PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 9514844-7 1998 The GH-releasing effect of DA was mimicked by the D1 agonists SKF38393 (0.1-10 muM) and SKF77434 (0.1-10 muM), but not by the D2 agonist LY171555 (3 muM). Dopamine 27-29 latexin Homo sapiens 79-82 21662891-4 1999 The response for dopamine was linear from 20 to 200 muM, with a LOD of 1.0 muM and a sensitivity of 52 pA/muM. Dopamine 17-25 latexin Homo sapiens 52-55 21662891-4 1999 The response for dopamine was linear from 20 to 200 muM, with a LOD of 1.0 muM and a sensitivity of 52 pA/muM. Dopamine 17-25 latexin Homo sapiens 75-78 21662891-4 1999 The response for dopamine was linear from 20 to 200 muM, with a LOD of 1.0 muM and a sensitivity of 52 pA/muM. Dopamine 17-25 latexin Homo sapiens 75-78 9514844-8 1998 In addition, the GH response to DA (1 muM) was blocked by the D1 antagonist SCH23390 (5 muM) but not by the D2 antagonist (+/-) sulpiride (5 muM), suggesting that the GH-releasing action of DA is mediated through receptors resembling mammalian D1 receptors. Dopamine 32-34 latexin Homo sapiens 38-41 9514844-8 1998 In addition, the GH response to DA (1 muM) was blocked by the D1 antagonist SCH23390 (5 muM) but not by the D2 antagonist (+/-) sulpiride (5 muM), suggesting that the GH-releasing action of DA is mediated through receptors resembling mammalian D1 receptors. Dopamine 32-34 latexin Homo sapiens 88-91 9514844-8 1998 In addition, the GH response to DA (1 muM) was blocked by the D1 antagonist SCH23390 (5 muM) but not by the D2 antagonist (+/-) sulpiride (5 muM), suggesting that the GH-releasing action of DA is mediated through receptors resembling mammalian D1 receptors. Dopamine 32-34 latexin Homo sapiens 88-91 31183562-5 2019 The voltammetric response to DA is linear in the 2.0 muM to 160 muM concentration range even in the presence of 1.0 mM ascorbic acid and 0.1 mM of UA. Dopamine 29-31 latexin Homo sapiens 53-56 8748684-5 1995 Conversely, at concentrations above 100 muM, salsolinol inhibited the uptake of noradrenaline and dopamine, with IC50 values of 411 muM and 379 muM, respectively. Dopamine 98-106 latexin Homo sapiens 132-135 8748684-5 1995 Conversely, at concentrations above 100 muM, salsolinol inhibited the uptake of noradrenaline and dopamine, with IC50 values of 411 muM and 379 muM, respectively. Dopamine 98-106 latexin Homo sapiens 132-135 32253397-5 2020 Under the optimal conditions, the DA sensor shows a linear response in the DA concentration ranges of 0.1 muM-0.08 mM and 0.08-0.41 mM with a low detection limit of 0.027 muM and a high sensitivity of 602.4 muA mM-1 cm-2. Dopamine 34-36 latexin Homo sapiens 106-109 32253397-5 2020 Under the optimal conditions, the DA sensor shows a linear response in the DA concentration ranges of 0.1 muM-0.08 mM and 0.08-0.41 mM with a low detection limit of 0.027 muM and a high sensitivity of 602.4 muA mM-1 cm-2. Dopamine 34-36 latexin Homo sapiens 171-174 32253397-5 2020 Under the optimal conditions, the DA sensor shows a linear response in the DA concentration ranges of 0.1 muM-0.08 mM and 0.08-0.41 mM with a low detection limit of 0.027 muM and a high sensitivity of 602.4 muA mM-1 cm-2. Dopamine 75-77 latexin Homo sapiens 106-109 32253397-5 2020 Under the optimal conditions, the DA sensor shows a linear response in the DA concentration ranges of 0.1 muM-0.08 mM and 0.08-0.41 mM with a low detection limit of 0.027 muM and a high sensitivity of 602.4 muA mM-1 cm-2. Dopamine 75-77 latexin Homo sapiens 171-174 32043995-7 2020 The detection limits of noradrenaline, adrenaline, and dopamine were 0.1, 0.1, and 0.2 muM, respectively. Dopamine 55-63 latexin Homo sapiens 87-90 31594597-6 2020 Acceptable linear response of AA, DA, UA and AC respectively have been ranged 0.01-900.0 muM, 0.01-700.0 muM, 0.01-900.0 muM, and 0.01-900.0 muM with determination limits (S/N = 3) of 3.1 nM, 2.6 nM, 2.4 nM and 4.4 nM. Dopamine 34-36 latexin Homo sapiens 89-92 31594597-6 2020 Acceptable linear response of AA, DA, UA and AC respectively have been ranged 0.01-900.0 muM, 0.01-700.0 muM, 0.01-900.0 muM, and 0.01-900.0 muM with determination limits (S/N = 3) of 3.1 nM, 2.6 nM, 2.4 nM and 4.4 nM. Dopamine 34-36 latexin Homo sapiens 105-108 31594597-6 2020 Acceptable linear response of AA, DA, UA and AC respectively have been ranged 0.01-900.0 muM, 0.01-700.0 muM, 0.01-900.0 muM, and 0.01-900.0 muM with determination limits (S/N = 3) of 3.1 nM, 2.6 nM, 2.4 nM and 4.4 nM. Dopamine 34-36 latexin Homo sapiens 105-108 31594597-6 2020 Acceptable linear response of AA, DA, UA and AC respectively have been ranged 0.01-900.0 muM, 0.01-700.0 muM, 0.01-900.0 muM, and 0.01-900.0 muM with determination limits (S/N = 3) of 3.1 nM, 2.6 nM, 2.4 nM and 4.4 nM. Dopamine 34-36 latexin Homo sapiens 105-108 31009905-6 2019 The amperometric detection of dopamine sensor, using this sensing element, exhibits a wide linear response of 0-0.8 muM with a low detection limit of 2.7 nM. Dopamine 30-38 latexin Homo sapiens 116-119 1165793-4 1975 Chlorpromazine, 1 muM, or pimozide, 1 muM, antagonized selectively the reduction in 3H-noradrenaline release obtained with dopamine or apomorphine, without affecting the inhibition obtained with (-)-noradrenaline. Dopamine 123-131 latexin Homo sapiens 18-21 1165793-4 1975 Chlorpromazine, 1 muM, or pimozide, 1 muM, antagonized selectively the reduction in 3H-noradrenaline release obtained with dopamine or apomorphine, without affecting the inhibition obtained with (-)-noradrenaline. Dopamine 123-131 latexin Homo sapiens 38-41 1165793-6 1975 Phenoxybenzamine, 0.29 muM, prevented the inhibition of 3H-transmitter overflow obtained with (-)-noradrenaline, dopamine or apomorphine. Dopamine 113-121 latexin Homo sapiens 23-26 31060035-5 2019 At optimized conditions, the calibration curve was linear in the concentration range of 0.81-16.80 muM of dopamine. Dopamine 106-114 latexin Homo sapiens 99-102 31183562-5 2019 The voltammetric response to DA is linear in the 2.0 muM to 160 muM concentration range even in the presence of 1.0 mM ascorbic acid and 0.1 mM of UA. Dopamine 29-31 latexin Homo sapiens 64-67 30999216-8 2019 Bath application of dopamine (1 muM), or the phosphatidylinositol (PI)-linked D1-like-receptor agonist SKF83959 (5 muM), induced reliable and reversible increases in fluo-4 fluorescence and excitatory postsynaptic currents in fan cells, but not in pyramidal cells. Dopamine 20-28 latexin Homo sapiens 32-35 30999216-8 2019 Bath application of dopamine (1 muM), or the phosphatidylinositol (PI)-linked D1-like-receptor agonist SKF83959 (5 muM), induced reliable and reversible increases in fluo-4 fluorescence and excitatory postsynaptic currents in fan cells, but not in pyramidal cells. Dopamine 20-28 latexin Homo sapiens 115-118 30999216-10 2019 Blocking release of calcium from internal stores by loading cells with the IP3 receptor blocker heparin (1 mM) or the ryanodine receptor blocker dantrolene (20 muM) abolished both the calcium transients and the facilitation of evoked synaptic currents induced by dopamine. Dopamine 263-271 latexin Homo sapiens 160-163 30782366-4 2019 The electrochemical investigation showed that the sensor performed good selectivity and wide linear ranges for the simultaneous detection of dopamine (0.5-150 muM) and uric acid (0.5-600 muM). Dopamine 141-149 latexin Homo sapiens 159-162 30782366-4 2019 The electrochemical investigation showed that the sensor performed good selectivity and wide linear ranges for the simultaneous detection of dopamine (0.5-150 muM) and uric acid (0.5-600 muM). Dopamine 141-149 latexin Homo sapiens 187-190 30218923-4 2018 The PtNPs@MnO2 nanocomposite was employed in a sensing assay for the determination of glutathione (GSH) and dopamine (DA) with a linear range of 0.2 muM-11 muM for GSH and 0.1 muM-1.1 muM for DA. Dopamine 108-116 latexin Homo sapiens 149-152 31049714-9 2019 The electrode respondsy to dopamine in the ranges of 0.15 to 45 muM, the detection limit is 8.2 nM (S/N = 3), and the sensitivity is 20.81 muA muM-1 cm-2. Dopamine 27-35 latexin Homo sapiens 64-67 30391428-4 2019 The results showed that bioluminescence emission of aequorin was effectively quenched by increasing concentration of dopamine at the range of 1 nM to 100 muM with a detection limit of 53 nM. Dopamine 117-125 latexin Homo sapiens 154-157 30597937-8 2018 The presence of ascorbic acid increased the sensitivity of dopamine determination at the modified electrode, and the detection limit was estimated to be 0.5 muM with 0.1 mM ascorbic acid to imitate physiological solutions. Dopamine 59-67 latexin Homo sapiens 157-160 30218923-4 2018 The PtNPs@MnO2 nanocomposite was employed in a sensing assay for the determination of glutathione (GSH) and dopamine (DA) with a linear range of 0.2 muM-11 muM for GSH and 0.1 muM-1.1 muM for DA. Dopamine 118-120 latexin Homo sapiens 149-152 29958556-5 2018 Considering dopamine detection in real biological fluid samples, the NUNCD electrode performed excellently with a detection limit of 0.39 muM and a high recovery ranging from 90-120%, revealing that NUNCD electrodes have promising use in the sensing of dopamine. Dopamine 253-261 latexin Homo sapiens 138-141 32528220-6 2018 Using cyclic voltammetry, the 100-nm "thin" film microelectrode produced the most favorable combination of DA sensitivity value of 36 +-2% muA/muM/cm2 with a linear range of 33 nM to 1 muM and a limit of detection (LOD) of 9.5 +- 1.2% nM. Dopamine 107-109 latexin Homo sapiens 143-146 29753967-6 2018 By taking advantage of this fact, a sensitive probe was designed for determination of dopamine, adrenaline and noradrenaline with a limit of detection of 0.07, 0.60 and 0.01 muM, respectively. Dopamine 86-94 latexin Homo sapiens 174-177 29891587-6 2018 Results indicate that solriamfetol has dual reuptake inhibition activity at dopamine (DA; IC50 = 2.9 muM) and norepinephrine (NE; IC50 = 4.4 muM) transporters, and this activity is associated in vivo with increased extracellular concentration of DA and NE as measured by microdialysis. Dopamine 86-88 latexin Homo sapiens 101-104 32254405-6 2018 The linear ranges are 0.001-1.5 mM, 0.05-160 muM and 0.001-0.875 mM for AA, DA and UA, and the corresponding detection limits are 0.3 muM, 20 nM and 0.3 muM, respectively. Dopamine 76-78 latexin Homo sapiens 134-137 32254405-6 2018 The linear ranges are 0.001-1.5 mM, 0.05-160 muM and 0.001-0.875 mM for AA, DA and UA, and the corresponding detection limits are 0.3 muM, 20 nM and 0.3 muM, respectively. Dopamine 76-78 latexin Homo sapiens 134-137 29704250-9 2018 Using this system with a gold microelectrode, dopamine, and epinephrine could be quantified within the concentration range of 1-500 muM and detected at a concentration of 0.3 muM. Dopamine 46-54 latexin Homo sapiens 132-135 29704250-9 2018 Using this system with a gold microelectrode, dopamine, and epinephrine could be quantified within the concentration range of 1-500 muM and detected at a concentration of 0.3 muM. Dopamine 46-54 latexin Homo sapiens 175-178 29958556-4 2018 Furthermore, the marked selectivity of the NUNCD electrode is very favorable for the determination of dopamine (DA) concentration (0.32 muM) in the presence of ascorbic acid (AA) and uric acid (UA). Dopamine 102-110 latexin Homo sapiens 136-139 29958556-4 2018 Furthermore, the marked selectivity of the NUNCD electrode is very favorable for the determination of dopamine (DA) concentration (0.32 muM) in the presence of ascorbic acid (AA) and uric acid (UA). Dopamine 112-114 latexin Homo sapiens 136-139 29958556-5 2018 Considering dopamine detection in real biological fluid samples, the NUNCD electrode performed excellently with a detection limit of 0.39 muM and a high recovery ranging from 90-120%, revealing that NUNCD electrodes have promising use in the sensing of dopamine. Dopamine 12-20 latexin Homo sapiens 138-141 29537707-5 2018 We found that blockade of D1/D5 receptors by SCH 23390 (20 muM) significantly reduced the magnitude of LTP in both DH and VH similarly suggesting that dopamine endogenously released during TBS, presumably mimicking low activity of DA neurons, exerts a homogeneous modulation of LTP along the hippocampal long axis. Dopamine 151-159 latexin Homo sapiens 59-62 29442624-5 2018 Linear analytical curves were obtained in the range from 2 to 120 muM for dopamine by differential pulse voltammetry, with the detection limit of 0.13 muM (signal-to-noise ratio of 3). Dopamine 74-82 latexin Homo sapiens 66-69 29442624-5 2018 Linear analytical curves were obtained in the range from 2 to 120 muM for dopamine by differential pulse voltammetry, with the detection limit of 0.13 muM (signal-to-noise ratio of 3). Dopamine 74-82 latexin Homo sapiens 151-154 32528220-6 2018 Using cyclic voltammetry, the 100-nm "thin" film microelectrode produced the most favorable combination of DA sensitivity value of 36 +-2% muA/muM/cm2 with a linear range of 33 nM to 1 muM and a limit of detection (LOD) of 9.5 +- 1.2% nM. Dopamine 107-109 latexin Homo sapiens 185-188 29226668-10 2017 However, it is still able to detect DA down to 1 pM range in the presence of [AA] = 100 muM and 100 pM in the presence of [UA] = 3 muM, these values for AA and UA being the physiological levels in the cerebrospinal fluid and the striatum, respectively. Dopamine 36-38 latexin Homo sapiens 88-91 29400087-4 2018 RESULTS: Heterocumulenes derived from dopamine and beta-O-methylnoradrenaline were strong antiproliferative agents (GI50<10 muM). Dopamine 38-46 latexin Homo sapiens 127-130 28960783-6 2018 Dopamine in the range 20-100 muM can be linearly detected by the fluorescence quenching ratio of gold nanoclusters. Dopamine 0-8 latexin Homo sapiens 29-32 29156235-5 2018 The linear detection range for dopamine is 2.5-1500 muM with detection limit of 1.5 muM. Dopamine 31-39 latexin Homo sapiens 52-55 29156235-5 2018 The linear detection range for dopamine is 2.5-1500 muM with detection limit of 1.5 muM. Dopamine 31-39 latexin Homo sapiens 84-87 29594573-5 2018 The peak current (best measured at a working voltage of 0.17 V vs. Ag/AgCl) increases linearly in the 0.1 to 400 muM DA concentration range, with a 40 nM detection limit (at S/N = 3). Dopamine 117-119 latexin Homo sapiens 113-116 29594573-9 2018 The determination of the DPV curves provides satisfactory results which can be characterized by a linear response for DA concentrations ranging from 0.1 to 400.0 muM with a 40 nM detection limit (at S/N = 3). Dopamine 118-120 latexin Homo sapiens 162-165 29226668-10 2017 However, it is still able to detect DA down to 1 pM range in the presence of [AA] = 100 muM and 100 pM in the presence of [UA] = 3 muM, these values for AA and UA being the physiological levels in the cerebrospinal fluid and the striatum, respectively. Dopamine 36-38 latexin Homo sapiens 131-134 28714693-5 2017 We find that in vivo imaging of neuromodulation requires simultaneous optimization of dopamine nanosensor reversibility and sensitivity: dopamine imaging in the striatum or nucleus accumbens requires nanosensors with an optimal dopamine dissociation constant (Kd) of 1 muM, whereas Kds above 10 muM are required for dopamine imaging in the prefrontal cortex. Dopamine 137-145 latexin Homo sapiens 269-272 29027462-3 2017 The proposed system responses specifically to dopamine with a detection limit of 1.43 muM. Dopamine 46-54 latexin Homo sapiens 86-89 28714693-5 2017 We find that in vivo imaging of neuromodulation requires simultaneous optimization of dopamine nanosensor reversibility and sensitivity: dopamine imaging in the striatum or nucleus accumbens requires nanosensors with an optimal dopamine dissociation constant (Kd) of 1 muM, whereas Kds above 10 muM are required for dopamine imaging in the prefrontal cortex. Dopamine 137-145 latexin Homo sapiens 295-298 28714693-5 2017 We find that in vivo imaging of neuromodulation requires simultaneous optimization of dopamine nanosensor reversibility and sensitivity: dopamine imaging in the striatum or nucleus accumbens requires nanosensors with an optimal dopamine dissociation constant (Kd) of 1 muM, whereas Kds above 10 muM are required for dopamine imaging in the prefrontal cortex. Dopamine 137-145 latexin Homo sapiens 269-272 28714693-5 2017 We find that in vivo imaging of neuromodulation requires simultaneous optimization of dopamine nanosensor reversibility and sensitivity: dopamine imaging in the striatum or nucleus accumbens requires nanosensors with an optimal dopamine dissociation constant (Kd) of 1 muM, whereas Kds above 10 muM are required for dopamine imaging in the prefrontal cortex. Dopamine 137-145 latexin Homo sapiens 295-298 28714693-5 2017 We find that in vivo imaging of neuromodulation requires simultaneous optimization of dopamine nanosensor reversibility and sensitivity: dopamine imaging in the striatum or nucleus accumbens requires nanosensors with an optimal dopamine dissociation constant (Kd) of 1 muM, whereas Kds above 10 muM are required for dopamine imaging in the prefrontal cortex. Dopamine 137-145 latexin Homo sapiens 269-272 28714693-5 2017 We find that in vivo imaging of neuromodulation requires simultaneous optimization of dopamine nanosensor reversibility and sensitivity: dopamine imaging in the striatum or nucleus accumbens requires nanosensors with an optimal dopamine dissociation constant (Kd) of 1 muM, whereas Kds above 10 muM are required for dopamine imaging in the prefrontal cortex. Dopamine 137-145 latexin Homo sapiens 295-298 28752884-4 2017 Amperometric dopamine biosensors based on dendritic Pt3Ni nanoalloy microelectrode exhibit a wide linear detection ranges from 0.5 muM to 250 muM with ultrahigh sensitivity, fast response, and excellent selectivity at a potential of 0.3 V in a 0.1 M phosphate buffered solution (pH = 7.2). Dopamine 13-21 latexin Homo sapiens 131-134 28752884-4 2017 Amperometric dopamine biosensors based on dendritic Pt3Ni nanoalloy microelectrode exhibit a wide linear detection ranges from 0.5 muM to 250 muM with ultrahigh sensitivity, fast response, and excellent selectivity at a potential of 0.3 V in a 0.1 M phosphate buffered solution (pH = 7.2). Dopamine 13-21 latexin Homo sapiens 142-145 32264558-7 2017 The relative intensity of restored fluorescence is proportional to the concentration of DA in the wide linear range of 0.25-50 muM with a low detection limit of 57 nM (S/N = 3). Dopamine 88-90 latexin Homo sapiens 127-130 28482573-6 2017 The composite of DAAO-Hb/MnO2 NPs/PTh to construct a bi-enzyme biosensor in this study showed a linear response with DA in the concentration range of 0.04-9.0muM with R-squared value of 0.994 (for S/N=3) and its sensitivity and detection limite were about 12.801muA/muM and 41nM respectively. Dopamine 17-19 latexin Homo sapiens 158-161 27770936-4 2017 Under optimum conditions with differential pulse voltammetry method, a broad linear response versus the concentrations of DA and AC has been observed in the ranges of 2.0 to 250.0muM and 5.0 to 300.0muM, respectively. Dopamine 122-124 latexin Homo sapiens 179-182 26530000-5 2015 This near-infrared quantum dot based ECL sensor displayed a linear response over the range 3.7 <= [dopamine] <= 450 muM, allowing the rapid detection of dopamine and providing a platform for future development. Dopamine 102-110 latexin Homo sapiens 122-125 27420730-3 2016 This treatment did suppress the detection of ascorbic acid oxidation signal, but only in a manner suitable for single-use detection of high concentrations of dopamine (i.e., > 1 muM). Dopamine 158-166 latexin Homo sapiens 181-184 27155116-9 2016 The DA concentration changed from 42.8 to 481.6muM when the MEA probe inserted from cortex into deep brain nucleus of striatum, which reflected the inhomogeneous distribution of DA in brains. Dopamine 4-6 latexin Homo sapiens 47-50 27455760-6 2016 These surface modified pin type electrode was applied to detect low concentration of DA and successfully detect various concentration of DA from 100 muM to 1 muM with linear relationship in the presence of ascorbic acid and uric acid. Dopamine 85-87 latexin Homo sapiens 149-152 27455760-6 2016 These surface modified pin type electrode was applied to detect low concentration of DA and successfully detect various concentration of DA from 100 muM to 1 muM with linear relationship in the presence of ascorbic acid and uric acid. Dopamine 85-87 latexin Homo sapiens 158-161 27455760-6 2016 These surface modified pin type electrode was applied to detect low concentration of DA and successfully detect various concentration of DA from 100 muM to 1 muM with linear relationship in the presence of ascorbic acid and uric acid. Dopamine 137-139 latexin Homo sapiens 149-152 27455760-6 2016 These surface modified pin type electrode was applied to detect low concentration of DA and successfully detect various concentration of DA from 100 muM to 1 muM with linear relationship in the presence of ascorbic acid and uric acid. Dopamine 137-139 latexin Homo sapiens 158-161 26530000-5 2015 This near-infrared quantum dot based ECL sensor displayed a linear response over the range 3.7 <= [dopamine] <= 450 muM, allowing the rapid detection of dopamine and providing a platform for future development. Dopamine 159-167 latexin Homo sapiens 122-125 25756085-4 2015 For simultaneous detection of AA, DA, and UA, the linear current-concentration responses were observed from 1.0 muM-4.1 mM, 0.05-112.0 muM, and 3.0-186.0 muM, with the detection limits of 0.185, 0.017, and 0.654 muM (S/N = 3), respectively. Dopamine 34-36 latexin Homo sapiens 112-115 26249610-3 2015 The resulting sensor exhibited a good response to hydrogen peroxide over linear range from 0.2 to 60.0muM with a limit of detection of 0.08muM, good reproducibility, long-term stability and negligible interference from ascorbic acid, uric acid and dopamine. Dopamine 248-256 latexin Homo sapiens 102-105 32262771-3 2015 The oxidation peak current of DA is linearly proportional to its concentration in the range from 1.5-215.56 muM, with a detection limit of 0.1 muM (at S/N = 3). Dopamine 30-32 latexin Homo sapiens 108-111 32262771-3 2015 The oxidation peak current of DA is linearly proportional to its concentration in the range from 1.5-215.56 muM, with a detection limit of 0.1 muM (at S/N = 3). Dopamine 30-32 latexin Homo sapiens 143-146 32262714-4 2015 The relevant detection limit toward dopamine is 0.3 nM with a wide linear range from 0.001 muM to 200 muM. Dopamine 36-44 latexin Homo sapiens 91-94 32262714-4 2015 The relevant detection limit toward dopamine is 0.3 nM with a wide linear range from 0.001 muM to 200 muM. Dopamine 36-44 latexin Homo sapiens 102-105 26212665-2 2015 High selectivity and sensitivity (detection limit of 0.5 muM, a linear range from 10 muM to 1 mM) have been achieved through the strong interaction between the nonstick PTFE and DA via its oxidative self-polymerization, and the output voltage and current of the developed TENS can reach 116 V and 33 muA, which is exceptionally attractive for the fabrication of self-powered and portable device toward the detection of dopamine. Dopamine 178-180 latexin Homo sapiens 57-60 26212665-2 2015 High selectivity and sensitivity (detection limit of 0.5 muM, a linear range from 10 muM to 1 mM) have been achieved through the strong interaction between the nonstick PTFE and DA via its oxidative self-polymerization, and the output voltage and current of the developed TENS can reach 116 V and 33 muA, which is exceptionally attractive for the fabrication of self-powered and portable device toward the detection of dopamine. Dopamine 178-180 latexin Homo sapiens 85-88 26184200-6 2015 The linear range of the constructed DA sensor was from 1.6 muM to 39.7 muM with a detection limit of 0.1 muM (S/N = 3). Dopamine 36-38 latexin Homo sapiens 59-62 26184200-6 2015 The linear range of the constructed DA sensor was from 1.6 muM to 39.7 muM with a detection limit of 0.1 muM (S/N = 3). Dopamine 36-38 latexin Homo sapiens 71-74 26184200-6 2015 The linear range of the constructed DA sensor was from 1.6 muM to 39.7 muM with a detection limit of 0.1 muM (S/N = 3). Dopamine 36-38 latexin Homo sapiens 71-74 25882962-5 2015 Therewith, the aptamer affinity for dopamine increases one order of magnitude due to electrostatically regulated immobilization, with the aptamer-dopamine dissociation constant of 0.12 +- 0.01 muM versus 1.6 +- 0.17 muM shown in solution. Dopamine 36-44 latexin Homo sapiens 193-196 25882962-5 2015 Therewith, the aptamer affinity for dopamine increases one order of magnitude due to electrostatically regulated immobilization, with the aptamer-dopamine dissociation constant of 0.12 +- 0.01 muM versus 1.6 +- 0.17 muM shown in solution. Dopamine 36-44 latexin Homo sapiens 216-219 25882962-5 2015 Therewith, the aptamer affinity for dopamine increases one order of magnitude due to electrostatically regulated immobilization, with the aptamer-dopamine dissociation constant of 0.12 +- 0.01 muM versus 1.6 +- 0.17 muM shown in solution. Dopamine 146-154 latexin Homo sapiens 193-196 25882962-5 2015 Therewith, the aptamer affinity for dopamine increases one order of magnitude due to electrostatically regulated immobilization, with the aptamer-dopamine dissociation constant of 0.12 +- 0.01 muM versus 1.6 +- 0.17 muM shown in solution. Dopamine 146-154 latexin Homo sapiens 216-219 25882962-6 2015 Under optimal conditions, 0.1-2 muM dopamine was specifically and 85.4 nA muM(-1) cm(-2) sensitively detected, with no interference from structurally related catecholamines. Dopamine 36-44 latexin Homo sapiens 32-35 25882962-7 2015 The results allow improvement of the robustness of dopamine monitoring by aptamer-modified electrodes in biological systems, within the 0.01-1 muM dopamine fluctuation range. Dopamine 51-59 latexin Homo sapiens 143-146 25882962-7 2015 The results allow improvement of the robustness of dopamine monitoring by aptamer-modified electrodes in biological systems, within the 0.01-1 muM dopamine fluctuation range. Dopamine 147-155 latexin Homo sapiens 143-146 25756085-4 2015 For simultaneous detection of AA, DA, and UA, the linear current-concentration responses were observed from 1.0 muM-4.1 mM, 0.05-112.0 muM, and 3.0-186.0 muM, with the detection limits of 0.185, 0.017, and 0.654 muM (S/N = 3), respectively. Dopamine 34-36 latexin Homo sapiens 135-138 25756085-4 2015 For simultaneous detection of AA, DA, and UA, the linear current-concentration responses were observed from 1.0 muM-4.1 mM, 0.05-112.0 muM, and 3.0-186.0 muM, with the detection limits of 0.185, 0.017, and 0.654 muM (S/N = 3), respectively. Dopamine 34-36 latexin Homo sapiens 135-138 25756085-4 2015 For simultaneous detection of AA, DA, and UA, the linear current-concentration responses were observed from 1.0 muM-4.1 mM, 0.05-112.0 muM, and 3.0-186.0 muM, with the detection limits of 0.185, 0.017, and 0.654 muM (S/N = 3), respectively. Dopamine 34-36 latexin Homo sapiens 135-138 25045758-5 2014 The ErGO-based biosensor exhibited a linear response towards dopamine in the concentration range of 0.1-10 muM with a low detection limit of 0.1 muM. Dopamine 61-69 latexin Homo sapiens 145-148 25804204-2 2015 The deposited graphene significantly increased the surface area of working electrode, which led to the nMEA (with diameter of 20 mum) with excellent selectivity and sensitivity to DA. Dopamine 180-182 latexin Homo sapiens 129-132 25576954-6 2015 The lowest detection limits (signal/noise=3) were 25.0, 0.04, and 0.2 muM for AA, DA, and UA, respectively. Dopamine 82-84 latexin Homo sapiens 70-73 28191042-8 2015 Limits of detection for dopamine and NO were 130 nM and 1.8 muM, respectively. Dopamine 24-32 latexin Homo sapiens 60-63 26294980-4 2015 Analyzing DA participation in striatal GABAergic plasticity we observed that high frequency stimulation (HFS) of GABAergic interneurons in the presence of DA at a low concentration (200 nM) favored the expression of inhibitory striatal LTD, whereas higher concentration of DA (20 muM) primarily induced LTP. Dopamine 155-157 latexin Homo sapiens 280-283 25045758-5 2014 The ErGO-based biosensor exhibited a linear response towards dopamine in the concentration range of 0.1-10 muM with a low detection limit of 0.1 muM. Dopamine 61-69 latexin Homo sapiens 107-110 25325840-5 2014 On the basis of large potential separation and high current response, selective and sensitive simultaneous determination of AA, DA, and UA was successfully accomplished by differential pulse voltammetry, displaying a linear response from 50 to 2000 muM, from 0.1 to 50 muM, and from 0.1 to 50 muM with a detection limit (S/N = 3) of 0.78, 0.02, and 0.06 muM. Dopamine 128-130 latexin Homo sapiens 249-252 25029608-5 2014 This novel sensor exhibits fast amperometric response and high sensitivity toward DA with a wide linear concentration range of 10-500 muM and a low detection limit of 0.11 muM (S/N = 3), wherein the interference of UA and AA can be eliminated effectively. Dopamine 82-84 latexin Homo sapiens 134-137 25029608-5 2014 This novel sensor exhibits fast amperometric response and high sensitivity toward DA with a wide linear concentration range of 10-500 muM and a low detection limit of 0.11 muM (S/N = 3), wherein the interference of UA and AA can be eliminated effectively. Dopamine 82-84 latexin Homo sapiens 172-175 25000168-6 2014 Under the optimum conditions, the oxidation peak currents of ACOP and DA were linearly correlated to their concentrations in the ranges of 1-100 and 1-50 muM, respectively. Dopamine 70-72 latexin Homo sapiens 154-157 24441542-5 2014 The voltammetric response of AA was well resolved from the responses of DA and UA, and the oxidation potential of AA was negatively shifted to -0.20 V. The biosensor tolerated a wide linear concentration range for AA, from 1.0 x 10(-5)M to 2.63 x 10(-3)M (R(2)=0.9929), with a detection limit of 0.5 muM (S/N = 3). Dopamine 72-74 latexin Homo sapiens 300-303 24140872-5 2014 By simultaneously changing the concentrations of AA, DA and UA, their oxidation peaks appeared at -0.05 V, 0.16 V and 2.6 V, and the good linear responses ranges were 73.52-2305.53 muM, 1.36-125.69 muM and 3.98-371.49 muM, respectively. Dopamine 53-55 latexin Homo sapiens 181-184 24140872-5 2014 By simultaneously changing the concentrations of AA, DA and UA, their oxidation peaks appeared at -0.05 V, 0.16 V and 2.6 V, and the good linear responses ranges were 73.52-2305.53 muM, 1.36-125.69 muM and 3.98-371.49 muM, respectively. Dopamine 53-55 latexin Homo sapiens 198-201 24140872-5 2014 By simultaneously changing the concentrations of AA, DA and UA, their oxidation peaks appeared at -0.05 V, 0.16 V and 2.6 V, and the good linear responses ranges were 73.52-2305.53 muM, 1.36-125.69 muM and 3.98-371.49 muM, respectively. Dopamine 53-55 latexin Homo sapiens 198-201 24725464-5 2014 In the presence of dopamine (25 muM), the translocation events at--85 pA shifted to--80 pA, which also represents translocation events, because the event time decreases with increasing voltage. Dopamine 19-27 latexin Homo sapiens 32-35 24725464-7 2014 Event profiles for an N-terminal 1-60-residue peptide and a C-terminal 96-140-residue peptide were both altered in the presence of 25 muM dopamine. Dopamine 138-146 latexin Homo sapiens 134-137 25325840-5 2014 On the basis of large potential separation and high current response, selective and sensitive simultaneous determination of AA, DA, and UA was successfully accomplished by differential pulse voltammetry, displaying a linear response from 50 to 2000 muM, from 0.1 to 50 muM, and from 0.1 to 50 muM with a detection limit (S/N = 3) of 0.78, 0.02, and 0.06 muM. Dopamine 128-130 latexin Homo sapiens 269-272 25325840-5 2014 On the basis of large potential separation and high current response, selective and sensitive simultaneous determination of AA, DA, and UA was successfully accomplished by differential pulse voltammetry, displaying a linear response from 50 to 2000 muM, from 0.1 to 50 muM, and from 0.1 to 50 muM with a detection limit (S/N = 3) of 0.78, 0.02, and 0.06 muM. Dopamine 128-130 latexin Homo sapiens 269-272 25325840-5 2014 On the basis of large potential separation and high current response, selective and sensitive simultaneous determination of AA, DA, and UA was successfully accomplished by differential pulse voltammetry, displaying a linear response from 50 to 2000 muM, from 0.1 to 50 muM, and from 0.1 to 50 muM with a detection limit (S/N = 3) of 0.78, 0.02, and 0.06 muM. Dopamine 128-130 latexin Homo sapiens 269-272 24308368-6 2013 Of all five carbon electrode types, freshly cleaved basal plane HOPG showed the clearest signal (distinct from the background) at low concentrations of DA (<10 muM) as a consequence of the low capacitance. Dopamine 152-154 latexin Homo sapiens 163-166 24012662-3 2013 Being employed as an electrochemical sensor for detection of dopamine, the modified electrode exhibits remarkable sensitivity (3.44muA/muM) and lower detection limit (5nM), with linear response in a range of 0.05-20muM. Dopamine 61-69 latexin Homo sapiens 135-138 24245864-6 2013 Optimization of FSCAV yielded a sensitivity of 81 +- 11 nA/muM for dopamine, corresponding to a limit of detection of 3.7 +- 0.5 nM. Dopamine 67-75 latexin Homo sapiens 59-62 23481547-9 2013 Exogenous DA (50 muM) also evoked a glutamate-receptor independent increase in firing and an inward current when D2 receptors were blocked. Dopamine 10-12 latexin Homo sapiens 17-20 24283218-5 2013 Dopamine (20 muM) and SKF 81297 (10 muM) caused inhibition of TTX-R sodium current in small DRG neurons by 23% and 37%, respectively. Dopamine 0-8 latexin Homo sapiens 13-16 23651571-5 2013 By simultaneously changing the concentrations of AA, DA, UA and AC, their electrochemical oxidation peaks appeared at -0.03, 0.15, 0.24 and 0.35 V, and good linear current responses were obtained in the concentration ranges of 6-350, 0.5-50, 1-90 and 0.4-32 muM with the detection limits of 1, 0.1, 0.2 and 0.05 muM (S/N=3), respectively. Dopamine 53-55 latexin Homo sapiens 258-261 23651571-5 2013 By simultaneously changing the concentrations of AA, DA, UA and AC, their electrochemical oxidation peaks appeared at -0.03, 0.15, 0.24 and 0.35 V, and good linear current responses were obtained in the concentration ranges of 6-350, 0.5-50, 1-90 and 0.4-32 muM with the detection limits of 1, 0.1, 0.2 and 0.05 muM (S/N=3), respectively. Dopamine 53-55 latexin Homo sapiens 312-315 23929722-5 2013 METHODS AND RESULTS: Nonivamide (1 muM) stimulated the Ca(2+) -dependent release of serotonin (272 +- 115%) and dopamine (646 +- 48%) in SH-SY5Y cells compared to nontreated cells (100%) to a similar extent as capsaicin. Dopamine 112-120 latexin Homo sapiens 35-38 22720719-6 2012 Specifically, good-quality signal of dopamine and its oxidized form, dopamine o-quinone, was obtained using 10 muL of 1 muM solution of dopamine on the IDA. Dopamine 37-45 latexin Homo sapiens 120-123 23202347-7 2013 The sensitivity of AA, DA and UA at the sensor is 0.012, 4.031, 0.605 muA/muM respectively. Dopamine 23-25 latexin Homo sapiens 74-77 23598203-6 2013 Under optimum conditions, the relative fluorescence intensity was linearly proportional to the concentration of DA within the range from 0.05 to 10 muM, with the detection limit down to 12 nM (n=3). Dopamine 112-114 latexin Homo sapiens 148-151 23020235-4 2012 The determined linear range for DA detection was 0.2-600 muM (N = 3) and the sensitivity was 178 nA/muM cm(2), while the detection limit was 26 nM (S/N = 3). Dopamine 32-34 latexin Homo sapiens 57-60 23020235-4 2012 The determined linear range for DA detection was 0.2-600 muM (N = 3) and the sensitivity was 178 nA/muM cm(2), while the detection limit was 26 nM (S/N = 3). Dopamine 32-34 latexin Homo sapiens 100-103 22590703-5 2012 Moreover, the excellent differential pulse voltammetric (DPV) response toward dopamine, hydroquinone and catechol was obtained and the detection limits was determined to be 0.337, 0.289 and 0.369 muM, respectively. Dopamine 78-86 latexin Homo sapiens 196-199 23397090-7 2013 This configuration achieved a detection limit of 0.454 +- 0.026 muM dopamine at the collector in the presence of 100 muM ascorbic acid in artificial cerebrospinal fluid buffer, concentrations that are consistent with physiological levels. Dopamine 68-76 latexin Homo sapiens 64-67 23397090-7 2013 This configuration achieved a detection limit of 0.454 +- 0.026 muM dopamine at the collector in the presence of 100 muM ascorbic acid in artificial cerebrospinal fluid buffer, concentrations that are consistent with physiological levels. Dopamine 68-76 latexin Homo sapiens 117-120 22512377-10 2012 In primary neonatal rat astrocyte cell cultures, dopamine (200 muM) significantly (P < 0.05) increased numbers of intracellular tachyzoites after 24 hr. Dopamine 49-57 latexin Homo sapiens 63-66 22570010-5 2012 After exposure of synaptosomes to Tat(1-86) (1 muM), DAT immunoreactivity was decreased in plasma membrane enriched fractions (P3) and increased in vesicle-enriched fractions (P4) relative to controls without change in total synaptosomal fractions (P2), suggesting that Tat-induced inhibition of DA uptake is attributable to DAT internalization. Dopamine 53-55 latexin Homo sapiens 47-50 22720719-6 2012 Specifically, good-quality signal of dopamine and its oxidized form, dopamine o-quinone, was obtained using 10 muL of 1 muM solution of dopamine on the IDA. Dopamine 69-77 latexin Homo sapiens 120-123 19225762-11 2009 Administration of alpha-flupenthixol (20 muM) attenuated eIPSC inhibition by dopamine but had no effect on met-enkephalin-induced inhibition. Dopamine 77-85 latexin Homo sapiens 41-44 22391457-7 2012 This, combined with the porosity that enhances the adsorption activity of C(60)-modified electrodes, enable the electrocatalytic analysis of target biomolecules with detection limit as low as 0.1 nM for DA in the presence of AA, and 1 muM for UA in the presence of RSH. Dopamine 203-205 latexin Homo sapiens 235-238 22299833-5 2012 Application of dopamine at a low concentration (1 muM) did not induce an increase in Ca(2+) signal frequency by itself. Dopamine 15-23 latexin Homo sapiens 50-53 22676038-6 2012 GSK-3beta inhibitors completely blocked high-dose (20 muM) DA-induced depressive effects on IPSCs but exhibited limited effects on the facilitating regulation of IPSC in low-dose DA (200 nM). Dopamine 59-61 latexin Homo sapiens 54-57 22224417-4 2012 In vivo, focal application of 100 muM d-kynurenine by reverse microdialysis led to a steady rise in extracellular kynurenic acid in the rat striatum, causing a 4-fold elevation after 2 h. Attesting to functional significance, this increase was accompanied by a 36% reduction in extracellular dopamine. Dopamine 292-300 latexin Homo sapiens 34-37 19195936-4 2009 The electrocatalytic currents increases linearly with the DA and UA concentrations in the ranges of 0.1-900 microM and 20-650 microM, and the detection limits for DA and UA, were 0.087 and 15 muM, respectively. Dopamine 58-60 latexin Homo sapiens 192-195 19074002-4 2009 Bromocriptine (100 nM) and dopamine (100 nM and 10 muM) increased tyrosine phosphorylation during the capacitation period. Dopamine 27-35 latexin Homo sapiens 51-54 19074002-7 2009 We also showed that bromocriptine (100 nM) and low-concentration dopamine (100 nM and 10 muM) increased total and progressive motility of sperm. Dopamine 65-73 latexin Homo sapiens 89-92 19195936-4 2009 The electrocatalytic currents increases linearly with the DA and UA concentrations in the ranges of 0.1-900 microM and 20-650 microM, and the detection limits for DA and UA, were 0.087 and 15 muM, respectively. Dopamine 163-165 latexin Homo sapiens 192-195 18542926-7 2008 However, when used in combination with ADP (10 muM), dopamine in a range of 1 to 100 muM significantly potentiated platelet microaggregate formation and adhesion to collagen under low shear flow conditions. Dopamine 53-61 latexin Homo sapiens 47-50 18542926-7 2008 However, when used in combination with ADP (10 muM), dopamine in a range of 1 to 100 muM significantly potentiated platelet microaggregate formation and adhesion to collagen under low shear flow conditions. Dopamine 53-61 latexin Homo sapiens 85-88