PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28554847-2	2017	Studies with drugs that interfere with serotonin-mediated neurotransmission suggest that the mu-opioid receptor (MOR) synergistically interacts with the 5-HT1A receptor in the dPAG to inhibit escape, a panic-related behavior.	dpag	176-180	opioid receptor mu 1	Homo sapiens	93-111	
32376258-4	2020	This study investigated the involvement and a likely interplay between CB1 and MOR receptors localized into the dPAG on the regulation of fear-like defensive responses and antinociception (evaluated in tail-flick test) evoked by dPAG chemical stimulation with N-methyl-d-aspartate (NMDA).	dpag	112-116	opioid receptor mu 1	Homo sapiens	79-82	
32376258-4	2020	This study investigated the involvement and a likely interplay between CB1 and MOR receptors localized into the dPAG on the regulation of fear-like defensive responses and antinociception (evaluated in tail-flick test) evoked by dPAG chemical stimulation with N-methyl-d-aspartate (NMDA).	dpag	229-233	opioid receptor mu 1	Homo sapiens	79-82	
28797641-7	2017	Therefore, the anti-escape effect of opiorphin in the dPAG seems to be mediated by endogenous bradykinin, acting on kinin B2 receptors, which previous results have shown to interact synergistically with MOR in the dPAG to restrain escape in two animal models of panic.	dpag	214-218	opioid receptor mu 1	Homo sapiens	203-206	
28554847-2	2017	Studies with drugs that interfere with serotonin-mediated neurotransmission suggest that the mu-opioid receptor (MOR) synergistically interacts with the 5-HT1A receptor in the dPAG to inhibit escape, a panic-related behavior.	dpag	176-180	opioid receptor mu 1	Homo sapiens	113-116	
28347824-7	2017	We also evaluated whether activation of the 5-HT1A-R or the mu-opioid receptor (MOR) in the dPAG contributes to the Nor-BNI effects.	dpag	92-96	opioid receptor mu 1	Homo sapiens	60-78	
28347824-7	2017	We also evaluated whether activation of the 5-HT1A-R or the mu-opioid receptor (MOR) in the dPAG contributes to the Nor-BNI effects.	dpag	92-96	opioid receptor mu 1	Homo sapiens	80-83	
26320545-2	2015	Additionally, reported evidence indicates that the mu-opioid receptor (MOR) interacts with the 5-HT1A-R in the dPAG.	dpag	111-115	opioid receptor mu 1	Homo sapiens	51-69	
26320545-2	2015	Additionally, reported evidence indicates that the mu-opioid receptor (MOR) interacts with the 5-HT1A-R in the dPAG.	dpag	111-115	opioid receptor mu 1	Homo sapiens	71-74	
26320545-3	2015	In the present work, pretreatment of the dPAG with the selective MOR blocker CTOP antagonized the anti-escape effect of chronic fluoxetine (10 mg/kg, i.p., daily, for 21 days), as measured in the elevated T-maze (ETM) test, indicating mediation of this effect by the MOR.	dpag	41-45	opioid receptor mu 1	Homo sapiens	65-68	
26320545-3	2015	In the present work, pretreatment of the dPAG with the selective MOR blocker CTOP antagonized the anti-escape effect of chronic fluoxetine (10 mg/kg, i.p., daily, for 21 days), as measured in the elevated T-maze (ETM) test, indicating mediation of this effect by the MOR.	dpag	41-45	opioid receptor mu 1	Homo sapiens	267-270	
26320545-4	2015	In addition, the combined administration of sub-effective doses of the selective MOR agonist DAMGO (intra-dPAG) and sub-effective doses of chronic as well as subchronic (7 days) fluoxetine increased avoidance and escape latencies, suggesting that the activation of MORs may facilitate and accelerate the effects of fluoxetine.	dpag	106-110	opioid receptor mu 1	Homo sapiens	81-84