PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26497474-5	2016	Here, we show that newly synthesized precursor sterols arriving at the plasma membrane (PM) are removed by extracellular apoA-I in a manner dependent on ABCA1, a key macromolecule for HDL biogenesis.	Sterols	47-54	apolipoprotein A1	Homo sapiens	121-127	
21215605-7	2012	Intake of rye bread enriched with 2g/d of plant sterols during two weeks reduced significantly serum total and LDL cholesterol, apoB/apoA1 and total cholesterol/HDL cholesterol ratios by 5.1%, 8.1%, 8.3% and 7.2%, respectively, compared to controls.	Sterols	48-55	apolipoprotein A1	Homo sapiens	133-138	
25329888-5	2014	ApoA-I-mediated cholesterol efflux was examined using the fluorescent sterol (BODIPY-cholesterol).	Sterols	21-27	apolipoprotein A1	Homo sapiens	0-6	
23826352-6	2013	Further analysis showed that rHDL containing the carboxyl-terminal deletion mutant apoA-I[Delta(185-243)] only slightly reduced (by 22%) the ABCG1-mediated efflux of 7-ketocholesterol, indicating that depending on the sterol type, structural changes in rHDL-associated apoA-I affect differently the ABCG1-mediated efflux of cholesterol and 7-ketocholesterol.	Sterols	177-183	apolipoprotein A1	Homo sapiens	83-89	
11939788-0	2002	Sterol efflux to apolipoprotein A-I originates from caveolin-rich microdomains and potentiates PDGF-dependent protein kinase activity.	Sterols	0-6	apolipoprotein A1	Homo sapiens	17-35	
12202492-13	2002	In conclusion, the apoA-I/ABCA1- and HDL/SR-BI-dependent pathways modulate polarized sterol mobilization at the BBB.	Sterols	85-91	apolipoprotein A1	Homo sapiens	19-25	
19145455-6	2009	Plant sterol intake lowered TC, LDL cholesterol, and Apo B by 9.0, 12.4 and 6.1% and TC/HDLC by 9.6% (P <or= 0.0001 for all), respectively, without affecting HDL cholesterol and Apo A1 (P = 0.2831 and 0.732).	Sterols	6-12	apolipoprotein A1	Homo sapiens	181-187	
18066136-0	2007	Modulation of apolipoprotein A1 and B, adiponectin, ghrelin, and growth hormone concentrations by plant sterols and exercise in previously sedentary humans.	Sterols	104-111	apolipoprotein A1	Homo sapiens	14-37	
7132726-5	1982	In normal plasma the major component of efflux of sterol radioactivity from labeled fibroblasts was dependent upon unassociated apo A-I.	Sterols	50-56	apolipoprotein A1	Homo sapiens	128-135	
10882340-3	2000	This implies that lipidation of apolipoprotein A-I by the ABCA1 pathway is required for generating HDL particles and clearing sterol from macrophages.	Sterols	126-132	apolipoprotein A1	Homo sapiens	32-50	
9017501-7	1996	Thus, an inability of nascent apoA-I to acquire cellular lipids results in a rapid clearance of apoA-I from the plasma, decreased production and increased clearance of LDL, and sterol deposition in tissue macrophages.	Sterols	177-183	apolipoprotein A1	Homo sapiens	30-36	
11349133-1	2001	ABCA1, the ATP-binding cassette protein mutated in Tangier disease, mediates the efflux of excess cellular sterol to apoA-I and thereby the formation of high density lipoprotein.	Sterols	107-113	apolipoprotein A1	Homo sapiens	117-123	
9922151-5	1998	We show that binding of high-density lipoprotein and apolipoprotein A-I to the brush border membrane-resident receptor inhibits uptake of cholesterol (sterol) into the brush border membrane from lipid donor particles.	Sterols	143-149	apolipoprotein A1	Homo sapiens	53-71	
6943589-2	1981	Sterol efflux was highly (approximately 80%) dependent upon a minor lipoprotein fraction containing apolipoprotein A-I unassociated with other apolipoproteins.	Sterols	0-6	apolipoprotein A1	Homo sapiens	100-118	
28205180-8	2017	Here, we describe several methods at the cell culture level to monitor ABCA1-dependent release of sterol molecules to apoA-I present at the cell exterior.	Sterols	98-104	apolipoprotein A1	Homo sapiens	118-124