PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
8733762-3	1996	p53 nuclear staining was weakly positive in 21% of the TCs, whereas strong nuclear staining was seen in 64% of the ACs and 88% of the SCLCs (P = 0.0047).	9-ethyl-N-(3,4,5-trimethoxyphenyl)carbazole-3-sulfonamide	55-58	tumor protein p53	Homo sapiens	0-3	
10391558-9	1999	p53 mutations were found in 4 of 16 GCCs (25%) and 8 of 18 TCs (44%).	9-ethyl-N-(3,4,5-trimethoxyphenyl)carbazole-3-sulfonamide	59-62	tumor protein p53	Homo sapiens	0-3	
10391558-11	1999	CONCLUSIONS: p53 mutations appear to play a role in the pathogenesis of some GCCs and in approximately 50% of TCs of the appendix, whereas mutations in the K-ras oncogene do not appear to be important in the development of these tumors.	9-ethyl-N-(3,4,5-trimethoxyphenyl)carbazole-3-sulfonamide	110-113	tumor protein p53	Homo sapiens	13-16	
29707344-7	2018	In the P53 (+) group, the rate of patients with PD-L1 (+) in TCs was significantly higher than in the P53 (-) group (85.3% vs. 45.5%, P=0.001).	9-ethyl-N-(3,4,5-trimethoxyphenyl)carbazole-3-sulfonamide	61-64	tumor protein p53	Homo sapiens	7-10