PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
7586135-12	1995	A comparison with previous in vivo data obtained by the same assays in T-lymphocytes of styrene-exposed workers suggests that chronic, low dose exposure to styrene in the work environment may be more efficient in inducing persistent DNA adducts and HPRT mutation than acute, short-term exposure.	Styrene	88-95	hypoxanthine phosphoribosyltransferase 1	Homo sapiens	249-253	
8597072-2	1995	Styrene exposed lamination workers in Bohemia showed a significantly higher frequency of hprt mutant cells than Swedish control populations studied simultaneously.	Styrene	0-7	hypoxanthine phosphoribosyltransferase 1	Homo sapiens	89-93	
11535253-5	2001	Moderately higher HPRT mutant frequencies were detected in styrene-exposed individuals (20.2 +/- 25.8 x 10(-6)) as compared to controls (13.3 +/- 6.3 x 10(-6)), but this difference was not significant.	Styrene	59-66	hypoxanthine phosphoribosyltransferase 1	Homo sapiens	18-22	
11535253-6	2001	ANOVA (in the whole set of data) revealed that mutant frequencies at the HPRT gene were significantly associated with years of employment (F=6.9, P=0.0001), styrene in blood (F=10.1, P=0.0001), and heterozygosity in CYP2E1 (intron 6; F=13.5, P=0.0008) and GSTP1 (exon 5; F=3.6, P=0.038).	Styrene	157-164	hypoxanthine phosphoribosyltransferase 1	Homo sapiens	73-77	
7586135-12	1995	A comparison with previous in vivo data obtained by the same assays in T-lymphocytes of styrene-exposed workers suggests that chronic, low dose exposure to styrene in the work environment may be more efficient in inducing persistent DNA adducts and HPRT mutation than acute, short-term exposure.	Styrene	156-163	hypoxanthine phosphoribosyltransferase 1	Homo sapiens	249-253