PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 9886834-4 1999 In CHO/rbGHR and CHO/hGHR cells, permeable sulfhydryl-reactive agents, like N-ethylmaleimide (NEM) and iodacetamide (IA), inhibited GHR internalization and induced an immediate dose-dependent loss of cellular GHR, associated with a concomitant marked increase in released GHBP. Ethylmaleimide 76-92 growth hormone receptor Homo sapiens 21-25 9886834-4 1999 In CHO/rbGHR and CHO/hGHR cells, permeable sulfhydryl-reactive agents, like N-ethylmaleimide (NEM) and iodacetamide (IA), inhibited GHR internalization and induced an immediate dose-dependent loss of cellular GHR, associated with a concomitant marked increase in released GHBP. Ethylmaleimide 76-92 growth hormone receptor Homo sapiens 272-276 9886834-4 1999 In CHO/rbGHR and CHO/hGHR cells, permeable sulfhydryl-reactive agents, like N-ethylmaleimide (NEM) and iodacetamide (IA), inhibited GHR internalization and induced an immediate dose-dependent loss of cellular GHR, associated with a concomitant marked increase in released GHBP. Ethylmaleimide 94-97 growth hormone receptor Homo sapiens 21-25 9886834-4 1999 In CHO/rbGHR and CHO/hGHR cells, permeable sulfhydryl-reactive agents, like N-ethylmaleimide (NEM) and iodacetamide (IA), inhibited GHR internalization and induced an immediate dose-dependent loss of cellular GHR, associated with a concomitant marked increase in released GHBP. Ethylmaleimide 94-97 growth hormone receptor Homo sapiens 272-276 8756574-5 1996 N-Ethylmaleimide (500 microM for 30 min at 30 C) markedly increased GHBP secretion, matched by a corresponding decrease in GHR. Ethylmaleimide 0-16 growth hormone receptor Homo sapiens 68-72 9528979-9 1998 Treatment with N-ethylmaleimide (NEM; 5 mM), an agent known to cause GHBP shedding from IM-9 cells, promoted a similar rapid loss of full-length GHRs and an accumulation of GHR cytoplasmic domain remnant proteins. Ethylmaleimide 33-36 growth hormone receptor Homo sapiens 145-148 9528979-9 1998 Treatment with N-ethylmaleimide (NEM; 5 mM), an agent known to cause GHBP shedding from IM-9 cells, promoted a similar rapid loss of full-length GHRs and an accumulation of GHR cytoplasmic domain remnant proteins. Ethylmaleimide 15-31 growth hormone receptor Homo sapiens 69-73 9528979-9 1998 Treatment with N-ethylmaleimide (NEM; 5 mM), an agent known to cause GHBP shedding from IM-9 cells, promoted a similar rapid loss of full-length GHRs and an accumulation of GHR cytoplasmic domain remnant proteins. Ethylmaleimide 15-31 growth hormone receptor Homo sapiens 145-148 9528979-9 1998 Treatment with N-ethylmaleimide (NEM; 5 mM), an agent known to cause GHBP shedding from IM-9 cells, promoted a similar rapid loss of full-length GHRs and an accumulation of GHR cytoplasmic domain remnant proteins. Ethylmaleimide 33-36 growth hormone receptor Homo sapiens 69-73