PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31936679-11	2020	To further substantiate the mechanism of cell death mediated by endoplasmic reticulum stress (ERS), we determined the expression of the inositol-requiring enzyme (IRE1), (PKR-like ER kinase) PERK, activating transcription factor 6 (ATF6), and activating transcription factor 4 ATF4, the apoptotic markers p53, Bax, and caspase 3, and the anti-apoptotic marker Bcl-2.	Inositol	136-144	activating transcription factor 4	Homo sapiens	243-276	
31936679-11	2020	To further substantiate the mechanism of cell death mediated by endoplasmic reticulum stress (ERS), we determined the expression of the inositol-requiring enzyme (IRE1), (PKR-like ER kinase) PERK, activating transcription factor 6 (ATF6), and activating transcription factor 4 ATF4, the apoptotic markers p53, Bax, and caspase 3, and the anti-apoptotic marker Bcl-2.	Inositol	136-144	activating transcription factor 4	Homo sapiens	277-281	
34368469-8	2021	Inositol requiring enzyme-1 alpha (IRE1alpha) was not activated; but hypoxia induced eif2alpha phosphorylation, increasing expression of ATF4 (activating transcription factor-4).	Inositol	0-8	activating transcription factor 4	Homo sapiens	137-141