PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32901839-7	2020	KEGG pathway analysis revealed that the lncRNAs were closely associated with fatty acid metabolism, apoptosis and the TGF-beta signaling pathway.	Fatty Acids	77-87	transforming growth factor alpha	Homo sapiens	118-126	
5480854-1	1970	Transport of plasma-free fatty acids (FFA) and of fatty acids in triglycerides of plasma very low density lipoproteins (VLDL-TGFA) was studied in two normal subjects, five patients with type IV hyperlipoproteinemia, and two patients with type I hyperlipoproteinemia.	Fatty Acids	50-61	transforming growth factor alpha	Homo sapiens	120-129	
33424924-7	2020	Gene Ontology (GO) and Kyoto Encyclopedia of Genes and Genomes (KEGG) pathway enrichment analysis of the host genes of the DE circRNAs showed that the host genes were enriched in lipid metabolism related GO terms (e.g., fatty acid beta-oxidation using acyl-CoA dehydrogenase and MLL3/4 complex), and signaling pathways (e.g., TGF-beta and lysine degradation signaling pathway).	Fatty Acids	220-230	transforming growth factor alpha	Homo sapiens	326-334	
32194897-0	2020	Fatty acid extracts facilitate cutaneous wound healing through activating AKT, ERK, and TGF-beta/Smad3 signaling and promoting angiogenesis.	Fatty Acids	0-10	transforming growth factor alpha	Homo sapiens	88-96	
32194897-8	2020	Our findings suggest that FAs can promote cutaneous wound healing by inducing angiogenesis, partly by activating AKT, ERK, and TGF-beta/Smad3 signaling.	Fatty Acids	26-29	transforming growth factor alpha	Homo sapiens	127-135	
20628023-1	2010	Cellular effects of FFA might differ from those of lipoprotein triglyceride (TG)-derived fatty acids (TGFA).	Fatty Acids	89-100	transforming growth factor alpha	Homo sapiens	102-106	
35517820-3	2022	PPARalpha, AMPK, sirtuins, HIF-1, and TGF-beta/SMAD3 activation have all been shown to play key roles in the regulation of fatty acid beta-oxidation in kidney diseases, and restoration of fatty acid beta-oxidation by modulation of these molecules can ameliorate the development of such diseases.	Fatty Acids	123-133	transforming growth factor alpha	Homo sapiens	38-46	
35517820-3	2022	PPARalpha, AMPK, sirtuins, HIF-1, and TGF-beta/SMAD3 activation have all been shown to play key roles in the regulation of fatty acid beta-oxidation in kidney diseases, and restoration of fatty acid beta-oxidation by modulation of these molecules can ameliorate the development of such diseases.	Fatty Acids	188-198	transforming growth factor alpha	Homo sapiens	38-46	
22343749-7	2012	Recombinant phosphatidylserine-specific phospholipase A(1) (PS-PLA(1)) that deacylates fatty acid at the sn-1 position of PS and produces 2-acyl-LysoPS, but not catalytically inactive mutant PS-PLA(1), stimulated the release of AP-TGFalpha from GPR34-expressing cells.	Fatty Acids	87-97	transforming growth factor alpha	Homo sapiens	231-239	
8910478-9	1996	The palmitoylation of these cysteines suggests a possibly dynamic role of fatty acid modification in the integrity and function of the transmembrane TGF-alpha complex.	Fatty Acids	74-84	transforming growth factor alpha	Homo sapiens	149-158	
35544303-4	2022	Here, we demonstrate that transforming growth factor beta (TGF-beta)-induced EMT is accompanied by decreased fatty acid oxidation (FAO) and reduced acetyl-coenzyme A (acetyl-CoA) levels.	Fatty Acids	109-119	transforming growth factor alpha	Homo sapiens	59-67	
35590477-5	2022	Using APQ to quantify the time-resolved proteomic profiles during a TGF-beta-induced epithelial-mesenchymal transition, we found many differentially expressed proteins associated with fatty acid degradation, indicating that fatty acid metabolism reprogramming occurred during the process.	Fatty Acids	184-194	transforming growth factor alpha	Homo sapiens	68-76	
35590477-5	2022	Using APQ to quantify the time-resolved proteomic profiles during a TGF-beta-induced epithelial-mesenchymal transition, we found many differentially expressed proteins associated with fatty acid degradation, indicating that fatty acid metabolism reprogramming occurred during the process.	Fatty Acids	224-234	transforming growth factor alpha	Homo sapiens	68-76