PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28859986-4	2017	While KRIBB11 is an inhibitor of HSF1, we found that KRIBB11 induced Mcl-1 degradation in an HSF1-independent manner.	N2-(1H-indazole-5-yl)-N6-methyl-3-nitropyridine-2,6-diamine	53-60	MCL1 apoptosis regulator, BCL2 family member	Homo sapiens	69-74	
28859986-8	2017	Taken together, our results suggest that targeting Mcl-1 with KRIBB11 treatment, while simultaneously downregulating BIS, could be a therapeutic strategy in NSCLC.	N2-(1H-indazole-5-yl)-N6-methyl-3-nitropyridine-2,6-diamine	62-69	MCL1 apoptosis regulator, BCL2 family member	Homo sapiens	51-56	
26100943-11	2016	Similar to knockdown of BAG3, pharmacological targeting of the BAG3/Mcl-1 pathway with KRIBB11 was capable to sensitize both cell lines to treatment with ABT-737.	N2-(1H-indazole-5-yl)-N6-methyl-3-nitropyridine-2,6-diamine	87-94	MCL1 apoptosis regulator, BCL2 family member	Homo sapiens	68-73