-bilosomes were found capable of inducing mucosal immune response, i.e. sIgA titre in salivary and intestinal secretions as well as cell mediated immune response (IL-2 and IFN-gamma levels in spleen homogenate) which was not induced by i.m. gm 18-20 interferon gamma Homo sapiens 192-201 24239836-1 2013 The distinguished alternative GM allotypes localized in immunoglobulin constant heavy G chain IGHG (Fcgamma) (GM) genes on chromosome 14q32.3 define two unique variants of respectively IgG3, IgG1 and IgG2 subclasses, with different structures and functions. gm 30-32 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 185-189 24113258-5 2013 The density of Olig2 positive cells in the GM was lower in DS brains at early stages, then showed a transient increase contrasting controls. gm 43-45 oligodendrocyte transcription factor 2 Homo sapiens 15-20 24067975-3 2013 We show that gM but not gB or gD efficiently removes tetherin from the plasma membrane and can functionally substitute for the human immunodeficiency virus type 1 (HIV-1) Vpu protein, the prototypic viral tetherin antagonist, in rescuing HIV-1 release from tetherin-expressing cells. gm 13-15 bone marrow stromal cell antigen 2 Homo sapiens 53-61 24067975-3 2013 We show that gM but not gB or gD efficiently removes tetherin from the plasma membrane and can functionally substitute for the human immunodeficiency virus type 1 (HIV-1) Vpu protein, the prototypic viral tetherin antagonist, in rescuing HIV-1 release from tetherin-expressing cells. gm 13-15 bone marrow stromal cell antigen 2 Homo sapiens 205-213 24067975-3 2013 We show that gM but not gB or gD efficiently removes tetherin from the plasma membrane and can functionally substitute for the human immunodeficiency virus type 1 (HIV-1) Vpu protein, the prototypic viral tetherin antagonist, in rescuing HIV-1 release from tetherin-expressing cells. gm 13-15 bone marrow stromal cell antigen 2 Homo sapiens 205-213 24204965-6 2013 However, HNE strongly potentiated the membrane conductance increase (Gm) mediated by different long-chain fatty acids in UCP-containing and in UCP-free membranes and this suggest the involvement of both lipid-mediated and protein-mediated mechanisms with FA playing the central role. gm 69-71 uncoupling protein 1 (mitochondrial, proton carrier) Mus musculus 121-124 24204965-6 2013 However, HNE strongly potentiated the membrane conductance increase (Gm) mediated by different long-chain fatty acids in UCP-containing and in UCP-free membranes and this suggest the involvement of both lipid-mediated and protein-mediated mechanisms with FA playing the central role. gm 69-71 uncoupling protein 1 (mitochondrial, proton carrier) Mus musculus 143-146 24124601-5 2013 The immunophenotype of GM-IMs is F4/80(high) CD11b(high) CD11c(low) Ly6C(low). gm 23-25 integrin alpha M Mus musculus 45-50 24124601-5 2013 The immunophenotype of GM-IMs is F4/80(high) CD11b(high) CD11c(low) Ly6C(low). gm 23-25 integrin alpha X Mus musculus 57-62 24124601-5 2013 The immunophenotype of GM-IMs is F4/80(high) CD11b(high) CD11c(low) Ly6C(low). gm 23-25 lymphocyte antigen 6 complex, locus C1 Mus musculus 68-72 24124601-12 2013 GM-IMs showed enhanced expression of M2 macrophage markers such as Arg1 and Retnla following stimulation by Th2 cytokines IL-4 and IL-13. gm 0-2 arginase, liver Mus musculus 67-71 24124601-12 2013 GM-IMs showed enhanced expression of M2 macrophage markers such as Arg1 and Retnla following stimulation by Th2 cytokines IL-4 and IL-13. gm 0-2 interleukin 4 Mus musculus 122-126 24124601-12 2013 GM-IMs showed enhanced expression of M2 macrophage markers such as Arg1 and Retnla following stimulation by Th2 cytokines IL-4 and IL-13. gm 0-2 interleukin 13 Mus musculus 131-136 24124601-15 2013 When GM-IMs were injected into clodronate-treated mice, they induced resident macrophage proliferation by producing M-CSF. gm 5-7 colony stimulating factor 1 (macrophage) Mus musculus 116-121 23208772-6 2013 In GM, the density of vWF-immunoreactive segments with cross-sectional areas greater than 800 microm2 was higher in MDD. gm 3-5 von Willebrand factor Homo sapiens 22-25 23686376-5 2013 The most significant factor affecting the release of BMP-2 from composites was the loading phase of the growth factor: GM loading reduced the burst release, increased BMP-2 release during the later phases of the experiment, and increased the cumulative release in faster degrading samples. gm 119-121 bone morphogenetic protein 2 Homo sapiens 53-58 23686376-5 2013 The most significant factor affecting the release of BMP-2 from composites was the loading phase of the growth factor: GM loading reduced the burst release, increased BMP-2 release during the later phases of the experiment, and increased the cumulative release in faster degrading samples. gm 119-121 bone morphogenetic protein 2 Homo sapiens 167-172 23658364-9 2013 However, the realized selection intensity for IMF in GM denotes that the restriction on IMF was incomplete [-0.18; HPD95 (-0.36, +0.02)]. gm 53-55 IMF Sus scrofa 46-49 23644832-10 2013 In cabins with textile seats, the geometric mean (GM) concentrations of Fel d1, Can f1 and Equ cx were 5359 ng g(-1), 6067 ng g(-1), and 13 703 ng g(-1) (GM) respectively. gm 50-52 major allergen Can f 1 Canis lupus familiaris 80-86 23379433-7 2013 In white subjects (n = 263), GM 23-carriers had higher levels of anti-HER2 antibodies than non-carriers of this allele (p = 0 004). gm 29-31 erb-b2 receptor tyrosine kinase 2 Homo sapiens 70-74 23379433-12 2013 These racially restricted contributions of GM and FcgammaR genotypes to humoral immunity to HER2 have potential implications for immunotherapy of breast cancer. gm 43-45 erb-b2 receptor tyrosine kinase 2 Homo sapiens 92-96 25151822-15 2013 Our results showed an increase of both activities (LOX and POX) in wheat infected by Bgt for both (M) and (NM) plants by the inoculum SZE (Ri+Gm) at P/5 phosphorus concentration. gm 142-144 LOC543232 Triticum aestivum 51-54 25151822-15 2013 Our results showed an increase of both activities (LOX and POX) in wheat infected by Bgt for both (M) and (NM) plants by the inoculum SZE (Ri+Gm) at P/5 phosphorus concentration. gm 142-144 peroxidase-like Triticum aestivum 59-62 22829048-7 2013 Similarly, the GM of total-blood THM concentration increased from 16.5 ng/l pre shower to 299 ng/l at 10 min post shower. gm 15-17 THM Homo sapiens 33-36 21895782-4 2012 The anti-inflammatory potential was evaluated by studying the effect of an ethanolic extract obtained from the GS and GM meal-based feed additive (GSGME) on the pro-inflammatory transcription factor NF-kappaB, which is considered to play a key role in the induction of weaning-associated intestinal inflammation. gm 118-120 nuclear factor kappa B subunit 1 Homo sapiens 199-208 22728082-5 2012 RESULTS: Significantly higher Can f 1 concentrations were found in hair and coat samples of hypoallergenic dogs (n = 196, geometric mean [GM], 2.26 mug/g, geometric standard deviation [GSD], 0.73, and GM, 27.04 mug/g, GSD, 0.57, respectively) than of non-hypoallergenic dogs (n = 160, GM, 0.77 mug/g, GSD, 0.71, and GM, 12.98 mug/g, GSD, 0.76, respectively). gm 138-140 major allergen Can f 1 Canis lupus familiaris 30-37 22728082-5 2012 RESULTS: Significantly higher Can f 1 concentrations were found in hair and coat samples of hypoallergenic dogs (n = 196, geometric mean [GM], 2.26 mug/g, geometric standard deviation [GSD], 0.73, and GM, 27.04 mug/g, GSD, 0.57, respectively) than of non-hypoallergenic dogs (n = 160, GM, 0.77 mug/g, GSD, 0.71, and GM, 12.98 mug/g, GSD, 0.76, respectively). gm 201-203 major allergen Can f 1 Canis lupus familiaris 30-37 22728082-5 2012 RESULTS: Significantly higher Can f 1 concentrations were found in hair and coat samples of hypoallergenic dogs (n = 196, geometric mean [GM], 2.26 mug/g, geometric standard deviation [GSD], 0.73, and GM, 27.04 mug/g, GSD, 0.57, respectively) than of non-hypoallergenic dogs (n = 160, GM, 0.77 mug/g, GSD, 0.71, and GM, 12.98 mug/g, GSD, 0.76, respectively). gm 201-203 major allergen Can f 1 Canis lupus familiaris 30-37 22961864-7 2012 By using synthetic peptides, the immunodominant IgE-binding epitope recognized by most GM-allergic/CM-tolerant patients was located in the caprine domain 49-79. gm 87-89 immunoglobulin heavy constant epsilon Homo sapiens 48-51 22961864-8 2012 CONCLUSION: The restricted specificity of the IgE response toward the caprine beta-CN in GM-allergic/CM-tolerant patients is mainly directed against the domain 49-79, which differs from its bovine counterpart by only three amino acid substitutions. gm 89-91 immunoglobulin heavy constant epsilon Homo sapiens 46-49 22920724-11 2012 When GM-grown ASC pellets were implanted in 1 mm non-critical hyaline cartilage defects in vivo, cartilage regeneration was inhibited as evaluated by radiographic and equilibrium partitioning of an ionic contrast agent via microCT imaging. gm 5-7 PYD and CARD domain containing Rattus norvegicus 14-17 22551852-11 2012 The GM of lead concentrations in tap water was below 1 mug/L; 58% of concentrations were lower than 1 mug/L and 2.9% were higher than or equal to 10 mug/L. gm 4-6 nuclear RNA export factor 1 Homo sapiens 33-36 22205204-4 2012 RESULTS: In maternal blood, the lowest concentrations of IL-10 (p = 0.0019) and TNF-alpha (p = 0.0185) were observed in the GM >=100-mg/dL group. gm 124-126 interleukin 10 Homo sapiens 57-62 22205204-4 2012 RESULTS: In maternal blood, the lowest concentrations of IL-10 (p = 0.0019) and TNF-alpha (p = 0.0185) were observed in the GM >=100-mg/dL group. gm 124-126 tumor necrosis factor Homo sapiens 80-89 22166939-5 2012 The simulations suggest that, overall, two-step methods provide higher power than one-step approaches and that combining gene-level P-values using the GM with a soft truncation threshold between 0.05 and 0.20 is a powerful approach for conducting GSA, relative to the competing approaches assessed. gm 151-153 GNAS complex locus Homo sapiens 247-250 22387106-9 2012 CONCLUSION: Expanding the role of the pharmacist in a GM-ACE unit has improved the medication use process in a high-risk population through improvements in medication overuse, medication underuse, dosing, medication reconciliation, patient education, and health care provider education. gm 54-56 angiotensin I converting enzyme Homo sapiens 57-60 22156919-7 2012 Overall, our findings contribute to better understanding interactions of gal-1 with larger, complex polysaccharides and to the development of GM-based therapeutics for clinical use. gm 142-144 galectin 1 Homo sapiens 73-78 22045592-7 2012 The greatest loss in CSA (mean[SD]) was seen in GM (-23.3(8.7)%), followed by SOL (-19.0(9.8)%), GL (-17.1(6.5)%), and TA (-10.7(5.9)%). gm 48-50 ERCC excision repair 8, CSA ubiquitin ligase complex subunit Homo sapiens 21-24 21957127-3 2012 We first observed that AnxA1(-/-) mice have significantly increased neutrophil numbers in their bone marrow while having normal levels of GM and G colony-forming units, monocytes, and macrophages. gm 138-140 annexin A1 Mus musculus 23-28 22573066-2 2012 Investigation of the alternative GM allotypes of gamma3, gamma1 and gamma2 chains has disclosed new structural and functional IgG subclasses and B-cell variants, with possible effects on childhood asthma. gm 33-35 gamma-aminobutyric acid (GABA) A receptor, subunit gamma 2 Mus musculus 49-74 23056268-8 2012 Significant correlations were found between GM composition and IL-1alpha, IFN-gamma, closed arm entries of Elevated Plus Maze, total time in Elevated Plus Maze, time spent in Light/Dark Box, and time spent in the inner zone of the Open Field as well as total time in the Open Field. gm 44-46 interleukin 1 alpha Mus musculus 63-72 21647756-6 2011 AR protein expressions in GM and GH were lower than those in GC (P < 0.05), and there was a dose-response relationship between Al-exposure doses and AR protein expressions. gm 26-28 androgen receptor Rattus norvegicus 0-2 21925209-5 2011 The results demonstrate that GM-CSF enhanced resistance to infection with 1.9x10(4) ffc of the mouse-adapted influenza A/Puerto Rico/8/34 (PR8) H1N1 strain, as indicated by significant differences in mortality and mean survival of GM-CSF-deficient (GM(-/-)) mice compared to GM(-/-) mice in which GM-CSF is expressed at increased levels. gm 29-31 colony stimulating factor 2 (granulocyte-macrophage) Mus musculus 231-237 21925209-5 2011 The results demonstrate that GM-CSF enhanced resistance to infection with 1.9x10(4) ffc of the mouse-adapted influenza A/Puerto Rico/8/34 (PR8) H1N1 strain, as indicated by significant differences in mortality and mean survival of GM-CSF-deficient (GM(-/-)) mice compared to GM(-/-) mice in which GM-CSF is expressed at increased levels. gm 231-233 colony stimulating factor 2 (granulocyte-macrophage) Mus musculus 29-35 21822343-1 2011 GM/CA CAT at Sector 23 of the Advanced Photon Source (APS) is an NIH funded facility for crystallographic structure determination of biological macromolecules by X-ray diffraction.A second generation Berkeley automounter is being integrated into the beamline control system at the 23-BM experimental station. gm 0-2 catalase Homo sapiens 6-9 20195920-5 2011 Correspondingly, GM values for alpha(1)-MG (2.29 and 1.99 mg/l vs. 2.17 mg/l for All Japan-A) and for beta(2)-MG (87 and 80 mug/l vs. 99 mug/l for All Japan-A) were not elevated, and NAG also stayed unchanged (2.89 and 2.87 units/l for prefecture 1 and prefecture 7, respectively). gm 17-19 O-GlcNAcase Homo sapiens 183-186 21755007-3 2011 This was most likely because SHIP-/- GM-DCs could not up-regulate MHCII and/or co-stimulatory receptors following TLR stimulation. gm 37-39 inositol polyphosphate-5-phosphatase D Mus musculus 29-33 21755007-7 2011 However, WT GM-DC suppression appeared to be mediated, at least in part, by nitric oxide (NO) production while SHIP-/- GM-DCs expressed high levels of arginase 1 and did not produce NO. gm 119-121 inositol polyphosphate-5-phosphatase D Mus musculus 111-115 21755007-7 2011 However, WT GM-DC suppression appeared to be mediated, at least in part, by nitric oxide (NO) production while SHIP-/- GM-DCs expressed high levels of arginase 1 and did not produce NO. gm 119-121 arginase, liver Mus musculus 151-161 21105651-8 2010 Moreover, GM significantly reduced the nuclear translocation of sterol regulatory element-binding protein-1 (nSREBP-1) in ethanol-treated mice. gm 10-12 sterol regulatory element binding transcription factor 1 Mus musculus 64-107 20736037-5 2010 Other GM allotypes, known to be in linkage disequilibrium with GM 13, were also associated with higher anti-HER-2 antibody levels, albeit not as strongly. gm 6-8 erb-b2 receptor tyrosine kinase 2 Homo sapiens 108-113 20736037-6 2010 These results show that GM allotypes are associated with humoral immunity to HER-2, a finding with potentially significant implications for immunotherapy of breast cancer. gm 24-26 erb-b2 receptor tyrosine kinase 2 Homo sapiens 77-82 20889541-5 2010 The adjuvant effect of the Gel/HBs+GM vaccine was dependent upon the local release of GM-CSF. gm 35-37 colony stimulating factor 2 Homo sapiens 86-92 20889541-7 2010 Analysis of the draining lymph nodes of Gel/HBs+GM vaccine-treated mice revealed an elevated number of CD11c(+) dendritic cells showing enhanced expression of MHC class II and a variety of costimulatory molecules. gm 48-50 integrin subunit alpha X Homo sapiens 103-108 20643815-6 2010 We show that although the adiponectin expression is low in lymphocytes, it is sufficient to induce a significant inhibitory effect on GM precursors (CFU-GM) and activate the AMPK pathway in these cells. gm 134-136 adiponectin, C1Q and collagen domain containing Homo sapiens 26-37 20643815-7 2010 The regulation of adiponectin production by lymphocytes and its detailed function in suppressing GM colony formation need to be elucidated now. gm 97-99 adiponectin, C1Q and collagen domain containing Homo sapiens 18-29 20024944-5 2010 In dancers compared with nondancers, decreased GM volumes were observed in the left premotor cortex, SMA, putamen, and superior frontal gyrus, and decreased WM volumes in both corticospinal tracts, both internal capsules, corpus callosum, and left anterior cingulum. gm 47-49 survival of motor neuron 1, telomeric Homo sapiens 101-104 20298794-5 2010 IL-6 was associated with less global gray and white matter (GM and WM), as well as smaller parietal and temporal GM volumes. gm 60-62 interleukin 6 Macaca mulatta 0-4 20393130-3 2010 Alternatively, FLT3 might be expressed at the earliest stages of GM development. gm 65-67 FMS-like tyrosine kinase 3 Mus musculus 15-19 19816937-7 2010 In vivo cytotoxic assay revealed high E7-specific cytolytic T lymphocytes activity in spleen and in genital draining lymph nodes (LN), and E7-specific CD8 T cells could be detected in GM by tetramer staining. gm 184-186 CD8a molecule Homo sapiens 151-154 20100023-7 2010 Serum ECP was only higher in children whose sleep was disturbed by asthma in the past year than those who were not (geometric mean [GM]: 18.9 vs. 11.1 microg/l, relative mean difference [RMD]: 1.71 [95%CI: 1.09-2.69]). gm 132-134 ribonuclease A family member 3 Homo sapiens 6-9 20100023-8 2010 Serum ECP was increased among asthmatic children on inhaled corticosteroid than those not using them (GM: 15.6 vs. 8.1 microg/l, RMD: 1.93 [95%CI: 1.11-3.39]). gm 102-104 ribonuclease A family member 3 Homo sapiens 6-9 20082487-4 2010 However, the pretreatment of GM significantly alleviated APAP-induced oxidative stress by increasing GSH content, decreasing serum ALT, AST and MDA, and retaining the activity of GSH-px and SOD in the liver. gm 29-31 glutamic pyruvic transaminase, soluble Mus musculus 131-134 20082487-4 2010 However, the pretreatment of GM significantly alleviated APAP-induced oxidative stress by increasing GSH content, decreasing serum ALT, AST and MDA, and retaining the activity of GSH-px and SOD in the liver. gm 29-31 solute carrier family 17 (anion/sugar transporter), member 5 Mus musculus 136-139 20082487-5 2010 Furthermore, GM pretreatment can inhibit caspase-3 activation and phosphorylation of c-Jun-NH2-terminal protein kinase 2 (JNK1/2) and extracellular signal-regulated kinase (ERK). gm 13-15 caspase 3 Mus musculus 41-50 20082487-5 2010 Furthermore, GM pretreatment can inhibit caspase-3 activation and phosphorylation of c-Jun-NH2-terminal protein kinase 2 (JNK1/2) and extracellular signal-regulated kinase (ERK). gm 13-15 mitogen-activated protein kinase 8 Mus musculus 122-128 20082487-5 2010 Furthermore, GM pretreatment can inhibit caspase-3 activation and phosphorylation of c-Jun-NH2-terminal protein kinase 2 (JNK1/2) and extracellular signal-regulated kinase (ERK). gm 13-15 mitogen-activated protein kinase 1 Mus musculus 134-171 20082487-5 2010 Furthermore, GM pretreatment can inhibit caspase-3 activation and phosphorylation of c-Jun-NH2-terminal protein kinase 2 (JNK1/2) and extracellular signal-regulated kinase (ERK). gm 13-15 mitogen-activated protein kinase 1 Mus musculus 173-176 20637454-4 2010 Upon LPS stimulation, GM.BMDCs became fully mature, expressed high levels of PD-L1 and produced more IL-10 and less IL-12p70 and IFN-gamma than PBS.BMDCs. gm 22-24 CD274 antigen Mus musculus 77-82 20637454-4 2010 Upon LPS stimulation, GM.BMDCs became fully mature, expressed high levels of PD-L1 and produced more IL-10 and less IL-12p70 and IFN-gamma than PBS.BMDCs. gm 22-24 interleukin 10 Mus musculus 101-106 20637454-4 2010 Upon LPS stimulation, GM.BMDCs became fully mature, expressed high levels of PD-L1 and produced more IL-10 and less IL-12p70 and IFN-gamma than PBS.BMDCs. gm 22-24 interferon gamma Mus musculus 129-138 19890886-8 2009 Transform growth factor-beta1 (TGF-beta1) was incorporated into the GM hydrogel to improve its bioactivity. gm 68-70 transforming growth factor beta 1 Homo sapiens 0-29 19890886-8 2009 Transform growth factor-beta1 (TGF-beta1) was incorporated into the GM hydrogel to improve its bioactivity. gm 68-70 transforming growth factor beta 1 Homo sapiens 31-40 19365631-2 2009 The aim of the present investigation was to determine whether GM and KM allotypes-genetic markers of IgG heavy chains and kappa-type light chains, respectively-contribute to the magnitude of natural antibody responsiveness to MUC1 in patients with breast cancer. gm 62-64 mucin 1, cell surface associated Homo sapiens 226-230 19717631-7 2009 Using lasI::Gm and ptsP::Gm inactivation M18 mutants, we further show that expression of the phzM gene is positively regulated by the quorum-sensing protein LasI and negatively regulated by the phosphoenolpyruvate phosphotransferase protein PtsP. gm 12-14 phenazine-specific methyltransferase Pseudomonas aeruginosa PAO1 93-97 19717631-7 2009 Using lasI::Gm and ptsP::Gm inactivation M18 mutants, we further show that expression of the phzM gene is positively regulated by the quorum-sensing protein LasI and negatively regulated by the phosphoenolpyruvate phosphotransferase protein PtsP. gm 25-27 phenazine-specific methyltransferase Pseudomonas aeruginosa PAO1 93-97 19761540-3 2009 Using yeast two-hybrid screening, we found that the gM carboxy-terminal cytoplasmic tail (gM-CT) interacts with FIP4, a Rab11-GTPase effector protein. gm 52-54 RAB11A, member RAS oncogene family Homo sapiens 120-125 19028845-10 2009 Fat over the GM muscle from the B treatment was more saturated and had decreased PUFA/SFA ratio than that from G1 with fat from G2 being intermediate (P<0.001). gm 13-15 Polyunsaturated fatty acid percentage Sus scrofa 81-85 19834281-7 2009 Current exposure workers with In-S of 3 ng/ml or above demonstrated a significant increase of KL-6 in both GM and prevalence exceeding the reference value. gm 107-109 mucin 1, cell surface associated Homo sapiens 94-98 19015737-7 2009 Only an increase in the MASP2 transcript was statistically significant (PPIB, P = 0.001; GM, P = 0.047; rbcL, P = 0.045). gm 89-91 MBL associated serine protease 2 Homo sapiens 24-29 19073762-3 2008 Functional integration of Gm PNC1 and At PNC2 into the cytoplasmic membranes of intact Escherichia coli cells revealed ATP and ADP import activities. gm 26-28 peroxisomal adenine nucleotide carrier 1 Arabidopsis thaliana 29-33 18842903-7 2008 In the GM, most reporter(+) cells remained NG2(+), even after injury, but stopped proliferating rather soon after recombination. gm 7-9 chondroitin sulfate proteoglycan 4 Mus musculus 43-46 18842903-8 2008 Thus, our results demonstrate the continuous generation of mature, myelinating oligodendrocytes in the WM, whereas cells in the GM generated mostly postmitotic NG2(+) glia. gm 128-130 chondroitin sulfate proteoglycan 4 Mus musculus 160-163 18801325-9 2008 The results among the GM-neutral farmers were in most cases closely related to the positive farmers" choices, implying that they believe that there are advantages with growing an IR GM crop, but also fear potential drawbacks. gm 22-24 immunity related GTPase M Homo sapiens 179-184 18519707-2 2008 The aim of the present investigation was to determine whether the variation in naturally occurring antibody levels to MUC1 in patients with gastric cancer is associated with GM and KM allotypes, genetic markers of IgG heavy chains and kappa-type light chains, respectively. gm 174-176 mucin 1, cell surface associated Homo sapiens 118-122 18519707-8 2008 These results show, for the first time, that GM and KM allotypes contribute to the interindividual differences in humoral immunity to MUC1. gm 45-47 mucin 1, cell surface associated Homo sapiens 134-138 18436863-4 2008 Decreased TAL1 expression in CMP resulted in rare erythroid colonies, in a 2-3 fold reduction of GM colony number in clonogenic assays and in a 3.6-5.6 decreased production of CD14(+)CD15(+) GM cells in liquid culture. gm 97-99 TAL bHLH transcription factor 1, erythroid differentiation factor Homo sapiens 10-14 18436863-5 2008 Moreover, analysis of transcript profile of gene involved in GM differentiation showed that GM cells expressing shRNA-TAL1 construct displayed decreased levels of g-csfr, c/ebpalpha, and mpo and high levels of gata-2 transcripts, indicating a blocking of GM differentiation. gm 61-63 TAL bHLH transcription factor 1, erythroid differentiation factor Homo sapiens 118-122 18436863-6 2008 In contrast, GM differentiation of GMP remained unaffected when TAL1 transcript levels were decreased. gm 13-15 5'-nucleotidase, cytosolic II Homo sapiens 35-38 18370581-13 2008 CONCLUSIONS: Both GM and TM treatments resulted in an improvement of subjective measures associated with CTS, but improvement in grip strength was only detected with the TM protocol. gm 18-20 transthyretin Homo sapiens 105-108 18299402-3 2008 Demonstrating the sufficiency of miR-155 to drive GM expansion, enforced expression in mouse bone marrow cells caused GM proliferation in a manner reminiscent of LPS treatment. gm 50-52 microRNA 155 Mus musculus 33-40 18299402-3 2008 Demonstrating the sufficiency of miR-155 to drive GM expansion, enforced expression in mouse bone marrow cells caused GM proliferation in a manner reminiscent of LPS treatment. gm 118-120 microRNA 155 Mus musculus 33-40 18299402-4 2008 However, the miR-155-induced GM populations displayed pathological features characteristic of myeloid neoplasia. gm 29-31 microRNA 155 Homo sapiens 13-20 18299402-7 2008 These data implicate miR-155 as a contributor to physiological GM expansion during inflammation and to certain pathological features associated with AML, emphasizing the importance of proper miR-155 regulation in developing myeloid cells during times of inflammatory stress. gm 63-65 microRNA 155 Homo sapiens 21-28 18041736-9 2008 Levels of PAP mRNA significantly increased in the GM diet between day 1 and day 3 and decreased to the basal level by day 15. gm 50-52 regenerating family member 3 beta Rattus norvegicus 10-13 18472508-5 2008 The number of CD34+ cell/ml in the preapheresis blood correlated closely with CD34+ cells/kg and, to a lesser degree, with GM-CFU/kg in the apheresis products (r = 0.81 and r = 0.67, respectively, P < 0.0001). gm 123-125 CD34 molecule Homo sapiens 14-18 17994293-8 2008 Since autoclaving of vip-s bearing GM leaf samples showed no deterioration/interference in detection efficacy, the assay seems to be suitable for processed food products as well. gm 35-37 vasoactive intestinal peptide Homo sapiens 21-24 17943303-11 2008 CONCLUSIONS: Variations in GM values for Cd-U, alpha 1-MG-U, beta 2-MG-U, and NAG-U at different time of sampling are small so that single measurement would be acceptable as far as the evaluation on a group basis is the study objective. gm 27-29 beta-2-microglobulin Homo sapiens 61-72 17943303-11 2008 CONCLUSIONS: Variations in GM values for Cd-U, alpha 1-MG-U, beta 2-MG-U, and NAG-U at different time of sampling are small so that single measurement would be acceptable as far as the evaluation on a group basis is the study objective. gm 27-29 O-GlcNAcase Homo sapiens 78-83 18569589-7 2008 The distribution of 8-oxodGuo adducts for the Ser326Cys variants of hOGG1 revealed geometric means (GM) of 5.83 (CC), 5.27 (CG), and 6.53 (GG) 8-oxodGuo adducts/10(6)dGuo. gm 100-102 8-oxoguanine DNA glycosylase Homo sapiens 68-73 17978043-5 2007 Notably, although VEGF suppression significantly enhanced pericyte coverage in the GM, it remained less than in other brain regions. gm 83-85 vascular endothelial growth factor A Homo sapiens 18-22 17978043-7 2007 Transforming growth factor-beta1 (TGF-beta1) protein expression was lower, whereas sphingosine-1-phosphate1 (S1P1) and N-cadherin levels were higher in the GM than in the cortex or white matter. gm 156-158 sphingosine-1-phosphate receptor 1 Homo sapiens 83-107 17978043-7 2007 Transforming growth factor-beta1 (TGF-beta1) protein expression was lower, whereas sphingosine-1-phosphate1 (S1P1) and N-cadherin levels were higher in the GM than in the cortex or white matter. gm 156-158 sphingosine-1-phosphate receptor 1 Homo sapiens 109-113 17978043-7 2007 Transforming growth factor-beta1 (TGF-beta1) protein expression was lower, whereas sphingosine-1-phosphate1 (S1P1) and N-cadherin levels were higher in the GM than in the cortex or white matter. gm 156-158 cadherin 2 Homo sapiens 119-129 17727418-6 2007 Transgenic tobacco plants differing in the amounts of aquaporin NtAQP1 showed different slopes of the gm-Ci response, suggesting a possible role for aquaporins in mediating CO2 responsiveness of gm. gm 102-104 probable aquaporin TIP1-1 Nicotiana tabacum 64-70 17340198-3 2007 IL-1alpha recognized a di-antennary N-glycan with two alpha2-3-linked sialic acid residues, whereas IL-1beta recognized the GM(4), a alpha2-3-linked sialylated glycosphingolipid. gm 124-126 interleukin 1 beta Homo sapiens 100-108 17879330-10 2007 We conclude that TPM allows claims to be made about subsets of p-values, while the claim of the RTP is, like GM, more appropriately about all L tests. gm 109-111 MORN repeat containing 4 Homo sapiens 96-99 17257813-6 2007 PP1-GM activity and the amount of PP1 bound to GM decreased 40% and 45% respectively, in response to adrenaline in control mice. gm 4-6 protein phosphatase 1 catalytic subunit gamma Mus musculus 0-3 16750632-6 2007 In the lower leg, longer total durations of GM activity were found during DS1 on the paretic side in people with stroke (51%) than in controls (38%). gm 44-46 mitochondrial ribosomal protein L58 Homo sapiens 74-77 17323156-6 2007 Also, bioactive GM increased this BMP effect. gm 16-18 bone morphogenetic protein 1 Homo sapiens 34-37 17323156-9 2007 In addition, pBMP-9 with bioactive GM generated less MMP-2 than did rhBMP-2 on days 3 and 5. gm 35-37 matrix metallopeptidase 2 Homo sapiens 53-58 17074305-9 2007 The rank order of MTLR-phosphorylation was: [1-22]>[1-14]>Phe(3)[1-22]=Phe(3)[1-14]>GM-109=MA-2029. gm 93-95 motilin receptor Homo sapiens 18-22 16597465-6 2007 Fourth, partitioning and trafficking of GM to the plasma membrane depended on the transport of ceramide precursors from endoplasmic reticulum to Golgi network, as well as on the synthesis, glycosylation and vesicular assembly in trans-Golgi, and less on the cytoskeleton architecture in both quiescent and activated CD4 thymic and splenic T cells. gm 40-42 CD4 molecule Homo sapiens 316-319 16597465-7 2007 Together, these findings suggest that the differential partitioning and intracellular trafficking of GM and cholesterol in thymic and splenic CD4 T cells may account for the stage of functional maturation. gm 101-103 CD4 molecule Homo sapiens 142-145 17040502-5 2006 The coexistence in a male of these two entities that predominantly affect females reinforces the hypothesis that a pathogenic link exists between GM and PBC. gm 146-148 dihydrolipoamide S-acetyltransferase Homo sapiens 153-156 17040502-6 2006 Consequently, PBC should be looked for in all patients with GM. gm 60-62 dihydrolipoamide S-acetyltransferase Homo sapiens 14-17 16817976-12 2006 IgE-immunoblot analysis demonstrated the presence of 14 IgE-binding components within the wild-type potato and 9 within the GM potato. gm 124-126 immunoglobulin heavy constant epsilon Homo sapiens 0-3 16551675-9 2006 The 8 h TWA levels of BaP ranged from <0.01 to 6.21 microg m(-3) with a GM of 0.036 microg m(-3); 90% were <0.75 microg m(-3) and 95% were <2.0 microg m(-3). gm 75-77 prohibitin 2 Homo sapiens 22-25 16731921-3 2006 Hence, it was investigated whether UL49.5 can combine the interactions with gM and the TAP complex. gm 76-78 envelope glycoprotein N Bovine alphaherpesvirus 1 35-41 16731921-4 2006 In cell lines constitutively expressing both UL49.5 and gM, UL49.5 appears to be required for functional processing of gM. gm 56-58 envelope glycoprotein N Bovine alphaherpesvirus 1 60-66 16731921-6 2006 Remarkably, expression of cloned gM results in the abrogation of the UL49.5-mediated inhibition of TAP and prevents the degradation of the transporter. gm 33-35 envelope glycoprotein N Bovine alphaherpesvirus 1 69-75 16731921-6 2006 Remarkably, expression of cloned gM results in the abrogation of the UL49.5-mediated inhibition of TAP and prevents the degradation of the transporter. gm 33-35 nuclear RNA export factor 1 Homo sapiens 99-102 16731921-8 2006 Moreover, in later periods, non-gM-associated UL49.5 can be detected in addition to the UL49.5/gM complex. gm 32-34 envelope glycoprotein N Bovine alphaherpesvirus 1 46-52 16731921-9 2006 Thus, it has been deduced that different functions of UL49.5, editing of gM processing and inhibition of TAP, can be combined during BHV-1 infection. gm 73-75 envelope glycoprotein N Bovine alphaherpesvirus 1 54-60 16627880-10 2006 More importantly, the lesser degree of GFAP expression in astrocyte end-feet of GM compared with cortex and WM may reflect a cytoskeletal structural difference that contributes to the fragility of GM vasculature and propensity to hemorrhage. gm 80-82 glial fibrillary acidic protein Homo sapiens 39-43 16688825-17 2006 In the second stage, TGF-beta1 increased significantly in GM group compared to GN(2). gm 58-60 transforming growth factor, beta 1 Rattus norvegicus 21-30 16173010-3 2006 In GM-847 cell study, we have found that hTERT is not required for tumorigenic growth in subrenal capsule transplantation, however, it is required in subcutaneous injection assay. gm 3-5 telomerase reverse transcriptase Homo sapiens 41-46 16469640-9 2006 Adenosine triphosphate/capsaicin application increased CGRP release by 75% over baseline (606 +/- 98 pg/gm, p < 0.005). gm 104-106 calcitonin-related polypeptide alpha Rattus norvegicus 55-59 16399110-8 2006 The preoperative administration of GM (GM group) substantially alleviated hepatic I/R injury compared with the untreated control group; postoperative serum transaminase levels were significantly decreased in association with marked suppression of IL-6 levels in blood circulation during surgery. gm 35-37 interleukin 6 Homo sapiens 247-251 16399110-8 2006 The preoperative administration of GM (GM group) substantially alleviated hepatic I/R injury compared with the untreated control group; postoperative serum transaminase levels were significantly decreased in association with marked suppression of IL-6 levels in blood circulation during surgery. gm 39-41 interleukin 6 Homo sapiens 247-251 16392918-5 2005 Despite this, the attraction basin around each GM is relatively large, since after all their atomic coordinates are randomly displaced by values as high as 2.0 bohrs, the perturbed structures, upon reoptimization, relax back to the GM in more than 50% of the cases (except for n=10 and 11). gm 47-49 nuclear receptor subfamily 4 group A member 1 Homo sapiens 277-288 16046524-2 2005 JMML cells are characterized by hypersensitivity to granulocyte-macrophage colony-stimulating factor (GM-CSF) caused by a continuously activated GM-CSF receptor-retrovirus-associated sequence (RAS) signal transduction pathway through various molecular mechanisms, resulting in spontaneous GM colony formation in vitro. gm 102-104 colony stimulating factor 2 Homo sapiens 52-100 16046524-2 2005 JMML cells are characterized by hypersensitivity to granulocyte-macrophage colony-stimulating factor (GM-CSF) caused by a continuously activated GM-CSF receptor-retrovirus-associated sequence (RAS) signal transduction pathway through various molecular mechanisms, resulting in spontaneous GM colony formation in vitro. gm 102-104 colony stimulating factor 2 Homo sapiens 145-151 16176266-9 2005 The Brij-96 domains retained their structural integrity after cholesterol depletion while, in contrast, the Triton X-100-based caveolin-1/GM(1) microdomains did not. gm 138-140 caveolin 1 Homo sapiens 127-137 16189211-6 2005 We found that claudin-5, occludin, and JAM-1 were expressed as early as 16 wk in GM, cortex, and white matter. gm 81-83 claudin 5 Homo sapiens 14-23 16189211-6 2005 We found that claudin-5, occludin, and JAM-1 were expressed as early as 16 wk in GM, cortex, and white matter. gm 81-83 occludin Homo sapiens 25-33 16189211-6 2005 We found that claudin-5, occludin, and JAM-1 were expressed as early as 16 wk in GM, cortex, and white matter. gm 81-83 F11 receptor Homo sapiens 39-44 16025152-3 2005 The CD34+ and CD3+ cell content of grafts were significantly lower following GM alone compared to G alone (P < 0.001 and 0.04, respectively). gm 77-79 CD34 molecule Homo sapiens 4-8 15961574-4 2005 GM and GM + IL-15 DCs expressed low levels of CD80/CD86 and MHC class II. gm 0-2 CD80 antigen Mus musculus 46-50 15961574-4 2005 GM and GM + IL-15 DCs expressed low levels of CD80/CD86 and MHC class II. gm 0-2 CD86 antigen Mus musculus 51-55 15961574-4 2005 GM and GM + IL-15 DCs expressed low levels of CD80/CD86 and MHC class II. gm 7-9 interleukin 15 Mus musculus 12-17 15961574-4 2005 GM and GM + IL-15 DCs expressed low levels of CD80/CD86 and MHC class II. gm 7-9 CD80 antigen Mus musculus 46-50 15961574-4 2005 GM and GM + IL-15 DCs expressed low levels of CD80/CD86 and MHC class II. gm 7-9 CD86 antigen Mus musculus 51-55 15961574-5 2005 The GM + IL-15 DCs produced high levels of IL-12p70 and interferon (IFN)-gamma, whereas GM DCs produced only high levels of IL-12p70. gm 4-6 interferon gamma Mus musculus 56-78 16085045-3 2005 METHODS: Expression of MCP-1 was evaluated in the myometrium, the placenta, the gestational membranes (GM) and the amniotic fluid (AF) by real time RT-PCR, Northern blot analysis and ELISA. gm 103-105 C-C motif chemokine ligand 2 Homo sapiens 23-28 16085045-6 2005 Increased MCP-1 transcripts were demonstrated in GM during term labor. gm 49-51 C-C motif chemokine ligand 2 Homo sapiens 10-15 15669088-7 2005 It was found that the amino-terminal GM-CSF fusion proteins, GM-VL-VH and GM-VH-VL, showed much higher activity than the carboxy-terminal GM-CSF fusion proteins, VL-VH-GM and VH-VL-GM, in stimulating the cell proliferation of a GM-CSF-dependent cell line, NFS-60. gm 61-63 colony stimulating factor 2 (granulocyte-macrophage) Mus musculus 37-43 15669088-7 2005 It was found that the amino-terminal GM-CSF fusion proteins, GM-VL-VH and GM-VH-VL, showed much higher activity than the carboxy-terminal GM-CSF fusion proteins, VL-VH-GM and VH-VL-GM, in stimulating the cell proliferation of a GM-CSF-dependent cell line, NFS-60. gm 61-63 colony stimulating factor 2 (granulocyte-macrophage) Mus musculus 138-144 15669088-7 2005 It was found that the amino-terminal GM-CSF fusion proteins, GM-VL-VH and GM-VH-VL, showed much higher activity than the carboxy-terminal GM-CSF fusion proteins, VL-VH-GM and VH-VL-GM, in stimulating the cell proliferation of a GM-CSF-dependent cell line, NFS-60. gm 61-63 colony stimulating factor 2 (granulocyte-macrophage) Mus musculus 138-144 15661843-6 2005 With the exception of the Q-motif mutant eIF4AP56L, the eIF4A mutants inactivated for Pdcd4 binding were inactivated for binding to eIF4G (GM, GC, or both) and for enhancing translation. gm 139-141 programmed cell death 4 Homo sapiens 86-91 15699110-6 2005 Thus, a population of cytokine-expanded GM precursors function as regulatory APCs, suggesting that G-CSF derivatives may have application in disorders characterized by a loss of self-tolerance. gm 40-42 colony stimulating factor 3 Homo sapiens 99-104 15590153-18 2005 t-PA was also involved in GM lesions, probably through an inflammatory process involving macrophages. gm 26-28 plasminogen activator, tissue Mus musculus 0-4 14502230-4 2003 Treatment of nude mice with anti-asialo GM(1) antibody temporally abrogated the growth retardation of AsPC-1/IL-21, but not AsPC-1/IL-23 tumors; however, the growth of AsPC-1/IL-21 tumors came to be retarded thereafter with the regeneration of natural killer (NK) cells. gm 40-42 interleukin 21 Mus musculus 109-114 14502230-4 2003 Treatment of nude mice with anti-asialo GM(1) antibody temporally abrogated the growth retardation of AsPC-1/IL-21, but not AsPC-1/IL-23 tumors; however, the growth of AsPC-1/IL-21 tumors came to be retarded thereafter with the regeneration of natural killer (NK) cells. gm 40-42 interleukin 21 Mus musculus 175-180 12920239-9 2003 rAAV-IkappaBalpha injection reduced infarct size (IF/area-at-risk = 19 +/- 3%; IF/left ventricle = 10 +/- 2%; p < 0.001), blocked NF-kappaB activation, diminished cardiac ICAM-1 expression (0.4 +/- 0.02 relative amount of cardiac ICAM-1 mRNA; p < 0.001), and blunted leukocyte accumulation (area-at-risk = 0.6 +/- 0.05 micro g/gm tissue; infarct area = 0.4 +/- 0.02 micro g/gm tissue; p < 0.001). gm 333-335 nuclear factor of kappa light polypeptide gene enhancer in B cells inhibitor, alpha Mus musculus 5-17 12920239-9 2003 rAAV-IkappaBalpha injection reduced infarct size (IF/area-at-risk = 19 +/- 3%; IF/left ventricle = 10 +/- 2%; p < 0.001), blocked NF-kappaB activation, diminished cardiac ICAM-1 expression (0.4 +/- 0.02 relative amount of cardiac ICAM-1 mRNA; p < 0.001), and blunted leukocyte accumulation (area-at-risk = 0.6 +/- 0.05 micro g/gm tissue; infarct area = 0.4 +/- 0.02 micro g/gm tissue; p < 0.001). gm 380-382 nuclear factor of kappa light polypeptide gene enhancer in B cells inhibitor, alpha Mus musculus 5-17 12747453-11 2003 Severe alcohol use as measured by GGTP >51 iu/l worsens PI (P<0.07), which adversely impacts GM, GI, PD, and ultimately AL. gm 99-101 inactive glutathione hydrolase 2 Homo sapiens 34-38 12618323-10 2003 The 2f(1)-f(2) DPOAE showed consistent regions of suppression that were approximately an octave above the GM for the 1-kHz, 65/55-dB SPL condition. gm 106-108 sphingosine-1-phosphate lyase 1 Homo sapiens 133-136 12571851-10 2003 The quantities of IGF-1 and IGFBP-3 present ex vivo were 11.3 and 78.7 ng/gm of cartilage in normal cartilage and 21.6 and 225.4 ng/gm in OA cartilage. gm 74-76 insulin like growth factor 1 Homo sapiens 18-23 12571851-10 2003 The quantities of IGF-1 and IGFBP-3 present ex vivo were 11.3 and 78.7 ng/gm of cartilage in normal cartilage and 21.6 and 225.4 ng/gm in OA cartilage. gm 74-76 insulin like growth factor binding protein 3 Homo sapiens 28-35 12571851-10 2003 The quantities of IGF-1 and IGFBP-3 present ex vivo were 11.3 and 78.7 ng/gm of cartilage in normal cartilage and 21.6 and 225.4 ng/gm in OA cartilage. gm 132-134 insulin like growth factor 1 Homo sapiens 18-23 12571851-10 2003 The quantities of IGF-1 and IGFBP-3 present ex vivo were 11.3 and 78.7 ng/gm of cartilage in normal cartilage and 21.6 and 225.4 ng/gm in OA cartilage. gm 132-134 insulin like growth factor binding protein 3 Homo sapiens 28-35 14674248-14 2003 4), whereas GM inhibits native PP1 beta more potently than native PP1 gamma 1 (not shown). gm 12-14 neuropeptide Y receptor Y4 Homo sapiens 31-34 14674248-22 2003 Moreover, only PP1 beta has been identified in complexes with GM in muscle extracts, although these data did not exclude the possibility that other isoforms were also present. gm 62-64 protein phosphatase 1 catalytic subunit beta Homo sapiens 15-23 12388794-9 2002 Moreover, compensatory downregulation of endogenous renin expression was more pronounced in mice containing the GM variant. gm 112-114 renin Homo sapiens 52-57 12393686-3 2002 Opsonophagocytosis by GM(-/-) AMs was severely impaired and was restored by pulmonary GM-CSF expression in vivo or by PU.1 expression in vitro. gm 22-24 colony stimulating factor 2 (granulocyte-macrophage) Mus musculus 86-92 12393686-3 2002 Opsonophagocytosis by GM(-/-) AMs was severely impaired and was restored by pulmonary GM-CSF expression in vivo or by PU.1 expression in vitro. gm 22-24 spleen focus forming virus (SFFV) proviral integration oncogene Mus musculus 118-122 12393686-10 2002 IL-18 expression by GM(-/-) AMs was severely impaired and was restored by pulmonary GM-CSF expression in vivo or by PU.1 expression in vitro. gm 20-22 interleukin 18 Mus musculus 0-5 12393686-10 2002 IL-18 expression by GM(-/-) AMs was severely impaired and was restored by pulmonary GM-CSF expression in vivo or by PU.1 expression in vitro. gm 20-22 colony stimulating factor 2 (granulocyte-macrophage) Mus musculus 84-90 12393686-10 2002 IL-18 expression by GM(-/-) AMs was severely impaired and was restored by pulmonary GM-CSF expression in vivo or by PU.1 expression in vitro. gm 20-22 spleen focus forming virus (SFFV) proviral integration oncogene Mus musculus 116-120 12444140-7 2002 Retroviral vector-mediated reconstitution of PU.1 expression in cultured GM(-/-) AMs restored phagocytosis of 4- micro m beads, endocytosis of adenovirus, and transferrin-coated 100-nm beads (independent of integrin alpha(V) and transferrin receptors, respectively), and restored normal cytoskeletal organization, filamentous actin distribution, and stimulated formation of filopodia. gm 73-75 spleen focus forming virus (SFFV) proviral integration oncogene Mus musculus 45-49 12444140-7 2002 Retroviral vector-mediated reconstitution of PU.1 expression in cultured GM(-/-) AMs restored phagocytosis of 4- micro m beads, endocytosis of adenovirus, and transferrin-coated 100-nm beads (independent of integrin alpha(V) and transferrin receptors, respectively), and restored normal cytoskeletal organization, filamentous actin distribution, and stimulated formation of filopodia. gm 73-75 transferrin Mus musculus 159-170 12198624-2 2002 Colonization by serotype VIII GBS was associated with significantly higher serum concentrations of serotype-specific antibodies (geometric mean [GM], 5.53 micro g/mL), compared with both noncolonization (1.53 micro g/mL) and colonization with other serotypes (2.19 micro g/mL). gm 145-147 cytochrome c oxidase subunit 8A Homo sapiens 25-29 12374203-3 2002 Anti-GM, IgM-antibodies in the normal human immunological repertoire are low affinity antibodies that cross-react with other glycoconjugates carrying Gal beta1-3GalNAc and probably do not have GM1-mediated biological activity. gm 5-7 UDP-GlcNAc:betaGal beta-1,3-N-acetylglucosaminyltransferase 2 Homo sapiens 154-161 11827518-6 2002 From kinetic measurements, we determined the rate constant for dissociation of a single site of FITC-CTB from microsphere-supported bilayers to be (3.21 +/- 0.03) x 10(-3) s(-1), and the rate of association of a site on FITC-CTB in solution to a GM(1) in the bilayer to be (2.8 +/- 0.4) x 10(4) M(-1) s(-1). gm 246-248 phosphate cytidylyltransferase 1B, choline Homo sapiens 101-104 11934394-1 2002 Addition of a small amount of ganglioside GM(1) to phosphatidylserine (PS) liposomes, a gradual increase of protein kinase C (PKC) activity was recorded up to about 2 mol% GM(1) where the maximal enzyme activity was obtained. gm 42-44 proline rich transmembrane protein 2 Homo sapiens 108-124 11934394-1 2002 Addition of a small amount of ganglioside GM(1) to phosphatidylserine (PS) liposomes, a gradual increase of protein kinase C (PKC) activity was recorded up to about 2 mol% GM(1) where the maximal enzyme activity was obtained. gm 42-44 proline rich transmembrane protein 2 Homo sapiens 126-129 11504698-4 2001 Expression of GM-CSF in lungs of SP-D(-/-),GM(-/-) mice corrected GM-CSF-dependent abnormalities in surfactant catabolism but did not correct lung pathology characteristic of SP-D deletion. gm 14-16 surfactant associated protein D Mus musculus 33-37 11504698-4 2001 Expression of GM-CSF in lungs of SP-D(-/-),GM(-/-) mice corrected GM-CSF-dependent abnormalities in surfactant catabolism but did not correct lung pathology characteristic of SP-D deletion. gm 14-16 colony stimulating factor 2 (granulocyte-macrophage) Mus musculus 66-72 19002915-7 2001 The secretion of two kinds of cytokines, Interleukin-6 and Tumor Necrosis Factor-alpha from human B cells was also enhanced in adding the crude extract, but not the standards such as Glycyrrhizin (GL) or 18,beta-glycyrrhetinic acid (GM). gm 233-235 tumor necrosis factor Homo sapiens 59-86 11288073-6 2001 The BET measured surface area of the fibers sintered at 900 degrees C was 0 m(2)/gm compared to a value of 200 m(2)/gm for a typical sol-gel-derived particle of similar composition. gm 81-83 delta/notch like EGF repeat containing Homo sapiens 4-7 11327710-2 2001 The assay establishes that recombinant human IL-2 binds to ganglioside GD(1b) but not to any other gangliosides (GM(1), GM(2), GM(3), GD(1a), GD(2), GD(3), and GT(1b)). gm 113-115 interleukin 2 Homo sapiens 45-49 11275537-8 2001 An expansion of the RRF for the TA and GM was seen when increasing the stimulus intensity from 0.8xPTh to 1.2xPTh and from 1.2xPTh to 1.6xPTh, indicating a gradually increasing reflex threshold towards the border, where TA contraction is inappropriate in a withdrawal reaction. gm 39-41 mitochondrial ribosome recycling factor Homo sapiens 20-23 11283248-1 2001 The regulatory-targeting subunit (RGL), also called GM) of the muscle-specific glycogen-associated protein phosphatase PP1G targets the enzyme to glycogen where it modulates the activity of glycogen-metabolizing enzymes. gm 52-54 protein phosphatase 1, regulatory subunit 3A Mus musculus 4-32 11283248-1 2001 The regulatory-targeting subunit (RGL), also called GM) of the muscle-specific glycogen-associated protein phosphatase PP1G targets the enzyme to glycogen where it modulates the activity of glycogen-metabolizing enzymes. gm 52-54 protein phosphatase 1, regulatory subunit 3A Mus musculus 34-37 11283248-1 2001 The regulatory-targeting subunit (RGL), also called GM) of the muscle-specific glycogen-associated protein phosphatase PP1G targets the enzyme to glycogen where it modulates the activity of glycogen-metabolizing enzymes. gm 52-54 protein phosphatase 1 catalytic subunit gamma Mus musculus 119-123 11159019-1 2001 Metabolism of surfactant protein (SP) A and dipalmitoylphosphatidylcholine (DPPC) was assessed in alveolar macrophages isolated from granulocyte-macrophage colony-stimulated factor (GM-CSF) gene-targeted [GM(-/-)] mice, wild-type mice, and GM(-/-) mice expressing GM-CSF under control of the SP-C promoter element (SP-C-GM). gm 182-184 surfactant associated protein A1 Mus musculus 14-39 11159019-2 2001 Although binding and uptake of (125)I-SP-A were significantly increased in alveolar macrophages from GM(-/-) compared with wild type or SP-C-GM mice, catabolism of (125)I-SP-A was markedly decreased in GM(-/-) mice. gm 101-103 surfactant associated protein A1 Mus musculus 38-42 11159019-3 2001 Association of [(3)H]DPPC with alveolar macrophages from GM(-/-), wild-type, and SP-C-GM mice was similar; however, catabolism of DPPC was markedly reduced in cells from GM(-/-) mice. gm 86-88 sparse coat Mus musculus 81-85 11222615-6 2001 An isogenic snr-1::Gm insertional mutant was unable to grow aerobically with nitrate as a sole nitrogen source or anaerobically with nitrate as an electron acceptor. gm 19-21 cytochrome C Snr1 Pseudomonas aeruginosa PAO1 12-17 11222615-7 2001 Complementation of the snr-1::Gm mutant with the snr-1 gene restored the wild-type phenotypes. gm 30-32 cytochrome C Snr1 Pseudomonas aeruginosa PAO1 23-28 11222615-7 2001 Complementation of the snr-1::Gm mutant with the snr-1 gene restored the wild-type phenotypes. gm 30-32 cytochrome C Snr1 Pseudomonas aeruginosa PAO1 49-54 11316271-6 2001 However, activation of CFTR with cAMP-cocktail increased Im and Gm 15- and 20-fold, respectively while membrane surface area increased by about 7%, indicating the functional insertion of preformed CFTR into the plasma membrane. gm 64-66 cystic fibrosis transmembrane conductance regulator (ATP-binding cassette sub-family C, member 7) Xenopus laevis 23-27 11920266-7 2001 rhSCGF-alpha was synergistic with interleukin-3 and the flt3 ligand to enhance GM colony-growth, but not synergistic with those inducing ex vivo expansion of GM progenitor cells. gm 79-81 fms related receptor tyrosine kinase 3 ligand Homo sapiens 56-67 11372759-0 2001 A functional role of mitogen-activated protein kinases, erk1 and erk2, in the differentiation of a human leukemia cell line, UT-7/GM: a possible key factor for cell fate determination toward erythroid and megakaryocytic lineages. gm 130-132 mitogen-activated protein kinase 3 Homo sapiens 56-60 11372759-0 2001 A functional role of mitogen-activated protein kinases, erk1 and erk2, in the differentiation of a human leukemia cell line, UT-7/GM: a possible key factor for cell fate determination toward erythroid and megakaryocytic lineages. gm 130-132 mitogen-activated protein kinase 1 Homo sapiens 65-69 11090188-8 2000 However, in the presence of gM, the UL73 gene product was posttranslationally modified to the 50- to 60-kDa species. gm 28-30 envelope glycoprotein N Human betaherpesvirus 5 36-40 11097318-3 2000 METHODS: An indirect competitive enzyme-linked immunosorbent assay for quantitation in serum of the alternative serum Gm allotypes from the gamma3-, gamma1-, and gamma2 loci and radial immunodiffusion for quantitation of IgG subclasses were used. gm 118-120 tryptophanyl-tRNA synthetase 1 Homo sapiens 140-168 10821951-8 2000 The addition of Stem Cell Factor (SCF) or IL-6 to the standard combination of flt-3L+/-GM-CSF produces a large increase in the proliferation of GM and DC progenitors (28 times and 11 times respectively) in the first step of the culture. gm 87-89 kit ligand Mus musculus 16-32 10821951-8 2000 The addition of Stem Cell Factor (SCF) or IL-6 to the standard combination of flt-3L+/-GM-CSF produces a large increase in the proliferation of GM and DC progenitors (28 times and 11 times respectively) in the first step of the culture. gm 87-89 kit ligand Mus musculus 34-37 10821951-8 2000 The addition of Stem Cell Factor (SCF) or IL-6 to the standard combination of flt-3L+/-GM-CSF produces a large increase in the proliferation of GM and DC progenitors (28 times and 11 times respectively) in the first step of the culture. gm 87-89 interleukin 6 Mus musculus 42-46 10868450-7 2000 In patients with DU the duodenal mucosal tissues with GM (MB-unstained mucosa) showed significantly higher levels of IL-8 than those without GM (MB-stained mucosa) or the antral mucosa. gm 54-56 C-X-C motif chemokine ligand 8 Homo sapiens 117-121 10868450-10 2000 CONCLUSIONS: Increased IL-8 activity in the duodenal mucosa with GM may be important for ulcerogenesis in H. pylori-positive DU patients. gm 65-67 C-X-C motif chemokine ligand 8 Homo sapiens 23-27 10737774-3 2000 Here we show by positional cloning that gm results from a G-->A substitution mutation in a splice acceptor site within the alpha-subunit of Rab geranylgeranyl transferase (Rabggta), an enzyme that attaches geranylgeranyl groups to Rab proteins. gm 40-42 Rab geranylgeranyl transferase, a subunit Mus musculus 143-173 10737774-3 2000 Here we show by positional cloning that gm results from a G-->A substitution mutation in a splice acceptor site within the alpha-subunit of Rab geranylgeranyl transferase (Rabggta), an enzyme that attaches geranylgeranyl groups to Rab proteins. gm 40-42 Rab geranylgeranyl transferase, a subunit Mus musculus 175-182 10764957-6 2000 Motilin binding to these membranes was determined by the displacement of (125)I MOT by the native peptide MOT 1-22, or by peptide analogues MOT 1-12 [CH(2)NH](10-11) or GM-109 and by erythromycin derivative GM-611. gm 169-171 promotilin Oryctolagus cuniculus 0-7 10764957-6 2000 Motilin binding to these membranes was determined by the displacement of (125)I MOT by the native peptide MOT 1-22, or by peptide analogues MOT 1-12 [CH(2)NH](10-11) or GM-109 and by erythromycin derivative GM-611. gm 207-209 promotilin Oryctolagus cuniculus 0-7 10567387-4 1999 In the present study, we define the molecular determinants required for ligand recognition and for stabilization of the complex through a convergence of several approaches, including the construction of chimeric receptors, the molecular dynamics of our three-dimensional model of the GM.GMR complex, and site-directed mutagenesis. gm 284-286 colony stimulating factor 2 receptor subunit alpha Homo sapiens 287-290 10567387-5 1999 The functional importance of individual residues was then investigated through ligand binding studies at equilibrium and through determination of the kinetic constants of the GM.GMR complex. gm 175-177 colony stimulating factor 2 receptor subunit alpha Homo sapiens 178-181 10502675-5 1999 In host cells (MA104 cells) pretreated with Arthrobacter ureafaciens neuraminidase, which were still infected by human rotaviruses (KUN and MO strains), GM(3) was hydrolyzed markedly by the neuraminidase, while GM(1a) was not hydrolyzed at all. gm 153-155 neuraminidase 1 Homo sapiens 69-82 10502675-5 1999 In host cells (MA104 cells) pretreated with Arthrobacter ureafaciens neuraminidase, which were still infected by human rotaviruses (KUN and MO strains), GM(3) was hydrolyzed markedly by the neuraminidase, while GM(1a) was not hydrolyzed at all. gm 153-155 neuraminidase 1 Homo sapiens 190-203 10520186-5 1999 Different maturation rates of the alternative Gm allotypes within IgG1, IgG2 and IgG3 were shown. gm 46-48 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 81-85 10554151-4 1999 Geometric mean (GM) concentrations of INAP in urine did not vary significantly among sampling cycles. gm 16-18 NFKB inhibitor zeta Homo sapiens 38-42 10519152-6 1999 However, in oocytes expressing CFTR, large simultaneous increases of Gm, Im and Cm occurred after stimulation with cAMP. gm 69-71 cystic fibrosis transmembrane conductance regulator L homeolog Xenopus laevis 31-35 10457894-3 1999 Among other candidate genes are the GM allotypes, which are the markers of the constant parts of heavy chains of the subclasses IgG1, IgG2 and IgG3. gm 36-38 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 143-147 10198353-3 1999 In the present study, GM(-/-) mice were treated daily or weekly with recombinant mouse GM-CSF by aerosol inhalation or intraperitoneal injection for 4-5 wk. gm 22-24 colony stimulating factor 2 (granulocyte-macrophage) Mus musculus 87-93 10052713-2 1999 The present study evaluated the effects of FL on the proliferation of granulocytemacrophage (GM) progenitor cells collected from PB of 24 patients with chronic myeloproliferative disorders (MPDs) by using a methylcellulose assay in serum-free culture conditions. gm 93-95 fms related receptor tyrosine kinase 3 ligand Homo sapiens 43-45 10030268-2 1999 This study tests the hypothesis that the response of cell lines derived from GM to fractionated radiotherapy depends on the function of wild-type p53 (wt p53), a tumor suppressor gene frequently mutated in GM tumors. gm 77-79 tumor protein p53 Homo sapiens 146-149 10030268-2 1999 This study tests the hypothesis that the response of cell lines derived from GM to fractionated radiotherapy depends on the function of wild-type p53 (wt p53), a tumor suppressor gene frequently mutated in GM tumors. gm 77-79 tumor protein p53 Homo sapiens 154-157 10030268-11 1999 CONCLUSIONS: The effect of fractionated radiotherapy in GM may depend on the function of the tumor suppressor gene p53. gm 56-58 tumor protein p53 Homo sapiens 115-118 9694504-10 1998 The numbers of CFU-Mk in TPO-containing and CFU-GM in G-CSF-containing cultures were larger under 5% O2. gm 48-50 colony stimulating factor 3 Homo sapiens 54-59 9657745-4 1998 UT-7/GM was used as a model system, because this cell line can differentiate into erythroid-lineage cells with EPO treatment (Komatsu et al, Blood 89:4021, 1997). gm 5-7 erythropoietin Homo sapiens 111-114 9596651-8 1998 The addition of TPO to the culture containing SCF or SCF + GM-CSF caused a significant increase in the production of GM colony-forming cells by JCML CD34+CD38neg/low population, indicating the stimulatory effects of TPO on JCML primitive hematopoietic progenitors. gm 59-61 thrombopoietin Homo sapiens 16-19 9596651-8 1998 The addition of TPO to the culture containing SCF or SCF + GM-CSF caused a significant increase in the production of GM colony-forming cells by JCML CD34+CD38neg/low population, indicating the stimulatory effects of TPO on JCML primitive hematopoietic progenitors. gm 59-61 CD34 molecule Homo sapiens 149-153 9552065-12 1998 CONCLUSION: This study shows that delayed hematologic recovery is frequent if less than 1 x 10(6)/kg CD34+ cells are infused after high-dose therapy, particularly with GM-CFC less than 1 x 10(5)/kg. gm 168-170 CD34 molecule Homo sapiens 101-105 9525625-3 1998 We sought to express BHV-1 open reading frame U(L)10, which encodes gM, and specifically identify the glycoprotein. gm 68-70 envelope glycoprotein M Bovine alphaherpesvirus 1 46-52 9525625-6 1998 The fusion protein containing the 63 C-terminal amino acids from the corrected gM sequence engendered antibodies that immunoprecipitated a 30-kDa protein from in vitro translation reactions programmed with the U(L)10 gene. gm 79-81 envelope glycoprotein M Bovine alphaherpesvirus 1 210-216 9619915-6 1998 In contrast to ATRA, GCSF increased Gm colony size but not numbers in semi-solid cultures of normal marrow MNC, which suggests the cytokine augments post-progenitor amplification. gm 36-38 colony stimulating factor 3 Homo sapiens 21-25 9537780-10 1998 High levels of Fel d 1 (GM 22.9 microg/g, range 4.5-58) and Can f 1 (GM 21.6 microg/g, range 4-63) were found in upholstered chairs. gm 69-71 major allergen Can f 1 Canis lupus familiaris 60-67 9345027-4 1997 Leptin also induced GM colony formation from BMC of db/db mutant mice whose leptin receptors were incomplete, but the responsiveness was significantly reduced. gm 20-22 leptin Mus musculus 0-6 9345027-4 1997 Leptin also induced GM colony formation from BMC of db/db mutant mice whose leptin receptors were incomplete, but the responsiveness was significantly reduced. gm 20-22 leptin Mus musculus 76-82 9357844-2 1997 Overexpression of GM-CSF only in respiratory epithelial cells of mice deficient in GM-CSF using the SP-C promotor (GM-/-,SP-C-GM+/+) resulted in increased type II cell numbers and normalization of alveolar Sat PC pool sizes. gm 18-20 colony stimulating factor 2 (granulocyte-macrophage) Mus musculus 83-89 9357844-2 1997 Overexpression of GM-CSF only in respiratory epithelial cells of mice deficient in GM-CSF using the SP-C promotor (GM-/-,SP-C-GM+/+) resulted in increased type II cell numbers and normalization of alveolar Sat PC pool sizes. gm 18-20 sparse coat Mus musculus 100-104 9357844-2 1997 Overexpression of GM-CSF only in respiratory epithelial cells of mice deficient in GM-CSF using the SP-C promotor (GM-/-,SP-C-GM+/+) resulted in increased type II cell numbers and normalization of alveolar Sat PC pool sizes. gm 18-20 sparse coat Mus musculus 121-125 9357844-4 1997 The clearance of dipalmitoylphosphatidylcholine and SP-B from the airspaces was more rapid for GM-/-,SP-C-GM+/+ mice than for GM+/+ mice. gm 95-97 surfactant associated protein B Mus musculus 52-56 9357844-4 1997 The clearance of dipalmitoylphosphatidylcholine and SP-B from the airspaces was more rapid for GM-/-,SP-C-GM+/+ mice than for GM+/+ mice. gm 95-97 sparse coat Mus musculus 101-105 9357844-4 1997 The clearance of dipalmitoylphosphatidylcholine and SP-B from the airspaces was more rapid for GM-/-,SP-C-GM+/+ mice than for GM+/+ mice. gm 106-108 surfactant associated protein B Mus musculus 52-56 9357844-4 1997 The clearance of dipalmitoylphosphatidylcholine and SP-B from the airspaces was more rapid for GM-/-,SP-C-GM+/+ mice than for GM+/+ mice. gm 106-108 sparse coat Mus musculus 101-105 9357845-2 1997 Lung weight and volume were significantly increased in SP-C-GM mice compared with GM+/+ or GM-/- control mice. gm 60-62 sparse coat Mus musculus 55-59 9357845-4 1997 Abundance of type II cells per mouse lung was increased three- to fourfold in SP-C-GM mice compared with GM+/+ and GM-/- mice. gm 83-85 sparse coat Mus musculus 78-82 9350619-2 1997 In mature mammalian muscle, the muscular chloride channel ClC-1 contributes about 75% of the sarcolemmal resting conductance (Gm). gm 126-128 chloride voltage-gated channel 1 Homo sapiens 58-63 9276733-7 1997 Mutant GH was less potent than wild-type GH not only in phosphorylation of tyrosine residues in GHR, janus kinase 2 (JAK2), and signal transducers and activators of transcription 5 (STAT5) in IM-9 cells, but also in metabolic responses of BaF/GM cells, a stable clone transfected with cDNA of the chimera of the extracellular domain of human GHR, the transmembrane and the cytoplasmic domain of the human thrombopoietin receptor. gm 243-245 growth hormone 1 Homo sapiens 7-9 9276733-7 1997 Mutant GH was less potent than wild-type GH not only in phosphorylation of tyrosine residues in GHR, janus kinase 2 (JAK2), and signal transducers and activators of transcription 5 (STAT5) in IM-9 cells, but also in metabolic responses of BaF/GM cells, a stable clone transfected with cDNA of the chimera of the extracellular domain of human GHR, the transmembrane and the cytoplasmic domain of the human thrombopoietin receptor. gm 243-245 growth hormone 1 Homo sapiens 41-43 9166841-2 1997 We isolated a novel subline, UT-7/GM after long-term culture of UT-7 with GM-CSF. gm 34-36 colony stimulating factor 2 Homo sapiens 74-80 9166841-6 1997 These findings indicate that UT-7/GM is a bipotential cell line that can be induced to differentiate into erythroid and megakaryocytic lineages by EPO and TPO, respectively. gm 34-36 erythropoietin Homo sapiens 147-150 9166841-6 1997 These findings indicate that UT-7/GM is a bipotential cell line that can be induced to differentiate into erythroid and megakaryocytic lineages by EPO and TPO, respectively. gm 34-36 thrombopoietin Homo sapiens 155-158 9129051-4 1997 Although FAK was upregulated in the cells stimulated by GM-CSF (GM-treated cells), the kinase was barely detectable in the cells cultured with IL-3 (IL-3-treated cells). gm 56-58 PTK2 protein tyrosine kinase 2 Mus musculus 9-12 9211403-4 1997 uPA overexpression enhanced the invasive capacity of GM 7373 cells through a mechanism which differs from that mediated by metalloproteases. gm 53-55 plasminogen activator, urokinase Bos taurus 0-3 9062859-14 1997 The density of PMN elastase positive cells was about 3 times higher in GM than in PIM (3.7 versus 1.2). gm 71-73 elastase, neutrophil expressed Homo sapiens 15-27 9071993-6 1997 Furthermore, the presence of a protein kinase C inhibitor, bisindolylmaleimide (GF 109203X), blocked hCSF-GM-mediated induction of focal adhesion kinase, implicating an important role for protein kinase C in the induction of pp125FAK. gm 106-108 protein tyrosine kinase 2 Homo sapiens 225-233 8955573-12 1996 Can f 1 was significantly higher in upholstered seats in public places than in sofas in homes without a dog (GM 1.8 micrograms/g; P < 0.001). gm 109-111 major allergen Can f 1 Canis lupus familiaris 0-7 9031077-5 1996 Normal CFU-GM responded to rhG+GM-CSF or IL-3 with increased kinetic activity and sensitivity to Ara-C. gm 11-13 colony stimulating factor 2 Homo sapiens 31-37 9031077-5 1996 Normal CFU-GM responded to rhG+GM-CSF or IL-3 with increased kinetic activity and sensitivity to Ara-C. gm 11-13 interleukin 3 Homo sapiens 41-45 8828497-9 1996 When [125I]IGFBP-3 was incubated with GM-10 fibroblasts or C6 glioma cells at 37 C for 4 h, only 10% of the bound ligand remained associated with the cell surface; approximately 90% of the cell-associated radio-activity was internalized and could be recovered in lysates of acid-washed cells. gm 38-40 insulin like growth factor binding protein 3 Homo sapiens 11-18 8876553-10 1996 VCAM-1 detected by ELISA in polyp tissues (6.8 +/- 1.3 micrograms/gm) was higher than that found in six S/T samples (1.2 +/- 0.3 micrograms/gm, p < 0.005) and in two NA samples (1.8 +/- 0.02 micrograms/gm, p = 0.08). gm 66-68 vascular cell adhesion molecule 1 Homo sapiens 0-6 8876553-10 1996 VCAM-1 detected by ELISA in polyp tissues (6.8 +/- 1.3 micrograms/gm) was higher than that found in six S/T samples (1.2 +/- 0.3 micrograms/gm, p < 0.005) and in two NA samples (1.8 +/- 0.02 micrograms/gm, p = 0.08). gm 140-142 vascular cell adhesion molecule 1 Homo sapiens 0-6 8876553-11 1996 E-selectin values in polyps (1.4 +/- 0.3 micrograms/gm) were not statistically different from those detected in six S/T samples (0.5 +/- 0.2 microgram/gm) or two NA samples (1.6 +/- 0.4 microgram/gm). gm 52-54 selectin E Homo sapiens 0-10 8938517-6 1996 Direct addition of KL or FL to colony assays resulted in only a 1.2-fold increase in CFU-GM outgrowth, suggesting that the effects on increased CFU-GM output were at the preprogenitor stage. gm 89-91 KIT ligand Homo sapiens 19-21 9086498-6 1996 Blood samples had been collected between 8:00 and 9:00 a.m. A lower DBH activity was found in exposed as compared to control workers (GM: 7.25 U/ml serum vs. 10.11 U/ml serum; p < 0.01), whereas MAO-B activity was significantly lower in a heavily exposed subgroup (10.1 vs. 13.8 U/10(7) platelets; p = 0.05), but not in the whole sample (p = 0.07). gm 134-136 dopamine beta-hydroxylase Homo sapiens 68-71 9086498-7 1996 Serum PRL was higher both in male (GM: 8.90 ng/ml vs. 6.05 ng/ml; p < 0.01) and female (GM: 12.6 ng/ml vs. 9.33 ng/ml; p < 0.05) styrene-exposed workers as compared to their respective controls. gm 35-37 prolactin Homo sapiens 6-9 9086498-7 1996 Serum PRL was higher both in male (GM: 8.90 ng/ml vs. 6.05 ng/ml; p < 0.01) and female (GM: 12.6 ng/ml vs. 9.33 ng/ml; p < 0.05) styrene-exposed workers as compared to their respective controls. gm 91-93 prolactin Homo sapiens 6-9 8706735-2 1996 Here, we identify PP1c-binding domains on GL and GM, the subunits that target PP1c to hepatic and muscle glycogen, respectively, and on M110, the subunit that targets PP1c to smooth muscle myosin. gm 49-51 protein phosphatase 1 catalytic subunit gamma Homo sapiens 18-22 8706735-2 1996 Here, we identify PP1c-binding domains on GL and GM, the subunits that target PP1c to hepatic and muscle glycogen, respectively, and on M110, the subunit that targets PP1c to smooth muscle myosin. gm 49-51 protein phosphatase 1 catalytic subunit gamma Homo sapiens 78-82 8706735-2 1996 Here, we identify PP1c-binding domains on GL and GM, the subunits that target PP1c to hepatic and muscle glycogen, respectively, and on M110, the subunit that targets PP1c to smooth muscle myosin. gm 49-51 protein phosphatase 1 catalytic subunit gamma Homo sapiens 78-82 8706735-6 1996 Thus, phosphorylation of Ser67 dissociates GM from PP1c because phosphate is inserted into the PP1c-binding domain of GM. gm 43-45 protein phosphatase 1 catalytic subunit gamma Homo sapiens 95-99 8706735-6 1996 Thus, phosphorylation of Ser67 dissociates GM from PP1c because phosphate is inserted into the PP1c-binding domain of GM. gm 118-120 protein phosphatase 1 catalytic subunit gamma Homo sapiens 51-55 8706735-6 1996 Thus, phosphorylation of Ser67 dissociates GM from PP1c because phosphate is inserted into the PP1c-binding domain of GM. gm 118-120 protein phosphatase 1 catalytic subunit gamma Homo sapiens 95-99 8706735-10 1996 M110-(M1-F38) displaced GL from PP1c, while GM-(G63-T93) displaced M110 from PP1c in vitro. gm 44-46 protein phosphatase 1 catalytic subunit gamma Homo sapiens 77-81 8639823-3 1996 We found that UT-7/GM, which is a subline of UT-7, but neither UT-7 nor UT-7/EPO, can proliferate in response to TPO. gm 19-21 thrombopoietin Homo sapiens 113-116 8609219-3 1996 Transgenic mice bearing the SP-C-GM-CSF construct (SP-C-GM+) were bred to GM-/- mice resulting in complete correction of alveolar proteinosis in bitransgenic GM-/-, SP-C-GM+ mice. gm 33-35 surfactant protein C Homo sapiens 28-32 8609219-6 1996 Surfactant proteins-A and -B and phospholipid in bronchoalveolar lavage fluid were normalized in the GM-/-, SP-C-GM+ mice. gm 101-103 surfactant protein C Homo sapiens 108-112 8609219-6 1996 Surfactant proteins-A and -B and phospholipid in bronchoalveolar lavage fluid were normalized in the GM-/-, SP-C-GM+ mice. gm 113-115 surfactant protein C Homo sapiens 108-112 8628019-3 1996 The incidence of spontaneous GM colonies correlated both with the number of blast cells and the amount of c-kit positive cells present in the initial sample. gm 29-31 KIT proto-oncogene, receptor tyrosine kinase Homo sapiens 106-111 8596698-3 1996 In the present study we examined the effects on whole-cell (wc) conductance (Gm) and voltage (Vm) of NAC and the congeners S-carboxymethyl-L-cysteine (CMC) and S-carbamyl-L-cysteine (CAC) and L-cysteine in normal and CF airway epithelial cells. gm 77-79 X-linked Kx blood group Homo sapiens 101-104 8596698-5 1996 The increase in Gm (delta Gm) by the other compounds was concentration dependent and was (all substances at 1 mmol/l) 3.8 +/- 1.4 nS (NAC; n = 11), 4.2 +/- 1.0 nS (CMC; n = 16) and 3.8 +/- 1.6 nS (CAC; n = 18), respectively. gm 16-18 X-linked Kx blood group Homo sapiens 134-137 8596698-5 1996 The increase in Gm (delta Gm) by the other compounds was concentration dependent and was (all substances at 1 mmol/l) 3.8 +/- 1.4 nS (NAC; n = 11), 4.2 +/- 1.0 nS (CMC; n = 16) and 3.8 +/- 1.6 nS (CAC; n = 18), respectively. gm 16-18 carbonic anhydrase 2 Homo sapiens 197-200 8596698-6 1996 The changes in Gm were paralleled by an increased depolarization (delta Vm) when extracellular Cl- concentration was reduced to 34 mmol/l: under control conditions = -4.1 +/- 2.1 versus 10.2 +/- 2.1 mV in the presence of NAC, CMC, CAC (n = 36). gm 15-17 X-linked Kx blood group Homo sapiens 221-224 8596698-6 1996 The changes in Gm were paralleled by an increased depolarization (delta Vm) when extracellular Cl- concentration was reduced to 34 mmol/l: under control conditions = -4.1 +/- 2.1 versus 10.2 +/- 2.1 mV in the presence of NAC, CMC, CAC (n = 36). gm 15-17 carbonic anhydrase 2 Homo sapiens 231-234 8596698-7 1996 In the presence of NAC, CMC and CAC, the reduction in Cl- concentration was paralleled by a reduction of Gm by 2.1 +/- 0.4 nS (n = 35), indicating that all substances acted by increasing the Cl- conductance. gm 105-107 X-linked Kx blood group Homo sapiens 19-22 8596698-7 1996 In the presence of NAC, CMC and CAC, the reduction in Cl- concentration was paralleled by a reduction of Gm by 2.1 +/- 0.4 nS (n = 35), indicating that all substances acted by increasing the Cl- conductance. gm 105-107 carbonic anhydrase 2 Homo sapiens 32-35 8596698-12 1996 In contrast, in CFTR-cRNA-injected oocytes Gm was enhanced when intracellular adenosine 3",5"-cyclic monophosphate (cAMP) was increased by forskolin and IBMX (Gm = 4.5 +/- 1.3 microS; n = 8). gm 43-45 CF transmembrane conductance regulator Homo sapiens 16-20 8596698-13 1996 Gm was significantly increased by 0.74 +/- 0.2 microS (n = 11) and 0.46 +/- 0.1 microS (n = 10) when oocytes were exposed to NAC and CMC, respectively (both 1 mmol/l). gm 0-2 X-linked Kx blood group Homo sapiens 125-128 9165266-5 1996 Elevated levels of IgG3 and IgG4 were associated with GM 1,17 5,6,13 and GM 1,3,17 23 5,13, respectively. gm 54-56 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 19-23 7498521-3 1995 The similarities between GM and GL were most striking between residues 63-86, 144-166 and 186-227 of human GM (approximately 40% identity), nearly all the identities with the putative yeast homologue GAC1 being located between 144-166 and 186-227. gm 25-27 protein phosphatase regulator GAC1 Saccharomyces cerevisiae S288C 200-204 7491938-5 1995 Cd2+, La3+, and Gd3+ had biphasic effects on membrane conductance (Gm). gm 67-69 CD2 molecule Homo sapiens 0-3 7579445-8 1995 Ex vivo expansion studies with isolated CD34+ bone marrow cells from normal donors showed that flt3L alone supported maintenance of both GM and HPP progenitors for 3 to 4 weeks in vitro. gm 137-139 fms related receptor tyrosine kinase 3 ligand Homo sapiens 95-100 7669673-7 1995 These results indicate that IL-13, alone and synergistically with the effect of IL-3, promotes MK colony formation, but it inhibits the growth of GM and erythroid progenitor cells in vitro. gm 146-148 interleukin 13 Homo sapiens 28-33 7638763-9 1995 The newborn myocardium also had increased catalase activity (1027.9 +/- 20.6 units/gm versus 707.3 +/- 38.7 units/gm; p < 0.05), whereas the activity of XO was decreased relative to the adult (0.23 +/- 0.01 mU/gm versus 7.6 +/- 1.4 mU/gm; p < 0.05). gm 83-85 catalase Rattus norvegicus 42-50 7722288-5 1995 These analyses also revealed that the CD4+CD27- population included significantly higher frequencies of cells that were IL-5+IFN-gamma- (GM, 4.9% vs 1.5%; p = 0.025), IL-4+IFN-gamma- (GM, 13.8% vs 3.5%; p = 0.025), and IFN-gamma+IL-4-IL-5- (GM, 27.3% vs 12.0%; p = 0.011) than the CD4+CD27+ population. gm 137-139 CD4 molecule Homo sapiens 38-41 7722288-5 1995 These analyses also revealed that the CD4+CD27- population included significantly higher frequencies of cells that were IL-5+IFN-gamma- (GM, 4.9% vs 1.5%; p = 0.025), IL-4+IFN-gamma- (GM, 13.8% vs 3.5%; p = 0.025), and IFN-gamma+IL-4-IL-5- (GM, 27.3% vs 12.0%; p = 0.011) than the CD4+CD27+ population. gm 137-139 CD27 molecule Homo sapiens 42-46 7722288-5 1995 These analyses also revealed that the CD4+CD27- population included significantly higher frequencies of cells that were IL-5+IFN-gamma- (GM, 4.9% vs 1.5%; p = 0.025), IL-4+IFN-gamma- (GM, 13.8% vs 3.5%; p = 0.025), and IFN-gamma+IL-4-IL-5- (GM, 27.3% vs 12.0%; p = 0.011) than the CD4+CD27+ population. gm 184-186 CD4 molecule Homo sapiens 38-41 7722288-5 1995 These analyses also revealed that the CD4+CD27- population included significantly higher frequencies of cells that were IL-5+IFN-gamma- (GM, 4.9% vs 1.5%; p = 0.025), IL-4+IFN-gamma- (GM, 13.8% vs 3.5%; p = 0.025), and IFN-gamma+IL-4-IL-5- (GM, 27.3% vs 12.0%; p = 0.011) than the CD4+CD27+ population. gm 184-186 CD27 molecule Homo sapiens 42-46 7722288-5 1995 These analyses also revealed that the CD4+CD27- population included significantly higher frequencies of cells that were IL-5+IFN-gamma- (GM, 4.9% vs 1.5%; p = 0.025), IL-4+IFN-gamma- (GM, 13.8% vs 3.5%; p = 0.025), and IFN-gamma+IL-4-IL-5- (GM, 27.3% vs 12.0%; p = 0.011) than the CD4+CD27+ population. gm 184-186 CD4 molecule Homo sapiens 38-41 7722288-5 1995 These analyses also revealed that the CD4+CD27- population included significantly higher frequencies of cells that were IL-5+IFN-gamma- (GM, 4.9% vs 1.5%; p = 0.025), IL-4+IFN-gamma- (GM, 13.8% vs 3.5%; p = 0.025), and IFN-gamma+IL-4-IL-5- (GM, 27.3% vs 12.0%; p = 0.011) than the CD4+CD27+ population. gm 184-186 CD27 molecule Homo sapiens 42-46 7850804-2 1995 The presence of these GM suppressor cells can be diminished by treatment of LLC-LN7-bearing mice with low doses of 100 units IFN-gamma plus 10 units tumor necrosis factor alpha (TNF-alpha). gm 22-24 interferon gamma Mus musculus 125-134 7850804-2 1995 The presence of these GM suppressor cells can be diminished by treatment of LLC-LN7-bearing mice with low doses of 100 units IFN-gamma plus 10 units tumor necrosis factor alpha (TNF-alpha). gm 22-24 tumor necrosis factor Mus musculus 149-176 7850804-2 1995 The presence of these GM suppressor cells can be diminished by treatment of LLC-LN7-bearing mice with low doses of 100 units IFN-gamma plus 10 units tumor necrosis factor alpha (TNF-alpha). gm 22-24 tumor necrosis factor Mus musculus 178-187 7850804-3 1995 The aim of this study was to determine whether treatment of LLC-LN7-bearing mice with IFN-gamma/TNF-alpha to diminish GM suppressor cell presence would increase the responsiveness to IL-2 immune stimulatory therapy (100-1000 IU, twice daily for 5 days). gm 118-120 interferon gamma Mus musculus 86-95 7850804-3 1995 The aim of this study was to determine whether treatment of LLC-LN7-bearing mice with IFN-gamma/TNF-alpha to diminish GM suppressor cell presence would increase the responsiveness to IL-2 immune stimulatory therapy (100-1000 IU, twice daily for 5 days). gm 118-120 tumor necrosis factor Mus musculus 96-105 7850804-3 1995 The aim of this study was to determine whether treatment of LLC-LN7-bearing mice with IFN-gamma/TNF-alpha to diminish GM suppressor cell presence would increase the responsiveness to IL-2 immune stimulatory therapy (100-1000 IU, twice daily for 5 days). gm 118-120 interleukin 2 Mus musculus 183-187 7850804-8 1995 These results show that treatment to minimize the presence of GM suppressor cells enhances the effectiveness of IL-2 to stimulate anti-tumor immune responses and to diminish tumor growth and metastasis. gm 62-64 interleukin 2 Mus musculus 112-116 7851013-4 1995 In this study the Gm allotypes, as genetic characteristics of the IgG1, IgG2 and IgG3, were analysed in 83 Caucasian IgAD individuals. gm 18-20 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 81-85 7851013-17 1995 The "compensatory" increase of IgG was significant for both IgG1 and IgG3 in all Gm phenotypes, but in the Gm(a,",g/f,",b). gm 81-83 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 69-73 7731032-5 1995 From these experiments, we observed that under some circumstances Vm varied with pHo but without Gm or pHi changes, whereas under other circumstances changes of Gm occurred during alterations of pHi while pHo and Vm remained unaltered. gm 161-163 glucose-6-phosphate isomerase Homo sapiens 195-198 7731032-6 1995 At pHi approximately 6.5 associated with Vm approximately -10 mV, Gm dramatically increased to quasi-infinite values. gm 66-68 glucose-6-phosphate isomerase Homo sapiens 3-6 7731032-8 1995 In conclusion, we demonstrate a differential regulation whereby Vm and Gm are controlled by pHo and pHi: pHo modulates mainly Vm and pHi modulates chiefly Gm. gm 71-73 glucose-6-phosphate isomerase Homo sapiens 100-103 7731032-8 1995 In conclusion, we demonstrate a differential regulation whereby Vm and Gm are controlled by pHo and pHi: pHo modulates mainly Vm and pHi modulates chiefly Gm. gm 71-73 glucose-6-phosphate isomerase Homo sapiens 133-136 7731032-8 1995 In conclusion, we demonstrate a differential regulation whereby Vm and Gm are controlled by pHo and pHi: pHo modulates mainly Vm and pHi modulates chiefly Gm. gm 155-157 glucose-6-phosphate isomerase Homo sapiens 100-103 7731032-8 1995 In conclusion, we demonstrate a differential regulation whereby Vm and Gm are controlled by pHo and pHi: pHo modulates mainly Vm and pHi modulates chiefly Gm. gm 155-157 glucose-6-phosphate isomerase Homo sapiens 133-136 7731032-9 1995 Furthermore, at pHi approximately 6.5 and Vm approximately -10 mV, our data reveal a large Gm that tends towards infinite values in a reversible fashion. gm 91-93 glucose-6-phosphate isomerase Homo sapiens 16-19 7802069-10 1994 Annexin-V concentrations in conditioned media were significantly lower in the presence of antiphospholipid antibodies immunoglobulin G compared with normal immunoglobulin G (49.4 +/- 8.9 ng/gm wet weight vs 57.2 +/- 11.5 ng/gm, respectively, p < 0.05). gm 190-192 annexin A5 Homo sapiens 0-9 7525726-9 1994 Most importantly, GM cultured in the presence of anti-IL-10 mAb, acquired the ability to stimulate egg Ag-specific T cells. gm 18-20 interleukin 10 Mus musculus 54-59 7525726-11 1994 In separate experiments, culture supernatants from GM exerted a powerful inhibition of costimulatory Ag expression on Con-A-stimulated peritoneal exudate cells in vivo, which could similarly be attributed to IL-10. gm 51-53 interleukin 10 Mus musculus 208-213 7963583-4 1994 In the sera of 160 patients having chronic/recurrent sinusitis, there was a highly significant correlation between the level of specific anti-M. catarrhalis IgG3 level and certain Gm phenotypes. gm 180-182 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 157-161 7980421-6 1994 Bovine lung heparins were also less effective than mucosal heparin as bFGF antagonists in GM 7373-cell-proliferation assays. gm 90-92 fibroblast growth factor 2 Bos taurus 70-74 7926812-2 1994 32D GM cells were previously shown to express significant levels of the Epo receptor mRNA and protein which was retained intracellularly and did not appear on the cell surface. gm 4-6 erythropoietin Mus musculus 72-75 7990815-5 1994 As shown by GM and competition experiments, both origins contain high-affinity binding sites for the transcription-replication factor Oct-1 and for two unknown nuclear factors. gm 12-14 POU class 2 homeobox 1 Homo sapiens 134-139 8178978-3 1994 PP-1 activities were reduced in the GM fraction from insulin-resistant subjects (54 +/- 2 vs. 61 +/- 1, P < 0.01) but, in contrast to our previously published results, were elevated in the extract (33 +/- 6 vs. 18 +/- 3, P < 0.05). gm 36-38 neuropeptide Y receptor Y4 Homo sapiens 0-4 8178978-3 1994 PP-1 activities were reduced in the GM fraction from insulin-resistant subjects (54 +/- 2 vs. 61 +/- 1, P < 0.01) but, in contrast to our previously published results, were elevated in the extract (33 +/- 6 vs. 18 +/- 3, P < 0.05). gm 36-38 insulin Homo sapiens 53-60 8178978-4 1994 Recombination of the cytosol and GM fractions (reconstituted extract) demonstrated that the low extract PP-1 activities could only be regenerated when the GM fraction from insulin-sensitive subjects was recombined with cytosol from either group. gm 33-35 neuropeptide Y receptor Y4 Homo sapiens 104-108 8178978-4 1994 Recombination of the cytosol and GM fractions (reconstituted extract) demonstrated that the low extract PP-1 activities could only be regenerated when the GM fraction from insulin-sensitive subjects was recombined with cytosol from either group. gm 155-157 insulin Homo sapiens 172-179 8299123-2 1994 The presence of these GM-suppressor cells in mice bearing LLC-LN7 tumors was associated with a reduced capacity of splenic T cells to proliferate in response to interleukin-2 (IL-2). gm 22-24 interleukin 2 Mus musculus 161-174 8299123-2 1994 The presence of these GM-suppressor cells in mice bearing LLC-LN7 tumors was associated with a reduced capacity of splenic T cells to proliferate in response to interleukin-2 (IL-2). gm 22-24 interleukin 2 Mus musculus 176-180 8170729-4 1994 In the PRP-OMP group the GM concentration was 0.44 microgram/ml and 85% had PRP antibodies > or = 0.15 microgram/ml. gm 25-27 prion protein Homo sapiens 7-10 8261414-6 1993 However, when tumor production of GM-CSF was inhibited by culture with both 1,25(OH)2D3 and IFN-gamma, the ability of the dissociated tumor culture to sustain the presence of GM-suppressor cells was blocked. gm 34-36 interferon gamma Mus musculus 92-101 8156144-14 1993 The phenotype Gm 3;5 has been associated with PM in caucasoids and may interact with DR3 in predisposing to disease. gm 14-16 TNF receptor superfamily member 25 Homo sapiens 85-88 7514454-7 1993 Furthermore, prolonged exposure of GM/SO cells to TNF-alpha for 5-7 days yielded a monocyte-macrophage-like morphology of some cells as these cells displayed an apparent glass and plastic adherence. gm 35-37 tumor necrosis factor Homo sapiens 50-59 8230568-8 1993 Conversely, the elastin content in the VV and PV was significantly reduced (VV = 53 +/- 3 mg/gm, PV = 50 +/- 4 mg/gm vs NV = 74 +/- 4 mg/gm, p < 0.05). gm 93-95 elastin Homo sapiens 16-23 8241948-8 1993 In highly purified HP/HSC undergoing E or GM differentiation, GATA-1 expression is characterized initially by proliferation-dependent activation and at later stages by sustained expression in the E pathway and suppression in the GM pathway. gm 42-44 GATA binding protein 1 Homo sapiens 62-68 8241948-8 1993 In highly purified HP/HSC undergoing E or GM differentiation, GATA-1 expression is characterized initially by proliferation-dependent activation and at later stages by sustained expression in the E pathway and suppression in the GM pathway. gm 229-231 GATA binding protein 1 Homo sapiens 62-68 8414921-6 1993 NT (10(-8) mol/l, n = 9) increased GM and CM significantly from 2.4 +/- 0.3 nS and 23.5 +/- 3 pF to 32 +/- 8 nS and 27.3 +/- 3.1 pF respectively. gm 35-37 neurotensin Homo sapiens 0-2 8414921-11 1993 With the PTM it was possible to monitor the rapid changes in GM and CM, as they were induced by ATP (n = 42) and NT (n = 29), with high time resolution. gm 61-63 neurotensin Homo sapiens 113-115 8490171-1 1993 Gunmetal (gm/gm) is a recessively inherited mouse pigment dilution mutant that has high mortality and poor reproductive rates. gm 10-12 Rab geranylgeranyl transferase, a subunit Mus musculus 0-8 8490171-1 1993 Gunmetal (gm/gm) is a recessively inherited mouse pigment dilution mutant that has high mortality and poor reproductive rates. gm 13-15 Rab geranylgeranyl transferase, a subunit Mus musculus 0-8 7686232-3 1993 In vitro colony assay also suggests an increase in sensitivity of GM progenitors to cytokines (GM-CSF, IL-3, G-CSF and/or SCF) in a patient with juvenile chronic myelogenous leukemia. gm 66-68 colony stimulating factor 2 Homo sapiens 95-101 7686232-3 1993 In vitro colony assay also suggests an increase in sensitivity of GM progenitors to cytokines (GM-CSF, IL-3, G-CSF and/or SCF) in a patient with juvenile chronic myelogenous leukemia. gm 66-68 interleukin 3 Homo sapiens 103-107 7686232-3 1993 In vitro colony assay also suggests an increase in sensitivity of GM progenitors to cytokines (GM-CSF, IL-3, G-CSF and/or SCF) in a patient with juvenile chronic myelogenous leukemia. gm 66-68 colony stimulating factor 3 Homo sapiens 109-114 7686232-3 1993 In vitro colony assay also suggests an increase in sensitivity of GM progenitors to cytokines (GM-CSF, IL-3, G-CSF and/or SCF) in a patient with juvenile chronic myelogenous leukemia. gm 66-68 KIT ligand Homo sapiens 122-125 8455636-10 1993 A urinary H chain fragment containing the VH region directly linked to C gamma 3 which contained the Gm(a) specific and Gm(x) specific amino acid residues was positive for Gm(a) but negative for Gm(x). gm 101-103 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 71-80 8455636-10 1993 A urinary H chain fragment containing the VH region directly linked to C gamma 3 which contained the Gm(a) specific and Gm(x) specific amino acid residues was positive for Gm(a) but negative for Gm(x). gm 120-122 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 71-80 8455636-10 1993 A urinary H chain fragment containing the VH region directly linked to C gamma 3 which contained the Gm(a) specific and Gm(x) specific amino acid residues was positive for Gm(a) but negative for Gm(x). gm 120-122 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 71-80 8455636-10 1993 A urinary H chain fragment containing the VH region directly linked to C gamma 3 which contained the Gm(a) specific and Gm(x) specific amino acid residues was positive for Gm(a) but negative for Gm(x). gm 120-122 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 71-80 8455636-11 1993 Another urinary H chain fragment containing only the C gamma 3 domain was negative for both Gm(a) and (x). gm 92-94 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 53-62 8455636-12 1993 These findings indicate that Gm allotypic markers may depend on conformational determinants in which strongest expression for Gm(a) and (x) depends on structures expressed by C gamma 3 linked to C gamma 2 domains. gm 29-31 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 175-184 8455636-12 1993 These findings indicate that Gm allotypic markers may depend on conformational determinants in which strongest expression for Gm(a) and (x) depends on structures expressed by C gamma 3 linked to C gamma 2 domains. gm 126-128 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 175-184 1384900-3 1992 In four patients where complete sets of serial samples were obtained, the appearance of CD34+ cells preceded the increase in CFU-GM by 24-48 h. Peak levels of CD34+ cells ranged from 0.6-5% and coincided with the peak increase in CFU-GM. gm 129-131 CD34 molecule Homo sapiens 88-92 1380541-0 1992 Heteroclitic polyclonal and monoclonal anti-Gm(a) and anti-Gm(g) human rheumatoid factors react with epitopes induced in Gm(a-), Gm(g-) IgG by interaction with antigen or by nonspecific aggregation. gm 44-46 myelin associated glycoprotein Homo sapiens 121-127 1380541-0 1992 Heteroclitic polyclonal and monoclonal anti-Gm(a) and anti-Gm(g) human rheumatoid factors react with epitopes induced in Gm(a-), Gm(g-) IgG by interaction with antigen or by nonspecific aggregation. gm 59-61 myelin associated glycoprotein Homo sapiens 121-127 1380541-0 1992 Heteroclitic polyclonal and monoclonal anti-Gm(a) and anti-Gm(g) human rheumatoid factors react with epitopes induced in Gm(a-), Gm(g-) IgG by interaction with antigen or by nonspecific aggregation. gm 59-61 myelin associated glycoprotein Homo sapiens 121-127 1380541-11 1992 The present data suggest that acquisition of the Gm(a) determinant by Gm(a-) IgG may result from subtle changes in the CH2-CH3 RF-binding region. gm 49-51 myelin associated glycoprotein Homo sapiens 70-76 1374649-5 1992 The percentage of CD34-positive cells clearly correlated with the growth of CFU-GEMM/GM and BFU-E (p less than 0.01). gm 85-87 CD34 molecule Homo sapiens 18-22 1292647-7 1992 With progenitor cell-enriched population, obtained by fluorescence-activated cell sorting (FACS) of 6-am bone marrow samples, in 3 out of 6 experiments Met- and Leu-enkephalin showed 30-35% inhibition of GM colony formation over a wide range of concentrations (10(-15)-10(-6)). gm 204-206 prodynorphin Mus musculus 161-175 22059752-6 1992 After butchery, subjective assessment of LD, GM or RF muscles could be used to identify both PSE and DFD carcasses. gm 45-47 PSE Sus scrofa 93-96 1722318-4 1991 EpoR mRNA (by RNase protection analysis) and EpoR protein (by specific antibody immunoprecipitation of metabolically labeled EpoR protein) were detectable not only in 32D and 32D Epo (an Epo-dependent subclone) but also in 32D GM, a subclone dependent for growth on granulocyte/macrophage colony-stimulating factor. gm 227-229 erythropoietin receptor Mus musculus 0-4 1722318-4 1991 EpoR mRNA (by RNase protection analysis) and EpoR protein (by specific antibody immunoprecipitation of metabolically labeled EpoR protein) were detectable not only in 32D and 32D Epo (an Epo-dependent subclone) but also in 32D GM, a subclone dependent for growth on granulocyte/macrophage colony-stimulating factor. gm 227-229 erythropoietin receptor Mus musculus 45-49 1722318-4 1991 EpoR mRNA (by RNase protection analysis) and EpoR protein (by specific antibody immunoprecipitation of metabolically labeled EpoR protein) were detectable not only in 32D and 32D Epo (an Epo-dependent subclone) but also in 32D GM, a subclone dependent for growth on granulocyte/macrophage colony-stimulating factor. gm 227-229 erythropoietin receptor Mus musculus 45-49 1722318-4 1991 EpoR mRNA (by RNase protection analysis) and EpoR protein (by specific antibody immunoprecipitation of metabolically labeled EpoR protein) were detectable not only in 32D and 32D Epo (an Epo-dependent subclone) but also in 32D GM, a subclone dependent for growth on granulocyte/macrophage colony-stimulating factor. gm 227-229 erythropoietin Mus musculus 0-3 1679291-6 1991 We have previously reported interactions between HLA, Gm (particularly G2m(23)), and IDDM and postulate that the TCRB-Gm-IDDM and HLA-Gm-IDDM interaction effects may be functionally related. gm 54-56 T cell receptor beta locus Homo sapiens 113-117 1774195-0 1991 Gm allotypes influence the production of IgG3 but the effect is age-dependent. gm 0-2 immunoglobulin heavy constant gamma 3 (G3m marker) Homo sapiens 41-45