PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16266291-9	2005	Based on the proposed mechanism for TTR amyloid fibril formation we discuss the action of some of the proposed TTR stabilizers such as derivatives of some NSAIDs (diflunisal, diclofenac, flufenamic acid, and derivatives) and the action of amyloid disrupters such as 4"-iodo-4"-deoxydoxorubicin (I-DOX) and tetracyclines.	Tetracyclines	306-319	transthyretin	Homo sapiens	111-114	
12724338-7	2003	The species generated upon I-DOX and tetracyclines treatments were nontoxic, as revealed by the lack of significant caspase-3 activation on a Schwannoma cell line, making them potential therapeutic drugs in TTR-related and other amyloidosis.	Tetracyclines	37-50	transthyretin	Homo sapiens	207-210