PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32487544-0	2020	Angiopoietin-like protein 8 differentially regulates ANGPTL3 and ANGPTL4 during postprandial partitioning of fatty acids.	Fatty Acids	109-120	angiopoietin like 3	Homo sapiens	53-60	
32022755-1	2020	PURPOSE OF REVIEW: Angiopoietin-like protein-3 (ANGPTL3) is emerging as a key player in lipoprotein transport with an expanding role on fatty acid and glucose metabolism.	Fatty Acids	136-146	angiopoietin like 3	Homo sapiens	19-46	
32022755-1	2020	PURPOSE OF REVIEW: Angiopoietin-like protein-3 (ANGPTL3) is emerging as a key player in lipoprotein transport with an expanding role on fatty acid and glucose metabolism.	Fatty Acids	136-146	angiopoietin like 3	Homo sapiens	48-55	
32022755-5	2020	Individuals with familial combined hypolipidemia due to homozygous loss-of-function mutations in ANGPTL3 gene showed improved metabolism of triglyceride-rich lipoproteins during fasting and postprandial state and increased fatty acid oxidation and insulin sensitivity.	Fatty Acids	223-233	angiopoietin like 3	Homo sapiens	97-104	
27040449-9	2016	Our findings demonstrate that complete ANGPTL3 deficiency associates with highly reduced postprandial lipemia probably due to faster catabolism of intestinally derived lipoproteins, larger expansion of the postprandial FFA pool, and decreased influx of dietary-derived fatty acids into the liver.	Fatty Acids	269-280	angiopoietin like 3	Homo sapiens	39-46	
32151767-5	2020	Accordingly, ANGPTL3 depleted IHH displayed changes in cellular overall fatty acid (FA) composition and in the lipid species composition of several lipid classes, characterized by abundant n-6 and n-3 polyunsaturated FAs (PUFAs).	Fatty Acids	72-82	angiopoietin like 3	Homo sapiens	13-20	
23839332-5	2013	SUMMARY: The identification of loss-of-function ANGPTL3 mutation is shedding light on a possible role of ANGPTL3 at the crossroads of lipoproteins, fatty acids, and glucose metabolism, thus making ANGPTL3 an attractive protein to target the cardio-metabolic risk.	Fatty Acids	148-159	angiopoietin like 3	Homo sapiens	48-55	
23839332-5	2013	SUMMARY: The identification of loss-of-function ANGPTL3 mutation is shedding light on a possible role of ANGPTL3 at the crossroads of lipoproteins, fatty acids, and glucose metabolism, thus making ANGPTL3 an attractive protein to target the cardio-metabolic risk.	Fatty Acids	148-159	angiopoietin like 3	Homo sapiens	105-112	
23839332-5	2013	SUMMARY: The identification of loss-of-function ANGPTL3 mutation is shedding light on a possible role of ANGPTL3 at the crossroads of lipoproteins, fatty acids, and glucose metabolism, thus making ANGPTL3 an attractive protein to target the cardio-metabolic risk.	Fatty Acids	148-159	angiopoietin like 3	Homo sapiens	105-112