PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32938730-8	2020	However, blocking mTOR, PPARgamma, SREBP1, or p38-MAPK signals with specific inhibitors could significantly relieve the enhanced fatty acid catabolism and mitochondrial capacity induced by ST-4.	Fatty Acids	129-139	mitogen-activated protein kinase 14	Mus musculus	46-54	
26040473-9	2015	In vivo administration of FGF21, but not FGF21 plus ERK1/2 inhibitor, to diabetic or fatty-acid-infused mice significantly prevented cardiac apoptosis and reduced inactivation of ERK1/2, p38 MAPK and AMPK and prevented cardiac remodelling and dysfunction.	Fatty Acids	85-95	mitogen-activated protein kinase 14	Mus musculus	187-195	
32463484-9	2020	Mechanistically, weak p38alpha activation in fatty liver upregulated the genes involved in fatty acid beta-oxidation via PPARalpha phosphorylation, thereby reducing steatosis.	Fatty Acids	91-101	mitogen-activated protein kinase 14	Mus musculus	22-30	
29550588-11	2018	CONCLUSION: Overall, these findings indicate that BMS309403 reduces fatty acid-induced ER stress-associated inflammation in skeletal muscle by reducing p38 MAPK activation.	Fatty Acids	68-78	mitogen-activated protein kinase 14	Mus musculus	152-155	
34675006-3	2021	Here, we demonstrate that hepatic p38 activation by MKK6 overexpression in the liver of mice induces severe liver steatosis, reduces fat mass, and elevates circulating fatty acid levels in a hepatic p38alpha- and FGF21-dependent manner.	Fatty Acids	168-178	mitogen-activated protein kinase 14	Mus musculus	34-37	
17416903-12	2007	These data indicate that obesity and saturated fatty acids decrease PGC-1 and mitochondrial gene expression and function via p38 MAPK-dependent transcriptional pathways.	Fatty Acids	37-58	mitogen-activated protein kinase 14	Mus musculus	125-133	
34917917-6	2021	This study has shown that mechanistically, via the elevation of hepatic FGF21, p38alpha activation increases the influx of fatty acids from the adipose tissue to liver, resulting in hepatic ectopic lipid accumulation and insulin resistance.	Fatty Acids	123-134	mitogen-activated protein kinase 14	Mus musculus	79-87	
34675006-3	2021	Here, we demonstrate that hepatic p38 activation by MKK6 overexpression in the liver of mice induces severe liver steatosis, reduces fat mass, and elevates circulating fatty acid levels in a hepatic p38alpha- and FGF21-dependent manner.	Fatty Acids	168-178	mitogen-activated protein kinase 14	Mus musculus	199-207	
34957482-3	2021	Here, we demonstrate that hepatic p38 activation by MKK6 overexpression in the liver of mice induces severe liver steatosis, reduces fat mass, and elevates circulating fatty acid levels in a hepatic p38alpha- and FGF21-dependent manner.	Fatty Acids	168-178	mitogen-activated protein kinase 14	Mus musculus	34-37	
34957482-3	2021	Here, we demonstrate that hepatic p38 activation by MKK6 overexpression in the liver of mice induces severe liver steatosis, reduces fat mass, and elevates circulating fatty acid levels in a hepatic p38alpha- and FGF21-dependent manner.	Fatty Acids	168-178	mitogen-activated protein kinase 14	Mus musculus	199-207	
34675006-4	2021	Mechanistically, through increasing the FGF21 production from liver, hepatic p38 activation increases the influx of fatty acids from adipose tissue to liver, leading to hepatic ectopic lipid accumulation and insulin resistance.	Fatty Acids	116-127	mitogen-activated protein kinase 14	Mus musculus	77-80	
34957482-4	2021	Mechanistically, through increasing the FGF21 production from liver, hepatic p38 activation increases the influx of fatty acids from adipose tissue to liver, leading to hepatic ectopic lipid accumulation and insulin resistance.	Fatty Acids	116-127	mitogen-activated protein kinase 14	Mus musculus	77-80