PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22147260-2	2012	We hypothesized that polymorphisms in oxidative stress (OS)-related genes, including estrogen-quinone metabolizing enzymes NQO2 and GSTM1-5, may influence disease progression and treatment response.	quinone	94-101	glutathione S-transferase mu 1	Homo sapiens	132-139	
21914835-8	2011	It is of interest that one of the reactive metabolites with a quinone structure was predominantly conjugated with GSH by GSTM1.	quinone	62-69	glutathione S-transferase mu 1	Homo sapiens	121-126	
21914835-9	2011	Thus, we demonstrated that the GST isoforms contributed differently to the GSH conjugation of individual reactive metabolites of troglitazone, and GSTM1 is the most important GST isoform in the GSH conjugation of a specific reactive metabolite produced from the cytotoxic, quinone-form metabolite of troglitazone.	quinone	273-280	glutathione S-transferase mu 1	Homo sapiens	147-152	
15607759-10	2004	Specific glutathione-conjugates of the estrogen quinone also potently inhibited hGSTM1-1 and hGSTA1-1.	quinone	48-55	glutathione S-transferase mu 1	Homo sapiens	80-88