PMID-sentid	Pub_year	Sent_text	compound_name	comp_offset	prot_official_name	organism	prot_offset
32261371-0	2014	Label-free aptamer biosensor for thrombin detection on a nanocomposite of graphene and plasma polymerized allylamine.	Allylamine	106-116	coagulation factor II, thrombin	Homo sapiens	33-41
26454886-4	2016	Out of a panel of 68 selected targets, we identified plasma miR-208a-3p as being significantly upregulated after single administration with either isoproterenol (ISO) or allylamine (AAM).	Allylamine	170-180	membrane associated ring-CH-type finger 8	Rattus norvegicus	60-63
26454886-4	2016	Out of a panel of 68 selected targets, we identified plasma miR-208a-3p as being significantly upregulated after single administration with either isoproterenol (ISO) or allylamine (AAM).	Allylamine	182-185	membrane associated ring-CH-type finger 8	Rattus norvegicus	60-63
24663102-1	2014	We have investigated the dynamics and mechanistic details of the interaction between bovine serum albumin (BSA) and allylamine-capped silicon quantum dots (Si QDs) by means of fluorescence spectroscopy, circular dichroism (CD), and FTIR spectroscopy.	Allylamine	116-126	albumin	Homo sapiens	92-105
32261371-1	2014	A label-free and effective aptasensor based on an amino-functionalized nanocomposite of graphene and plasma-polymerized allylamine (G-PPAA) was developed for thrombin detection.	Allylamine	120-130	coagulation factor II, thrombin	Homo sapiens	158-166
24320984-0	2013	Effect of self-association of bovine serum albumin on the stability of surfactant-induced aggregates of allylamine-capped silicon quantum dots.	Allylamine	104-114	albumin	Homo sapiens	37-50
22113586-1	2012	An efficient synthetic method for stereoselective construction of asymmetric quaternary carbon stereocenters, bearing nitrogen in the form of Boc-protected allyl amines, has been developed.	Allylamine	156-168	BOC cell adhesion associated, oncogene regulated	Homo sapiens	142-145
22136802-0	2012	Layer-by-layer films composed of poly(allylamine) and insulin for pH-triggered release of insulin.	Allylamine	38-48	insulin	Homo sapiens	90-97
24171660-7	2013	The composition of PAH/CD-S sprayed films determined by X-ray photoelectron spectroscopy is independent of the spraying rate ratio of the two constituents and corresponds to one allylamine for one sulfate group.	Allylamine	178-188	phenylalanine hydroxylase	Homo sapiens	19-22
24171660-7	2013	The composition of PAH/CD-S sprayed films determined by X-ray photoelectron spectroscopy is independent of the spraying rate ratio of the two constituents and corresponds to one allylamine for one sulfate group.	Allylamine	178-188	CDP-diacylglycerol synthase 1	Homo sapiens	23-27
22877639-0	2012	Direct thrombin inhibitor-bivalirudin functionalized plasma polymerized allylamine coating for improved biocompatibility of vascular devices.	Allylamine	72-82	coagulation factor II, thrombin	Homo sapiens	7-15
22032345-2	2012	Previous studies have shown that OPN (osteopontin) mRNA and protein levels increase significantly on induction of proliferative activity by allylamine (an atherogenic amine) and that this response can be inhibited by OPN antibodies.	Allylamine	140-150	secreted phosphoprotein 1	Mus musculus	33-36
22032345-2	2012	Previous studies have shown that OPN (osteopontin) mRNA and protein levels increase significantly on induction of proliferative activity by allylamine (an atherogenic amine) and that this response can be inhibited by OPN antibodies.	Allylamine	140-150	secreted phosphoprotein 1	Mus musculus	38-49
19941016-1	2010	A simple approach to a new family of enantiomerically enriched polyunsaturated t-Boc-protected-delta-amino esters is described, via microwave promoted Stille coupling of (Z)-methyl-2-bromobutenoate with stannylated allylamines.	Allylamine	215-226	BOC cell adhesion associated, oncogene regulated	Homo sapiens	81-84
22315656-1	2012	Repeated cycles of oxidative injury by allylamine in vivo induce a proliferative rat vascular (aortic) smooth muscle cell (vSMC) phenotype characterized by matrix-dependent enhancement of mitogenic sensitivity, changes in cell surface integrin expression, and osteopontin (opn) overexpression.	Allylamine	39-49	secreted phosphoprotein 1	Rattus norvegicus	260-271
22315656-1	2012	Repeated cycles of oxidative injury by allylamine in vivo induce a proliferative rat vascular (aortic) smooth muscle cell (vSMC) phenotype characterized by matrix-dependent enhancement of mitogenic sensitivity, changes in cell surface integrin expression, and osteopontin (opn) overexpression.	Allylamine	39-49	secreted phosphoprotein 1	Rattus norvegicus	273-276
22315656-4	2012	The NF-kappaB DNA binding element located at -1943 in the 5"-UTR strongly inhibited opn promoter activity in allylamine vSMCs, and this response was regulated by the extracellular matrix.	Allylamine	109-119	secreted phosphoprotein 1	Rattus norvegicus	84-87
21968664-1	2011	OBJECTIVE: Naftifine HCl 2% cream (NAFT-2%) is a topical allylamine antifungal preparation under development in the U.S.	Allylamine	57-67	HCL2	Homo sapiens	21-26
18799315-1	2008	The parkinsonian inducing agent, 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP), is a cyclic tertiary allylamine exhibiting good monoamine oxidase B (MAO-B) substrate properties.	Allylamine	107-117	monoamine oxidase B	Bos taurus	134-153
19766178-0	2010	The influence of polymer architecture on the protective effect of novel comb shaped amphiphilic poly(allylamine) against in vitro enzymatic degradation of insulin--towards oral insulin delivery.	Allylamine	101-111	insulin	Homo sapiens	155-162
19766178-0	2010	The influence of polymer architecture on the protective effect of novel comb shaped amphiphilic poly(allylamine) against in vitro enzymatic degradation of insulin--towards oral insulin delivery.	Allylamine	101-111	insulin	Homo sapiens	177-184
19053775-6	2008	The X-ray crystal structure of the mofegiline-MAO-B adduct shows a covalent bond between the flavin cofactor N(5) with the distal allylamine carbon atom as well as the absence of the fluorine atom.	Allylamine	130-140	monoamine oxidase B	Homo sapiens	46-51
18799315-1	2008	The parkinsonian inducing agent, 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP), is a cyclic tertiary allylamine exhibiting good monoamine oxidase B (MAO-B) substrate properties.	Allylamine	107-117	monoamine oxidase B	Bos taurus	155-160
18357783-4	2007	Allylamine, diethylaminoethylmethacrylate, and N,N"-methylenebisacrylamide, providing high binding energies with aflatoxins B1, B2, and G2 were selected as functional monomers for the formation of aflatoxin B1-imprinted polymer membranes.	Allylamine	0-10	immunoglobulin kappa variable 7-3 (pseudogene)	Homo sapiens	124-138
17981471-5	2008	The in vitro activities of these novel compounds were superior to that of nifurtimox, a nitrofuran currently used in the treatment of human Chagas" disease, and terbinafine, a commercially available allylamine-based squalene epoxidase inhibitor.	Allylamine	199-209	squalene epoxidase	Homo sapiens	216-234
16019066-3	2005	The EGF was covalently tethered to PDMS substrates aminated by plasma polymerization of allylamine via a homobifunctional polyethylene glycol (PEG) spacer.	Allylamine	88-98	epidermal growth factor	Homo sapiens	4-7
17492711-1	2007	The adsorption of BSA and fibrinogen onto plasma-polymerized di-(ethylene glycol) vinyl ether, allylamine, and maleic anhydride films were investigated in detail by surface plasmon resonance spectroscopy (SPR).	Allylamine	95-105	fibrinogen beta chain	Homo sapiens	26-36
17043127-10	2007	In addition, the amino acids responsible for terbinafine resistance, although they are distributed along the sequence of Erg1p, cluster on the surface of the Erg1p model, giving rise to a putative binding site for allylamines.	Allylamine	214-225	squalene monooxygenase	Saccharomyces cerevisiae S288C	121-126
17043127-10	2007	In addition, the amino acids responsible for terbinafine resistance, although they are distributed along the sequence of Erg1p, cluster on the surface of the Erg1p model, giving rise to a putative binding site for allylamines.	Allylamine	214-225	squalene monooxygenase	Saccharomyces cerevisiae S288C	158-163
16817187-3	2006	In this work, covalent tethering of epidermal growth factor (EGF) to allylamine plasma modified polydimethylsiloxane (PDMS) substrates is characterized to determine the nature of the bound growth factor and to optimize the conditions for the reaction.	Allylamine	69-79	epidermal growth factor	Homo sapiens	36-59
16817187-3	2006	In this work, covalent tethering of epidermal growth factor (EGF) to allylamine plasma modified polydimethylsiloxane (PDMS) substrates is characterized to determine the nature of the bound growth factor and to optimize the conditions for the reaction.	Allylamine	69-79	epidermal growth factor	Homo sapiens	61-64
16808534-2	2006	The diastereoselection is dependent on the temperature of the reaction and the structure of the substituent at C-2 and can be rationalized by accepting a 1,4-asymmetric induction process after coordination of the selenium to the nitrogen atom of the allylamine system.	Allylamine	250-260	complement C2	Homo sapiens	111-114
17095030-3	2006	Allylamine induces hypercontraction in isolated rat coronary artery in a semicarbazide-sensitive amine oxidase activity (SSAO) dependent manner.	Allylamine	0-10	amine oxidase, copper containing 3	Rattus norvegicus	121-125
17095030-8	2006	In isolated aorta of spontaneously hypertensive rat, allylamine-induced an SSAO-dependent contraction and enhanced norepinephrine sensitivity but not in Sprague-Dawley rat aorta.	Allylamine	53-63	amine oxidase, copper containing 3	Rattus norvegicus	75-79
16986970-1	2006	Terminal alkynes react with allylamine in the presence of a RhCl(PPh3)3 catalyst to give (E)-3-alkylidene-3,4-dihydro-2H-pyrroles.	Allylamine	28-38	caveolin 1	Homo sapiens	65-69
15252491-8	2004	In contrast, the reaction with allylamine gave only the disubstituted complex [(ReCl2[eta1-N2C(O)Ph][eta1-NH2CH2CH=CH2]2(PPh3)] (8).	Allylamine	31-41	caveolin 1	Homo sapiens	121-125
15048878-0	2004	Modulation of cyclin dependent kinase inhibitor proteins and ERK1/2 activity in allylamine-injured vascular smooth muscle cells.	Allylamine	80-90	mitogen-activated protein kinase 3	Homo sapiens	61-67
7814369-7	1995	This deduced rat SE sequence is 30.2% identical to the ERG 1 gene, which encodes SE from an allylamine-resistant Saccharomyces cerevisiae mutant.	Allylamine	92-102	squalene epoxidase	Rattus norvegicus	17-19
14697907-12	2004	Recent studies showed that vascular SSAO metabolizes endogenous primary amines, allylamine, methylamine and aminoacetone, to the corresponding cytotoxic aldehydes.	Allylamine	80-90	amine oxidase copper containing 2	Homo sapiens	36-40
11996948-1	2002	Repeated cycles of oxidative injury by allylamine induce proliferative rat vascular smooth muscle cell (vSMC) phenotypes characterized by enhanced secretion of osteopontin (OPN).	Allylamine	39-49	secreted phosphoprotein 1	Rattus norvegicus	160-171
11996948-1	2002	Repeated cycles of oxidative injury by allylamine induce proliferative rat vascular smooth muscle cell (vSMC) phenotypes characterized by enhanced secretion of osteopontin (OPN).	Allylamine	39-49	secreted phosphoprotein 1	Rattus norvegicus	173-176
9226551-2	1997	Under the CZE conditions employed, a peaks of beta-trace protein (beta TP), which is the most abundant low MW protein in CSF, was clearly detected on the electropherograms of all the samples examined, and the CSF beta TP level could be tentatively determined using allylamine added at a constant concentration as the internal standard.	Allylamine	265-275	prostaglandin D2 synthase	Homo sapiens	46-64
9226551-2	1997	Under the CZE conditions employed, a peaks of beta-trace protein (beta TP), which is the most abundant low MW protein in CSF, was clearly detected on the electropherograms of all the samples examined, and the CSF beta TP level could be tentatively determined using allylamine added at a constant concentration as the internal standard.	Allylamine	265-275	prostaglandin D2 synthase	Homo sapiens	66-73
9136084-0	1997	Differential processing of osteopontin characterizes the proliferative vascular smooth muscle cell phenotype induced by allylamine.	Allylamine	120-130	secreted phosphoprotein 1	Homo sapiens	27-38
9136084-12	1997	We conclude that enhanced proteolytic cleavage of OPN may characterize the modulation of vascular SMCs to a more proliferative phenotype following chemical injury by allylamine.	Allylamine	166-176	secreted phosphoprotein 1	Homo sapiens	50-53
9143330-1	1997	The allylamine class of antifungal compounds are specific inhibitors of squalene epoxidase (SE).	Allylamine	4-14	squalene epoxidase	Homo sapiens	72-90
9143330-1	1997	The allylamine class of antifungal compounds are specific inhibitors of squalene epoxidase (SE).	Allylamine	4-14	squalene epoxidase	Homo sapiens	92-94
11667225-1	1996	beta-Nitrogen-functionalized vinylic organolithium compounds derived from secondary aliphatic allylamines have been found to undergo upon heating (reflux of THF) either a dimerization or a regio- and stereoselective cyclodimerization reaction affording diamino 1,4-dienes or cis-2,3-disubstituted 4-methylenepyrrolidines, respectively, according to reaction time.	Allylamine	94-105	suppressor of cytokine signaling 2	Homo sapiens	275-280
12941564-7	2003	We also used the positively charged functional monomer allylamine as functional monomer in order to increase the selectivity of the MIC toward Fru-val.	Allylamine	55-65	zinc finger and BTB domain containing 22	Homo sapiens	143-146
12941564-8	2003	The selectivity of the electrode immobilizing the allylamine-containing polymer showed 1.7-fold higher response toward Fru-val than toward Fru-epsilon-lys.	Allylamine	50-60	zinc finger and BTB domain containing 22	Homo sapiens	119-122
12941564-8	2003	The selectivity of the electrode immobilizing the allylamine-containing polymer showed 1.7-fold higher response toward Fru-val than toward Fru-epsilon-lys.	Allylamine	50-60	zinc finger and BTB domain containing 22	Homo sapiens	139-142
12941564-9	2003	By combining the use of both allylamine as the functional monomer and m-val as the template molecule, an even better MIC-immobilized electrode was produced with a Fru-val selectivity comparable to that constructed by imprinting with Fru-val.	Allylamine	29-39	zinc finger and BTB domain containing 22	Homo sapiens	163-166
12926386-1	2003	alpha-Keto amides 10a,b, formed from reaction of pyruvic or benzoylformic acid with allyl amine are found to present as single rotameric forms whilst their tertiary amido analogues 10c, d present as two rotamers in solution at rt.	Allylamine	84-95	Rho GTPase activating protein 9	Homo sapiens	181-184
9503571-1	1997	The ability of allylamine (AA) administration to produce vascular lesions resembling atherosclerotic disease in animals, has been linked to metabolism of AA to the toxic aldehyde acrolein (ACR) by a semicarbazide-sensitive amine oxidase (SSAO) found in plasma and in vascular smooth muscle.	Allylamine	15-25	amine oxidase copper containing 2	Homo sapiens	199-236
9503571-1	1997	The ability of allylamine (AA) administration to produce vascular lesions resembling atherosclerotic disease in animals, has been linked to metabolism of AA to the toxic aldehyde acrolein (ACR) by a semicarbazide-sensitive amine oxidase (SSAO) found in plasma and in vascular smooth muscle.	Allylamine	15-25	amine oxidase copper containing 2	Homo sapiens	238-242
8920635-9	1996	Vascular SSAO can metabolize the xenobiotic aliphatic amine, allylamine, to the cytotoxic aldehyde acrolein and this has been linked to the ability of allylamine administration to produce cardiovascular lesions in experimental animals, sometimes mimicking features of atherosclerotic disease.	Allylamine	61-71	amine oxidase copper containing 2	Homo sapiens	9-13
8920635-9	1996	Vascular SSAO can metabolize the xenobiotic aliphatic amine, allylamine, to the cytotoxic aldehyde acrolein and this has been linked to the ability of allylamine administration to produce cardiovascular lesions in experimental animals, sometimes mimicking features of atherosclerotic disease.	Allylamine	151-161	amine oxidase copper containing 2	Homo sapiens	9-13
7814369-7	1995	This deduced rat SE sequence is 30.2% identical to the ERG 1 gene, which encodes SE from an allylamine-resistant Saccharomyces cerevisiae mutant.	Allylamine	92-102	potassium voltage-gated channel subfamily H member 2	Rattus norvegicus	55-60
7814369-7	1995	This deduced rat SE sequence is 30.2% identical to the ERG 1 gene, which encodes SE from an allylamine-resistant Saccharomyces cerevisiae mutant.	Allylamine	92-102	squalene epoxidase	Rattus norvegicus	81-83
8430435-2	1993	Previous studies showed that allylamine-induced chronic lesions are markedly reduced by semicarbazide, an inhibitor of semicarbazide-sensitive amine oxidase (SSAO), and that allylamine is metabolized to the aldehyde, acrolein, by SSAO.	Allylamine	29-39	amine oxidase, copper containing 3	Rattus norvegicus	119-156
8584667-5	1995	The possibility that SSAO enzymes can convert amine substrates to highly toxic metabolites is illustrated by the production of acrolein from the xenobiotic amine, allylamine and formaldehyde and methylglyoxal from methylamine and aminoacetone, respectively.	Allylamine	163-173	amine oxidase copper containing 2	Homo sapiens	21-25
7931255-4	1994	Also the xenobiotic aliphatic amine allylamine produces cardiovascular damage in experimental animals by a mechanism which involves its deamination by SSAO to acrolein.	Allylamine	36-46	amine oxidase copper containing 2	Homo sapiens	151-155
8430435-2	1993	Previous studies showed that allylamine-induced chronic lesions are markedly reduced by semicarbazide, an inhibitor of semicarbazide-sensitive amine oxidase (SSAO), and that allylamine is metabolized to the aldehyde, acrolein, by SSAO.	Allylamine	29-39	amine oxidase, copper containing 3	Rattus norvegicus	158-162
8430435-2	1993	Previous studies showed that allylamine-induced chronic lesions are markedly reduced by semicarbazide, an inhibitor of semicarbazide-sensitive amine oxidase (SSAO), and that allylamine is metabolized to the aldehyde, acrolein, by SSAO.	Allylamine	29-39	amine oxidase, copper containing 3	Rattus norvegicus	230-234
8430435-2	1993	Previous studies showed that allylamine-induced chronic lesions are markedly reduced by semicarbazide, an inhibitor of semicarbazide-sensitive amine oxidase (SSAO), and that allylamine is metabolized to the aldehyde, acrolein, by SSAO.	Allylamine	174-184	amine oxidase, copper containing 3	Rattus norvegicus	158-162
8430435-2	1993	Previous studies showed that allylamine-induced chronic lesions are markedly reduced by semicarbazide, an inhibitor of semicarbazide-sensitive amine oxidase (SSAO), and that allylamine is metabolized to the aldehyde, acrolein, by SSAO.	Allylamine	174-184	amine oxidase, copper containing 3	Rattus norvegicus	230-234
8430435-3	1993	We hypothesized that inhibitors of SSAO might reduce the acute cardiovascular toxicity of allylamine.	Allylamine	90-100	amine oxidase, copper containing 3	Rattus norvegicus	35-39
3945966-8	1986	Growth studies of cells surviving an 8-h exposure to allylamine indicate that surviving endothelial cells have better growth characteristics than surviving smooth muscle cells; both cell lines are also apparently injured at concentrations of allylamine much lower than the CT50.	Allylamine	53-63	LEM domain containing 1	Homo sapiens	273-277
1446001-3	1992	In an attempt to exploit the special interactions between this cyclic tertiary allylamine and MAO-B, we have initiated studies to evaluate the enzymatic and biological properties of MPTP analogs bearing functional groups which are known to mediate the metabolism-dependent inactivation of this enzyme.	Allylamine	79-89	monoamine oxidase B	Homo sapiens	94-99
1606650-1	1992	Allylamine (ALAM) film was plasma-polymerized on a flat glass (referred to as ALAM(GLA): GLA refers to a flat glass plate) for use as a solid phase in two-site immunoradiometric assay (two-site IRMA).	Allylamine	0-10	galactosidase alpha	Homo sapiens	83-86
1606650-1	1992	Allylamine (ALAM) film was plasma-polymerized on a flat glass (referred to as ALAM(GLA): GLA refers to a flat glass plate) for use as a solid phase in two-site immunoradiometric assay (two-site IRMA).	Allylamine	0-10	galactosidase alpha	Homo sapiens	89-92
1606650-1	1992	Allylamine (ALAM) film was plasma-polymerized on a flat glass (referred to as ALAM(GLA): GLA refers to a flat glass plate) for use as a solid phase in two-site immunoradiometric assay (two-site IRMA).	Allylamine	12-16	galactosidase alpha	Homo sapiens	83-86
1606650-1	1992	Allylamine (ALAM) film was plasma-polymerized on a flat glass (referred to as ALAM(GLA): GLA refers to a flat glass plate) for use as a solid phase in two-site immunoradiometric assay (two-site IRMA).	Allylamine	12-16	galactosidase alpha	Homo sapiens	89-92
1311944-4	1992	In contrast, allylamines, which have a different mode of action and a weaker ability to bind to cytochrome P-450, are not expected to inhibit clinical drug oxidation.	Allylamine	13-24	cytochrome P450 family 4 subfamily F member 3	Homo sapiens	96-112
1600034-3	1992	Fibrinogen adsorption from a series of dilute plasma solutions to radio-frequency glow discharge/allylamine, measured using 125I radiolabelled baboon fibrinogen, increased with decreasing plasma dilution to a level much higher than that previously observed on polyurethanes.	Allylamine	97-107	fibrinogen beta chain	Homo sapiens	0-10
1600034-3	1992	Fibrinogen adsorption from a series of dilute plasma solutions to radio-frequency glow discharge/allylamine, measured using 125I radiolabelled baboon fibrinogen, increased with decreasing plasma dilution to a level much higher than that previously observed on polyurethanes.	Allylamine	97-107	fibrinogen beta chain	Homo sapiens	150-160
1600034-11	1992	The plasma preadsorption studies with fibrinogen deficient media suggested that adsorbed fibrinogen is necessary for platelet adhesion to the radio-frequency glow discharge/allylamine substrate at high protein coverage.	Allylamine	173-183	fibrinogen beta chain	Homo sapiens	89-99
2104785-2	1990	In this study, we demonstrate that allylamine, a selective cardiovascular toxin in vivo, is actively metabolized in vitro by a purified vascular enzyme (semicarbazide-sensitive amine oxidase), which has been localized recently to vascular smooth muscle cells.	Allylamine	35-45	amine oxidase copper containing 2	Homo sapiens	153-190
2104785-3	1990	Oxidative deamination of allylamine to a highly toxic aldehyde, acrolein, was blocked through enzyme inhibition by semicarbazide-sensitive amine oxidase suggests that this vascular enzyme"s physiological role may include metabolism of exogenous amines.	Allylamine	25-35	amine oxidase copper containing 2	Homo sapiens	115-152
2842890-0	1988	Allylamine-induced vascular toxicity in vitro: prevention by semicarbazide-sensitive amine oxidase inhibitors.	Allylamine	0-10	amine oxidase, copper containing 3	Rattus norvegicus	61-98
2842890-11	1988	Allylamine-induced cytotoxicity was partially prevented by catalase (2500 U/ml).	Allylamine	0-10	catalase	Rattus norvegicus	59-67
31956732-4	2020	This new concept is realized by employing the surface amine groups of plasma polymerized allylamine (PPAm) film for grafting a molecule e.g., thrombin inhibitor, bivalirudin (BVLD), meanwhile its bulk amine groups is used as a universal depot for storing and releasing therapeutic nitric oxide (NO) gas as supplement to the functions of BVLD.	Allylamine	89-99	coagulation factor II, thrombin	Homo sapiens	142-150
2862241-0	1985	An allylamine derivative (MDL 72145) with potent irreversible inhibitory actions on rat aorta semicarbazide-sensitive amine oxidase.	Allylamine	3-13	amine oxidase, copper containing 3	Rattus norvegicus	94-131
1094115-3	1975	DesGly10-LH-RH hydrazide was used as a precursor in the synthesis of desGly10-LH-RH allylamide and desGly10-LH-RH propargylamide by conversion to the azide and reaction with allylamine and propargylamine, respectively.	Allylamine	174-184	gonadotropin releasing hormone 1	Rattus norvegicus	9-14
1094115-3	1975	DesGly10-LH-RH hydrazide was used as a precursor in the synthesis of desGly10-LH-RH allylamide and desGly10-LH-RH propargylamide by conversion to the azide and reaction with allylamine and propargylamine, respectively.	Allylamine	174-184	gonadotropin releasing hormone 1	Rattus norvegicus	78-83
1094115-3	1975	DesGly10-LH-RH hydrazide was used as a precursor in the synthesis of desGly10-LH-RH allylamide and desGly10-LH-RH propargylamide by conversion to the azide and reaction with allylamine and propargylamine, respectively.	Allylamine	174-184	gonadotropin releasing hormone 1	Rattus norvegicus	78-83
33450973-3	2021	The allylamine class of antimycotics targets the enzyme squalene monooxygenase.	Allylamine	4-14	squalene epoxidase	Homo sapiens	56-78
32770418-5	2020	Resistance to terbinafine and other allylamines is very rare and usually correlated with point mutations in the squalene epoxidase gene resulting in single amino acid substitutions in the enzyme, which is crucial in the ergosterol synthesis pathway.	Allylamine	36-47	squalene epoxidase	Homo sapiens	112-130
29575717-7	2018	RESULTS: Treatment with azole-based and allylamine antifungals was associated with improved asthma control (mean change in asthma control 1.72-2.25; p = 0.004), increased PEFR (69.4% predicted to 79.3% predicted, p = 0.0011) and markedly reduced serum IgE levels (1,075 kU/L to 463 kU/L, p = 0.0005) and blood eosinophil counts (Mean absolute count 530-275, p = 0.0095).	Allylamine	40-50	immunoglobulin heavy constant epsilon	Homo sapiens	252-255
29623974-5	2018	Subsequently, for the Pd(0)-catalyzed intermolecular hydroamination of 1,1-diphenyl MCP with 2-pyrrolidone, it is more favorable for the C1 of the metallacyclobutane intermediate to undergo protonation to yield a pi-allylpalladium intermediate, from which the final allylamine product is afforded via reductive elimination.	Allylamine	266-276	capping actin protein, gelsolin like	Homo sapiens	84-87