PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25009983-8	2014	When Pten was overexpressed, proliferation of DCMECs and concentrations for beta-casein, triglyceride, and lactose were significantly decreased.	Triglycerides	89-101	phosphatase and tensin homolog	Bos taurus	5-9	
32952018-6	2020	Furthermore, PTEN knockdown decreased mRNA and protein abundance of VLDL assembly-related molecules, including apolipoprotein B100, apolipoprotein E, microsomal triglyceride transfer protein, and low density lipoprotein receptor.	Triglycerides	161-173	phosphatase and tensin homolog	Bos taurus	13-17	
32952018-7	2020	Importantly, PTEN knockdown promoted triglyceride accumulation in hepatocytes and reduced the VLDL content in culture medium.	Triglycerides	37-49	phosphatase and tensin homolog	Bos taurus	13-17	
32952018-10	2020	Importantly, PTEN knockdown heightened the increase in triglyceride accumulation of hepatocytes and the decrease in VLDL content in culture medium induced by FFA.	Triglycerides	55-67	phosphatase and tensin homolog	Bos taurus	13-17	
32952018-11	2020	Overall, these in vitro data indicate that FFA inhibits PTEN expression, leading to triglyceride accumulation and the inhibition of VLDL assembly in calf hepatocytes.	Triglycerides	84-96	phosphatase and tensin homolog	Bos taurus	56-60