PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
8858023-8	1996	RESULTS: Treatment with verapamil, TMB-8, U73122, or W7 markedly inhibited TNF release by LPSa, but had little effect on IL-1 release.	8-(N,N-diethylamino)octyl-3,4,5-trimethoxybenzoate	35-40	tumor necrosis factor	Mus musculus	75-78	
9010769-3	1996	In concordance with this, production of TNF-alpha was inhibited by EGTA and/or TMB-8.	8-(N,N-diethylamino)octyl-3,4,5-trimethoxybenzoate	79-84	tumor necrosis factor	Mus musculus	40-49	
7705961-3	1994	Supernatants collected from macrophages treated with cisplatin and EGTA, nifedipine, TMB-8 or W-7 demonstrated decreased tumor necrosis factor (TNF) and interleukin-1 (IL-1) activity in comparison to supernatants collected from macrophages treated with cisplatin alone.	8-(N,N-diethylamino)octyl-3,4,5-trimethoxybenzoate	85-90	tumor necrosis factor	Mus musculus	121-142	
7705961-3	1994	Supernatants collected from macrophages treated with cisplatin and EGTA, nifedipine, TMB-8 or W-7 demonstrated decreased tumor necrosis factor (TNF) and interleukin-1 (IL-1) activity in comparison to supernatants collected from macrophages treated with cisplatin alone.	8-(N,N-diethylamino)octyl-3,4,5-trimethoxybenzoate	85-90	tumor necrosis factor	Mus musculus	144-147	
1906437-5	1991	Both the calcium chelator, EGTA, and the intracellular calcium antagonist, TMB-8, inhibited TNF production stimulated by all agents, indicating that TNF stimulation by all agents was calcium dependent.	8-(N,N-diethylamino)octyl-3,4,5-trimethoxybenzoate	75-80	tumor necrosis factor	Mus musculus	92-95	
1906437-5	1991	Both the calcium chelator, EGTA, and the intracellular calcium antagonist, TMB-8, inhibited TNF production stimulated by all agents, indicating that TNF stimulation by all agents was calcium dependent.	8-(N,N-diethylamino)octyl-3,4,5-trimethoxybenzoate	75-80	tumor necrosis factor	Mus musculus	149-152