PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9549041-6	1997	An inhibitory study showed that the presence of cycloheximide (3 micrograms/mL) or actinomycin D (1 microgram/mL) blocked both the stimulatory effect of IGF-II on progesterone accumulation and the amplification of GH on IGF-II induced production (P > 0.01), suggesting GH amplification of IGF-II-induced progesterone accumulation is a process involving gene transcription and translation.	Cycloheximide	48-61	insulin like growth factor 2	Homo sapiens	153-159	
9378983-5	1997	IGF-I and IGF-II (but not basic fibroblast growth factor) were also able to protect cells from apoptosis induced by etoposide or cycloheximide.	Cycloheximide	129-142	insulin like growth factor 2	Homo sapiens	10-16	
9549041-6	1997	An inhibitory study showed that the presence of cycloheximide (3 micrograms/mL) or actinomycin D (1 microgram/mL) blocked both the stimulatory effect of IGF-II on progesterone accumulation and the amplification of GH on IGF-II induced production (P > 0.01), suggesting GH amplification of IGF-II-induced progesterone accumulation is a process involving gene transcription and translation.	Cycloheximide	48-61	insulin like growth factor 2	Homo sapiens	220-226	
9549041-6	1997	An inhibitory study showed that the presence of cycloheximide (3 micrograms/mL) or actinomycin D (1 microgram/mL) blocked both the stimulatory effect of IGF-II on progesterone accumulation and the amplification of GH on IGF-II induced production (P > 0.01), suggesting GH amplification of IGF-II-induced progesterone accumulation is a process involving gene transcription and translation.	Cycloheximide	48-61	insulin like growth factor 2	Homo sapiens	220-226	
2226300-10	1990	IGF-II-stimulated 3 beta HSD activity was completely inhibited by concurrent treatment with either actinomycin D or cycloheximide, suggesting that new mRNA and protein synthesis are required for IGF-II to exert its stimulatory effect.	Cycloheximide	116-129	insulin like growth factor 2	Homo sapiens	0-6	
2226300-10	1990	IGF-II-stimulated 3 beta HSD activity was completely inhibited by concurrent treatment with either actinomycin D or cycloheximide, suggesting that new mRNA and protein synthesis are required for IGF-II to exert its stimulatory effect.	Cycloheximide	116-129	insulin like growth factor 2	Homo sapiens	195-201	
17082775-5	2006	Inhibition of the synthesis of putative trans imprinting factors with cycloheximide led to loss of IGF2 imprinting in normal cultured fibroblasts, suggesting that normal cells produce proteins that act in trans to induce or maintain genomic imprinting.	Cycloheximide	70-83	insulin like growth factor 2	Homo sapiens	99-103	
1693565-4	1990	The release of IGF-II and IGF binding proteins into serum-free conditioned medium (1.7 pmol/10(6) cells.24 h and 0.8 pmol binding sites/10(6) cells.24 h for 3 days, respectively) is inhibited 80% by cycloheximide (10 micrograms/ml).	Cycloheximide	199-212	insulin like growth factor 2	Homo sapiens	15-21