PMID-sentid	Pub_year	Sent_text	compound_name	comp_offset	prot_official_name	organism	prot_offset
12914535-4	2003	This study was designed to examine the effects of a D(2) DA receptor agonist (D(2)AG; R(-)-propylnorapomorphine HCl) on basal and VIP-stimulated PRL gene transcription rate, PRL mRNA steady-state levels, PRL mRNA stability and PRL release from cultured turkey anterior pituitary cells.	alpha-naphthylphthalamic acid	86-115	VIP peptides	Meleagris gallopavo	130-133
16159377-6	2005	VIP or the L-type Ca2+ channel activator, Bay K8644 (Bay) increased [Ca2+]i in a concentration- and time-dependent fashion, an effect abolished by preincubating the cells with R(-)-propylnorapomorphine HCl, a D2 dopamine receptor agonist (D2AG) or Verapamil (VR), a specific L-type Ca2+ channel blocker.	alpha-naphthylphthalamic acid	176-205	VIP peptides	Meleagris gallopavo	0-3
15634200-5	2005	1-N-naphthylphthalamic acid (NPA) and triiodobenzoic acid lead to overexpression of the GA-biosynthetic gene AtGA20ox1 comparable in magnitude to the overexpression observed in seedlings treated with paclobutrazol, a GA biosynthesis inhibitor.	alpha-naphthylphthalamic acid	0-27	2-oxoglutarate (2OG) and Fe(II)-dependent oxygenase superfamily protein	Arabidopsis thaliana	109-118
15634200-5	2005	1-N-naphthylphthalamic acid (NPA) and triiodobenzoic acid lead to overexpression of the GA-biosynthetic gene AtGA20ox1 comparable in magnitude to the overexpression observed in seedlings treated with paclobutrazol, a GA biosynthesis inhibitor.	alpha-naphthylphthalamic acid	29-32	2-oxoglutarate (2OG) and Fe(II)-dependent oxygenase superfamily protein	Arabidopsis thaliana	109-118
14617075-4	2003	Previous studies have suggested that AtGSTF2 interacts with indole-3-acetic acid (IAA) and the auxin transport inhibitor 1-N-naphthylphthalamic acid (NPA).	alpha-naphthylphthalamic acid	121-148	glutathione S-transferase PHI 2	Arabidopsis thaliana	37-44
14617075-4	2003	Previous studies have suggested that AtGSTF2 interacts with indole-3-acetic acid (IAA) and the auxin transport inhibitor 1-N-naphthylphthalamic acid (NPA).	alpha-naphthylphthalamic acid	150-153	glutathione S-transferase PHI 2	Arabidopsis thaliana	37-44
17337532-6	2007	The bundling of actin impairs the synchrony of cell division and increases the sensitivity to 1-N-naphthylphthalamic acid.	alpha-naphthylphthalamic acid	94-121	actin	Nicotiana tabacum	16-21
12914535-4	2003	This study was designed to examine the effects of a D(2) DA receptor agonist (D(2)AG; R(-)-propylnorapomorphine HCl) on basal and VIP-stimulated PRL gene transcription rate, PRL mRNA steady-state levels, PRL mRNA stability and PRL release from cultured turkey anterior pituitary cells.	alpha-naphthylphthalamic acid	86-115	prolactin	Meleagris gallopavo	145-148
11842162-7	2002	First, the auxin transport inhibitor, N-1-naphthylphthalamic acid, inhibited gravistimulated curvature and Ivr2 transcript accumulation.	alpha-naphthylphthalamic acid	38-65	invertase 2	Zea mays	107-111
12586904-5	2003	The auxin polar transport inhibitors N(1)-naphthylphthalamic acid or 2,3,5-triiodobenzoic acid caused a further increase of PAO expression in outer tissues after light treatment.	alpha-naphthylphthalamic acid	37-65	polyamine oxidase 1	Zea mays	124-127
12586904-6	2003	The small increase of PAO expression, normally occurring in the mesocotyl epidermis during plant development in the dark, was also inhibited by auxin, although to a lesser extent with respect to light-exposed tissue, and was stimulated by N(1)-naphthylphthalamic acid or 2,3,5-triiodobenzoic acid, thus suggesting a complex regulation of PAO expression.	alpha-naphthylphthalamic acid	239-267	polyamine oxidase 1	Zea mays	22-25
11408534-4	2001	However, the dopamine D2 receptor agonists R-(-)-propylnorapomorphine hydrochloride (NPA) and quinpirole decreased the half-life of DAT.	alpha-naphthylphthalamic acid	85-88	solute carrier family 6 member 3	Rattus norvegicus	132-135
10695501-2	1999	Lanoteplase or n-PA, a third-generation plasminogen activator consisting of a deletion and point mutant of tissue-type plasminogen activator (t-PA), is a promising agent to approach this therapeutic goal.	alpha-naphthylphthalamic acid	15-19	plasminogen activator, tissue type	Homo sapiens	107-140
11042902-3	2000	Biochemically modified mutants of wild-type t-PA, e.g. r-PA (reteplase), n-PA (lanoteplase), and TNK-t-PA have altered biochemical and pharmacokinetic characteristics which make them more easy to use (as double or single bolus injection) and share a theoretical efficacy benefit.	alpha-naphthylphthalamic acid	73-77	plasminogen activator, tissue type	Homo sapiens	44-48
10695501-2	1999	Lanoteplase or n-PA, a third-generation plasminogen activator consisting of a deletion and point mutant of tissue-type plasminogen activator (t-PA), is a promising agent to approach this therapeutic goal.	alpha-naphthylphthalamic acid	15-19	plasminogen activator, tissue type	Homo sapiens	142-146
2963185-1	1988	The dopamine D-1 agonist SK&F 38393 (10 mg/kg) the D-2 agonist (-)-NPA (0.04 mg/kg) and d-amphetamine (1.0 mg/kg) were established as discriminative stimuli versus saline in rats.	alpha-naphthylphthalamic acid	67-74	solute carrier family 3 member 1	Rattus norvegicus	55-58
9351244-1	1997	In order to clarify the mechanism underlying the polar auxin transport system, the pis1 mutant in Arabidopsis thaliana that is hypersensitive to N-1-naphthylphthalamic acid (NPA), an auxin transport inhibitor was isolated and characterized.	alpha-naphthylphthalamic acid	145-172	phosphatidylinositol synthase 1	Arabidopsis thaliana	83-87
9351244-1	1997	In order to clarify the mechanism underlying the polar auxin transport system, the pis1 mutant in Arabidopsis thaliana that is hypersensitive to N-1-naphthylphthalamic acid (NPA), an auxin transport inhibitor was isolated and characterized.	alpha-naphthylphthalamic acid	174-177	phosphatidylinositol synthase 1	Arabidopsis thaliana	83-87
7798946-7	1995	Direct measurement of the effect of 3-NPA on SDH activity showed that 3-NPA dose-dependently inhibited SDH in vitro in a manner commensurate with the loss of dopamine uptake by 3-NPA.	alpha-naphthylphthalamic acid	37-41	succinate dehydrogenase complex iron sulfur subunit B	Homo sapiens	45-48
7798946-7	1995	Direct measurement of the effect of 3-NPA on SDH activity showed that 3-NPA dose-dependently inhibited SDH in vitro in a manner commensurate with the loss of dopamine uptake by 3-NPA.	alpha-naphthylphthalamic acid	37-41	succinate dehydrogenase complex iron sulfur subunit B	Homo sapiens	103-106
34895546-6	2022	Dose-response analysis supplementing indole-3-acetic acid (IAA), or the auxin transport inhibitor N-1-naphthylphthalamic acid (NPA), indicated normal responses to auxin in the med16-2 and med16-3 mutants regarding primary root growth and lateral root formation, but strong resistance to NPA in primary roots, which could be correlated with cell division and elongation.	alpha-naphthylphthalamic acid	98-125	sensitive to freezing 6	Arabidopsis thaliana	176-181
34895546-6	2022	Dose-response analysis supplementing indole-3-acetic acid (IAA), or the auxin transport inhibitor N-1-naphthylphthalamic acid (NPA), indicated normal responses to auxin in the med16-2 and med16-3 mutants regarding primary root growth and lateral root formation, but strong resistance to NPA in primary roots, which could be correlated with cell division and elongation.	alpha-naphthylphthalamic acid	98-125	sensitive to freezing 6	Arabidopsis thaliana	188-195
34895546-6	2022	Dose-response analysis supplementing indole-3-acetic acid (IAA), or the auxin transport inhibitor N-1-naphthylphthalamic acid (NPA), indicated normal responses to auxin in the med16-2 and med16-3 mutants regarding primary root growth and lateral root formation, but strong resistance to NPA in primary roots, which could be correlated with cell division and elongation.	alpha-naphthylphthalamic acid	127-130	sensitive to freezing 6	Arabidopsis thaliana	176-181
34895546-6	2022	Dose-response analysis supplementing indole-3-acetic acid (IAA), or the auxin transport inhibitor N-1-naphthylphthalamic acid (NPA), indicated normal responses to auxin in the med16-2 and med16-3 mutants regarding primary root growth and lateral root formation, but strong resistance to NPA in primary roots, which could be correlated with cell division and elongation.	alpha-naphthylphthalamic acid	127-130	sensitive to freezing 6	Arabidopsis thaliana	188-195
2963185-6	1988	The (-)-NPA stimulus was dependent on postsynaptic D-2 receptors: It was mimicked by quinpirole and pergolide in stimulant dosages, whereas the partial D-2 agonist (-)-3-PPP inhibited the effect of (-)-NPA.	alpha-naphthylphthalamic acid	8-11	solute carrier family 3 member 1	Rattus norvegicus	51-54
2963185-6	1988	The (-)-NPA stimulus was dependent on postsynaptic D-2 receptors: It was mimicked by quinpirole and pergolide in stimulant dosages, whereas the partial D-2 agonist (-)-3-PPP inhibited the effect of (-)-NPA.	alpha-naphthylphthalamic acid	8-11	solute carrier family 3 member 1	Rattus norvegicus	152-155
6617745-1	1983	NCA, the chloro analog of the potent dopamine agonist NPA is an irreversible ligand at dopamine receptors in mammalian brain.	alpha-naphthylphthalamic acid	54-57	CEA cell adhesion molecule 6	Homo sapiens	0-3
31377726-8	2019	The inhibition effect of naphthylphthalamic acid treatment and exogenous IAA application depends on a known auxin signaling pathway involving AUXIN RESPONSE FACTOR7 (ARF7)/ARF19 and LBD29.	alpha-naphthylphthalamic acid	25-48	Transcriptional factor B3 family protein / auxin-responsive factor AUX/IAA-like protein	Arabidopsis thaliana	142-164
33081176-4	2020	1-N-naphthylphtalamic acid (NPA) disrupted the dynamic distribution of the WUS signal induced by early 24 h R treatment, and NPA plus 24 R treatment increased the average shoot number compared with early 24 h R alone.	alpha-naphthylphthalamic acid	0-26	Homeodomain-like superfamily protein	Arabidopsis thaliana	75-78
33081176-4	2020	1-N-naphthylphtalamic acid (NPA) disrupted the dynamic distribution of the WUS signal induced by early 24 h R treatment, and NPA plus 24 R treatment increased the average shoot number compared with early 24 h R alone.	alpha-naphthylphthalamic acid	28-31	Homeodomain-like superfamily protein	Arabidopsis thaliana	75-78
31377726-8	2019	The inhibition effect of naphthylphthalamic acid treatment and exogenous IAA application depends on a known auxin signaling pathway involving AUXIN RESPONSE FACTOR7 (ARF7)/ARF19 and LBD29.	alpha-naphthylphthalamic acid	25-48	Transcriptional factor B3 family protein / auxin-responsive factor AUX/IAA-like protein	Arabidopsis thaliana	166-170
31377726-8	2019	The inhibition effect of naphthylphthalamic acid treatment and exogenous IAA application depends on a known auxin signaling pathway involving AUXIN RESPONSE FACTOR7 (ARF7)/ARF19 and LBD29.	alpha-naphthylphthalamic acid	25-48	auxin response factor 19	Arabidopsis thaliana	172-177
31377726-8	2019	The inhibition effect of naphthylphthalamic acid treatment and exogenous IAA application depends on a known auxin signaling pathway involving AUXIN RESPONSE FACTOR7 (ARF7)/ARF19 and LBD29.	alpha-naphthylphthalamic acid	25-48	lateral organ boundaries-domain 29	Arabidopsis thaliana	182-187
31347842-2	2019	Eight novel peptidomimetics were tested for their ability to enhance the maximal effect of tritiated N-propylapomorphine ([3H]-NPA) at dopamine D2 receptors (D2R).	alpha-naphthylphthalamic acid	127-130	dopamine receptor D2	Homo sapiens	144-156
31347842-2	2019	Eight novel peptidomimetics were tested for their ability to enhance the maximal effect of tritiated N-propylapomorphine ([3H]-NPA) at dopamine D2 receptors (D2R).	alpha-naphthylphthalamic acid	127-130	dopamine receptor D2	Homo sapiens	158-161
30496625-5	2019	The root elongation of plc2 mutants was less sensitive to the high concentration of exogenous auxins, and the application of 1-naphthaleneacetic acid or the auxin transport inhibitor N-1-naphthylphthalamic acid could rescue the root hair growth of plc2 mutants.	alpha-naphthylphthalamic acid	183-210	phospholipase C 2	Arabidopsis thaliana	23-27
30496625-5	2019	The root elongation of plc2 mutants was less sensitive to the high concentration of exogenous auxins, and the application of 1-naphthaleneacetic acid or the auxin transport inhibitor N-1-naphthylphthalamic acid could rescue the root hair growth of plc2 mutants.	alpha-naphthylphthalamic acid	183-210	phospholipase C 2	Arabidopsis thaliana	248-252
29114079-9	2018	Moreover, gif1 gif2 double mutations and 35S:MIR396 also acted synergistically with 1-N-naphthylphthalamic acid in forming aberrant gynoecia.	alpha-naphthylphthalamic acid	84-111	SSXT family protein	Arabidopsis thaliana	10-19
27770560-7	2017	Conversely, auxin efflux inhibitors N-1-naphthylphthalamic acid and 2,3,5-triiodobenzoic acid affect BL-induced relocalization of clathrin, endocytosis and lateralization of PIN3 as well as asymmetric distribution of auxin.	alpha-naphthylphthalamic acid	36-63	Auxin efflux carrier family protein	Arabidopsis thaliana	174-178
28780645-3	2017	An analysis of primary root growth of WT, med12, aux1-7 and med12 aux1 single and double mutants in response to sucrose and/or N-1-naphthylphthalamic acid (NPA) placed MED12 upstream of auxin transport for the sugar modulation of root growth.	alpha-naphthylphthalamic acid	127-154	RNA polymerase II transcription mediator	Arabidopsis thaliana	42-47
28780645-3	2017	An analysis of primary root growth of WT, med12, aux1-7 and med12 aux1 single and double mutants in response to sucrose and/or N-1-naphthylphthalamic acid (NPA) placed MED12 upstream of auxin transport for the sugar modulation of root growth.	alpha-naphthylphthalamic acid	127-154	Transmembrane amino acid transporter family protein	Arabidopsis thaliana	49-55
28780645-3	2017	An analysis of primary root growth of WT, med12, aux1-7 and med12 aux1 single and double mutants in response to sucrose and/or N-1-naphthylphthalamic acid (NPA) placed MED12 upstream of auxin transport for the sugar modulation of root growth.	alpha-naphthylphthalamic acid	127-154	RNA polymerase II transcription mediator	Arabidopsis thaliana	60-65
28780645-3	2017	An analysis of primary root growth of WT, med12, aux1-7 and med12 aux1 single and double mutants in response to sucrose and/or N-1-naphthylphthalamic acid (NPA) placed MED12 upstream of auxin transport for the sugar modulation of root growth.	alpha-naphthylphthalamic acid	127-154	Transmembrane amino acid transporter family protein	Arabidopsis thaliana	49-53
28780645-3	2017	An analysis of primary root growth of WT, med12, aux1-7 and med12 aux1 single and double mutants in response to sucrose and/or N-1-naphthylphthalamic acid (NPA) placed MED12 upstream of auxin transport for the sugar modulation of root growth.	alpha-naphthylphthalamic acid	127-154	RNA polymerase II transcription mediator	Arabidopsis thaliana	168-173
27342309-6	2016	SOB3-D and sob3-6 hypocotyls also exhibited altered sensitivity to the polar auxin transport inhibitor N-1-napthylphthalamic acid, suggesting a critical connection between auxin and the modulation of seedling elongation by SOB3 Finally, we found that overexpression of GREEN FLUORESCENT PROTEIN-SAUR19 in the SOB3-D line partially rescued defects in hypocotyl elongation, and SOB3 bound directly to the promoters of YUC8 and SAUR19 subfamily members.	alpha-naphthylphthalamic acid	103-129	Putative AT-hook DNA-binding family protein	Arabidopsis thaliana	0-4
27342309-6	2016	SOB3-D and sob3-6 hypocotyls also exhibited altered sensitivity to the polar auxin transport inhibitor N-1-napthylphthalamic acid, suggesting a critical connection between auxin and the modulation of seedling elongation by SOB3 Finally, we found that overexpression of GREEN FLUORESCENT PROTEIN-SAUR19 in the SOB3-D line partially rescued defects in hypocotyl elongation, and SOB3 bound directly to the promoters of YUC8 and SAUR19 subfamily members.	alpha-naphthylphthalamic acid	103-129	Putative AT-hook DNA-binding family protein	Arabidopsis thaliana	11-15
27342309-6	2016	SOB3-D and sob3-6 hypocotyls also exhibited altered sensitivity to the polar auxin transport inhibitor N-1-napthylphthalamic acid, suggesting a critical connection between auxin and the modulation of seedling elongation by SOB3 Finally, we found that overexpression of GREEN FLUORESCENT PROTEIN-SAUR19 in the SOB3-D line partially rescued defects in hypocotyl elongation, and SOB3 bound directly to the promoters of YUC8 and SAUR19 subfamily members.	alpha-naphthylphthalamic acid	103-129	Putative AT-hook DNA-binding family protein	Arabidopsis thaliana	223-227
27342309-6	2016	SOB3-D and sob3-6 hypocotyls also exhibited altered sensitivity to the polar auxin transport inhibitor N-1-napthylphthalamic acid, suggesting a critical connection between auxin and the modulation of seedling elongation by SOB3 Finally, we found that overexpression of GREEN FLUORESCENT PROTEIN-SAUR19 in the SOB3-D line partially rescued defects in hypocotyl elongation, and SOB3 bound directly to the promoters of YUC8 and SAUR19 subfamily members.	alpha-naphthylphthalamic acid	103-129	SAUR-like auxin-responsive protein family	Arabidopsis thaliana	287-301
27342309-6	2016	SOB3-D and sob3-6 hypocotyls also exhibited altered sensitivity to the polar auxin transport inhibitor N-1-napthylphthalamic acid, suggesting a critical connection between auxin and the modulation of seedling elongation by SOB3 Finally, we found that overexpression of GREEN FLUORESCENT PROTEIN-SAUR19 in the SOB3-D line partially rescued defects in hypocotyl elongation, and SOB3 bound directly to the promoters of YUC8 and SAUR19 subfamily members.	alpha-naphthylphthalamic acid	103-129	Putative AT-hook DNA-binding family protein	Arabidopsis thaliana	223-227
27342309-6	2016	SOB3-D and sob3-6 hypocotyls also exhibited altered sensitivity to the polar auxin transport inhibitor N-1-napthylphthalamic acid, suggesting a critical connection between auxin and the modulation of seedling elongation by SOB3 Finally, we found that overexpression of GREEN FLUORESCENT PROTEIN-SAUR19 in the SOB3-D line partially rescued defects in hypocotyl elongation, and SOB3 bound directly to the promoters of YUC8 and SAUR19 subfamily members.	alpha-naphthylphthalamic acid	103-129	Putative AT-hook DNA-binding family protein	Arabidopsis thaliana	223-227
27342309-6	2016	SOB3-D and sob3-6 hypocotyls also exhibited altered sensitivity to the polar auxin transport inhibitor N-1-napthylphthalamic acid, suggesting a critical connection between auxin and the modulation of seedling elongation by SOB3 Finally, we found that overexpression of GREEN FLUORESCENT PROTEIN-SAUR19 in the SOB3-D line partially rescued defects in hypocotyl elongation, and SOB3 bound directly to the promoters of YUC8 and SAUR19 subfamily members.	alpha-naphthylphthalamic acid	103-129	Flavin-binding monooxygenase family protein	Arabidopsis thaliana	416-420
27342309-6	2016	SOB3-D and sob3-6 hypocotyls also exhibited altered sensitivity to the polar auxin transport inhibitor N-1-napthylphthalamic acid, suggesting a critical connection between auxin and the modulation of seedling elongation by SOB3 Finally, we found that overexpression of GREEN FLUORESCENT PROTEIN-SAUR19 in the SOB3-D line partially rescued defects in hypocotyl elongation, and SOB3 bound directly to the promoters of YUC8 and SAUR19 subfamily members.	alpha-naphthylphthalamic acid	103-129	SAUR-like auxin-responsive protein family	Arabidopsis thaliana	295-301
21156857-6	2011	N-1-Naphthylphthalamic acid, an inhibitor of auxin efflux activity, reduced the Pi starvation-induced LR root formation of siz1 seedlings to a level equivalent to that seen in the wild type.	alpha-naphthylphthalamic acid	0-27	DNA-binding protein with MIZ/SP-RING zinc finger, PHD-finger and SAP domain-containing protein	Arabidopsis thaliana	123-127
25870601-8	2015	Indole-3-acetic acid (IAA) and N-1-napthylphthalamic acid (NPA) treatment experiments revealed differential up-regulation and down-regulation, respectively, of the 19 citrus ARF genes in the callus of sweet orange.	alpha-naphthylphthalamic acid	31-57	auxin-response factor	Citrus sinensis	174-177
25870601-8	2015	Indole-3-acetic acid (IAA) and N-1-napthylphthalamic acid (NPA) treatment experiments revealed differential up-regulation and down-regulation, respectively, of the 19 citrus ARF genes in the callus of sweet orange.	alpha-naphthylphthalamic acid	59-62	auxin-response factor	Citrus sinensis	174-177
24803136-3	2015	However, treatment with the auxin transport inhibitor N-1-naphthylphthalamic acid significantly reduced PHOT1 expression in this zone.	alpha-naphthylphthalamic acid	54-81	phototropin 1	Arabidopsis thaliana	104-109
23147222-7	2012	Further functional analysis of ABCB21 using yeast import and export assays showed that ABCB21 mediates the 1-N-naphthylphthalamic acid (NPA)-sensitive translocation of auxin in an inward direction when the cytoplasmic IAA concentration is low, whereas this transporter mediates outward transport under high internal IAA.	alpha-naphthylphthalamic acid	107-134	P-glycoprotein 21	Arabidopsis thaliana	31-37
23147222-7	2012	Further functional analysis of ABCB21 using yeast import and export assays showed that ABCB21 mediates the 1-N-naphthylphthalamic acid (NPA)-sensitive translocation of auxin in an inward direction when the cytoplasmic IAA concentration is low, whereas this transporter mediates outward transport under high internal IAA.	alpha-naphthylphthalamic acid	107-134	P-glycoprotein 21	Arabidopsis thaliana	87-93
23147222-7	2012	Further functional analysis of ABCB21 using yeast import and export assays showed that ABCB21 mediates the 1-N-naphthylphthalamic acid (NPA)-sensitive translocation of auxin in an inward direction when the cytoplasmic IAA concentration is low, whereas this transporter mediates outward transport under high internal IAA.	alpha-naphthylphthalamic acid	136-139	P-glycoprotein 21	Arabidopsis thaliana	31-37
23147222-7	2012	Further functional analysis of ABCB21 using yeast import and export assays showed that ABCB21 mediates the 1-N-naphthylphthalamic acid (NPA)-sensitive translocation of auxin in an inward direction when the cytoplasmic IAA concentration is low, whereas this transporter mediates outward transport under high internal IAA.	alpha-naphthylphthalamic acid	136-139	P-glycoprotein 21	Arabidopsis thaliana	87-93
22025721-4	2011	Here, we show that all of the taa mutant phenotypes, including defects in shade avoidance, root resistance to ethylene and N-1-naphthylphthalamic acid (NPA), are phenocopied by inactivating YUC genes.	alpha-naphthylphthalamic acid	123-150	Flavin-binding monooxygenase family protein	Arabidopsis thaliana	190-193
22025721-4	2011	Here, we show that all of the taa mutant phenotypes, including defects in shade avoidance, root resistance to ethylene and N-1-naphthylphthalamic acid (NPA), are phenocopied by inactivating YUC genes.	alpha-naphthylphthalamic acid	152-155	Flavin-binding monooxygenase family protein	Arabidopsis thaliana	190-193
21533019-6	2011	Here we show that specific ACS isozymes are targets for regulation by protein phosphatase 2A (PP2A) during Arabidopsis thaliana seedling growth and that reduced PP2A function causes increased ACS activity in the roots curl in 1-N-naphthylphthalamic acid 1 (rcn1) mutant.	alpha-naphthylphthalamic acid	226-253	serine/threonine protein phosphatase 2A	Arabidopsis thaliana	78-92
21533019-6	2011	Here we show that specific ACS isozymes are targets for regulation by protein phosphatase 2A (PP2A) during Arabidopsis thaliana seedling growth and that reduced PP2A function causes increased ACS activity in the roots curl in 1-N-naphthylphthalamic acid 1 (rcn1) mutant.	alpha-naphthylphthalamic acid	226-253	serine/threonine protein phosphatase 2A	Arabidopsis thaliana	94-98
21533019-6	2011	Here we show that specific ACS isozymes are targets for regulation by protein phosphatase 2A (PP2A) during Arabidopsis thaliana seedling growth and that reduced PP2A function causes increased ACS activity in the roots curl in 1-N-naphthylphthalamic acid 1 (rcn1) mutant.	alpha-naphthylphthalamic acid	226-253	serine/threonine protein phosphatase 2A	Arabidopsis thaliana	161-165
21533019-6	2011	Here we show that specific ACS isozymes are targets for regulation by protein phosphatase 2A (PP2A) during Arabidopsis thaliana seedling growth and that reduced PP2A function causes increased ACS activity in the roots curl in 1-N-naphthylphthalamic acid 1 (rcn1) mutant.	alpha-naphthylphthalamic acid	226-253	ARM repeat superfamily protein	Arabidopsis thaliana	257-261
24164597-7	2014	Moreover, the rel3 mutant was more resistant than wild-type to 1-naphthaleneacetic acid (NAA) and N-1-naphthylphthalamic acid (NPA) in root growth, and exhibited insensitivity to auxins but greater sensitivity to auxin transport inhibitors during nodulation.	alpha-naphthylphthalamic acid	98-125	REL3	Lotus japonicus	14-18
24164597-7	2014	Moreover, the rel3 mutant was more resistant than wild-type to 1-naphthaleneacetic acid (NAA) and N-1-naphthylphthalamic acid (NPA) in root growth, and exhibited insensitivity to auxins but greater sensitivity to auxin transport inhibitors during nodulation.	alpha-naphthylphthalamic acid	127-130	REL3	Lotus japonicus	14-18
23065197-1	2012	Aminopeptidase M1 (APM1) was the first M1 metallopeptidase family member identified in Arabidopsis, isolated by its affinity for the auxin transport inhibitor N-1-naphthylphthalamic acid (NPA).	alpha-naphthylphthalamic acid	159-186	aminopeptidase M1	Arabidopsis thaliana	0-17
23065197-1	2012	Aminopeptidase M1 (APM1) was the first M1 metallopeptidase family member identified in Arabidopsis, isolated by its affinity for the auxin transport inhibitor N-1-naphthylphthalamic acid (NPA).	alpha-naphthylphthalamic acid	159-186	aminopeptidase M1	Arabidopsis thaliana	19-23
23065197-1	2012	Aminopeptidase M1 (APM1) was the first M1 metallopeptidase family member identified in Arabidopsis, isolated by its affinity for the auxin transport inhibitor N-1-naphthylphthalamic acid (NPA).	alpha-naphthylphthalamic acid	188-191	aminopeptidase M1	Arabidopsis thaliana	0-17
23065197-1	2012	Aminopeptidase M1 (APM1) was the first M1 metallopeptidase family member identified in Arabidopsis, isolated by its affinity for the auxin transport inhibitor N-1-naphthylphthalamic acid (NPA).	alpha-naphthylphthalamic acid	188-191	aminopeptidase M1	Arabidopsis thaliana	19-23
21926336-5	2011	In Arabidopsis thaliana inflorescence stems, 1-N-naphthylphthalamic acid-induced auxin accumulation stimulates cambium activity in the wild type but not in wox4 mutants, although basal cambium activity is not abolished.	alpha-naphthylphthalamic acid	45-72	WUSCHEL related homeobox 4	Arabidopsis thaliana	156-160
19704429-1	2008	A new Arabidopsis mutant is characterized (rha1) that shows, in the roots, reduced right-handed slanting, reduced gravitropism and resistance to 2,4-D, TIBA, NPA and ethylene.	alpha-naphthylphthalamic acid	158-161	Ras small GTP-binding family protein	Arabidopsis thaliana	43-47
19736566-3	2009	The altered responses of 5pt13 mutants to 1-N-naphthylphthalamic acid (an auxin transport inhibitor) indicate that 5PTase13 might be involved in the regulation of auxin transport.	alpha-naphthylphthalamic acid	42-69	Endonuclease/exonuclease/phosphatase family protein	Arabidopsis thaliana	115-123
19825605-2	2008	The inhibitory effects by 1-N-naphthylphthalamic acid (NPA), an inhibitor of polar auxin transport, are suppressed under the MSBP1 overexpression, suggesting the positive effects of MSBP1 on polar auxin transport.	alpha-naphthylphthalamic acid	26-53	minisatellite binding protein 1	Homo sapiens	125-130
19825605-2	2008	The inhibitory effects by 1-N-naphthylphthalamic acid (NPA), an inhibitor of polar auxin transport, are suppressed under the MSBP1 overexpression, suggesting the positive effects of MSBP1 on polar auxin transport.	alpha-naphthylphthalamic acid	26-53	minisatellite binding protein 1	Homo sapiens	182-187
19825605-2	2008	The inhibitory effects by 1-N-naphthylphthalamic acid (NPA), an inhibitor of polar auxin transport, are suppressed under the MSBP1 overexpression, suggesting the positive effects of MSBP1 on polar auxin transport.	alpha-naphthylphthalamic acid	55-58	minisatellite binding protein 1	Homo sapiens	125-130
19825605-2	2008	The inhibitory effects by 1-N-naphthylphthalamic acid (NPA), an inhibitor of polar auxin transport, are suppressed under the MSBP1 overexpression, suggesting the positive effects of MSBP1 on polar auxin transport.	alpha-naphthylphthalamic acid	55-58	minisatellite binding protein 1	Homo sapiens	182-187