PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29518282-11	2018	INTERPRETATION: mI/Cr has significant temporal and spatial associations with Abeta and could potentially be considered as a disease state biomarker.	Inositol	16-18	amyloid beta precursor protein	Homo sapiens	77-82	
10764800-9	2000	The attenuation of toxicity is the result of Abeta-inositol interaction, as inositol uptake inhibitors had no effect on neuronal survival.	Inositol	51-59	amyloid beta precursor protein	Homo sapiens	45-50	
10764800-11	2000	Inositol stereoisomers are naturally occurring molecules that readily cross the blood-brain barrier and may represent a viable treatment for AD through the complexation of Abeta and attenuation of Abeta neurotoxic effects.	Inositol	0-8	amyloid beta precursor protein	Homo sapiens	172-177	
32418522-9	2020	Inositol stereoisomer"s namely, scylloinositol and Myo-inositol, have also been seen to inhibit Abeta production and plaque accumulation in the brain, inhibiting AD pathogenesis.	Inositol	0-8	amyloid beta precursor protein	Homo sapiens	96-101	
32418522-9	2020	Inositol stereoisomer"s namely, scylloinositol and Myo-inositol, have also been seen to inhibit Abeta production and plaque accumulation in the brain, inhibiting AD pathogenesis.	Inositol	51-63	amyloid beta precursor protein	Homo sapiens	96-101	
29518282-7	2018	RESULTS: Abeta + regions had significantly higher (p = 0.02) mI/Cr and lower tNA/Cr (p = 0.02), whereas in hypometabolic areas only tNA/Cr was reduced (p = 0.003).	Inositol	61-63	amyloid beta precursor protein	Homo sapiens	9-14	
29310864-5	2018	Abeta (beta = 0.45, p = 0.018) and white matter hyperintensities (beta = 0.40, p = 0.046) were independently and interactively (beta = -0.49, p = 0.026) associated with a higher ratio of mI over NAA (mI/NAA) in the posterior cingulate and precuneus gray matter but not in the white matter.	Inositol	187-189	amyloid beta precursor protein	Homo sapiens	0-5	
29310864-5	2018	Abeta (beta = 0.45, p = 0.018) and white matter hyperintensities (beta = 0.40, p = 0.046) were independently and interactively (beta = -0.49, p = 0.026) associated with a higher ratio of mI over NAA (mI/NAA) in the posterior cingulate and precuneus gray matter but not in the white matter.	Inositol	200-202	amyloid beta precursor protein	Homo sapiens	0-5	
25471565-9	2014	Higher amyloid-beta burden was associated with elevated mI/Cr and lower NAA/mI ratios, but not with NAA/Cr.	Inositol	56-58	amyloid beta precursor protein	Homo sapiens	7-19	
25471565-9	2014	Higher amyloid-beta burden was associated with elevated mI/Cr and lower NAA/mI ratios, but not with NAA/Cr.	Inositol	76-78	amyloid beta precursor protein	Homo sapiens	7-19	
23627280-0	2013	Binding mechanism of inositol stereoisomers to monomers and aggregates of Abeta(16-22).	Inositol	21-29	amyloid beta precursor protein	Homo sapiens	74-79	
23065792-6	2012	Moreover, inhibition of amyloid-beta peptide oligomerization and fibrillization via post-weaning administration of scyllo-inositol, a naturally occurring stereoisomer of myo-inositol, rescued both structural and functional impairment of the cortical microvasculature in this Alzheimer"s disease model.	Inositol	170-182	amyloid beta precursor protein	Homo sapiens	24-36