PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24632207-6	2014	In addition, the activation of NR5A2 produced a reduction in the oxidation of fatty acids, an effect which disappeared in low-glucose conditions.	Fatty Acids	78-89	nuclear receptor subfamily 5, group A, member 2	Mus musculus	31-36	
28552526-7	2017	These results suggest that Sod2 is a target gene of LRH-1, and that LRH-1 agonists can mediate a reduction in ROS production and oxidative stress driven by an excess of fatty acids, as exhibited in nonalcoholic fatty liver disease.	Fatty Acids	169-180	nuclear receptor subfamily 5, group A, member 2	Mus musculus	68-73	
22772466-2	2012	Although LRH-1 activation may protect the liver against diet-induced steatosis and insulin resistance, little is known about how LRH-1 controls hepatic glucose and fatty acid metabolism under physiological conditions.	Fatty Acids	164-174	nuclear receptor subfamily 5, group A, member 2	Mus musculus	129-134	
17265163-4	2007	Nuclear LRH-1 immunoreactivity was also localized in cells involved in fatty acid/glucose metabolism, including hepatocytes, brown adipocytes, and cardiomyocytes, and neurons involved in the regulation of food intake, including the arcuate nucleus in the hypothalamus and paraventricular nucleus of thalamus.	Fatty Acids	71-81	nuclear receptor subfamily 5, group A, member 2	Mus musculus	8-13