PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16847153-3	2006	METHODS AND RESULTS: BMC- and CD34+-derived progenitor cells interacted with ischemic femoral arteries through SDF-1 and CXCR4 signaling and released nitric oxide (NO) via an endothelial nitric oxide synthase (eNOS)-dependent pathway.	bmc	21-24	C-X-C motif chemokine ligand 12	Homo sapiens	111-116	
24569031-8	2014	In contrast, in the BMC group, only the basal (P = 0.02) and the stromal cell-derived factor-1-induced (P = 0.05) migratory capacity of the administered BMC were associated with improved clinical outcome.	bmc	20-23	C-X-C motif chemokine ligand 12	Homo sapiens	65-94	
24569031-8	2014	In contrast, in the BMC group, only the basal (P = 0.02) and the stromal cell-derived factor-1-induced (P = 0.05) migratory capacity of the administered BMC were associated with improved clinical outcome.	bmc	153-156	C-X-C motif chemokine ligand 12	Homo sapiens	65-94	
22129485-7	2012	Extensive studies have demonstrated that the SDF-1/CXCR4 axis is a key regulator for BMC mobilisation.	bmc	85-88	C-X-C motif chemokine ligand 12	Homo sapiens	45-50	
22129485-8	2012	Moreover, abrogation of the SDF-1/CXCR4 axis by proteolytic modification can efficiently increase BMC mobilisation.	bmc	98-101	C-X-C motif chemokine ligand 12	Homo sapiens	28-33