PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32367036-4	2020	Here we identify disulfiram, a drug for treating alcohol addiction, as an inhibitor of pore formation by GSDMD but not other members of the GSDM family.	Alcohols	49-56	gasdermin D	Homo sapiens	105-110	
32524615-7	2020	RESULTS: Alcohol-induced in vivo pyroptosis occurs because of an increase in the levels of pyroptotic proteins such as nucleotide-binding domain (NOD)-like receptor protein 3 (NLRP3), caspase-1, gasdermin-D (GSDMD), and amplified inflammatory response.	Alcohols	9-16	gasdermin D	Homo sapiens	195-206	
32524615-7	2020	RESULTS: Alcohol-induced in vivo pyroptosis occurs because of an increase in the levels of pyroptotic proteins such as nucleotide-binding domain (NOD)-like receptor protein 3 (NLRP3), caspase-1, gasdermin-D (GSDMD), and amplified inflammatory response.	Alcohols	9-16	gasdermin D	Homo sapiens	208-213	
30940212-12	2019	Evidence from DMR and FL analyses indicated that dietary folate and alcohol intake may be associated with genomic regions with tumor suppressor activity such as the GSDMD and HOXA5 genes.	Alcohols	68-75	gasdermin D	Homo sapiens	165-170	
34561552-2	2021	Disulfiram (DSF), an old drug that has been used to treat alcohol addiction for decades, has recently been identified as a potent inhibitor of the gasdermin D (GSDMD)-induced pore formation that causes pyroptosis and inflammatory cytokine release.	Alcohols	58-65	gasdermin D	Homo sapiens	147-158