PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 2561349-2 1989 It was revealed, that under conditions promoting DS (hyperpolarization, muscle warming, rise of calcium concentration as well as treatment by the DS--potentiating agent--proadifen) decrease of the postsynaptic membrane sensitivity to the acetylcholine can develop after few multiquantal end-plate currents--and when acetylcholinesterase is inhibited--during the response to a single quantum of acetylcholine. Proadifen 170-179 acetylcholinesterase (Cartwright blood group) Homo sapiens 316-336 9279814-9 1997 The desensitization of ACh receptors potentiated by proadifen, prevented completely the 6- to 8-fold prolongation of EPC which was induced by neostigmine inhibition of synaptic AChE. Proadifen 52-61 acetylcholinesterase (Cartwright blood group) Homo sapiens 177-181 2600828-2 1989 The amplitudes of end-plate currents (EPCs) in short trains of fifteen to seventeen EPCs at 10 Hz were depressed in the presence of 10 microM-proadifen when acetylcholinesterase (AChE) was inhibited. Proadifen 142-151 acetylcholinesterase (Cartwright blood group) Homo sapiens 157-177 2600828-2 1989 The amplitudes of end-plate currents (EPCs) in short trains of fifteen to seventeen EPCs at 10 Hz were depressed in the presence of 10 microM-proadifen when acetylcholinesterase (AChE) was inhibited. Proadifen 142-151 acetylcholinesterase (Cartwright blood group) Homo sapiens 179-183