PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32213513-3	2020	Streptozotocin-induced diabetic rats and mice were orally treated with PPARalpha agonist fenofibrate.	Streptozocin	0-14	peroxisome proliferator activated receptor alpha	Mus musculus	71-80	
33214136-7	2021	Peripheral ischemic recovery and PPARalpha signaling was further compromised by Streptozotocin-induced diabetes, and ameliorated by feeding fenofibrate.	Streptozocin	80-94	peroxisome proliferator activated receptor alpha	Mus musculus	33-42	
22084061-0	2012	Red clover extract ameliorates dyslipidemia in streptozotocin-induced diabetic C57BL/6 mice by activating hepatic PPARalpha.	Streptozocin	47-61	peroxisome proliferator activated receptor alpha	Mus musculus	114-123	
30129056-12	2018	CONCLUSIONS AND IMPLICATIONS: Nuciferine improved lipid profile and attenuated hepatic steatosis in HFD/STZ-induced diabetic mice by activating the PPARalpha/PGC1alpha pathway.	Streptozocin	104-107	peroxisome proliferator activated receptor alpha	Mus musculus	148-157	
28271032-1	2017	OBJECTIVE: Recently, we have shown that Bezafibrate (BEZ), the pan-PPAR (peroxisome proliferator-activated receptor) activator, ameliorated diabetes in insulin deficient streptozotocin treated diabetic mice.	Streptozocin	170-184	peroxisome proliferator activated receptor alpha	Mus musculus	67-71	
22084061-8	2012	Thus we conclude that red clover extract and biochanin A or formononetin significantly ameliorate the lipid profiles of STZ-diabetic mice and these effects are achieved at least in part by activating hepatic PPARalpha.	Streptozocin	120-123	peroxisome proliferator activated receptor alpha	Mus musculus	208-217	
22110479-1	2012	We previously demonstrated in streptozotocin-induced diabetic mice that deficiency or inhibition of aldose reductase (AR) caused significant dephosphorylation of hepatic transcriptional factor PPARalpha, leading to its activation and significant reductions in serum lipid levels.	Streptozocin	30-44	peroxisome proliferator activated receptor alpha	Mus musculus	193-202	
17897018-7	2007	Here, we will review the anti-atherosclerotic potential of PPAR agonists with particular emphasis on recent studies in an animal model of diabetes-associated atherosclerosis, the streptozotocin diabetic apolipoprotein E deficient mouse.	Streptozocin	179-193	peroxisome proliferator activated receptor alpha	Mus musculus	59-63	
12958197-0	2003	Activation of PPAR-alpha in streptozotocin-induced diabetes is essential for resistance against acetaminophen toxicity.	Streptozocin	28-42	peroxisome proliferator activated receptor alpha	Mus musculus	14-24	
11259402-7	2001	However, when fatty acid (the natural ligand for PPARalpha) supply was increased (high-fat feeding, fasting, and STZ-induced diabetes), cardiac UCP-3 but not UCP-2 expression increased.	Streptozocin	113-116	peroxisome proliferator activated receptor alpha	Mus musculus	49-58