PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10607731-13	2000	Higher levels of BPDE-DNA adducts in individuals with the combined CYP1A1(1/*2 or *2A/*2A)-GSTM1*0/*0 genotype suggest that these genotype combinations are at increased risk for contracting lung cancer when exposed to PAH.	7,8-Dihydro-7,8-dihydroxybenzo(a)pyrene 9,10-oxide	17-21	phenylalanine hydroxylase	Homo sapiens	218-221	
18489080-2	2008	P4501A1/P4501B1 plus epoxide hydrolase activate PAH to (+/-)- anti-benzo[ a]pyrene diol epoxide ((+/-)- anti-BPDE), which causes bulky DNA adducts.	7,8-Dihydro-7,8-dihydroxybenzo(a)pyrene 9,10-oxide	109-113	phenylalanine hydroxylase	Homo sapiens	48-51	
10491778-0	1999	Influence of GSTM1 genotypes on anti-BPDE-DNA adduct levels in mononuclear white blood cells of humans exposed to PAH.	7,8-Dihydro-7,8-dihydroxybenzo(a)pyrene 9,10-oxide	37-41	phenylalanine hydroxylase	Homo sapiens	114-117	
10491778-9	1999	In coke-oven workers, chimney sweeps, and aluminum workers, respectively, the multiplicative effect of the null genotype with occupational PAH exposure gives risks of 162 (= 27.2 x 5.94), 10 (= 1.70 x 5.94), and 3 (= 0.50 x 5.94) times higher probability (risk) of high BPDE-DNA adduct formation than that of non-exposed subjects with the active GSTM1 genotype.	7,8-Dihydro-7,8-dihydroxybenzo(a)pyrene 9,10-oxide	270-274	phenylalanine hydroxylase	Homo sapiens	139-142	
10491778-10	1999	CONCLUSION: Our results indicate a greater risk of anti-BPDE-DNA adduct formation resulting from occupational high-level PAH-exposure in GSTM1 null (GSTM1 *0/*0) workers.	7,8-Dihydro-7,8-dihydroxybenzo(a)pyrene 9,10-oxide	56-60	phenylalanine hydroxylase	Homo sapiens	121-124