PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
2561349-2	1989	It was revealed, that under conditions promoting DS (hyperpolarization, muscle warming, rise of calcium concentration as well as treatment by the DS--potentiating agent--proadifen) decrease of the postsynaptic membrane sensitivity to the acetylcholine can develop after few multiquantal end-plate currents--and when acetylcholinesterase is inhibited--during the response to a single quantum of acetylcholine.	Proadifen	170-179	acetylcholinesterase (Cartwright blood group)	Homo sapiens	316-336	
2600828-2	1989	The amplitudes of end-plate currents (EPCs) in short trains of fifteen to seventeen EPCs at 10 Hz were depressed in the presence of 10 microM-proadifen when acetylcholinesterase (AChE) was inhibited.	Proadifen	142-151	acetylcholinesterase (Cartwright blood group)	Homo sapiens	157-177	
2600828-2	1989	The amplitudes of end-plate currents (EPCs) in short trains of fifteen to seventeen EPCs at 10 Hz were depressed in the presence of 10 microM-proadifen when acetylcholinesterase (AChE) was inhibited.	Proadifen	142-151	acetylcholinesterase (Cartwright blood group)	Homo sapiens	179-183	
9279814-9	1997	The desensitization of ACh receptors potentiated by proadifen, prevented completely the 6- to 8-fold prolongation of EPC which was induced by neostigmine inhibition of synaptic AChE.	Proadifen	52-61	acetylcholinesterase (Cartwright blood group)	Homo sapiens	177-181