PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32516834-4	2020	RESULTS: Densities of CD3+ and CD8+ TILs and PD-L1 expressions on TCs and ICs were significantly higher in p16+/HPV-mediated OPSCC.	9-ethyl-N-(3,4,5-trimethoxyphenyl)carbazole-3-sulfonamide	66-69	cyclin dependent kinase inhibitor 2A	Homo sapiens	107-110	
32928147-15	2020	The relative expression of CDKN2A/p16INK4a in T cells was increased in TCS (mean 1.54; 95% CI of the mean: 1.074-2.005; p = 0.048).	9-ethyl-N-(3,4,5-trimethoxyphenyl)carbazole-3-sulfonamide	71-74	cyclin dependent kinase inhibitor 2A	Homo sapiens	34-42	
32928147-15	2020	The relative expression of CDKN2A/p16INK4a in T cells was increased in TCS (mean 1.54; 95% CI of the mean: 1.074-2.005; p = 0.048).	9-ethyl-N-(3,4,5-trimethoxyphenyl)carbazole-3-sulfonamide	71-74	cyclin dependent kinase inhibitor 2A	Homo sapiens	27-33	
32928147-16	2020	CONCLUSION: In this exploratory study, TCS showed increased expression of CDKN2A/p16INK4a and a lymphocyte phenotype that has been associated with immunosenescence.	9-ethyl-N-(3,4,5-trimethoxyphenyl)carbazole-3-sulfonamide	39-42	cyclin dependent kinase inhibitor 2A	Homo sapiens	74-80	
32928147-16	2020	CONCLUSION: In this exploratory study, TCS showed increased expression of CDKN2A/p16INK4a and a lymphocyte phenotype that has been associated with immunosenescence.	9-ethyl-N-(3,4,5-trimethoxyphenyl)carbazole-3-sulfonamide	39-42	cyclin dependent kinase inhibitor 2A	Homo sapiens	81-89	
12612881-5	2003	P16 was expressed in 91% of TCs, 77% of ACs, 78% of LCNECs, and 93% of SCLCs.	9-ethyl-N-(3,4,5-trimethoxyphenyl)carbazole-3-sulfonamide	28-31	cyclin dependent kinase inhibitor 2A	Homo sapiens	0-3	
12612881-8	2003	Overall, the Rb pathway (Rb/P16(INK4)/cyclin D1) was altered more frequently in ACs than in TCs (P = 0.001) and more frequently in LCNECs than in ACs (P = 0.001).	9-ethyl-N-(3,4,5-trimethoxyphenyl)carbazole-3-sulfonamide	92-95	cyclin dependent kinase inhibitor 2A	Homo sapiens	28-31