PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
18625081-13	2008	The lack of any aberrant morphological and metabolite phenotype in the seeds of the arr22 mutants indicates that ARR22 is probably primarily responsible for the fine tuning of specific branches of chalaza-based TCS signalling.	9-ethyl-N-(3,4,5-trimethoxyphenyl)carbazole-3-sulfonamide	211-214	response regulator 22	Arabidopsis thaliana	113-118	
30742656-6	2019	In congruency with the proposed function of ARR22, overexpression of ARR22 blocked the activation of all B-type ARRs in this study in a TCS dependent manner.	9-ethyl-N-(3,4,5-trimethoxyphenyl)carbazole-3-sulfonamide	136-139	response regulator 22	Arabidopsis thaliana	44-49	
30742656-6	2019	In congruency with the proposed function of ARR22, overexpression of ARR22 blocked the activation of all B-type ARRs in this study in a TCS dependent manner.	9-ethyl-N-(3,4,5-trimethoxyphenyl)carbazole-3-sulfonamide	136-139	response regulator 22	Arabidopsis thaliana	69-74	
30742656-9	2019	Thus, our approach can be used to investigate gene signaling networks involving the TCS by leveraging ARR22 as a TCS inhibitor along with B-type ARR overexpression.	9-ethyl-N-(3,4,5-trimethoxyphenyl)carbazole-3-sulfonamide	84-87	response regulator 22	Arabidopsis thaliana	102-107	
30742656-9	2019	Thus, our approach can be used to investigate gene signaling networks involving the TCS by leveraging ARR22 as a TCS inhibitor along with B-type ARR overexpression.	9-ethyl-N-(3,4,5-trimethoxyphenyl)carbazole-3-sulfonamide	113-116	response regulator 22	Arabidopsis thaliana	102-107	
26769709-7	2017	Consequently, we propose that ARR22, by acting as a phosphatase on specific AHPs, disrupts the TCS phosphorelay and prevents B-type ARR phosphorylation, and thus their activation as transcription factors, explaining the observed deactivation of cytokinin-responsive genes.	9-ethyl-N-(3,4,5-trimethoxyphenyl)carbazole-3-sulfonamide	95-98	response regulator 22	Arabidopsis thaliana	30-35