PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
7508936-7	1994	MA-alpha 2M also blocks NGF-promoted incorporation of 32P from [32P]ATP into trk receptors in vitro.	Phosphorus-32	54-57	nerve growth factor	Rattus norvegicus	24-27	
7508936-7	1994	MA-alpha 2M also blocks NGF-promoted incorporation of 32P from [32P]ATP into trk receptors in vitro.	Phosphorus-32	64-67	nerve growth factor	Rattus norvegicus	24-27	
1861152-2	1991	We found that NGF and TPA, alone or in combination, increased (a) the incorporation of [32P]Pi into NF-M and (b) the rate of conversion of NF-M from a poorly phosphorylated to a more highly phosphorylated form.	Phosphorus-32	88-91	nerve growth factor	Rattus norvegicus	14-17	
1319464-5	1992	Incorporation of 32P into ERK1 and ERK2 occurred primarily on tyrosine and threonine residues and was associated with a single tryptic peptide, which is identical to one whose phosphorylation is increased by treatment of intact PC12 cells with NGF.	Phosphorus-32	17-20	nerve growth factor	Rattus norvegicus	244-247	
3106121-11	1987	By immunoprecipitation of vinculin from 32P-labeled cells, vinculin phosphorylation was found to be increased within 1 hr of NGF treatment.	Phosphorus-32	40-43	nerve growth factor	Rattus norvegicus	125-128	
2720800-1	1989	It has been shown that in PC12 and its subclone PC12h treatment of the cells with nerve growth factor (NGF) induces a selective decrease in the incorporation of radioactive phosphate into a 100,000-dalton protein, designated in an earlier study as Nsp100, in the subsequent phosphorylation of soluble extracts from cells with (gamma-32P)ATP.	Phosphorus-32	333-336	nerve growth factor	Rattus norvegicus	82-101	
2720800-1	1989	It has been shown that in PC12 and its subclone PC12h treatment of the cells with nerve growth factor (NGF) induces a selective decrease in the incorporation of radioactive phosphate into a 100,000-dalton protein, designated in an earlier study as Nsp100, in the subsequent phosphorylation of soluble extracts from cells with (gamma-32P)ATP.	Phosphorus-32	333-336	nerve growth factor	Rattus norvegicus	103-106	
2538812-5	1989	NGF stimulates [3H]myristate- or [32P]phosphate-labeled phosphatidic acid production over the same time course.	Phosphorus-32	34-38	nerve growth factor	Rattus norvegicus	0-3	
3372590-6	1988	As measured by incorporation of [32P]phosphate, NGF had a dual effect on MAP1.2.	Phosphorus-32	33-36	nerve growth factor	Rattus norvegicus	48-51	
3804999-7	1987	NGF also increased the stability of the modified form of M. All three subunits were 32P-labeled, and NGF increased the incorporation of 32P into M and H. Neurofilament subunits were also immunoprecipitated from a soluble fraction of [35S]methionine-labeled PC12 cells.	Phosphorus-32	84-87	nerve growth factor	Rattus norvegicus	0-3	
3804999-7	1987	NGF also increased the stability of the modified form of M. All three subunits were 32P-labeled, and NGF increased the incorporation of 32P into M and H. Neurofilament subunits were also immunoprecipitated from a soluble fraction of [35S]methionine-labeled PC12 cells.	Phosphorus-32	136-139	nerve growth factor	Rattus norvegicus	0-3	
3804999-7	1987	NGF also increased the stability of the modified form of M. All three subunits were 32P-labeled, and NGF increased the incorporation of 32P into M and H. Neurofilament subunits were also immunoprecipitated from a soluble fraction of [35S]methionine-labeled PC12 cells.	Phosphorus-32	136-139	nerve growth factor	Rattus norvegicus	101-104	
3772374-3	1986	Half-maximal incorporation of 32P from [32P]ATP into S6 occurred after 4-7 min of NGF treatment.	Phosphorus-32	30-33	nerve growth factor	Rattus norvegicus	82-85	
3772374-3	1986	Half-maximal incorporation of 32P from [32P]ATP into S6 occurred after 4-7 min of NGF treatment.	Phosphorus-32	40-43	nerve growth factor	Rattus norvegicus	82-85	
3948029-2	1986	Here we show, using dot hybridization of mRNA with a 32P-labelled cDNA probe for beta-nerve growth factor (beta NGF), that mRNA sequences homologous to beta NGF appear in muscle in parallel with increased motoneurone survival activity.	Phosphorus-32	53-56	nerve growth factor	Rattus norvegicus	81-105	
3948029-2	1986	Here we show, using dot hybridization of mRNA with a 32P-labelled cDNA probe for beta-nerve growth factor (beta NGF), that mRNA sequences homologous to beta NGF appear in muscle in parallel with increased motoneurone survival activity.	Phosphorus-32	53-56	nerve growth factor	Rattus norvegicus	107-115	
3948029-2	1986	Here we show, using dot hybridization of mRNA with a 32P-labelled cDNA probe for beta-nerve growth factor (beta NGF), that mRNA sequences homologous to beta NGF appear in muscle in parallel with increased motoneurone survival activity.	Phosphorus-32	53-56	nerve growth factor	Rattus norvegicus	152-160	
15372494-6	2004	In response to ATP, NGF, or NGF + ATP, MAPK activity (measured by 32P incorporation) was maximal within 2 hr and remained statistically elevated over control levels throughout the 24 hr monitored.	Phosphorus-32	66-69	nerve growth factor	Rattus norvegicus	20-23	
15372494-6	2004	In response to ATP, NGF, or NGF + ATP, MAPK activity (measured by 32P incorporation) was maximal within 2 hr and remained statistically elevated over control levels throughout the 24 hr monitored.	Phosphorus-32	66-69	nerve growth factor	Rattus norvegicus	28-31	
15372494-7	2004	At maximal 32P incorporation, MAPK activity in response to ATP, NGF, and NGF + ATP was two-, four-, and sixfold higher, respectively, than control values; the observed increase was qualitatively confirmed using Western blots.	Phosphorus-32	11-14	nerve growth factor	Rattus norvegicus	64-67	
15372494-7	2004	At maximal 32P incorporation, MAPK activity in response to ATP, NGF, and NGF + ATP was two-, four-, and sixfold higher, respectively, than control values; the observed increase was qualitatively confirmed using Western blots.	Phosphorus-32	11-14	nerve growth factor	Rattus norvegicus	73-76