PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31064834-10	2019	Our findings support the conclusion that selective suppression of CXCL1/CXCL2 represents an IFN-beta-mediated "training" of the macrophage transcriptional response to TLR2 agonists and that blocking of TLR4 therapeutically with Eritoran after influenza virus infection reverses this suppression by blunting influenza-induced IFN-beta.	eritoran	228-236	interferon beta 1, fibroblast	Mus musculus	92-100